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Showing 133 to 141 of 141 entries
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White matter microstructure associations to amyloid burden in adults with Down syndrome.

NeuroImage. Clinical

Bazydlo AM, Zammit MD, Wu M, Lao PJ, Dean DC, Johnson SC, Tudorascu DL, Cohen A, Cody KA, Ances B, Laymon CM, Klunk WE, Zaman S, Handen BL, Hartley SL, Alexander AL, Christian BT.
PMID: 34902714
Neuroimage Clin. 2021 Dec 10;33:102908. doi: 10.1016/j.nicl.2021.102908. Epub 2021 Dec 10.

INTRODUCTION: Individuals with Down syndrome (DS) are at an increased risk of developing Alzheimer's Disease (AD). One of the early underlying mechanisms in AD pathology is the accumulation of amyloid protein plaques, which are deposited in extracellular gray matter...

Cite
Bazydlo AM, Zammit MD, Wu M, et al. White matter microstructure associations to amyloid burden in adults with Down syndrome. Neuroimage Clin. 2021;33:102908doi: 10.1016/j.nicl.2021.102908.
Bazydlo, A. M., Zammit, M. D., Wu, M., Lao, P. J., Dean, D. C., Johnson, S. C., Tudorascu, D. L., Cohen, A., Cody, K. A., Ances, B., Laymon, C. M., Klunk, W. E., Zaman, S., Handen, B. L., Hartley, S. L., Alexander, A. L., & Christian, B. T. (2021). White matter microstructure associations to amyloid burden in adults with Down syndrome. NeuroImage. Clinical, 33102908. https://doi.org/10.1016/j.nicl.2021.102908
Bazydlo, Austin M, et al. "White matter microstructure associations to amyloid burden in adults with Down syndrome." NeuroImage. Clinical vol. 33 (2021): 102908. doi: https://doi.org/10.1016/j.nicl.2021.102908
Bazydlo AM, Zammit MD, Wu M, Lao PJ, Dean DC, Johnson SC, Tudorascu DL, Cohen A, Cody KA, Ances B, Laymon CM, Klunk WE, Zaman S, Handen BL, Hartley SL, Alexander AL, Christian BT. White matter microstructure associations to amyloid burden in adults with Down syndrome. Neuroimage Clin. 2021 Dec 10;33:102908. doi: 10.1016/j.nicl.2021.102908. Epub 2021 Dec 10. PMID: 34902714; PMCID: PMC8672096.
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White matter microstructure associations to amyloid burden in adults with Down syndrome.

NeuroImage. Clinical

Bazydlo AM, Zammit MD, Wu M, Lao PJ, Dean DC, Johnson SC, Tudorascu DL, Cohen A, Cody KA, Ances B, Laymon CM, Klunk WE, Zaman S, Handen BL, Hartley SL, Alexander AL, Christian BT.
PMID: 34902714
Neuroimage Clin. 2021 Dec 10;33:102908. doi: 10.1016/j.nicl.2021.102908. Epub 2021 Dec 10.

INTRODUCTION: Individuals with Down syndrome (DS) are at an increased risk of developing Alzheimer's Disease (AD). One of the early underlying mechanisms in AD pathology is the accumulation of amyloid protein plaques, which are deposited in extracellular gray matter...

Cite
Bazydlo AM, Zammit MD, Wu M, et al. White matter microstructure associations to amyloid burden in adults with Down syndrome. Neuroimage Clin. 2021;33:102908doi: 10.1016/j.nicl.2021.102908.
Bazydlo, A. M., Zammit, M. D., Wu, M., Lao, P. J., Dean, D. C., Johnson, S. C., Tudorascu, D. L., Cohen, A., Cody, K. A., Ances, B., Laymon, C. M., Klunk, W. E., Zaman, S., Handen, B. L., Hartley, S. L., Alexander, A. L., & Christian, B. T. (2021). White matter microstructure associations to amyloid burden in adults with Down syndrome. NeuroImage. Clinical, 33102908. https://doi.org/10.1016/j.nicl.2021.102908
Bazydlo, Austin M, et al. "White matter microstructure associations to amyloid burden in adults with Down syndrome." NeuroImage. Clinical vol. 33 (2021): 102908. doi: https://doi.org/10.1016/j.nicl.2021.102908
Bazydlo AM, Zammit MD, Wu M, Lao PJ, Dean DC, Johnson SC, Tudorascu DL, Cohen A, Cody KA, Ances B, Laymon CM, Klunk WE, Zaman S, Handen BL, Hartley SL, Alexander AL, Christian BT. White matter microstructure associations to amyloid burden in adults with Down syndrome. Neuroimage Clin. 2021 Dec 10;33:102908. doi: 10.1016/j.nicl.2021.102908. Epub 2021 Dec 10. PMID: 34902714; PMCID: PMC8672096.
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Recrystallization inhibition in ice due to ice binding protein activity detected by nuclear magnetic resonance.

Biotechnology reports (Amsterdam, Netherlands)

Brown JR, Seymour JD, Brox TI, Skidmore ML, Wang C, Christner BC, Luo BH, Codd SL.
PMID: 28626650
Biotechnol Rep (Amst). 2014 Jun 20;3:60-64. doi: 10.1016/j.btre.2014.06.005. eCollection 2014 Sep.

Liquid water present in polycrystalline ice at the interstices between ice crystals results in a network of liquid-filled veins and nodes within a solid ice matrix, making ice a low porosity porous media. Here we used nuclear magnetic resonance...

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Brown JR, Seymour JD, Brox TI, et al. Recrystallization inhibition in ice due to ice binding protein activity detected by nuclear magnetic resonance. Biotechnol Rep (Amst). 2014;3:60-64doi: 10.1016/j.btre.2014.06.005.
Brown, J. R., Seymour, J. D., Brox, T. I., Skidmore, M. L., Wang, C., Christner, B. C., Luo, B. H., & Codd, S. L. (2014). Recrystallization inhibition in ice due to ice binding protein activity detected by nuclear magnetic resonance. Biotechnology reports (Amsterdam, Netherlands), 360-64. https://doi.org/10.1016/j.btre.2014.06.005
Brown, Jennifer R, et al. "Recrystallization inhibition in ice due to ice binding protein activity detected by nuclear magnetic resonance." Biotechnology reports (Amsterdam, Netherlands) vol. 3 (2014): 60-64. doi: https://doi.org/10.1016/j.btre.2014.06.005
Brown JR, Seymour JD, Brox TI, Skidmore ML, Wang C, Christner BC, Luo BH, Codd SL. Recrystallization inhibition in ice due to ice binding protein activity detected by nuclear magnetic resonance. Biotechnol Rep (Amst). 2014 Jun 20;3:60-64. doi: 10.1016/j.btre.2014.06.005. eCollection 2014 Sep. PMID: 28626650; PMCID: PMC5466106.
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Tuft cell-produced cysteinyl leukotrienes and IL-25 synergistically initiate lung type 2 inflammation.

Science immunology

Ualiyeva S, Lemire E, Aviles EC, Wong C, Boyd AA, Lai J, Liu T, Matsumoto I, Barrett NA, Boyce JA, Haber AL, Bankova LG.
PMID: 34932383
Sci Immunol. 2021 Dec 24;6(66):eabj0474. doi: 10.1126/sciimmunol.abj0474. Epub 2021 Dec 24.

[Figure: see text].

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Ualiyeva S, Lemire E, Aviles EC, et al. Tuft cell-produced cysteinyl leukotrienes and IL-25 synergistically initiate lung type 2 inflammation. Sci Immunol. 2021;6(66):eabj0474doi: 10.1126/sciimmunol.abj0474.
Ualiyeva, S., Lemire, E., Aviles, E. C., Wong, C., Boyd, A. A., Lai, J., Liu, T., Matsumoto, I., Barrett, N. A., Boyce, J. A., Haber, A. L., & Bankova, L. G. (2021). Tuft cell-produced cysteinyl leukotrienes and IL-25 synergistically initiate lung type 2 inflammation. Science immunology, 6(66), eabj0474. https://doi.org/10.1126/sciimmunol.abj0474
Ualiyeva, Saltanat, et al. "Tuft cell-produced cysteinyl leukotrienes and IL-25 synergistically initiate lung type 2 inflammation." Science immunology vol. 6,66 (2021): eabj0474. doi: https://doi.org/10.1126/sciimmunol.abj0474
Ualiyeva S, Lemire E, Aviles EC, Wong C, Boyd AA, Lai J, Liu T, Matsumoto I, Barrett NA, Boyce JA, Haber AL, Bankova LG. Tuft cell-produced cysteinyl leukotrienes and IL-25 synergistically initiate lung type 2 inflammation. Sci Immunol. 2021 Dec 24;6(66):eabj0474. doi: 10.1126/sciimmunol.abj0474. Epub 2021 Dec 24. PMID: 34932383.
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White matter microstructure associations to amyloid burden in adults with Down syndrome.

NeuroImage. Clinical

Bazydlo AM, Zammit MD, Wu M, Lao PJ, Dean DC, Johnson SC, Tudorascu DL, Cohen A, Cody KA, Ances B, Laymon CM, Klunk WE, Zaman S, Handen BL, Hartley SL, Alexander AL, Christian BT.
PMID: 34902714
Neuroimage Clin. 2021 Dec 10;33:102908. doi: 10.1016/j.nicl.2021.102908. Epub 2021 Dec 10.

INTRODUCTION: Individuals with Down syndrome (DS) are at an increased risk of developing Alzheimer's Disease (AD). One of the early underlying mechanisms in AD pathology is the accumulation of amyloid protein plaques, which are deposited in extracellular gray matter...

Cite
Bazydlo AM, Zammit MD, Wu M, et al. White matter microstructure associations to amyloid burden in adults with Down syndrome. Neuroimage Clin. 2021;33:102908doi: 10.1016/j.nicl.2021.102908.
Bazydlo, A. M., Zammit, M. D., Wu, M., Lao, P. J., Dean, D. C., Johnson, S. C., Tudorascu, D. L., Cohen, A., Cody, K. A., Ances, B., Laymon, C. M., Klunk, W. E., Zaman, S., Handen, B. L., Hartley, S. L., Alexander, A. L., & Christian, B. T. (2021). White matter microstructure associations to amyloid burden in adults with Down syndrome. NeuroImage. Clinical, 33102908. https://doi.org/10.1016/j.nicl.2021.102908
Bazydlo, Austin M, et al. "White matter microstructure associations to amyloid burden in adults with Down syndrome." NeuroImage. Clinical vol. 33 (2021): 102908. doi: https://doi.org/10.1016/j.nicl.2021.102908
Bazydlo AM, Zammit MD, Wu M, Lao PJ, Dean DC, Johnson SC, Tudorascu DL, Cohen A, Cody KA, Ances B, Laymon CM, Klunk WE, Zaman S, Handen BL, Hartley SL, Alexander AL, Christian BT. White matter microstructure associations to amyloid burden in adults with Down syndrome. Neuroimage Clin. 2021 Dec 10;33:102908. doi: 10.1016/j.nicl.2021.102908. Epub 2021 Dec 10. PMID: 34902714; PMCID: PMC8672096.
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Creation of an Automated Fluorescence Guided Tumor Ablation System.

IEEE journal of translational engineering in health and medicine

Tucker M, Ma G, Ross W, Buckland DM, Codd PJ.
PMID: 34765325
IEEE J Transl Eng Health Med. 2021 Jul 21;9:4300109. doi: 10.1109/JTEHM.2021.3097210. eCollection 2021.

OBJECTIVE: Create a device that improves the identification and extent of resection at the interface between healthy and tumor tissue; ultimately, using this device would improve surgical outcomes for patients and increase survival.METHODS: We have created a contactless tumor...

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Tucker M, Ma G, Ross W, et al. Creation of an Automated Fluorescence Guided Tumor Ablation System. IEEE J Transl Eng Health Med. 2021;9:4300109doi: 10.1109/JTEHM.2021.3097210.
Tucker, M., Ma, G., Ross, W., Buckland, D. M., & Codd, P. J. (2021). Creation of an Automated Fluorescence Guided Tumor Ablation System. IEEE journal of translational engineering in health and medicine, 94300109. https://doi.org/10.1109/JTEHM.2021.3097210
Tucker, Matthew, et al. "Creation of an Automated Fluorescence Guided Tumor Ablation System." IEEE journal of translational engineering in health and medicine vol. 9 (2021): 4300109. doi: https://doi.org/10.1109/JTEHM.2021.3097210
Tucker M, Ma G, Ross W, Buckland DM, Codd PJ. Creation of an Automated Fluorescence Guided Tumor Ablation System. IEEE J Transl Eng Health Med. 2021 Jul 21;9:4300109. doi: 10.1109/JTEHM.2021.3097210. eCollection 2021. PMID: 34765325; PMCID: PMC8577571.
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T cell phenotypes in COVID-19 - a living review.

Oxford open immunology

Hanna SJ, Codd AS, Gea-Mallorqui E, Scourfield DO, Richter FC, Ladell K, Borsa M, Compeer EB, Moon OR, Galloway SAE, Dimonte S, Capitani L, Shepherd FR, Wilson JD, Uhl LFK, Gallimore AM, Milicic A.
PMID: 33575657
Oxf Open Immunol. 2020 Dec 29;2(1):iqaa007. doi: 10.1093/oxfimm/iqaa007. eCollection 2021.

COVID-19 is characterized by profound lymphopenia in the peripheral blood, and the remaining T cells display altered phenotypes, characterized by a spectrum of activation and exhaustion. However, antigen-specific T cell responses are emerging as a crucial mechanism for both...

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Hanna SJ, Codd AS, Gea-Mallorqui E, et al. T cell phenotypes in COVID-19 - a living review. Oxf Open Immunol. 2020;2(1):iqaa007doi: 10.1093/oxfimm/iqaa007.
Hanna, S. J., Codd, A. S., Gea-Mallorqui, E., Scourfield, D. O., Richter, F. C., Ladell, K., Borsa, M., Compeer, E. B., Moon, O. R., Galloway, S. A. E., Dimonte, S., Capitani, L., Shepherd, F. R., Wilson, J. D., Uhl, L. F. K., Gallimore, A. M., & Milicic, A. (2021). T cell phenotypes in COVID-19 - a living review. Oxford open immunology, 2(1), iqaa007. https://doi.org/10.1093/oxfimm/iqaa007
Hanna, Stephanie J, et al. "T cell phenotypes in COVID-19 - a living review." Oxford open immunology vol. 2,1 (2021): iqaa007. doi: https://doi.org/10.1093/oxfimm/iqaa007
Hanna SJ, Codd AS, Gea-Mallorqui E, Scourfield DO, Richter FC, Ladell K, Borsa M, Compeer EB, Moon OR, Galloway SAE, Dimonte S, Capitani L, Shepherd FR, Wilson JD, Uhl LFK, Gallimore AM, Milicic A. T cell phenotypes in COVID-19 - a living review. Oxf Open Immunol. 2020 Dec 29;2(1):iqaa007. doi: 10.1093/oxfimm/iqaa007. eCollection 2021. PMID: 33575657; PMCID: PMC7798577.
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Cerebrovascular stiffness and flow dynamics in the presence of amyloid and tau biomarkers.

Alzheimer's & dementia (Amsterdam, Netherlands)

Rivera-Rivera LA, Eisenmenger L, Cody KA, Reher T, Betthauser T, Cadman RV, Rowley HA, Carlsson CM, Chin NA, Johnson SC, Johnson KM.
PMID: 35005194
Alzheimers Dement (Amst). 2021 Dec 31;13(1):e12253. doi: 10.1002/dad2.12253. eCollection 2021.

INTRODUCTION: This work investigated the relationship between cerebrovascular disease (CVD) markers and Alzheimer's disease (AD) biomarkers of amyloid beta deposition, and neurofibrillary tau tangles in subjects spanning the AD clinical spectrum.METHODS: A total of 136 subjects participated in this...

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Rivera-Rivera LA, Eisenmenger L, Cody KA, et al. Cerebrovascular stiffness and flow dynamics in the presence of amyloid and tau biomarkers. Alzheimers Dement (Amst). 2021;13(1):e12253doi: 10.1002/dad2.12253.
Rivera-Rivera, L. A., Eisenmenger, L., Cody, K. A., Reher, T., Betthauser, T., Cadman, R. V., Rowley, H. A., Carlsson, C. M., Chin, N. A., Johnson, S. C., & Johnson, K. M. (2021). Cerebrovascular stiffness and flow dynamics in the presence of amyloid and tau biomarkers. Alzheimer's & dementia (Amsterdam, Netherlands), 13(1), e12253. https://doi.org/10.1002/dad2.12253
Rivera-Rivera, Leonardo A, et al. "Cerebrovascular stiffness and flow dynamics in the presence of amyloid and tau biomarkers." Alzheimer's & dementia (Amsterdam, Netherlands) vol. 13,1 (2021): e12253. doi: https://doi.org/10.1002/dad2.12253
Rivera-Rivera LA, Eisenmenger L, Cody KA, Reher T, Betthauser T, Cadman RV, Rowley HA, Carlsson CM, Chin NA, Johnson SC, Johnson KM. Cerebrovascular stiffness and flow dynamics in the presence of amyloid and tau biomarkers. Alzheimers Dement (Amst). 2021 Dec 31;13(1):e12253. doi: 10.1002/dad2.12253. eCollection 2021. PMID: 35005194; PMCID: PMC8719432.
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Population pharmacokinetic-pharmacodynamic model of oxfendazole in healthy adults in a multiple ascending dose and food effect study and target attainment analysis.

Antimicrobial agents and chemotherapy

Bach T, Deye GA, Codd EE, Horton J, Winokur P, An G.
PMID: 34606333
Antimicrob Agents Chemother. 2021 Oct 04;AAC0143221. doi: 10.1128/AAC.01432-21. Epub 2021 Oct 04.

Oxfendazole is a potent veterinary antiparasitic drug undergoing development for human use to treat multiple parasitic infections. Results from two recently completed Phase I clinical trials conducted in healthy adults showed that the pharmacokinetics of oxfendazole is nonlinear, affected...

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Bach T, Deye GA, Codd EE, et al. Population pharmacokinetic-pharmacodynamic model of oxfendazole in healthy adults in a multiple ascending dose and food effect study and target attainment analysis. Antimicrob Agents Chemother. 2021;AAC0143221doi: 10.1128/AAC.01432-21.
Bach, T., Deye, G. A., Codd, E. E., Horton, J., Winokur, P., & An, G. (2021). Population pharmacokinetic-pharmacodynamic model of oxfendazole in healthy adults in a multiple ascending dose and food effect study and target attainment analysis. Antimicrobial agents and chemotherapy, AAC0143221. https://doi.org/10.1128/AAC.01432-21
Bach, Thanh, et al. "Population pharmacokinetic-pharmacodynamic model of oxfendazole in healthy adults in a multiple ascending dose and food effect study and target attainment analysis." Antimicrobial agents and chemotherapy vol. (2021): AAC0143221. doi: https://doi.org/10.1128/AAC.01432-21
Bach T, Deye GA, Codd EE, Horton J, Winokur P, An G. Population pharmacokinetic-pharmacodynamic model of oxfendazole in healthy adults in a multiple ascending dose and food effect study and target attainment analysis. Antimicrob Agents Chemother. 2021 Oct 04;AAC0143221. doi: 10.1128/AAC.01432-21. Epub 2021 Oct 04. PMID: 34606333.
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Showing 133 to 141 of 141 entries