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Showing 1 to 11 of 11 entries
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Arl13b regulates Shh signaling from both inside and outside the cilium.

Molecular biology of the cell

Mariani LE, Bijlsma MF, Ivanova AA, Suciu SK, Kahn RA, Caspary T.
PMID: 27682584
Mol Biol Cell. 2016 Sep 28; doi: 10.1091/mbc.E16-03-0189. Epub 2016 Sep 28.

The regulatory GTPase Arl13b localizes to primary cilia, where it regulates Sonic hedgehog (Shh) signaling. Missense mutations in ARL13B can cause the ciliopathy Joubert syndrome, while the mouse null allele is embryonic lethal. We used mouse embryonic fibroblasts as...

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Mariani LE, Bijlsma MF, Ivanova AA, et al. Arl13b regulates Shh signaling from both inside and outside the cilium. Mol Biol Cell. 2016;doi: 10.1091/mbc.E16-03-0189.
Mariani, L. E., Bijlsma, M. F., Ivanova, A. A., Suciu, S. K., Kahn, R. A., & Caspary, T. (2016). Arl13b regulates Shh signaling from both inside and outside the cilium. Molecular biology of the cell, . https://doi.org/10.1091/mbc.E16-03-0189
Mariani, Laura E, et al. "Arl13b regulates Shh signaling from both inside and outside the cilium." Molecular biology of the cell vol. (2016). doi: https://doi.org/10.1091/mbc.E16-03-0189
Mariani LE, Bijlsma MF, Ivanova AA, Suciu SK, Kahn RA, Caspary T. Arl13b regulates Shh signaling from both inside and outside the cilium. Mol Biol Cell. 2016 Sep 28; doi: 10.1091/mbc.E16-03-0189. Epub 2016 Sep 28. PMID: 27682584; PMCID: PMC5170560.
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Neurobiological mechanisms of social attachment and pair bonding.

Current opinion in behavioral sciences

Johnson ZV, Young LJ.
PMID: 26146650
Curr Opin Behav Sci. 2015 Jun;3:38-44. doi: 10.1016/j.cobeha.2015.01.009.

Species have evolved diverse social behavior and mating strategies in response to selective forces in their environments. While promiscuity is the predominant mating strategy across most vertebrate taxa, convergent evolution of monogamous mating systems has occurred multiple times across...

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Johnson ZV, Young LJ. Neurobiological mechanisms of social attachment and pair bonding. Curr Opin Behav Sci. 2015;3:38-44doi: 10.1016/j.cobeha.2015.01.009.
Johnson, Z. V., & Young, L. J. (2015). Neurobiological mechanisms of social attachment and pair bonding. Current opinion in behavioral sciences, 338-44. https://doi.org/10.1016/j.cobeha.2015.01.009
Johnson, Zachary V, and Young, Larry J. "Neurobiological mechanisms of social attachment and pair bonding." Current opinion in behavioral sciences vol. 3 (2015): 38-44. doi: https://doi.org/10.1016/j.cobeha.2015.01.009
Johnson ZV, Young LJ. Neurobiological mechanisms of social attachment and pair bonding. Curr Opin Behav Sci. 2015 Jun;3:38-44. doi: 10.1016/j.cobeha.2015.01.009. PMID: 26146650; PMCID: PMC4486624.
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What's That (Blue) Spot on my MRI? Multimodal Neuroimaging of the Locus Coeruleus in Neurodegenerative Disease.

Frontiers in neuroscience

Kelberman M, Keilholz S, Weinshenker D.
PMID: 33122996
Front Neurosci. 2020 Oct 06;14:583421. doi: 10.3389/fnins.2020.583421. eCollection 2020.

The locus coeruleus (LC) has long been underappreciated for its role in the pathophysiology of Alzheimer's disease (AD), Parkinson's disease (PD), and other neurodegenerative disorders. While AD and PD are distinct in clinical presentation, both are characterized by prodromal...

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Kelberman M, Keilholz S, Weinshenker D. What's That (Blue) Spot on my MRI? Multimodal Neuroimaging of the Locus Coeruleus in Neurodegenerative Disease. Front Neurosci. 2020;14:583421doi: 10.3389/fnins.2020.583421.
Kelberman, M., Keilholz, S., & Weinshenker, D. (2020). What's That (Blue) Spot on my MRI? Multimodal Neuroimaging of the Locus Coeruleus in Neurodegenerative Disease. Frontiers in neuroscience, 14583421. https://doi.org/10.3389/fnins.2020.583421
Kelberman, Michael, et al. "What's That (Blue) Spot on my MRI? Multimodal Neuroimaging of the Locus Coeruleus in Neurodegenerative Disease." Frontiers in neuroscience vol. 14 (2020): 583421. doi: https://doi.org/10.3389/fnins.2020.583421
Kelberman M, Keilholz S, Weinshenker D. What's That (Blue) Spot on my MRI? Multimodal Neuroimaging of the Locus Coeruleus in Neurodegenerative Disease. Front Neurosci. 2020 Oct 06;14:583421. doi: 10.3389/fnins.2020.583421. eCollection 2020. PMID: 33122996; PMCID: PMC7573566.
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Sciatic Nerve Cut and Repair Using Fibrin Glue in Adult Mice.

Bio-protocol

Akhter ET, Rotterman TM, English AW, Alvarez FJ.
PMID: 31788507
Bio Protoc. 2019 Sep 20;9(18). doi: 10.21769/BioProtoc.3363.

Peripheral nerve injury (PNI) is an excellent model for studying neural responses to injury and elucidating the mechanisms that can facilitate axon regeneration. As such, several animal models have been employed to study regenerative mechanisms after PNI, including Aplysia,...

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Akhter ET, Rotterman TM, English AW, et al. Sciatic Nerve Cut and Repair Using Fibrin Glue in Adult Mice. Bio Protoc. 2019;9(18)doi: 10.21769/BioProtoc.3363.
Akhter, E. T., Rotterman, T. M., English, A. W., & Alvarez, F. J. (2019). Sciatic Nerve Cut and Repair Using Fibrin Glue in Adult Mice. Bio-protocol, 9(18), . https://doi.org/10.21769/BioProtoc.3363
Akhter, Erica T, et al. "Sciatic Nerve Cut and Repair Using Fibrin Glue in Adult Mice." Bio-protocol vol. 9,18 (2019). doi: https://doi.org/10.21769/BioProtoc.3363
Akhter ET, Rotterman TM, English AW, Alvarez FJ. Sciatic Nerve Cut and Repair Using Fibrin Glue in Adult Mice. Bio Protoc. 2019 Sep 20;9(18). doi: 10.21769/BioProtoc.3363. PMID: 31788507; PMCID: PMC6884152.
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In Reply.

Anesthesiology

Safavynia SA, Keating G, Speigel I, Fidler JA, Kreuzer M, Rye DB, Jenkins A, García PS.
PMID: 28098616
Anesthesiology. 2017 Feb;126(2):352-353. doi: 10.1097/ALN.0000000000001446.

No abstract available.

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Safavynia SA, Keating G, Speigel I, et al. In Reply. Anesthesiology. 2017;126(2):352-353doi: 10.1097/ALN.0000000000001446.
Safavynia, S. A., Keating, G., Speigel, I., Fidler, J. A., Kreuzer, M., Rye, D. B., Jenkins, A., & García, P. S. (2017). In Reply. Anesthesiology, 126(2), 352-353. https://doi.org/10.1097/ALN.0000000000001446
Safavynia, Seyed A, et al. "In Reply." Anesthesiology vol. 126,2 (2017): 352-353. doi: https://doi.org/10.1097/ALN.0000000000001446
Safavynia SA, Keating G, Speigel I, Fidler JA, Kreuzer M, Rye DB, Jenkins A, García PS. In Reply. Anesthesiology. 2017 Feb;126(2):352-353. doi: 10.1097/ALN.0000000000001446. PMID: 28098616; PMCID: PMC5340312.
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Transplantation of iPS cell-derived neural progenitors overexpressing SDF-1α increases regeneration and functional recovery after ischemic stroke.

Oncotarget

Chau M, Deveau TC, Song M, Wei ZZ, Gu X, Yu SP, Wei L.
PMID: 29228630
Oncotarget. 2017 Oct 31;8(57):97537-97553. doi: 10.18632/oncotarget.22180. eCollection 2017 Nov 14.

Ischemic stroke is a leading cause of human death and disability while clinical treatments are limited. The adult brain possesses endogenous regenerative activities that may benefit tissue repair after stroke. Trophic factors such as stromal cell-derived factor 1 alpha...

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Chau M, Deveau TC, Song M, et al. Transplantation of iPS cell-derived neural progenitors overexpressing SDF-1α increases regeneration and functional recovery after ischemic stroke. Oncotarget. 2017;8(57):97537-97553doi: 10.18632/oncotarget.22180.
Chau, M., Deveau, T. C., Song, M., Wei, Z. Z., Gu, X., Yu, S. P., & Wei, L. (2017). Transplantation of iPS cell-derived neural progenitors overexpressing SDF-1α increases regeneration and functional recovery after ischemic stroke. Oncotarget, 8(57), 97537-97553. https://doi.org/10.18632/oncotarget.22180
Chau, Monica, et al. "Transplantation of iPS cell-derived neural progenitors overexpressing SDF-1α increases regeneration and functional recovery after ischemic stroke." Oncotarget vol. 8,57 (2017): 97537-97553. doi: https://doi.org/10.18632/oncotarget.22180
Chau M, Deveau TC, Song M, Wei ZZ, Gu X, Yu SP, Wei L. Transplantation of iPS cell-derived neural progenitors overexpressing SDF-1α increases regeneration and functional recovery after ischemic stroke. Oncotarget. 2017 Oct 31;8(57):97537-97553. doi: 10.18632/oncotarget.22180. eCollection 2017 Nov 14. PMID: 29228630; PMCID: PMC5722582.
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A commentary on the utility of a new L-DOPA-responsive dystonia mouse model.

Rare diseases (Austin, Tex.)

Rose SJ, Hess EJ.
PMID: 27141408
Rare Dis. 2015 Dec 29;4(1):e1128617. doi: 10.1080/21675511.2015.1128617. eCollection 2016.

In a recent issue of Brain, we reported on the generation and characterization of a mouse model of the rare disease L-DOPA-responsive dystonia (DRD). Here, we discuss the utility of these mice for understanding broader disease processes and treatment...

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Rose SJ, Hess EJ. A commentary on the utility of a new L-DOPA-responsive dystonia mouse model. Rare Dis. 2015;4(1):e1128617doi: 10.1080/21675511.2015.1128617.
Rose, S. J., & Hess, E. J. (2016). A commentary on the utility of a new L-DOPA-responsive dystonia mouse model. Rare diseases (Austin, Tex.), 4(1), e1128617. https://doi.org/10.1080/21675511.2015.1128617
Rose, Samuel J, and Hess, Ellen J. "A commentary on the utility of a new L-DOPA-responsive dystonia mouse model." Rare diseases (Austin, Tex.) vol. 4,1 (2016): e1128617. doi: https://doi.org/10.1080/21675511.2015.1128617
Rose SJ, Hess EJ. A commentary on the utility of a new L-DOPA-responsive dystonia mouse model. Rare Dis. 2015 Dec 29;4(1):e1128617. doi: 10.1080/21675511.2015.1128617. eCollection 2016. PMID: 27141408; PMCID: PMC4838313.
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Alzheimer's pathology causes impaired inhibitory connections and reactivation of spatial codes during spatial navigation.

Cell reports

Prince SM, Paulson AL, Jeong N, Zhang L, Amigues S, Singer AC.
PMID: 33882308
Cell Rep. 2021 Apr 20;35(3):109008. doi: 10.1016/j.celrep.2021.109008.

Synapse loss and altered synaptic strength are thought to underlie cognitive impairment in Alzheimer's disease (AD) by disrupting neural activity essential for memory. While synaptic dysfunction in AD has been well characterized in anesthetized animals and in vitro, it...

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Prince SM, Paulson AL, Jeong N, et al. Alzheimer's pathology causes impaired inhibitory connections and reactivation of spatial codes during spatial navigation. Cell Rep. 2021;35(3):109008doi: 10.1016/j.celrep.2021.109008.
Prince, S. M., Paulson, A. L., Jeong, N., Zhang, L., Amigues, S., & Singer, A. C. (2021). Alzheimer's pathology causes impaired inhibitory connections and reactivation of spatial codes during spatial navigation. Cell reports, 35(3), 109008. https://doi.org/10.1016/j.celrep.2021.109008
Prince, Stephanie M, et al. "Alzheimer's pathology causes impaired inhibitory connections and reactivation of spatial codes during spatial navigation." Cell reports vol. 35,3 (2021): 109008. doi: https://doi.org/10.1016/j.celrep.2021.109008
Prince SM, Paulson AL, Jeong N, Zhang L, Amigues S, Singer AC. Alzheimer's pathology causes impaired inhibitory connections and reactivation of spatial codes during spatial navigation. Cell Rep. 2021 Apr 20;35(3):109008. doi: 10.1016/j.celrep.2021.109008. PMID: 33882308; PMCID: PMC8139125.
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The Role of BDNF in Peripheral Nerve Regeneration: Activity-Dependent Treatments and Val66Met.

Frontiers in cellular neuroscience

McGregor CE, English AW.
PMID: 30687012
Front Cell Neurosci. 2019 Jan 11;12:522. doi: 10.3389/fncel.2018.00522. eCollection 2018.

Despite the ability of peripheral nerves to spontaneously regenerate after injury, recovery is generally very poor. The neurotrophins have emerged as an important modulator of axon regeneration, particularly brain derived neurotrophic factor (BDNF). BDNF regulation and signaling, as well...

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McGregor CE, English AW. The Role of BDNF in Peripheral Nerve Regeneration: Activity-Dependent Treatments and Val66Met. Front Cell Neurosci. 2019;12:522doi: 10.3389/fncel.2018.00522.
McGregor, C. E., & English, A. W. (2018). The Role of BDNF in Peripheral Nerve Regeneration: Activity-Dependent Treatments and Val66Met. Frontiers in cellular neuroscience, 12522. https://doi.org/10.3389/fncel.2018.00522
McGregor, Claire Emma, and English, Arthur W. "The Role of BDNF in Peripheral Nerve Regeneration: Activity-Dependent Treatments and Val66Met." Frontiers in cellular neuroscience vol. 12 (2018): 522. doi: https://doi.org/10.3389/fncel.2018.00522
McGregor CE, English AW. The Role of BDNF in Peripheral Nerve Regeneration: Activity-Dependent Treatments and Val66Met. Front Cell Neurosci. 2019 Jan 11;12:522. doi: 10.3389/fncel.2018.00522. eCollection 2018. PMID: 30687012; PMCID: PMC6336700.
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Extracellular vesicles in the treatment of neurological disorders.

Neurobiology of disease

Reed SL, Escayg A.
PMID: 34271084
Neurobiol Dis. 2021 Sep;157:105445. doi: 10.1016/j.nbd.2021.105445. Epub 2021 Jul 14.

Extracellular vesicles (EVs) are small, cell-derived membranous particles containing various nucleic acids, proteins, and lipids that play essential roles in intercellular communication. Evidence indicating that part of the regenerative benefit from stem cell therapy arises through EVs released from...

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Reed SL, Escayg A. Extracellular vesicles in the treatment of neurological disorders. Neurobiol Dis. 2021;157:105445doi: 10.1016/j.nbd.2021.105445.
Reed, S. L., & Escayg, A. (2021). Extracellular vesicles in the treatment of neurological disorders. Neurobiology of disease, 157105445. https://doi.org/10.1016/j.nbd.2021.105445
Reed, Samantha L, and Escayg, Andrew. "Extracellular vesicles in the treatment of neurological disorders." Neurobiology of disease vol. 157 (2021): 105445. doi: https://doi.org/10.1016/j.nbd.2021.105445
Reed SL, Escayg A. Extracellular vesicles in the treatment of neurological disorders. Neurobiol Dis. 2021 Sep;157:105445. doi: 10.1016/j.nbd.2021.105445. Epub 2021 Jul 14. PMID: 34271084.
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microRNAs Sculpt Neuronal Communication in a Tight Balance That Is Lost in Neurological Disease.

Frontiers in molecular neuroscience

Thomas KT, Gross C, Bassell GJ.
PMID: 30618607
Front Mol Neurosci. 2018 Dec 12;11:455. doi: 10.3389/fnmol.2018.00455. eCollection 2018.

Since the discovery of the first microRNA 25 years ago, microRNAs (miRNAs) have emerged as critical regulators of gene expression within the mammalian brain. miRNAs are small non-coding RNAs that direct the RNA induced silencing complex to complementary sites...

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Thomas KT, Gross C, Bassell GJ. microRNAs Sculpt Neuronal Communication in a Tight Balance That Is Lost in Neurological Disease. Front Mol Neurosci. 2018;11:455doi: 10.3389/fnmol.2018.00455.
Thomas, K. T., Gross, C., & Bassell, G. J. (2018). microRNAs Sculpt Neuronal Communication in a Tight Balance That Is Lost in Neurological Disease. Frontiers in molecular neuroscience, 11455. https://doi.org/10.3389/fnmol.2018.00455
Thomas, Kristen T, et al. "microRNAs Sculpt Neuronal Communication in a Tight Balance That Is Lost in Neurological Disease." Frontiers in molecular neuroscience vol. 11 (2018): 455. doi: https://doi.org/10.3389/fnmol.2018.00455
Thomas KT, Gross C, Bassell GJ. microRNAs Sculpt Neuronal Communication in a Tight Balance That Is Lost in Neurological Disease. Front Mol Neurosci. 2018 Dec 12;11:455. doi: 10.3389/fnmol.2018.00455. eCollection 2018. PMID: 30618607; PMCID: PMC6299112.
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Showing 1 to 11 of 11 entries