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Showing 1 to 12 of 23 entries
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Methylglyoxal-Induced Retinal Angiogenesis in Zebrafish Embryo: A Potential Animal Model of Neovascular Retinopathy.

Journal of ophthalmology

Li Y, Zhao Y, Sang S, Leung T.
PMID: 31143470
J Ophthalmol. 2019 Apr 17;2019:2746735. doi: 10.1155/2019/2746735. eCollection 2019.

Methylglyoxal (MG) is an intermediate of glucose metabolism and the precursor of advanced glycation end products (AGEs) found in high levels in blood or tissue of diabetic patients. MG and AGEs are thought to play a major role in...

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Li Y, Zhao Y, Sang S, et al. Methylglyoxal-Induced Retinal Angiogenesis in Zebrafish Embryo: A Potential Animal Model of Neovascular Retinopathy. J Ophthalmol. 2019;2019:2746735doi: 10.1155/2019/2746735.
Li, Y., Zhao, Y., Sang, S., & Leung, T. (2019). Methylglyoxal-Induced Retinal Angiogenesis in Zebrafish Embryo: A Potential Animal Model of Neovascular Retinopathy. Journal of ophthalmology, 20192746735. https://doi.org/10.1155/2019/2746735
Li, Ying, et al. "Methylglyoxal-Induced Retinal Angiogenesis in Zebrafish Embryo: A Potential Animal Model of Neovascular Retinopathy." Journal of ophthalmology vol. 2019 (2019): 2746735. doi: https://doi.org/10.1155/2019/2746735
Li Y, Zhao Y, Sang S, Leung T. Methylglyoxal-Induced Retinal Angiogenesis in Zebrafish Embryo: A Potential Animal Model of Neovascular Retinopathy. J Ophthalmol. 2019 Apr 17;2019:2746735. doi: 10.1155/2019/2746735. eCollection 2019. PMID: 31143470; PMCID: PMC6501125.
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TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study.

Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology

Hurson AN, Abubakar M, Hamilton AM, Conway K, Hoadley KA, Love MI, Olshan AF, Perou CM, Garcia-Closas M, Troester MA.
PMID: 34737209
Cancer Epidemiol Biomarkers Prev. 2022 Jan;31(1):124-131. doi: 10.1158/1055-9965.EPI-21-0661. Epub 2021 Nov 04.

BACKGROUND: TP53 and estrogen receptor (ER) both play essential roles in breast cancer development and progression, with recent research revealing cross-talk between TP53 and ER signaling pathways. Although many studies have demonstrated heterogeneity of risk factor associations across ER...

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Hurson AN, Abubakar M, Hamilton AM, et al. TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study. Cancer Epidemiol Biomarkers Prev. 2021;31(1):124-131doi: 10.1158/1055-9965.EPI-21-0661.
Hurson, A. N., Abubakar, M., Hamilton, A. M., Conway, K., Hoadley, K. A., Love, M. I., Olshan, A. F., Perou, C. M., Garcia-Closas, M., & Troester, M. A. (2022). TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study. Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology, 31(1), 124-131. https://doi.org/10.1158/1055-9965.EPI-21-0661
Hurson, Amber N, et al. "TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study." Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology vol. 31,1 (2022): 124-131. doi: https://doi.org/10.1158/1055-9965.EPI-21-0661
Hurson AN, Abubakar M, Hamilton AM, Conway K, Hoadley KA, Love MI, Olshan AF, Perou CM, Garcia-Closas M, Troester MA. TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study. Cancer Epidemiol Biomarkers Prev. 2022 Jan;31(1):124-131. doi: 10.1158/1055-9965.EPI-21-0661. Epub 2021 Nov 04. PMID: 34737209.
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Antiapoptotic Bcl-2 family proteins BCL-xL and MCL-1 integrate neural progenitor survival and proliferation during postnatal cerebellar neurogenesis.

Cell death and differentiation

Veleta KA, Cleveland AH, Babcock BR, He YW, Hwang D, Sokolsky-Papkov M, Gershon TR.
PMID: 33293647
Cell Death Differ. 2021 May;28(5):1579-1592. doi: 10.1038/s41418-020-00687-7. Epub 2020 Dec 08.

The tendency of brain cells to undergo apoptosis in response to exogenous events varies across neural development, with apoptotic threshold dependent on proliferation state. Proliferative neural progenitors show a low threshold for apoptosis, while terminally differentiated neurons are relatively...

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Veleta KA, Cleveland AH, Babcock BR, et al. Antiapoptotic Bcl-2 family proteins BCL-xL and MCL-1 integrate neural progenitor survival and proliferation during postnatal cerebellar neurogenesis. Cell Death Differ. 2020;28(5):1579-1592doi: 10.1038/s41418-020-00687-7.
Veleta, K. A., Cleveland, A. H., Babcock, B. R., He, Y. W., Hwang, D., Sokolsky-Papkov, M., & Gershon, T. R. (2021). Antiapoptotic Bcl-2 family proteins BCL-xL and MCL-1 integrate neural progenitor survival and proliferation during postnatal cerebellar neurogenesis. Cell death and differentiation, 28(5), 1579-1592. https://doi.org/10.1038/s41418-020-00687-7
Veleta, Katherine A, et al. "Antiapoptotic Bcl-2 family proteins BCL-xL and MCL-1 integrate neural progenitor survival and proliferation during postnatal cerebellar neurogenesis." Cell death and differentiation vol. 28,5 (2021): 1579-1592. doi: https://doi.org/10.1038/s41418-020-00687-7
Veleta KA, Cleveland AH, Babcock BR, He YW, Hwang D, Sokolsky-Papkov M, Gershon TR. Antiapoptotic Bcl-2 family proteins BCL-xL and MCL-1 integrate neural progenitor survival and proliferation during postnatal cerebellar neurogenesis. Cell Death Differ. 2021 May;28(5):1579-1592. doi: 10.1038/s41418-020-00687-7. Epub 2020 Dec 08. PMID: 33293647; PMCID: PMC8166857.
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Racial differences in breast cancer outcomes by hepatocyte growth factor pathway expression.

Breast cancer research and treatment

Jones GS, Hoadley KA, Benefield H, Olsson LT, Hamilton AM, Bhattacharya A, Kirk EL, Tipaldos HJ, Fleming JM, Williams KP, Love MI, Nichols HB, Olshan AF, Troester MA.
PMID: 35034243
Breast Cancer Res Treat. 2022 Jan 16; doi: 10.1007/s10549-021-06497-w. Epub 2022 Jan 16.

PURPOSE: Black women have a 40% increased risk of breast cancer-related mortality. These outcome disparities may reflect differences in tumor pathways and a lack of targetable therapies for specific subtypes that are more common in Black women. Hepatocyte growth...

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Jones GS, Hoadley KA, Benefield H, et al. Racial differences in breast cancer outcomes by hepatocyte growth factor pathway expression. Breast Cancer Res Treat. 2022;doi: 10.1007/s10549-021-06497-w.
Jones, G. S., Hoadley, K. A., Benefield, H., Olsson, L. T., Hamilton, A. M., Bhattacharya, A., Kirk, E. L., Tipaldos, H. J., Fleming, J. M., Williams, K. P., Love, M. I., Nichols, H. B., Olshan, A. F., & Troester, M. A. (2022). Racial differences in breast cancer outcomes by hepatocyte growth factor pathway expression. Breast cancer research and treatment, . https://doi.org/10.1007/s10549-021-06497-w
Jones, Gieira S, et al. "Racial differences in breast cancer outcomes by hepatocyte growth factor pathway expression." Breast cancer research and treatment vol. (2022). doi: https://doi.org/10.1007/s10549-021-06497-w
Jones GS, Hoadley KA, Benefield H, Olsson LT, Hamilton AM, Bhattacharya A, Kirk EL, Tipaldos HJ, Fleming JM, Williams KP, Love MI, Nichols HB, Olshan AF, Troester MA. Racial differences in breast cancer outcomes by hepatocyte growth factor pathway expression. Breast Cancer Res Treat. 2022 Jan 16; doi: 10.1007/s10549-021-06497-w. Epub 2022 Jan 16. PMID: 35034243.
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TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study.

Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology

Hurson AN, Abubakar M, Hamilton AM, Conway K, Hoadley KA, Love MI, Olshan AF, Perou CM, Garcia-Closas M, Troester MA.
PMID: 34737209
Cancer Epidemiol Biomarkers Prev. 2022 Jan;31(1):124-131. doi: 10.1158/1055-9965.EPI-21-0661. Epub 2021 Nov 04.

BACKGROUND: TP53 and estrogen receptor (ER) both play essential roles in breast cancer development and progression, with recent research revealing cross-talk between TP53 and ER signaling pathways. Although many studies have demonstrated heterogeneity of risk factor associations across ER...

Cite
Hurson AN, Abubakar M, Hamilton AM, et al. TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study. Cancer Epidemiol Biomarkers Prev. 2021;31(1):124-131doi: 10.1158/1055-9965.EPI-21-0661.
Hurson, A. N., Abubakar, M., Hamilton, A. M., Conway, K., Hoadley, K. A., Love, M. I., Olshan, A. F., Perou, C. M., Garcia-Closas, M., & Troester, M. A. (2022). TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study. Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology, 31(1), 124-131. https://doi.org/10.1158/1055-9965.EPI-21-0661
Hurson, Amber N, et al. "TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study." Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology vol. 31,1 (2022): 124-131. doi: https://doi.org/10.1158/1055-9965.EPI-21-0661
Hurson AN, Abubakar M, Hamilton AM, Conway K, Hoadley KA, Love MI, Olshan AF, Perou CM, Garcia-Closas M, Troester MA. TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study. Cancer Epidemiol Biomarkers Prev. 2022 Jan;31(1):124-131. doi: 10.1158/1055-9965.EPI-21-0661. Epub 2021 Nov 04. PMID: 34737209; PMCID: PMC8755611.
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Extended HPV Genotyping to Compare HPV Type Distribution in Self- and Provider-Collected Samples for Cervical Cancer Screening.

Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology

Rohner E, Edelman C, Sanusi B, Schmitt JW, Baker A, Chesko K, Faherty B, Gregory SM, Romocki LS, Sivaraman V, Nelson JAE, O'Connor S, Hudgens MG, Knittel AK, Rahangdale L, Smith JS.
PMID: 32943435
Cancer Epidemiol Biomarkers Prev. 2020 Dec;29(12):2651-2661. doi: 10.1158/1055-9965.EPI-20-0674. Epub 2020 Sep 17.

BACKGROUND: Primary high-risk human papillomavirus (hr-HPV) testing of self-collected cervico-vaginal swabs could increase cervical cancer screening coverage, although triage strategies are needed to reduce unnecessary colposcopies. We evaluated the use of extended hr-HPV genotyping of self-collected samples for cervical...

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Rohner E, Edelman C, Sanusi B, et al. Extended HPV Genotyping to Compare HPV Type Distribution in Self- and Provider-Collected Samples for Cervical Cancer Screening. Cancer Epidemiol Biomarkers Prev. 2020;29(12):2651-2661doi: 10.1158/1055-9965.EPI-20-0674.
Rohner, E., Edelman, C., Sanusi, B., Schmitt, J. W., Baker, A., Chesko, K., Faherty, B., Gregory, S. M., Romocki, L. S., Sivaraman, V., Nelson, J. A. E., O'Connor, S., Hudgens, M. G., Knittel, A. K., Rahangdale, L., & Smith, J. S. (2020). Extended HPV Genotyping to Compare HPV Type Distribution in Self- and Provider-Collected Samples for Cervical Cancer Screening. Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology, 29(12), 2651-2661. https://doi.org/10.1158/1055-9965.EPI-20-0674
Rohner, Eliane, et al. "Extended HPV Genotyping to Compare HPV Type Distribution in Self- and Provider-Collected Samples for Cervical Cancer Screening." Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology vol. 29,12 (2020): 2651-2661. doi: https://doi.org/10.1158/1055-9965.EPI-20-0674
Rohner E, Edelman C, Sanusi B, Schmitt JW, Baker A, Chesko K, Faherty B, Gregory SM, Romocki LS, Sivaraman V, Nelson JAE, O'Connor S, Hudgens MG, Knittel AK, Rahangdale L, Smith JS. Extended HPV Genotyping to Compare HPV Type Distribution in Self- and Provider-Collected Samples for Cervical Cancer Screening. Cancer Epidemiol Biomarkers Prev. 2020 Dec;29(12):2651-2661. doi: 10.1158/1055-9965.EPI-20-0674. Epub 2020 Sep 17. PMID: 32943435; PMCID: PMC7710587.
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Genome-wide cancer-specific chromatin accessibility patterns derived from archival processed xenograft tumors.

Genome research

Marcel SS, Quimby AL, Noel MP, Jaimes OC, Mehrab-Mohseni M, Ashur SA, Velasco B, Tsuruta JK, Kasoji SK, Santos CM, Dayton PA, Parker JS, Davis IJ, Pattenden SG.
PMID: 34815311
Genome Res. 2021 Nov 23; doi: 10.1101/gr.275219.121. Epub 2021 Nov 23.

Chromatin accessibility states that influence gene expression and other nuclear processes can be altered in disease. The constellation of transcription factors and chromatin regulatory complexes in cells results in characteristic patterns of chromatin accessibility. The study of these patterns...

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Marcel SS, Quimby AL, Noel MP, et al. Genome-wide cancer-specific chromatin accessibility patterns derived from archival processed xenograft tumors. Genome Res. 2021;doi: 10.1101/gr.275219.121.
Marcel, S. S., Quimby, A. L., Noel, M. P., Jaimes, O. C., Mehrab-Mohseni, M., Ashur, S. A., Velasco, B., Tsuruta, J. K., Kasoji, S. K., Santos, C. M., Dayton, P. A., Parker, J. S., Davis, I. J., & Pattenden, S. G. (2021). Genome-wide cancer-specific chromatin accessibility patterns derived from archival processed xenograft tumors. Genome research, . https://doi.org/10.1101/gr.275219.121
Marcel, Shelsa S, et al. "Genome-wide cancer-specific chromatin accessibility patterns derived from archival processed xenograft tumors." Genome research vol. (2021). doi: https://doi.org/10.1101/gr.275219.121
Marcel SS, Quimby AL, Noel MP, Jaimes OC, Mehrab-Mohseni M, Ashur SA, Velasco B, Tsuruta JK, Kasoji SK, Santos CM, Dayton PA, Parker JS, Davis IJ, Pattenden SG. Genome-wide cancer-specific chromatin accessibility patterns derived from archival processed xenograft tumors. Genome Res. 2021 Nov 23; doi: 10.1101/gr.275219.121. Epub 2021 Nov 23. PMID: 34815311; PMCID: PMC8647830.
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Lysyl hydroxylase 2-induced collagen cross-link switching promotes metastasis in head and neck squamous cell carcinomas.

Neoplasia (New York, N.Y.)

Sato K, Parag-Sharma K, Terajima M, Musicant AM, Murphy RM, Ramsey MR, Hibi H, Yamauchi M, Amelio AL.
PMID: 34107376
Neoplasia. 2021 Jun;23(6):594-606. doi: 10.1016/j.neo.2021.05.014. Epub 2021 Jun 06.

Head and neck squamous cell carcinoma (HNSCC) is the 6th most common cancer worldwide and incidence rates are continuing to rise globally. HNSCC patient prognosis is closely related to the occurrence of tumor metastases, and collagen within the tumor...

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Sato K, Parag-Sharma K, Terajima M, et al. Lysyl hydroxylase 2-induced collagen cross-link switching promotes metastasis in head and neck squamous cell carcinomas. Neoplasia. 2021;23(6):594-606doi: 10.1016/j.neo.2021.05.014.
Sato, K., Parag-Sharma, K., Terajima, M., Musicant, A. M., Murphy, R. M., Ramsey, M. R., Hibi, H., Yamauchi, M., & Amelio, A. L. (2021). Lysyl hydroxylase 2-induced collagen cross-link switching promotes metastasis in head and neck squamous cell carcinomas. Neoplasia (New York, N.Y.), 23(6), 594-606. https://doi.org/10.1016/j.neo.2021.05.014
Sato, Kotaro, et al. "Lysyl hydroxylase 2-induced collagen cross-link switching promotes metastasis in head and neck squamous cell carcinomas." Neoplasia (New York, N.Y.) vol. 23,6 (2021): 594-606. doi: https://doi.org/10.1016/j.neo.2021.05.014
Sato K, Parag-Sharma K, Terajima M, Musicant AM, Murphy RM, Ramsey MR, Hibi H, Yamauchi M, Amelio AL. Lysyl hydroxylase 2-induced collagen cross-link switching promotes metastasis in head and neck squamous cell carcinomas. Neoplasia. 2021 Jun;23(6):594-606. doi: 10.1016/j.neo.2021.05.014. Epub 2021 Jun 06. PMID: 34107376; PMCID: PMC8192727.
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Tumor Necrosis Factor Alpha-Induced Interleukin-1 Alpha Synthesis and Cell Death Is Increased in Mouse Epithelial Cells Infected With Chlamydia muridarum.

The Journal of infectious diseases

Nagarajan UM, Cho C, Gyorke CE, Nagarajan S, Ezzell JA, Brochu H, Huntress I, Harrell E, Peng X.
PMID: 34396406
J Infect Dis. 2021 Aug 16;224(12):S47-S55. doi: 10.1093/infdis/jiab168.

Chlamydia trachomatis-genital infection in women can be modeled in mice using Chlamydia muridarum. Using this model, it has been shown that the cytokines tumor necrosis factor (TNF)α and interleukin (IL)-1α lead to irreversible tissue damage in the oviducts. In...

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Nagarajan UM, Cho C, Gyorke CE, et al. Tumor Necrosis Factor Alpha-Induced Interleukin-1 Alpha Synthesis and Cell Death Is Increased in Mouse Epithelial Cells Infected With Chlamydia muridarum. J Infect Dis. 2021;224(12):S47-S55doi: 10.1093/infdis/jiab168.
Nagarajan, U. M., Cho, C., Gyorke, C. E., Nagarajan, S., Ezzell, J. A., Brochu, H., Huntress, I., Harrell, E., & Peng, X. (2021). Tumor Necrosis Factor Alpha-Induced Interleukin-1 Alpha Synthesis and Cell Death Is Increased in Mouse Epithelial Cells Infected With Chlamydia muridarum. The Journal of infectious diseases, 224(12), S47-S55. https://doi.org/10.1093/infdis/jiab168
Nagarajan, Uma M, et al. "Tumor Necrosis Factor Alpha-Induced Interleukin-1 Alpha Synthesis and Cell Death Is Increased in Mouse Epithelial Cells Infected With Chlamydia muridarum." The Journal of infectious diseases vol. 224,12 (2021): S47-S55. doi: https://doi.org/10.1093/infdis/jiab168
Nagarajan UM, Cho C, Gyorke CE, Nagarajan S, Ezzell JA, Brochu H, Huntress I, Harrell E, Peng X. Tumor Necrosis Factor Alpha-Induced Interleukin-1 Alpha Synthesis and Cell Death Is Increased in Mouse Epithelial Cells Infected With Chlamydia muridarum. J Infect Dis. 2021 Aug 16;224(12):S47-S55. doi: 10.1093/infdis/jiab168. PMID: 34396406; PMCID: PMC8365113.
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Cellular effects of nicotine salt-containing e-liquids.

Journal of applied toxicology : JAT

Ghosh A, Beyazcicek O, Davis ES, Onyenwoke RU, Tarran R.
PMID: 33034066
J Appl Toxicol. 2021 Mar;41(3):493-505. doi: 10.1002/jat.4060. Epub 2020 Oct 09.

"Pod-based" e-cigarettes such as JUUL are currently the most prevalent electronic nicotine delivery systems (ENDS) in the United States. JUUL-type ENDS utilize nicotine salts protonated with benzoic acid rather than freebase nicotine. However, limited information is available on the...

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Ghosh A, Beyazcicek O, Davis ES, et al. Cellular effects of nicotine salt-containing e-liquids. J Appl Toxicol. 2020;41(3):493-505doi: 10.1002/jat.4060.
Ghosh, A., Beyazcicek, O., Davis, E. S., Onyenwoke, R. U., & Tarran, R. (2021). Cellular effects of nicotine salt-containing e-liquids. Journal of applied toxicology : JAT, 41(3), 493-505. https://doi.org/10.1002/jat.4060
Ghosh, Arunava, et al. "Cellular effects of nicotine salt-containing e-liquids." Journal of applied toxicology : JAT vol. 41,3 (2021): 493-505. doi: https://doi.org/10.1002/jat.4060
Ghosh A, Beyazcicek O, Davis ES, Onyenwoke RU, Tarran R. Cellular effects of nicotine salt-containing e-liquids. J Appl Toxicol. 2021 Mar;41(3):493-505. doi: 10.1002/jat.4060. Epub 2020 Oct 09. PMID: 33034066.
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MicroRNA-29 is an essential regulator of brain maturation through regulation of CH methylation.

Cell reports

Swahari V, Nakamura A, Hollville E, Stroud H, Simon JM, Ptacek TS, Beck MV, Flowers C, Guo J, Plestant C, Liang J, Kurtz CL, Kanke M, Hammond SM, He YW, Anton ES, Sethupathy P, Moy SS, Greenberg ME, Deshmukh M.
PMID: 33826889
Cell Rep. 2021 Apr 06;35(1):108946. doi: 10.1016/j.celrep.2021.108946.

Although embryonic brain development and neurodegeneration have received considerable attention, the events that govern postnatal brain maturation are less understood. Here, we identify the miR-29 family to be strikingly induced during the late stages of brain maturation. Brain maturation...

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Swahari V, Nakamura A, Hollville E, et al. MicroRNA-29 is an essential regulator of brain maturation through regulation of CH methylation. Cell Rep. 2021;35(1):108946doi: 10.1016/j.celrep.2021.108946.
Swahari, V., Nakamura, A., Hollville, E., Stroud, H., Simon, J. M., Ptacek, T. S., Beck, M. V., Flowers, C., Guo, J., Plestant, C., Liang, J., Kurtz, C. L., Kanke, M., Hammond, S. M., He, Y. W., Anton, E. S., Sethupathy, P., Moy, S. S., Greenberg, M. E., & Deshmukh, M. (2021). MicroRNA-29 is an essential regulator of brain maturation through regulation of CH methylation. Cell reports, 35(1), 108946. https://doi.org/10.1016/j.celrep.2021.108946
Swahari, Vijay, et al. "MicroRNA-29 is an essential regulator of brain maturation through regulation of CH methylation." Cell reports vol. 35,1 (2021): 108946. doi: https://doi.org/10.1016/j.celrep.2021.108946
Swahari V, Nakamura A, Hollville E, Stroud H, Simon JM, Ptacek TS, Beck MV, Flowers C, Guo J, Plestant C, Liang J, Kurtz CL, Kanke M, Hammond SM, He YW, Anton ES, Sethupathy P, Moy SS, Greenberg ME, Deshmukh M. MicroRNA-29 is an essential regulator of brain maturation through regulation of CH methylation. Cell Rep. 2021 Apr 06;35(1):108946. doi: 10.1016/j.celrep.2021.108946. PMID: 33826889; PMCID: PMC8103628.
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TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study.

Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology

Hurson AN, Abubakar M, Hamilton AM, Conway K, Hoadley KA, Love MI, Olshan AF, Perou CM, Garcia-Closas M, Troester MA.
PMID: 34737209
Cancer Epidemiol Biomarkers Prev. 2022 Jan;31(1):124-131. doi: 10.1158/1055-9965.EPI-21-0661. Epub 2021 Nov 04.

BACKGROUND: TP53 and estrogen receptor (ER) both play essential roles in breast cancer development and progression, with recent research revealing cross-talk between TP53 and ER signaling pathways. Although many studies have demonstrated heterogeneity of risk factor associations across ER...

Cite
Hurson AN, Abubakar M, Hamilton AM, et al. TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study. Cancer Epidemiol Biomarkers Prev. 2021;31(1):124-131doi: 10.1158/1055-9965.EPI-21-0661.
Hurson, A. N., Abubakar, M., Hamilton, A. M., Conway, K., Hoadley, K. A., Love, M. I., Olshan, A. F., Perou, C. M., Garcia-Closas, M., & Troester, M. A. (2022). TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study. Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology, 31(1), 124-131. https://doi.org/10.1158/1055-9965.EPI-21-0661
Hurson, Amber N, et al. "TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study." Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology vol. 31,1 (2022): 124-131. doi: https://doi.org/10.1158/1055-9965.EPI-21-0661
Hurson AN, Abubakar M, Hamilton AM, Conway K, Hoadley KA, Love MI, Olshan AF, Perou CM, Garcia-Closas M, Troester MA. TP53 Pathway Function, Estrogen Receptor Status, and Breast Cancer Risk Factors in the Carolina Breast Cancer Study. Cancer Epidemiol Biomarkers Prev. 2022 Jan;31(1):124-131. doi: 10.1158/1055-9965.EPI-21-0661. Epub 2021 Nov 04. PMID: 34737209; PMCID: PMC8755611.
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Showing 1 to 12 of 23 entries