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Takagi M, Uno H, Nishi R, et al. ATM Regulates Adipocyte Differentiation and Contributes to Glucose Homeostasis. Cell Rep. 2015;10(6):957-967doi: 10.1016/j.celrep.2015.01.027.
Takagi, M., Uno, H., Nishi, R., Sugimoto, M., Hasegawa, S., Piao, J., Ihara, N., Kanai, S., Kakei, S., Tamura, Y., Suganami, T., Kamei, Y., Shimizu, T., Yasuda, A., Ogawa, Y., & Mizutani, S. (2015). ATM Regulates Adipocyte Differentiation and Contributes to Glucose Homeostasis. Cell reports, 10(6), 957-967. https://doi.org/10.1016/j.celrep.2015.01.027
Takagi, Masatoshi, et al. "ATM Regulates Adipocyte Differentiation and Contributes to Glucose Homeostasis." Cell reports vol. 10,6 (2015): 957-967. doi: https://doi.org/10.1016/j.celrep.2015.01.027
Takagi M, Uno H, Nishi R, Sugimoto M, Hasegawa S, Piao J, Ihara N, Kanai S, Kakei S, Tamura Y, Suganami T, Kamei Y, Shimizu T, Yasuda A, Ogawa Y, Mizutani S. ATM Regulates Adipocyte Differentiation and Contributes to Glucose Homeostasis. Cell Rep. 2015 Feb 17;10(6):957-967. doi: 10.1016/j.celrep.2015.01.027. Epub 2015 Feb 12. PMID: 25683718.
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Cell Rep. 2015 Feb 17;10(6):957-967. doi: 10.1016/j.celrep.2015.01.027. Epub 2015 Feb 12.

ATM Regulates Adipocyte Differentiation and Contributes to Glucose Homeostasis.

Cell reports

Masatoshi Takagi, Hatsume Uno, Rina Nishi, Masataka Sugimoto, Setsuko Hasegawa, Jinhua Piao, Norimasa Ihara, Sayaka Kanai, Saori Kakei, Yoshifumi Tamura, Takayoshi Suganami, Yasutomi Kamei, Toshiaki Shimizu, Akio Yasuda, Yoshihiro Ogawa, Shuki Mizutani

Affiliations

  1. Department of Pediatrics and Developmental Biology, Graduate School of Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo 113-8519, Japan. Electronic address: [email protected].
  2. Department of Pediatrics and Developmental Biology, Graduate School of Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo 113-8519, Japan; Medical Business Unit, Sony Corporation, 1-5-45 Yushima, Bunkyo-ku, Tokyo 113-8519, Japan.
  3. Department of Pediatrics and Developmental Biology, Graduate School of Medicine, Tokyo Medical and Dental University, 1-5-45 Yushima, Bunkyo-ku, Tokyo 113-8519, Japan.
  4. Section of Biochemistry, National Institute for Longevity Sciences, NCGG, 36-3 Gengo, Moriokacho, Obu, Aichi 474-8522, Japan.
  5. Department of Reproductive Biology, National Research Institute for Child Health and Development, 2-10-1 Ohkura, Setagaya-ku, Tokyo 157-0074, Japan.
  6. Department of Molecular Endocrinology and Metabolism, Graduate School of Medicine, Tokyo Medical and Dental University, Japan Science and Technology Agency, CREST, 1-5-45 Yushima, Bunkyo-ku, Tokyo 113-8519, Japan.
  7. Department of Medicine, Metabolism & Endocrinology, Juntendo University, School of Medicine, 2-1-1 Hongo, Bunkyo-ku, Tokyo, 113-0033, Japan.
  8. Department of Organ Network and Metabolism, Graduate School of Medical and Dental Sciences, Tokyo Medical and Dental University, Japan Science and Technology Agency, PRESTO, 1-5-45 Yushima, Bunkyo-ku, Tokyo 113-8519, Japan.
  9. Laboratory of Molecular Nutrition, Graduate School of Environmental and Life Science, Kyoto Prefectural University, Hangicho 1-5, Shimogamo, Kyotoshi, Kyoto 606-8522, Japan.
  10. Department of Pediatrics and Adolescent Medicine, Juntendo University, School of Medicine, 2-1-1 Hongo, Bunkyo-ku, Tokyo 113-0033, Japan.

PMID: 25683718 DOI: 10.1016/j.celrep.2015.01.027

Abstract

Ataxia-telangiectasia (A-T) patients occasionally develop diabetes mellitus. However, only limited attempts have been made to gain insight into the molecular mechanism of diabetes mellitus development in A-T patients. We found that Atm

Copyright © 2015 The Authors. Published by Elsevier Inc. All rights reserved.

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