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Effects of a Flavonoid-Rich Fraction on the Acquisition and Extinction of Fear Memory: Pharmacological and Molecular Approaches.

Frontiers in behavioral neuroscience

de Oliveira DR, Zamberlam CR, Rêgo GM, Cavalheiro A, Cerutti JM, Cerutti SM.
PMID: 26778988
Front Behav Neurosci. 2016 Jan 05;9:345. doi: 10.3389/fnbeh.2015.00345. eCollection 2015.

The effects of flavonoids have been correlated with their ability to modulate the glutamatergic, serotoninergic, and GABAergic neurotransmission; the major targets of these substances are N-methyl-D-aspartic acid receptor (NMDARs), serotonin type1A receptor (5-HT1ARs), and the gamma-aminobutyric acid type A...

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de Oliveira DR, Zamberlam CR, Rêgo GM, et al. Effects of a Flavonoid-Rich Fraction on the Acquisition and Extinction of Fear Memory: Pharmacological and Molecular Approaches. Front Behav Neurosci. 2016;9:345doi: 10.3389/fnbeh.2015.00345.
de Oliveira, D. R., Zamberlam, C. R., Rêgo, G. M., Cavalheiro, A., Cerutti, J. M., & Cerutti, S. M. (2015). Effects of a Flavonoid-Rich Fraction on the Acquisition and Extinction of Fear Memory: Pharmacological and Molecular Approaches. Frontiers in behavioral neuroscience, 9345. https://doi.org/10.3389/fnbeh.2015.00345
de Oliveira, Daniela R, et al. "Effects of a Flavonoid-Rich Fraction on the Acquisition and Extinction of Fear Memory: Pharmacological and Molecular Approaches." Frontiers in behavioral neuroscience vol. 9 (2015): 345. doi: https://doi.org/10.3389/fnbeh.2015.00345
de Oliveira DR, Zamberlam CR, Rêgo GM, Cavalheiro A, Cerutti JM, Cerutti SM. Effects of a Flavonoid-Rich Fraction on the Acquisition and Extinction of Fear Memory: Pharmacological and Molecular Approaches. Front Behav Neurosci. 2016 Jan 05;9:345. doi: 10.3389/fnbeh.2015.00345. eCollection 2015. PMID: 26778988; PMCID: PMC4700274.
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Neurally Released GABA Acts via GABA.

Frontiers in physiology

Koussoulas K, Swaminathan M, Fung C, Bornstein JC, Foong JPP.
PMID: 29487540
Front Physiol. 2018 Feb 13;9:97. doi: 10.3389/fphys.2018.00097. eCollection 2018.

γ-Aminobutyric Acid (GABA) and its receptors, GABA

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Koussoulas K, Swaminathan M, Fung C, et al. Neurally Released GABA Acts via GABA. Front Physiol. 2018;9:97doi: 10.3389/fphys.2018.00097.
Koussoulas, K., Swaminathan, M., Fung, C., Bornstein, J. C., & Foong, J. P. P. (2018). Neurally Released GABA Acts via GABA. Frontiers in physiology, 997. https://doi.org/10.3389/fphys.2018.00097
Koussoulas, Katerina, et al. "Neurally Released GABA Acts via GABA." Frontiers in physiology vol. 9 (2018): 97. doi: https://doi.org/10.3389/fphys.2018.00097
Koussoulas K, Swaminathan M, Fung C, Bornstein JC, Foong JPP. Neurally Released GABA Acts via GABA. Front Physiol. 2018 Feb 13;9:97. doi: 10.3389/fphys.2018.00097. eCollection 2018. PMID: 29487540; PMCID: PMC5816811.
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Potentiation of Gamma Aminobutyric Acid Receptors (GABAAR) by Ethanol: How Are Inhibitory Receptors Affected?.

Frontiers in cellular neuroscience

Förstera B, Castro PA, Moraga-Cid G, Aguayo LG.
PMID: 27199667
Front Cell Neurosci. 2016 May 06;10:114. doi: 10.3389/fncel.2016.00114. eCollection 2016.

In recent years there has been an increase in the understanding of ethanol actions on the type A γ-aminobutyric acid chloride channel (GABAAR), a member of the pentameric ligand gated ion channels (pLGICs). However, the mechanism by which ethanol...

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Förstera B, Castro PA, Moraga-Cid G, et al. Potentiation of Gamma Aminobutyric Acid Receptors (GABAAR) by Ethanol: How Are Inhibitory Receptors Affected?. Front Cell Neurosci. 2016;10:114doi: 10.3389/fncel.2016.00114.
Förstera, B., Castro, P. A., Moraga-Cid, G., & Aguayo, L. G. (2016). Potentiation of Gamma Aminobutyric Acid Receptors (GABAAR) by Ethanol: How Are Inhibitory Receptors Affected?. Frontiers in cellular neuroscience, 10114. https://doi.org/10.3389/fncel.2016.00114
Förstera, Benjamin, et al. "Potentiation of Gamma Aminobutyric Acid Receptors (GABAAR) by Ethanol: How Are Inhibitory Receptors Affected?." Frontiers in cellular neuroscience vol. 10 (2016): 114. doi: https://doi.org/10.3389/fncel.2016.00114
Förstera B, Castro PA, Moraga-Cid G, Aguayo LG. Potentiation of Gamma Aminobutyric Acid Receptors (GABAAR) by Ethanol: How Are Inhibitory Receptors Affected?. Front Cell Neurosci. 2016 May 06;10:114. doi: 10.3389/fncel.2016.00114. eCollection 2016. PMID: 27199667; PMCID: PMC4858537.
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Voluntary Ethanol Consumption Induced by Social Isolation Reverses the Increase of α(4)/δ GABA(A) Receptor Gene Expression and Function in the Hippocampus of C57BL/6J Mice.

Frontiers in neuroscience

Sanna E, Talani G, Obili N, Mascia MP, Mostallino MC, Secci PP, Pisu MG, Biggio F, Utzeri C, Olla P, Biggio G, Follesa P.
PMID: 21347217
Front Neurosci. 2011 Feb 10;5:15. doi: 10.3389/fnins.2011.00015. eCollection 2011.

Post-weaning social isolation (SI) is a model of prolonged mild stress characterized by behavioral and neurochemical alterations. We used SI in C57BL/6J mice to investigate the effects of ethanol (EtOH) in the free-choice drinking paradigm on gene expression and...

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Sanna E, Talani G, Obili N, et al. Voluntary Ethanol Consumption Induced by Social Isolation Reverses the Increase of α(4)/δ GABA(A) Receptor Gene Expression and Function in the Hippocampus of C57BL/6J Mice. Front Neurosci. 2011;5:15doi: 10.3389/fnins.2011.00015.
Sanna, E., Talani, G., Obili, N., Mascia, M. P., Mostallino, M. C., Secci, P. P., Pisu, M. G., Biggio, F., Utzeri, C., Olla, P., Biggio, G., & Follesa, P. (2011). Voluntary Ethanol Consumption Induced by Social Isolation Reverses the Increase of α(4)/δ GABA(A) Receptor Gene Expression and Function in the Hippocampus of C57BL/6J Mice. Frontiers in neuroscience, 515. https://doi.org/10.3389/fnins.2011.00015
Sanna, Enrico, et al. "Voluntary Ethanol Consumption Induced by Social Isolation Reverses the Increase of α(4)/δ GABA(A) Receptor Gene Expression and Function in the Hippocampus of C57BL/6J Mice." Frontiers in neuroscience vol. 5 (2011): 15. doi: https://doi.org/10.3389/fnins.2011.00015
Sanna E, Talani G, Obili N, Mascia MP, Mostallino MC, Secci PP, Pisu MG, Biggio F, Utzeri C, Olla P, Biggio G, Follesa P. Voluntary Ethanol Consumption Induced by Social Isolation Reverses the Increase of α(4)/δ GABA(A) Receptor Gene Expression and Function in the Hippocampus of C57BL/6J Mice. Front Neurosci. 2011 Feb 10;5:15. doi: 10.3389/fnins.2011.00015. eCollection 2011. PMID: 21347217; PMCID: PMC3039156.
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Modulation of cell surface GABA(B) receptors by desensitization, trafficking and regulated degradation.

World journal of biological chemistry

Benke D, Zemoura K, Maier PJ.
PMID: 22558486
World J Biol Chem. 2012 Apr 26;3(4):61-72. doi: 10.4331/wjbc.v3.i4.61.

Inhibitory neurotransmission ensures normal brain function by counteracting and integrating excitatory activity. γ-Aminobutyric acid (GABA) is the main inhibitory neurotransmitter in the mammalian central nervous system, and mediates its effects via two classes of receptors: the GABA(A) and GABA(B)...

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Benke D, Zemoura K, Maier PJ. Modulation of cell surface GABA(B) receptors by desensitization, trafficking and regulated degradation. World J Biol Chem. 2012;3(4):61-72doi: 10.4331/wjbc.v3.i4.61.
Benke, D., Zemoura, K., & Maier, P. J. (2012). Modulation of cell surface GABA(B) receptors by desensitization, trafficking and regulated degradation. World journal of biological chemistry, 3(4), 61-72. https://doi.org/10.4331/wjbc.v3.i4.61
Benke, Dietmar, et al. "Modulation of cell surface GABA(B) receptors by desensitization, trafficking and regulated degradation." World journal of biological chemistry vol. 3,4 (2012): 61-72. doi: https://doi.org/10.4331/wjbc.v3.i4.61
Benke D, Zemoura K, Maier PJ. Modulation of cell surface GABA(B) receptors by desensitization, trafficking and regulated degradation. World J Biol Chem. 2012 Apr 26;3(4):61-72. doi: 10.4331/wjbc.v3.i4.61. PMID: 22558486; PMCID: PMC3342575.
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γ1-Containing GABA-A Receptors Cluster at Synapses Where they Mediate Slower Synaptic Currents than γ2-Containing GABA-A Receptors.

Frontiers in molecular neuroscience

Dixon CL, Sah P, Keramidas A, Lynch JW, Durisic N.
PMID: 28642681
Front Mol Neurosci. 2017 Jun 08;10:178. doi: 10.3389/fnmol.2017.00178. eCollection 2017.

GABA-A receptors (GABA

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Dixon CL, Sah P, Keramidas A, et al. γ1-Containing GABA-A Receptors Cluster at Synapses Where they Mediate Slower Synaptic Currents than γ2-Containing GABA-A Receptors. Front Mol Neurosci. 2017;10:178doi: 10.3389/fnmol.2017.00178.
Dixon, C. L., Sah, P., Keramidas, A., Lynch, J. W., & Durisic, N. (2017). γ1-Containing GABA-A Receptors Cluster at Synapses Where they Mediate Slower Synaptic Currents than γ2-Containing GABA-A Receptors. Frontiers in molecular neuroscience, 10178. https://doi.org/10.3389/fnmol.2017.00178
Dixon, Christine L, et al. "γ1-Containing GABA-A Receptors Cluster at Synapses Where they Mediate Slower Synaptic Currents than γ2-Containing GABA-A Receptors." Frontiers in molecular neuroscience vol. 10 (2017): 178. doi: https://doi.org/10.3389/fnmol.2017.00178
Dixon CL, Sah P, Keramidas A, Lynch JW, Durisic N. γ1-Containing GABA-A Receptors Cluster at Synapses Where they Mediate Slower Synaptic Currents than γ2-Containing GABA-A Receptors. Front Mol Neurosci. 2017 Jun 08;10:178. doi: 10.3389/fnmol.2017.00178. eCollection 2017. PMID: 28642681; PMCID: PMC5462899.
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The effect of age and sex on the expression of GABA signaling components in the human hippocampus and entorhinal cortex.

Scientific reports

Ethiraj J, Palpagama TH, Turner C, van der Werf B, Waldvogel HJ, Faull RLM, Kwakowsky A.
PMID: 34728681
Sci Rep. 2021 Nov 02;11(1):21470. doi: 10.1038/s41598-021-00792-8.

Gamma-aminobutyric acid (GABA) is the primary inhibitory neurotransmitter in the nervous system. The GABA signaling system in the brain is comprised of GABA synthesizing enzymes, transporters, GABAA and GABAB receptors (GABA

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Ethiraj J, Palpagama TH, Turner C, et al. The effect of age and sex on the expression of GABA signaling components in the human hippocampus and entorhinal cortex. Sci Rep. 2021;11(1):21470doi: 10.1038/s41598-021-00792-8.
Ethiraj, J., Palpagama, T. H., Turner, C., van der Werf, B., Waldvogel, H. J., Faull, R. L. M., & Kwakowsky, A. (2021). The effect of age and sex on the expression of GABA signaling components in the human hippocampus and entorhinal cortex. Scientific reports, 11(1), 21470. https://doi.org/10.1038/s41598-021-00792-8
Ethiraj, Jayarjun, et al. "The effect of age and sex on the expression of GABA signaling components in the human hippocampus and entorhinal cortex." Scientific reports vol. 11,1 (2021): 21470. doi: https://doi.org/10.1038/s41598-021-00792-8
Ethiraj J, Palpagama TH, Turner C, van der Werf B, Waldvogel HJ, Faull RLM, Kwakowsky A. The effect of age and sex on the expression of GABA signaling components in the human hippocampus and entorhinal cortex. Sci Rep. 2021 Nov 02;11(1):21470. doi: 10.1038/s41598-021-00792-8. PMID: 34728681; PMCID: PMC8563768.
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Combination of antiseizure medications phenobarbital, ketamine, and midazolam reduces soman-induced epileptogenesis and brain pathology in rats.

Epilepsia open

Lumley LA, Marrero-Rosado B, Rossetti F, Schultz CR, Stone MF, Niquet J, Wasterlain CG.
PMID: 34657398
Epilepsia Open. 2021 Dec;6(4):757-769. doi: 10.1002/epi4.12552. Epub 2021 Oct 23.

OBJECTIVE: Cholinergic-induced status epilepticus (SE) is associated with a loss of synaptic gamma-aminobutyric acid A receptors (GABAMETHODS: We investigated the efficacy of combining antiseizure medications to include a benzodiazepine and a barbiturate allosteric GABARESULTS: The triple therapy combination of...

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Lumley LA, Marrero-Rosado B, Rossetti F, et al. Combination of antiseizure medications phenobarbital, ketamine, and midazolam reduces soman-induced epileptogenesis and brain pathology in rats. Epilepsia Open. 2021;6(4):757-769doi: 10.1002/epi4.12552.
Lumley, L. A., Marrero-Rosado, B., Rossetti, F., Schultz, C. R., Stone, M. F., Niquet, J., & Wasterlain, C. G. (2021). Combination of antiseizure medications phenobarbital, ketamine, and midazolam reduces soman-induced epileptogenesis and brain pathology in rats. Epilepsia open, 6(4), 757-769. https://doi.org/10.1002/epi4.12552
Lumley, Lucille A, et al. "Combination of antiseizure medications phenobarbital, ketamine, and midazolam reduces soman-induced epileptogenesis and brain pathology in rats." Epilepsia open vol. 6,4 (2021): 757-769. doi: https://doi.org/10.1002/epi4.12552
Lumley LA, Marrero-Rosado B, Rossetti F, Schultz CR, Stone MF, Niquet J, Wasterlain CG. Combination of antiseizure medications phenobarbital, ketamine, and midazolam reduces soman-induced epileptogenesis and brain pathology in rats. Epilepsia Open. 2021 Dec;6(4):757-769. doi: 10.1002/epi4.12552. Epub 2021 Oct 23. PMID: 34657398; PMCID: PMC8633481.
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Azobenzene-isoxazoline as photopharmacological ligand for optical control of insect GABA receptor and behavior.

Pest management science

Qiao Z, Ji Y, Zhang Y, Li Z, Xu Z, Shao X.
PMID: 34516709
Pest Manag Sci. 2021 Sep 13; doi: 10.1002/ps.6641. Epub 2021 Sep 13.

BACKGROUND: Photopharmacology is a fast-growing photonics-based technology, which realizes the high-resolution regulation of drugs in time and space through light. The purpose of this research was to introduce photochromic groups into the isoxazoline structure to realize the regulation of...

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Qiao Z, Ji Y, Zhang Y, et al. Azobenzene-isoxazoline as photopharmacological ligand for optical control of insect GABA receptor and behavior. Pest Manag Sci. 2021;doi: 10.1002/ps.6641.
Qiao, Z., Ji, Y., Zhang, Y., Li, Z., Xu, Z., & Shao, X. (2021). Azobenzene-isoxazoline as photopharmacological ligand for optical control of insect GABA receptor and behavior. Pest management science, . https://doi.org/10.1002/ps.6641
Qiao, Zhi, et al. "Azobenzene-isoxazoline as photopharmacological ligand for optical control of insect GABA receptor and behavior." Pest management science vol. (2021). doi: https://doi.org/10.1002/ps.6641
Qiao Z, Ji Y, Zhang Y, Li Z, Xu Z, Shao X. Azobenzene-isoxazoline as photopharmacological ligand for optical control of insect GABA receptor and behavior. Pest Manag Sci. 2021 Sep 13; doi: 10.1002/ps.6641. Epub 2021 Sep 13. PMID: 34516709.
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Potential Therapeutic Use of Neurosteroids for Hypertension.

Frontiers in physiology

Head GA, Jackson KL, Gueguen C.
PMID: 31920690
Front Physiol. 2019 Dec 12;10:1477. doi: 10.3389/fphys.2019.01477. eCollection 2019.

The sympathetic nervous system (SNS) contribution to long-term setting of blood pressure (BP) and hence hypertension has been a continuing controversy over many decades. However, the contribution of increased sympathetic vasomotor tone to the heart, kidney, and blood vessels...

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Head GA, Jackson KL, Gueguen C. Potential Therapeutic Use of Neurosteroids for Hypertension. Front Physiol. 2019;10:1477doi: 10.3389/fphys.2019.01477.
Head, G. A., Jackson, K. L., & Gueguen, C. (2019). Potential Therapeutic Use of Neurosteroids for Hypertension. Frontiers in physiology, 101477. https://doi.org/10.3389/fphys.2019.01477
Head, Geoffrey A, et al. "Potential Therapeutic Use of Neurosteroids for Hypertension." Frontiers in physiology vol. 10 (2019): 1477. doi: https://doi.org/10.3389/fphys.2019.01477
Head GA, Jackson KL, Gueguen C. Potential Therapeutic Use of Neurosteroids for Hypertension. Front Physiol. 2019 Dec 12;10:1477. doi: 10.3389/fphys.2019.01477. eCollection 2019. PMID: 31920690; PMCID: PMC6920208.
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Mechanisms and Regulation of Neuronal GABA.

Current topics in behavioral neurosciences

Rose TR, Wickman K.
PMID: 32808092
Curr Top Behav Neurosci. 2022;52:39-79. doi: 10.1007/7854_2020_129.

γ-Aminobutyric acid B receptors (GABA

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Rose TR, Wickman K. Mechanisms and Regulation of Neuronal GABA. Curr Top Behav Neurosci. 2022;52:39-79doi: 10.1007/7854_2020_129.
Rose, T. R., & Wickman, K. (2022). Mechanisms and Regulation of Neuronal GABA. Current topics in behavioral neurosciences, 5239-79. https://doi.org/10.1007/7854_2020_129
Rose, Timothy R, and Wickman, Kevin. "Mechanisms and Regulation of Neuronal GABA." Current topics in behavioral neurosciences vol. 52 (2022): 39-79. doi: https://doi.org/10.1007/7854_2020_129
Rose TR, Wickman K. Mechanisms and Regulation of Neuronal GABA. Curr Top Behav Neurosci. 2022;52:39-79. doi: 10.1007/7854_2020_129. PMID: 32808092.
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Intravenous Ketamine Bolus(es) for the Treatment of Status Epilepticus, Refractory Status Epilepticus, and Cluster Seizures: A Retrospective Study of 15 Dogs.

Frontiers in veterinary science

Roynard P, Bilderback A, Dewey CW.
PMID: 33681317
Front Vet Sci. 2021 Feb 17;8:547279. doi: 10.3389/fvets.2021.547279. eCollection 2021.

Status epilepticus (SE) and cluster seizures (CS) are common occurrences in veterinary neurology and frequent reasons of admission to veterinary hospitals. With prolonged seizure activity, gamma amino-butyric acid (GABA) receptors (GABAa receptors) become inactive, leading to a state of...

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Roynard P, Bilderback A, Dewey CW. Intravenous Ketamine Bolus(es) for the Treatment of Status Epilepticus, Refractory Status Epilepticus, and Cluster Seizures: A Retrospective Study of 15 Dogs. Front Vet Sci. 2021;8:547279doi: 10.3389/fvets.2021.547279.
Roynard, P., Bilderback, A., & Dewey, C. W. (2021). Intravenous Ketamine Bolus(es) for the Treatment of Status Epilepticus, Refractory Status Epilepticus, and Cluster Seizures: A Retrospective Study of 15 Dogs. Frontiers in veterinary science, 8547279. https://doi.org/10.3389/fvets.2021.547279
Roynard, Patrick, et al. "Intravenous Ketamine Bolus(es) for the Treatment of Status Epilepticus, Refractory Status Epilepticus, and Cluster Seizures: A Retrospective Study of 15 Dogs." Frontiers in veterinary science vol. 8 (2021): 547279. doi: https://doi.org/10.3389/fvets.2021.547279
Roynard P, Bilderback A, Dewey CW. Intravenous Ketamine Bolus(es) for the Treatment of Status Epilepticus, Refractory Status Epilepticus, and Cluster Seizures: A Retrospective Study of 15 Dogs. Front Vet Sci. 2021 Feb 17;8:547279. doi: 10.3389/fvets.2021.547279. eCollection 2021. PMID: 33681317; PMCID: PMC7925624.
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Showing 1 to 12 of 85 entries