Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 13 to 24 of 1303 entries
Sorted by: Best Match Show Resources per page
Relationship between triterpenoid anticancer drug resistance, autophagy, and caspase-1 in adult T-cell leukemia.

PeerJ

Nakanishi T, Song Y, He C, Wang D, Morita K, Tsukada J, Kanazawa T, Yoshida Y.
PMID: 27190722
PeerJ. 2016 May 12;4:e2026. doi: 10.7717/peerj.2026. eCollection 2016.

We previously reported that the inflammasome inhibitor cucurbitacin D (CuD) induces apoptosis in human leukemia cell lines. Here, we investigated the effects of CuD and a B-cell lymphoma extra-large (Bcl-xL) inhibitor on autophagy in peripheral blood lymphocytes (PBL) isolated...

Cite
Nakanishi T, Song Y, He C, et al. Relationship between triterpenoid anticancer drug resistance, autophagy, and caspase-1 in adult T-cell leukemia. PeerJ. 2016;4:e2026doi: 10.7717/peerj.2026.
Nakanishi, T., Song, Y., He, C., Wang, D., Morita, K., Tsukada, J., Kanazawa, T., & Yoshida, Y. (2016). Relationship between triterpenoid anticancer drug resistance, autophagy, and caspase-1 in adult T-cell leukemia. PeerJ, 4e2026. https://doi.org/10.7717/peerj.2026
Nakanishi, Tsukasa, et al. "Relationship between triterpenoid anticancer drug resistance, autophagy, and caspase-1 in adult T-cell leukemia." PeerJ vol. 4 (2016): e2026. doi: https://doi.org/10.7717/peerj.2026
Nakanishi T, Song Y, He C, Wang D, Morita K, Tsukada J, Kanazawa T, Yoshida Y. Relationship between triterpenoid anticancer drug resistance, autophagy, and caspase-1 in adult T-cell leukemia. PeerJ. 2016 May 12;4:e2026. doi: 10.7717/peerj.2026. eCollection 2016. PMID: 27190722; PMCID: PMC4868592.
Copy Download .nbib Format: NLM
Bioinformatic Studies to Predict MicroRNAs with the Potential of Uncoupling RECK Expression from Epithelial-Mesenchymal Transition in Cancer Cells.

Cancer informatics

Wang Z, Murakami R, Yuki K, Yoshida Y, Noda M.
PMID: 27226706
Cancer Inform. 2016 May 19;15:91-102. doi: 10.4137/CIN.S34141. eCollection 2016.

RECK is downregulated in many tumors, and forced RECK expression in tumor cells often results in suppression of malignant phenotypes. Recent findings suggest that RECK is upregulated after epithelial-mesenchymal transition (EMT) in normal epithelium-derived cells but not in cancer...

Cite
Wang Z, Murakami R, Yuki K, et al. Bioinformatic Studies to Predict MicroRNAs with the Potential of Uncoupling RECK Expression from Epithelial-Mesenchymal Transition in Cancer Cells. Cancer Inform. 2016;15:91-102doi: 10.4137/CIN.S34141.
Wang, Z., Murakami, R., Yuki, K., Yoshida, Y., & Noda, M. (2016). Bioinformatic Studies to Predict MicroRNAs with the Potential of Uncoupling RECK Expression from Epithelial-Mesenchymal Transition in Cancer Cells. Cancer informatics, 1591-102. https://doi.org/10.4137/CIN.S34141
Wang, Zhipeng, et al. "Bioinformatic Studies to Predict MicroRNAs with the Potential of Uncoupling RECK Expression from Epithelial-Mesenchymal Transition in Cancer Cells." Cancer informatics vol. 15 (2016): 91-102. doi: https://doi.org/10.4137/CIN.S34141
Wang Z, Murakami R, Yuki K, Yoshida Y, Noda M. Bioinformatic Studies to Predict MicroRNAs with the Potential of Uncoupling RECK Expression from Epithelial-Mesenchymal Transition in Cancer Cells. Cancer Inform. 2016 May 19;15:91-102. doi: 10.4137/CIN.S34141. eCollection 2016. PMID: 27226706; PMCID: PMC4874744.
Copy Download .nbib Format: NLM
Transient changes in electric fields induced by interaction of ultraintense laser pulses with insulator and metal foils: Sustainable fields spanning several millimeters.

Physical review. E, Statistical, nonlinear, and soft matter physics

Inoue S, Tokita S, Hashida M, Sakabe S.
PMID: 25974596
Phys Rev E Stat Nonlin Soft Matter Phys. 2015 Apr;91(4):043101. doi: 10.1103/PhysRevE.91.043101. Epub 2015 Apr 01.

The temporal evolutions of electromagnetic fields generated by the interaction between ultraintense lasers (1.3×10(18) and 8.2×10(18)W/cm(2)) and solid targets at a distance of several millimeters from the laser-irradiated region have been investigated by electron deflectometry. For three types of...

Cite
Inoue S, Tokita S, Hashida M, et al. Transient changes in electric fields induced by interaction of ultraintense laser pulses with insulator and metal foils: Sustainable fields spanning several millimeters. Phys Rev E Stat Nonlin Soft Matter Phys. 2015;91(4):043101doi: 10.1103/PhysRevE.91.043101.
Inoue, S., Tokita, S., Hashida, M., & Sakabe, S. (2015). Transient changes in electric fields induced by interaction of ultraintense laser pulses with insulator and metal foils: Sustainable fields spanning several millimeters. Physical review. E, Statistical, nonlinear, and soft matter physics, 91(4), 043101. https://doi.org/10.1103/PhysRevE.91.043101
Inoue, Shunsuke, et al. "Transient changes in electric fields induced by interaction of ultraintense laser pulses with insulator and metal foils: Sustainable fields spanning several millimeters." Physical review. E, Statistical, nonlinear, and soft matter physics vol. 91,4 (2015): 043101. doi: https://doi.org/10.1103/PhysRevE.91.043101
Inoue S, Tokita S, Hashida M, Sakabe S. Transient changes in electric fields induced by interaction of ultraintense laser pulses with insulator and metal foils: Sustainable fields spanning several millimeters. Phys Rev E Stat Nonlin Soft Matter Phys. 2015 Apr;91(4):043101. doi: 10.1103/PhysRevE.91.043101. Epub 2015 Apr 01. PMID: 25974596.
Copy Download .nbib Format: NLM
Aptamer-labeled PLGA nanoparticles for targeting cancer cells.

Cancer nanotechnology

Aravind A, Varghese SH, Veeranarayanan S, Mathew A, Nagaoka Y, Iwai S, Fukuda T, Hasumura T, Yoshida Y, Maekawa T, Kumar DS.
PMID: 26069492
Cancer Nanotechnol. 2012;3(1):1-12. doi: 10.1007/s12645-011-0024-6. Epub 2012 Jan 19.

Cancer is one of the leading causes of death in most parts of the world and is a very serious cause of concern particularly in developing countries. In this work, we prepared and evaluated the aptamer-labeled paclitaxel-loaded poly(lactic-

Cite
Aravind A, Varghese SH, Veeranarayanan S, et al. Aptamer-labeled PLGA nanoparticles for targeting cancer cells. Cancer Nanotechnol. 2012;3(1):1-12doi: 10.1007/s12645-011-0024-6.
Aravind, A., Varghese, S. H., Veeranarayanan, S., Mathew, A., Nagaoka, Y., Iwai, S., Fukuda, T., Hasumura, T., Yoshida, Y., Maekawa, T., & Kumar, D. S. (2012). Aptamer-labeled PLGA nanoparticles for targeting cancer cells. Cancer nanotechnology, 3(1), 1-12. https://doi.org/10.1007/s12645-011-0024-6
Aravind, Athulya, et al. "Aptamer-labeled PLGA nanoparticles for targeting cancer cells." Cancer nanotechnology vol. 3,1 (2012): 1-12. doi: https://doi.org/10.1007/s12645-011-0024-6
Aravind A, Varghese SH, Veeranarayanan S, Mathew A, Nagaoka Y, Iwai S, Fukuda T, Hasumura T, Yoshida Y, Maekawa T, Kumar DS. Aptamer-labeled PLGA nanoparticles for targeting cancer cells. Cancer Nanotechnol. 2012;3(1):1-12. doi: 10.1007/s12645-011-0024-6. Epub 2012 Jan 19. PMID: 26069492; PMCID: PMC4452037.
Copy Download .nbib Format: NLM
Communication: Salt-induced water orientation at a surface of non-ionic surfactant in relation to a mechanism of Hofmeister effect.

The Journal of chemical physics

Hishida M, Kaneko Y, Okuno M, Yamamura Y, Ishibashi TA, Saito K.
PMID: 25956079
J Chem Phys. 2015 May 07;142(17):171101. doi: 10.1063/1.4919664.

The behavior of water molecules at the surface of nonionic surfactant (monomyristolein) and effects of monovalent ions on the behavior are investigated using the heterodyne-detected vibrational sum frequency generation spectroscopy. It is found that water molecules at the surface...

Cite
Hishida M, Kaneko Y, Okuno M, et al. Communication: Salt-induced water orientation at a surface of non-ionic surfactant in relation to a mechanism of Hofmeister effect. J Chem Phys. 2015;142(17):171101doi: 10.1063/1.4919664.
Hishida, M., Kaneko, Y., Okuno, M., Yamamura, Y., Ishibashi, T. A., & Saito, K. (2015). Communication: Salt-induced water orientation at a surface of non-ionic surfactant in relation to a mechanism of Hofmeister effect. The Journal of chemical physics, 142(17), 171101. https://doi.org/10.1063/1.4919664
Hishida, Mafumi, et al. "Communication: Salt-induced water orientation at a surface of non-ionic surfactant in relation to a mechanism of Hofmeister effect." The Journal of chemical physics vol. 142,17 (2015): 171101. doi: https://doi.org/10.1063/1.4919664
Hishida M, Kaneko Y, Okuno M, Yamamura Y, Ishibashi TA, Saito K. Communication: Salt-induced water orientation at a surface of non-ionic surfactant in relation to a mechanism of Hofmeister effect. J Chem Phys. 2015 May 07;142(17):171101. doi: 10.1063/1.4919664. PMID: 25956079.
Copy Download .nbib Format: NLM
Oral therapy for type 1 diabetes mellitus using a novel immunomodulator, FTY720 (fingolimod), in combination with sitagliptin, a dipeptidyl peptidase-4 inhibitor, examined in non-obese diabetic mice.

Journal of diabetes investigation

Tsuji T, Yoshida Y, Fujita T, Kohno T.
PMID: 24843604
J Diabetes Investig. 2012 Oct 18;3(5):441-8. doi: 10.1111/j.2040-1124.2012.00218.x.

UNLABELLED: Aims/Introduction:  The therapeutic effectiveness against type 1 diabetes mellitus of a novel immunomodulator, FTY720 (fingolimod), in combination with sitagliptin, a dipeptidyl peptidase-4 inhibitor, was examined in the non-obese diabetic (NOD) mouse model.MATERIALS AND METHODS:   Female NOD mice that...

Cite
Tsuji T, Yoshida Y, Fujita T, et al. Oral therapy for type 1 diabetes mellitus using a novel immunomodulator, FTY720 (fingolimod), in combination with sitagliptin, a dipeptidyl peptidase-4 inhibitor, examined in non-obese diabetic mice. J Diabetes Investig. 2012;3(5):441-8doi: 10.1111/j.2040-1124.2012.00218.x.
Tsuji, T., Yoshida, Y., Fujita, T., & Kohno, T. (2012). Oral therapy for type 1 diabetes mellitus using a novel immunomodulator, FTY720 (fingolimod), in combination with sitagliptin, a dipeptidyl peptidase-4 inhibitor, examined in non-obese diabetic mice. Journal of diabetes investigation, 3(5), 441-8. https://doi.org/10.1111/j.2040-1124.2012.00218.x
Tsuji, Takumi, et al. "Oral therapy for type 1 diabetes mellitus using a novel immunomodulator, FTY720 (fingolimod), in combination with sitagliptin, a dipeptidyl peptidase-4 inhibitor, examined in non-obese diabetic mice." Journal of diabetes investigation vol. 3,5 (2012): 441-8. doi: https://doi.org/10.1111/j.2040-1124.2012.00218.x
Tsuji T, Yoshida Y, Fujita T, Kohno T. Oral therapy for type 1 diabetes mellitus using a novel immunomodulator, FTY720 (fingolimod), in combination with sitagliptin, a dipeptidyl peptidase-4 inhibitor, examined in non-obese diabetic mice. J Diabetes Investig. 2012 Oct 18;3(5):441-8. doi: 10.1111/j.2040-1124.2012.00218.x. PMID: 24843604; PMCID: PMC4019244.
Copy Download .nbib Format: NLM
Cytological Features of a Variant NUT Midline Carcinoma of the Lung Harboring the NSD3-NUT Fusion Gene: A Case Report and Literature Review.

Case reports in pathology

Kuroda S, Suzuki S, Kurita A, Muraki M, Aoshima Y, Tanioka F, Sugimura H.
PMID: 25685583
Case Rep Pathol. 2015;2015:572951. doi: 10.1155/2015/572951. Epub 2015 Jan 19.

Background. Nuclear protein in testis (NUT) midline carcinoma (NMC) is a very rare and aggressive malignancy. In more than two-thirds of these NMC cases, a fusion between NUT and BRD4 or BRD3 has been documented; other variants are rare....

Cite
Kuroda S, Suzuki S, Kurita A, et al. Cytological Features of a Variant NUT Midline Carcinoma of the Lung Harboring the NSD3-NUT Fusion Gene: A Case Report and Literature Review. Case Rep Pathol. 2015;2015:572951doi: 10.1155/2015/572951.
Kuroda, S., Suzuki, S., Kurita, A., Muraki, M., Aoshima, Y., Tanioka, F., & Sugimura, H. (2015). Cytological Features of a Variant NUT Midline Carcinoma of the Lung Harboring the NSD3-NUT Fusion Gene: A Case Report and Literature Review. Case reports in pathology, 2015572951. https://doi.org/10.1155/2015/572951
Kuroda, Shiho, et al. "Cytological Features of a Variant NUT Midline Carcinoma of the Lung Harboring the NSD3-NUT Fusion Gene: A Case Report and Literature Review." Case reports in pathology vol. 2015 (2015): 572951. doi: https://doi.org/10.1155/2015/572951
Kuroda S, Suzuki S, Kurita A, Muraki M, Aoshima Y, Tanioka F, Sugimura H. Cytological Features of a Variant NUT Midline Carcinoma of the Lung Harboring the NSD3-NUT Fusion Gene: A Case Report and Literature Review. Case Rep Pathol. 2015;2015:572951. doi: 10.1155/2015/572951. Epub 2015 Jan 19. PMID: 25685583; PMCID: PMC4320876.
Copy Download .nbib Format: NLM
Corrigendum: The tumour suppressor LKB1 regulates myelination through mitochondrial metabolism.

Nature communications

Pooya S, Liu X, Kumar VB, Anderson J, Imai F, Zhang W, Ciraolo G, Ratner N, Setchell KD, Yoshida Y, Jankowski MP, Dasgupta B.
PMID: 25574931
Nat Commun. 2015 Jan 09;6:6075. doi: 10.1038/ncomms7075.

No abstract available.

Cite
Pooya S, Liu X, Kumar VB, et al. Corrigendum: The tumour suppressor LKB1 regulates myelination through mitochondrial metabolism. Nat Commun. 2015;6:6075doi: 10.1038/ncomms7075.
Pooya, S., Liu, X., Kumar, V. B., Anderson, J., Imai, F., Zhang, W., Ciraolo, G., Ratner, N., Setchell, K. D., Yoshida, Y., Jankowski, M. P., & Dasgupta, B. (2015). Corrigendum: The tumour suppressor LKB1 regulates myelination through mitochondrial metabolism. Nature communications, 66075. https://doi.org/10.1038/ncomms7075
Pooya, Shabnam, et al. "Corrigendum: The tumour suppressor LKB1 regulates myelination through mitochondrial metabolism." Nature communications vol. 6 (2015): 6075. doi: https://doi.org/10.1038/ncomms7075
Pooya S, Liu X, Kumar VB, Anderson J, Imai F, Zhang W, Ciraolo G, Ratner N, Setchell KD, Yoshida Y, Jankowski MP, Dasgupta B. Corrigendum: The tumour suppressor LKB1 regulates myelination through mitochondrial metabolism. Nat Commun. 2015 Jan 09;6:6075. doi: 10.1038/ncomms7075. PMID: 25574931.
Copy Download .nbib Format: NLM
Genetic alterations in lung adenocarcinoma with a micropapillary component.

Molecular and clinical oncology

Furukawa M, Toyooka S, Ichimura K, Yamamoto H, Soh J, Hashida S, Ouchida M, Shien K, Asano H, Tsukuda K, Miyoshi S.
PMID: 26893860
Mol Clin Oncol. 2016 Feb;4(2):195-200. doi: 10.3892/mco.2015.690. Epub 2015 Dec 02.

Pulmonary adenocarcinoma (PA) with a micropapillary component (PA-MPC) is known as an aggressive subtype of PA. The molecular profiles of PA-MPC have not been well characterized. the pathological reports of patients who underwent surgical resection for lung cancer between...

Cite
Furukawa M, Toyooka S, Ichimura K, et al. Genetic alterations in lung adenocarcinoma with a micropapillary component. Mol Clin Oncol. 2015;4(2):195-200doi: 10.3892/mco.2015.690.
Furukawa, M., Toyooka, S., Ichimura, K., Yamamoto, H., Soh, J., Hashida, S., Ouchida, M., Shien, K., Asano, H., Tsukuda, K., & Miyoshi, S. (2016). Genetic alterations in lung adenocarcinoma with a micropapillary component. Molecular and clinical oncology, 4(2), 195-200. https://doi.org/10.3892/mco.2015.690
Furukawa, Masashi, et al. "Genetic alterations in lung adenocarcinoma with a micropapillary component." Molecular and clinical oncology vol. 4,2 (2016): 195-200. doi: https://doi.org/10.3892/mco.2015.690
Furukawa M, Toyooka S, Ichimura K, Yamamoto H, Soh J, Hashida S, Ouchida M, Shien K, Asano H, Tsukuda K, Miyoshi S. Genetic alterations in lung adenocarcinoma with a micropapillary component. Mol Clin Oncol. 2016 Feb;4(2):195-200. doi: 10.3892/mco.2015.690. Epub 2015 Dec 02. PMID: 26893860; PMCID: PMC4734214.
Copy Download .nbib Format: NLM
Reply.

The Annals of thoracic surgery

Hishida T, Tsuboi M, Okumura T, Boku N, Hyodo I, Mori K, Kondo H.
PMID: 28935314
Ann Thorac Surg. 2017 Oct;104(4):1435-1436. doi: 10.1016/j.athoracsur.2017.03.047.

No abstract available.

Cite
Hishida T, Tsuboi M, Okumura T, et al. Reply. Ann Thorac Surg. 2017;104(4):1435-1436doi: 10.1016/j.athoracsur.2017.03.047.
Hishida, T., Tsuboi, M., Okumura, T., Boku, N., Hyodo, I., Mori, K., & Kondo, H. (2017). Reply. The Annals of thoracic surgery, 104(4), 1435-1436. https://doi.org/10.1016/j.athoracsur.2017.03.047
Hishida, Tomoyuki, et al. "Reply." The Annals of thoracic surgery vol. 104,4 (2017): 1435-1436. doi: https://doi.org/10.1016/j.athoracsur.2017.03.047
Hishida T, Tsuboi M, Okumura T, Boku N, Hyodo I, Mori K, Kondo H. Reply. Ann Thorac Surg. 2017 Oct;104(4):1435-1436. doi: 10.1016/j.athoracsur.2017.03.047. PMID: 28935314.
Copy Download .nbib Format: NLM
Reduction in gastrointestinal bleeding by development and implementation of a protocol for stress ulcer prophylaxis: a before-after study.

Journal of pharmaceutical health care and sciences

Ikemura M, Nakasako S, Seo R, Atsumi T, Ariyoshi K, Hashida T.
PMID: 26819744
J Pharm Health Care Sci. 2015 Dec 15;1:33. doi: 10.1186/s40780-015-0034-3. eCollection 2015.

BACKGROUND: The implementation of a protocol has been associated with improvements in the processes of care in clinical settings. Although stress ulcer prophylaxis is recommended for critically ill patients at high risk, there is currently no consensus on its...

Cite
Ikemura M, Nakasako S, Seo R, et al. Reduction in gastrointestinal bleeding by development and implementation of a protocol for stress ulcer prophylaxis: a before-after study. J Pharm Health Care Sci. 2015;1:33doi: 10.1186/s40780-015-0034-3.
Ikemura, M., Nakasako, S., Seo, R., Atsumi, T., Ariyoshi, K., & Hashida, T. (2015). Reduction in gastrointestinal bleeding by development and implementation of a protocol for stress ulcer prophylaxis: a before-after study. Journal of pharmaceutical health care and sciences, 133. https://doi.org/10.1186/s40780-015-0034-3
Ikemura, Mai, et al. "Reduction in gastrointestinal bleeding by development and implementation of a protocol for stress ulcer prophylaxis: a before-after study." Journal of pharmaceutical health care and sciences vol. 1 (2015): 33. doi: https://doi.org/10.1186/s40780-015-0034-3
Ikemura M, Nakasako S, Seo R, Atsumi T, Ariyoshi K, Hashida T. Reduction in gastrointestinal bleeding by development and implementation of a protocol for stress ulcer prophylaxis: a before-after study. J Pharm Health Care Sci. 2015 Dec 15;1:33. doi: 10.1186/s40780-015-0034-3. eCollection 2015. PMID: 26819744; PMCID: PMC4729099.
Copy Download .nbib Format: NLM
Who are appropriate surgical candidates among patients with oligometastatic non-small cell lung cancer?.

Journal of thoracic disease

Hishida T.
PMID: 28275501
J Thorac Dis. 2017 Feb;9(2):E149-E151. doi: 10.21037/jtd.2017.02.25.

No abstract available.

Cite
Hishida T. Who are appropriate surgical candidates among patients with oligometastatic non-small cell lung cancer?. J Thorac Dis. 2017;9(2):E149-E151doi: 10.21037/jtd.2017.02.25.
Hishida, T. (2017). Who are appropriate surgical candidates among patients with oligometastatic non-small cell lung cancer?. Journal of thoracic disease, 9(2), E149-E151. https://doi.org/10.21037/jtd.2017.02.25
Hishida, Tomoyuki. "Who are appropriate surgical candidates among patients with oligometastatic non-small cell lung cancer?." Journal of thoracic disease vol. 9,2 (2017): E149-E151. doi: https://doi.org/10.21037/jtd.2017.02.25
Hishida T. Who are appropriate surgical candidates among patients with oligometastatic non-small cell lung cancer?. J Thorac Dis. 2017 Feb;9(2):E149-E151. doi: 10.21037/jtd.2017.02.25. PMID: 28275501; PMCID: PMC5334073.
Copy Download .nbib Format: NLM
Showing 13 to 24 of 1303 entries