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Ma L, Ainsworth HC, Snipes JA, et al. APOL1 Kidney-Risk Variants Induce Mitochondrial Fission . Kidney Int Rep. 2020;5(6):891-904doi: 10.1016/j.ekir.2020.03.020.
Ma, L., Ainsworth, H. C., Snipes, J. A., Murea, M., Choi, Y. A., Langefeld, C. D., Parks, J. S., Bharadwaj, M. S., Chou, J. W., Hemal, A. K., Petrovic, S., Craddock, A. L., Cheng, D., Hawkins, G. A., Miller, L. D., Hicks, P. J., Saleem, M. A., Divers, J., Molina, A. J. A., & Freedman, B. I. (2020). APOL1 Kidney-Risk Variants Induce Mitochondrial Fission . Kidney international reports, 5(6), 891-904. https://doi.org/10.1016/j.ekir.2020.03.020
Ma, Lijun, et al. "APOL1 Kidney-Risk Variants Induce Mitochondrial Fission ." Kidney international reports vol. 5,6 (2020): 891-904. doi: https://doi.org/10.1016/j.ekir.2020.03.020
Ma L, Ainsworth HC, Snipes JA, Murea M, Choi YA, Langefeld CD, Parks JS, Bharadwaj MS, Chou JW, Hemal AK, Petrovic S, Craddock AL, Cheng D, Hawkins GA, Miller LD, Hicks PJ, Saleem MA, Divers J, Molina AJA, Freedman BI. APOL1 Kidney-Risk Variants Induce Mitochondrial Fission . Kidney Int Rep. 2020 Mar 30;5(6):891-904. doi: 10.1016/j.ekir.2020.03.020. eCollection 2020 Jun. PMID: 32518871; PMCID: PMC7271005.
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Kidney Int Rep. 2020 Mar 30;5(6):891-904. doi: 10.1016/j.ekir.2020.03.020. eCollection 2020 Jun.

APOL1 Kidney-Risk Variants Induce Mitochondrial Fission .

Kidney international reports

Lijun Ma, Hannah C Ainsworth, James A Snipes, Mariana Murea, Young A Choi, Carl D Langefeld, John S Parks, Manish S Bharadwaj, Jeff W Chou, Ashok K Hemal, Snezana Petrovic, Ann L Craddock, Dongmei Cheng, Gregory A Hawkins, Lance D Miller, Pamela J Hicks, Moin A Saleem, Jasmin Divers, Anthony J A Molina, Barry I Freedman

Affiliations

  1. Department of Internal Medicine, Section on Nephrology, Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  2. Division of Public Health Sciences, Department of Biostatistics and Data Science, Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  3. Department of Internal Medicine, Section on Molecular Medicine, Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  4. Department of Internal Medicine, Section on Gerontology and Geriatric Medicine, Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  5. Department of Urology, Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  6. Department of Cancer Biology, Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  7. Department of Biochemistry, Wake Forest School of Medicine, Winston-Salem, North Carolina, USA.
  8. Children's Renal Unit, Bristol Royal Hospital for Children, University of Bristol, Bristol, United Kingdom.

PMID: 32518871 PMCID: PMC7271005 DOI: 10.1016/j.ekir.2020.03.020

Abstract

CONCLUSION: Results suggest the mitochondrial fusion/fission pathway may be a therapeutic target in

© 2020 International Society of Nephrology. Published by Elsevier Inc.

Keywords: APOL1; African Americans; FSGS; chronic kidney disease; mitochondria

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