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Application of high-intensity focused ultrasound for fetal therapy: experimental study using an animal model of lower urinary tract obstruction.

Journal of medical ultrasonics (2001)

Aoki H, Ichizuka K, Ichihara M, Matsuoka R, Hasegawa J, Okai T, Umemura S.
PMID: 27277098
J Med Ultrason (2001). 2013 Apr;40(2):107-10. doi: 10.1007/s10396-012-0398-z. Epub 2012 Sep 08.

OBJECTIVE: The purpose of this study is to investigate whether high-intensity focused ultrasound (HIFU) exposure is able to produce a fistula between the bladder and abdominal wall of a fetus with lower urinary tract obstruction (LUTO).MATERIALS AND METHODS: We...

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Aoki H, Ichizuka K, Ichihara M, et al. Application of high-intensity focused ultrasound for fetal therapy: experimental study using an animal model of lower urinary tract obstruction. J Med Ultrason (2001). 2012;40(2):107-10doi: 10.1007/s10396-012-0398-z.
Aoki, H., Ichizuka, K., Ichihara, M., Matsuoka, R., Hasegawa, J., Okai, T., & Umemura, S. (2013). Application of high-intensity focused ultrasound for fetal therapy: experimental study using an animal model of lower urinary tract obstruction. Journal of medical ultrasonics (2001), 40(2), 107-10. https://doi.org/10.1007/s10396-012-0398-z
Aoki, Hiroko, et al. "Application of high-intensity focused ultrasound for fetal therapy: experimental study using an animal model of lower urinary tract obstruction." Journal of medical ultrasonics (2001) vol. 40,2 (2013): 107-10. doi: https://doi.org/10.1007/s10396-012-0398-z
Aoki H, Ichizuka K, Ichihara M, Matsuoka R, Hasegawa J, Okai T, Umemura S. Application of high-intensity focused ultrasound for fetal therapy: experimental study using an animal model of lower urinary tract obstruction. J Med Ultrason (2001). 2013 Apr;40(2):107-10. doi: 10.1007/s10396-012-0398-z. Epub 2012 Sep 08. PMID: 27277098.
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TNFα in liver fibrosis.

Current pathobiology reports

Yang YM, Seki E.
PMID: 26726307
Curr Pathobiol Rep. 2015 Dec;3(4):253-261. doi: 10.1007/s40139-015-0093-z. Epub 2015 Sep 30.

Hepatocyte death, inflammation, and liver fibrosis are the hallmarks of chronic liver disease. Tumor necrosis factor-α (TNFα) is an inflammatory cytokine involved in liver inflammation and sustained liver inflammation leads to liver fibrosis. TNFα exerts inflammation, proliferation, and apoptosis....

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Yang YM, Seki E. TNFα in liver fibrosis. Curr Pathobiol Rep. 2015;3(4):253-261doi: 10.1007/s40139-015-0093-z.
Yang, Y. M., & Seki, E. (2015). TNFα in liver fibrosis. Current pathobiology reports, 3(4), 253-261. https://doi.org/10.1007/s40139-015-0093-z
Yang, Yoon Mee, and Seki, Ekihiro. "TNFα in liver fibrosis." Current pathobiology reports vol. 3,4 (2015): 253-261. doi: https://doi.org/10.1007/s40139-015-0093-z
Yang YM, Seki E. TNFα in liver fibrosis. Curr Pathobiol Rep. 2015 Dec;3(4):253-261. doi: 10.1007/s40139-015-0093-z. Epub 2015 Sep 30. PMID: 26726307; PMCID: PMC4693602.
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Interstitial pneumonia induced by sorafenib in a patient with hepatocellular carcinoma: An autopsy case report.

Oncology letters

Yamaguchi T, Seki T, Miyasaka C, Inokuchi R, Kawamura R, Sakaguchi Y, Murata M, Matsuzaki K, Nakano Y, Uemura Y, Okazaki K.
PMID: 25789013
Oncol Lett. 2015 Apr;9(4):1633-1636. doi: 10.3892/ol.2015.2934. Epub 2015 Feb 05.

Sorafenib is a multikinase inhibitor currently approved in Japan for the treatment of unresectable hepatocellular carcinoma. Interstitial pneumonia induced by sorafenib may have a fatal outcome, and therefore, has recently been the focus of many studies. The current report...

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Yamaguchi T, Seki T, Miyasaka C, et al. Interstitial pneumonia induced by sorafenib in a patient with hepatocellular carcinoma: An autopsy case report. Oncol Lett. 2015;9(4):1633-1636doi: 10.3892/ol.2015.2934.
Yamaguchi, T., Seki, T., Miyasaka, C., Inokuchi, R., Kawamura, R., Sakaguchi, Y., Murata, M., Matsuzaki, K., Nakano, Y., Uemura, Y., & Okazaki, K. (2015). Interstitial pneumonia induced by sorafenib in a patient with hepatocellular carcinoma: An autopsy case report. Oncology letters, 9(4), 1633-1636. https://doi.org/10.3892/ol.2015.2934
Yamaguchi, Takashi, et al. "Interstitial pneumonia induced by sorafenib in a patient with hepatocellular carcinoma: An autopsy case report." Oncology letters vol. 9,4 (2015): 1633-1636. doi: https://doi.org/10.3892/ol.2015.2934
Yamaguchi T, Seki T, Miyasaka C, Inokuchi R, Kawamura R, Sakaguchi Y, Murata M, Matsuzaki K, Nakano Y, Uemura Y, Okazaki K. Interstitial pneumonia induced by sorafenib in a patient with hepatocellular carcinoma: An autopsy case report. Oncol Lett. 2015 Apr;9(4):1633-1636. doi: 10.3892/ol.2015.2934. Epub 2015 Feb 05. PMID: 25789013; PMCID: PMC4356407.
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Light-melt adhesive based on dynamic carbon frameworks in a columnar liquid-crystal phase.

Nature communications

Saito S, Nobusue S, Tsuzaka E, Yuan C, Mori C, Hara M, Seki T, Camacho C, Irle S, Yamaguchi S.
PMID: 27373592
Nat Commun. 2016 Jul 04;7:12094. doi: 10.1038/ncomms12094.

Liquid crystal (LC) provides a suitable platform to exploit structural motions of molecules in a condensed phase. Amplification of the structural changes enables a variety of technologies not only in LC displays but also in other applications. Until very...

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Saito S, Nobusue S, Tsuzaka E, et al. Light-melt adhesive based on dynamic carbon frameworks in a columnar liquid-crystal phase. Nat Commun. 2016;7:12094doi: 10.1038/ncomms12094.
Saito, S., Nobusue, S., Tsuzaka, E., Yuan, C., Mori, C., Hara, M., Seki, T., Camacho, C., Irle, S., & Yamaguchi, S. (2016). Light-melt adhesive based on dynamic carbon frameworks in a columnar liquid-crystal phase. Nature communications, 712094. https://doi.org/10.1038/ncomms12094
Saito, Shohei, et al. "Light-melt adhesive based on dynamic carbon frameworks in a columnar liquid-crystal phase." Nature communications vol. 7 (2016): 12094. doi: https://doi.org/10.1038/ncomms12094
Saito S, Nobusue S, Tsuzaka E, Yuan C, Mori C, Hara M, Seki T, Camacho C, Irle S, Yamaguchi S. Light-melt adhesive based on dynamic carbon frameworks in a columnar liquid-crystal phase. Nat Commun. 2016 Jul 04;7:12094. doi: 10.1038/ncomms12094. PMID: 27373592; PMCID: PMC4932191.
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Efficacy of tolvaptan in patients with refractory ascites in a clinical setting.

World journal of hepatology

Ohki T, Sato K, Yamada T, Yamagami M, Ito D, Kawanishi K, Kojima K, Seki M, Toda N, Tagawa K.
PMID: 26140088
World J Hepatol. 2015 Jun 28;7(12):1685-93. doi: 10.4254/wjh.v7.i12.1685.

AIM: To elucidate the efficacies of tolvaptan (TLV) as a treatment for refractory ascites compared with conventional treatment.METHODS: We retrospectively enrolled 120 refractory ascites patients between January 1, 2009 and September 31, 2014. Sixty patients were treated with oral...

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Ohki T, Sato K, Yamada T, et al. Efficacy of tolvaptan in patients with refractory ascites in a clinical setting. World J Hepatol. 2015;7(12):1685-93doi: 10.4254/wjh.v7.i12.1685.
Ohki, T., Sato, K., Yamada, T., Yamagami, M., Ito, D., Kawanishi, K., Kojima, K., Seki, M., Toda, N., & Tagawa, K. (2015). Efficacy of tolvaptan in patients with refractory ascites in a clinical setting. World journal of hepatology, 7(12), 1685-93. https://doi.org/10.4254/wjh.v7.i12.1685
Ohki, Takamasa, et al. "Efficacy of tolvaptan in patients with refractory ascites in a clinical setting." World journal of hepatology vol. 7,12 (2015): 1685-93. doi: https://doi.org/10.4254/wjh.v7.i12.1685
Ohki T, Sato K, Yamada T, Yamagami M, Ito D, Kawanishi K, Kojima K, Seki M, Toda N, Tagawa K. Efficacy of tolvaptan in patients with refractory ascites in a clinical setting. World J Hepatol. 2015 Jun 28;7(12):1685-93. doi: 10.4254/wjh.v7.i12.1685. PMID: 26140088; PMCID: PMC4483550.
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Assessment of the features of serum apolipoprotein profiles in chronic HCV infection: difference between HCV genotypes 1b and 2.

Hepatology international

Seki N, Sugita T, Aida Y, Itagaki M, Ishiguro H, Sutoh S, Abe H, Tsubota A, Matsushima M, Aizawa Y.
PMID: 26202760
Hepatol Int. 2014 Oct;8(4):550-9. doi: 10.1007/s12072-014-9572-2. Epub 2014 Sep 26.

BACKGROUND: The life cycle of hepatitis C virus (HCV) is tightly associated with host lipoprotein metabolic pathways. Apolipoprotein is present on the outer surface of lipoprotein particles and plays an important role in lipoprotein metabolism. We aimed to elucidate...

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Seki N, Sugita T, Aida Y, et al. Assessment of the features of serum apolipoprotein profiles in chronic HCV infection: difference between HCV genotypes 1b and 2. Hepatol Int. 2014;8(4):550-9doi: 10.1007/s12072-014-9572-2.
Seki, N., Sugita, T., Aida, Y., Itagaki, M., Ishiguro, H., Sutoh, S., Abe, H., Tsubota, A., Matsushima, M., & Aizawa, Y. (2014). Assessment of the features of serum apolipoprotein profiles in chronic HCV infection: difference between HCV genotypes 1b and 2. Hepatology international, 8(4), 550-9. https://doi.org/10.1007/s12072-014-9572-2
Seki, Nobuyoshi, et al. "Assessment of the features of serum apolipoprotein profiles in chronic HCV infection: difference between HCV genotypes 1b and 2." Hepatology international vol. 8,4 (2014): 550-9. doi: https://doi.org/10.1007/s12072-014-9572-2
Seki N, Sugita T, Aida Y, Itagaki M, Ishiguro H, Sutoh S, Abe H, Tsubota A, Matsushima M, Aizawa Y. Assessment of the features of serum apolipoprotein profiles in chronic HCV infection: difference between HCV genotypes 1b and 2. Hepatol Int. 2014 Oct;8(4):550-9. doi: 10.1007/s12072-014-9572-2. Epub 2014 Sep 26. PMID: 26202760.
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17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice.

Physiological reports

Yatomi M, Hisada T, Ishizuka T, Koga Y, Ono A, Kamide Y, Seki K, Aoki-Saito H, Tsurumaki H, Sunaga N, Kaira K, Dobashi K, Yamada M, Okajima F.
PMID: 26660549
Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12628.

Idiopathic pulmonary fibrosis (IPF) is a destructive inflammatory disease with limited therapeutic options. Inflammation plays an integral role in the development of pulmonary fibrosis. Unresolved inflammatory responses can lead to substantial tissue injury, chronic inflammation, and fibrosis. The resolvins...

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Yatomi M, Hisada T, Ishizuka T, et al. 17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice. Physiol Rep. 2015;3(12)doi: 10.14814/phy2.12628.
Yatomi, M., Hisada, T., Ishizuka, T., Koga, Y., Ono, A., Kamide, Y., Seki, K., Aoki-Saito, H., Tsurumaki, H., Sunaga, N., Kaira, K., Dobashi, K., Yamada, M., & Okajima, F. (2015). 17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice. Physiological reports, 3(12), . https://doi.org/10.14814/phy2.12628
Yatomi, Masakiyo, et al. "17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice." Physiological reports vol. 3,12 (2015). doi: https://doi.org/10.14814/phy2.12628
Yatomi M, Hisada T, Ishizuka T, Koga Y, Ono A, Kamide Y, Seki K, Aoki-Saito H, Tsurumaki H, Sunaga N, Kaira K, Dobashi K, Yamada M, Okajima F. 17(R)-resolvin D1 ameliorates bleomycin-induced pulmonary fibrosis in mice. Physiol Rep. 2015 Dec;3(12). doi: 10.14814/phy2.12628. PMID: 26660549; PMCID: PMC4760456.
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Functional Changes in the Care-needing Elderly after Surface Electrical Stimulation to the Abdomen.

Journal of the Japanese Physical Therapy Association = Rigaku ryoho

Miura M, Seki K, Ito O, Handa Y, Kohzuki M.
PMID: 25792897
J Jpn Phys Ther Assoc. 2012;15(1):15-20. doi: 10.1298/jjpta.Vol15_003.

BACKGROUND: Strength of the trunk muscles is a key component of motor control, but it declines easily with the process of aging and/or disuse.OBJECTIVES: To investigate the effects of surface electrical stimulation (ES) to the abdominal muscles and the...

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Miura M, Seki K, Ito O, et al. Functional Changes in the Care-needing Elderly after Surface Electrical Stimulation to the Abdomen. J Jpn Phys Ther Assoc. 2012;15(1):15-20doi: 10.1298/jjpta.Vol15_003.
Miura, M., Seki, K., Ito, O., Handa, Y., & Kohzuki, M. (2012). Functional Changes in the Care-needing Elderly after Surface Electrical Stimulation to the Abdomen. Journal of the Japanese Physical Therapy Association = Rigaku ryoho, 15(1), 15-20. https://doi.org/10.1298/jjpta.Vol15_003
Miura, Misa, et al. "Functional Changes in the Care-needing Elderly after Surface Electrical Stimulation to the Abdomen." Journal of the Japanese Physical Therapy Association = Rigaku ryoho vol. 15,1 (2012): 15-20. doi: https://doi.org/10.1298/jjpta.Vol15_003
Miura M, Seki K, Ito O, Handa Y, Kohzuki M. Functional Changes in the Care-needing Elderly after Surface Electrical Stimulation to the Abdomen. J Jpn Phys Ther Assoc. 2012;15(1):15-20. doi: 10.1298/jjpta.Vol15_003. PMID: 25792897; PMCID: PMC4316540.
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Persistent hyperglycemia modulates gut immune function and microbiota in rats.

Journal of intensive care

Mori K, Suzuki T, Igarashi T, Inoue K, Asahara T, Nomoto K, Seki H, Yamada T, Minamishima S, Kosugi S, Katori N, Morisaki H.
PMID: 26207186
J Intensive Care. 2015 Jul 23;3(1):34. doi: 10.1186/s40560-015-0101-8. eCollection 2015.

BACKGROUND: Since hyperglycemia-induced cellular dysfunction could be associated with alterations of the immune system, we tested the hypothesis that hyperglycemia augments the aberrant immune responses such as inflammation and differentiation of CD4(+) T lymphocytes in the mesenteric lymph nodes...

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Mori K, Suzuki T, Igarashi T, et al. Persistent hyperglycemia modulates gut immune function and microbiota in rats. J Intensive Care. 2015;3(1):34doi: 10.1186/s40560-015-0101-8.
Mori, K., Suzuki, T., Igarashi, T., Inoue, K., Asahara, T., Nomoto, K., Seki, H., Yamada, T., Minamishima, S., Kosugi, S., Katori, N., & Morisaki, H. (2015). Persistent hyperglycemia modulates gut immune function and microbiota in rats. Journal of intensive care, 3(1), 34. https://doi.org/10.1186/s40560-015-0101-8
Mori, Katsuya, et al. "Persistent hyperglycemia modulates gut immune function and microbiota in rats." Journal of intensive care vol. 3,1 (2015): 34. doi: https://doi.org/10.1186/s40560-015-0101-8
Mori K, Suzuki T, Igarashi T, Inoue K, Asahara T, Nomoto K, Seki H, Yamada T, Minamishima S, Kosugi S, Katori N, Morisaki H. Persistent hyperglycemia modulates gut immune function and microbiota in rats. J Intensive Care. 2015 Jul 23;3(1):34. doi: 10.1186/s40560-015-0101-8. eCollection 2015. PMID: 26207186; PMCID: PMC4511975.
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Traumatic sinus of valsalva aneurysm repaired by partial david operation.

The Thoracic and cardiovascular surgeon reports

Seki H, Borger M, Mohr F, Misfeld M.
PMID: 25798351
Thorac Cardiovasc Surg Rep. 2014 Dec;3(1):13-5. doi: 10.1055/s-0034-1383431. Epub 2014 Jul 16.

We report a case of chronic type A dissection and aneurysm of the sinus of Valsalva after a motor bike accident which remained undetected for 20 years. It was successfully treated with a modified valve-preserving technique. The noncoronary sinus...

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Seki H, Borger M, Mohr F, et al. Traumatic sinus of valsalva aneurysm repaired by partial david operation. Thorac Cardiovasc Surg Rep. 2014;3(1):13-5doi: 10.1055/s-0034-1383431.
Seki, H., Borger, M., Mohr, F., & Misfeld, M. (2014). Traumatic sinus of valsalva aneurysm repaired by partial david operation. The Thoracic and cardiovascular surgeon reports, 3(1), 13-5. https://doi.org/10.1055/s-0034-1383431
Seki, Hiroshi, et al. "Traumatic sinus of valsalva aneurysm repaired by partial david operation." The Thoracic and cardiovascular surgeon reports vol. 3,1 (2014): 13-5. doi: https://doi.org/10.1055/s-0034-1383431
Seki H, Borger M, Mohr F, Misfeld M. Traumatic sinus of valsalva aneurysm repaired by partial david operation. Thorac Cardiovasc Surg Rep. 2014 Dec;3(1):13-5. doi: 10.1055/s-0034-1383431. Epub 2014 Jul 16. PMID: 25798351; PMCID: PMC4360744.
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Mismatched changes of the photoluminescence and crystalline structure of a mechanochromic gold(I) isocyanide complex.

Chemical communications (Cambridge, England)

Seki T, Sakurada K, Ito H.
PMID: 26242610
Chem Commun (Camb). 2015 Sep 21;51(73):13933-6. doi: 10.1039/c5cc04609b.

The mechanochromic compound 1 forms green-emitting crystals (1g) and blue-emitting crystals (1b) that exhibit two intriguing responses to mechanical stimulation. Upon mechanical stimulation, the emission color of 1g does not change but its crystalline structure does. Conversely, 1b shows...

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Seki T, Sakurada K, Ito H. Mismatched changes of the photoluminescence and crystalline structure of a mechanochromic gold(I) isocyanide complex. Chem Commun (Camb). 2015;51(73):13933-6doi: 10.1039/c5cc04609b.
Seki, T., Sakurada, K., & Ito, H. (2015). Mismatched changes of the photoluminescence and crystalline structure of a mechanochromic gold(I) isocyanide complex. Chemical communications (Cambridge, England), 51(73), 13933-6. https://doi.org/10.1039/c5cc04609b
Seki, Tomohiro, et al. "Mismatched changes of the photoluminescence and crystalline structure of a mechanochromic gold(I) isocyanide complex." Chemical communications (Cambridge, England) vol. 51,73 (2015): 13933-6. doi: https://doi.org/10.1039/c5cc04609b
Seki T, Sakurada K, Ito H. Mismatched changes of the photoluminescence and crystalline structure of a mechanochromic gold(I) isocyanide complex. Chem Commun (Camb). 2015 Sep 21;51(73):13933-6. doi: 10.1039/c5cc04609b. PMID: 26242610.
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PRDM14 Is a Unique Epigenetic Regulator Stabilizing Transcriptional Networks for Pluripotency.

Frontiers in cell and developmental biology

Seki Y.
PMID: 29487849
Front Cell Dev Biol. 2018 Feb 13;6:12. doi: 10.3389/fcell.2018.00012. eCollection 2018.

PR-domain containing protein 14 (PRDM14) is a site-specific DNA-binding protein and is required for establishment of pluripotency in embryonic stem cells (ESCs) and primordial germ cells (PGCs) in mice. DNA methylation status is regulated by the balance between

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Seki Y. PRDM14 Is a Unique Epigenetic Regulator Stabilizing Transcriptional Networks for Pluripotency. Front Cell Dev Biol. 2018;6:12doi: 10.3389/fcell.2018.00012.
Seki, Y. (2018). PRDM14 Is a Unique Epigenetic Regulator Stabilizing Transcriptional Networks for Pluripotency. Frontiers in cell and developmental biology, 612. https://doi.org/10.3389/fcell.2018.00012
Seki, Yoshiyuki. "PRDM14 Is a Unique Epigenetic Regulator Stabilizing Transcriptional Networks for Pluripotency." Frontiers in cell and developmental biology vol. 6 (2018): 12. doi: https://doi.org/10.3389/fcell.2018.00012
Seki Y. PRDM14 Is a Unique Epigenetic Regulator Stabilizing Transcriptional Networks for Pluripotency. Front Cell Dev Biol. 2018 Feb 13;6:12. doi: 10.3389/fcell.2018.00012. eCollection 2018. PMID: 29487849; PMCID: PMC5816753.
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Showing 13 to 24 of 2349 entries