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Evaluation of the results of treatment of morbid obesity by the endoscopic intragastric balloon implantation method.

Wideochirurgia i inne techniki maloinwazyjne = Videosurgery and other miniinvasive techniques

Żurawiński W, Sokołowski D, Krupa-Kotara K, Czech E, Sosada K.
PMID: 28446931
Wideochir Inne Tech Maloinwazyjne. 2017;12(1):37-48. doi: 10.5114/wiitm.2017.66856. Epub 2017 Mar 30.

INTRODUCTION: Overweight and obesity are ranked in the fifth place among the risk factors responsible for the greatest number of deaths in the world.AIM: To assess the effects of treatment of patients with morbid obesity using endoscopic intragastric balloon...

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Żurawiński W, Sokołowski D, Krupa-Kotara K, et al. Evaluation of the results of treatment of morbid obesity by the endoscopic intragastric balloon implantation method. Wideochir Inne Tech Maloinwazyjne. 2017;12(1):37-48doi: 10.5114/wiitm.2017.66856.
Żurawiński, W., Sokołowski, D., Krupa-Kotara, K., Czech, E., & Sosada, K. (2017). Evaluation of the results of treatment of morbid obesity by the endoscopic intragastric balloon implantation method. Wideochirurgia i inne techniki maloinwazyjne = Videosurgery and other miniinvasive techniques, 12(1), 37-48. https://doi.org/10.5114/wiitm.2017.66856
Żurawiński, Wojciech, et al. "Evaluation of the results of treatment of morbid obesity by the endoscopic intragastric balloon implantation method." Wideochirurgia i inne techniki maloinwazyjne = Videosurgery and other miniinvasive techniques vol. 12,1 (2017): 37-48. doi: https://doi.org/10.5114/wiitm.2017.66856
Żurawiński W, Sokołowski D, Krupa-Kotara K, Czech E, Sosada K. Evaluation of the results of treatment of morbid obesity by the endoscopic intragastric balloon implantation method. Wideochir Inne Tech Maloinwazyjne. 2017;12(1):37-48. doi: 10.5114/wiitm.2017.66856. Epub 2017 Mar 30. PMID: 28446931; PMCID: PMC5397553.
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Single plasmon spatial and spectral sorting on a crystalline two-dimensional plasmonic platform.

Nanoscale

Kumar U, Bolisetty S, Mezzenga R, Girard C, Dujardin E, Cuche A.
PMID: 32614011
Nanoscale. 2020 Jul 02;12(25):13414-13420. doi: 10.1039/d0nr02066d.

In the context of the emerging field of quantum plasmonics, we demonstrate in this manuscript the wavelength-dependent propagation and sorting of single plasmons launched in a two-dimensional crystalline gold flake by a broadband quantum nanoemitter. The stream of single...

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Kumar U, Bolisetty S, Mezzenga R, et al. Single plasmon spatial and spectral sorting on a crystalline two-dimensional plasmonic platform. Nanoscale. 2020;12(25):13414-13420doi: 10.1039/d0nr02066d.
Kumar, U., Bolisetty, S., Mezzenga, R., Girard, C., Dujardin, E., & Cuche, A. (2020). Single plasmon spatial and spectral sorting on a crystalline two-dimensional plasmonic platform. Nanoscale, 12(25), 13414-13420. https://doi.org/10.1039/d0nr02066d
Kumar, Upkar, et al. "Single plasmon spatial and spectral sorting on a crystalline two-dimensional plasmonic platform." Nanoscale vol. 12,25 (2020): 13414-13420. doi: https://doi.org/10.1039/d0nr02066d
Kumar U, Bolisetty S, Mezzenga R, Girard C, Dujardin E, Cuche A. Single plasmon spatial and spectral sorting on a crystalline two-dimensional plasmonic platform. Nanoscale. 2020 Jul 02;12(25):13414-13420. doi: 10.1039/d0nr02066d. PMID: 32614011.
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Symposium 3: Non-enzymatic biocatalysts in nature and biotechnology.

Fresenius' journal of analytical chemistry

Woodson SA, Cech TR, Young M, Perriman R, Kelly L, Graf L, Llewellyn D, Gerlach W, Plückthun A, Collins J.
PMID: 32226232
Fresenius J Anal Chem. 1990;337(1):12-14. doi: 10.1007/BF00325711.

No abstract available.

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Woodson SA, Cech TR, Young M, et al. Symposium 3: Non-enzymatic biocatalysts in nature and biotechnology. Fresenius J Anal Chem. 1990;337(1):12-14doi: 10.1007/BF00325711.
Woodson, S. A., Cech, T. R., Young, M., Perriman, R., Kelly, L., Graf, L., Llewellyn, D., Gerlach, W., Plückthun, A., & Collins, J. (1990). Symposium 3: Non-enzymatic biocatalysts in nature and biotechnology. Fresenius' journal of analytical chemistry, 337(1), 12-14. https://doi.org/10.1007/BF00325711
Woodson, Sarah A, et al. "Symposium 3: Non-enzymatic biocatalysts in nature and biotechnology." Fresenius' journal of analytical chemistry vol. 337,1 (1990): 12-14. doi: https://doi.org/10.1007/BF00325711
Woodson SA, Cech TR, Young M, Perriman R, Kelly L, Graf L, Llewellyn D, Gerlach W, Plückthun A, Collins J. Symposium 3: Non-enzymatic biocatalysts in nature and biotechnology. Fresenius J Anal Chem. 1990;337(1):12-14. doi: 10.1007/BF00325711. PMID: 32226232; PMCID: PMC7100648.
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Commentary: A statistical revolution: Channeling frustration to integration.

The Journal of thoracic and cardiovascular surgery

Cech A, Mokadam NA.
PMID: 32741626
J Thorac Cardiovasc Surg. 2021 Oct;162(4):1171-1172. doi: 10.1016/j.jtcvs.2020.07.015. Epub 2020 Jul 10.

No abstract available.

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Cech A, Mokadam NA. Commentary: A statistical revolution: Channeling frustration to integration. J Thorac Cardiovasc Surg. 2020;162(4):1171-1172doi: 10.1016/j.jtcvs.2020.07.015.
Cech, A., & Mokadam, N. A. (2021). Commentary: A statistical revolution: Channeling frustration to integration. The Journal of thoracic and cardiovascular surgery, 162(4), 1171-1172. https://doi.org/10.1016/j.jtcvs.2020.07.015
Cech, Austin, and Mokadam, Nahush A. "Commentary: A statistical revolution: Channeling frustration to integration." The Journal of thoracic and cardiovascular surgery vol. 162,4 (2021): 1171-1172. doi: https://doi.org/10.1016/j.jtcvs.2020.07.015
Cech A, Mokadam NA. Commentary: A statistical revolution: Channeling frustration to integration. J Thorac Cardiovasc Surg. 2021 Oct;162(4):1171-1172. doi: 10.1016/j.jtcvs.2020.07.015. Epub 2020 Jul 10. PMID: 32741626.
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Homozygous WASHC4 variant in two sisters causes a syndromic phenotype defined by dysmorphisms, intellectual disability, profound developmental disorder, and skeletal muscle involvement.

The Journal of pathology

Gangfuß A, Czech A, Hentschel A, Münchberg U, Horvath R, Töpf A, O'Heir E, Lochmüller H, Stehling F, Kiewert C, Sickmann A, Kuechler A, Kaiser FJ, Kölbel H, Christiansen J, Schara-Schmidt U, Roos A.
PMID: 34599609
J Pathol. 2022 Jan;256(1):93-107. doi: 10.1002/path.5812. Epub 2021 Nov 18.

Recessive variants in WASHC4 are linked to intellectual disability complicated by poor language skills, short stature, and dysmorphic features. The protein encoded by WASHC4 is part of the Wiskott-Aldrich syndrome protein and SCAR homolog family, co-localizes with actin in...

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Gangfuß A, Czech A, Hentschel A, et al. Homozygous WASHC4 variant in two sisters causes a syndromic phenotype defined by dysmorphisms, intellectual disability, profound developmental disorder, and skeletal muscle involvement. J Pathol. 2021;256(1):93-107doi: 10.1002/path.5812.
Gangfuß, A., Czech, A., Hentschel, A., Münchberg, U., Horvath, R., Töpf, A., O'Heir, E., Lochmüller, H., Stehling, F., Kiewert, C., Sickmann, A., Kuechler, A., Kaiser, F. J., Kölbel, H., Christiansen, J., Schara-Schmidt, U., & Roos, A. (2022). Homozygous WASHC4 variant in two sisters causes a syndromic phenotype defined by dysmorphisms, intellectual disability, profound developmental disorder, and skeletal muscle involvement. The Journal of pathology, 256(1), 93-107. https://doi.org/10.1002/path.5812
Gangfuß, Andrea, et al. "Homozygous WASHC4 variant in two sisters causes a syndromic phenotype defined by dysmorphisms, intellectual disability, profound developmental disorder, and skeletal muscle involvement." The Journal of pathology vol. 256,1 (2022): 93-107. doi: https://doi.org/10.1002/path.5812
Gangfuß A, Czech A, Hentschel A, Münchberg U, Horvath R, Töpf A, O'Heir E, Lochmüller H, Stehling F, Kiewert C, Sickmann A, Kuechler A, Kaiser FJ, Kölbel H, Christiansen J, Schara-Schmidt U, Roos A. Homozygous WASHC4 variant in two sisters causes a syndromic phenotype defined by dysmorphisms, intellectual disability, profound developmental disorder, and skeletal muscle involvement. J Pathol. 2022 Jan;256(1):93-107. doi: 10.1002/path.5812. Epub 2021 Nov 18. PMID: 34599609.
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Interlaboratory Comparison of Untargeted Mass Spectrometry Data Uncovers Underlying Causes for Variability.

Journal of natural products

Clark TN, Houriet J, Vidar WS, Kellogg JJ, Todd DA, Cech NB, Linington RG.
PMID: 33666420
J Nat Prod. 2021 Mar 26;84(3):824-835. doi: 10.1021/acs.jnatprod.0c01376. Epub 2021 Mar 05.

Despite the value of mass spectrometry in modern natural products discovery workflows, it remains very difficult to compare data sets between laboratories. In this study we compared mass spectrometry data for the same sample set from two different laboratories...

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Clark TN, Houriet J, Vidar WS, et al. Interlaboratory Comparison of Untargeted Mass Spectrometry Data Uncovers Underlying Causes for Variability. J Nat Prod. 2021;84(3):824-835doi: 10.1021/acs.jnatprod.0c01376.
Clark, T. N., Houriet, J., Vidar, W. S., Kellogg, J. J., Todd, D. A., Cech, N. B., & Linington, R. G. (2021). Interlaboratory Comparison of Untargeted Mass Spectrometry Data Uncovers Underlying Causes for Variability. Journal of natural products, 84(3), 824-835. https://doi.org/10.1021/acs.jnatprod.0c01376
Clark, Trevor N, et al. "Interlaboratory Comparison of Untargeted Mass Spectrometry Data Uncovers Underlying Causes for Variability." Journal of natural products vol. 84,3 (2021): 824-835. doi: https://doi.org/10.1021/acs.jnatprod.0c01376
Clark TN, Houriet J, Vidar WS, Kellogg JJ, Todd DA, Cech NB, Linington RG. Interlaboratory Comparison of Untargeted Mass Spectrometry Data Uncovers Underlying Causes for Variability. J Nat Prod. 2021 Mar 26;84(3):824-835. doi: 10.1021/acs.jnatprod.0c01376. Epub 2021 Mar 05. PMID: 33666420; PMCID: PMC8326878.
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RNA in biological condensates.

RNA (New York, N.Y.)

Cech TR.
PMID: 34903621
RNA. 2022 Jan;28(1):1-2. doi: 10.1261/rna.079051.121.

No abstract available.

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Cech TR. RNA in biological condensates. RNA. 2022;28(1):1-2doi: 10.1261/rna.079051.121.
Cech, T. R. (2022). RNA in biological condensates. RNA (New York, N.Y.), 28(1), 1-2. https://doi.org/10.1261/rna.079051.121
Cech, Thomas R. "RNA in biological condensates." RNA (New York, N.Y.) vol. 28,1 (2022): 1-2. doi: https://doi.org/10.1261/rna.079051.121
Cech TR. RNA in biological condensates. RNA. 2022 Jan;28(1):1-2. doi: 10.1261/rna.079051.121. PMID: 34903621.
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chinmo-mutant spermatogonial stem cells cause mitotic drive by evicting non-mutant neighbors from the niche.

Developmental cell

Tseng CY, Burel M, Cammer M, Harsh S, Flaherty MS, Baumgartner S, Bach EA.
PMID: 34942115
Dev Cell. 2022 Jan 10;57(1):80-94.e7. doi: 10.1016/j.devcel.2021.12.004. Epub 2021 Dec 22.

Niches maintain a finite pool of stem cells via restricted space and short-range signals. Stem cells compete for limited niche resources, but the mechanisms regulating competition are poorly understood. Using the Drosophila testis model, we show that germline stem...

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Tseng CY, Burel M, Cammer M, et al. chinmo-mutant spermatogonial stem cells cause mitotic drive by evicting non-mutant neighbors from the niche. Dev Cell. 2021;57(1):80-94.e7doi: 10.1016/j.devcel.2021.12.004.
Tseng, C. Y., Burel, M., Cammer, M., Harsh, S., Flaherty, M. S., Baumgartner, S., & Bach, E. A. (2022). chinmo-mutant spermatogonial stem cells cause mitotic drive by evicting non-mutant neighbors from the niche. Developmental cell, 57(1), 80-94.e7. https://doi.org/10.1016/j.devcel.2021.12.004
Tseng, Chen-Yuan, et al. "chinmo-mutant spermatogonial stem cells cause mitotic drive by evicting non-mutant neighbors from the niche." Developmental cell vol. 57,1 (2022): 80-94.e7. doi: https://doi.org/10.1016/j.devcel.2021.12.004
Tseng CY, Burel M, Cammer M, Harsh S, Flaherty MS, Baumgartner S, Bach EA. chinmo-mutant spermatogonial stem cells cause mitotic drive by evicting non-mutant neighbors from the niche. Dev Cell. 2022 Jan 10;57(1):80-94.e7. doi: 10.1016/j.devcel.2021.12.004. Epub 2021 Dec 22. PMID: 34942115; PMCID: PMC8752517.
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chinmo-mutant spermatogonial stem cells cause mitotic drive by evicting non-mutant neighbors from the niche.

Developmental cell

Tseng CY, Burel M, Cammer M, Harsh S, Flaherty MS, Baumgartner S, Bach EA.
PMID: 34942115
Dev Cell. 2022 Jan 10;57(1):80-94.e7. doi: 10.1016/j.devcel.2021.12.004. Epub 2021 Dec 22.

Niches maintain a finite pool of stem cells via restricted space and short-range signals. Stem cells compete for limited niche resources, but the mechanisms regulating competition are poorly understood. Using the Drosophila testis model, we show that germline stem...

Cite
Tseng CY, Burel M, Cammer M, et al. chinmo-mutant spermatogonial stem cells cause mitotic drive by evicting non-mutant neighbors from the niche. Dev Cell. 2021;57(1):80-94.e7doi: 10.1016/j.devcel.2021.12.004.
Tseng, C. Y., Burel, M., Cammer, M., Harsh, S., Flaherty, M. S., Baumgartner, S., & Bach, E. A. (2022). chinmo-mutant spermatogonial stem cells cause mitotic drive by evicting non-mutant neighbors from the niche. Developmental cell, 57(1), 80-94.e7. https://doi.org/10.1016/j.devcel.2021.12.004
Tseng, Chen-Yuan, et al. "chinmo-mutant spermatogonial stem cells cause mitotic drive by evicting non-mutant neighbors from the niche." Developmental cell vol. 57,1 (2022): 80-94.e7. doi: https://doi.org/10.1016/j.devcel.2021.12.004
Tseng CY, Burel M, Cammer M, Harsh S, Flaherty MS, Baumgartner S, Bach EA. chinmo-mutant spermatogonial stem cells cause mitotic drive by evicting non-mutant neighbors from the niche. Dev Cell. 2022 Jan 10;57(1):80-94.e7. doi: 10.1016/j.devcel.2021.12.004. Epub 2021 Dec 22. PMID: 34942115; PMCID: PMC8752517.
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The association between body composition, leptin levels and glucose dysregulation in youth with cystic fibrosis.

Journal of cystic fibrosis : official journal of the European Cystic Fibrosis Society

Granados A, Beach EA, Christiansen AJ, Patterson BW, Wallendorf M, Arbeláez AM.
PMID: 34183284
J Cyst Fibros. 2021 Sep;20(5):796-802. doi: 10.1016/j.jcf.2021.06.004. Epub 2021 Jun 26.

BACKGROUND: Optimization of nutritional status is recommended in patients with cystic fibrosis (CF) given the association between lower body mass index (BMI) and poor clinical outcomes. However, higher BMI and body fat correlate with glucose impairment and higher leptin...

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Granados A, Beach EA, Christiansen AJ, et al. The association between body composition, leptin levels and glucose dysregulation in youth with cystic fibrosis. J Cyst Fibros. 2021;20(5):796-802doi: 10.1016/j.jcf.2021.06.004.
Granados, A., Beach, E. A., Christiansen, A. J., Patterson, B. W., Wallendorf, M., & Arbeláez, A. M. (2021). The association between body composition, leptin levels and glucose dysregulation in youth with cystic fibrosis. Journal of cystic fibrosis : official journal of the European Cystic Fibrosis Society, 20(5), 796-802. https://doi.org/10.1016/j.jcf.2021.06.004
Granados, Andrea, et al. "The association between body composition, leptin levels and glucose dysregulation in youth with cystic fibrosis." Journal of cystic fibrosis : official journal of the European Cystic Fibrosis Society vol. 20,5 (2021): 796-802. doi: https://doi.org/10.1016/j.jcf.2021.06.004
Granados A, Beach EA, Christiansen AJ, Patterson BW, Wallendorf M, Arbeláez AM. The association between body composition, leptin levels and glucose dysregulation in youth with cystic fibrosis. J Cyst Fibros. 2021 Sep;20(5):796-802. doi: 10.1016/j.jcf.2021.06.004. Epub 2021 Jun 26. PMID: 34183284; PMCID: PMC8552309.
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Publisher Correction: Nuclear compartmentalization of TERT mRNA and TUG1 lncRNA is driven by intron retention.

Nature communications

Dumbović G, Braunschweig U, Langner HK, Smallegan M, Biayna J, Hass EP, Jastrzebska K, Blencowe B, Cech TR, Caruthers MH, Rinn JL.
PMID: 34675241
Nat Commun. 2021 Oct 21;12(1):6189. doi: 10.1038/s41467-021-26492-5.

No abstract available.

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Dumbović G, Braunschweig U, Langner HK, et al. Publisher Correction: Nuclear compartmentalization of TERT mRNA and TUG1 lncRNA is driven by intron retention. Nat Commun. 2021;12(1):6189doi: 10.1038/s41467-021-26492-5.
Dumbović, G., Braunschweig, U., Langner, H. K., Smallegan, M., Biayna, J., Hass, E. P., Jastrzebska, K., Blencowe, B., Cech, T. R., Caruthers, M. H., & Rinn, J. L. (2021). Publisher Correction: Nuclear compartmentalization of TERT mRNA and TUG1 lncRNA is driven by intron retention. Nature communications, 12(1), 6189. https://doi.org/10.1038/s41467-021-26492-5
Dumbović, Gabrijela, et al. "Publisher Correction: Nuclear compartmentalization of TERT mRNA and TUG1 lncRNA is driven by intron retention." Nature communications vol. 12,1 (2021): 6189. doi: https://doi.org/10.1038/s41467-021-26492-5
Dumbović G, Braunschweig U, Langner HK, Smallegan M, Biayna J, Hass EP, Jastrzebska K, Blencowe B, Cech TR, Caruthers MH, Rinn JL. Publisher Correction: Nuclear compartmentalization of TERT mRNA and TUG1 lncRNA is driven by intron retention. Nat Commun. 2021 Oct 21;12(1):6189. doi: 10.1038/s41467-021-26492-5. PMID: 34675241; PMCID: PMC8531028.
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Phenotypical and Myopathological Consequences of Compound Heterozygous Missense and Nonsense Variants in .

Cells

Della Marina A, Arlt A, Schara-Schmidt U, Depienne C, Gangfuß A, Kölbel H, Sickmann A, Freier E, Kohlschmidt N, Hentschel A, Weis J, Czech A, Grüneboom A, Roos A.
PMID: 34943989
Cells. 2021 Dec 09;10(12). doi: 10.3390/cells10123481.

BACKGROUND: Presynaptic forms of congenital myasthenic syndromes (CMS) due to pathogenic variants in METHODS: Exome sequencing (ES) was carried out to identify the molecular genetic cause of the disease in a 5-year-old male patient and histological, immunofluorescence as well...

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Della Marina A, Arlt A, Schara-Schmidt U, et al. Phenotypical and Myopathological Consequences of Compound Heterozygous Missense and Nonsense Variants in . Cells. 2021;10(12)doi: 10.3390/cells10123481.
Della Marina, A., Arlt, A., Schara-Schmidt, U., Depienne, C., Gangfuß, A., Kölbel, H., Sickmann, A., Freier, E., Kohlschmidt, N., Hentschel, A., Weis, J., Czech, A., Grüneboom, A., & Roos, A. (2021). Phenotypical and Myopathological Consequences of Compound Heterozygous Missense and Nonsense Variants in . Cells, 10(12), . https://doi.org/10.3390/cells10123481
Della Marina, Adela, et al. "Phenotypical and Myopathological Consequences of Compound Heterozygous Missense and Nonsense Variants in ." Cells vol. 10,12 (2021). doi: https://doi.org/10.3390/cells10123481
Della Marina A, Arlt A, Schara-Schmidt U, Depienne C, Gangfuß A, Kölbel H, Sickmann A, Freier E, Kohlschmidt N, Hentschel A, Weis J, Czech A, Grüneboom A, Roos A. Phenotypical and Myopathological Consequences of Compound Heterozygous Missense and Nonsense Variants in . Cells. 2021 Dec 09;10(12). doi: 10.3390/cells10123481. PMID: 34943989.
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Showing 25 to 36 of 177 entries