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Crystalline free energies of micelles of diblock copolymer solutions.

The Journal of chemical physics

D'Adamo G, Pierleoni C.
PMID: 21133452
J Chem Phys. 2010 Nov 28;133(20):204902. doi: 10.1063/1.3509391.

We report a characterization of the relative stability and structural behavior of various micellar crystals of an athermal model of AB-diblock copolymers in solution. We adopt a previously developed coarse-graining representation of the chains which maps each copolymer on...

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D'Adamo G, Pierleoni C. Crystalline free energies of micelles of diblock copolymer solutions. J Chem Phys. 2010;133(20):204902doi: 10.1063/1.3509391.
D'Adamo, G., & Pierleoni, C. (2010). Crystalline free energies of micelles of diblock copolymer solutions. The Journal of chemical physics, 133(20), 204902. https://doi.org/10.1063/1.3509391
D'Adamo, Giuseppe, and Pierleoni, Carlo. "Crystalline free energies of micelles of diblock copolymer solutions." The Journal of chemical physics vol. 133,20 (2010): 204902. doi: https://doi.org/10.1063/1.3509391
D'Adamo G, Pierleoni C. Crystalline free energies of micelles of diblock copolymer solutions. J Chem Phys. 2010 Nov 28;133(20):204902. doi: 10.1063/1.3509391. PMID: 21133452.
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The interactome of the amyloid beta precursor protein family members is shaped by phosphorylation of their intracellular domains.

Molecular neurodegeneration

Tamayev R, Zhou D, D'Adamio L.
PMID: 19602287
Mol Neurodegener. 2009 Jul 14;4:28. doi: 10.1186/1750-1326-4-28.

BACKGROUND: Brain tissue from patients with Alzheimer's disease has shown an increase of phosphorylation of Tyr-682, located on the conserved Y682ENPTY motif, and Thr-668 residues, both in the intracellular domain (AID) of amyloid beta precursor protein (APP), although the...

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Tamayev R, Zhou D, D'Adamio L. The interactome of the amyloid beta precursor protein family members is shaped by phosphorylation of their intracellular domains. Mol Neurodegener. 2009;4:28doi: 10.1186/1750-1326-4-28.
Tamayev, R., Zhou, D., & D'Adamio, L. (2009). The interactome of the amyloid beta precursor protein family members is shaped by phosphorylation of their intracellular domains. Molecular neurodegeneration, 428. https://doi.org/10.1186/1750-1326-4-28
Tamayev, Robert, et al. "The interactome of the amyloid beta precursor protein family members is shaped by phosphorylation of their intracellular domains." Molecular neurodegeneration vol. 4 (2009): 28. doi: https://doi.org/10.1186/1750-1326-4-28
Tamayev R, Zhou D, D'Adamio L. The interactome of the amyloid beta precursor protein family members is shaped by phosphorylation of their intracellular domains. Mol Neurodegener. 2009 Jul 14;4:28. doi: 10.1186/1750-1326-4-28. PMID: 19602287; PMCID: PMC2723102.
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Adherence to an Exercise Intervention Among Older Women Post Hip Fracture.

Journal of clinical sport psychology

Resnick B, D'Adamo C, Shardell M, Orwig D, Hawkes W, Hebel JR, Golden J, Magaziner J, Zimmerman S, Yu-Yahiro J.
PMID: 20428489
J Clin Sport Psychol. 2008 Mar;2(1):41-56. doi: 10.1123/jcsp.2.1.41.

The purpose of this study was to evaluate adherence to home-based exercise interventions among older women post hip fracture that were randomized to one of three exercise intervention groups or a routine care group. A total of 157 female...

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Resnick B, D'Adamo C, Shardell M, et al. Adherence to an Exercise Intervention Among Older Women Post Hip Fracture. J Clin Sport Psychol. 2008;2(1):41-56doi: 10.1123/jcsp.2.1.41.
Resnick, B., D'Adamo, C., Shardell, M., Orwig, D., Hawkes, W., Hebel, J. R., Golden, J., Magaziner, J., Zimmerman, S., & Yu-Yahiro, J. (2008). Adherence to an Exercise Intervention Among Older Women Post Hip Fracture. Journal of clinical sport psychology, 2(1), 41-56. https://doi.org/10.1123/jcsp.2.1.41
Resnick, Barbara, et al. "Adherence to an Exercise Intervention Among Older Women Post Hip Fracture." Journal of clinical sport psychology vol. 2,1 (2008): 41-56. doi: https://doi.org/10.1123/jcsp.2.1.41
Resnick B, D'Adamo C, Shardell M, Orwig D, Hawkes W, Hebel JR, Golden J, Magaziner J, Zimmerman S, Yu-Yahiro J. Adherence to an Exercise Intervention Among Older Women Post Hip Fracture. J Clin Sport Psychol. 2008 Mar;2(1):41-56. doi: 10.1123/jcsp.2.1.41. PMID: 20428489; PMCID: PMC2859720.
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Mapping populations at risk: improving spatial demographic data for infectious disease modeling and metric derivation.

Population health metrics

Tatem AJ, Adamo S, Bharti N, Burgert CR, Castro M, Dorelien A, Fink G, Linard C, John M, Montana L, Montgomery MR, Nelson A, Noor AM, Pindolia D, Yetman G, Balk D.
PMID: 22591595
Popul Health Metr. 2012 May 16;10(1):8. doi: 10.1186/1478-7954-10-8.

The use of Global Positioning Systems (GPS) and Geographical Information Systems (GIS) in disease surveys and reporting is becoming increasingly routine, enabling a better understanding of spatial epidemiology and the improvement of surveillance and control strategies. In turn, the...

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Tatem AJ, Adamo S, Bharti N, et al. Mapping populations at risk: improving spatial demographic data for infectious disease modeling and metric derivation. Popul Health Metr. 2012;10(1):8doi: 10.1186/1478-7954-10-8.
Tatem, A. J., Adamo, S., Bharti, N., Burgert, C. R., Castro, M., Dorelien, A., Fink, G., Linard, C., John, M., Montana, L., Montgomery, M. R., Nelson, A., Noor, A. M., Pindolia, D., Yetman, G., & Balk, D. (2012). Mapping populations at risk: improving spatial demographic data for infectious disease modeling and metric derivation. Population health metrics, 10(1), 8. https://doi.org/10.1186/1478-7954-10-8
Tatem, Andrew J, et al. "Mapping populations at risk: improving spatial demographic data for infectious disease modeling and metric derivation." Population health metrics vol. 10,1 (2012): 8. doi: https://doi.org/10.1186/1478-7954-10-8
Tatem AJ, Adamo S, Bharti N, Burgert CR, Castro M, Dorelien A, Fink G, Linard C, John M, Montana L, Montgomery MR, Nelson A, Noor AM, Pindolia D, Yetman G, Balk D. Mapping populations at risk: improving spatial demographic data for infectious disease modeling and metric derivation. Popul Health Metr. 2012 May 16;10(1):8. doi: 10.1186/1478-7954-10-8. PMID: 22591595; PMCID: PMC3487779.
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Polymers as compressible soft spheres.

The Journal of chemical physics

D'Adamo G, Pelissetto A, Pierleoni C.
PMID: 22713071
J Chem Phys. 2012 Jun 14;136(22):224905. doi: 10.1063/1.4728338.

We consider a coarse-grained model in which polymers under good-solvent conditions are represented by soft spheres whose radii, which should be identified with the polymer radii of gyrations, are allowed to fluctuate. The corresponding pair potential depends on the...

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D'Adamo G, Pelissetto A, Pierleoni C. Polymers as compressible soft spheres. J Chem Phys. 2012;136(22):224905doi: 10.1063/1.4728338.
D'Adamo, G., Pelissetto, A., & Pierleoni, C. (2012). Polymers as compressible soft spheres. The Journal of chemical physics, 136(22), 224905. https://doi.org/10.1063/1.4728338
D'Adamo, Giuseppe, et al. "Polymers as compressible soft spheres." The Journal of chemical physics vol. 136,22 (2012): 224905. doi: https://doi.org/10.1063/1.4728338
D'Adamo G, Pelissetto A, Pierleoni C. Polymers as compressible soft spheres. J Chem Phys. 2012 Jun 14;136(22):224905. doi: 10.1063/1.4728338. PMID: 22713071.
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Skeletal Muscle: A Significant Novel Neurohypophyseal Hormone-Secreting Organ.

Frontiers in physiology

Adamo S, Pigna E, Lugarà R, Moresi V, Coletti D, Bouché M.
PMID: 30670984
Front Physiol. 2019 Jan 08;9:1885. doi: 10.3389/fphys.2018.01885. eCollection 2018.

No abstract available.

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Adamo S, Pigna E, Lugarà R, et al. Skeletal Muscle: A Significant Novel Neurohypophyseal Hormone-Secreting Organ. Front Physiol. 2019;9:1885doi: 10.3389/fphys.2018.01885.
Adamo, S., Pigna, E., Lugarà, R., Moresi, V., Coletti, D., & Bouché, M. (2018). Skeletal Muscle: A Significant Novel Neurohypophyseal Hormone-Secreting Organ. Frontiers in physiology, 91885. https://doi.org/10.3389/fphys.2018.01885
Adamo, Sergio, et al. "Skeletal Muscle: A Significant Novel Neurohypophyseal Hormone-Secreting Organ." Frontiers in physiology vol. 9 (2018): 1885. doi: https://doi.org/10.3389/fphys.2018.01885
Adamo S, Pigna E, Lugarà R, Moresi V, Coletti D, Bouché M. Skeletal Muscle: A Significant Novel Neurohypophyseal Hormone-Secreting Organ. Front Physiol. 2019 Jan 08;9:1885. doi: 10.3389/fphys.2018.01885. eCollection 2018. PMID: 30670984; PMCID: PMC6331439.
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HDAC4 Regulates Skeletal Muscle Regeneration via Soluble Factors.

Frontiers in physiology

Renzini A, Marroncelli N, Noviello C, Moresi V, Adamo S.
PMID: 30319457
Front Physiol. 2018 Sep 27;9:1387. doi: 10.3389/fphys.2018.01387. eCollection 2018.

Skeletal muscle possesses a high ability to regenerate after an insult or in pathological conditions, relying on satellite cells, the skeletal muscle stem cells. Satellite cell behavior is tightly regulated by the surrounding microenvironment, which provides multiple signals derived...

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Renzini A, Marroncelli N, Noviello C, et al. HDAC4 Regulates Skeletal Muscle Regeneration via Soluble Factors. Front Physiol. 2018;9:1387doi: 10.3389/fphys.2018.01387.
Renzini, A., Marroncelli, N., Noviello, C., Moresi, V., & Adamo, S. (2018). HDAC4 Regulates Skeletal Muscle Regeneration via Soluble Factors. Frontiers in physiology, 91387. https://doi.org/10.3389/fphys.2018.01387
Renzini, Alessandra, et al. "HDAC4 Regulates Skeletal Muscle Regeneration via Soluble Factors." Frontiers in physiology vol. 9 (2018): 1387. doi: https://doi.org/10.3389/fphys.2018.01387
Renzini A, Marroncelli N, Noviello C, Moresi V, Adamo S. HDAC4 Regulates Skeletal Muscle Regeneration via Soluble Factors. Front Physiol. 2018 Sep 27;9:1387. doi: 10.3389/fphys.2018.01387. eCollection 2018. PMID: 30319457; PMCID: PMC6171007.
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Jumping Translocations of 1q in Myelodysplastic Syndrome and Acute Myeloid Leukemia: Report of Three Cases and Review of Literature.

Case reports in genetics

Couture T, Amato K, DiAdamo A, Li P.
PMID: 30271640
Case Rep Genet. 2018 Sep 09;2018:8296478. doi: 10.1155/2018/8296478. eCollection 2018.

Jumping translocations of 1q refer to the break-off of chromosome 1q as a donor fusing to two or more recipient chromosomes. We detected jumping translocations of 1q in three patients with initial diagnosis of myelodysplastic syndrome (MDS) and later...

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Couture T, Amato K, DiAdamo A, et al. Jumping Translocations of 1q in Myelodysplastic Syndrome and Acute Myeloid Leukemia: Report of Three Cases and Review of Literature. Case Rep Genet. 2018;2018:8296478doi: 10.1155/2018/8296478.
Couture, T., Amato, K., DiAdamo, A., & Li, P. (2018). Jumping Translocations of 1q in Myelodysplastic Syndrome and Acute Myeloid Leukemia: Report of Three Cases and Review of Literature. Case reports in genetics, 20188296478. https://doi.org/10.1155/2018/8296478
Couture, T, et al. "Jumping Translocations of 1q in Myelodysplastic Syndrome and Acute Myeloid Leukemia: Report of Three Cases and Review of Literature." Case reports in genetics vol. 2018 (2018): 8296478. doi: https://doi.org/10.1155/2018/8296478
Couture T, Amato K, DiAdamo A, Li P. Jumping Translocations of 1q in Myelodysplastic Syndrome and Acute Myeloid Leukemia: Report of Three Cases and Review of Literature. Case Rep Genet. 2018 Sep 09;2018:8296478. doi: 10.1155/2018/8296478. eCollection 2018. PMID: 30271640; PMCID: PMC6151220.
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Increasing autophagy does not affect neurogenic muscle atrophy.

European journal of translational myology

Pigna E, Sanna K, Coletti D, Li Z, Parlakian A, Adamo S, Moresi V.
PMID: 30344980
Eur J Transl Myol. 2018 Aug 23;28(3):7687. doi: 10.4081/ejtm.2018.7687. eCollection 2018 Jul 10.

Physiological autophagy plays a crucial role in the regulation of muscle mass and metabolism, while the excessive induction or the inhibition of the autophagic flux contributes to the progression of several diseases. Autophagy can be activated by different stimuli,...

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Pigna E, Sanna K, Coletti D, et al. Increasing autophagy does not affect neurogenic muscle atrophy. Eur J Transl Myol. 2018;28(3):7687doi: 10.4081/ejtm.2018.7687.
Pigna, E., Sanna, K., Coletti, D., Li, Z., Parlakian, A., Adamo, S., & Moresi, V. (2018). Increasing autophagy does not affect neurogenic muscle atrophy. European journal of translational myology, 28(3), 7687. https://doi.org/10.4081/ejtm.2018.7687
Pigna, Eva, et al. "Increasing autophagy does not affect neurogenic muscle atrophy." European journal of translational myology vol. 28,3 (2018): 7687. doi: https://doi.org/10.4081/ejtm.2018.7687
Pigna E, Sanna K, Coletti D, Li Z, Parlakian A, Adamo S, Moresi V. Increasing autophagy does not affect neurogenic muscle atrophy. Eur J Transl Myol. 2018 Aug 23;28(3):7687. doi: 10.4081/ejtm.2018.7687. eCollection 2018 Jul 10. PMID: 30344980; PMCID: PMC6176397.
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Analytical validation and chromosomal distribution of regions of homozygosity by oligonucleotide array comparative genomic hybridization from normal prenatal and postnatal case series.

Molecular cytogenetics

Wen J, Comerford K, Xu Z, Wu W, Amato K, Grommisch B, DiAdamo A, Xu F, Chai H, Li P.
PMID: 30886647
Mol Cytogenet. 2019 Mar 06;12:12. doi: 10.1186/s13039-019-0424-6. eCollection 2019.

BACKGROUND: Regions of homozygosity (ROH) are continuous homozygous segments commonly seen in the human genome. The integration of single nucleotide polymorphism (SNP) probes into current array comparative genomic hybridization (aCGH) analysis has enabled the detection of the ROH. However,...

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Wen J, Comerford K, Xu Z, et al. Analytical validation and chromosomal distribution of regions of homozygosity by oligonucleotide array comparative genomic hybridization from normal prenatal and postnatal case series. Mol Cytogenet. 2019;12:12doi: 10.1186/s13039-019-0424-6.
Wen, J., Comerford, K., Xu, Z., Wu, W., Amato, K., Grommisch, B., DiAdamo, A., Xu, F., Chai, H., & Li, P. (2019). Analytical validation and chromosomal distribution of regions of homozygosity by oligonucleotide array comparative genomic hybridization from normal prenatal and postnatal case series. Molecular cytogenetics, 1212. https://doi.org/10.1186/s13039-019-0424-6
Wen, Jiadi, et al. "Analytical validation and chromosomal distribution of regions of homozygosity by oligonucleotide array comparative genomic hybridization from normal prenatal and postnatal case series." Molecular cytogenetics vol. 12 (2019): 12. doi: https://doi.org/10.1186/s13039-019-0424-6
Wen J, Comerford K, Xu Z, Wu W, Amato K, Grommisch B, DiAdamo A, Xu F, Chai H, Li P. Analytical validation and chromosomal distribution of regions of homozygosity by oligonucleotide array comparative genomic hybridization from normal prenatal and postnatal case series. Mol Cytogenet. 2019 Mar 06;12:12. doi: 10.1186/s13039-019-0424-6. eCollection 2019. PMID: 30886647; PMCID: PMC6404290.
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Soft tissue adhesion patterns over Trevira tube on modular endoprosthesis for malignant bone tumours: an in vitro study.

Journal of biological regulators and homeostatic agents

D’Adamio S, Cazzato G, Ziranu A, Sgambato A, Rosa MA, Maccauro G.
PMID: 29181955
J Biol Regul Homeost Agents. 2017 Oct-Dec,;31(4):37-42.

A reliable and effective technique in case of limb salvage surgery after resection of extensive bone tumors is represented by the implant of modular or custom-made megaprosthesis. Fixation of the residual surrounding soft tissue on the implant represent a...

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D’Adamio S, Cazzato G, Ziranu A, et al. Soft tissue adhesion patterns over Trevira tube on modular endoprosthesis for malignant bone tumours: an in vitro study. J Biol Regul Homeost Agents. 2017;31(4):37-42
D’Adamio, S., Cazzato, G., Ziranu, A., Sgambato, A., Rosa, M. A., & Maccauro, G. (2017). Soft tissue adhesion patterns over Trevira tube on modular endoprosthesis for malignant bone tumours: an in vitro study. Journal of biological regulators and homeostatic agents, 31(4), 37-42.
D’Adamio, S, et al. "Soft tissue adhesion patterns over Trevira tube on modular endoprosthesis for malignant bone tumours: an in vitro study." Journal of biological regulators and homeostatic agents vol. 31,4 (2017): 37-42.
D’Adamio S, Cazzato G, Ziranu A, Sgambato A, Rosa MA, Maccauro G. Soft tissue adhesion patterns over Trevira tube on modular endoprosthesis for malignant bone tumours: an in vitro study. J Biol Regul Homeost Agents. 2017 Oct-Dec,;31(4):37-42. PMID: 29181955.
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[No title available]

Praxis

Adamo S, Gian Vital D, Zinkernagel AS, Kollias S, Steiner UC.
PMID: 29183214
Praxis (Bern 1994). 2017 Nov;106(24):1350-1353. doi: 10.1024/1661-8157/a002837.

No abstract available.

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Adamo S, Gian Vital D, Zinkernagel AS, et al. Carotidynie: eine seltene Manifestation einer nicht so seltenen Erkrankung. . Praxis (Bern 1994). 2017;106(24):1350-1353doi: 10.1024/1661-8157/a002837.
Adamo, S., Gian Vital, D., Zinkernagel, A. S., Kollias, S., & Steiner, U. C. (2017). Carotidynie: eine seltene Manifestation einer nicht so seltenen Erkrankung. . Praxis, 106(24), 1350-1353. https://doi.org/10.1024/1661-8157/a002837
Adamo, Sarah, et al. "Carotidynie: eine seltene Manifestation einer nicht so seltenen Erkrankung." . Praxis vol. 106,24 (2017): 1350-1353. doi: https://doi.org/10.1024/1661-8157/a002837
Adamo S, Gian Vital D, Zinkernagel AS, Kollias S, Steiner UC. Carotidynie: eine seltene Manifestation einer nicht so seltenen Erkrankung. . Praxis (Bern 1994). 2017 Nov;106(24):1350-1353. doi: 10.1024/1661-8157/a002837. German. PMID: 29183214.
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Showing 13 to 24 of 202 entries