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DNA methylation in blood as a mediator of the association of mid-childhood body mass index with cardio-metabolic risk score in early adolescence.

Epigenetics

Huang JV, Cardenas A, Colicino E, Schooling CM, Rifas-Shiman SL, Agha G, Zheng Y, Hou L, Just AC, Litonjua AA, DeMeo DL, Lin X, Oken E, Hivert MF, Baccarelli AA.
PMID: 30412002
Epigenetics. 2018;13(10):1072-1087. doi: 10.1080/15592294.2018.1543503. Epub 2018 Nov 13.

Obesity is associated with higher cardio-metabolic risk even in childhood and adolescence; whether this association is mediated by epigenetic mechanisms remains unclear. We examined the extent to which mid-childhood body mass index (BMI) z-score (median age 7.7 years) was...

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Huang JV, Cardenas A, Colicino E, et al. DNA methylation in blood as a mediator of the association of mid-childhood body mass index with cardio-metabolic risk score in early adolescence. Epigenetics. 2018;13(10):1072-1087doi: 10.1080/15592294.2018.1543503.
Huang, J. V., Cardenas, A., Colicino, E., Schooling, C. M., Rifas-Shiman, S. L., Agha, G., Zheng, Y., Hou, L., Just, A. C., Litonjua, A. A., DeMeo, D. L., Lin, X., Oken, E., Hivert, M. F., & Baccarelli, A. A. (2018). DNA methylation in blood as a mediator of the association of mid-childhood body mass index with cardio-metabolic risk score in early adolescence. Epigenetics, 13(10), 1072-1087. https://doi.org/10.1080/15592294.2018.1543503
Huang, Jian V, et al. "DNA methylation in blood as a mediator of the association of mid-childhood body mass index with cardio-metabolic risk score in early adolescence." Epigenetics vol. 13,10 (2018): 1072-1087. doi: https://doi.org/10.1080/15592294.2018.1543503
Huang JV, Cardenas A, Colicino E, Schooling CM, Rifas-Shiman SL, Agha G, Zheng Y, Hou L, Just AC, Litonjua AA, DeMeo DL, Lin X, Oken E, Hivert MF, Baccarelli AA. DNA methylation in blood as a mediator of the association of mid-childhood body mass index with cardio-metabolic risk score in early adolescence. Epigenetics. 2018;13(10):1072-1087. doi: 10.1080/15592294.2018.1543503. Epub 2018 Nov 13. PMID: 30412002; PMCID: PMC6342073.
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Prospective Associations of Early Pregnancy Metal Mixtures with Mitochondria DNA Copy Number and Telomere Length in Maternal and Cord Blood.

Environmental health perspectives

Smith AR, Lin PD, Rifas-Shiman SL, Rahman ML, Gold DR, Baccarelli AA, Claus Henn B, Amarasiriwardena C, Wright RO, Coull B, Hivert MF, Oken E, Cardenas A.
PMID: 34797165
Environ Health Perspect. 2021 Nov;129(11):117007. doi: 10.1289/EHP9294. Epub 2021 Nov 19.

BACKGROUND: Metal exposure during pregnancy influences maternal and child health. Oxidative stress and inflammation may mediate adverse effects of heavy metals, whereas essential metals may act as antioxidants. Mitochondrial DNA is a prime target for metal-induced oxidative damage. Telomere...

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Smith AR, Lin PD, Rifas-Shiman SL, et al. Prospective Associations of Early Pregnancy Metal Mixtures with Mitochondria DNA Copy Number and Telomere Length in Maternal and Cord Blood. Environ Health Perspect. 2021;129(11):117007doi: 10.1289/EHP9294.
Smith, A. R., Lin, P. D., Rifas-Shiman, S. L., Rahman, M. L., Gold, D. R., Baccarelli, A. A., Claus Henn, B., Amarasiriwardena, C., Wright, R. O., Coull, B., Hivert, M. F., Oken, E., & Cardenas, A. (2021). Prospective Associations of Early Pregnancy Metal Mixtures with Mitochondria DNA Copy Number and Telomere Length in Maternal and Cord Blood. Environmental health perspectives, 129(11), 117007. https://doi.org/10.1289/EHP9294
Smith, Anna R, et al. "Prospective Associations of Early Pregnancy Metal Mixtures with Mitochondria DNA Copy Number and Telomere Length in Maternal and Cord Blood." Environmental health perspectives vol. 129,11 (2021): 117007. doi: https://doi.org/10.1289/EHP9294
Smith AR, Lin PD, Rifas-Shiman SL, Rahman ML, Gold DR, Baccarelli AA, Claus Henn B, Amarasiriwardena C, Wright RO, Coull B, Hivert MF, Oken E, Cardenas A. Prospective Associations of Early Pregnancy Metal Mixtures with Mitochondria DNA Copy Number and Telomere Length in Maternal and Cord Blood. Environ Health Perspect. 2021 Nov;129(11):117007. doi: 10.1289/EHP9294. Epub 2021 Nov 19. PMID: 34797165; PMCID: PMC8604047.
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Associations of midchildhood to early adolescence central adiposity gain with cardiometabolic health in early adolescence.

Obesity (Silver Spring, Md.)

Wu AJ, Aris IM, Rifas-Shiman SL, Oken E, Taveras EM, Chavarro JE, Hivert MF.
PMID: 34529343
Obesity (Silver Spring). 2021 Nov;29(11):1882-1891. doi: 10.1002/oby.23261. Epub 2021 Sep 16.

OBJECTIVE: This study examined the associations of central adiposity gain from midchildhood to early adolescence with cardiometabolic health markers in early adolescence.METHODS: A total of 620 participants were studied in Project Viva. In midchildhood (mean age = 7.8 years)...

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Wu AJ, Aris IM, Rifas-Shiman SL, et al. Associations of midchildhood to early adolescence central adiposity gain with cardiometabolic health in early adolescence. Obesity (Silver Spring). 2021;29(11):1882-1891doi: 10.1002/oby.23261.
Wu, A. J., Aris, I. M., Rifas-Shiman, S. L., Oken, E., Taveras, E. M., Chavarro, J. E., & Hivert, M. F. (2021). Associations of midchildhood to early adolescence central adiposity gain with cardiometabolic health in early adolescence. Obesity (Silver Spring, Md.), 29(11), 1882-1891. https://doi.org/10.1002/oby.23261
Wu, Allison J, et al. "Associations of midchildhood to early adolescence central adiposity gain with cardiometabolic health in early adolescence." Obesity (Silver Spring, Md.) vol. 29,11 (2021): 1882-1891. doi: https://doi.org/10.1002/oby.23261
Wu AJ, Aris IM, Rifas-Shiman SL, Oken E, Taveras EM, Chavarro JE, Hivert MF. Associations of midchildhood to early adolescence central adiposity gain with cardiometabolic health in early adolescence. Obesity (Silver Spring). 2021 Nov;29(11):1882-1891. doi: 10.1002/oby.23261. Epub 2021 Sep 16. PMID: 34529343; PMCID: PMC8571062.
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DNA Methylation Architecture of the ACE2 gene in Nasal Cells.

medRxiv : the preprint server for health sciences

Cardenas A, Rifas-Shiman SL, Sordillo JE, DeMeo DL, Baccarelli AA, Hivert MF, Gold DR, Oken E.
PMID: 32995808
medRxiv. 2020 Sep 16; doi: 10.1101/2020.08.25.20182105.

Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) has led to the global coronavirus disease 2019 (COVID-19) pandemic. SARS-CoV-2 enters cells via angiotensin-Converting Enzyme 2 (ACE2) receptors, highly expressed in nasal epithelium with parallel high infectivity.1,2 The nasal epigenome is...

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Cardenas A, Rifas-Shiman SL, Sordillo JE, et al. DNA Methylation Architecture of the ACE2 gene in Nasal Cells. medRxiv. 2020;doi: 10.1101/2020.08.25.20182105.
Cardenas, A., Rifas-Shiman, S. L., Sordillo, J. E., DeMeo, D. L., Baccarelli, A. A., Hivert, M. F., Gold, D. R., & Oken, E. (2020). DNA Methylation Architecture of the ACE2 gene in Nasal Cells. medRxiv : the preprint server for health sciences, . https://doi.org/10.1101/2020.08.25.20182105
Cardenas, Andres, et al. "DNA Methylation Architecture of the ACE2 gene in Nasal Cells." medRxiv : the preprint server for health sciences vol. (2020). doi: https://doi.org/10.1101/2020.08.25.20182105
Cardenas A, Rifas-Shiman SL, Sordillo JE, DeMeo DL, Baccarelli AA, Hivert MF, Gold DR, Oken E. DNA Methylation Architecture of the ACE2 gene in Nasal Cells. medRxiv. 2020 Sep 16; doi: 10.1101/2020.08.25.20182105. PMID: 32995808; PMCID: PMC7523147.
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Maternal anxiety during pregnancy and newborn epigenome-wide DNA methylation.

Molecular psychiatry

Sammallahti S, Cortes Hidalgo AP, Tuominen S, Malmberg A, Mulder RH, Brunst KJ, Alemany S, McBride NS, Yousefi P, Heiss JA, McRae N, Page CM, Jin J, Pesce G, Caramaschi D, Rifas-Shiman SL, Koen N, Adams CD, Magnus MC, Baïz N, Ratanatharathorn A, Czamara D, Håberg SE, Colicino E, Baccarelli AA, Cardenas A, DeMeo DL, Lawlor DA, Relton CL, Felix JF, van IJzendoorn MH, Bakermans-Kranenburg MJ, Kajantie E, Räikkönen K, Sunyer J, Sharp GC, Houtepen LC, Nohr EA, Sørensen TIA, Téllez-Rojo MM, Wright RO, Annesi-Maesano I, Wright J, Hivert MF, Wright RJ, Zar HJ, Stein DJ, London SJ, Cecil CAM, Tiemeier H, Lahti J.
PMID: 33414500
Mol Psychiatry. 2021 Jun;26(6):1832-1845. doi: 10.1038/s41380-020-00976-0. Epub 2021 Jan 07.

Maternal anxiety during pregnancy is associated with adverse foetal, neonatal, and child outcomes, but biological mechanisms remain unclear. Altered foetal DNA methylation (DNAm) has been proposed as a potential underlying mechanism. In the current study, we performed a meta-analysis...

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Sammallahti S, Cortes Hidalgo AP, Tuominen S, et al. Maternal anxiety during pregnancy and newborn epigenome-wide DNA methylation. Mol Psychiatry. 2021;26(6):1832-1845doi: 10.1038/s41380-020-00976-0.
Sammallahti, S., Cortes Hidalgo, A. P., Tuominen, S., Malmberg, A., Mulder, R. H., Brunst, K. J., Alemany, S., McBride, N. S., Yousefi, P., Heiss, J. A., McRae, N., Page, C. M., Jin, J., Pesce, G., Caramaschi, D., Rifas-Shiman, S. L., Koen, N., Adams, C. D., Magnus, M. C., Baïz, N., Ratanatharathorn, A., Czamara, D., Håberg, S. E., Colicino, E., Baccarelli, A. A., Cardenas, A., DeMeo, D. L., Lawlor, D. A., Relton, C. L., Felix, J. F., van IJzendoorn, M. H., Bakermans-Kranenburg, M. J., Kajantie, E., Räikkönen, K., Sunyer, J., Sharp, G. C., Houtepen, L. C., Nohr, E. A., Sørensen, T. I. A., Téllez-Rojo, M. M., Wright, R. O., Annesi-Maesano, I., Wright, J., Hivert, M. F., Wright, R. J., Zar, H. J., Stein, D. J., London, S. J., Cecil, C. A. M., Tiemeier, H., & Lahti, J. (2021). Maternal anxiety during pregnancy and newborn epigenome-wide DNA methylation. Molecular psychiatry, 26(6), 1832-1845. https://doi.org/10.1038/s41380-020-00976-0
Sammallahti, Sara, et al. "Maternal anxiety during pregnancy and newborn epigenome-wide DNA methylation." Molecular psychiatry vol. 26,6 (2021): 1832-1845. doi: https://doi.org/10.1038/s41380-020-00976-0
Sammallahti S, Cortes Hidalgo AP, Tuominen S, Malmberg A, Mulder RH, Brunst KJ, Alemany S, McBride NS, Yousefi P, Heiss JA, McRae N, Page CM, Jin J, Pesce G, Caramaschi D, Rifas-Shiman SL, Koen N, Adams CD, Magnus MC, Baïz N, Ratanatharathorn A, Czamara D, Håberg SE, Colicino E, Baccarelli AA, Cardenas A, DeMeo DL, Lawlor DA, Relton CL, Felix JF, van IJzendoorn MH, Bakermans-Kranenburg MJ, Kajantie E, Räikkönen K, Sunyer J, Sharp GC, Houtepen LC, Nohr EA, Sørensen TIA, Téllez-Rojo MM, Wright RO, Annesi-Maesano I, Wright J, Hivert MF, Wright RJ, Zar HJ, Stein DJ, London SJ, Cecil CAM, Tiemeier H, Lahti J. Maternal anxiety during pregnancy and newborn epigenome-wide DNA methylation. Mol Psychiatry. 2021 Jun;26(6):1832-1845. doi: 10.1038/s41380-020-00976-0. Epub 2021 Jan 07. PMID: 33414500; PMCID: PMC8595870.
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Associations of maternal non-nutritive sweetener intake during pregnancy with offspring body mass index and body fat from birth to adolescence.

International journal of obesity (2005)

Plows JF, Aris IM, Rifas-Shiman SL, Goran MI, Oken E.
PMID: 34611285
Int J Obes (Lond). 2022 Jan;46(1):186-193. doi: 10.1038/s41366-021-00897-0. Epub 2021 Oct 05.

BACKGROUND/OBJECTIVE: The evidence that maternal non-nutritive sweetener (NNS) intake during pregnancy increases childhood obesity risk is conflicting. A potential reason for this is that all prior studies examined childhood body mass index (BMI) at only one timepoint and at...

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Plows JF, Aris IM, Rifas-Shiman SL, et al. Associations of maternal non-nutritive sweetener intake during pregnancy with offspring body mass index and body fat from birth to adolescence. Int J Obes (Lond). 2021;46(1):186-193doi: 10.1038/s41366-021-00897-0.
Plows, J. F., Aris, I. M., Rifas-Shiman, S. L., Goran, M. I., & Oken, E. (2022). Associations of maternal non-nutritive sweetener intake during pregnancy with offspring body mass index and body fat from birth to adolescence. International journal of obesity (2005), 46(1), 186-193. https://doi.org/10.1038/s41366-021-00897-0
Plows, Jasmine F, et al. "Associations of maternal non-nutritive sweetener intake during pregnancy with offspring body mass index and body fat from birth to adolescence." International journal of obesity (2005) vol. 46,1 (2022): 186-193. doi: https://doi.org/10.1038/s41366-021-00897-0
Plows JF, Aris IM, Rifas-Shiman SL, Goran MI, Oken E. Associations of maternal non-nutritive sweetener intake during pregnancy with offspring body mass index and body fat from birth to adolescence. Int J Obes (Lond). 2022 Jan;46(1):186-193. doi: 10.1038/s41366-021-00897-0. Epub 2021 Oct 05. PMID: 34611285.
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Associations of maternal non-nutritive sweetener intake during pregnancy with offspring body mass index and body fat from birth to adolescence.

International journal of obesity (2005)

Plows JF, Aris IM, Rifas-Shiman SL, Goran MI, Oken E.
PMID: 34611285
Int J Obes (Lond). 2021 Oct 05; doi: 10.1038/s41366-021-00897-0. Epub 2021 Oct 05.

BACKGROUND/OBJECTIVE: The evidence that maternal non-nutritive sweetener (NNS) intake during pregnancy increases childhood obesity risk is conflicting. A potential reason for this is that all prior studies examined childhood body mass index (BMI) at only one timepoint and at...

Cite
Plows JF, Aris IM, Rifas-Shiman SL, et al. Associations of maternal non-nutritive sweetener intake during pregnancy with offspring body mass index and body fat from birth to adolescence. Int J Obes (Lond). 2021;doi: 10.1038/s41366-021-00897-0.
Plows, J. F., Aris, I. M., Rifas-Shiman, S. L., Goran, M. I., & Oken, E. (2021). Associations of maternal non-nutritive sweetener intake during pregnancy with offspring body mass index and body fat from birth to adolescence. International journal of obesity (2005), . https://doi.org/10.1038/s41366-021-00897-0
Plows, Jasmine F, et al. "Associations of maternal non-nutritive sweetener intake during pregnancy with offspring body mass index and body fat from birth to adolescence." International journal of obesity (2005) vol. (2021). doi: https://doi.org/10.1038/s41366-021-00897-0
Plows JF, Aris IM, Rifas-Shiman SL, Goran MI, Oken E. Associations of maternal non-nutritive sweetener intake during pregnancy with offspring body mass index and body fat from birth to adolescence. Int J Obes (Lond). 2021 Oct 05; doi: 10.1038/s41366-021-00897-0. Epub 2021 Oct 05. PMID: 34611285.
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Contributions of asthma, rhinitis and IgE to exhaled nitric oxide in adolescents.

ERJ open research

Flashner BM, Rifas-Shiman SL, Oken E, Camargo CA, Platts-Mills TAE, Workman L, Litonjua AA, Gold DR, Rice MB.
PMID: 33898613
ERJ Open Res. 2021 Apr 19;7(2). doi: 10.1183/23120541.00945-2020. eCollection 2021 Apr.

Exhaled nitric oxide fraction (

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Flashner BM, Rifas-Shiman SL, Oken E, et al. Contributions of asthma, rhinitis and IgE to exhaled nitric oxide in adolescents. ERJ Open Res. 2021;7(2)doi: 10.1183/23120541.00945-2020.
Flashner, B. M., Rifas-Shiman, S. L., Oken, E., Camargo, C. A., Platts-Mills, T. A. E., Workman, L., Litonjua, A. A., Gold, D. R., & Rice, M. B. (2021). Contributions of asthma, rhinitis and IgE to exhaled nitric oxide in adolescents. ERJ open research, 7(2), . https://doi.org/10.1183/23120541.00945-2020
Flashner, Bess M, et al. "Contributions of asthma, rhinitis and IgE to exhaled nitric oxide in adolescents." ERJ open research vol. 7,2 (2021). doi: https://doi.org/10.1183/23120541.00945-2020
Flashner BM, Rifas-Shiman SL, Oken E, Camargo CA, Platts-Mills TAE, Workman L, Litonjua AA, Gold DR, Rice MB. Contributions of asthma, rhinitis and IgE to exhaled nitric oxide in adolescents. ERJ Open Res. 2021 Apr 19;7(2). doi: 10.1183/23120541.00945-2020. eCollection 2021 Apr. PMID: 33898613; PMCID: PMC8053905.
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SPR perspectives: Environmental influences on Child Health Outcomes (ECHO) Program: overcoming challenges to generate engaged, multidisciplinary science.

Pediatric research

LeWinn KZ, Caretta E, Davis A, Anderson AL, Oken E.
PMID: 34131290
Pediatr Res. 2021 Jun 15; doi: 10.1038/s41390-021-01598-0. Epub 2021 Jun 15.

The US National Institutes of Health-funded Environmental influences on Child Health Outcomes (ECHO) Program brings together 69 cohorts and over 57,000 children from across the nation to address five key pediatric outcome areas with high public health impact: pre-,...

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LeWinn KZ, Caretta E, Davis A, et al. SPR perspectives: Environmental influences on Child Health Outcomes (ECHO) Program: overcoming challenges to generate engaged, multidisciplinary science. Pediatr Res. 2021;doi: 10.1038/s41390-021-01598-0.
LeWinn, K. Z., Caretta, E., Davis, A., Anderson, A. L., Oken, E. (2021). SPR perspectives: Environmental influences on Child Health Outcomes (ECHO) Program: overcoming challenges to generate engaged, multidisciplinary science. Pediatric research, . https://doi.org/10.1038/s41390-021-01598-0
LeWinn, Kaja Z, et al. "SPR perspectives: Environmental influences on Child Health Outcomes (ECHO) Program: overcoming challenges to generate engaged, multidisciplinary science." Pediatric research vol. (2021). doi: https://doi.org/10.1038/s41390-021-01598-0
LeWinn KZ, Caretta E, Davis A, Anderson AL, Oken E. SPR perspectives: Environmental influences on Child Health Outcomes (ECHO) Program: overcoming challenges to generate engaged, multidisciplinary science. Pediatr Res. 2021 Jun 15; doi: 10.1038/s41390-021-01598-0. Epub 2021 Jun 15. PMID: 34131290; PMCID: PMC8204620.
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Prenatal metal exposure, cord blood DNA methylation and persistence in childhood: an epigenome-wide association study of 12 metals.

Clinical epigenetics

Bozack AK, Rifas-Shiman SL, Coull BA, Baccarelli AA, Wright RO, Amarasiriwardena C, Gold DR, Oken E, Hivert MF, Cardenas A.
PMID: 34798907
Clin Epigenetics. 2021 Nov 19;13(1):208. doi: 10.1186/s13148-021-01198-z.

BACKGROUND: Prenatal exposure to essential and non-essential metals impacts birth and child health, including fetal growth and neurodevelopment. DNA methylation (DNAm) may be involved in pathways linking prenatal metal exposure and health. In the Project Viva cohort, we analyzed...

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Bozack AK, Rifas-Shiman SL, Coull BA, et al. Prenatal metal exposure, cord blood DNA methylation and persistence in childhood: an epigenome-wide association study of 12 metals. Clin Epigenetics. 2021;13(1):208doi: 10.1186/s13148-021-01198-z.
Bozack, A. K., Rifas-Shiman, S. L., Coull, B. A., Baccarelli, A. A., Wright, R. O., Amarasiriwardena, C., Gold, D. R., Oken, E., Hivert, M. F., & Cardenas, A. (2021). Prenatal metal exposure, cord blood DNA methylation and persistence in childhood: an epigenome-wide association study of 12 metals. Clinical epigenetics, 13(1), 208. https://doi.org/10.1186/s13148-021-01198-z
Bozack, Anne K, et al. "Prenatal metal exposure, cord blood DNA methylation and persistence in childhood: an epigenome-wide association study of 12 metals." Clinical epigenetics vol. 13,1 (2021): 208. doi: https://doi.org/10.1186/s13148-021-01198-z
Bozack AK, Rifas-Shiman SL, Coull BA, Baccarelli AA, Wright RO, Amarasiriwardena C, Gold DR, Oken E, Hivert MF, Cardenas A. Prenatal metal exposure, cord blood DNA methylation and persistence in childhood: an epigenome-wide association study of 12 metals. Clin Epigenetics. 2021 Nov 19;13(1):208. doi: 10.1186/s13148-021-01198-z. PMID: 34798907; PMCID: PMC8605513.
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Association of mode of delivery with offspring pubertal development in Project Viva: a prospective pre-birth cohort study in the USA.

Human reproduction (Oxford, England)

Aris IM, Rifas-Shiman SL, Mínguez-Alarcón L, Sordillo JE, Hivert MF, Oken E, Chavarro JE.
PMID: 34755186
Hum Reprod. 2021 Dec 27;37(1):54-65. doi: 10.1093/humrep/deab235.

STUDY QUESTION: Is cesarean delivery associated with earlier offspring pubertal development?SUMMARY ANSWER: We identified that boys born by cesarean delivery developed puberty earlier, evidenced by an earlier age at peak height velocity and earlier attainment of puberty score >...

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Aris IM, Rifas-Shiman SL, Mínguez-Alarcón L, et al. Association of mode of delivery with offspring pubertal development in Project Viva: a prospective pre-birth cohort study in the USA. Hum Reprod. 2021;37(1):54-65doi: 10.1093/humrep/deab235.
Aris, I. M., Rifas-Shiman, S. L., Mínguez-Alarcón, L., Sordillo, J. E., Hivert, M. F., Oken, E., & Chavarro, J. E. (2021). Association of mode of delivery with offspring pubertal development in Project Viva: a prospective pre-birth cohort study in the USA. Human reproduction (Oxford, England), 37(1), 54-65. https://doi.org/10.1093/humrep/deab235
Aris, Izzuddin M, et al. "Association of mode of delivery with offspring pubertal development in Project Viva: a prospective pre-birth cohort study in the USA." Human reproduction (Oxford, England) vol. 37,1 (2021): 54-65. doi: https://doi.org/10.1093/humrep/deab235
Aris IM, Rifas-Shiman SL, Mínguez-Alarcón L, Sordillo JE, Hivert MF, Oken E, Chavarro JE. Association of mode of delivery with offspring pubertal development in Project Viva: a prospective pre-birth cohort study in the USA. Hum Reprod. 2021 Dec 27;37(1):54-65. doi: 10.1093/humrep/deab235. PMID: 34755186.
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Detecting differentially methylated regions with multiple distinct associations.

Epigenomics

Lent S, Cardenas A, Rifas-Shiman SL, Perron P, Bouchard L, Liu CT, Hivert MF, Dupuis J.
PMID: 33641349
Epigenomics. 2021 Mar;13(6):451-464. doi: 10.2217/epi-2020-0344. Epub 2021 Mar 01.

No abstract available.

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Lent S, Cardenas A, Rifas-Shiman SL, et al. Detecting differentially methylated regions with multiple distinct associations. Epigenomics. 2021;13(6):451-464doi: 10.2217/epi-2020-0344.
Lent, S., Cardenas, A., Rifas-Shiman, S. L., Perron, P., Bouchard, L., Liu, C. T., Hivert, M. F., & Dupuis, J. (2021). Detecting differentially methylated regions with multiple distinct associations. Epigenomics, 13(6), 451-464. https://doi.org/10.2217/epi-2020-0344
Lent, Samantha, et al. "Detecting differentially methylated regions with multiple distinct associations." Epigenomics vol. 13,6 (2021): 451-464. doi: https://doi.org/10.2217/epi-2020-0344
Lent S, Cardenas A, Rifas-Shiman SL, Perron P, Bouchard L, Liu CT, Hivert MF, Dupuis J. Detecting differentially methylated regions with multiple distinct associations. Epigenomics. 2021 Mar;13(6):451-464. doi: 10.2217/epi-2020-0344. Epub 2021 Mar 01. PMID: 33641349; PMCID: PMC8023344.
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Showing 1 to 12 of 34 entries