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Mizoguchi T, MacDonald BT, Bhandary B, et al. Coronary Disease Association With ADAMTS7 Is Due to Protease Activity. Circ Res. 2021;129(4):458-470doi: 10.1161/CIRCRESAHA.121.319163.
Mizoguchi, T., MacDonald, B. T., Bhandary, B., Popp, N. R., Laprise, D., Arduini, A., Lai, D., Zhu, Q. M., Xing, Y., Kaushik, V. K., Kathiresan, S., & Ellinor, P. T. (2021). Coronary Disease Association With ADAMTS7 Is Due to Protease Activity. Circulation research, 129(4), 458-470. https://doi.org/10.1161/CIRCRESAHA.121.319163
Mizoguchi, Taiji, et al. "Coronary Disease Association With ADAMTS7 Is Due to Protease Activity." Circulation research vol. 129,4 (2021): 458-470. doi: https://doi.org/10.1161/CIRCRESAHA.121.319163
Mizoguchi T, MacDonald BT, Bhandary B, Popp NR, Laprise D, Arduini A, Lai D, Zhu QM, Xing Y, Kaushik VK, Kathiresan S, Ellinor PT. Coronary Disease Association With ADAMTS7 Is Due to Protease Activity. Circ Res. 2021 Aug 06;129(4):458-470. doi: 10.1161/CIRCRESAHA.121.319163. Epub 2021 Jun 28. PMID: 34176299; PMCID: PMC8349873.
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Circ Res. 2021 Aug 06;129(4):458-470. doi: 10.1161/CIRCRESAHA.121.319163. Epub 2021 Jun 28.

Coronary Disease Association With ADAMTS7 Is Due to Protease Activity.

Circulation research

Taiji Mizoguchi, Bryan T MacDonald, Bidur Bhandary, Nicholas R Popp, Dylan Laprise, Alessandro Arduini, Daniel Lai, Qiuyu Martin Zhu, Yi Xing, Virendar K Kaushik, Sekar Kathiresan, Patrick T Ellinor

Affiliations

  1. Cardiovascular Disease Initiative (T.M., B.T.M., B.B., N.R.P., A.A., D.L., Q.M.Z., S.K., P.T.E.), Broad Institute of MIT and Harvard, Cambridge, MA.
  2. Now with Verve Therapeutics, Cambridge, MA, USA (T.M., S.K.).
  3. Center for the Development of Therapeutics (D.L., Y.X., V.K.K.), Broad Institute of MIT and Harvard, Cambridge, MA.
  4. Center for Genomic Medicine (Q.M.Z., S.K.), Massachusetts General Hospital, Boston.
  5. Cardiovascular Research Center (P.T.E.), Massachusetts General Hospital, Boston.

PMID: 34176299 PMCID: PMC8349873 DOI: 10.1161/CIRCRESAHA.121.319163

Abstract

[Figure: see text].

Keywords: atherosclerosis; coronary artery disease; phenotype; protease; wound healing

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