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Risk of perforation in balloon dilation associated with steroid injection for preventing esophageal stricture after endoscopic submucosal dissection.

Endoscopy international open

Tsujii Y, Hayashi Y, Kawai N, Yamada T, Yamamoto K, Hayashi S, Yoshii S, Nagai K, Inoue T, Nishida T, Iijima H, Mita E, Inoue A, Takehara T.
PMID: 28670613
Endosc Int Open. 2017 Jul;5(7):E573-E579. doi: 10.1055/s-0043-110077. Epub 2017 Jun 23.

BACKGROUND AND STUDY AIMS:  Endoscopic local steroid injection (LSI) has been used to prevent esophageal strictures after endoscopic submucosal dissection (ESD) for superficial esophageal neoplasms (SENs). This study aimed to evaluate the safety and efficacy of LSI therapy.PATIENTS AND...

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Tsujii Y, Hayashi Y, Kawai N, et al. Risk of perforation in balloon dilation associated with steroid injection for preventing esophageal stricture after endoscopic submucosal dissection. Endosc Int Open. 2017;5(7):E573-E579doi: 10.1055/s-0043-110077.
Tsujii, Y., Hayashi, Y., Kawai, N., Yamada, T., Yamamoto, K., Hayashi, S., Yoshii, S., Nagai, K., Inoue, T., Nishida, T., Iijima, H., Mita, E., Inoue, A., & Takehara, T. (2017). Risk of perforation in balloon dilation associated with steroid injection for preventing esophageal stricture after endoscopic submucosal dissection. Endoscopy international open, 5(7), E573-E579. https://doi.org/10.1055/s-0043-110077
Tsujii, Yoshiki, et al. "Risk of perforation in balloon dilation associated with steroid injection for preventing esophageal stricture after endoscopic submucosal dissection." Endoscopy international open vol. 5,7 (2017): E573-E579. doi: https://doi.org/10.1055/s-0043-110077
Tsujii Y, Hayashi Y, Kawai N, Yamada T, Yamamoto K, Hayashi S, Yoshii S, Nagai K, Inoue T, Nishida T, Iijima H, Mita E, Inoue A, Takehara T. Risk of perforation in balloon dilation associated with steroid injection for preventing esophageal stricture after endoscopic submucosal dissection. Endosc Int Open. 2017 Jul;5(7):E573-E579. doi: 10.1055/s-0043-110077. Epub 2017 Jun 23. PMID: 28670613; PMCID: PMC5482748.
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New Insight into Atherosclerosis in Hemodialysis Patients: Overexpression of Scavenger Receptor and Macrophage Colony-Stimulating Factor Genes.

Nephron extra

Nishida M, Ando M, Iwamoto Y, Tsuchiya K, Nitta K.
PMID: 27721822
Nephron Extra. 2016 Aug 27;6(2):22-30. doi: 10.1159/000448486. eCollection 2016.

BACKGROUND: Scavenger receptors (SRs) play a pivotal role in atherogenesis. The mechanism of atherosclerosis, which is specific to hemodialysis (HD) patients, was studied on the basis of SR gene expressions.METHODS: The gene expressions of SR type A (SR-A) and...

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Nishida M, Ando M, Iwamoto Y, et al. New Insight into Atherosclerosis in Hemodialysis Patients: Overexpression of Scavenger Receptor and Macrophage Colony-Stimulating Factor Genes. Nephron Extra. 2016;6(2):22-30doi: 10.1159/000448486.
Nishida, M., Ando, M., Iwamoto, Y., Tsuchiya, K., & Nitta, K. (2016). New Insight into Atherosclerosis in Hemodialysis Patients: Overexpression of Scavenger Receptor and Macrophage Colony-Stimulating Factor Genes. Nephron extra, 6(2), 22-30. https://doi.org/10.1159/000448486
Nishida, Miki, et al. "New Insight into Atherosclerosis in Hemodialysis Patients: Overexpression of Scavenger Receptor and Macrophage Colony-Stimulating Factor Genes." Nephron extra vol. 6,2 (2016): 22-30. doi: https://doi.org/10.1159/000448486
Nishida M, Ando M, Iwamoto Y, Tsuchiya K, Nitta K. New Insight into Atherosclerosis in Hemodialysis Patients: Overexpression of Scavenger Receptor and Macrophage Colony-Stimulating Factor Genes. Nephron Extra. 2016 Aug 27;6(2):22-30. doi: 10.1159/000448486. eCollection 2016. PMID: 27721822; PMCID: PMC5040927.
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Enhanced heterologous production of eicosapentaenoic acid in Escherichia coli cells that co-express eicosapentaenoic acid biosynthesis pfa genes and foreign DNA fragments including a high-performance catalase gene, vktA.

Biotechnology letters

Orikasa Y, Ito Y, Nishida T, Watanabe K, Morita N, Ohwada T, Yumoto I, Okuyama H.
PMID: 27519303
Biotechnol Lett. 2007 May;29(5):811-2. doi: 10.1007/s10529-007-9343-4.

No abstract available.

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Orikasa Y, Ito Y, Nishida T, et al. Enhanced heterologous production of eicosapentaenoic acid in Escherichia coli cells that co-express eicosapentaenoic acid biosynthesis pfa genes and foreign DNA fragments including a high-performance catalase gene, vktA. Biotechnol Lett. 2007;29(5):811-2doi: 10.1007/s10529-007-9343-4.
Orikasa, Y., Ito, Y., Nishida, T., Watanabe, K., Morita, N., Ohwada, T., Yumoto, I., & Okuyama, H. (2007). Enhanced heterologous production of eicosapentaenoic acid in Escherichia coli cells that co-express eicosapentaenoic acid biosynthesis pfa genes and foreign DNA fragments including a high-performance catalase gene, vktA. Biotechnology letters, 29(5), 811-2. https://doi.org/10.1007/s10529-007-9343-4
Orikasa, Yoshitake, et al. "Enhanced heterologous production of eicosapentaenoic acid in Escherichia coli cells that co-express eicosapentaenoic acid biosynthesis pfa genes and foreign DNA fragments including a high-performance catalase gene, vktA." Biotechnology letters vol. 29,5 (2007): 811-2. doi: https://doi.org/10.1007/s10529-007-9343-4
Orikasa Y, Ito Y, Nishida T, Watanabe K, Morita N, Ohwada T, Yumoto I, Okuyama H. Enhanced heterologous production of eicosapentaenoic acid in Escherichia coli cells that co-express eicosapentaenoic acid biosynthesis pfa genes and foreign DNA fragments including a high-performance catalase gene, vktA. Biotechnol Lett. 2007 May;29(5):811-2. doi: 10.1007/s10529-007-9343-4. PMID: 27519303.
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Downregulation of miR-186 is associated with metastatic recurrence of gastrointestinal stromal tumors.

Oncology letters

Niinuma T, Kai M, Kitajima H, Yamamoto E, Harada T, Maruyama R, Nobuoka T, Nishida T, Kanda T, Hasegawa T, Tokino T, Sugai T, Shinomura Y, Nakase H, Suzuki H.
PMID: 29113198
Oncol Lett. 2017 Nov;14(5):5703-5710. doi: 10.3892/ol.2017.6911. Epub 2017 Sep 08.

Although dysregulation of microRNAs (miRNAs/miRs) is a common feature of human malignancies, its involvement in gastrointestinal stromal tumors (GISTs) is not fully understood. The present study aimed to identify the miRNAs that perform a role in GIST metastasis. miRNA...

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Niinuma T, Kai M, Kitajima H, et al. Downregulation of miR-186 is associated with metastatic recurrence of gastrointestinal stromal tumors. Oncol Lett. 2017;14(5):5703-5710doi: 10.3892/ol.2017.6911.
Niinuma, T., Kai, M., Kitajima, H., Yamamoto, E., Harada, T., Maruyama, R., Nobuoka, T., Nishida, T., Kanda, T., Hasegawa, T., Tokino, T., Sugai, T., Shinomura, Y., Nakase, H., & Suzuki, H. (2017). Downregulation of miR-186 is associated with metastatic recurrence of gastrointestinal stromal tumors. Oncology letters, 14(5), 5703-5710. https://doi.org/10.3892/ol.2017.6911
Niinuma, Takeshi, et al. "Downregulation of miR-186 is associated with metastatic recurrence of gastrointestinal stromal tumors." Oncology letters vol. 14,5 (2017): 5703-5710. doi: https://doi.org/10.3892/ol.2017.6911
Niinuma T, Kai M, Kitajima H, Yamamoto E, Harada T, Maruyama R, Nobuoka T, Nishida T, Kanda T, Hasegawa T, Tokino T, Sugai T, Shinomura Y, Nakase H, Suzuki H. Downregulation of miR-186 is associated with metastatic recurrence of gastrointestinal stromal tumors. Oncol Lett. 2017 Nov;14(5):5703-5710. doi: 10.3892/ol.2017.6911. Epub 2017 Sep 08. PMID: 29113198; PMCID: PMC5661378.
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The E3 ubiquitin ligase Hace1 is required for early embryonic development in Xenopus laevis.

BMC developmental biology

Iimura A, Yamazaki F, Suzuki T, Endo T, Nishida E, Kusakabe M.
PMID: 27653971
BMC Dev Biol. 2016 Sep 21;16(1):31. doi: 10.1186/s12861-016-0132-y.

BACKGROUND: HECT domain and ankyrin repeat containing E3 ubiquitin protein ligase 1 (HACE1) regulates a wide variety of cellular processes. It has been shown that one of the targets of HACE1 is the GTP-bound form of the small GTPase...

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Iimura A, Yamazaki F, Suzuki T, et al. The E3 ubiquitin ligase Hace1 is required for early embryonic development in Xenopus laevis. BMC Dev Biol. 2016;16(1):31doi: 10.1186/s12861-016-0132-y.
Iimura, A., Yamazaki, F., Suzuki, T., Endo, T., Nishida, E., & Kusakabe, M. (2016). The E3 ubiquitin ligase Hace1 is required for early embryonic development in Xenopus laevis. BMC developmental biology, 16(1), 31. https://doi.org/10.1186/s12861-016-0132-y
Iimura, Akira, et al. "The E3 ubiquitin ligase Hace1 is required for early embryonic development in Xenopus laevis." BMC developmental biology vol. 16,1 (2016): 31. doi: https://doi.org/10.1186/s12861-016-0132-y
Iimura A, Yamazaki F, Suzuki T, Endo T, Nishida E, Kusakabe M. The E3 ubiquitin ligase Hace1 is required for early embryonic development in Xenopus laevis. BMC Dev Biol. 2016 Sep 21;16(1):31. doi: 10.1186/s12861-016-0132-y. PMID: 27653971; PMCID: PMC5031333.
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Late-onset myasthenia gravis is predisposed to become generalized in the elderly.

eNeurologicalSci

Sakai W, Matsui N, Ishida M, Furukawa T, Miyazaki Y, Fujita K, Miyamoto R, Yamamoto N, Sako W, Sato K, Kondo K, Nishida Y, Mitsui T, Izumi Y, Kaji R.
PMID: 29473057
eNeurologicalSci. 2016 Feb 11;2:17-20. doi: 10.1016/j.ensci.2016.02.004. eCollection 2016 Mar.

OBJECTIVE: The continuous increase in the number of patients presenting with late-onset myasthenia gravis (LOMG) underscores the need for a better understanding of the clinical course and the establishment of an optimal therapeutic strategy. We aimed to clarify factors...

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Sakai W, Matsui N, Ishida M, et al. Late-onset myasthenia gravis is predisposed to become generalized in the elderly. eNeurologicalSci. 2016;2:17-20doi: 10.1016/j.ensci.2016.02.004.
Sakai, W., Matsui, N., Ishida, M., Furukawa, T., Miyazaki, Y., Fujita, K., Miyamoto, R., Yamamoto, N., Sako, W., Sato, K., Kondo, K., Nishida, Y., Mitsui, T., Izumi, Y., & Kaji, R. (2016). Late-onset myasthenia gravis is predisposed to become generalized in the elderly. eNeurologicalSci, 217-20. https://doi.org/10.1016/j.ensci.2016.02.004
Sakai, Waka, et al. "Late-onset myasthenia gravis is predisposed to become generalized in the elderly." eNeurologicalSci vol. 2 (2016): 17-20. doi: https://doi.org/10.1016/j.ensci.2016.02.004
Sakai W, Matsui N, Ishida M, Furukawa T, Miyazaki Y, Fujita K, Miyamoto R, Yamamoto N, Sako W, Sato K, Kondo K, Nishida Y, Mitsui T, Izumi Y, Kaji R. Late-onset myasthenia gravis is predisposed to become generalized in the elderly. eNeurologicalSci. 2016 Feb 11;2:17-20. doi: 10.1016/j.ensci.2016.02.004. eCollection 2016 Mar. PMID: 29473057; PMCID: PMC5818137.
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Cardiac arrest caused by sibutramine obtained over the Internet: a case of a young woman without pre-existing cardiovascular disease successfully resuscitated using extracorporeal membrane oxygenation.

Acute medicine & surgery

Bunya N, Sawamoto K, Uemura S, Kyan R, Inoue H, Nishida J, Kouzu H, Kokubu N, Miura T, Narimatsu E.
PMID: 29123885
Acute Med Surg. 2017 Mar 29;4(3):334-337. doi: 10.1002/ams2.275. eCollection 2017 Jul.

CASE: Sibutramine is a weight loss agent that was withdrawn from the market in the USA and European Union because it increases adverse events in patients with cardiovascular diseases. However, non-prescription weight loss pills containing sibutramine can be still...

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Bunya N, Sawamoto K, Uemura S, et al. Cardiac arrest caused by sibutramine obtained over the Internet: a case of a young woman without pre-existing cardiovascular disease successfully resuscitated using extracorporeal membrane oxygenation. Acute Med Surg. 2017;4(3):334-337doi: 10.1002/ams2.275.
Bunya, N., Sawamoto, K., Uemura, S., Kyan, R., Inoue, H., Nishida, J., Kouzu, H., Kokubu, N., Miura, T., & Narimatsu, E. (2017). Cardiac arrest caused by sibutramine obtained over the Internet: a case of a young woman without pre-existing cardiovascular disease successfully resuscitated using extracorporeal membrane oxygenation. Acute medicine & surgery, 4(3), 334-337. https://doi.org/10.1002/ams2.275
Bunya, Naofumi, et al. "Cardiac arrest caused by sibutramine obtained over the Internet: a case of a young woman without pre-existing cardiovascular disease successfully resuscitated using extracorporeal membrane oxygenation." Acute medicine & surgery vol. 4,3 (2017): 334-337. doi: https://doi.org/10.1002/ams2.275
Bunya N, Sawamoto K, Uemura S, Kyan R, Inoue H, Nishida J, Kouzu H, Kokubu N, Miura T, Narimatsu E. Cardiac arrest caused by sibutramine obtained over the Internet: a case of a young woman without pre-existing cardiovascular disease successfully resuscitated using extracorporeal membrane oxygenation. Acute Med Surg. 2017 Mar 29;4(3):334-337. doi: 10.1002/ams2.275. eCollection 2017 Jul. PMID: 29123885; PMCID: PMC5674461.
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Corrigendum: Reconsidering Tonotopic Maps in the Auditory Cortex and Lemniscal Auditory Thalamus in Mice.

Frontiers in neural circuits

Tsukano H, Horie M, Ohga S, Takahashi K, Kubota Y, Hishida R, Takebayashi H, Shibuki K.
PMID: 28579946
Front Neural Circuits. 2017 May 31;11:39. doi: 10.3389/fncir.2017.00039. eCollection 2017.

[This corrects the article on p. 14 in vol. 11, PMID: 28293178.].

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Tsukano H, Horie M, Ohga S, et al. Corrigendum: Reconsidering Tonotopic Maps in the Auditory Cortex and Lemniscal Auditory Thalamus in Mice. Front Neural Circuits. 2017;11:39doi: 10.3389/fncir.2017.00039.
Tsukano, H., Horie, M., Ohga, S., Takahashi, K., Kubota, Y., Hishida, R., Takebayashi, H., & Shibuki, K. (2017). Corrigendum: Reconsidering Tonotopic Maps in the Auditory Cortex and Lemniscal Auditory Thalamus in Mice. Frontiers in neural circuits, 1139. https://doi.org/10.3389/fncir.2017.00039
Tsukano, Hiroaki, et al. "Corrigendum: Reconsidering Tonotopic Maps in the Auditory Cortex and Lemniscal Auditory Thalamus in Mice." Frontiers in neural circuits vol. 11 (2017): 39. doi: https://doi.org/10.3389/fncir.2017.00039
Tsukano H, Horie M, Ohga S, Takahashi K, Kubota Y, Hishida R, Takebayashi H, Shibuki K. Corrigendum: Reconsidering Tonotopic Maps in the Auditory Cortex and Lemniscal Auditory Thalamus in Mice. Front Neural Circuits. 2017 May 31;11:39. doi: 10.3389/fncir.2017.00039. eCollection 2017. PMID: 28579946; PMCID: PMC5449952.
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A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report.

Journal of thoracic disease

Shiroyama T, Tamiya M, Hayama M, Nishihara T, Nishida T, Tanaka A, Morishita N, Suzuki H, Okamoto N, Kawahara K, Hirashima T.
PMID: 27162697
J Thorac Dis. 2016 May;8(5):E345-8. doi: 10.21037/jtd.2016.03.43.

An 87-year-old man with postoperative recurrent lung adenocarcinoma was treated with gefitinib. At the beginning of treatment, surgically resected archived tumor tissue revealed a deletion in exon 19 of the EGFR gene, but no ALK gene rearrangement. After gefitinib...

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Shiroyama T, Tamiya M, Hayama M, et al. A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report. J Thorac Dis. 2016;8(5):E345-8doi: 10.21037/jtd.2016.03.43.
Shiroyama, T., Tamiya, M., Hayama, M., Nishihara, T., Nishida, T., Tanaka, A., Morishita, N., Suzuki, H., Okamoto, N., Kawahara, K., & Hirashima, T. (2016). A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report. Journal of thoracic disease, 8(5), E345-8. https://doi.org/10.21037/jtd.2016.03.43
Shiroyama, Takayuki, et al. "A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report." Journal of thoracic disease vol. 8,5 (2016): E345-8. doi: https://doi.org/10.21037/jtd.2016.03.43
Shiroyama T, Tamiya M, Hayama M, Nishihara T, Nishida T, Tanaka A, Morishita N, Suzuki H, Okamoto N, Kawahara K, Hirashima T. A heterochronic genetic change from an EGFR mutation to an ALK rearrangement in a patient with lung adenocarcinoma: a case report. J Thorac Dis. 2016 May;8(5):E345-8. doi: 10.21037/jtd.2016.03.43. PMID: 27162697; PMCID: PMC4842787.
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A humanized anti-CD26 monoclonal antibody inhibits cell growth of malignant mesothelioma via retarded G2/M cell cycle transition.

Cancer cell international

Hayashi M, Madokoro H, Yamada K, Nishida H, Morimoto C, Sakamoto M, Yamada T.
PMID: 27134571
Cancer Cell Int. 2016 Apr 30;16:35. doi: 10.1186/s12935-016-0310-9. eCollection 2016.

BACKGROUND: Malignant Mesothelioma (MM) is a highly aggressive tumor with poor prognosis. Multimodal treatments and novel molecular targeted therapies against MM are in high demand in order treat this disease effectively. We have developed a humanized monoclonal antibody YS110...

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Hayashi M, Madokoro H, Yamada K, et al. A humanized anti-CD26 monoclonal antibody inhibits cell growth of malignant mesothelioma via retarded G2/M cell cycle transition. Cancer Cell Int. 2016;16:35doi: 10.1186/s12935-016-0310-9.
Hayashi, M., Madokoro, H., Yamada, K., Nishida, H., Morimoto, C., Sakamoto, M., & Yamada, T. (2016). A humanized anti-CD26 monoclonal antibody inhibits cell growth of malignant mesothelioma via retarded G2/M cell cycle transition. Cancer cell international, 1635. https://doi.org/10.1186/s12935-016-0310-9
Hayashi, Mutsumi, et al. "A humanized anti-CD26 monoclonal antibody inhibits cell growth of malignant mesothelioma via retarded G2/M cell cycle transition." Cancer cell international vol. 16 (2016): 35. doi: https://doi.org/10.1186/s12935-016-0310-9
Hayashi M, Madokoro H, Yamada K, Nishida H, Morimoto C, Sakamoto M, Yamada T. A humanized anti-CD26 monoclonal antibody inhibits cell growth of malignant mesothelioma via retarded G2/M cell cycle transition. Cancer Cell Int. 2016 Apr 30;16:35. doi: 10.1186/s12935-016-0310-9. eCollection 2016. PMID: 27134571; PMCID: PMC4851800.
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Metabolic regulation of the CCN family genes by glycolysis in chondrocytes.

Journal of cell communication and signaling

Akashi S, Nishida T, El-Seoudi A, Takigawa M, Iida S, Kubota S.
PMID: 29129024
J Cell Commun Signal. 2018 Mar;12(1):245-252. doi: 10.1007/s12079-017-0420-8. Epub 2017 Nov 11.

The CCN family consists of 6 genes in the mammalian genome and produces multifunctional proteins involved in a variety of biological processes. Recent reports indicate the profound roles of CCN2 in energy metabolism in chondrocytes, and Ccn2 deficiency is...

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Akashi S, Nishida T, El-Seoudi A, et al. Metabolic regulation of the CCN family genes by glycolysis in chondrocytes. J Cell Commun Signal. 2017;12(1):245-252doi: 10.1007/s12079-017-0420-8.
Akashi, S., Nishida, T., El-Seoudi, A., Takigawa, M., Iida, S., & Kubota, S. (2018). Metabolic regulation of the CCN family genes by glycolysis in chondrocytes. Journal of cell communication and signaling, 12(1), 245-252. https://doi.org/10.1007/s12079-017-0420-8
Akashi, Sho, et al. "Metabolic regulation of the CCN family genes by glycolysis in chondrocytes." Journal of cell communication and signaling vol. 12,1 (2018): 245-252. doi: https://doi.org/10.1007/s12079-017-0420-8
Akashi S, Nishida T, El-Seoudi A, Takigawa M, Iida S, Kubota S. Metabolic regulation of the CCN family genes by glycolysis in chondrocytes. J Cell Commun Signal. 2018 Mar;12(1):245-252. doi: 10.1007/s12079-017-0420-8. Epub 2017 Nov 11. PMID: 29129024; PMCID: PMC5842183.
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A Comparative Effectiveness Study of Renal Parameters Between Imidapril and Amlodipine in Patients with Hypertension: A Retrospective Cohort Study.

Cardiology and therapy

Nishida Y, Takahashi Y, Tezuka K, Takeuchi S, Nakayama T, Asai S.
PMID: 28044266
Cardiol Ther. 2017 Jun;6(1):69-80. doi: 10.1007/s40119-016-0080-4. Epub 2017 Jan 02.

INTRODUCTION: Imidapril is an angiotensin converting enzyme inhibitor (ACEI) that is frequently used as an antihypertensive drug in Japan. Although ACEIs are known to have adverse effects of decreasing glomerular filtration rate (GFR) and causing hyperkalemia, there are very...

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Nishida Y, Takahashi Y, Tezuka K, et al. A Comparative Effectiveness Study of Renal Parameters Between Imidapril and Amlodipine in Patients with Hypertension: A Retrospective Cohort Study. Cardiol Ther. 2017;6(1):69-80doi: 10.1007/s40119-016-0080-4.
Nishida, Y., Takahashi, Y., Tezuka, K., Takeuchi, S., Nakayama, T., & Asai, S. (2017). A Comparative Effectiveness Study of Renal Parameters Between Imidapril and Amlodipine in Patients with Hypertension: A Retrospective Cohort Study. Cardiology and therapy, 6(1), 69-80. https://doi.org/10.1007/s40119-016-0080-4
Nishida, Yayoi, et al. "A Comparative Effectiveness Study of Renal Parameters Between Imidapril and Amlodipine in Patients with Hypertension: A Retrospective Cohort Study." Cardiology and therapy vol. 6,1 (2017): 69-80. doi: https://doi.org/10.1007/s40119-016-0080-4
Nishida Y, Takahashi Y, Tezuka K, Takeuchi S, Nakayama T, Asai S. A Comparative Effectiveness Study of Renal Parameters Between Imidapril and Amlodipine in Patients with Hypertension: A Retrospective Cohort Study. Cardiol Ther. 2017 Jun;6(1):69-80. doi: 10.1007/s40119-016-0080-4. Epub 2017 Jan 02. PMID: 28044266; PMCID: PMC5446813.
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