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The validity and internal structure of the Bipolar Depression Rating Scale: data from a clinical trial of N-acetylcysteine as adjunctive therapy in bipolar disorder.

Acta neuropsychiatrica

Berk M, Dodd S, Dean OM, Kohlmann K, Berk L, Malhi GS.
PMID: 26952834
Acta Neuropsychiatr. 2010 Oct;22(5):237-42. doi: 10.1111/j.1601-5215.2010.00472.x.

UNLABELLED: Berk M, Dodd S, Dean OM, Kohlmann K, Berk L, Malhi GS. The validity and internal structure of the Bipolar Depression Rating Scale: data from a clinical trial of N-acetylcysteine as adjunctive therapy in bipolar disorder.BACKGROUND: The phenomenology...

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Berk M, Dodd S, Dean OM, et al. The validity and internal structure of the Bipolar Depression Rating Scale: data from a clinical trial of N-acetylcysteine as adjunctive therapy in bipolar disorder. Acta Neuropsychiatr. 2010;22(5):237-42doi: 10.1111/j.1601-5215.2010.00472.x.
Berk, M., Dodd, S., Dean, O. M., Kohlmann, K., Berk, L., & Malhi, G. S. (2010). The validity and internal structure of the Bipolar Depression Rating Scale: data from a clinical trial of N-acetylcysteine as adjunctive therapy in bipolar disorder. Acta neuropsychiatrica, 22(5), 237-42. https://doi.org/10.1111/j.1601-5215.2010.00472.x
Berk, Michael, et al. "The validity and internal structure of the Bipolar Depression Rating Scale: data from a clinical trial of N-acetylcysteine as adjunctive therapy in bipolar disorder." Acta neuropsychiatrica vol. 22,5 (2010): 237-42. doi: https://doi.org/10.1111/j.1601-5215.2010.00472.x
Berk M, Dodd S, Dean OM, Kohlmann K, Berk L, Malhi GS. The validity and internal structure of the Bipolar Depression Rating Scale: data from a clinical trial of N-acetylcysteine as adjunctive therapy in bipolar disorder. Acta Neuropsychiatr. 2010 Oct;22(5):237-42. doi: 10.1111/j.1601-5215.2010.00472.x. PMID: 26952834.
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Supplementation with Phycocyanobilin, Citrulline, Taurine, and Supranutritional Doses of Folic Acid and Biotin-Potential for Preventing or Slowing the Progression of Diabetic Complications.

Healthcare (Basel, Switzerland)

McCarty MF.
PMID: 28335416
Healthcare (Basel). 2017 Mar 14;5(1). doi: 10.3390/healthcare5010015.

Oxidative stress, the resulting uncoupling of endothelial nitric oxide synthase (eNOS), and loss of nitric oxide (NO) bioactivity, are key mediators of the vascular and microvascular complications of diabetes. Much of this oxidative stress arises from up-regulated nicotinamide adenine...

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McCarty MF. Supplementation with Phycocyanobilin, Citrulline, Taurine, and Supranutritional Doses of Folic Acid and Biotin-Potential for Preventing or Slowing the Progression of Diabetic Complications. Healthcare (Basel). 2017;5(1)doi: 10.3390/healthcare5010015.
McCarty, M. F. (2017). Supplementation with Phycocyanobilin, Citrulline, Taurine, and Supranutritional Doses of Folic Acid and Biotin-Potential for Preventing or Slowing the Progression of Diabetic Complications. Healthcare (Basel, Switzerland), 5(1), . https://doi.org/10.3390/healthcare5010015
McCarty, Mark F. "Supplementation with Phycocyanobilin, Citrulline, Taurine, and Supranutritional Doses of Folic Acid and Biotin-Potential for Preventing or Slowing the Progression of Diabetic Complications." Healthcare (Basel, Switzerland) vol. 5,1 (2017). doi: https://doi.org/10.3390/healthcare5010015
McCarty MF. Supplementation with Phycocyanobilin, Citrulline, Taurine, and Supranutritional Doses of Folic Acid and Biotin-Potential for Preventing or Slowing the Progression of Diabetic Complications. Healthcare (Basel). 2017 Mar 14;5(1). doi: 10.3390/healthcare5010015. PMID: 28335416; PMCID: PMC5371921.
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Estrogen inhibits starvation-induced apoptosis in osteocytes by a redox-independent process involving association of JNK and glutathione S-transferase P1-1.

FEBS open bio

Domazetovic V, Fontani F, Marcucci G, Iantomasi T, Brandi ML, Vincenzini MT.
PMID: 28469982
FEBS Open Bio. 2017 Apr 05;7(5):705-718. doi: 10.1002/2211-5463.12216. eCollection 2017 May.

Estrogen deficiency causes bone loss as a result of microdamage, oxidative stress, and osteocyte apoptosis. A relationship between oxidative stress-induced apoptosis, c-Jun N-terminal kinase (JNK) activation, and expression of factors involved in bone remodeling has been demonstrated in osteocytes....

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Domazetovic V, Fontani F, Marcucci G, et al. Estrogen inhibits starvation-induced apoptosis in osteocytes by a redox-independent process involving association of JNK and glutathione S-transferase P1-1. FEBS Open Bio. 2017;7(5):705-718doi: 10.1002/2211-5463.12216.
Domazetovic, V., Fontani, F., Marcucci, G., Iantomasi, T., Brandi, M. L., & Vincenzini, M. T. (2017). Estrogen inhibits starvation-induced apoptosis in osteocytes by a redox-independent process involving association of JNK and glutathione S-transferase P1-1. FEBS open bio, 7(5), 705-718. https://doi.org/10.1002/2211-5463.12216
Domazetovic, Vladana, et al. "Estrogen inhibits starvation-induced apoptosis in osteocytes by a redox-independent process involving association of JNK and glutathione S-transferase P1-1." FEBS open bio vol. 7,5 (2017): 705-718. doi: https://doi.org/10.1002/2211-5463.12216
Domazetovic V, Fontani F, Marcucci G, Iantomasi T, Brandi ML, Vincenzini MT. Estrogen inhibits starvation-induced apoptosis in osteocytes by a redox-independent process involving association of JNK and glutathione S-transferase P1-1. FEBS Open Bio. 2017 Apr 05;7(5):705-718. doi: 10.1002/2211-5463.12216. eCollection 2017 May. PMID: 28469982; PMCID: PMC5407897.
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NADPH Oxidase Activity in Cerebral Arterioles Is a Key Mediator of Cerebral Small Vessel Disease-Implications for Prevention.

Healthcare (Basel, Switzerland)

McCarty MF.
PMID: 27417759
Healthcare (Basel). 2015 Apr 15;3(2):233-51. doi: 10.3390/healthcare3020233.

Cerebral small vessel disease (SVD), a common feature of brain aging, is characterized by lacunar infarcts, microbleeds, leukoaraiosis, and a leaky blood-brain barrier. Functionally, it is associated with cognitive decline, dementia, depression, gait abnormalities, and increased risk for stroke....

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McCarty MF. NADPH Oxidase Activity in Cerebral Arterioles Is a Key Mediator of Cerebral Small Vessel Disease-Implications for Prevention. Healthcare (Basel). 2015;3(2):233-51doi: 10.3390/healthcare3020233.
McCarty, M. F. (2015). NADPH Oxidase Activity in Cerebral Arterioles Is a Key Mediator of Cerebral Small Vessel Disease-Implications for Prevention. Healthcare (Basel, Switzerland), 3(2), 233-51. https://doi.org/10.3390/healthcare3020233
McCarty, Mark F. "NADPH Oxidase Activity in Cerebral Arterioles Is a Key Mediator of Cerebral Small Vessel Disease-Implications for Prevention." Healthcare (Basel, Switzerland) vol. 3,2 (2015): 233-51. doi: https://doi.org/10.3390/healthcare3020233
McCarty MF. NADPH Oxidase Activity in Cerebral Arterioles Is a Key Mediator of Cerebral Small Vessel Disease-Implications for Prevention. Healthcare (Basel). 2015 Apr 15;3(2):233-51. doi: 10.3390/healthcare3020233. PMID: 27417759; PMCID: PMC4939544.
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Organochlorine Pesticide-Mediated Induction of NADPH Oxidase and Nitric-Oxide Synthase in Endothelial Cell.

Journal of clinical and diagnostic research : JCDR

Ghosh R, Siddharth M, Singh N, Kare PK, Banerjee BD, Wadhwa N, Tripathi AK.
PMID: 28273962
J Clin Diagn Res. 2017 Jan;11(1):BC09-BC12. doi: 10.7860/JCDR/2017/25276.9315. Epub 2017 Jan 01.

INTRODUCTION: Organochlorine Pesticides (OCPs) are detected ubiquitously in human and have been shown to be associated with Cardiovascular Disease (CVD) and atherosclerosis.AIM: To find out the effect of organochlorine pesticides in endothelial cell with regard to oxidative stress and...

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Ghosh R, Siddharth M, Singh N, et al. Organochlorine Pesticide-Mediated Induction of NADPH Oxidase and Nitric-Oxide Synthase in Endothelial Cell. J Clin Diagn Res. 2017;11(1):BC09-BC12doi: 10.7860/JCDR/2017/25276.9315.
Ghosh, R., Siddharth, M., Singh, N., Kare, P. K., Banerjee, B. D., Wadhwa, N., & Tripathi, A. K. (2017). Organochlorine Pesticide-Mediated Induction of NADPH Oxidase and Nitric-Oxide Synthase in Endothelial Cell. Journal of clinical and diagnostic research : JCDR, 11(1), BC09-BC12. https://doi.org/10.7860/JCDR/2017/25276.9315
Ghosh, Rishila, et al. "Organochlorine Pesticide-Mediated Induction of NADPH Oxidase and Nitric-Oxide Synthase in Endothelial Cell." Journal of clinical and diagnostic research : JCDR vol. 11,1 (2017): BC09-BC12. doi: https://doi.org/10.7860/JCDR/2017/25276.9315
Ghosh R, Siddharth M, Singh N, Kare PK, Banerjee BD, Wadhwa N, Tripathi AK. Organochlorine Pesticide-Mediated Induction of NADPH Oxidase and Nitric-Oxide Synthase in Endothelial Cell. J Clin Diagn Res. 2017 Jan;11(1):BC09-BC12. doi: 10.7860/JCDR/2017/25276.9315. Epub 2017 Jan 01. PMID: 28273962; PMCID: PMC5324407.
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N-acetylcysteine prevents TNF-induced mitochondrial damage, apoptosis and viral particle production in HIV-infected U937 cells.

Redox report : communications in free radical research

Malorni W, Rivabene R, Santini MT, Rainaldi G, Donelli G.
PMID: 27414179
Redox Rep. 1994 Oct;1(1):57-64. doi: 10.1080/13510002.1994.11746957.

It has been hypothesized that reactive oxygen intermediates (ROI) can activate human immunodeficiency virus (HIV) replication and that HIV can trigger programmed cell death (PCD). In this work, we studied PCD in U937 cultured cells chronically infected with HIV...

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Malorni W, Rivabene R, Santini MT, et al. N-acetylcysteine prevents TNF-induced mitochondrial damage, apoptosis and viral particle production in HIV-infected U937 cells. Redox Rep. 1994;1(1):57-64doi: 10.1080/13510002.1994.11746957.
Malorni, W., Rivabene, R., Santini, M. T., Rainaldi, G., & Donelli, G. (1994). N-acetylcysteine prevents TNF-induced mitochondrial damage, apoptosis and viral particle production in HIV-infected U937 cells. Redox report : communications in free radical research, 1(1), 57-64. https://doi.org/10.1080/13510002.1994.11746957
Malorni, W, et al. "N-acetylcysteine prevents TNF-induced mitochondrial damage, apoptosis and viral particle production in HIV-infected U937 cells." Redox report : communications in free radical research vol. 1,1 (1994): 57-64. doi: https://doi.org/10.1080/13510002.1994.11746957
Malorni W, Rivabene R, Santini MT, Rainaldi G, Donelli G. N-acetylcysteine prevents TNF-induced mitochondrial damage, apoptosis and viral particle production in HIV-infected U937 cells. Redox Rep. 1994 Oct;1(1):57-64. doi: 10.1080/13510002.1994.11746957. PMID: 27414179.
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A Case Report of Toxic Brain Syndrome Caused by Methyl Bromide.

Turkish journal of anaesthesiology and reanimation

Büyükçoban S, Arıcı MA, Koca U, Kalkan Ş.
PMID: 27366483
Turk J Anaesthesiol Reanim. 2015 Apr;43(2):134-7. doi: 10.5152/TJAR.2014.84756. Epub 2015 Feb 05.

Methyl bromide (CH3Br) is a halogenated aliphatic hydrocarbon that may cause acute and chronic toxicities. We describe a case of a 44-year-old male patient who developed toxic brain syndrome (TBS) and central nervous system (CNS) toxicity after exposure to...

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Büyükçoban S, Arıcı MA, Koca U, et al. A Case Report of Toxic Brain Syndrome Caused by Methyl Bromide. Turk J Anaesthesiol Reanim. 2015;43(2):134-7doi: 10.5152/TJAR.2014.84756.
Büyükçoban, S., Arıcı, M. A., Koca, U., & Kalkan, �. �. (2015). A Case Report of Toxic Brain Syndrome Caused by Methyl Bromide. Turkish journal of anaesthesiology and reanimation, 43(2), 134-7. https://doi.org/10.5152/TJAR.2014.84756
Büyükçoban, Sibel, et al. "A Case Report of Toxic Brain Syndrome Caused by Methyl Bromide." Turkish journal of anaesthesiology and reanimation vol. 43,2 (2015): 134-7. doi: https://doi.org/10.5152/TJAR.2014.84756
Büyükçoban S, Arıcı MA, Koca U, Kalkan Ş. A Case Report of Toxic Brain Syndrome Caused by Methyl Bromide. Turk J Anaesthesiol Reanim. 2015 Apr;43(2):134-7. doi: 10.5152/TJAR.2014.84756. Epub 2015 Feb 05. PMID: 27366483; PMCID: PMC4917157.
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Absorption of Etoposide (VP-16-213) from the Small Intestine of the Rat. The Potential Role of Mucus as an Absorption Rate Limiting Barrier.

Pharmaceutical research

Schurgers N, de Blaey CJ, Crommelin DJ.
PMID: 24272806
Pharm Res. 1985 Jul;2(4):162-5. doi: 10.1023/A:1016331904482.

The absorption of etoposide (VP-16-213) was investigated in a perfused intestinal loop. The absorbed amount of drug was determined by collecting the blood draining the loop via cannulation of the efferent jejunal vein. The absorption rate of VP-16-213 strongly...

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Schurgers N, de Blaey CJ, Crommelin DJ. Absorption of Etoposide (VP-16-213) from the Small Intestine of the Rat. The Potential Role of Mucus as an Absorption Rate Limiting Barrier. Pharm Res. 1985;2(4):162-5doi: 10.1023/A:1016331904482.
Schurgers, N., de Blaey, C. J., & Crommelin, D. J. (1985). Absorption of Etoposide (VP-16-213) from the Small Intestine of the Rat. The Potential Role of Mucus as an Absorption Rate Limiting Barrier. Pharmaceutical research, 2(4), 162-5. https://doi.org/10.1023/A:1016331904482
Schurgers, N, et al. "Absorption of Etoposide (VP-16-213) from the Small Intestine of the Rat. The Potential Role of Mucus as an Absorption Rate Limiting Barrier." Pharmaceutical research vol. 2,4 (1985): 162-5. doi: https://doi.org/10.1023/A:1016331904482
Schurgers N, de Blaey CJ, Crommelin DJ. Absorption of Etoposide (VP-16-213) from the Small Intestine of the Rat. The Potential Role of Mucus as an Absorption Rate Limiting Barrier. Pharm Res. 1985 Jul;2(4):162-5. doi: 10.1023/A:1016331904482. PMID: 24272806.
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Protective effect of NAC against malathion-induced oxidative stress in freshly isolated rat hepatocytes.

Advanced pharmaceutical bulletin

Mostafalou S, Abdollahi M, Eghbal MA, Saeedi Kouzehkonani N.
PMID: 24312774
Adv Pharm Bull. 2012;2(1):79-88. doi: 10.5681/apb.2012.011. Epub 2012 Mar 15.

PURPOSE: Induction of oxidative stress by Organophosphate compounds (OPs) has been previously reported. In the present work, the mechanism of protective effects of N-acetylcysteine as a glutathion (GSH) prodrug against malathion-induced cell toxicity was investigated. In this work, freshly...

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Mostafalou S, Abdollahi M, Eghbal MA, et al. Protective effect of NAC against malathion-induced oxidative stress in freshly isolated rat hepatocytes. Adv Pharm Bull. 2012;2(1):79-88doi: 10.5681/apb.2012.011.
Mostafalou, S., Abdollahi, M., Eghbal, M. A., & Saeedi Kouzehkonani, N. (2012). Protective effect of NAC against malathion-induced oxidative stress in freshly isolated rat hepatocytes. Advanced pharmaceutical bulletin, 2(1), 79-88. https://doi.org/10.5681/apb.2012.011
Mostafalou, Sara, et al. "Protective effect of NAC against malathion-induced oxidative stress in freshly isolated rat hepatocytes." Advanced pharmaceutical bulletin vol. 2,1 (2012): 79-88. doi: https://doi.org/10.5681/apb.2012.011
Mostafalou S, Abdollahi M, Eghbal MA, Saeedi Kouzehkonani N. Protective effect of NAC against malathion-induced oxidative stress in freshly isolated rat hepatocytes. Adv Pharm Bull. 2012;2(1):79-88. doi: 10.5681/apb.2012.011. Epub 2012 Mar 15. PMID: 24312774; PMCID: PMC3846007.
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Exposed subcutaneous implantable devices: an operative protocol for management and salvage.

Plastic and reconstructive surgery. Global open

Toia F, D'Arpa S, Cordova A, Moschella F.
PMID: 26034650
Plast Reconstr Surg Glob Open. 2015 Apr 07;3(3):e343. doi: 10.1097/GOX.0000000000000303. eCollection 2015 Mar.

BACKGROUND: Implantable venous and electrical devices are prone to exposure and infection. Indications for management are controversial, but-especially if infected-exposed devices are often removed and an additional operation is needed to replace the device, causing a delay in chemotherapy...

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Toia F, D'Arpa S, Cordova A, et al. Exposed subcutaneous implantable devices: an operative protocol for management and salvage. Plast Reconstr Surg Glob Open. 2015;3(3):e343doi: 10.1097/GOX.0000000000000303.
Toia, F., D'Arpa, S., Cordova, A., & Moschella, F. (2015). Exposed subcutaneous implantable devices: an operative protocol for management and salvage. Plastic and reconstructive surgery. Global open, 3(3), e343. https://doi.org/10.1097/GOX.0000000000000303
Toia, Francesca, et al. "Exposed subcutaneous implantable devices: an operative protocol for management and salvage." Plastic and reconstructive surgery. Global open vol. 3,3 (2015): e343. doi: https://doi.org/10.1097/GOX.0000000000000303
Toia F, D'Arpa S, Cordova A, Moschella F. Exposed subcutaneous implantable devices: an operative protocol for management and salvage. Plast Reconstr Surg Glob Open. 2015 Apr 07;3(3):e343. doi: 10.1097/GOX.0000000000000303. eCollection 2015 Mar. PMID: 26034650; PMCID: PMC4448718.
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Oxidative stress modulation in hepatitis C virus infected cells.

World journal of hepatology

Lozano-Sepulveda SA, Bryan-Marrugo OL, Cordova-Fletes C, Gutierrez-Ruiz MC, Rivas-Estilla AM.
PMID: 26692473
World J Hepatol. 2015 Dec 18;7(29):2880-9. doi: 10.4254/wjh.v7.i29.2880.

Hepatitis C virus (HCV) replication is associated with the endoplasmic reticulum, where the virus can induce cellular stress. Oxidative cell damage plays an important role in HCV physiopathology. Oxidative stress is triggered when the concentration of oxygen species in...

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Lozano-Sepulveda SA, Bryan-Marrugo OL, Cordova-Fletes C, et al. Oxidative stress modulation in hepatitis C virus infected cells. World J Hepatol. 2015;7(29):2880-9doi: 10.4254/wjh.v7.i29.2880.
Lozano-Sepulveda, S. A., Bryan-Marrugo, O. L., Cordova-Fletes, C., Gutierrez-Ruiz, M. C., & Rivas-Estilla, A. M. (2015). Oxidative stress modulation in hepatitis C virus infected cells. World journal of hepatology, 7(29), 2880-9. https://doi.org/10.4254/wjh.v7.i29.2880
Lozano-Sepulveda, Sonia A, et al. "Oxidative stress modulation in hepatitis C virus infected cells." World journal of hepatology vol. 7,29 (2015): 2880-9. doi: https://doi.org/10.4254/wjh.v7.i29.2880
Lozano-Sepulveda SA, Bryan-Marrugo OL, Cordova-Fletes C, Gutierrez-Ruiz MC, Rivas-Estilla AM. Oxidative stress modulation in hepatitis C virus infected cells. World J Hepatol. 2015 Dec 18;7(29):2880-9. doi: 10.4254/wjh.v7.i29.2880. PMID: 26692473; PMCID: PMC4678374.
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The effect of N-acetylcysteine on the microscopic fluid dynamics of rat tears.

Clinical & experimental optometry

Tragoulias ST, Anderton PJ.
PMID: 12366370
Clin Exp Optom. 2001 Sep;84(5):282-286. doi: 10.1111/j.1444-0938.2001.tb05039.x.

BACKGROUND: The classical view of the tear film is of a 10-micron film of aqueous tears, sandwiched between thin layers of lipid and mucus. This has been challenged recently by the revelation that the tear film may be considerably...

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Tragoulias ST, Anderton PJ. The effect of N-acetylcysteine on the microscopic fluid dynamics of rat tears. Clin Exp Optom. 2001;84(5):282-286doi: 10.1111/j.1444-0938.2001.tb05039.x.
Tragoulias, S. T., & Anderton, P. J. (2001). The effect of N-acetylcysteine on the microscopic fluid dynamics of rat tears. Clinical & experimental optometry, 84(5), 282-286. https://doi.org/10.1111/j.1444-0938.2001.tb05039.x
Tragoulias, Sophia T, and Anderton, Philip J. "The effect of N-acetylcysteine on the microscopic fluid dynamics of rat tears." Clinical & experimental optometry vol. 84,5 (2001): 282-286. doi: https://doi.org/10.1111/j.1444-0938.2001.tb05039.x
Tragoulias ST, Anderton PJ. The effect of N-acetylcysteine on the microscopic fluid dynamics of rat tears. Clin Exp Optom. 2001 Sep;84(5):282-286. doi: 10.1111/j.1444-0938.2001.tb05039.x. PMID: 12366370.
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Showing 25 to 36 of 1089 entries