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Wu SY, Huang JH, Chen WY, et al. Cell Intrinsic Galectin-3 Attenuates Neutrophil ROS-Dependent Killing of . Front Immunol. 2017;8:48doi: 10.3389/fimmu.2017.00048.
Wu, S. Y., Huang, J. H., Chen, W. Y., Chan, Y. C., Lin, C. H., Chen, Y. C., Liu, F. T., & Wu-Hsieh, B. A. (2017). Cell Intrinsic Galectin-3 Attenuates Neutrophil ROS-Dependent Killing of . Frontiers in immunology, 848. https://doi.org/10.3389/fimmu.2017.00048
Wu, Sheng-Yang, et al. "Cell Intrinsic Galectin-3 Attenuates Neutrophil ROS-Dependent Killing of ." Frontiers in immunology vol. 8 (2017): 48. doi: https://doi.org/10.3389/fimmu.2017.00048
Wu SY, Huang JH, Chen WY, Chan YC, Lin CH, Chen YC, Liu FT, Wu-Hsieh BA. Cell Intrinsic Galectin-3 Attenuates Neutrophil ROS-Dependent Killing of . Front Immunol. 2017 Feb 03;8:48. doi: 10.3389/fimmu.2017.00048. eCollection 2017. PMID: 28217127; PMCID: PMC5289966.
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Front Immunol. 2017 Feb 03;8:48. doi: 10.3389/fimmu.2017.00048. eCollection 2017.

Cell Intrinsic Galectin-3 Attenuates Neutrophil ROS-Dependent Killing of .

Frontiers in immunology

Sheng-Yang Wu, Juin-Hua Huang, Wen-Yu Chen, Yi-Chen Chan, Chun-Hung Lin, Yee-Chun Chen, Fu-Tong Liu, Betty A Wu-Hsieh

Affiliations

  1. Graduate Institute of Immunology, National Taiwan University College of Medicine , Taipei , Taiwan.
  2. Institute of Biological Chemistry, Academia Sinica , Taipei , Taiwan.
  3. Department of Internal Medicine, National Taiwan University Hospital , Taipei , Taiwan.
  4. Institute of Biomedical Sciences, Academia Sinica , Taipei , Taiwan.

PMID: 28217127 PMCID: PMC5289966 DOI: 10.3389/fimmu.2017.00048

Abstract

Invasive candidiasis is a leading cause of nosocomial bloodstream infection. Neutrophils are the important effector cells in host resistance to candidiasis. To investigate the modulation of neutrophil fungicidal function will advance our knowledge on the control of candidiasis. While recombinant galectin-3 enhances neutrophil phagocytosis of

Keywords: Candida albicans; M2 macrophage; Syk kinase; TD139; galectin-3; monocyte/dendritic cell recruitment; neutrophil; reactive oxygen species

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