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Showing 1 to 9 of 9 entries
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STIM2 Contributes to Enhanced Store-operated Ca Entry in Pulmonary Artery Smooth Muscle Cells from Patients with Idiopathic Pulmonary Arterial Hypertension.

Pulmonary circulation

Song MY, Makino A, Yuan JX.
PMID: 21709766
Pulm Circ. 2011;1(1):84-94. doi: 10.4103/2045-8932.78106.

Pulmonary vasoconstriction and vascular remodeling are two major causes for elevated pulmonary vascular resistance and pulmonary arterial pressure in patients with idiopathic pulmonary arterial hypertension (IPAH). An increase in cytosolic free Ca(2+) concentration ([Ca(2+)](cyt)) in pulmonary artery smooth muscle...

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Song MY, Makino A, Yuan JX. STIM2 Contributes to Enhanced Store-operated Ca Entry in Pulmonary Artery Smooth Muscle Cells from Patients with Idiopathic Pulmonary Arterial Hypertension. Pulm Circ. 2011;1(1):84-94doi: 10.4103/2045-8932.78106.
Song, M. Y., Makino, A., & Yuan, J. X. (2011). STIM2 Contributes to Enhanced Store-operated Ca Entry in Pulmonary Artery Smooth Muscle Cells from Patients with Idiopathic Pulmonary Arterial Hypertension. Pulmonary circulation, 1(1), 84-94. https://doi.org/10.4103/2045-8932.78106
Song, Michael Y, et al. "STIM2 Contributes to Enhanced Store-operated Ca Entry in Pulmonary Artery Smooth Muscle Cells from Patients with Idiopathic Pulmonary Arterial Hypertension." Pulmonary circulation vol. 1,1 (2011): 84-94. doi: https://doi.org/10.4103/2045-8932.78106
Song MY, Makino A, Yuan JX. STIM2 Contributes to Enhanced Store-operated Ca Entry in Pulmonary Artery Smooth Muscle Cells from Patients with Idiopathic Pulmonary Arterial Hypertension. Pulm Circ. 2011;1(1):84-94. doi: 10.4103/2045-8932.78106. PMID: 21709766; PMCID: PMC3121304.
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Optimization of isolated perfused/ventilated mouse lung to study hypoxic pulmonary vasoconstriction.

Pulmonary circulation

Yoo HY, Zeifman A, Ko EA, Smith KA, Chen J, Machado RF, Zhao YY, Minshall RD, Yuan JX.
PMID: 24015341
Pulm Circ. 2013 Apr;3(2):396-405. doi: 10.4103/2045-8932.114776.

Hypoxic pulmonary vasoconstriction (HPV) is a compensatory physiological mechanism in the lung that optimizes the matching of ventilation to perfusion and thereby maximizes gas exchange. Historically, HPV has been primarily studied in isolated perfused/ventilated lungs; however, the results of...

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Yoo HY, Zeifman A, Ko EA, et al. Optimization of isolated perfused/ventilated mouse lung to study hypoxic pulmonary vasoconstriction. Pulm Circ. 2013;3(2):396-405doi: 10.4103/2045-8932.114776.
Yoo, H. Y., Zeifman, A., Ko, E. A., Smith, K. A., Chen, J., Machado, R. F., Zhao, Y. Y., Minshall, R. D., & Yuan, J. X. (2013). Optimization of isolated perfused/ventilated mouse lung to study hypoxic pulmonary vasoconstriction. Pulmonary circulation, 3(2), 396-405. https://doi.org/10.4103/2045-8932.114776
Yoo, Hae Young, et al. "Optimization of isolated perfused/ventilated mouse lung to study hypoxic pulmonary vasoconstriction." Pulmonary circulation vol. 3,2 (2013): 396-405. doi: https://doi.org/10.4103/2045-8932.114776
Yoo HY, Zeifman A, Ko EA, Smith KA, Chen J, Machado RF, Zhao YY, Minshall RD, Yuan JX. Optimization of isolated perfused/ventilated mouse lung to study hypoxic pulmonary vasoconstriction. Pulm Circ. 2013 Apr;3(2):396-405. doi: 10.4103/2045-8932.114776. PMID: 24015341; PMCID: PMC3757835.
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Functional ion channels in human pulmonary artery smooth muscle cells: Voltage-dependent cation channels.

Pulmonary circulation

Firth AL, Remillard CV, Platoshyn O, Fantozzi I, Ko EA, Yuan JX.
PMID: 21927714
Pulm Circ. 2011 Jan 01;1(1):48-71. doi: 10.4103/2045-8932.78103.

The activity of voltage-gated ion channels is critical for the maintenance of cellular membrane potential and generation of action potentials. In turn, membrane potential regulates cellular ion homeostasis, triggering the opening and closing of ion channels in the plasma...

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Firth AL, Remillard CV, Platoshyn O, et al. Functional ion channels in human pulmonary artery smooth muscle cells: Voltage-dependent cation channels. Pulm Circ. 2011;1(1):48-71doi: 10.4103/2045-8932.78103.
Firth, A. L., Remillard, C. V., Platoshyn, O., Fantozzi, I., Ko, E. A., & Yuan, J. X. (2011). Functional ion channels in human pulmonary artery smooth muscle cells: Voltage-dependent cation channels. Pulmonary circulation, 1(1), 48-71. https://doi.org/10.4103/2045-8932.78103
Firth, Amy L, et al. "Functional ion channels in human pulmonary artery smooth muscle cells: Voltage-dependent cation channels." Pulmonary circulation vol. 1,1 (2011): 48-71. doi: https://doi.org/10.4103/2045-8932.78103
Firth AL, Remillard CV, Platoshyn O, Fantozzi I, Ko EA, Yuan JX. Functional ion channels in human pulmonary artery smooth muscle cells: Voltage-dependent cation channels. Pulm Circ. 2011 Jan 01;1(1):48-71. doi: 10.4103/2045-8932.78103. PMID: 21927714; PMCID: PMC3173772.
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Pathogenic role of store-operated and receptor-operated ca(2+) channels in pulmonary arterial hypertension.

Journal of signal transduction

Fernandez RA, Sundivakkam P, Smith KA, Zeifman AS, Drennan AR, Yuan JX.
PMID: 23056939
J Signal Transduct. 2012;2012:951497. doi: 10.1155/2012/951497. Epub 2012 Sep 27.

Pulmonary circulation is an important circulatory system in which the body brings in oxygen. Pulmonary arterial hypertension (PAH) is a progressive and fatal disease that predominantly affects women. Sustained pulmonary vasoconstriction, excessive pulmonary vascular remodeling, in situ thrombosis, and...

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Fernandez RA, Sundivakkam P, Smith KA, et al. Pathogenic role of store-operated and receptor-operated ca(2+) channels in pulmonary arterial hypertension. J Signal Transduct. 2012;2012:951497doi: 10.1155/2012/951497.
Fernandez, R. A., Sundivakkam, P., Smith, K. A., Zeifman, A. S., Drennan, A. R., & Yuan, J. X. (2012). Pathogenic role of store-operated and receptor-operated ca(2+) channels in pulmonary arterial hypertension. Journal of signal transduction, 2012951497. https://doi.org/10.1155/2012/951497
Fernandez, Ruby A, et al. "Pathogenic role of store-operated and receptor-operated ca(2+) channels in pulmonary arterial hypertension." Journal of signal transduction vol. 2012 (2012): 951497. doi: https://doi.org/10.1155/2012/951497
Fernandez RA, Sundivakkam P, Smith KA, Zeifman AS, Drennan AR, Yuan JX. Pathogenic role of store-operated and receptor-operated ca(2+) channels in pulmonary arterial hypertension. J Signal Transduct. 2012;2012:951497. doi: 10.1155/2012/951497. Epub 2012 Sep 27. PMID: 23056939; PMCID: PMC3465915.
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Tension Measurement in Isolated Rat and Mouse Pulmonary Artery.

Drug discovery today. Disease models

Ko EA, Song MY, Donthamsetty R, Makino A, Yuan JX.
PMID: 23175638
Drug Discov Today Dis Models. 2010;7(3):123-130. doi: 10.1016/j.ddmod.2011.04.001. Epub 2011 May 06.

Arterial vasoconstriction is an important physiological process in regulating blood pressure, and is involved in pathologies. The isolation of arteries from rats and mice, as well as the measurement of vascular tension in an ex vivo preparation, are important...

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Ko EA, Song MY, Donthamsetty R, et al. Tension Measurement in Isolated Rat and Mouse Pulmonary Artery. Drug Discov Today Dis Models. 2011;7(3):123-130doi: 10.1016/j.ddmod.2011.04.001.
Ko, E. A., Song, M. Y., Donthamsetty, R., Makino, A., & Yuan, J. X. (2010). Tension Measurement in Isolated Rat and Mouse Pulmonary Artery. Drug discovery today. Disease models, 7(3), 123-130. https://doi.org/10.1016/j.ddmod.2011.04.001
Ko, Eun A, et al. "Tension Measurement in Isolated Rat and Mouse Pulmonary Artery." Drug discovery today. Disease models vol. 7,3 (2010): 123-130. doi: https://doi.org/10.1016/j.ddmod.2011.04.001
Ko EA, Song MY, Donthamsetty R, Makino A, Yuan JX. Tension Measurement in Isolated Rat and Mouse Pulmonary Artery. Drug Discov Today Dis Models. 2010;7(3):123-130. doi: 10.1016/j.ddmod.2011.04.001. Epub 2011 May 06. PMID: 23175638; PMCID: PMC3501740.
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Identification of functional progenitor cells in the pulmonary vasculature.

Pulmonary circulation

Firth AL, Yuan JX.
PMID: 22558524
Pulm Circ. 2012 Jan-Mar;2(1):84-100. doi: 10.4103/2045-8932.94841.

The pulmonary vasculature comprises a complex network of branching arteries and veins all functioning to reoxygenate the blood for circulation around the body. The cell types of the pulmonary artery are able to respond to changes in oxygen tension...

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Firth AL, Yuan JX. Identification of functional progenitor cells in the pulmonary vasculature. Pulm Circ. 2012;2(1):84-100doi: 10.4103/2045-8932.94841.
Firth, A. L., & Yuan, J. X. (2012). Identification of functional progenitor cells in the pulmonary vasculature. Pulmonary circulation, 2(1), 84-100. https://doi.org/10.4103/2045-8932.94841
Firth, Amy L, and Yuan, Jason X-J. "Identification of functional progenitor cells in the pulmonary vasculature." Pulmonary circulation vol. 2,1 (2012): 84-100. doi: https://doi.org/10.4103/2045-8932.94841
Firth AL, Yuan JX. Identification of functional progenitor cells in the pulmonary vasculature. Pulm Circ. 2012 Jan-Mar;2(1):84-100. doi: 10.4103/2045-8932.94841. PMID: 22558524; PMCID: PMC3342753.
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Activity of Ca -activated Cl channels contributes to regulating receptor- and store-operated Ca entry in human pulmonary artery smooth muscle cells.

Pulmonary circulation

Yamamura A, Yamamura H, Zeifman A, Yuan JX.
PMID: 22034612
Pulm Circ. 2011 Apr-Jun;1(2):269-79. doi: 10.4103/2045-8932.83447.

Intracellular Ca(2+) plays a fundamental role in regulating cell functions in pulmonary arterial smooth muscle cells (PASMCs). A rise in cytosolic Ca(2+) concentration ([Ca(2+)](cyt)) triggers pulmonary vasoconstriction and stimulates PASMC proliferation. [Ca(2+)](cyt) is increased mainly by Ca(2+) release from...

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Yamamura A, Yamamura H, Zeifman A, et al. Activity of Ca -activated Cl channels contributes to regulating receptor- and store-operated Ca entry in human pulmonary artery smooth muscle cells. Pulm Circ. 2011;1(2):269-79doi: 10.4103/2045-8932.83447.
Yamamura, A., Yamamura, H., Zeifman, A., & Yuan, J. X. (2011). Activity of Ca -activated Cl channels contributes to regulating receptor- and store-operated Ca entry in human pulmonary artery smooth muscle cells. Pulmonary circulation, 1(2), 269-79. https://doi.org/10.4103/2045-8932.83447
Yamamura, Aya, et al. "Activity of Ca -activated Cl channels contributes to regulating receptor- and store-operated Ca entry in human pulmonary artery smooth muscle cells." Pulmonary circulation vol. 1,2 (2011): 269-79. doi: https://doi.org/10.4103/2045-8932.83447
Yamamura A, Yamamura H, Zeifman A, Yuan JX. Activity of Ca -activated Cl channels contributes to regulating receptor- and store-operated Ca entry in human pulmonary artery smooth muscle cells. Pulm Circ. 2011 Apr-Jun;1(2):269-79. doi: 10.4103/2045-8932.83447. PMID: 22034612; PMCID: PMC3198647.
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Fenfluramine-induced gene dysregulation in human pulmonary artery smooth muscle and endothelial cells.

Pulmonary circulation

Yao W, Mu W, Zeifman A, Lofti M, Remillard CV, Makino A, Perkins DL, Garcia JG, Yuan JX, Zhang W.
PMID: 22140631
Pulm Circ. 2011 Jul-Sep;1(3):405-18. doi: 10.4103/2045-8932.87310.

Fenfluramine is prescribed either alone or in combination with phentermine as part of Fen-Phen, an anti-obesity medication. Fenfluramine was withdrawn from the US market in 1997 due to reports of heart valvular disease, pulmonary arterial hypertension, and cardiac fibrosis....

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Yao W, Mu W, Zeifman A, et al. Fenfluramine-induced gene dysregulation in human pulmonary artery smooth muscle and endothelial cells. Pulm Circ. 2011;1(3):405-18doi: 10.4103/2045-8932.87310.
Yao, W., Mu, W., Zeifman, A., Lofti, M., Remillard, C. V., Makino, A., Perkins, D. L., Garcia, J. G., Yuan, J. X., & Zhang, W. (2011). Fenfluramine-induced gene dysregulation in human pulmonary artery smooth muscle and endothelial cells. Pulmonary circulation, 1(3), 405-18. https://doi.org/10.4103/2045-8932.87310
Yao, Weijuan, et al. "Fenfluramine-induced gene dysregulation in human pulmonary artery smooth muscle and endothelial cells." Pulmonary circulation vol. 1,3 (2011): 405-18. doi: https://doi.org/10.4103/2045-8932.87310
Yao W, Mu W, Zeifman A, Lofti M, Remillard CV, Makino A, Perkins DL, Garcia JG, Yuan JX, Zhang W. Fenfluramine-induced gene dysregulation in human pulmonary artery smooth muscle and endothelial cells. Pulm Circ. 2011 Jul-Sep;1(3):405-18. doi: 10.4103/2045-8932.87310. PMID: 22140631; PMCID: PMC3224433.
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Thrombin-mediated activation of Akt signaling contributes to pulmonary vascular remodeling in pulmonary hypertension.

Physiological reports

Ogawa A, Firth AL, Ariyasu S, Yamadori I, Matsubara H, Song S, Fraidenburg DR, Yuan JX.
PMID: 24744867
Physiol Rep. 2013 Dec 29;1(7):e00190. doi: 10.1002/phy2.190. eCollection 2013 Dec 01.

Chronic thromboembolic pulmonary hypertension (CTEPH) has been increasingly recognized as a common source of elevated pulmonary vascular resistance and pulmonary hypertension. It is clear that development of pulmonary thromboemboli is the inciting event for this process, yet it remains...

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Ogawa A, Firth AL, Ariyasu S, et al. Thrombin-mediated activation of Akt signaling contributes to pulmonary vascular remodeling in pulmonary hypertension. Physiol Rep. 2013;1(7):e00190doi: 10.1002/phy2.190.
Ogawa, A., Firth, A. L., Ariyasu, S., Yamadori, I., Matsubara, H., Song, S., Fraidenburg, D. R., & Yuan, J. X. (2013). Thrombin-mediated activation of Akt signaling contributes to pulmonary vascular remodeling in pulmonary hypertension. Physiological reports, 1(7), e00190. https://doi.org/10.1002/phy2.190
Ogawa, Aiko, et al. "Thrombin-mediated activation of Akt signaling contributes to pulmonary vascular remodeling in pulmonary hypertension." Physiological reports vol. 1,7 (2013): e00190. doi: https://doi.org/10.1002/phy2.190
Ogawa A, Firth AL, Ariyasu S, Yamadori I, Matsubara H, Song S, Fraidenburg DR, Yuan JX. Thrombin-mediated activation of Akt signaling contributes to pulmonary vascular remodeling in pulmonary hypertension. Physiol Rep. 2013 Dec 29;1(7):e00190. doi: 10.1002/phy2.190. eCollection 2013 Dec 01. PMID: 24744867; PMCID: PMC3970741.
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Showing 1 to 9 of 9 entries