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Predicting amyloid status in corticobasal syndrome using modified clinical criteria, magnetic resonance imaging and fluorodeoxyglucose positron emission tomography.

Alzheimer's research & therapy

Sha SJ, Ghosh PM, Lee SE, Corbetta-Rastelli C, Jagust WJ, Kornak J, Rankin KP, Grinberg LT, Vinters HV, Mendez MF, Dickson DW, Seeley WW, Gorno-Tempini M, Kramer J, Miller BL, Boxer AL, Rabinovici GD.
PMID: 25733984
Alzheimers Res Ther. 2015 Mar 02;7(1):8. doi: 10.1186/s13195-014-0093-y. eCollection 2015.

INTRODUCTION: Group comparisons demonstrate greater visuospatial and memory deficits and temporoparietal-predominant degeneration on neuroimaging in patients with corticobasal syndrome (CBS) found to have Alzheimer's disease (AD) pathology versus those with underlying frontotemporal lobar degeneration (FTLD). The value of these...

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Sha SJ, Ghosh PM, Lee SE, et al. Predicting amyloid status in corticobasal syndrome using modified clinical criteria, magnetic resonance imaging and fluorodeoxyglucose positron emission tomography. Alzheimers Res Ther. 2015;7(1):8doi: 10.1186/s13195-014-0093-y.
Sha, S. J., Ghosh, P. M., Lee, S. E., Corbetta-Rastelli, C., Jagust, W. J., Kornak, J., Rankin, K. P., Grinberg, L. T., Vinters, H. V., Mendez, M. F., Dickson, D. W., Seeley, W. W., Gorno-Tempini, M., Kramer, J., Miller, B. L., Boxer, A. L., & Rabinovici, G. D. (2015). Predicting amyloid status in corticobasal syndrome using modified clinical criteria, magnetic resonance imaging and fluorodeoxyglucose positron emission tomography. Alzheimer's research & therapy, 7(1), 8. https://doi.org/10.1186/s13195-014-0093-y
Sha, Sharon J, et al. "Predicting amyloid status in corticobasal syndrome using modified clinical criteria, magnetic resonance imaging and fluorodeoxyglucose positron emission tomography." Alzheimer's research & therapy vol. 7,1 (2015): 8. doi: https://doi.org/10.1186/s13195-014-0093-y
Sha SJ, Ghosh PM, Lee SE, Corbetta-Rastelli C, Jagust WJ, Kornak J, Rankin KP, Grinberg LT, Vinters HV, Mendez MF, Dickson DW, Seeley WW, Gorno-Tempini M, Kramer J, Miller BL, Boxer AL, Rabinovici GD. Predicting amyloid status in corticobasal syndrome using modified clinical criteria, magnetic resonance imaging and fluorodeoxyglucose positron emission tomography. Alzheimers Res Ther. 2015 Mar 02;7(1):8. doi: 10.1186/s13195-014-0093-y. eCollection 2015. PMID: 25733984; PMCID: PMC4346122.
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The role of apolipoprotein E (APOE) genotype in early mild cognitive impairment (E-MCI).

Frontiers in aging neuroscience

Risacher SL, Kim S, Shen L, Nho K, Foroud T, Green RC, Petersen RC, Jack CR, Aisen PS, Koeppe RA, Jagust WJ, Shaw LM, Trojanowski JQ, Weiner MW, Saykin AJ.
PMID: 23554593
Front Aging Neurosci. 2013 Apr 01;5:11. doi: 10.3389/fnagi.2013.00011. eCollection 2013.

OBJECTIVE: Our goal was to evaluate the association of APOE with amyloid deposition, cerebrospinal fluid levels (CSF) of Aβ, tau, and p-tau, brain atrophy, cognition and cognitive complaints in E-MCI patients and cognitively healthy older adults (HC) in the...

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Risacher SL, Kim S, Shen L, et al. The role of apolipoprotein E (APOE) genotype in early mild cognitive impairment (E-MCI). Front Aging Neurosci. 2013;5:11doi: 10.3389/fnagi.2013.00011.
Risacher, S. L., Kim, S., Shen, L., Nho, K., Foroud, T., Green, R. C., Petersen, R. C., Jack, C. R., Aisen, P. S., Koeppe, R. A., Jagust, W. J., Shaw, L. M., Trojanowski, J. Q., Weiner, M. W., Saykin, A. J. (2013). The role of apolipoprotein E (APOE) genotype in early mild cognitive impairment (E-MCI). Frontiers in aging neuroscience, 511. https://doi.org/10.3389/fnagi.2013.00011
Risacher, Shannon L, et al. "The role of apolipoprotein E (APOE) genotype in early mild cognitive impairment (E-MCI)." Frontiers in aging neuroscience vol. 5 (2013): 11. doi: https://doi.org/10.3389/fnagi.2013.00011
Risacher SL, Kim S, Shen L, Nho K, Foroud T, Green RC, Petersen RC, Jack CR, Aisen PS, Koeppe RA, Jagust WJ, Shaw LM, Trojanowski JQ, Weiner MW, Saykin AJ. The role of apolipoprotein E (APOE) genotype in early mild cognitive impairment (E-MCI). Front Aging Neurosci. 2013 Apr 01;5:11. doi: 10.3389/fnagi.2013.00011. eCollection 2013. PMID: 23554593; PMCID: PMC3612590.
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Blood protein predictors of brain amyloid for enrichment in clinical trials?.

Alzheimer's & dementia (Amsterdam, Netherlands)

Ashton NJ, Kiddle SJ, Graf J, Ward M, Baird AL, Hye A, Westwood S, Wong KV, Dobson RJ, Rabinovici GD, Miller BL, Rosen HJ, Torres A, Zhang Z, Thurfjell L, Covin A, Hehir CT, Baker D, Bazenet C, Lovestone S.
PMID: 27239491
Alzheimers Dement (Amst). 2015 Mar 29;1(1):48-60. doi: 10.1016/j.dadm.2014.11.005. eCollection 2015 Mar.

BACKGROUND: Measures of neocortical amyloid burden (NAB) identify individuals who are at substantially greater risk of developing Alzheimer's disease (AD). Blood-based biomarkers predicting NAB would have great utility for the enrichment of AD clinical trials, including large-scale prevention trials.METHODS:...

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Ashton NJ, Kiddle SJ, Graf J, et al. Blood protein predictors of brain amyloid for enrichment in clinical trials?. Alzheimers Dement (Amst). 2015;1(1):48-60doi: 10.1016/j.dadm.2014.11.005.
Ashton, N. J., Kiddle, S. J., Graf, J., Ward, M., Baird, A. L., Hye, A., Westwood, S., Wong, K. V., Dobson, R. J., Rabinovici, G. D., Miller, B. L., Rosen, H. J., Torres, A., Zhang, Z., Thurfjell, L., Covin, A., Hehir, C. T., Baker, D., Bazenet, C., Lovestone, S. (2015). Blood protein predictors of brain amyloid for enrichment in clinical trials?. Alzheimer's & dementia (Amsterdam, Netherlands), 1(1), 48-60. https://doi.org/10.1016/j.dadm.2014.11.005
Ashton, Nicholas J, et al. "Blood protein predictors of brain amyloid for enrichment in clinical trials?." Alzheimer's & dementia (Amsterdam, Netherlands) vol. 1,1 (2015): 48-60. doi: https://doi.org/10.1016/j.dadm.2014.11.005
Ashton NJ, Kiddle SJ, Graf J, Ward M, Baird AL, Hye A, Westwood S, Wong KV, Dobson RJ, Rabinovici GD, Miller BL, Rosen HJ, Torres A, Zhang Z, Thurfjell L, Covin A, Hehir CT, Baker D, Bazenet C, Lovestone S. Blood protein predictors of brain amyloid for enrichment in clinical trials?. Alzheimers Dement (Amst). 2015 Mar 29;1(1):48-60. doi: 10.1016/j.dadm.2014.11.005. eCollection 2015 Mar. PMID: 27239491; PMCID: PMC4876903.
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