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Tight junction physiology of pleural mesothelium.

Frontiers in physiology

Markov AG, Amasheh S.
PMID: 25009499
Front Physiol. 2014 Jun 24;5:221. doi: 10.3389/fphys.2014.00221. eCollection 2014.

Pleura consists of visceral and parietal cell layers, producing a fluid, which is necessary for lubrication of the pleural space. Function of both mesothelial cell layers is necessary for the regulation of a constant pleural fluid volume and composition...

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Markov AG, Amasheh S. Tight junction physiology of pleural mesothelium. Front Physiol. 2014;5:221doi: 10.3389/fphys.2014.00221.
Markov, A. G., & Amasheh, S. (2014). Tight junction physiology of pleural mesothelium. Frontiers in physiology, 5221. https://doi.org/10.3389/fphys.2014.00221
Markov, Alexander G, and Amasheh, Salah. "Tight junction physiology of pleural mesothelium." Frontiers in physiology vol. 5 (2014): 221. doi: https://doi.org/10.3389/fphys.2014.00221
Markov AG, Amasheh S. Tight junction physiology of pleural mesothelium. Front Physiol. 2014 Jun 24;5:221. doi: 10.3389/fphys.2014.00221. eCollection 2014. PMID: 25009499; PMCID: PMC4067758.
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Mouse Adenovirus Type 1 Early Region 1A Effects on the Blood-Brain Barrier.

mSphere

Tirumuru N, Pretto CD, Castro Jorge LA, Spindler KR.
PMID: 27303733
mSphere. 2016 Apr 20;1(2). doi: 10.1128/mSphere.00079-16. eCollection 2016.

Mouse adenovirus type 1 (MAV-1) infects endothelial cells and disrupts the blood-brain barrier (BBB), causing encephalitis in inbred and outbred mice. Using a virus mutant that does not produce the early region 1A protein E1A, we investigated whether the...

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Tirumuru N, Pretto CD, Castro Jorge LA, et al. Mouse Adenovirus Type 1 Early Region 1A Effects on the Blood-Brain Barrier. mSphere. 2016;1(2)doi: 10.1128/mSphere.00079-16.
Tirumuru, N., Pretto, C. D., Castro Jorge, L. A., & Spindler, K. R. (2016). Mouse Adenovirus Type 1 Early Region 1A Effects on the Blood-Brain Barrier. mSphere, 1(2), . https://doi.org/10.1128/mSphere.00079-16
Tirumuru, Nagaraja, et al. "Mouse Adenovirus Type 1 Early Region 1A Effects on the Blood-Brain Barrier." mSphere vol. 1,2 (2016). doi: https://doi.org/10.1128/mSphere.00079-16
Tirumuru N, Pretto CD, Castro Jorge LA, Spindler KR. Mouse Adenovirus Type 1 Early Region 1A Effects on the Blood-Brain Barrier. mSphere. 2016 Apr 20;1(2). doi: 10.1128/mSphere.00079-16. eCollection 2016. PMID: 27303733; PMCID: PMC4894691.
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Soft TCPTP Agonism-Novel Target to Rescue Airway Epithelial Integrity by Exogenous Spermidine.

Frontiers in pharmacology

Ghisalberti CA, Borzì RM, Cetrullo S, Flamigni F, Cairo G.
PMID: 27375482
Front Pharmacol. 2016 Jun 03;7:147. doi: 10.3389/fphar.2016.00147. eCollection 2016.

A reparative approach of disrupted epithelium in obstructive airway diseases, namely asthma and chronic obstructive pulmonary disease (COPD), may afford protection and long-lasting results compared to conventional therapies, e.g., corticosteroids or immunosuppressant drugs. Here, we propose the polyamine spermidine...

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Ghisalberti CA, Borzì RM, Cetrullo S, et al. Soft TCPTP Agonism-Novel Target to Rescue Airway Epithelial Integrity by Exogenous Spermidine. Front Pharmacol. 2016;7:147doi: 10.3389/fphar.2016.00147.
Ghisalberti, C. A., Borzì, R. M., Cetrullo, S., Flamigni, F., & Cairo, G. (2016). Soft TCPTP Agonism-Novel Target to Rescue Airway Epithelial Integrity by Exogenous Spermidine. Frontiers in pharmacology, 7147. https://doi.org/10.3389/fphar.2016.00147
Ghisalberti, Carlo A, et al. "Soft TCPTP Agonism-Novel Target to Rescue Airway Epithelial Integrity by Exogenous Spermidine." Frontiers in pharmacology vol. 7 (2016): 147. doi: https://doi.org/10.3389/fphar.2016.00147
Ghisalberti CA, Borzì RM, Cetrullo S, Flamigni F, Cairo G. Soft TCPTP Agonism-Novel Target to Rescue Airway Epithelial Integrity by Exogenous Spermidine. Front Pharmacol. 2016 Jun 03;7:147. doi: 10.3389/fphar.2016.00147. eCollection 2016. PMID: 27375482; PMCID: PMC4892113.
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Effect of Polysaccharides from Acanthopanax senticosus on Intestinal Mucosal Barrier of Escherichia coli Lipopolysaccharide Challenged Mice.

Asian-Australasian journal of animal sciences

Han J, Xu Y, Yang D, Yu N, Bai Z, Bian L.
PMID: 26732337
Asian-Australas J Anim Sci. 2016 Jan;29(1):134-41. doi: 10.5713/ajas.15.0534.

To investigate the role of polysaccharide from Acanthopanax senticosus (ASPS) in preventing lipopolysaccharide (LPS)-induced intestinal injury, 18 mice (at 5 wk of age) were assigned to three groups with 6 replicates of one mouse each. Mice were administrated by...

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Han J, Xu Y, Yang D, et al. Effect of Polysaccharides from Acanthopanax senticosus on Intestinal Mucosal Barrier of Escherichia coli Lipopolysaccharide Challenged Mice. Asian-Australas J Anim Sci. 2016;29(1):134-41doi: 10.5713/ajas.15.0534.
Han, J., Xu, Y., Yang, D., Yu, N., Bai, Z., & Bian, L. (2016). Effect of Polysaccharides from Acanthopanax senticosus on Intestinal Mucosal Barrier of Escherichia coli Lipopolysaccharide Challenged Mice. Asian-Australasian journal of animal sciences, 29(1), 134-41. https://doi.org/10.5713/ajas.15.0534
Han, Jie, et al. "Effect of Polysaccharides from Acanthopanax senticosus on Intestinal Mucosal Barrier of Escherichia coli Lipopolysaccharide Challenged Mice." Asian-Australasian journal of animal sciences vol. 29,1 (2016): 134-41. doi: https://doi.org/10.5713/ajas.15.0534
Han J, Xu Y, Yang D, Yu N, Bai Z, Bian L. Effect of Polysaccharides from Acanthopanax senticosus on Intestinal Mucosal Barrier of Escherichia coli Lipopolysaccharide Challenged Mice. Asian-Australas J Anim Sci. 2016 Jan;29(1):134-41. doi: 10.5713/ajas.15.0534. PMID: 26732337; PMCID: PMC4698680.
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Entry and Release of Hepatitis C Virus in Polarized Human Hepatocytes.

Journal of virology

Belouzard S, Danneels A, Fénéant L, Séron K, Rouillé Y, Dubuisson J.
PMID: 28659476
J Virol. 2017 Aug 24;91(18). doi: 10.1128/JVI.00478-17. Print 2017 Sep 15.

Hepatitis C virus (HCV) primarily infects hepatocytes, which are highly polarized cells. The relevance of cell polarity in the HCV life cycle has been addressed only in distantly related models and remains poorly understood. Although polarized epithelial cells have...

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Belouzard S, Danneels A, Fénéant L, et al. Entry and Release of Hepatitis C Virus in Polarized Human Hepatocytes. J Virol. 2017;91(18)doi: 10.1128/JVI.00478-17.
Belouzard, S., Danneels, A., Fénéant, L., Séron, K., Rouillé, Y., & Dubuisson, J. (2017). Entry and Release of Hepatitis C Virus in Polarized Human Hepatocytes. Journal of virology, 91(18), . https://doi.org/10.1128/JVI.00478-17
Belouzard, Sandrine, et al. "Entry and Release of Hepatitis C Virus in Polarized Human Hepatocytes." Journal of virology vol. 91,18 (2017). doi: https://doi.org/10.1128/JVI.00478-17
Belouzard S, Danneels A, Fénéant L, Séron K, Rouillé Y, Dubuisson J. Entry and Release of Hepatitis C Virus in Polarized Human Hepatocytes. J Virol. 2017 Aug 24;91(18). doi: 10.1128/JVI.00478-17. Print 2017 Sep 15. PMID: 28659476; PMCID: PMC5571255.
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Celiac Disease: Role of the Epithelial Barrier.

Cellular and molecular gastroenterology and hepatology

Schumann M, Siegmund B, Schulzke JD, Fromm M.
PMID: 28275682
Cell Mol Gastroenterol Hepatol. 2017 Jan 14;3(2):150-162. doi: 10.1016/j.jcmgh.2016.12.006. eCollection 2017 Mar.

In celiac disease (CD) a T-cell-mediated response to gluten is mounted in genetically predisposed individuals, resulting in a malabsorptive enteropathy histologically highlighted by villous atrophy and crypt hyperplasia. Recent data point to the epithelial layer as an under-rated hot...

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Schumann M, Siegmund B, Schulzke JD, et al. Celiac Disease: Role of the Epithelial Barrier. Cell Mol Gastroenterol Hepatol. 2017;3(2):150-162doi: 10.1016/j.jcmgh.2016.12.006.
Schumann, M., Siegmund, B., Schulzke, J. D., & Fromm, M. (2017). Celiac Disease: Role of the Epithelial Barrier. Cellular and molecular gastroenterology and hepatology, 3(2), 150-162. https://doi.org/10.1016/j.jcmgh.2016.12.006
Schumann, Michael, et al. "Celiac Disease: Role of the Epithelial Barrier." Cellular and molecular gastroenterology and hepatology vol. 3,2 (2017): 150-162. doi: https://doi.org/10.1016/j.jcmgh.2016.12.006
Schumann M, Siegmund B, Schulzke JD, Fromm M. Celiac Disease: Role of the Epithelial Barrier. Cell Mol Gastroenterol Hepatol. 2017 Jan 14;3(2):150-162. doi: 10.1016/j.jcmgh.2016.12.006. eCollection 2017 Mar. PMID: 28275682; PMCID: PMC5331784.
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Occludin expression in brain tumors and its relevance to peritumoral edema and survival.

Cancer research and treatment

Park MW, Kim CH, Cheong JH, Bak KH, Kim JM, Oh SJ.
PMID: 19771274
Cancer Res Treat. 2006;38(3):139-43. doi: 10.4143/crt.2006.38.3.139. Epub 2006 Sep 30.

PURPOSE: Peritumoral brain edema (PTBE) is a serious causative factor that contributes the morbidity or mortality of brain tumors. The development of PTBE is influenced by many factors, including such tight junction proteins as occludin. We evaluated the PTBE...

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Park MW, Kim CH, Cheong JH, et al. Occludin expression in brain tumors and its relevance to peritumoral edema and survival. Cancer Res Treat. 2006;38(3):139-43doi: 10.4143/crt.2006.38.3.139.
Park, M. W., Kim, C. H., Cheong, J. H., Bak, K. H., Kim, J. M., & Oh, S. J. (2006). Occludin expression in brain tumors and its relevance to peritumoral edema and survival. Cancer research and treatment, 38(3), 139-43. https://doi.org/10.4143/crt.2006.38.3.139
Park, Min-Woo, et al. "Occludin expression in brain tumors and its relevance to peritumoral edema and survival." Cancer research and treatment vol. 38,3 (2006): 139-43. doi: https://doi.org/10.4143/crt.2006.38.3.139
Park MW, Kim CH, Cheong JH, Bak KH, Kim JM, Oh SJ. Occludin expression in brain tumors and its relevance to peritumoral edema and survival. Cancer Res Treat. 2006;38(3):139-43. doi: 10.4143/crt.2006.38.3.139. Epub 2006 Sep 30. PMID: 19771274; PMCID: PMC2741679.
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The onset of enhanced intestinal permeability and food sensitivity triggered by medication used in dental procedures: a case report.

Case reports in gastrointestinal medicine

Vojdani A, Lambert J.
PMID: 23008786
Case Rep Gastrointest Med. 2012;2012:265052. doi: 10.1155/2012/265052. Epub 2012 Sep 12.

Enhanced intestinal permeability and food sensitivity are two of the many proven causes of gastrointestinal disorders. This present report describes a woman with no previous gastrointestinal (GI) complaints, who underwent dental root canal, bone graft, and implant procedures. Postsurgery...

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Vojdani A, Lambert J. The onset of enhanced intestinal permeability and food sensitivity triggered by medication used in dental procedures: a case report. Case Rep Gastrointest Med. 2012;2012:265052doi: 10.1155/2012/265052.
Vojdani, A., & Lambert, J. (2012). The onset of enhanced intestinal permeability and food sensitivity triggered by medication used in dental procedures: a case report. Case reports in gastrointestinal medicine, 2012265052. https://doi.org/10.1155/2012/265052
Vojdani, Aristo, and Lambert, Jama. "The onset of enhanced intestinal permeability and food sensitivity triggered by medication used in dental procedures: a case report." Case reports in gastrointestinal medicine vol. 2012 (2012): 265052. doi: https://doi.org/10.1155/2012/265052
Vojdani A, Lambert J. The onset of enhanced intestinal permeability and food sensitivity triggered by medication used in dental procedures: a case report. Case Rep Gastrointest Med. 2012;2012:265052. doi: 10.1155/2012/265052. Epub 2012 Sep 12. PMID: 23008786; PMCID: PMC3447324.
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Resolution of central nervous system astrocytic and endothelial sources of CCL2 gene expression during evolving neuroinflammation.

Fluids and barriers of the CNS

Shrestha B, Ge S, Pachter JS.
PMID: 24589378
Fluids Barriers CNS. 2014 Mar 04;11(1):6. doi: 10.1186/2045-8118-11-6.

BACKGROUND: The chemokine CCL2 is a critical mediator of neuroinflammation in diseases such as multiple sclerosis (MS) and its animal model, experimental autoimmune encephalomyelitis (EAE). CCL2 drives mononuclear cell infiltration into the central nervous system (CNS), alters expression and...

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Shrestha B, Ge S, Pachter JS. Resolution of central nervous system astrocytic and endothelial sources of CCL2 gene expression during evolving neuroinflammation. Fluids Barriers CNS. 2014;11(1):6doi: 10.1186/2045-8118-11-6.
Shrestha, B., Ge, S., & Pachter, J. S. (2014). Resolution of central nervous system astrocytic and endothelial sources of CCL2 gene expression during evolving neuroinflammation. Fluids and barriers of the CNS, 11(1), 6. https://doi.org/10.1186/2045-8118-11-6
Shrestha, Bandana, et al. "Resolution of central nervous system astrocytic and endothelial sources of CCL2 gene expression during evolving neuroinflammation." Fluids and barriers of the CNS vol. 11,1 (2014): 6. doi: https://doi.org/10.1186/2045-8118-11-6
Shrestha B, Ge S, Pachter JS. Resolution of central nervous system astrocytic and endothelial sources of CCL2 gene expression during evolving neuroinflammation. Fluids Barriers CNS. 2014 Mar 04;11(1):6. doi: 10.1186/2045-8118-11-6. PMID: 24589378; PMCID: PMC3944978.
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Disruption of blood-brain barrier in Alzheimer disease pathogenesis.

Tissue barriers

Kook SY, Seok Hong H, Moon M, Mook-Jung I.
PMID: 24665385
Tissue Barriers. 2013 Apr 01;1(2):e23993. doi: 10.4161/tisb.23993.

Blood-brain barrier (BBB) regulates transport of various molecules and maintains brain homeostasis. Perturbed intracellular Ca(2+) homeostasis and BBB damage have been implicated in the pathogenesis of Alzheimer disease (AD). Although receptor for advanced glycation end products (RAGE) is known...

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Kook SY, Seok Hong H, Moon M, et al. Disruption of blood-brain barrier in Alzheimer disease pathogenesis. Tissue Barriers. 2013;1(2):e23993doi: 10.4161/tisb.23993.
Kook, S. Y., Seok Hong, H., Moon, M., & Mook-Jung, I. (2013). Disruption of blood-brain barrier in Alzheimer disease pathogenesis. Tissue barriers, 1(2), e23993. https://doi.org/10.4161/tisb.23993
Kook, Sun-Young, et al. "Disruption of blood-brain barrier in Alzheimer disease pathogenesis." Tissue barriers vol. 1,2 (2013): e23993. doi: https://doi.org/10.4161/tisb.23993
Kook SY, Seok Hong H, Moon M, Mook-Jung I. Disruption of blood-brain barrier in Alzheimer disease pathogenesis. Tissue Barriers. 2013 Apr 01;1(2):e23993. doi: 10.4161/tisb.23993. PMID: 24665385; PMCID: PMC3887048.
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Qihuang decoction promotes the recovery of intestinal immune barrier dysfunction after gastrectomy in rats.

American journal of translational research

Peng H, Shen Y, Zhang Q, Liu J, Wang Z, Huang L, Zhou F, Yu J, Liu M, Yuan Y, Yu S, Yu Q.
PMID: 29636872
Am J Transl Res. 2018 Mar 15;10(3):827-836. eCollection 2018.

OBJECTIVE: This study aims to observe the effect of Qihuang decoction on small intestinal mucosal barrier after gastrectomy in rats.MATERIALS AND METHODS: A total of 80 Wistar rats were randomly divided into normal group, sham operation group, enteral nutrition...

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Peng H, Shen Y, Zhang Q, et al. Qihuang decoction promotes the recovery of intestinal immune barrier dysfunction after gastrectomy in rats. Am J Transl Res. 2018;10(3):827-836
Peng, H., Shen, Y., Zhang, Q., Liu, J., Wang, Z., Huang, L., Zhou, F., Yu, J., Liu, M., Yuan, Y., Yu, S., & Yu, Q. (2018). Qihuang decoction promotes the recovery of intestinal immune barrier dysfunction after gastrectomy in rats. American journal of translational research, 10(3), 827-836.
Peng, Hui, et al. "Qihuang decoction promotes the recovery of intestinal immune barrier dysfunction after gastrectomy in rats." American journal of translational research vol. 10,3 (2018): 827-836.
Peng H, Shen Y, Zhang Q, Liu J, Wang Z, Huang L, Zhou F, Yu J, Liu M, Yuan Y, Yu S, Yu Q. Qihuang decoction promotes the recovery of intestinal immune barrier dysfunction after gastrectomy in rats. Am J Transl Res. 2018 Mar 15;10(3):827-836. eCollection 2018. PMID: 29636872; PMCID: PMC5883123.
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Structural and Functional Alterations in the Microbial Community and Immunological Consequences in a Mouse Model of Antibiotic-Induced Dysbiosis.

Frontiers in microbiology

Shi Y, Kellingray L, Zhai Q, Gall GL, Narbad A, Zhao J, Zhang H, Chen W.
PMID: 30186263
Front Microbiol. 2018 Aug 21;9:1948. doi: 10.3389/fmicb.2018.01948. eCollection 2018.

The aim of this study was to establish continuous therapeutic-dose ampicillin (CTDA)-induced dysbiosis in a mouse model, mimicking typical adult exposure, with a view to using this to assess its impact on gut microbiota, intestinal metabolites and host immune...

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Shi Y, Kellingray L, Zhai Q, et al. Structural and Functional Alterations in the Microbial Community and Immunological Consequences in a Mouse Model of Antibiotic-Induced Dysbiosis. Front Microbiol. 2018;9:1948doi: 10.3389/fmicb.2018.01948.
Shi, Y., Kellingray, L., Zhai, Q., Gall, G. L., Narbad, A., Zhao, J., Zhang, H., & Chen, W. (2018). Structural and Functional Alterations in the Microbial Community and Immunological Consequences in a Mouse Model of Antibiotic-Induced Dysbiosis. Frontiers in microbiology, 91948. https://doi.org/10.3389/fmicb.2018.01948
Shi, Ying, et al. "Structural and Functional Alterations in the Microbial Community and Immunological Consequences in a Mouse Model of Antibiotic-Induced Dysbiosis." Frontiers in microbiology vol. 9 (2018): 1948. doi: https://doi.org/10.3389/fmicb.2018.01948
Shi Y, Kellingray L, Zhai Q, Gall GL, Narbad A, Zhao J, Zhang H, Chen W. Structural and Functional Alterations in the Microbial Community and Immunological Consequences in a Mouse Model of Antibiotic-Induced Dysbiosis. Front Microbiol. 2018 Aug 21;9:1948. doi: 10.3389/fmicb.2018.01948. eCollection 2018. PMID: 30186263; PMCID: PMC6110884.
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