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Showing 1 to 12 of 36 entries
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Cumulative neonatal oxygen exposure predicts response of adult mice infected with influenza A virus.

Pediatric pulmonology

Maduekwe ET, Buczynski BW, Yee M, Rangasamy T, Stevens TP, Lawrence BP, O'Reilly MA.
PMID: 24850805
Pediatr Pulmonol. 2015 Mar;50(3):222-230. doi: 10.1002/ppul.23063. Epub 2014 May 22.

An acceptable level of oxygen exposure in preterm infants that maximizes efficacy and minimizes harm has yet to be determined. Quantifying oxygen exposure as an area-under-the curve (O

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Maduekwe ET, Buczynski BW, Yee M, et al. Cumulative neonatal oxygen exposure predicts response of adult mice infected with influenza A virus. Pediatr Pulmonol. 2014;50(3):222-230doi: 10.1002/ppul.23063.
Maduekwe, E. T., Buczynski, B. W., Yee, M., Rangasamy, T., Stevens, T. P., Lawrence, B. P., & O'Reilly, M. A. (2015). Cumulative neonatal oxygen exposure predicts response of adult mice infected with influenza A virus. Pediatric pulmonology, 50(3), 222-230. https://doi.org/10.1002/ppul.23063
Maduekwe, Echezona T, et al. "Cumulative neonatal oxygen exposure predicts response of adult mice infected with influenza A virus." Pediatric pulmonology vol. 50,3 (2015): 222-230. doi: https://doi.org/10.1002/ppul.23063
Maduekwe ET, Buczynski BW, Yee M, Rangasamy T, Stevens TP, Lawrence BP, O'Reilly MA. Cumulative neonatal oxygen exposure predicts response of adult mice infected with influenza A virus. Pediatr Pulmonol. 2015 Mar;50(3):222-230. doi: 10.1002/ppul.23063. Epub 2014 May 22. PMID: 24850805; PMCID: PMC4334747.
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PPARγ and the Innate Immune System Mediate the Resolution of Inflammation.

PPAR research

Croasdell A, Duffney PF, Kim N, Lacy SH, Sime PJ, Phipps RP.
PMID: 26713087
PPAR Res. 2015;2015:549691. doi: 10.1155/2015/549691. Epub 2015 Dec 02.

The resolution of inflammation is an active and dynamic process, mediated in large part by the innate immune system. Resolution represents not only an increase in anti-inflammatory actions, but also a paradigm shift in immune cell function to restore...

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Croasdell A, Duffney PF, Kim N, et al. PPARγ and the Innate Immune System Mediate the Resolution of Inflammation. PPAR Res. 2015;2015:549691doi: 10.1155/2015/549691.
Croasdell, A., Duffney, P. F., Kim, N., Lacy, S. H., Sime, P. J., & Phipps, R. P. (2015). PPARγ and the Innate Immune System Mediate the Resolution of Inflammation. PPAR research, 2015549691. https://doi.org/10.1155/2015/549691
Croasdell, Amanda, et al. "PPARγ and the Innate Immune System Mediate the Resolution of Inflammation." PPAR research vol. 2015 (2015): 549691. doi: https://doi.org/10.1155/2015/549691
Croasdell A, Duffney PF, Kim N, Lacy SH, Sime PJ, Phipps RP. PPARγ and the Innate Immune System Mediate the Resolution of Inflammation. PPAR Res. 2015;2015:549691. doi: 10.1155/2015/549691. Epub 2015 Dec 02. PMID: 26713087; PMCID: PMC4680113.
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The PPAR-Platelet Connection: Modulators of Inflammation and Potential Cardiovascular Effects.

PPAR research

Spinelli SL, O'Brien JJ, Bancos S, Lehmann GM, Springer DL, Blumberg N, Francis CW, Taubman MB, Phipps RP.
PMID: 18288284
PPAR Res. 2008;2008:328172. doi: 10.1155/2008/328172.

Historically, platelets were viewed as simple anucleate cells responsible for initiating thrombosis and maintaining hemostasis, but clearly they are also key mediators of inflammation and immune cell activation. An emerging body of evidence links platelet function and thrombosis to...

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Spinelli SL, O'Brien JJ, Bancos S, et al. The PPAR-Platelet Connection: Modulators of Inflammation and Potential Cardiovascular Effects. PPAR Res. 2008;2008:328172doi: 10.1155/2008/328172.
Spinelli, S. L., O'Brien, J. J., Bancos, S., Lehmann, G. M., Springer, D. L., Blumberg, N., Francis, C. W., Taubman, M. B., & Phipps, R. P. (2008). The PPAR-Platelet Connection: Modulators of Inflammation and Potential Cardiovascular Effects. PPAR research, 2008328172. https://doi.org/10.1155/2008/328172
Spinelli, S L, et al. "The PPAR-Platelet Connection: Modulators of Inflammation and Potential Cardiovascular Effects." PPAR research vol. 2008 (2008): 328172. doi: https://doi.org/10.1155/2008/328172
Spinelli SL, O'Brien JJ, Bancos S, Lehmann GM, Springer DL, Blumberg N, Francis CW, Taubman MB, Phipps RP. The PPAR-Platelet Connection: Modulators of Inflammation and Potential Cardiovascular Effects. PPAR Res. 2008;2008:328172. doi: 10.1155/2008/328172. PMID: 18288284; PMCID: PMC2233896.
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Inflammatory and Oxidative Responses Induced by Exposure to Commonly Used e-Cigarette Flavoring Chemicals and Flavored e-Liquids without Nicotine.

Frontiers in physiology

Muthumalage T, Prinz M, Ansah KO, Gerloff J, Sundar IK, Rahman I.
PMID: 29375399
Front Physiol. 2018 Jan 11;8:1130. doi: 10.3389/fphys.2017.01130. eCollection 2017.

No abstract available.

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Muthumalage T, Prinz M, Ansah KO, et al. Inflammatory and Oxidative Responses Induced by Exposure to Commonly Used e-Cigarette Flavoring Chemicals and Flavored e-Liquids without Nicotine. Front Physiol. 2018;8:1130doi: 10.3389/fphys.2017.01130.
Muthumalage, T., Prinz, M., Ansah, K. O., Gerloff, J., Sundar, I. K., & Rahman, I. (2017). Inflammatory and Oxidative Responses Induced by Exposure to Commonly Used e-Cigarette Flavoring Chemicals and Flavored e-Liquids without Nicotine. Frontiers in physiology, 81130. https://doi.org/10.3389/fphys.2017.01130
Muthumalage, Thivanka, et al. "Inflammatory and Oxidative Responses Induced by Exposure to Commonly Used e-Cigarette Flavoring Chemicals and Flavored e-Liquids without Nicotine." Frontiers in physiology vol. 8 (2017): 1130. doi: https://doi.org/10.3389/fphys.2017.01130
Muthumalage T, Prinz M, Ansah KO, Gerloff J, Sundar IK, Rahman I. Inflammatory and Oxidative Responses Induced by Exposure to Commonly Used e-Cigarette Flavoring Chemicals and Flavored e-Liquids without Nicotine. Front Physiol. 2018 Jan 11;8:1130. doi: 10.3389/fphys.2017.01130. eCollection 2017. PMID: 29375399; PMCID: PMC5768608.
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The Ancestral Environment Shapes Antiviral CD8.

iScience

Post CM, Boule LA, Burke CG, O'Dell CT, Winans B, Lawrence BP.
PMID: 31569050
iScience. 2019 Oct 25;20:168-183. doi: 10.1016/j.isci.2019.09.014. Epub 2019 Sep 14.

Recent studies have linked health fates of children to environmental exposures of their great grandparents. However, few studies have considered whether ancestral exposures influence immune function across generations. Here, we report transgenerational inheritance of altered T cell responses resulting...

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Post CM, Boule LA, Burke CG, et al. The Ancestral Environment Shapes Antiviral CD8. iScience. 2019;20:168-183doi: 10.1016/j.isci.2019.09.014.
Post, C. M., Boule, L. A., Burke, C. G., O'Dell, C. T., Winans, B., & Lawrence, B. P. (2019). The Ancestral Environment Shapes Antiviral CD8. iScience, 20168-183. https://doi.org/10.1016/j.isci.2019.09.014
Post, Christina M, et al. "The Ancestral Environment Shapes Antiviral CD8." iScience vol. 20 (2019): 168-183. doi: https://doi.org/10.1016/j.isci.2019.09.014
Post CM, Boule LA, Burke CG, O'Dell CT, Winans B, Lawrence BP. The Ancestral Environment Shapes Antiviral CD8. iScience. 2019 Oct 25;20:168-183. doi: 10.1016/j.isci.2019.09.014. Epub 2019 Sep 14. PMID: 31569050; PMCID: PMC6817732.
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Current Perspectives on Characteristics, Compositions, and Toxicological Effects of E-Cigarettes Containing Tobacco and Menthol/Mint Flavors.

Frontiers in physiology

Kaur G, Gaurav A, Lamb T, Perkins M, Muthumalage T, Rahman I.
PMID: 33329065
Front Physiol. 2020 Nov 19;11:613948. doi: 10.3389/fphys.2020.613948. eCollection 2020.

Electronic nicotine delivery systems/devices (ENDS) such as electronic cigarettes (e-cigarettes) have been made available globally, with the intent to reduce tobacco smoking. To make these products more appealing to young adults, many brands have added flavoring agents. However, these...

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Kaur G, Gaurav A, Lamb T, et al. Current Perspectives on Characteristics, Compositions, and Toxicological Effects of E-Cigarettes Containing Tobacco and Menthol/Mint Flavors. Front Physiol. 2020;11:613948doi: 10.3389/fphys.2020.613948.
Kaur, G., Gaurav, A., Lamb, T., Perkins, M., Muthumalage, T., & Rahman, I. (2020). Current Perspectives on Characteristics, Compositions, and Toxicological Effects of E-Cigarettes Containing Tobacco and Menthol/Mint Flavors. Frontiers in physiology, 11613948. https://doi.org/10.3389/fphys.2020.613948
Kaur, Gurjot, et al. "Current Perspectives on Characteristics, Compositions, and Toxicological Effects of E-Cigarettes Containing Tobacco and Menthol/Mint Flavors." Frontiers in physiology vol. 11 (2020): 613948. doi: https://doi.org/10.3389/fphys.2020.613948
Kaur G, Gaurav A, Lamb T, Perkins M, Muthumalage T, Rahman I. Current Perspectives on Characteristics, Compositions, and Toxicological Effects of E-Cigarettes Containing Tobacco and Menthol/Mint Flavors. Front Physiol. 2020 Nov 19;11:613948. doi: 10.3389/fphys.2020.613948. eCollection 2020. PMID: 33329065; PMCID: PMC7710937.
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E-Liquid Containing a Mixture of Coconut, Vanilla, and Cookie Flavors Causes Cellular Senescence and Dysregulated Repair in Pulmonary Fibroblasts: Implications on Premature Aging.

Frontiers in physiology

Lucas JH, Muthumalage T, Wang Q, Friedman MR, Friedman AE, Rahman I.
PMID: 33013432
Front Physiol. 2020 Sep 04;11:924. doi: 10.3389/fphys.2020.00924. eCollection 2020.

Electronic cigarette (e-cig) usage has risen dramatically worldwide over the past decade. While they are touted as a safe alternative to cigarettes, recent studies indicate that high levels of nicotine and flavoring chemicals present in e-cigs may still cause...

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Lucas JH, Muthumalage T, Wang Q, et al. E-Liquid Containing a Mixture of Coconut, Vanilla, and Cookie Flavors Causes Cellular Senescence and Dysregulated Repair in Pulmonary Fibroblasts: Implications on Premature Aging. Front Physiol. 2020;11:924doi: 10.3389/fphys.2020.00924.
Lucas, J. H., Muthumalage, T., Wang, Q., Friedman, M. R., Friedman, A. E., & Rahman, I. (2020). E-Liquid Containing a Mixture of Coconut, Vanilla, and Cookie Flavors Causes Cellular Senescence and Dysregulated Repair in Pulmonary Fibroblasts: Implications on Premature Aging. Frontiers in physiology, 11924. https://doi.org/10.3389/fphys.2020.00924
Lucas, Joseph H, et al. "E-Liquid Containing a Mixture of Coconut, Vanilla, and Cookie Flavors Causes Cellular Senescence and Dysregulated Repair in Pulmonary Fibroblasts: Implications on Premature Aging." Frontiers in physiology vol. 11 (2020): 924. doi: https://doi.org/10.3389/fphys.2020.00924
Lucas JH, Muthumalage T, Wang Q, Friedman MR, Friedman AE, Rahman I. E-Liquid Containing a Mixture of Coconut, Vanilla, and Cookie Flavors Causes Cellular Senescence and Dysregulated Repair in Pulmonary Fibroblasts: Implications on Premature Aging. Front Physiol. 2020 Sep 04;11:924. doi: 10.3389/fphys.2020.00924. eCollection 2020. PMID: 33013432; PMCID: PMC7500211.
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Effects of Chlorpyrifos on Cholinesterase and Serine Lipase Activities and Lipid Metabolism in Brains of Rainbow Trout (Oncorhynchus mykiss).

Toxicological sciences : an official journal of the Society of Toxicology

Greer JB, Magnuson JT, Hester K, Giroux M, Pope C, Anderson T, Liu J, Dang V, Denslow ND, Schlenk D.
PMID: 31359069
Toxicol Sci. 2019 Jul 30; doi: 10.1093/toxsci/kfz167. Epub 2019 Jul 30.

Chlorpyrifos is an organophosphorus insecticide that elicits acute toxicity through inhibition of acetylcholinesterase (AChE), leading to acetylcholine accumulation and prolonged stimulation of cholinergic receptors throughout the central and peripheral nervous systems. Previous studies have indicated that neurodevelopment may also...

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Greer JB, Magnuson JT, Hester K, et al. Effects of Chlorpyrifos on Cholinesterase and Serine Lipase Activities and Lipid Metabolism in Brains of Rainbow Trout (Oncorhynchus mykiss). Toxicol Sci. 2019;doi: 10.1093/toxsci/kfz167.
Greer, J. B., Magnuson, J. T., Hester, K., Giroux, M., Pope, C., Anderson, T., Liu, J., Dang, V., Denslow, N. D., & Schlenk, D. (2019). Effects of Chlorpyrifos on Cholinesterase and Serine Lipase Activities and Lipid Metabolism in Brains of Rainbow Trout (Oncorhynchus mykiss). Toxicological sciences : an official journal of the Society of Toxicology, . https://doi.org/10.1093/toxsci/kfz167
Greer, J B, et al. "Effects of Chlorpyrifos on Cholinesterase and Serine Lipase Activities and Lipid Metabolism in Brains of Rainbow Trout (Oncorhynchus mykiss)." Toxicological sciences : an official journal of the Society of Toxicology vol. (2019). doi: https://doi.org/10.1093/toxsci/kfz167
Greer JB, Magnuson JT, Hester K, Giroux M, Pope C, Anderson T, Liu J, Dang V, Denslow ND, Schlenk D. Effects of Chlorpyrifos on Cholinesterase and Serine Lipase Activities and Lipid Metabolism in Brains of Rainbow Trout (Oncorhynchus mykiss). Toxicol Sci. 2019 Jul 30; doi: 10.1093/toxsci/kfz167. Epub 2019 Jul 30. PMID: 31359069; PMCID: PMC6813751.
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Exposure to a mixture of 23 chemicals associated with unconventional oil and gas operations alters immune response to challenge in adult mice.

Journal of immunotoxicology

O'Dell CT, Boule LA, Robert J, Georas SN, Eliseeva S, Lawrence BP.
PMID: 34455897
J Immunotoxicol. 2021 Dec;18(1):105-117. doi: 10.1080/1547691X.2021.1965677.

The prevalence of unconventional oil and gas (UOG) operations raises concerns regarding the potential for adverse health outcomes following exposure to water tainted by mixtures of UOG associated chemicals. The potential effects that exposure to complex chemical mixtures has...

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O'Dell CT, Boule LA, Robert J, et al. Exposure to a mixture of 23 chemicals associated with unconventional oil and gas operations alters immune response to challenge in adult mice. J Immunotoxicol. 2021;18(1):105-117doi: 10.1080/1547691X.2021.1965677.
O'Dell, C. T., Boule, L. A., Robert, J., Georas, S. N., Eliseeva, S., & Lawrence, B. P. (2021). Exposure to a mixture of 23 chemicals associated with unconventional oil and gas operations alters immune response to challenge in adult mice. Journal of immunotoxicology, 18(1), 105-117. https://doi.org/10.1080/1547691X.2021.1965677
O'Dell, Colleen T, et al. "Exposure to a mixture of 23 chemicals associated with unconventional oil and gas operations alters immune response to challenge in adult mice." Journal of immunotoxicology vol. 18,1 (2021): 105-117. doi: https://doi.org/10.1080/1547691X.2021.1965677
O'Dell CT, Boule LA, Robert J, Georas SN, Eliseeva S, Lawrence BP. Exposure to a mixture of 23 chemicals associated with unconventional oil and gas operations alters immune response to challenge in adult mice. J Immunotoxicol. 2021 Dec;18(1):105-117. doi: 10.1080/1547691X.2021.1965677. PMID: 34455897.
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The Aryl Hydrocarbon Receptor Modulates Murine Hematopoietic Stem Cell Homeostasis and Influences Lineage-Biased Stem and Progenitor Cells.

Stem cells and development

Vaughan KL, Franchini AM, Kern HG, Lawrence BP.
PMID: 34428990
Stem Cells Dev. 2021 Oct 01;30(19):970-980. doi: 10.1089/scd.2021.0096. Epub 2021 Sep 20.

The core function of hematopoietic stem and progenitor cells (HSPCs) is to provide lifelong production of all lineages of the blood and immune cells. The mechanisms that modulate HSPC homeostasis and lineage biasing are not fully understood. Growing evidence...

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Vaughan KL, Franchini AM, Kern HG, et al. The Aryl Hydrocarbon Receptor Modulates Murine Hematopoietic Stem Cell Homeostasis and Influences Lineage-Biased Stem and Progenitor Cells. Stem Cells Dev. 2021;30(19):970-980doi: 10.1089/scd.2021.0096.
Vaughan, K. L., Franchini, A. M., Kern, H. G., & Lawrence, B. P. (2021). The Aryl Hydrocarbon Receptor Modulates Murine Hematopoietic Stem Cell Homeostasis and Influences Lineage-Biased Stem and Progenitor Cells. Stem cells and development, 30(19), 970-980. https://doi.org/10.1089/scd.2021.0096
Vaughan, Keegan L, et al. "The Aryl Hydrocarbon Receptor Modulates Murine Hematopoietic Stem Cell Homeostasis and Influences Lineage-Biased Stem and Progenitor Cells." Stem cells and development vol. 30,19 (2021): 970-980. doi: https://doi.org/10.1089/scd.2021.0096
Vaughan KL, Franchini AM, Kern HG, Lawrence BP. The Aryl Hydrocarbon Receptor Modulates Murine Hematopoietic Stem Cell Homeostasis and Influences Lineage-Biased Stem and Progenitor Cells. Stem Cells Dev. 2021 Oct 01;30(19):970-980. doi: 10.1089/scd.2021.0096. Epub 2021 Sep 20. PMID: 34428990.
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Proteolytic regulation of synaptic plasticity in the mouse primary visual cortex: analysis of matrix metalloproteinase 9 deficient mice.

Frontiers in cellular neuroscience

Kelly EA, Russo AS, Jackson CD, Lamantia CE, Majewska AK.
PMID: 26441540
Front Cell Neurosci. 2015 Sep 22;9:369. doi: 10.3389/fncel.2015.00369. eCollection 2015.

The extracellular matrix (ECM) is known to play important roles in regulating neuronal recovery from injury. The ECM can also impact physiological synaptic plasticity, although this process is less well understood. To understand the impact of the ECM on...

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Kelly EA, Russo AS, Jackson CD, et al. Proteolytic regulation of synaptic plasticity in the mouse primary visual cortex: analysis of matrix metalloproteinase 9 deficient mice. Front Cell Neurosci. 2015;9:369doi: 10.3389/fncel.2015.00369.
Kelly, E. A., Russo, A. S., Jackson, C. D., Lamantia, C. E., & Majewska, A. K. (2015). Proteolytic regulation of synaptic plasticity in the mouse primary visual cortex: analysis of matrix metalloproteinase 9 deficient mice. Frontiers in cellular neuroscience, 9369. https://doi.org/10.3389/fncel.2015.00369
Kelly, Emily A, et al. "Proteolytic regulation of synaptic plasticity in the mouse primary visual cortex: analysis of matrix metalloproteinase 9 deficient mice." Frontiers in cellular neuroscience vol. 9 (2015): 369. doi: https://doi.org/10.3389/fncel.2015.00369
Kelly EA, Russo AS, Jackson CD, Lamantia CE, Majewska AK. Proteolytic regulation of synaptic plasticity in the mouse primary visual cortex: analysis of matrix metalloproteinase 9 deficient mice. Front Cell Neurosci. 2015 Sep 22;9:369. doi: 10.3389/fncel.2015.00369. eCollection 2015. PMID: 26441540; PMCID: PMC4585116.
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Emerging PPARγ-Independent Role of PPARγ Ligands in Lung Diseases.

PPAR research

Kulkarni AA, Woeller CF, Thatcher TH, Ramon S, Phipps RP, Sime PJ.
PMID: 22778711
PPAR Res. 2012;2012:705352. doi: 10.1155/2012/705352. Epub 2012 Jun 18.

Peroxisome proliferator activated receptor (PPAR)-γ is a nuclear hormone receptor that is activated by multiple agonists including thiazolidinediones, prostaglandins, and synthetic oleanolic acids. Many PPARγ ligands are under investigation as potential therapies for human diseases. These ligands modulate multiple...

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Kulkarni AA, Woeller CF, Thatcher TH, et al. Emerging PPARγ-Independent Role of PPARγ Ligands in Lung Diseases. PPAR Res. 2012;2012:705352doi: 10.1155/2012/705352.
Kulkarni, A. A., Woeller, C. F., Thatcher, T. H., Ramon, S., Phipps, R. P., & Sime, P. J. (2012). Emerging PPARγ-Independent Role of PPARγ Ligands in Lung Diseases. PPAR research, 2012705352. https://doi.org/10.1155/2012/705352
Kulkarni, Ajit A, et al. "Emerging PPARγ-Independent Role of PPARγ Ligands in Lung Diseases." PPAR research vol. 2012 (2012): 705352. doi: https://doi.org/10.1155/2012/705352
Kulkarni AA, Woeller CF, Thatcher TH, Ramon S, Phipps RP, Sime PJ. Emerging PPARγ-Independent Role of PPARγ Ligands in Lung Diseases. PPAR Res. 2012;2012:705352. doi: 10.1155/2012/705352. Epub 2012 Jun 18. PMID: 22778711; PMCID: PMC3385049.
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Showing 1 to 12 of 36 entries