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Functionalization of silicone rubber for the covalent immobilization of fibronectin.

Journal of materials science. Materials in medicine

Völcker N, Klee D, Höcker H, Langefeld S.
PMID: 15348316
J Mater Sci Mater Med. 2001 Feb;12(2):111-9. doi: 10.1023/a:1008938525489.

Surface modification techniques were employed in order to provide functionalized silicone rubber with enhanced cytocompatibility. Acrylic acid (AAc), methacrylic acid (MAAc) and glycidylmethacrylate (GMA) were graft-co-polymerized onto the surface of silicone induced by an argon plasma and thermal initiation....

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Völcker N, Klee D, Höcker H, et al. Functionalization of silicone rubber for the covalent immobilization of fibronectin. J Mater Sci Mater Med. 2001;12(2):111-9doi: 10.1023/a:1008938525489.
Völcker, N., Klee, D., Höcker, H., & Langefeld, S. (2001). Functionalization of silicone rubber for the covalent immobilization of fibronectin. Journal of materials science. Materials in medicine, 12(2), 111-9. https://doi.org/10.1023/a:1008938525489
Völcker, N, et al. "Functionalization of silicone rubber for the covalent immobilization of fibronectin." Journal of materials science. Materials in medicine vol. 12,2 (2001): 111-9. doi: https://doi.org/10.1023/a:1008938525489
Völcker N, Klee D, Höcker H, Langefeld S. Functionalization of silicone rubber for the covalent immobilization of fibronectin. J Mater Sci Mater Med. 2001 Feb;12(2):111-9. doi: 10.1023/a:1008938525489. PMID: 15348316.
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Genes associated with SLE are targets of recent positive selection.

Autoimmune diseases

Ramos PS, Shaftman SR, Ward RC, Langefeld CD.
PMID: 24587899
Autoimmune Dis. 2014;2014:203435. doi: 10.1155/2014/203435. Epub 2014 Jan 23.

The reasons for the ethnic disparities in the prevalence of systemic lupus erythematosus (SLE) and the relative high frequency of SLE risk alleles in the population are not fully understood. Population genetic factors such as natural selection alter allele...

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Ramos PS, Shaftman SR, Ward RC, et al. Genes associated with SLE are targets of recent positive selection. Autoimmune Dis. 2014;2014:203435doi: 10.1155/2014/203435.
Ramos, P. S., Shaftman, S. R., Ward, R. C., & Langefeld, C. D. (2014). Genes associated with SLE are targets of recent positive selection. Autoimmune diseases, 2014203435. https://doi.org/10.1155/2014/203435
Ramos, Paula S, et al. "Genes associated with SLE are targets of recent positive selection." Autoimmune diseases vol. 2014 (2014): 203435. doi: https://doi.org/10.1155/2014/203435
Ramos PS, Shaftman SR, Ward RC, Langefeld CD. Genes associated with SLE are targets of recent positive selection. Autoimmune Dis. 2014;2014:203435. doi: 10.1155/2014/203435. Epub 2014 Jan 23. PMID: 24587899; PMCID: PMC3920976.
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APOL1 Kidney-Risk Variants Induce Mitochondrial Fission .

Kidney international reports

Ma L, Ainsworth HC, Snipes JA, Murea M, Choi YA, Langefeld CD, Parks JS, Bharadwaj MS, Chou JW, Hemal AK, Petrovic S, Craddock AL, Cheng D, Hawkins GA, Miller LD, Hicks PJ, Saleem MA, Divers J, Molina AJA, Freedman BI.
PMID: 32518871
Kidney Int Rep. 2020 Mar 30;5(6):891-904. doi: 10.1016/j.ekir.2020.03.020. eCollection 2020 Jun.

CONCLUSION: Results suggest the mitochondrial fusion/fission pathway may be a therapeutic target in

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Ma L, Ainsworth HC, Snipes JA, et al. APOL1 Kidney-Risk Variants Induce Mitochondrial Fission . Kidney Int Rep. 2020;5(6):891-904doi: 10.1016/j.ekir.2020.03.020.
Ma, L., Ainsworth, H. C., Snipes, J. A., Murea, M., Choi, Y. A., Langefeld, C. D., Parks, J. S., Bharadwaj, M. S., Chou, J. W., Hemal, A. K., Petrovic, S., Craddock, A. L., Cheng, D., Hawkins, G. A., Miller, L. D., Hicks, P. J., Saleem, M. A., Divers, J., Molina, A. J. A., & Freedman, B. I. (2020). APOL1 Kidney-Risk Variants Induce Mitochondrial Fission . Kidney international reports, 5(6), 891-904. https://doi.org/10.1016/j.ekir.2020.03.020
Ma, Lijun, et al. "APOL1 Kidney-Risk Variants Induce Mitochondrial Fission ." Kidney international reports vol. 5,6 (2020): 891-904. doi: https://doi.org/10.1016/j.ekir.2020.03.020
Ma L, Ainsworth HC, Snipes JA, Murea M, Choi YA, Langefeld CD, Parks JS, Bharadwaj MS, Chou JW, Hemal AK, Petrovic S, Craddock AL, Cheng D, Hawkins GA, Miller LD, Hicks PJ, Saleem MA, Divers J, Molina AJA, Freedman BI. APOL1 Kidney-Risk Variants Induce Mitochondrial Fission . Kidney Int Rep. 2020 Mar 30;5(6):891-904. doi: 10.1016/j.ekir.2020.03.020. eCollection 2020 Jun. PMID: 32518871; PMCID: PMC7271005.
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TMTC2 variant associated with sensorineural hearing loss and auditory neuropathy spectrum disorder in a family dyad.

Molecular genetics & genomic medicine

Guillen-Ahlers H, Erbe CB, Chevalier FD, Montoya MJ, Zimmerman KD, Langefeld CD, Olivier M, Runge CL.
PMID: 29671961
Mol Genet Genomic Med. 2018 Apr 19; doi: 10.1002/mgg3.397. Epub 2018 Apr 19.

BACKGROUND: Sensorineural hearing loss (SNHL) is a common form of hearing loss that can be inherited or triggered by environmental insults; auditory neuropathy spectrum disorder (ANSD) is a SNHL subtype with unique diagnostic criteria. The genetic factors associated with...

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Guillen-Ahlers H, Erbe CB, Chevalier FD, et al. TMTC2 variant associated with sensorineural hearing loss and auditory neuropathy spectrum disorder in a family dyad. Mol Genet Genomic Med. 2018;doi: 10.1002/mgg3.397.
Guillen-Ahlers, H., Erbe, C. B., Chevalier, F. D., Montoya, M. J., Zimmerman, K. D., Langefeld, C. D., Olivier, M., & Runge, C. L. (2018). TMTC2 variant associated with sensorineural hearing loss and auditory neuropathy spectrum disorder in a family dyad. Molecular genetics & genomic medicine, . https://doi.org/10.1002/mgg3.397
Guillen-Ahlers, Hector, et al. "TMTC2 variant associated with sensorineural hearing loss and auditory neuropathy spectrum disorder in a family dyad." Molecular genetics & genomic medicine vol. (2018). doi: https://doi.org/10.1002/mgg3.397
Guillen-Ahlers H, Erbe CB, Chevalier FD, Montoya MJ, Zimmerman KD, Langefeld CD, Olivier M, Runge CL. TMTC2 variant associated with sensorineural hearing loss and auditory neuropathy spectrum disorder in a family dyad. Mol Genet Genomic Med. 2018 Apr 19; doi: 10.1002/mgg3.397. Epub 2018 Apr 19. PMID: 29671961; PMCID: PMC6081214.
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THE STRUCTURE AND FUNCTION OF DEPARTMENTS OF MEDICINE.

Transactions of the American Clinical and Climatological Association

Landefeld CS.
PMID: 28066053
Trans Am Clin Climatol Assoc. 2016;127:196-211.

The structure and function of departments of medicine are important for several reasons. First, departments of medicine are the biggest departments in virtually every medical school and in most universities with a medical school, and they are the largest...

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Landefeld CS. THE STRUCTURE AND FUNCTION OF DEPARTMENTS OF MEDICINE. Trans Am Clin Climatol Assoc. 2016;127:196-211
Landefeld, C. S. (2016). THE STRUCTURE AND FUNCTION OF DEPARTMENTS OF MEDICINE. Transactions of the American Clinical and Climatological Association, 127196-211.
Landefeld, C Seth. "THE STRUCTURE AND FUNCTION OF DEPARTMENTS OF MEDICINE." Transactions of the American Clinical and Climatological Association vol. 127 (2016): 196-211.
Landefeld CS. THE STRUCTURE AND FUNCTION OF DEPARTMENTS OF MEDICINE. Trans Am Clin Climatol Assoc. 2016;127:196-211. PMID: 28066053; PMCID: PMC5216493.
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Comparison of Genetic and Self-Identified Ancestry in Modeling Intracerebral Hemorrhage Risk.

Frontiers in neurology

Marini S, Lena UK, Crawford KM, Moomaw CJ, Testai FD, Kittner SJ, James ML, Woo D, Langefeld CD, Rosand J, Anderson CD.
PMID: 30034361
Front Neurol. 2018 Jul 06;9:514. doi: 10.3389/fneur.2018.00514. eCollection 2018.

No abstract available.

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Marini S, Lena UK, Crawford KM, et al. Comparison of Genetic and Self-Identified Ancestry in Modeling Intracerebral Hemorrhage Risk. Front Neurol. 2018;9:514doi: 10.3389/fneur.2018.00514.
Marini, S., Lena, U. K., Crawford, K. M., Moomaw, C. J., Testai, F. D., Kittner, S. J., James, M. L., Woo, D., Langefeld, C. D., Rosand, J., & Anderson, C. D. (2018). Comparison of Genetic and Self-Identified Ancestry in Modeling Intracerebral Hemorrhage Risk. Frontiers in neurology, 9514. https://doi.org/10.3389/fneur.2018.00514
Marini, Sandro, et al. "Comparison of Genetic and Self-Identified Ancestry in Modeling Intracerebral Hemorrhage Risk." Frontiers in neurology vol. 9 (2018): 514. doi: https://doi.org/10.3389/fneur.2018.00514
Marini S, Lena UK, Crawford KM, Moomaw CJ, Testai FD, Kittner SJ, James ML, Woo D, Langefeld CD, Rosand J, Anderson CD. Comparison of Genetic and Self-Identified Ancestry in Modeling Intracerebral Hemorrhage Risk. Front Neurol. 2018 Jul 06;9:514. doi: 10.3389/fneur.2018.00514. eCollection 2018. PMID: 30034361; PMCID: PMC6043667.
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Systemic inflammatory response syndrome, infection, and outcome in intracerebral hemorrhage.

Neurology(R) neuroimmunology & neuroinflammation

Boehme AK, Comeau ME, Langefeld CD, Lord A, Moomaw CJ, Osborne J, James ML, Martini S, Testai FD, Woo D, Elkind MSV.
PMID: 29318180
Neurol Neuroimmunol Neuroinflamm. 2017 Dec 22;5(2):e428. doi: 10.1212/NXI.0000000000000428. eCollection 2018 Mar.

OBJECTIVE: Systemic inflammatory response syndrome (SIRS) may be related to poor outcomes after intracerebral hemorrhage (ICH).METHODS: The Ethnic/Racial Variations of Intracerebral Hemorrhage study is an observational study of ICH in whites, blacks, and Hispanics throughout the United Sates. SIRS...

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Boehme AK, Comeau ME, Langefeld CD, et al. Systemic inflammatory response syndrome, infection, and outcome in intracerebral hemorrhage. Neurol Neuroimmunol Neuroinflamm. 2017;5(2):e428doi: 10.1212/NXI.0000000000000428.
Boehme, A. K., Comeau, M. E., Langefeld, C. D., Lord, A., Moomaw, C. J., Osborne, J., James, M. L., Martini, S., Testai, F. D., Woo, D., & Elkind, M. S. V. (2018). Systemic inflammatory response syndrome, infection, and outcome in intracerebral hemorrhage. Neurology(R) neuroimmunology & neuroinflammation, 5(2), e428. https://doi.org/10.1212/NXI.0000000000000428
Boehme, Amelia K, et al. "Systemic inflammatory response syndrome, infection, and outcome in intracerebral hemorrhage." Neurology(R) neuroimmunology & neuroinflammation vol. 5,2 (2018): e428. doi: https://doi.org/10.1212/NXI.0000000000000428
Boehme AK, Comeau ME, Langefeld CD, Lord A, Moomaw CJ, Osborne J, James ML, Martini S, Testai FD, Woo D, Elkind MSV. Systemic inflammatory response syndrome, infection, and outcome in intracerebral hemorrhage. Neurol Neuroimmunol Neuroinflamm. 2017 Dec 22;5(2):e428. doi: 10.1212/NXI.0000000000000428. eCollection 2018 Mar. PMID: 29318180; PMCID: PMC5745360.
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[No title available]

Kidney international reports

Freedman BI, Moxey-Mims MM, Alexander AA, Astor BC, Birdwell KA, Bowden DW, Bowen G, Bromberg J, Craven TE, Dadhania DM, Divers J, Doshi MD, Eidbo E, Fornoni A, Gautreaux MD, Gbadegesin RA, Gee PO, Guerra G, Hsu CY, Iltis AS, Jefferson N, Julian BA, Klassen DK, Koty PP, Langefeld CD, Lentine KL, Ma L, Mannon RB, Menon MC, Mohan S, Moore JB, Murphy B, Newell KA, Odim J, Ortigosa-Goggins M, Palmer ND, Park M, Parsa A, Pastan SO, Poggio ED, Rajapakse N, Reeves-Daniel AM, Rosas SE, Russell LP, Sawinski D, Smith SC, Spainhour M, Stratta RJ, Weir MR, Reboussin DM, Kimmel PL, Brennan DC.
PMID: 32154449
Kidney Int Rep. 2019 Dec 13;5(3):278-288. doi: 10.1016/j.ekir.2019.11.022. eCollection 2020 Mar.

INTRODUCTION: Much of the higher risk for end-stage kidney disease (ESKD) in African American individuals relates to ancestry-specific variation in the apolipoprotein L1 gene (METHODS: APOLLO will evaluate outcomes from 2614 deceased kidney donor-recipient pairs, as well as additional...

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Freedman BI, Moxey-Mims MM, Alexander AA, et al. . Kidney Int Rep. 2019;5(3):278-288doi: 10.1016/j.ekir.2019.11.022.
Freedman, B. I., Moxey-Mims, M. M., Alexander, A. A., Astor, B. C., Birdwell, K. A., Bowden, D. W., Bowen, G., Bromberg, J., Craven, T. E., Dadhania, D. M., Divers, J., Doshi, M. D., Eidbo, E., Fornoni, A., Gautreaux, M. D., Gbadegesin, R. A., Gee, P. O., Guerra, G., Hsu, C. Y., Iltis, A. S., Jefferson, N., Julian, B. A., Klassen, D. K., Koty, P. P., Langefeld, C. D., Lentine, K. L., Ma, L., Mannon, R. B., Menon, M. C., Mohan, S., Moore, J. B., Murphy, B., Newell, K. A., Odim, J., Ortigosa-Goggins, M., Palmer, N. D., Park, M., Parsa, A., Pastan, S. O., Poggio, E. D., Rajapakse, N., Reeves-Daniel, A. M., Rosas, S. E., Russell, L. P., Sawinski, D., Smith, S. C., Spainhour, M., Stratta, R. J., Weir, M. R., Reboussin, D. M., Kimmel, P. L., & Brennan, D. C. (2020). . Kidney international reports, 5(3), 278-288. https://doi.org/10.1016/j.ekir.2019.11.022
Freedman, Barry I, et al. "." Kidney international reports vol. 5,3 (2020): 278-288. doi: https://doi.org/10.1016/j.ekir.2019.11.022
Freedman BI, Moxey-Mims MM, Alexander AA, Astor BC, Birdwell KA, Bowden DW, Bowen G, Bromberg J, Craven TE, Dadhania DM, Divers J, Doshi MD, Eidbo E, Fornoni A, Gautreaux MD, Gbadegesin RA, Gee PO, Guerra G, Hsu CY, Iltis AS, Jefferson N, Julian BA, Klassen DK, Koty PP, Langefeld CD, Lentine KL, Ma L, Mannon RB, Menon MC, Mohan S, Moore JB, Murphy B, Newell KA, Odim J, Ortigosa-Goggins M, Palmer ND, Park M, Parsa A, Pastan SO, Poggio ED, Rajapakse N, Reeves-Daniel AM, Rosas SE, Russell LP, Sawinski D, Smith SC, Spainhour M, Stratta RJ, Weir MR, Reboussin DM, Kimmel PL, Brennan DC. . Kidney Int Rep. 2019 Dec 13;5(3):278-288. doi: 10.1016/j.ekir.2019.11.022. eCollection 2020 Mar. PMID: 32154449; PMCID: PMC7056919.
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Integrated Omics: Tools, Advances, and Future Approaches.

Journal of molecular endocrinology

Misra BB, Langefeld CD, Olivier M, Cox LA.
PMID: 30006342
J Mol Endocrinol. 2018 Jul 13; doi: 10.1530/JME-18-0055. Epub 2018 Jul 13.

With the rapid adoption of high-throughput omic approaches to analyze biological samples such as genomics, transcriptomics, proteomics, and metabolomics, each analysis can generate tera- to peta-byte sized data files on a daily basis. These data file sizes, together with...

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Misra BB, Langefeld CD, Olivier M, et al. Integrated Omics: Tools, Advances, and Future Approaches. J Mol Endocrinol. 2018;doi: 10.1530/JME-18-0055.
Misra, B. B., Langefeld, C. D., Olivier, M., & Cox, L. A. (2018). Integrated Omics: Tools, Advances, and Future Approaches. Journal of molecular endocrinology, . https://doi.org/10.1530/JME-18-0055
Misra, Biswapriya B, et al. "Integrated Omics: Tools, Advances, and Future Approaches." Journal of molecular endocrinology vol. (2018). doi: https://doi.org/10.1530/JME-18-0055
Misra BB, Langefeld CD, Olivier M, Cox LA. Integrated Omics: Tools, Advances, and Future Approaches. J Mol Endocrinol. 2018 Jul 13; doi: 10.1530/JME-18-0055. Epub 2018 Jul 13. PMID: 30006342.
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Antifibrotic factor KLF4 is repressed by the miR-10/TFAP2A/TBX5 axis in dermal fibroblasts: insights from twins discordant for systemic sclerosis.

Annals of the rheumatic diseases

Malaab M, Renaud L, Takamura N, Zimmerman KD, da Silveira WA, Ramos PS, Haddad S, Peters-Golden M, Penke LR, Wolf B, Hardiman G, Langefeld CD, Medsger TA, Feghali-Bostwick CA.
PMID: 34750102
Ann Rheum Dis. 2021 Nov 08; doi: 10.1136/annrheumdis-2021-221050. Epub 2021 Nov 08.

OBJECTIVES: Systemic sclerosis (SSc) is a complex disease of unknown aetiology in which inflammation and fibrosis lead to multiple organ damage. There is currently no effective therapy that can halt the progression of fibrosis or reverse it, thus studies...

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Malaab M, Renaud L, Takamura N, et al. Antifibrotic factor KLF4 is repressed by the miR-10/TFAP2A/TBX5 axis in dermal fibroblasts: insights from twins discordant for systemic sclerosis. Ann Rheum Dis. 2021;doi: 10.1136/annrheumdis-2021-221050.
Malaab, M., Renaud, L., Takamura, N., Zimmerman, K. D., da Silveira, W. A., Ramos, P. S., Haddad, S., Peters-Golden, M., Penke, L. R., Wolf, B., Hardiman, G., Langefeld, C. D., Medsger, T. A., & Feghali-Bostwick, C. A. (2021). Antifibrotic factor KLF4 is repressed by the miR-10/TFAP2A/TBX5 axis in dermal fibroblasts: insights from twins discordant for systemic sclerosis. Annals of the rheumatic diseases, . https://doi.org/10.1136/annrheumdis-2021-221050
Malaab, Maya, et al. "Antifibrotic factor KLF4 is repressed by the miR-10/TFAP2A/TBX5 axis in dermal fibroblasts: insights from twins discordant for systemic sclerosis." Annals of the rheumatic diseases vol. (2021). doi: https://doi.org/10.1136/annrheumdis-2021-221050
Malaab M, Renaud L, Takamura N, Zimmerman KD, da Silveira WA, Ramos PS, Haddad S, Peters-Golden M, Penke LR, Wolf B, Hardiman G, Langefeld CD, Medsger TA, Feghali-Bostwick CA. Antifibrotic factor KLF4 is repressed by the miR-10/TFAP2A/TBX5 axis in dermal fibroblasts: insights from twins discordant for systemic sclerosis. Ann Rheum Dis. 2021 Nov 08; doi: 10.1136/annrheumdis-2021-221050. Epub 2021 Nov 08. PMID: 34750102.
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miR-181a Mediates Inflammatory Gene Expression After Intracerebral Hemorrhage: An Integrated Analysis of miRNA-seq and mRNA-seq in a Swine ICH Model.

Journal of molecular neuroscience : MN

Walsh KB, Zimmerman KD, Zhang X, Demel SL, Luo Y, Langefeld CD, Wohleb E, Schulert G, Woo D, Adeoye O.
PMID: 33755911
J Mol Neurosci. 2021 Sep;71(9):1802-1814. doi: 10.1007/s12031-021-01815-9. Epub 2021 Mar 23.

Intracerebral hemorrhage (ICH) is a severe neurological disorder with no proven treatment. Inflammation after ICH contributes to clinical outcomes, but the relevant molecular mechanisms remain poorly understood. In studies of peripheral leukocyte counts and mRNA-sequencing (mRNA-seq), our group previously...

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Walsh KB, Zimmerman KD, Zhang X, et al. miR-181a Mediates Inflammatory Gene Expression After Intracerebral Hemorrhage: An Integrated Analysis of miRNA-seq and mRNA-seq in a Swine ICH Model. J Mol Neurosci. 2021;71(9):1802-1814doi: 10.1007/s12031-021-01815-9.
Walsh, K. B., Zimmerman, K. D., Zhang, X., Demel, S. L., Luo, Y., Langefeld, C. D., Wohleb, E., Schulert, G., Woo, D., & Adeoye, O. (2021). miR-181a Mediates Inflammatory Gene Expression After Intracerebral Hemorrhage: An Integrated Analysis of miRNA-seq and mRNA-seq in a Swine ICH Model. Journal of molecular neuroscience : MN, 71(9), 1802-1814. https://doi.org/10.1007/s12031-021-01815-9
Walsh, Kyle B, et al. "miR-181a Mediates Inflammatory Gene Expression After Intracerebral Hemorrhage: An Integrated Analysis of miRNA-seq and mRNA-seq in a Swine ICH Model." Journal of molecular neuroscience : MN vol. 71,9 (2021): 1802-1814. doi: https://doi.org/10.1007/s12031-021-01815-9
Walsh KB, Zimmerman KD, Zhang X, Demel SL, Luo Y, Langefeld CD, Wohleb E, Schulert G, Woo D, Adeoye O. miR-181a Mediates Inflammatory Gene Expression After Intracerebral Hemorrhage: An Integrated Analysis of miRNA-seq and mRNA-seq in a Swine ICH Model. J Mol Neurosci. 2021 Sep;71(9):1802-1814. doi: 10.1007/s12031-021-01815-9. Epub 2021 Mar 23. PMID: 33755911.
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Multiethnic Genome-Wide Association Study of Subclinical Atherosclerosis in Individuals With Type 2 Diabetes.

Circulation. Genomic and precision medicine

Lu Y, Dimitrov L, Chen SH, Bielak LF, Bis JC, Feitosa MF, Lu L, Kavousi M, Raffield LM, Smith AV, Wang L, Weiss S, Yao J, Zhu J, Gudmundsson EF, Gudmundsdottir V, Bos D, Ghanbari M, Ikram MA, Hwang SJ, Taylor KD, Budoff MJ, Gíslason GK, O'Donnell CJ, An P, Franceschini N, Freedman BI, Fu YP, Guo X, Heiss G, Kardia SLR, Wilson JG, Langefeld CD, Schminke U, Uitterlinden AG, Lange LA, Peyser PA, Gudnason VG, Psaty BM, Rotter JI, Bowden DW, Ng MCY.
PMID: 34241534
Circ Genom Precis Med. 2021 Aug;14(4):e003258. doi: 10.1161/CIRCGEN.120.003258. Epub 2021 Jul 09.

BACKGROUND: Coronary artery calcification (CAC) and carotid artery intima-media thickness (cIMT) are measures of subclinical atherosclerosis in asymptomatic individuals and strong risk factors for cardiovascular disease. Type 2 diabetes (T2D) is an independent cardiovascular disease risk factor that accelerates...

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Lu Y, Dimitrov L, Chen SH, et al. Multiethnic Genome-Wide Association Study of Subclinical Atherosclerosis in Individuals With Type 2 Diabetes. Circ Genom Precis Med. 2021;14(4):e003258doi: 10.1161/CIRCGEN.120.003258.
Lu, Y., Dimitrov, L., Chen, S. H., Bielak, L. F., Bis, J. C., Feitosa, M. F., Lu, L., Kavousi, M., Raffield, L. M., Smith, A. V., Wang, L., Weiss, S., Yao, J., Zhu, J., Gudmundsson, E. F., Gudmundsdottir, V., Bos, D., Ghanbari, M., Ikram, M. A., Hwang, S. J., Taylor, K. D., Budoff, M. J., Gíslason, G. K., O'Donnell, C. J., An, P., Franceschini, N., Freedman, B. I., Fu, Y. P., Guo, X., Heiss, G., Kardia, S. L. R., Wilson, J. G., Langefeld, C. D., Schminke, U., Uitterlinden, A. G., Lange, L. A., Peyser, P. A., Gudnason, V. G., Psaty, B. M., Rotter, J. I., Bowden, D. W., & Ng, M. C. Y. (2021). Multiethnic Genome-Wide Association Study of Subclinical Atherosclerosis in Individuals With Type 2 Diabetes. Circulation. Genomic and precision medicine, 14(4), e003258. https://doi.org/10.1161/CIRCGEN.120.003258
Lu, Yingchang, et al. "Multiethnic Genome-Wide Association Study of Subclinical Atherosclerosis in Individuals With Type 2 Diabetes." Circulation. Genomic and precision medicine vol. 14,4 (2021): e003258. doi: https://doi.org/10.1161/CIRCGEN.120.003258
Lu Y, Dimitrov L, Chen SH, Bielak LF, Bis JC, Feitosa MF, Lu L, Kavousi M, Raffield LM, Smith AV, Wang L, Weiss S, Yao J, Zhu J, Gudmundsson EF, Gudmundsdottir V, Bos D, Ghanbari M, Ikram MA, Hwang SJ, Taylor KD, Budoff MJ, Gíslason GK, O'Donnell CJ, An P, Franceschini N, Freedman BI, Fu YP, Guo X, Heiss G, Kardia SLR, Wilson JG, Langefeld CD, Schminke U, Uitterlinden AG, Lange LA, Peyser PA, Gudnason VG, Psaty BM, Rotter JI, Bowden DW, Ng MCY. Multiethnic Genome-Wide Association Study of Subclinical Atherosclerosis in Individuals With Type 2 Diabetes. Circ Genom Precis Med. 2021 Aug;14(4):e003258. doi: 10.1161/CIRCGEN.120.003258. Epub 2021 Jul 09. PMID: 34241534; PMCID: PMC8435075.
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Showing 1 to 12 of 66 entries