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Bone morphogenetic protein-4 and transforming growth factor-beta1 mechanisms in acute valvular response to supra-physiologic hemodynamic stresses.

World journal of cardiology

Sun L, Sucosky P.
PMID: 26131338
World J Cardiol. 2015 Jun 26;7(6):331-43. doi: 10.4330/wjc.v7.i6.331.

AIM: To explore ex vivo the role of bone morphogenetic protein-4 (BMP-4) and transforming growth factor-beta1 (TGF-β1) in acute valvular response to fluid shear stress (FSS) abnormalities.METHODS: Porcine valve leaflets were subjected ex vivo to physiologic FSS, supra-physiologic FSS...

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Sun L, Sucosky P. Bone morphogenetic protein-4 and transforming growth factor-beta1 mechanisms in acute valvular response to supra-physiologic hemodynamic stresses. World J Cardiol. 2015;7(6):331-43doi: 10.4330/wjc.v7.i6.331.
Sun, L., & Sucosky, P. (2015). Bone morphogenetic protein-4 and transforming growth factor-beta1 mechanisms in acute valvular response to supra-physiologic hemodynamic stresses. World journal of cardiology, 7(6), 331-43. https://doi.org/10.4330/wjc.v7.i6.331
Sun, Ling, and Sucosky, Philippe. "Bone morphogenetic protein-4 and transforming growth factor-beta1 mechanisms in acute valvular response to supra-physiologic hemodynamic stresses." World journal of cardiology vol. 7,6 (2015): 331-43. doi: https://doi.org/10.4330/wjc.v7.i6.331
Sun L, Sucosky P. Bone morphogenetic protein-4 and transforming growth factor-beta1 mechanisms in acute valvular response to supra-physiologic hemodynamic stresses. World J Cardiol. 2015 Jun 26;7(6):331-43. doi: 10.4330/wjc.v7.i6.331. PMID: 26131338; PMCID: PMC4478568.
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Effect of Atorvastatin and Pioglitazone on Plasma Levels of Adhesion Molecules in Non-Diabetic Patients With Hypertension or Stable Angina or Both.

Journal of clinical medicine research

Pattan V, Seth S, Jehangir W, Bhargava B, Maulik SK.
PMID: 26124907
J Clin Med Res. 2015 Aug;7(8):613-9. doi: 10.14740/jocmr2178e. Epub 2015 Jun 09.

BACKGROUND: It was to study the effect of atorvastatin, pioglitazone and their combination on plasma levels of adhesion molecules in patients with hypertension or stable angina or both.METHODS: It was an open-label, randomized parallel-group study. Forty-five atorvastatin-naive patients with...

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Pattan V, Seth S, Jehangir W, et al. Effect of Atorvastatin and Pioglitazone on Plasma Levels of Adhesion Molecules in Non-Diabetic Patients With Hypertension or Stable Angina or Both. J Clin Med Res. 2015;7(8):613-9doi: 10.14740/jocmr2178e.
Pattan, V., Seth, S., Jehangir, W., Bhargava, B., & Maulik, S. K. (2015). Effect of Atorvastatin and Pioglitazone on Plasma Levels of Adhesion Molecules in Non-Diabetic Patients With Hypertension or Stable Angina or Both. Journal of clinical medicine research, 7(8), 613-9. https://doi.org/10.14740/jocmr2178e
Pattan, Vishwanath, et al. "Effect of Atorvastatin and Pioglitazone on Plasma Levels of Adhesion Molecules in Non-Diabetic Patients With Hypertension or Stable Angina or Both." Journal of clinical medicine research vol. 7,8 (2015): 613-9. doi: https://doi.org/10.14740/jocmr2178e
Pattan V, Seth S, Jehangir W, Bhargava B, Maulik SK. Effect of Atorvastatin and Pioglitazone on Plasma Levels of Adhesion Molecules in Non-Diabetic Patients With Hypertension or Stable Angina or Both. J Clin Med Res. 2015 Aug;7(8):613-9. doi: 10.14740/jocmr2178e. Epub 2015 Jun 09. PMID: 26124907; PMCID: PMC4471748.
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Anti-inflammatory, tissue remodeling, immunomodulatory, and anticancer activities of oregano (.

Biochimie open

Han X, Parker TL.
PMID: 29450144
Biochim Open. 2017 Mar 03;4:73-77. doi: 10.1016/j.biopen.2017.02.005. eCollection 2017 Jun.

The use of oregano (

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Han X, Parker TL. Anti-inflammatory, tissue remodeling, immunomodulatory, and anticancer activities of oregano (. Biochim Open. 2017;4:73-77doi: 10.1016/j.biopen.2017.02.005.
Han, X., & Parker, T. L. (2017). Anti-inflammatory, tissue remodeling, immunomodulatory, and anticancer activities of oregano (. Biochimie open, 473-77. https://doi.org/10.1016/j.biopen.2017.02.005
Han, Xuesheng, and Parker, Tory L. "Anti-inflammatory, tissue remodeling, immunomodulatory, and anticancer activities of oregano (." Biochimie open vol. 4 (2017): 73-77. doi: https://doi.org/10.1016/j.biopen.2017.02.005
Han X, Parker TL. Anti-inflammatory, tissue remodeling, immunomodulatory, and anticancer activities of oregano (. Biochim Open. 2017 Mar 03;4:73-77. doi: 10.1016/j.biopen.2017.02.005. eCollection 2017 Jun. PMID: 29450144; PMCID: PMC5801825.
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Alterations in Circulating Adhesion Molecules in Acute Myocardial Infarction before and after Thrombolysis with Streptokinase.

Journal of cardiovascular and thoracic research

Saidi H, Vakilian M, Noori GH, Ghafouri HB, Abazarian N.
PMID: 24404343
J Cardiovasc Thorac Res. 2013;5(4):139-41. doi: 10.5681/jcvtr.2013.030. Epub 2013 Dec 05.

INTRODUCTION: The role of adhesion molecules in the development and progression of coronary atherosclerosis is inevitable. It is not clear yet whether these molecules increase or decrease in level after thrombolytic therapy. This study was designed to compare concentrations...

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Saidi H, Vakilian M, Noori GH, et al. Alterations in Circulating Adhesion Molecules in Acute Myocardial Infarction before and after Thrombolysis with Streptokinase. J Cardiovasc Thorac Res. 2013;5(4):139-41doi: 10.5681/jcvtr.2013.030.
Saidi, H., Vakilian, M., Noori, G. H., Ghafouri, H. B., & Abazarian, N. (2013). Alterations in Circulating Adhesion Molecules in Acute Myocardial Infarction before and after Thrombolysis with Streptokinase. Journal of cardiovascular and thoracic research, 5(4), 139-41. https://doi.org/10.5681/jcvtr.2013.030
Saidi, Hossein, et al. "Alterations in Circulating Adhesion Molecules in Acute Myocardial Infarction before and after Thrombolysis with Streptokinase." Journal of cardiovascular and thoracic research vol. 5,4 (2013): 139-41. doi: https://doi.org/10.5681/jcvtr.2013.030
Saidi H, Vakilian M, Noori GH, Ghafouri HB, Abazarian N. Alterations in Circulating Adhesion Molecules in Acute Myocardial Infarction before and after Thrombolysis with Streptokinase. J Cardiovasc Thorac Res. 2013;5(4):139-41. doi: 10.5681/jcvtr.2013.030. Epub 2013 Dec 05. PMID: 24404343; PMCID: PMC3883535.
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Cardiac troponin I exacerbates myocardial ischaemia/reperfusion injury by inducing the adhesion of monocytes to vascular endothelial cells via a TLR4/NF-κB-dependent pathway.

Clinical science (London, England : 1979)

Han Y, Liao X, Gao Z, Yang S, Chen C, Liu Y, Wang WE, Wu G, Chen X, Jose PA, Zhang Y, Zeng C.
PMID: 27682003
Clin Sci (Lond). 2016 Dec 01;130(24):2279-2293. doi: 10.1042/CS20160373. Epub 2016 Sep 28.

Cardiac troponin I (cTnI), a biomarker for myocardial damage and risk stratification, may be involved in the pathogenesis of cardiovascular diseases, which was ascribed to the effect of cTnI auto-antibodies. Whether or not cTnI itself has a direct impact...

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Han Y, Liao X, Gao Z, et al. Cardiac troponin I exacerbates myocardial ischaemia/reperfusion injury by inducing the adhesion of monocytes to vascular endothelial cells via a TLR4/NF-κB-dependent pathway. Clin Sci (Lond). 2016;130(24):2279-2293doi: 10.1042/CS20160373.
Han, Y., Liao, X., Gao, Z., Yang, S., Chen, C., Liu, Y., Wang, W. E., Wu, G., Chen, X., Jose, P. A., Zhang, Y., & Zeng, C. (2016). Cardiac troponin I exacerbates myocardial ischaemia/reperfusion injury by inducing the adhesion of monocytes to vascular endothelial cells via a TLR4/NF-κB-dependent pathway. Clinical science (London, England : 1979), 130(24), 2279-2293. https://doi.org/10.1042/CS20160373
Han, Yu, et al. "Cardiac troponin I exacerbates myocardial ischaemia/reperfusion injury by inducing the adhesion of monocytes to vascular endothelial cells via a TLR4/NF-κB-dependent pathway." Clinical science (London, England : 1979) vol. 130,24 (2016): 2279-2293. doi: https://doi.org/10.1042/CS20160373
Han Y, Liao X, Gao Z, Yang S, Chen C, Liu Y, Wang WE, Wu G, Chen X, Jose PA, Zhang Y, Zeng C. Cardiac troponin I exacerbates myocardial ischaemia/reperfusion injury by inducing the adhesion of monocytes to vascular endothelial cells via a TLR4/NF-κB-dependent pathway. Clin Sci (Lond). 2016 Dec 01;130(24):2279-2293. doi: 10.1042/CS20160373. Epub 2016 Sep 28. PMID: 27682003.
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A novel class of copper(II)- and zinc(II)-bound non-steroidal anti-inflammatory drugs that inhibits acute inflammation in vivo.

Cell & bioscience

Puranik R, Bao S, Bonin AM, Kaur R, Weder JE, Casbolt L, Hambley TW, Lay PA, Barter PJ, Rye KA.
PMID: 26855766
Cell Biosci. 2016 Feb 06;6:9. doi: 10.1186/s13578-016-0076-8. eCollection 2016.

BACKGROUND: The ability of Zn(II) and Cu(II) metal complexes of non-steroidal anti-inflammatory drugs (NSAIDs) to inhibit acute arterial inflammation in vivo has been studied.RESULTS: When acute vascular inflammation was induced in normocholesterolemic New Zealand White rabbits by inserting a...

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Puranik R, Bao S, Bonin AM, et al. A novel class of copper(II)- and zinc(II)-bound non-steroidal anti-inflammatory drugs that inhibits acute inflammation in vivo. Cell Biosci. 2016;6:9doi: 10.1186/s13578-016-0076-8.
Puranik, R., Bao, S., Bonin, A. M., Kaur, R., Weder, J. E., Casbolt, L., Hambley, T. W., Lay, P. A., Barter, P. J., & Rye, K. A. (2016). A novel class of copper(II)- and zinc(II)-bound non-steroidal anti-inflammatory drugs that inhibits acute inflammation in vivo. Cell & bioscience, 69. https://doi.org/10.1186/s13578-016-0076-8
Puranik, Rajesh, et al. "A novel class of copper(II)- and zinc(II)-bound non-steroidal anti-inflammatory drugs that inhibits acute inflammation in vivo." Cell & bioscience vol. 6 (2016): 9. doi: https://doi.org/10.1186/s13578-016-0076-8
Puranik R, Bao S, Bonin AM, Kaur R, Weder JE, Casbolt L, Hambley TW, Lay PA, Barter PJ, Rye KA. A novel class of copper(II)- and zinc(II)-bound non-steroidal anti-inflammatory drugs that inhibits acute inflammation in vivo. Cell Biosci. 2016 Feb 06;6:9. doi: 10.1186/s13578-016-0076-8. eCollection 2016. PMID: 26855766; PMCID: PMC4744413.
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Therapeutic effects of topical netrin-4 in a corneal acute inflammatory model.

International journal of ophthalmology

Han Y, Shao Y, Liu TT, Li SM, Li W, Liu ZG.
PMID: 25938032
Int J Ophthalmol. 2015 Apr 18;8(2):228-33. doi: 10.3980/j.issn.2222-3959.2015.02.03. eCollection 2015.

AIM: To evaluate the therapeutic effect of netrin-4 on the early acute phase of inflammation in the alkali-burned eye.METHODS: Eye drops containing netrin-4 or phosphate buffered saline (PBS) were administered to a alkali-burn-induced corneal acute inflammatory model four times...

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Han Y, Shao Y, Liu TT, et al. Therapeutic effects of topical netrin-4 in a corneal acute inflammatory model. Int J Ophthalmol. 2015;8(2):228-33doi: 10.3980/j.issn.2222-3959.2015.02.03.
Han, Y., Shao, Y., Liu, T. T., Li, S. M., Li, W., & Liu, Z. G. (2015). Therapeutic effects of topical netrin-4 in a corneal acute inflammatory model. International journal of ophthalmology, 8(2), 228-33. https://doi.org/10.3980/j.issn.2222-3959.2015.02.03
Han, Yun, et al. "Therapeutic effects of topical netrin-4 in a corneal acute inflammatory model." International journal of ophthalmology vol. 8,2 (2015): 228-33. doi: https://doi.org/10.3980/j.issn.2222-3959.2015.02.03
Han Y, Shao Y, Liu TT, Li SM, Li W, Liu ZG. Therapeutic effects of topical netrin-4 in a corneal acute inflammatory model. Int J Ophthalmol. 2015 Apr 18;8(2):228-33. doi: 10.3980/j.issn.2222-3959.2015.02.03. eCollection 2015. PMID: 25938032; PMCID: PMC4413598.
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Circulating cellular adhesion molecules ICAM-1, VCAM-1, P- and E-selectin in the prediction of cardiovascular disease in diabetes mellitus.

European journal of internal medicine

Jude EB, Douglas JT, Anderson SG, Young MJ, Boulton AJ.
PMID: 12020626
Eur J Intern Med. 2002 May;13(3):185-189. doi: 10.1016/s0953-6205(02)00014-6.

BACKGROUND: Macrovascular disease in diabetes mellitus is a multifactorial process resulting in part from accelerated atherosclerosis and increased thrombosis. The resultant cardiovascular mortality is up to five times that of an age-matched non-diabetic population. Recently, cell adhesion molecules (CAMs)...

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Jude EB, Douglas JT, Anderson SG, et al. Circulating cellular adhesion molecules ICAM-1, VCAM-1, P- and E-selectin in the prediction of cardiovascular disease in diabetes mellitus. Eur J Intern Med. 2002;13(3):185-189doi: 10.1016/s0953-6205(02)00014-6.
Jude, E. B., Douglas, J. T., Anderson, S. G., Young, M. J., & Boulton, A. J. (2002). Circulating cellular adhesion molecules ICAM-1, VCAM-1, P- and E-selectin in the prediction of cardiovascular disease in diabetes mellitus. European journal of internal medicine, 13(3), 185-189. https://doi.org/10.1016/s0953-6205(02)00014-6
Jude, Edward B., et al. "Circulating cellular adhesion molecules ICAM-1, VCAM-1, P- and E-selectin in the prediction of cardiovascular disease in diabetes mellitus." European journal of internal medicine vol. 13,3 (2002): 185-189. doi: https://doi.org/10.1016/s0953-6205(02)00014-6
Jude EB, Douglas JT, Anderson SG, Young MJ, Boulton AJ. Circulating cellular adhesion molecules ICAM-1, VCAM-1, P- and E-selectin in the prediction of cardiovascular disease in diabetes mellitus. Eur J Intern Med. 2002 May;13(3):185-189. doi: 10.1016/s0953-6205(02)00014-6. PMID: 12020626.
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Atheroprotective Mechanisms Activated by Fluid Shear Stress in Endothelial Cells.

Drug news & perspectives

Berk BC, Min W, Yan C, Surapisitchat J, Liu Y, Hoefen R.
PMID: 12677255
Drug News Perspect. 2002 Apr;15(3):133-139. doi: 10.1358/dnp.2002.15.3.704684.

Atherosclerosis preferentially occurs in areas of turbulent flow and low fluid shear stress, while laminar flow and high shear stress are atheroprotective. Well characterized atheroprotective mechanisms include inhibition of thrombosis (increased tissue-type plasminogen activator and decreased plasminogen activator inhibitor-1),...

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Berk BC, Min W, Yan C, et al. Atheroprotective Mechanisms Activated by Fluid Shear Stress in Endothelial Cells. Drug News Perspect. 2002;15(3):133-139doi: 10.1358/dnp.2002.15.3.704684.
Berk, B. C., Min, W., Yan, C., Surapisitchat, J., Liu, Y., & Hoefen, R. (2002). Atheroprotective Mechanisms Activated by Fluid Shear Stress in Endothelial Cells. Drug news & perspectives, 15(3), 133-139. https://doi.org/10.1358/dnp.2002.15.3.704684
Berk, Bradford C., et al. "Atheroprotective Mechanisms Activated by Fluid Shear Stress in Endothelial Cells." Drug news & perspectives vol. 15,3 (2002): 133-139. doi: https://doi.org/10.1358/dnp.2002.15.3.704684
Berk BC, Min W, Yan C, Surapisitchat J, Liu Y, Hoefen R. Atheroprotective Mechanisms Activated by Fluid Shear Stress in Endothelial Cells. Drug News Perspect. 2002 Apr;15(3):133-139. doi: 10.1358/dnp.2002.15.3.704684. PMID: 12677255.
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Plasma levels of soluble cellular adhesion molecules in patients with arterial hypertension. Correlations with plasma endothelin-1.

European journal of internal medicine

Parissis JT, Venetsanou KF, Mentzikof DG, Kalantzi MV, Georgopoulou MV, Chrisopoulos N, Karas SM.
PMID: 11395298
Eur J Intern Med. 2001 Jul;12(4):350-356. doi: 10.1016/s0953-6205(01)00125-x.

Background: Several reports have shown that circulating, soluble cellular adhesion molecules and endothelin-1 (ET-1) are implicated in the pathophysiological events of atherosclerosis and may reflect the endothelial dysfunction characterizing this disorder. Methods: To evaluate the expression of these factors...

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Parissis JT, Venetsanou KF, Mentzikof DG, et al. Plasma levels of soluble cellular adhesion molecules in patients with arterial hypertension. Correlations with plasma endothelin-1. Eur J Intern Med. 2001;12(4):350-356doi: 10.1016/s0953-6205(01)00125-x.
Parissis, J. T., Venetsanou, K. F., Mentzikof, D. G., Kalantzi, M. V., Georgopoulou, M. V., Chrisopoulos, N., & Karas, S. M. (2001). Plasma levels of soluble cellular adhesion molecules in patients with arterial hypertension. Correlations with plasma endothelin-1. European journal of internal medicine, 12(4), 350-356. https://doi.org/10.1016/s0953-6205(01)00125-x
Parissis, J T., et al. "Plasma levels of soluble cellular adhesion molecules in patients with arterial hypertension. Correlations with plasma endothelin-1." European journal of internal medicine vol. 12,4 (2001): 350-356. doi: https://doi.org/10.1016/s0953-6205(01)00125-x
Parissis JT, Venetsanou KF, Mentzikof DG, Kalantzi MV, Georgopoulou MV, Chrisopoulos N, Karas SM. Plasma levels of soluble cellular adhesion molecules in patients with arterial hypertension. Correlations with plasma endothelin-1. Eur J Intern Med. 2001 Jul;12(4):350-356. doi: 10.1016/s0953-6205(01)00125-x. PMID: 11395298.
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Monocyte/macrophage and T-cell infiltrates in peritoneum of patients with ovarian cancer or benign pelvic disease.

Journal of translational medicine

Wang X, Deavers M, Patenia R, Bassett RL, Mueller P, Ma Q, Wang E, Freedman RS.
PMID: 16824216
J Transl Med. 2006 Jul 06;4:30. doi: 10.1186/1479-5876-4-30.

BACKGROUND: We previously showed that tumor-free peritoneum of patients with epithelial ovarian cancer (EOC) exhibited enhanced expression of several inflammatory response genes compared to peritoneum of benign disease. Here, we examined peritoneal inflammatory cell patterns to determine their concordance...

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Wang X, Deavers M, Patenia R, et al. Monocyte/macrophage and T-cell infiltrates in peritoneum of patients with ovarian cancer or benign pelvic disease. J Transl Med. 2006;4:30doi: 10.1186/1479-5876-4-30.
Wang, X., Deavers, M., Patenia, R., Bassett, R. L., Mueller, P., Ma, Q., Wang, E., & Freedman, R. S. (2006). Monocyte/macrophage and T-cell infiltrates in peritoneum of patients with ovarian cancer or benign pelvic disease. Journal of translational medicine, 430. https://doi.org/10.1186/1479-5876-4-30
Wang, Xipeng, et al. "Monocyte/macrophage and T-cell infiltrates in peritoneum of patients with ovarian cancer or benign pelvic disease." Journal of translational medicine vol. 4 (2006): 30. doi: https://doi.org/10.1186/1479-5876-4-30
Wang X, Deavers M, Patenia R, Bassett RL, Mueller P, Ma Q, Wang E, Freedman RS. Monocyte/macrophage and T-cell infiltrates in peritoneum of patients with ovarian cancer or benign pelvic disease. J Transl Med. 2006 Jul 06;4:30. doi: 10.1186/1479-5876-4-30. PMID: 16824216; PMCID: PMC1550428.
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Heparin Attenuates the Expression of TNFalpha-induced Cerebral Endothelial Cell Adhesion Molecule.

The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Lee JH, Kim CH, Seo GH, Lee J, Kim JH, Kim DG, Ahn YS.
PMID: 19967061
Korean J Physiol Pharmacol. 2008 Oct;12(5):231-6. doi: 10.4196/kjpp.2008.12.5.231. Epub 2008 Oct 31.

Heparin is a well-known anticoagulant widely used in various clinical settings. Interestingly, recent studies have indicated that heparin also has anti-inflammatory effects on neuroinflammation-related diseases, such as Alzheimer's disease and meningitis. However, the underlying mechanism of its actions remains...

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Lee JH, Kim CH, Seo GH, et al. Heparin Attenuates the Expression of TNFalpha-induced Cerebral Endothelial Cell Adhesion Molecule. Korean J Physiol Pharmacol. 2008;12(5):231-6doi: 10.4196/kjpp.2008.12.5.231.
Lee, J. H., Kim, C. H., Seo, G. H., Lee, J., Kim, J. H., Kim, D. G., & Ahn, Y. S. (2008). Heparin Attenuates the Expression of TNFalpha-induced Cerebral Endothelial Cell Adhesion Molecule. The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology, 12(5), 231-6. https://doi.org/10.4196/kjpp.2008.12.5.231
Lee, Jeong Ho, et al. "Heparin Attenuates the Expression of TNFalpha-induced Cerebral Endothelial Cell Adhesion Molecule." The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology vol. 12,5 (2008): 231-6. doi: https://doi.org/10.4196/kjpp.2008.12.5.231
Lee JH, Kim CH, Seo GH, Lee J, Kim JH, Kim DG, Ahn YS. Heparin Attenuates the Expression of TNFalpha-induced Cerebral Endothelial Cell Adhesion Molecule. Korean J Physiol Pharmacol. 2008 Oct;12(5):231-6. doi: 10.4196/kjpp.2008.12.5.231. Epub 2008 Oct 31. PMID: 19967061; PMCID: PMC2788641.
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Showing 1 to 12 of 457 entries