Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 159 entries
Sorted by: Best Match Show Resources per page
Expression of estrogen receptor, progesterone receptor, and Ki67 in normal breast tissue in relation to subsequent risk of breast cancer.

NPJ breast cancer

Oh H, Eliassen AH, Wang M, Smith-Warner SA, Beck AH, Schnitt SJ, Collins LC, Connolly JL, Montaser-Kouhsari L, Polyak K, Tamimi RM.
PMID: 28111631
NPJ Breast Cancer. 2016;2. doi: 10.1038/npjbcancer.2016.32. Epub 2016 Oct 26.

Although expression of estrogen receptor (ER), progesterone receptor (PR), and cell proliferation marker Ki67 serve as predictive and prognostic factors in breast cancers, little is known about their roles in normal breast tissue. Here in a nested case-control study...

Cite
Oh H, Eliassen AH, Wang M, et al. Expression of estrogen receptor, progesterone receptor, and Ki67 in normal breast tissue in relation to subsequent risk of breast cancer. NPJ Breast Cancer. 2016;2doi: 10.1038/npjbcancer.2016.32.
Oh, H., Eliassen, A. H., Wang, M., Smith-Warner, S. A., Beck, A. H., Schnitt, S. J., Collins, L. C., Connolly, J. L., Montaser-Kouhsari, L., Polyak, K., & Tamimi, R. M. (2016). Expression of estrogen receptor, progesterone receptor, and Ki67 in normal breast tissue in relation to subsequent risk of breast cancer. NPJ breast cancer, 2. https://doi.org/10.1038/npjbcancer.2016.32
Oh, Hannah, et al. "Expression of estrogen receptor, progesterone receptor, and Ki67 in normal breast tissue in relation to subsequent risk of breast cancer." NPJ breast cancer vol. 2 (2016). doi: https://doi.org/10.1038/npjbcancer.2016.32
Oh H, Eliassen AH, Wang M, Smith-Warner SA, Beck AH, Schnitt SJ, Collins LC, Connolly JL, Montaser-Kouhsari L, Polyak K, Tamimi RM. Expression of estrogen receptor, progesterone receptor, and Ki67 in normal breast tissue in relation to subsequent risk of breast cancer. NPJ Breast Cancer. 2016;2. doi: 10.1038/npjbcancer.2016.32. Epub 2016 Oct 26. PMID: 28111631; PMCID: PMC5243126.
Copy Download .nbib Format: NLM
Continuity of transcriptomes among colorectal cancer subtypes based on meta-analysis.

Genome biology

Ma S, Ogino S, Parsana P, Nishihara R, Qian Z, Shen J, Mima K, Masugi Y, Cao Y, Nowak JA, Shima K, Hoshida Y, Giovannucci EL, Gala MK, Chan AT, Fuchs CS, Parmigiani G, Huttenhower C, Waldron L.
PMID: 30253799
Genome Biol. 2018 Sep 25;19(1):142. doi: 10.1186/s13059-018-1511-4.

BACKGROUND: Previous approaches to defining subtypes of colorectal carcinoma (CRC) and other cancers based on transcriptomes have assumed the existence of discrete subtypes. We analyze gene expression patterns of colorectal tumors from a large number of patients to test...

Cite
Ma S, Ogino S, Parsana P, et al. Continuity of transcriptomes among colorectal cancer subtypes based on meta-analysis. Genome Biol. 2018;19(1):142doi: 10.1186/s13059-018-1511-4.
Ma, S., Ogino, S., Parsana, P., Nishihara, R., Qian, Z., Shen, J., Mima, K., Masugi, Y., Cao, Y., Nowak, J. A., Shima, K., Hoshida, Y., Giovannucci, E. L., Gala, M. K., Chan, A. T., Fuchs, C. S., Parmigiani, G., Huttenhower, C., & Waldron, L. (2018). Continuity of transcriptomes among colorectal cancer subtypes based on meta-analysis. Genome biology, 19(1), 142. https://doi.org/10.1186/s13059-018-1511-4
Ma, Siyuan, et al. "Continuity of transcriptomes among colorectal cancer subtypes based on meta-analysis." Genome biology vol. 19,1 (2018): 142. doi: https://doi.org/10.1186/s13059-018-1511-4
Ma S, Ogino S, Parsana P, Nishihara R, Qian Z, Shen J, Mima K, Masugi Y, Cao Y, Nowak JA, Shima K, Hoshida Y, Giovannucci EL, Gala MK, Chan AT, Fuchs CS, Parmigiani G, Huttenhower C, Waldron L. Continuity of transcriptomes among colorectal cancer subtypes based on meta-analysis. Genome Biol. 2018 Sep 25;19(1):142. doi: 10.1186/s13059-018-1511-4. PMID: 30253799; PMCID: PMC6154428.
Copy Download .nbib Format: NLM
Exploratory Genome-Wide Interaction Analysis of Nonsteroidal Anti-inflammatory Drugs and Predicted Gene Expression on Colorectal Cancer Risk.

Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology

Wang X, Su YR, Petersen PS, Bien S, Schmit SL, Drew DA, Albanes D, Berndt SI, Brenner H, Campbell PT, Casey G, Chang-Claude J, Gallinger SJ, Gruber SB, Haile RW, Harrison TA, Hoffmeister M, Jacobs EJ, Jenkins MA, Joshi AD, Li L, Lin Y, Lindor NM, Marchand LL, Martin V, Milne R, Maclnnis R, Moreno V, Nan H, Newcomb PA, Potter JD, Rennert G, Rennert H, Slattery ML, Thibodeau SN, Weinstein SJ, Woods MO, Chan AT, White E, Hsu L, Peters U.
PMID: 32651213
Cancer Epidemiol Biomarkers Prev. 2020 Sep;29(9):1800-1808. doi: 10.1158/1055-9965.EPI-19-1018. Epub 2020 Jul 10.

BACKGROUND: Regular use of nonsteroidal anti-inflammatory drugs (NSAID) is associated with lower risk of colorectal cancer. Genome-wide interaction analysis on single variants (G × E) has identified several SNPs that may interact with NSAIDs to confer colorectal cancer risk,...

Cite
Wang X, Su YR, Petersen PS, et al. Exploratory Genome-Wide Interaction Analysis of Nonsteroidal Anti-inflammatory Drugs and Predicted Gene Expression on Colorectal Cancer Risk. Cancer Epidemiol Biomarkers Prev. 2020;29(9):1800-1808doi: 10.1158/1055-9965.EPI-19-1018.
Wang, X., Su, Y. R., Petersen, P. S., Bien, S., Schmit, S. L., Drew, D. A., Albanes, D., Berndt, S. I., Brenner, H., Campbell, P. T., Casey, G., Chang-Claude, J., Gallinger, S. J., Gruber, S. B., Haile, R. W., Harrison, T. A., Hoffmeister, M., Jacobs, E. J., Jenkins, M. A., Joshi, A. D., Li, L., Lin, Y., Lindor, N. M., Marchand, L. L., Martin, V., Milne, R., Maclnnis, R., Moreno, V., Nan, H., Newcomb, P. A., Potter, J. D., Rennert, G., Rennert, H., Slattery, M. L., Thibodeau, S. N., Weinstein, S. J., Woods, M. O., Chan, A. T., White, E., Hsu, L., & Peters, U. (2020). Exploratory Genome-Wide Interaction Analysis of Nonsteroidal Anti-inflammatory Drugs and Predicted Gene Expression on Colorectal Cancer Risk. Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology, 29(9), 1800-1808. https://doi.org/10.1158/1055-9965.EPI-19-1018
Wang, Xiaoliang, et al. "Exploratory Genome-Wide Interaction Analysis of Nonsteroidal Anti-inflammatory Drugs and Predicted Gene Expression on Colorectal Cancer Risk." Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology vol. 29,9 (2020): 1800-1808. doi: https://doi.org/10.1158/1055-9965.EPI-19-1018
Wang X, Su YR, Petersen PS, Bien S, Schmit SL, Drew DA, Albanes D, Berndt SI, Brenner H, Campbell PT, Casey G, Chang-Claude J, Gallinger SJ, Gruber SB, Haile RW, Harrison TA, Hoffmeister M, Jacobs EJ, Jenkins MA, Joshi AD, Li L, Lin Y, Lindor NM, Marchand LL, Martin V, Milne R, Maclnnis R, Moreno V, Nan H, Newcomb PA, Potter JD, Rennert G, Rennert H, Slattery ML, Thibodeau SN, Weinstein SJ, Woods MO, Chan AT, White E, Hsu L, Peters U. Exploratory Genome-Wide Interaction Analysis of Nonsteroidal Anti-inflammatory Drugs and Predicted Gene Expression on Colorectal Cancer Risk. Cancer Epidemiol Biomarkers Prev. 2020 Sep;29(9):1800-1808. doi: 10.1158/1055-9965.EPI-19-1018. Epub 2020 Jul 10. PMID: 32651213; PMCID: PMC7556991.
Copy Download .nbib Format: NLM
Association of .

Clinical cancer research : an official journal of the American Association for Cancer Research

Borowsky J, Haruki K, Lau MC, Dias Costa A, Väyrynen JP, Ugai T, Arima K, da Silva A, Felt KD, Zhao M, Gurjao C, Twombly TS, Fujiyoshi K, Väyrynen SA, Hamada T, Mima K, Bullman S, Harrison TA, Phipps AI, Peters U, Ng K, Meyerhardt JA, Song M, Giovannucci EL, Wu K, Zhang X, Freeman GJ, Huttenhower C, Garrett WS, Chan AT, Leggett BA, Whitehall VLJ, Walker N, Brown I, Bettington M, Nishihara R, Fuchs CS, Lennerz JK, Giannakis M, Nowak JA, Ogino S.
PMID: 33632927
Clin Cancer Res. 2021 May 15;27(10):2816-2826. doi: 10.1158/1078-0432.CCR-20-4009. Epub 2021 Feb 25.

PURPOSE: While evidence indicates that EXPERIMENTAL DESIGN: We measured RESULTS: The amount of CONCLUSIONS: The amount of tissue

Cite
Borowsky J, Haruki K, Lau MC, et al. Association of . Clin Cancer Res. 2021;27(10):2816-2826doi: 10.1158/1078-0432.CCR-20-4009.
Borowsky, J., Haruki, K., Lau, M. C., Dias Costa, A., Väyrynen, J. P., Ugai, T., Arima, K., da Silva, A., Felt, K. D., Zhao, M., Gurjao, C., Twombly, T. S., Fujiyoshi, K., Väyrynen, S. A., Hamada, T., Mima, K., Bullman, S., Harrison, T. A., Phipps, A. I., Peters, U., Ng, K., Meyerhardt, J. A., Song, M., Giovannucci, E. L., Wu, K., Zhang, X., Freeman, G. J., Huttenhower, C., Garrett, W. S., Chan, A. T., Leggett, B. A., Whitehall, V. L. J., Walker, N., Brown, I., Bettington, M., Nishihara, R., Fuchs, C. S., Lennerz, J. K., Giannakis, M., Nowak, J. A., & Ogino, S. (2021). Association of . Clinical cancer research : an official journal of the American Association for Cancer Research, 27(10), 2816-2826. https://doi.org/10.1158/1078-0432.CCR-20-4009
Borowsky, Jennifer, et al. "Association of ." Clinical cancer research : an official journal of the American Association for Cancer Research vol. 27,10 (2021): 2816-2826. doi: https://doi.org/10.1158/1078-0432.CCR-20-4009
Borowsky J, Haruki K, Lau MC, Dias Costa A, Väyrynen JP, Ugai T, Arima K, da Silva A, Felt KD, Zhao M, Gurjao C, Twombly TS, Fujiyoshi K, Väyrynen SA, Hamada T, Mima K, Bullman S, Harrison TA, Phipps AI, Peters U, Ng K, Meyerhardt JA, Song M, Giovannucci EL, Wu K, Zhang X, Freeman GJ, Huttenhower C, Garrett WS, Chan AT, Leggett BA, Whitehall VLJ, Walker N, Brown I, Bettington M, Nishihara R, Fuchs CS, Lennerz JK, Giannakis M, Nowak JA, Ogino S. Association of . Clin Cancer Res. 2021 May 15;27(10):2816-2826. doi: 10.1158/1078-0432.CCR-20-4009. Epub 2021 Feb 25. PMID: 33632927; PMCID: PMC8127352.
Copy Download .nbib Format: NLM
Genetic architectures of proximal and distal colorectal cancer are partly distinct.

Gut

Huyghe JR, Harrison TA, Bien SA, Hampel H, Figueiredo JC, Schmit SL, Conti DV, Chen S, Qu C, Lin Y, Barfield R, Baron JA, Cross AJ, Diergaarde B, Duggan D, Harlid S, Imaz L, Kang HM, Levine DM, Perduca V, Perez-Cornago A, Sakoda LC, Schumacher FR, Slattery ML, Toland AE, van Duijnhoven FJB, Van Guelpen B, Agudo A, Albanes D, Alonso MH, Anderson K, Arnau-Collell C, Arndt V, Banbury BL, Bassik MC, Berndt SI, Bézieau S, Bishop DT, Boehm J, Boeing H, Boutron-Ruault MC, Brenner H, Brezina S, Buch S, Buchanan DD, Burnett-Hartman A, Caan BJ, Campbell PT, Carr PR, Castells A, Castellví-Bel S, Chan AT, Chang-Claude J, Chanock SJ, Curtis KR, de la Chapelle A, Easton DF, English DR, Feskens EJM, Gala M, Gallinger SJ, Gauderman WJ, Giles GG, Goodman PJ, Grady WM, Grove JS, Gsur A, Gunter MJ, Haile RW, Hampe J, Hoffmeister M, Hopper JL, Hsu WL, Huang WY, Hudson TJ, Jenab M, Jenkins MA, Joshi AD, Keku TO, Kooperberg C, Kühn T, Küry S, Le Marchand L, Lejbkowicz F, Li CI, Li L, Lieb W, Lindblom A, Lindor NM, Männistö S, Markowitz SD, Milne RL, Moreno L, Murphy N, Nassir R, Offit K, Ogino S, Panico S, Parfrey PS, Pearlman R, Pharoah PDP, Phipps AI, Platz EA, Potter JD, Prentice RL, Qi L, Raskin L, Rennert G, Rennert HS, Riboli E, Schafmayer C, Schoen RE, Seminara D, Song M, Su YR, Tangen CM, Thibodeau SN, Thomas DC, Trichopoulou A, Ulrich CM, Visvanathan K, Vodicka P, Vodickova L, Vymetalkova V, Weigl K, Weinstein SJ, White E, Wolk A, Woods MO, Wu AH, Abecasis GR, Nickerson DA, Scacheri PC, Kundaje A, Casey G, Gruber SB, Hsu L, Moreno V, Hayes RB, Newcomb PA, Peters U.
PMID: 33632709
Gut. 2021 Jul;70(7):1325-1334. doi: 10.1136/gutjnl-2020-321534. Epub 2021 Feb 25.

OBJECTIVE: An understanding of the etiologic heterogeneity of colorectal cancer (CRC) is critical for improving precision prevention, including individualized screening recommendations and the discovery of novel drug targets and repurposable drug candidates for chemoprevention. Known differences in molecular characteristics...

Cite
Huyghe JR, Harrison TA, Bien SA, et al. Genetic architectures of proximal and distal colorectal cancer are partly distinct. Gut. 2021;70(7):1325-1334doi: 10.1136/gutjnl-2020-321534.
Huyghe, J. R., Harrison, T. A., Bien, S. A., Hampel, H., Figueiredo, J. C., Schmit, S. L., Conti, D. V., Chen, S., Qu, C., Lin, Y., Barfield, R., Baron, J. A., Cross, A. J., Diergaarde, B., Duggan, D., Harlid, S., Imaz, L., Kang, H. M., Levine, D. M., Perduca, V., Perez-Cornago, A., Sakoda, L. C., Schumacher, F. R., Slattery, M. L., Toland, A. E., van Duijnhoven, F. J. B., Van Guelpen, B., Agudo, A., Albanes, D., Alonso, M. H., Anderson, K., Arnau-Collell, C., Arndt, V., Banbury, B. L., Bassik, M. C., Berndt, S. I., Bézieau, S., Bishop, D. T., Boehm, J., Boeing, H., Boutron-Ruault, M. C., Brenner, H., Brezina, S., Buch, S., Buchanan, D. D., Burnett-Hartman, A., Caan, B. J., Campbell, P. T., Carr, P. R., Castells, A., Castellví-Bel, S., Chan, A. T., Chang-Claude, J., Chanock, S. J., Curtis, K. R., de la Chapelle, A., Easton, D. F., English, D. R., Feskens, E. J. M., Gala, M., Gallinger, S. J., Gauderman, W. J., Giles, G. G., Goodman, P. J., Grady, W. M., Grove, J. S., Gsur, A., Gunter, M. J., Haile, R. W., Hampe, J., Hoffmeister, M., Hopper, J. L., Hsu, W. L., Huang, W. Y., Hudson, T. J., Jenab, M., Jenkins, M. A., Joshi, A. D., Keku, T. O., Kooperberg, C., Kühn, T., Küry, S., Le Marchand, L., Lejbkowicz, F., Li, C. I., Li, L., Lieb, W., Lindblom, A., Lindor, N. M., Männistö, S., Markowitz, S. D., Milne, R. L., Moreno, L., Murphy, N., Nassir, R., Offit, K., Ogino, S., Panico, S., Parfrey, P. S., Pearlman, R., Pharoah, P. D. P., Phipps, A. I., Platz, E. A., Potter, J. D., Prentice, R. L., Qi, L., Raskin, L., Rennert, G., Rennert, H. S., Riboli, E., Schafmayer, C., Schoen, R. E., Seminara, D., Song, M., Su, Y. R., Tangen, C. M., Thibodeau, S. N., Thomas, D. C., Trichopoulou, A., Ulrich, C. M., Visvanathan, K., Vodicka, P., Vodickova, L., Vymetalkova, V., Weigl, K., Weinstein, S. J., White, E., Wolk, A., Woods, M. O., Wu, A. H., Abecasis, G. R., Nickerson, D. A., Scacheri, P. C., Kundaje, A., Casey, G., Gruber, S. B., Hsu, L., Moreno, V., Hayes, R. B., Newcomb, P. A., & Peters, U. (2021). Genetic architectures of proximal and distal colorectal cancer are partly distinct. Gut, 70(7), 1325-1334. https://doi.org/10.1136/gutjnl-2020-321534
Huyghe, Jeroen R, et al. "Genetic architectures of proximal and distal colorectal cancer are partly distinct." Gut vol. 70,7 (2021): 1325-1334. doi: https://doi.org/10.1136/gutjnl-2020-321534
Huyghe JR, Harrison TA, Bien SA, Hampel H, Figueiredo JC, Schmit SL, Conti DV, Chen S, Qu C, Lin Y, Barfield R, Baron JA, Cross AJ, Diergaarde B, Duggan D, Harlid S, Imaz L, Kang HM, Levine DM, Perduca V, Perez-Cornago A, Sakoda LC, Schumacher FR, Slattery ML, Toland AE, van Duijnhoven FJB, Van Guelpen B, Agudo A, Albanes D, Alonso MH, Anderson K, Arnau-Collell C, Arndt V, Banbury BL, Bassik MC, Berndt SI, Bézieau S, Bishop DT, Boehm J, Boeing H, Boutron-Ruault MC, Brenner H, Brezina S, Buch S, Buchanan DD, Burnett-Hartman A, Caan BJ, Campbell PT, Carr PR, Castells A, Castellví-Bel S, Chan AT, Chang-Claude J, Chanock SJ, Curtis KR, de la Chapelle A, Easton DF, English DR, Feskens EJM, Gala M, Gallinger SJ, Gauderman WJ, Giles GG, Goodman PJ, Grady WM, Grove JS, Gsur A, Gunter MJ, Haile RW, Hampe J, Hoffmeister M, Hopper JL, Hsu WL, Huang WY, Hudson TJ, Jenab M, Jenkins MA, Joshi AD, Keku TO, Kooperberg C, Kühn T, Küry S, Le Marchand L, Lejbkowicz F, Li CI, Li L, Lieb W, Lindblom A, Lindor NM, Männistö S, Markowitz SD, Milne RL, Moreno L, Murphy N, Nassir R, Offit K, Ogino S, Panico S, Parfrey PS, Pearlman R, Pharoah PDP, Phipps AI, Platz EA, Potter JD, Prentice RL, Qi L, Raskin L, Rennert G, Rennert HS, Riboli E, Schafmayer C, Schoen RE, Seminara D, Song M, Su YR, Tangen CM, Thibodeau SN, Thomas DC, Trichopoulou A, Ulrich CM, Visvanathan K, Vodicka P, Vodickova L, Vymetalkova V, Weigl K, Weinstein SJ, White E, Wolk A, Woods MO, Wu AH, Abecasis GR, Nickerson DA, Scacheri PC, Kundaje A, Casey G, Gruber SB, Hsu L, Moreno V, Hayes RB, Newcomb PA, Peters U. Genetic architectures of proximal and distal colorectal cancer are partly distinct. Gut. 2021 Jul;70(7):1325-1334. doi: 10.1136/gutjnl-2020-321534. Epub 2021 Feb 25. PMID: 33632709; PMCID: PMC8223655.
Copy Download .nbib Format: NLM
Dietary Gluten Intake Is Not Associated With Risk of Inflammatory Bowel Disease in US Adults Without Celiac Disease.

Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association

Lopes EW, Lebwohl B, Burke KE, Ivey KL, Ananthakrishnan AN, Lochhead P, Richter JM, Ludvigsson JF, Willett WC, Chan AT, Khalili H.
PMID: 33775898
Clin Gastroenterol Hepatol. 2021 Mar 26; doi: 10.1016/j.cgh.2021.03.029. Epub 2021 Mar 26.

BACKGROUND & AIMS: Diet is thought to play a role in the development of inflammatory bowel disease (IBD), though it is unknown whether gluten intake confers risk of IBD. The aim of this study was to determine the relationship...

Cite
Lopes EW, Lebwohl B, Burke KE, et al. Dietary Gluten Intake Is Not Associated With Risk of Inflammatory Bowel Disease in US Adults Without Celiac Disease. Clin Gastroenterol Hepatol. 2021;doi: 10.1016/j.cgh.2021.03.029.
Lopes, E. W., Lebwohl, B., Burke, K. E., Ivey, K. L., Ananthakrishnan, A. N., Lochhead, P., Richter, J. M., Ludvigsson, J. F., Willett, W. C., Chan, A. T., & Khalili, H. (2021). Dietary Gluten Intake Is Not Associated With Risk of Inflammatory Bowel Disease in US Adults Without Celiac Disease. Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association, . https://doi.org/10.1016/j.cgh.2021.03.029
Lopes, Emily W, et al. "Dietary Gluten Intake Is Not Associated With Risk of Inflammatory Bowel Disease in US Adults Without Celiac Disease." Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association vol. (2021). doi: https://doi.org/10.1016/j.cgh.2021.03.029
Lopes EW, Lebwohl B, Burke KE, Ivey KL, Ananthakrishnan AN, Lochhead P, Richter JM, Ludvigsson JF, Willett WC, Chan AT, Khalili H. Dietary Gluten Intake Is Not Associated With Risk of Inflammatory Bowel Disease in US Adults Without Celiac Disease. Clin Gastroenterol Hepatol. 2021 Mar 26; doi: 10.1016/j.cgh.2021.03.029. Epub 2021 Mar 26. PMID: 33775898; PMCID: PMC8586848.
Copy Download .nbib Format: NLM
Long-term diet quality and its change in relation to late-life subjective cognitive decline.

The American journal of clinical nutrition

Yuan C, Cao Y, Ascherio A, Okereke OI, Zong G, Grodstein F, Hofman A, Willett WC.
PMID: 34562097
Am J Clin Nutr. 2022 Jan 11;115(1):232-243. doi: 10.1093/ajcn/nqab326.

BACKGROUND: Evidence regarding the role of diet quality, especially its change, in subjective cognitive decline (SCD) is scarce.OBJECTIVES: We aimed to examine associations of long-term diet quality scores, including the Alternate Mediterranean Diet (AMED), Dietary Approaches to Stop Hypertension...

Cite
Yuan C, Cao Y, Ascherio A, et al. Long-term diet quality and its change in relation to late-life subjective cognitive decline. Am J Clin Nutr. 2022;115(1):232-243doi: 10.1093/ajcn/nqab326.
Yuan, C., Cao, Y., Ascherio, A., Okereke, O. I., Zong, G., Grodstein, F., Hofman, A., & Willett, W. C. (2022). Long-term diet quality and its change in relation to late-life subjective cognitive decline. The American journal of clinical nutrition, 115(1), 232-243. https://doi.org/10.1093/ajcn/nqab326
Yuan, Changzheng, et al. "Long-term diet quality and its change in relation to late-life subjective cognitive decline." The American journal of clinical nutrition vol. 115,1 (2022): 232-243. doi: https://doi.org/10.1093/ajcn/nqab326
Yuan C, Cao Y, Ascherio A, Okereke OI, Zong G, Grodstein F, Hofman A, Willett WC. Long-term diet quality and its change in relation to late-life subjective cognitive decline. Am J Clin Nutr. 2022 Jan 11;115(1):232-243. doi: 10.1093/ajcn/nqab326. PMID: 34562097; PMCID: PMC8755100.
Copy Download .nbib Format: NLM
Prolactin and Risk of Epithelial Ovarian Cancer.

Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology

Hathaway CA, Rice MS, Townsend MK, Hankinson SE, Arslan AA, Buring JE, Hallmans G, Idahl A, Kubzansky LD, Lee IM, Lundin EA, Sluss PM, Zeleniuch-Jacquotte A, Tworoger SS.
PMID: 34244157
Cancer Epidemiol Biomarkers Prev. 2021 Sep;30(9):1652-1659. doi: 10.1158/1055-9965.EPI-21-0139. Epub 2021 Jul 08.

BACKGROUND: Prolactin is synthesized in the ovaries and may play a role in ovarian cancer etiology. One prior prospective study observed a suggestive positive association between prolactin levels and risk of ovarian cancer.METHODS: We conducted a pooled case-control study...

Cite
Hathaway CA, Rice MS, Townsend MK, et al. Prolactin and Risk of Epithelial Ovarian Cancer. Cancer Epidemiol Biomarkers Prev. 2021;30(9):1652-1659doi: 10.1158/1055-9965.EPI-21-0139.
Hathaway, C. A., Rice, M. S., Townsend, M. K., Hankinson, S. E., Arslan, A. A., Buring, J. E., Hallmans, G., Idahl, A., Kubzansky, L. D., Lee, I. M., Lundin, E. A., Sluss, P. M., Zeleniuch-Jacquotte, A., & Tworoger, S. S. (2021). Prolactin and Risk of Epithelial Ovarian Cancer. Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology, 30(9), 1652-1659. https://doi.org/10.1158/1055-9965.EPI-21-0139
Hathaway, Cassandra A, et al. "Prolactin and Risk of Epithelial Ovarian Cancer." Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology vol. 30,9 (2021): 1652-1659. doi: https://doi.org/10.1158/1055-9965.EPI-21-0139
Hathaway CA, Rice MS, Townsend MK, Hankinson SE, Arslan AA, Buring JE, Hallmans G, Idahl A, Kubzansky LD, Lee IM, Lundin EA, Sluss PM, Zeleniuch-Jacquotte A, Tworoger SS. Prolactin and Risk of Epithelial Ovarian Cancer. Cancer Epidemiol Biomarkers Prev. 2021 Sep;30(9):1652-1659. doi: 10.1158/1055-9965.EPI-21-0139. Epub 2021 Jul 08. PMID: 34244157; PMCID: PMC8419083.
Copy Download .nbib Format: NLM
Hair color and risk of keratinocyte carcinoma in US women and men.

Journal of the American Academy of Dermatology

Dai J, Li TY, Cho E, Qureshi AA, Li WQ.
PMID: 33716057
J Am Acad Dermatol. 2022 Jan;86(1):234-237. doi: 10.1016/j.jaad.2021.01.099. Epub 2021 Mar 11.

No abstract available.

Cite
Dai J, Li TY, Cho E, et al. Hair color and risk of keratinocyte carcinoma in US women and men. J Am Acad Dermatol. 2021;86(1):234-237doi: 10.1016/j.jaad.2021.01.099.
Dai, J., Li, T. Y., Cho, E., Qureshi, A. A., & Li, W. Q. (2022). Hair color and risk of keratinocyte carcinoma in US women and men. Journal of the American Academy of Dermatology, 86(1), 234-237. https://doi.org/10.1016/j.jaad.2021.01.099
Dai, Jin, et al. "Hair color and risk of keratinocyte carcinoma in US women and men." Journal of the American Academy of Dermatology vol. 86,1 (2022): 234-237. doi: https://doi.org/10.1016/j.jaad.2021.01.099
Dai J, Li TY, Cho E, Qureshi AA, Li WQ. Hair color and risk of keratinocyte carcinoma in US women and men. J Am Acad Dermatol. 2022 Jan;86(1):234-237. doi: 10.1016/j.jaad.2021.01.099. Epub 2021 Mar 11. PMID: 33716057; PMCID: PMC8433260.
Copy Download .nbib Format: NLM
Preexisting Type 2 Diabetes and Survival among Patients with Colorectal Cancer.

Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology

Yuan C, Zhang X, Babic A, Morales-Oyarvide V, Zhang Y, Smith-Warner SA, Wu K, Wang M, Wolpin BM, Meyerhardt JA, Chan AT, Hu FB, Fuchs CS, Ogino S, Giovannucci EL, Ng K.
PMID: 33531435
Cancer Epidemiol Biomarkers Prev. 2021 Apr;30(4):757-764. doi: 10.1158/1055-9965.EPI-20-1083. Epub 2021 Feb 02.

BACKGROUND: Type 2 diabetes increases risk of developing colorectal cancer, but the association of preexisting diabetes with colorectal cancer survival remains unclear.METHODS: We analyzed survival by diabetes status at cancer diagnosis among 4,038 patients with colorectal cancer from two...

Cite
Yuan C, Zhang X, Babic A, et al. Preexisting Type 2 Diabetes and Survival among Patients with Colorectal Cancer. Cancer Epidemiol Biomarkers Prev. 2021;30(4):757-764doi: 10.1158/1055-9965.EPI-20-1083.
Yuan, C., Zhang, X., Babic, A., Morales-Oyarvide, V., Zhang, Y., Smith-Warner, S. A., Wu, K., Wang, M., Wolpin, B. M., Meyerhardt, J. A., Chan, A. T., Hu, F. B., Fuchs, C. S., Ogino, S., Giovannucci, E. L., & Ng, K. (2021). Preexisting Type 2 Diabetes and Survival among Patients with Colorectal Cancer. Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology, 30(4), 757-764. https://doi.org/10.1158/1055-9965.EPI-20-1083
Yuan, Chen, et al. "Preexisting Type 2 Diabetes and Survival among Patients with Colorectal Cancer." Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology vol. 30,4 (2021): 757-764. doi: https://doi.org/10.1158/1055-9965.EPI-20-1083
Yuan C, Zhang X, Babic A, Morales-Oyarvide V, Zhang Y, Smith-Warner SA, Wu K, Wang M, Wolpin BM, Meyerhardt JA, Chan AT, Hu FB, Fuchs CS, Ogino S, Giovannucci EL, Ng K. Preexisting Type 2 Diabetes and Survival among Patients with Colorectal Cancer. Cancer Epidemiol Biomarkers Prev. 2021 Apr;30(4):757-764. doi: 10.1158/1055-9965.EPI-20-1083. Epub 2021 Feb 02. PMID: 33531435; PMCID: PMC8026573.
Copy Download .nbib Format: NLM
Timing of Aspirin Use in Colorectal Cancer Chemoprevention: A Prospective Cohort Study.

Journal of the National Cancer Institute

Zhang Y, Chan AT, Meyerhardt JA, Giovannucci EL.
PMID: 33528007
J Natl Cancer Inst. 2021 Jul 01;113(7):841-851. doi: 10.1093/jnci/djab009.

BACKGROUND: Prior epidemiological and intervention studies have not been able to separate independent effects of dose, timing, and duration of aspirin use in colorectal cancer (CRC) chemoprevention. We examined aspirin-based CRC chemoprevention according to timing in the Nurses' Health...

Cite
Zhang Y, Chan AT, Meyerhardt JA, et al. Timing of Aspirin Use in Colorectal Cancer Chemoprevention: A Prospective Cohort Study. J Natl Cancer Inst. 2021;113(7):841-851doi: 10.1093/jnci/djab009.
Zhang, Y., Chan, A. T., Meyerhardt, J. A., & Giovannucci, E. L. (2021). Timing of Aspirin Use in Colorectal Cancer Chemoprevention: A Prospective Cohort Study. Journal of the National Cancer Institute, 113(7), 841-851. https://doi.org/10.1093/jnci/djab009
Zhang, Yin, et al. "Timing of Aspirin Use in Colorectal Cancer Chemoprevention: A Prospective Cohort Study." Journal of the National Cancer Institute vol. 113,7 (2021): 841-851. doi: https://doi.org/10.1093/jnci/djab009
Zhang Y, Chan AT, Meyerhardt JA, Giovannucci EL. Timing of Aspirin Use in Colorectal Cancer Chemoprevention: A Prospective Cohort Study. J Natl Cancer Inst. 2021 Jul 01;113(7):841-851. doi: 10.1093/jnci/djab009. PMID: 33528007; PMCID: PMC8246825.
Copy Download .nbib Format: NLM
Automated percent mammographic density, mammographic texture variation, and risk of breast cancer: a nested case-control study.

NPJ breast cancer

Warner ET, Rice MS, Zeleznik OA, Fowler EE, Murthy D, Vachon CM, Bertrand KA, Rosner BA, Heine J, Tamimi RM.
PMID: 34059687
NPJ Breast Cancer. 2021 May 31;7(1):68. doi: 10.1038/s41523-021-00272-2.

Percent mammographic density (PMD) is a strong breast cancer risk factor, however, other mammographic features, such as V, the standard deviation (SD) of pixel intensity, may be associated with risk. We assessed whether PMD, automated PMD (APD), and V,...

Cite
Warner ET, Rice MS, Zeleznik OA, et al. Automated percent mammographic density, mammographic texture variation, and risk of breast cancer: a nested case-control study. NPJ Breast Cancer. 2021;7(1):68doi: 10.1038/s41523-021-00272-2.
Warner, E. T., Rice, M. S., Zeleznik, O. A., Fowler, E. E., Murthy, D., Vachon, C. M., Bertrand, K. A., Rosner, B. A., Heine, J., & Tamimi, R. M. (2021). Automated percent mammographic density, mammographic texture variation, and risk of breast cancer: a nested case-control study. NPJ breast cancer, 7(1), 68. https://doi.org/10.1038/s41523-021-00272-2
Warner, Erica T, et al. "Automated percent mammographic density, mammographic texture variation, and risk of breast cancer: a nested case-control study." NPJ breast cancer vol. 7,1 (2021): 68. doi: https://doi.org/10.1038/s41523-021-00272-2
Warner ET, Rice MS, Zeleznik OA, Fowler EE, Murthy D, Vachon CM, Bertrand KA, Rosner BA, Heine J, Tamimi RM. Automated percent mammographic density, mammographic texture variation, and risk of breast cancer: a nested case-control study. NPJ Breast Cancer. 2021 May 31;7(1):68. doi: 10.1038/s41523-021-00272-2. PMID: 34059687; PMCID: PMC8166859.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 159 entries