Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 112 entries
Sorted by: Best Match Show Resources per page
Phenotypes and Virulence among Staphylococcus aureus USA100, USA200, USA300, USA400, and USA600 Clonal Lineages.

mSphere

King JM, Kulhankova K, Stach CS, Vu BG, Salgado-Pabón W.
PMID: 27303750
mSphere. 2016 Jun 08;1(3). doi: 10.1128/mSphere.00071-16. eCollection 2016.

Staphylococcus aureus diseases affect ~500,000 individuals per year in the United States. Worldwide, the USA100, USA200, USA400, and USA600 lineages cause many of the life-threatening S. aureus infections, such as bacteremia, infective endocarditis, pneumonia, toxic shock syndrome, and surgical...

Cite
King JM, Kulhankova K, Stach CS, et al. Phenotypes and Virulence among Staphylococcus aureus USA100, USA200, USA300, USA400, and USA600 Clonal Lineages. mSphere. 2016;1(3)doi: 10.1128/mSphere.00071-16.
King, J. M., Kulhankova, K., Stach, C. S., Vu, B. G., & Salgado-Pabón, W. (2016). Phenotypes and Virulence among Staphylococcus aureus USA100, USA200, USA300, USA400, and USA600 Clonal Lineages. mSphere, 1(3), . https://doi.org/10.1128/mSphere.00071-16
King, Jessica M, et al. "Phenotypes and Virulence among Staphylococcus aureus USA100, USA200, USA300, USA400, and USA600 Clonal Lineages." mSphere vol. 1,3 (2016). doi: https://doi.org/10.1128/mSphere.00071-16
King JM, Kulhankova K, Stach CS, Vu BG, Salgado-Pabón W. Phenotypes and Virulence among Staphylococcus aureus USA100, USA200, USA300, USA400, and USA600 Clonal Lineages. mSphere. 2016 Jun 08;1(3). doi: 10.1128/mSphere.00071-16. eCollection 2016. PMID: 27303750; PMCID: PMC4899884.
Copy Download .nbib Format: NLM
The Antistaphylococcal Activity of Citropin 1.1 and Temporin A against Planktonic Cells and Biofilms Formed by Isolates from Patients with Atopic Dermatitis: An Assessment of Their Potential to Induce Microbial Resistance Compared to Conventional Antimicrobials.

Pharmaceuticals (Basel, Switzerland)

Dawgul M, Baranska-Rybak W, Piechowicz L, Bauer M, Neubauer D, Nowicki R, Kamysz W.
PMID: 27231918
Pharmaceuticals (Basel). 2016 May 25;9(2). doi: 10.3390/ph9020030.

Staphylococcus aureus (SA) colonizes the vast majority of patients with atopic dermatitis (AD). Its resistance to antibiotics and ability to form biofilms are the main origins of therapeutic complications. Endogenous antimicrobial peptides (AMPs) exhibit strong activity against SA, including...

Cite
Dawgul M, Baranska-Rybak W, Piechowicz L, et al. The Antistaphylococcal Activity of Citropin 1.1 and Temporin A against Planktonic Cells and Biofilms Formed by Isolates from Patients with Atopic Dermatitis: An Assessment of Their Potential to Induce Microbial Resistance Compared to Conventional Antimicrobials. Pharmaceuticals (Basel). 2016;9(2)doi: 10.3390/ph9020030.
Dawgul, M., Baranska-Rybak, W., Piechowicz, L., Bauer, M., Neubauer, D., Nowicki, R., & Kamysz, W. (2016). The Antistaphylococcal Activity of Citropin 1.1 and Temporin A against Planktonic Cells and Biofilms Formed by Isolates from Patients with Atopic Dermatitis: An Assessment of Their Potential to Induce Microbial Resistance Compared to Conventional Antimicrobials. Pharmaceuticals (Basel, Switzerland), 9(2), . https://doi.org/10.3390/ph9020030
Dawgul, Malgorzata, et al. "The Antistaphylococcal Activity of Citropin 1.1 and Temporin A against Planktonic Cells and Biofilms Formed by Isolates from Patients with Atopic Dermatitis: An Assessment of Their Potential to Induce Microbial Resistance Compared to Conventional Antimicrobials." Pharmaceuticals (Basel, Switzerland) vol. 9,2 (2016). doi: https://doi.org/10.3390/ph9020030
Dawgul M, Baranska-Rybak W, Piechowicz L, Bauer M, Neubauer D, Nowicki R, Kamysz W. The Antistaphylococcal Activity of Citropin 1.1 and Temporin A against Planktonic Cells and Biofilms Formed by Isolates from Patients with Atopic Dermatitis: An Assessment of Their Potential to Induce Microbial Resistance Compared to Conventional Antimicrobials. Pharmaceuticals (Basel). 2016 May 25;9(2). doi: 10.3390/ph9020030. PMID: 27231918; PMCID: PMC4932548.
Copy Download .nbib Format: NLM
Association Between PTPN22 Polymorphisms and IgE Responses to Staphylococcal Superantigens in Chronic Urticaria.

Allergy, asthma & immunology research

Palikhe S, Kim SH, Pham le D, Ye YM, Park HS.
PMID: 25749762
Allergy Asthma Immunol Res. 2015 May;7(3):290-4. doi: 10.4168/aair.2015.7.3.290. Epub 2014 Oct 15.

Protein tyrosine phosphatase-22 (PTPN22) gene encodes lymphoid-specific tyrosine phosphatase (Lyp), an inhibitor of T cell activation. A polymorphism of the PTPN22 gene has been found to be associated with chronic urticaria (CU). We investigated the associations between PTPN22 gene...

Cite
Palikhe S, Kim SH, Pham le D, et al. Association Between PTPN22 Polymorphisms and IgE Responses to Staphylococcal Superantigens in Chronic Urticaria. Allergy Asthma Immunol Res. 2014;7(3):290-4doi: 10.4168/aair.2015.7.3.290.
Palikhe, S., Kim, S. H., Pham, l. e. . D., Ye, Y. M., & Park, H. S. (2015). Association Between PTPN22 Polymorphisms and IgE Responses to Staphylococcal Superantigens in Chronic Urticaria. Allergy, asthma & immunology research, 7(3), 290-4. https://doi.org/10.4168/aair.2015.7.3.290
Palikhe, Sailesh, et al. "Association Between PTPN22 Polymorphisms and IgE Responses to Staphylococcal Superantigens in Chronic Urticaria." Allergy, asthma & immunology research vol. 7,3 (2015): 290-4. doi: https://doi.org/10.4168/aair.2015.7.3.290
Palikhe S, Kim SH, Pham le D, Ye YM, Park HS. Association Between PTPN22 Polymorphisms and IgE Responses to Staphylococcal Superantigens in Chronic Urticaria. Allergy Asthma Immunol Res. 2015 May;7(3):290-4. doi: 10.4168/aair.2015.7.3.290. Epub 2014 Oct 15. PMID: 25749762; PMCID: PMC4397370.
Copy Download .nbib Format: NLM
Overexpression of Metastatic Related MicroRNAs, Mir-335 and Mir-10b, by Staphylococcal Enterotoxin B in the Metastatic Breast Cancer Cell Line.

Advanced pharmaceutical bulletin

Heidary MF, Mahmoodzadeh Hosseini H, Mehdizadeh Aghdam E, Nourani MR, Ranjbar R, Mirnejad R, Imani Fooladi AA.
PMID: 26236665
Adv Pharm Bull. 2015 Jun;5(2):255-9. doi: 10.15171/apb.2015.035. Epub 2015 Jun 01.

PURPOSE: One of the advanced cancer therapy strategies is immune-stimulating compound based immunotherapy Staphylococcal enterotoxin B (SEB) is one of the potent superantigens, which can efficiently activate antitumor immune response to eradicate tumor growth and inhibit metastasis. Herein, we...

Cite
Heidary MF, Mahmoodzadeh Hosseini H, Mehdizadeh Aghdam E, et al. Overexpression of Metastatic Related MicroRNAs, Mir-335 and Mir-10b, by Staphylococcal Enterotoxin B in the Metastatic Breast Cancer Cell Line. Adv Pharm Bull. 2015;5(2):255-9doi: 10.15171/apb.2015.035.
Heidary, M. F., Mahmoodzadeh Hosseini, H., Mehdizadeh Aghdam, E., Nourani, M. R., Ranjbar, R., Mirnejad, R., & Imani Fooladi, A. A. (2015). Overexpression of Metastatic Related MicroRNAs, Mir-335 and Mir-10b, by Staphylococcal Enterotoxin B in the Metastatic Breast Cancer Cell Line. Advanced pharmaceutical bulletin, 5(2), 255-9. https://doi.org/10.15171/apb.2015.035
Heidary, Mohammad Foad, et al. "Overexpression of Metastatic Related MicroRNAs, Mir-335 and Mir-10b, by Staphylococcal Enterotoxin B in the Metastatic Breast Cancer Cell Line." Advanced pharmaceutical bulletin vol. 5,2 (2015): 255-9. doi: https://doi.org/10.15171/apb.2015.035
Heidary MF, Mahmoodzadeh Hosseini H, Mehdizadeh Aghdam E, Nourani MR, Ranjbar R, Mirnejad R, Imani Fooladi AA. Overexpression of Metastatic Related MicroRNAs, Mir-335 and Mir-10b, by Staphylococcal Enterotoxin B in the Metastatic Breast Cancer Cell Line. Adv Pharm Bull. 2015 Jun;5(2):255-9. doi: 10.15171/apb.2015.035. Epub 2015 Jun 01. PMID: 26236665; PMCID: PMC4517087.
Copy Download .nbib Format: NLM
Central paraneoplastic neurological syndromes. Part 2. Practical, theoretical and mechanistic considerations of treatment with drugs and biologicals.

Drugs of today (Barcelona, Spain : 1998)

Pranzatelli MR.
PMID: 15010712
Drugs Today (Barc). 1998 Jun;34(6):507-23. doi: 10.1358/dot.1998.34.6.485248.

New information about paraneoplastic neurological syndromes, or the remote effects of cancer, has raised more questions than answers. The underlying mechanisms of brain injury remain to be elucidated. A brief review of autoimmunity provides a basis for exploring hypotheses...

Cite
Pranzatelli MR. Central paraneoplastic neurological syndromes. Part 2. Practical, theoretical and mechanistic considerations of treatment with drugs and biologicals. Drugs Today (Barc). 1998;34(6):507-23doi: 10.1358/dot.1998.34.6.485248.
Pranzatelli, M. R. (1998). Central paraneoplastic neurological syndromes. Part 2. Practical, theoretical and mechanistic considerations of treatment with drugs and biologicals. Drugs of today (Barcelona, Spain : 1998), 34(6), 507-23. https://doi.org/10.1358/dot.1998.34.6.485248
Pranzatelli, M R. "Central paraneoplastic neurological syndromes. Part 2. Practical, theoretical and mechanistic considerations of treatment with drugs and biologicals." Drugs of today (Barcelona, Spain : 1998) vol. 34,6 (1998): 507-23. doi: https://doi.org/10.1358/dot.1998.34.6.485248
Pranzatelli MR. Central paraneoplastic neurological syndromes. Part 2. Practical, theoretical and mechanistic considerations of treatment with drugs and biologicals. Drugs Today (Barc). 1998 Jun;34(6):507-23. doi: 10.1358/dot.1998.34.6.485248. PMID: 15010712.
Copy Download .nbib Format: NLM
Therapeutic Approaches to Streptococcal Toxic Shock Syndrome.

Current infectious disease reports

Baracco GJ, Bisno AL.
PMID: 11095793
Curr Infect Dis Rep. 1999 Aug;1(3):230-237. doi: 10.1007/s11908-999-0024-4.

The streptococcal toxic shock syndrome (STSS) is a severe, life-threatening condition characterized by hypotension and multiorgan system dysfunction associated with infection by the group A Streptococcus (GAS) or rarely by streptococci of other Lancefield serogroups. It is associated with...

Cite
Baracco GJ, Bisno AL. Therapeutic Approaches to Streptococcal Toxic Shock Syndrome. Curr Infect Dis Rep. 1999;1(3):230-237doi: 10.1007/s11908-999-0024-4.
Baracco, G. J., & Bisno, A. L. (1999). Therapeutic Approaches to Streptococcal Toxic Shock Syndrome. Current infectious disease reports, 1(3), 230-237. https://doi.org/10.1007/s11908-999-0024-4
Baracco, and Bisno. "Therapeutic Approaches to Streptococcal Toxic Shock Syndrome." Current infectious disease reports vol. 1,3 (1999): 230-237. doi: https://doi.org/10.1007/s11908-999-0024-4
Baracco GJ, Bisno AL. Therapeutic Approaches to Streptococcal Toxic Shock Syndrome. Curr Infect Dis Rep. 1999 Aug;1(3):230-237. doi: 10.1007/s11908-999-0024-4. PMID: 11095793.
Copy Download .nbib Format: NLM
Superantigens and autoimmune disease: are they involved?.

Current opinion in infectious diseases

Sawitzke AD, Mu HH, Cole BC.
PMID: 17035782
Curr Opin Infect Dis. 1999 Jun;12(3):213-9. doi: 10.1097/00001432-199906000-00010.

In over 10 years since the definition of superantigens, much has been learned about host cell-superantigen interactions. The initial simple set of rules used to define these interactions has given way to a more complex system, in which the...

Cite
Sawitzke AD, Mu HH, Cole BC. Superantigens and autoimmune disease: are they involved?. Curr Opin Infect Dis. 1999;12(3):213-9doi: 10.1097/00001432-199906000-00010.
Sawitzke, A. D., Mu, H. H., & Cole, B. C. (1999). Superantigens and autoimmune disease: are they involved?. Current opinion in infectious diseases, 12(3), 213-9. https://doi.org/10.1097/00001432-199906000-00010
Sawitzke, A D, et al. "Superantigens and autoimmune disease: are they involved?." Current opinion in infectious diseases vol. 12,3 (1999): 213-9. doi: https://doi.org/10.1097/00001432-199906000-00010
Sawitzke AD, Mu HH, Cole BC. Superantigens and autoimmune disease: are they involved?. Curr Opin Infect Dis. 1999 Jun;12(3):213-9. doi: 10.1097/00001432-199906000-00010. PMID: 17035782.
Copy Download .nbib Format: NLM
Overexpressed Bcl-x(L) prevents bacterial superantigen-induced apoptosis of thymocytes in vitro.

Cell death and differentiation

Takahashi T, Honda H, Hirai H, Tsujimoto Y.
PMID: 16465222
Cell Death Differ. 1997 Feb;4(2):159-65. doi: 10.1038/sj.cdd.4400214.

bcl-x, a homologous gene of bcl-2, has an anti-apoptotic function and appears to play a critical role in the development of lymphoid systems. To investigate the effect of overexpressed Bcl-x(L) on the development of T lymphocytes, we established two...

Cite
Takahashi T, Honda H, Hirai H, et al. Overexpressed Bcl-x(L) prevents bacterial superantigen-induced apoptosis of thymocytes in vitro. Cell Death Differ. 1997;4(2):159-65doi: 10.1038/sj.cdd.4400214.
Takahashi, T., Honda, H., Hirai, H., & Tsujimoto, Y. (1997). Overexpressed Bcl-x(L) prevents bacterial superantigen-induced apoptosis of thymocytes in vitro. Cell death and differentiation, 4(2), 159-65. https://doi.org/10.1038/sj.cdd.4400214
Takahashi, T, et al. "Overexpressed Bcl-x(L) prevents bacterial superantigen-induced apoptosis of thymocytes in vitro." Cell death and differentiation vol. 4,2 (1997): 159-65. doi: https://doi.org/10.1038/sj.cdd.4400214
Takahashi T, Honda H, Hirai H, Tsujimoto Y. Overexpressed Bcl-x(L) prevents bacterial superantigen-induced apoptosis of thymocytes in vitro. Cell Death Differ. 1997 Feb;4(2):159-65. doi: 10.1038/sj.cdd.4400214. PMID: 16465222.
Copy Download .nbib Format: NLM
HLA allele associations and V-beta T-lymphocyte expansions in patients with psoriasis, harboring toxin-producing Staphylococcus aureus.

Journal of biomedicine & biotechnology

Ajib R, Janbazian L, Rahal E, Matar GM, Zaynoun S, Kibbi AG, Abdelnoor AM.
PMID: 16489264
J Biomed Biotechnol. 2005;2005(4):310-5. doi: 10.1155/JBB.2005.310.

HLA alleles have been associated with psoriasis. Toxin-producing strains of Staphylococcus aureus behave as superantigens, and if present in patients, might play a role in the exacerbation of psoriatic lesions by activating certain V-beta (V beta) T-lymphocyte subsets. Allele...

Cite
Ajib R, Janbazian L, Rahal E, et al. HLA allele associations and V-beta T-lymphocyte expansions in patients with psoriasis, harboring toxin-producing Staphylococcus aureus. J Biomed Biotechnol. 2005;2005(4):310-5doi: 10.1155/JBB.2005.310.
Ajib, R., Janbazian, L., Rahal, E., Matar, G. M., Zaynoun, S., Kibbi, A. G., & Abdelnoor, A. M. (2005). HLA allele associations and V-beta T-lymphocyte expansions in patients with psoriasis, harboring toxin-producing Staphylococcus aureus. Journal of biomedicine & biotechnology, 2005(4), 310-5. https://doi.org/10.1155/JBB.2005.310
Ajib, Rola, et al. "HLA allele associations and V-beta T-lymphocyte expansions in patients with psoriasis, harboring toxin-producing Staphylococcus aureus." Journal of biomedicine & biotechnology vol. 2005,4 (2005): 310-5. doi: https://doi.org/10.1155/JBB.2005.310
Ajib R, Janbazian L, Rahal E, Matar GM, Zaynoun S, Kibbi AG, Abdelnoor AM. HLA allele associations and V-beta T-lymphocyte expansions in patients with psoriasis, harboring toxin-producing Staphylococcus aureus. J Biomed Biotechnol. 2005;2005(4):310-5. doi: 10.1155/JBB.2005.310. PMID: 16489264; PMCID: PMC1361490.
Copy Download .nbib Format: NLM
Searching the Staphylococcal Superantigens: Enterotoxins A, B, C, and TSST1 in Synovial Fluid of Cases With Negative Culture Inflammatory Arthritis.

Jundishapur journal of microbiology

Shokrollahi MR, Noorbakhsh S, Aliakbari M, Tabatabaei A.
PMID: 25368802
Jundishapur J Microbiol. 2014 Jul;7(7):e11647. doi: 10.5812/jjm.11647. Epub 2014 Jul 01.

BACKGROUND: Accurate and rapid diagnosis of bacterial arthritis is not always possible in unvaccinated (Streptococcus pneumoniae and Haemophilus influenzae type B) children in Iran.OBJECTIVES: Searching the staphylococcal superantigen (entrotoxin A, B, C and TSST1) in synovial fluid of cases...

Cite
Shokrollahi MR, Noorbakhsh S, Aliakbari M, et al. Searching the Staphylococcal Superantigens: Enterotoxins A, B, C, and TSST1 in Synovial Fluid of Cases With Negative Culture Inflammatory Arthritis. Jundishapur J Microbiol. 2014;7(7):e11647doi: 10.5812/jjm.11647.
Shokrollahi, M. R., Noorbakhsh, S., Aliakbari, M., & Tabatabaei, A. (2014). Searching the Staphylococcal Superantigens: Enterotoxins A, B, C, and TSST1 in Synovial Fluid of Cases With Negative Culture Inflammatory Arthritis. Jundishapur journal of microbiology, 7(7), e11647. https://doi.org/10.5812/jjm.11647
Shokrollahi, Mohammad Reza, et al. "Searching the Staphylococcal Superantigens: Enterotoxins A, B, C, and TSST1 in Synovial Fluid of Cases With Negative Culture Inflammatory Arthritis." Jundishapur journal of microbiology vol. 7,7 (2014): e11647. doi: https://doi.org/10.5812/jjm.11647
Shokrollahi MR, Noorbakhsh S, Aliakbari M, Tabatabaei A. Searching the Staphylococcal Superantigens: Enterotoxins A, B, C, and TSST1 in Synovial Fluid of Cases With Negative Culture Inflammatory Arthritis. Jundishapur J Microbiol. 2014 Jul;7(7):e11647. doi: 10.5812/jjm.11647. Epub 2014 Jul 01. PMID: 25368802; PMCID: PMC4216582.
Copy Download .nbib Format: NLM
Superantigen involvement and susceptibility factors in Kawasaki disease: profiles of TCR Vβ2+ T cells and HLA-DRB1, TNF-α and ITPKC genes among Filipino patients.

International journal of molecular epidemiology and genetics

Natividad MF, Torres-Villanueva CA, Saloma CP.
PMID: 23565324
Int J Mol Epidemiol Genet. 2013;4(1):70-6. Epub 2013 Mar 18.

Superantigens and genetic factors may play roles in the etiology and susceptibility to Kawasaki disease (KD). To investigate these roles, percentages of TCR-Vβ2+ T cells were compared by flow cytometry using anti-Vβ2 monoclonal antibodies and genotyping was done on...

Cite
Natividad MF, Torres-Villanueva CA, Saloma CP. Superantigen involvement and susceptibility factors in Kawasaki disease: profiles of TCR Vβ2+ T cells and HLA-DRB1, TNF-α and ITPKC genes among Filipino patients. Int J Mol Epidemiol Genet. 2013;4(1):70-6
Natividad, M. F., Torres-Villanueva, C. A., & Saloma, C. P. (2013). Superantigen involvement and susceptibility factors in Kawasaki disease: profiles of TCR Vβ2+ T cells and HLA-DRB1, TNF-α and ITPKC genes among Filipino patients. International journal of molecular epidemiology and genetics, 4(1), 70-6.
Natividad, Magdalena F, et al. "Superantigen involvement and susceptibility factors in Kawasaki disease: profiles of TCR Vβ2+ T cells and HLA-DRB1, TNF-α and ITPKC genes among Filipino patients." International journal of molecular epidemiology and genetics vol. 4,1 (2013): 70-6.
Natividad MF, Torres-Villanueva CA, Saloma CP. Superantigen involvement and susceptibility factors in Kawasaki disease: profiles of TCR Vβ2+ T cells and HLA-DRB1, TNF-α and ITPKC genes among Filipino patients. Int J Mol Epidemiol Genet. 2013;4(1):70-6. Epub 2013 Mar 18. PMID: 23565324; PMCID: PMC3612456.
Copy Download .nbib Format: NLM
Serum capacity to neutralize superantigens does not affect the outcome of Staphylococcus aureus bacteremia.

European journal of clinical microbiology & infectious diseases : official publication of the European Society of Clinical Microbiology

Yi J, Park JS, Hong KH, Lee SH, Kim EC.
PMID: 22290348
Eur J Clin Microbiol Infect Dis. 2012 Jan 31; doi: 10.1007/s10096-011-1541-2. Epub 2012 Jan 31.

Staphylococcal superantigens (SAg) could play an important role in sepsis by activating numerous T cells. We investigated whether serum capacity to neutralize SAgs can be a prognostic factor in Staphylococcus aureus bacteremia (SAB). In a university hospital, 105 consecutive...

Cite
Yi J, Park JS, Hong KH, et al. Serum capacity to neutralize superantigens does not affect the outcome of Staphylococcus aureus bacteremia. Eur J Clin Microbiol Infect Dis. 2012;doi: 10.1007/s10096-011-1541-2.
Yi, J., Park, J. S., Hong, K. H., Lee, S. H., & Kim, E. C. (2012). Serum capacity to neutralize superantigens does not affect the outcome of Staphylococcus aureus bacteremia. European journal of clinical microbiology & infectious diseases : official publication of the European Society of Clinical Microbiology, . https://doi.org/10.1007/s10096-011-1541-2
Yi, J, et al. "Serum capacity to neutralize superantigens does not affect the outcome of Staphylococcus aureus bacteremia." European journal of clinical microbiology & infectious diseases : official publication of the European Society of Clinical Microbiology vol. (2012). doi: https://doi.org/10.1007/s10096-011-1541-2
Yi J, Park JS, Hong KH, Lee SH, Kim EC. Serum capacity to neutralize superantigens does not affect the outcome of Staphylococcus aureus bacteremia. Eur J Clin Microbiol Infect Dis. 2012 Jan 31; doi: 10.1007/s10096-011-1541-2. Epub 2012 Jan 31. PMID: 22290348.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 112 entries