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Genome-wide association study and functional validation implicates JADE1 in tauopathy.

Acta neuropathologica

Farrell K, Kim S, Han N, Iida MA, Gonzalez EM, Otero-Garcia M, Walker JM, Richardson TE, Renton AE, Andrews SJ, Fulton-Howard B, Humphrey J, Vialle RA, Bowles KR, de Paiva Lopes K, Whitney K, Dangoor DK, Walsh H, Marcora E, Hefti MM, Casella A, Sissoko CT, Kapoor M, Novikova G, Udine E, Wong G, Tang W, Bhangale T, Hunkapiller J, Ayalon G, Graham RR, Cherry JD, Cortes EP, Borukov VY, McKee AC, Stein TD, Vonsattel JP, Teich AF, Gearing M, Glass J, Troncoso JC, Frosch MP, Hyman BT, Dickson DW, Murray ME, Attems J, Flanagan ME, Mao Q, Mesulam MM, Weintraub S, Woltjer RL, Pham T, Kofler J, Schneider JA, Yu L, Purohit DP, Haroutunian V, Hof PR, Gandy S, Sano M, Beach TG, Poon W, Kawas CH, Corrada MM, Rissman RA, Metcalf J, Shuldberg S, Salehi B, Nelson PT, Trojanowski JQ, Lee EB, Wolk DA, McMillan CT, Keene CD, Latimer CS, Montine TJ, Kovacs GG, Lutz MI, Fischer P, Perrin RJ, Cairns NJ, Franklin EE, Cohen HT, Raj T, Cobos I, Frost B, Goate A, White Iii CL, Crary JF.
PMID: 34719765
Acta Neuropathol. 2022 Jan;143(1):33-53. doi: 10.1007/s00401-021-02379-z. Epub 2021 Nov 01.

Primary age-related tauopathy (PART) is a neurodegenerative pathology with features distinct from but also overlapping with Alzheimer disease (AD). While both exhibit Alzheimer-type temporal lobe neurofibrillary degeneration alongside amnestic cognitive impairment, PART develops independently of amyloid-β (Aβ) plaques. The...

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Farrell K, Kim S, Han N, et al. Genome-wide association study and functional validation implicates JADE1 in tauopathy. Acta Neuropathol. 2021;143(1):33-53doi: 10.1007/s00401-021-02379-z.
Farrell, K., Kim, S., Han, N., Iida, M. A., Gonzalez, E. M., Otero-Garcia, M., Walker, J. M., Richardson, T. E., Renton, A. E., Andrews, S. J., Fulton-Howard, B., Humphrey, J., Vialle, R. A., Bowles, K. R., de Paiva Lopes, K., Whitney, K., Dangoor, D. K., Walsh, H., Marcora, E., Hefti, M. M., Casella, A., Sissoko, C. T., Kapoor, M., Novikova, G., Udine, E., Wong, G., Tang, W., Bhangale, T., Hunkapiller, J., Ayalon, G., Graham, R. R., Cherry, J. D., Cortes, E. P., Borukov, V. Y., McKee, A. C., Stein, T. D., Vonsattel, J. P., Teich, A. F., Gearing, M., Glass, J., Troncoso, J. C., Frosch, M. P., Hyman, B. T., Dickson, D. W., Murray, M. E., Attems, J., Flanagan, M. E., Mao, Q., Mesulam, M. M., Weintraub, S., Woltjer, R. L., Pham, T., Kofler, J., Schneider, J. A., Yu, L., Purohit, D. P., Haroutunian, V., Hof, P. R., Gandy, S., Sano, M., Beach, T. G., Poon, W., Kawas, C. H., Corrada, M. M., Rissman, R. A., Metcalf, J., Shuldberg, S., Salehi, B., Nelson, P. T., Trojanowski, J. Q., Lee, E. B., Wolk, D. A., McMillan, C. T., Keene, C. D., Latimer, C. S., Montine, T. J., Kovacs, G. G., Lutz, M. I., Fischer, P., Perrin, R. J., Cairns, N. J., Franklin, E. E., Cohen, H. T., Raj, T., Cobos, I., Frost, B., Goate, A., White Iii, C. L., & Crary, J. F. (2022). Genome-wide association study and functional validation implicates JADE1 in tauopathy. Acta neuropathologica, 143(1), 33-53. https://doi.org/10.1007/s00401-021-02379-z
Farrell, Kurt, et al. "Genome-wide association study and functional validation implicates JADE1 in tauopathy." Acta neuropathologica vol. 143,1 (2022): 33-53. doi: https://doi.org/10.1007/s00401-021-02379-z
Farrell K, Kim S, Han N, Iida MA, Gonzalez EM, Otero-Garcia M, Walker JM, Richardson TE, Renton AE, Andrews SJ, Fulton-Howard B, Humphrey J, Vialle RA, Bowles KR, de Paiva Lopes K, Whitney K, Dangoor DK, Walsh H, Marcora E, Hefti MM, Casella A, Sissoko CT, Kapoor M, Novikova G, Udine E, Wong G, Tang W, Bhangale T, Hunkapiller J, Ayalon G, Graham RR, Cherry JD, Cortes EP, Borukov VY, McKee AC, Stein TD, Vonsattel JP, Teich AF, Gearing M, Glass J, Troncoso JC, Frosch MP, Hyman BT, Dickson DW, Murray ME, Attems J, Flanagan ME, Mao Q, Mesulam MM, Weintraub S, Woltjer RL, Pham T, Kofler J, Schneider JA, Yu L, Purohit DP, Haroutunian V, Hof PR, Gandy S, Sano M, Beach TG, Poon W, Kawas CH, Corrada MM, Rissman RA, Metcalf J, Shuldberg S, Salehi B, Nelson PT, Trojanowski JQ, Lee EB, Wolk DA, McMillan CT, Keene CD, Latimer CS, Montine TJ, Kovacs GG, Lutz MI, Fischer P, Perrin RJ, Cairns NJ, Franklin EE, Cohen HT, Raj T, Cobos I, Frost B, Goate A, White Iii CL, Crary JF. Genome-wide association study and functional validation implicates JADE1 in tauopathy. Acta Neuropathol. 2022 Jan;143(1):33-53. doi: 10.1007/s00401-021-02379-z. Epub 2021 Nov 01. PMID: 34719765.
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Ex vivo MRI and histopathology detect novel iron-rich cortical inflammation in frontotemporal lobar degeneration with tau versus TDP-43 pathology.

NeuroImage. Clinical

Tisdall MD, Ohm DT, Lobrovich R, Das SR, Mizsei G, Prabhakaran K, Ittyerah R, Lim S, McMillan CT, Wolk DA, Gee J, Trojanowski JQ, Lee EB, Detre JA, Yushkevich P, Grossman M, Irwin DJ.
PMID: 34952351
Neuroimage Clin. 2021 Dec 14;33:102913. doi: 10.1016/j.nicl.2021.102913. Epub 2021 Dec 14.

Frontotemporal lobar degeneration (FTLD) is a heterogeneous spectrum of age-associated neurodegenerative diseases that include two main pathologic categories of tau (FTLD-Tau) and TDP-43 (FTLD-TDP) proteinopathies. These distinct proteinopathies are often clinically indistinguishable during life, posing a major obstacle for...

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Tisdall MD, Ohm DT, Lobrovich R, et al. Ex vivo MRI and histopathology detect novel iron-rich cortical inflammation in frontotemporal lobar degeneration with tau versus TDP-43 pathology. Neuroimage Clin. 2021;33:102913doi: 10.1016/j.nicl.2021.102913.
Tisdall, M. D., Ohm, D. T., Lobrovich, R., Das, S. R., Mizsei, G., Prabhakaran, K., Ittyerah, R., Lim, S., McMillan, C. T., Wolk, D. A., Gee, J., Trojanowski, J. Q., Lee, E. B., Detre, J. A., Yushkevich, P., Grossman, M., & Irwin, D. J. (2021). Ex vivo MRI and histopathology detect novel iron-rich cortical inflammation in frontotemporal lobar degeneration with tau versus TDP-43 pathology. NeuroImage. Clinical, 33102913. https://doi.org/10.1016/j.nicl.2021.102913
Tisdall, M Dylan, et al. "Ex vivo MRI and histopathology detect novel iron-rich cortical inflammation in frontotemporal lobar degeneration with tau versus TDP-43 pathology." NeuroImage. Clinical vol. 33 (2021): 102913. doi: https://doi.org/10.1016/j.nicl.2021.102913
Tisdall MD, Ohm DT, Lobrovich R, Das SR, Mizsei G, Prabhakaran K, Ittyerah R, Lim S, McMillan CT, Wolk DA, Gee J, Trojanowski JQ, Lee EB, Detre JA, Yushkevich P, Grossman M, Irwin DJ. Ex vivo MRI and histopathology detect novel iron-rich cortical inflammation in frontotemporal lobar degeneration with tau versus TDP-43 pathology. Neuroimage Clin. 2021 Dec 14;33:102913. doi: 10.1016/j.nicl.2021.102913. Epub 2021 Dec 14. PMID: 34952351; PMCID: PMC8715243.
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Alpha-synuclein from patient Lewy bodies exhibits distinct pathological activity that can be propagated in vitro.

Acta neuropathologica communications

Marotta NP, Ara J, Uemura N, Lougee MG, Meymand ES, Zhang B, Petersson EJ, Trojanowski JQ, Lee VM.
PMID: 34819159
Acta Neuropathol Commun. 2021 Nov 24;9(1):188. doi: 10.1186/s40478-021-01288-2.

Lewy bodies (LBs) are complex, intracellular inclusions that are common pathological features of many neurodegenerative diseases. They consist largely of aggregated forms of the protein alpha-Synuclein (α-Syn), which misfolds to give rise to beta-sheet rich amyloid fibrils. The aggregation...

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Marotta NP, Ara J, Uemura N, et al. Alpha-synuclein from patient Lewy bodies exhibits distinct pathological activity that can be propagated in vitro. Acta Neuropathol Commun. 2021;9(1):188doi: 10.1186/s40478-021-01288-2.
Marotta, N. P., Ara, J., Uemura, N., Lougee, M. G., Meymand, E. S., Zhang, B., Petersson, E. J., Trojanowski, J. Q., & Lee, V. M. (2021). Alpha-synuclein from patient Lewy bodies exhibits distinct pathological activity that can be propagated in vitro. Acta neuropathologica communications, 9(1), 188. https://doi.org/10.1186/s40478-021-01288-2
Marotta, Nicholas P, et al. "Alpha-synuclein from patient Lewy bodies exhibits distinct pathological activity that can be propagated in vitro." Acta neuropathologica communications vol. 9,1 (2021): 188. doi: https://doi.org/10.1186/s40478-021-01288-2
Marotta NP, Ara J, Uemura N, Lougee MG, Meymand ES, Zhang B, Petersson EJ, Trojanowski JQ, Lee VM. Alpha-synuclein from patient Lewy bodies exhibits distinct pathological activity that can be propagated in vitro. Acta Neuropathol Commun. 2021 Nov 24;9(1):188. doi: 10.1186/s40478-021-01288-2. PMID: 34819159; PMCID: PMC8611971.
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Distinct microglial response against Alzheimer's amyloid and tau pathologies characterized by P2Y12 receptor.

Brain communications

Maeda J, Minamihisamatsu T, Shimojo M, Zhou X, Ono M, Matsuba Y, Ji B, Ishii H, Ogawa M, Akatsu H, Kaneda D, Hashizume Y, Robinson JL, Lee VM, Saito T, Saido TC, Trojanowski JQ, Zhang MR, Suhara T, Higuchi M, Sahara N.
PMID: 33644757
Brain Commun. 2021 Jan 29;3(1):fcab011. doi: 10.1093/braincomms/fcab011. eCollection 2021.

Microglia are the resident phagocytes of the central nervous system, and microglial activation is considered to play an important role in the pathogenesis of neurodegenerative diseases. Recent studies with single-cell RNA analysis of CNS cells in Alzheimer's disease and...

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Maeda J, Minamihisamatsu T, Shimojo M, et al. Distinct microglial response against Alzheimer's amyloid and tau pathologies characterized by P2Y12 receptor. Brain Commun. 2021;3(1):fcab011doi: 10.1093/braincomms/fcab011.
Maeda, J., Minamihisamatsu, T., Shimojo, M., Zhou, X., Ono, M., Matsuba, Y., Ji, B., Ishii, H., Ogawa, M., Akatsu, H., Kaneda, D., Hashizume, Y., Robinson, J. L., Lee, V. M., Saito, T., Saido, T. C., Trojanowski, J. Q., Zhang, M. R., Suhara, T., Higuchi, M., & Sahara, N. (2021). Distinct microglial response against Alzheimer's amyloid and tau pathologies characterized by P2Y12 receptor. Brain communications, 3(1), fcab011. https://doi.org/10.1093/braincomms/fcab011
Maeda, Jun, et al. "Distinct microglial response against Alzheimer's amyloid and tau pathologies characterized by P2Y12 receptor." Brain communications vol. 3,1 (2021): fcab011. doi: https://doi.org/10.1093/braincomms/fcab011
Maeda J, Minamihisamatsu T, Shimojo M, Zhou X, Ono M, Matsuba Y, Ji B, Ishii H, Ogawa M, Akatsu H, Kaneda D, Hashizume Y, Robinson JL, Lee VM, Saito T, Saido TC, Trojanowski JQ, Zhang MR, Suhara T, Higuchi M, Sahara N. Distinct microglial response against Alzheimer's amyloid and tau pathologies characterized by P2Y12 receptor. Brain Commun. 2021 Jan 29;3(1):fcab011. doi: 10.1093/braincomms/fcab011. eCollection 2021. PMID: 33644757; PMCID: PMC7901060.
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Three-dimensional mapping of neurofibrillary tangle burden in the human medial temporal lobe.

Brain : a journal of neurology

Yushkevich PA, Muñoz López M, Iñiguez de Onzoño Martin MM, Ittyerah R, Lim S, Ravikumar S, Bedard ML, Pickup S, Liu W, Wang J, Hung LY, Lasserve J, Vergnet N, Xie L, Dong M, Cui S, McCollum L, Robinson JL, Schuck T, de Flores R, Grossman M, Tisdall MD, Prabhakaran K, Mizsei G, Das SR, Artacho-Pérula E, Arroyo Jiménez MDM, Marcos Raba MP, Molina Romero FJ, Cebada Sánchez S, Delgado González JC, de la Rosa-Prieto C, Córcoles Parada M, Lee EB, Trojanowski JQ, Ohm DT, Wisse LEM, Wolk DA, Irwin DJ, Insausti R.
PMID: 34259858
Brain. 2021 Oct 22;144(9):2784-2797. doi: 10.1093/brain/awab262.

Tau protein neurofibrillary tangles are closely linked to neuronal/synaptic loss and cognitive decline in Alzheimer's disease and related dementias. Our knowledge of the pattern of neurofibrillary tangle progression in the human brain, critical to the development of imaging biomarkers...

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Yushkevich PA, Muñoz López M, Iñiguez de Onzoño Martin MM, et al. Three-dimensional mapping of neurofibrillary tangle burden in the human medial temporal lobe. Brain. 2021;144(9):2784-2797doi: 10.1093/brain/awab262.
Yushkevich, P. A., Muñoz López, M., Iñiguez de Onzoño Martin, M. M., Ittyerah, R., Lim, S., Ravikumar, S., Bedard, M. L., Pickup, S., Liu, W., Wang, J., Hung, L. Y., Lasserve, J., Vergnet, N., Xie, L., Dong, M., Cui, S., McCollum, L., Robinson, J. L., Schuck, T., de Flores, R., Grossman, M., Tisdall, M. D., Prabhakaran, K., Mizsei, G., Das, S. R., Artacho-Pérula, E., Arroyo Jiménez, M. D. M., Marcos Raba, M. P., Molina Romero, F. J., Cebada Sánchez, S., Delgado González, J. C., de la Rosa-Prieto, C., Córcoles Parada, M., Lee, E. B., Trojanowski, J. Q., Ohm, D. T., Wisse, L. E. M., Wolk, D. A., Irwin, D. J., & Insausti, R. (2021). Three-dimensional mapping of neurofibrillary tangle burden in the human medial temporal lobe. Brain : a journal of neurology, 144(9), 2784-2797. https://doi.org/10.1093/brain/awab262
Yushkevich, Paul A, et al. "Three-dimensional mapping of neurofibrillary tangle burden in the human medial temporal lobe." Brain : a journal of neurology vol. 144,9 (2021): 2784-2797. doi: https://doi.org/10.1093/brain/awab262
Yushkevich PA, Muñoz López M, Iñiguez de Onzoño Martin MM, Ittyerah R, Lim S, Ravikumar S, Bedard ML, Pickup S, Liu W, Wang J, Hung LY, Lasserve J, Vergnet N, Xie L, Dong M, Cui S, McCollum L, Robinson JL, Schuck T, de Flores R, Grossman M, Tisdall MD, Prabhakaran K, Mizsei G, Das SR, Artacho-Pérula E, Arroyo Jiménez MDM, Marcos Raba MP, Molina Romero FJ, Cebada Sánchez S, Delgado González JC, de la Rosa-Prieto C, Córcoles Parada M, Lee EB, Trojanowski JQ, Ohm DT, Wisse LEM, Wolk DA, Irwin DJ, Insausti R. Three-dimensional mapping of neurofibrillary tangle burden in the human medial temporal lobe. Brain. 2021 Oct 22;144(9):2784-2797. doi: 10.1093/brain/awab262. PMID: 34259858.
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Computational modeling of tau pathology spread reveals patterns of regional vulnerability and the impact of a genetic risk factor.

Science advances

Cornblath EJ, Li HL, Changolkar L, Zhang B, Brown HJ, Gathagan RJ, Olufemi MF, Trojanowski JQ, Bassett DS, Lee VMY, Henderson MX.
PMID: 34108219
Sci Adv. 2021 Jun 09;7(24). doi: 10.1126/sciadv.abg6677. Print 2021 Jun.

Neuropathological staging studies have suggested that tau pathology spreads through the brain in Alzheimer's disease (AD) and other tauopathies, but it is unclear how neuroanatomical connections, spatial proximity, and regional vulnerability contribute. In this study, we seed tau pathology...

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Cornblath EJ, Li HL, Changolkar L, et al. Computational modeling of tau pathology spread reveals patterns of regional vulnerability and the impact of a genetic risk factor. Sci Adv. 2021;7(24)doi: 10.1126/sciadv.abg6677.
Cornblath, E. J., Li, H. L., Changolkar, L., Zhang, B., Brown, H. J., Gathagan, R. J., Olufemi, M. F., Trojanowski, J. Q., Bassett, D. S., Lee, V. M. Y., & Henderson, M. X. (2021). Computational modeling of tau pathology spread reveals patterns of regional vulnerability and the impact of a genetic risk factor. Science advances, 7(24), . https://doi.org/10.1126/sciadv.abg6677
Cornblath, Eli J, et al. "Computational modeling of tau pathology spread reveals patterns of regional vulnerability and the impact of a genetic risk factor." Science advances vol. 7,24 (2021). doi: https://doi.org/10.1126/sciadv.abg6677
Cornblath EJ, Li HL, Changolkar L, Zhang B, Brown HJ, Gathagan RJ, Olufemi MF, Trojanowski JQ, Bassett DS, Lee VMY, Henderson MX. Computational modeling of tau pathology spread reveals patterns of regional vulnerability and the impact of a genetic risk factor. Sci Adv. 2021 Jun 09;7(24). doi: 10.1126/sciadv.abg6677. Print 2021 Jun. PMID: 34108219; PMCID: PMC8189700.
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Parameter Identification of Cutting Forces in Crankshaft Grinding Using Artificial Neural Networks.

Materials (Basel, Switzerland)

Pavlenko I, Saga M, Kuric I, Kotliar A, Basova Y, Trojanowska J, Ivanov V.
PMID: 33255880
Materials (Basel). 2020 Nov 26;13(23). doi: 10.3390/ma13235357.

The intensifying of the manufacturing process and increasing the efficiency of production planning of precise and non-rigid parts, mainly crankshafts, are the first-priority task in modern manufacturing. The use of various methods for controlling the cutting force under cylindrical...

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Pavlenko I, Saga M, Kuric I, et al. Parameter Identification of Cutting Forces in Crankshaft Grinding Using Artificial Neural Networks. Materials (Basel). 2020;13(23)doi: 10.3390/ma13235357.
Pavlenko, I., Saga, M., Kuric, I., Kotliar, A., Basova, Y., Trojanowska, J., & Ivanov, V. (2020). Parameter Identification of Cutting Forces in Crankshaft Grinding Using Artificial Neural Networks. Materials (Basel, Switzerland), 13(23), . https://doi.org/10.3390/ma13235357
Pavlenko, Ivan, et al. "Parameter Identification of Cutting Forces in Crankshaft Grinding Using Artificial Neural Networks." Materials (Basel, Switzerland) vol. 13,23 (2020). doi: https://doi.org/10.3390/ma13235357
Pavlenko I, Saga M, Kuric I, Kotliar A, Basova Y, Trojanowska J, Ivanov V. Parameter Identification of Cutting Forces in Crankshaft Grinding Using Artificial Neural Networks. Materials (Basel). 2020 Nov 26;13(23). doi: 10.3390/ma13235357. PMID: 33255880; PMCID: PMC7728348.
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Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease.

Movement disorders : official journal of the Movement Disorder Society

Aamodt WW, Waligorska T, Shen J, Tropea TF, Siderowf A, Weintraub D, Grossman M, Irwin D, Wolk DA, Xie SX, Trojanowski JQ, Shaw LM, Chen-Plotkin AS.
PMID: 34480363
Mov Disord. 2021 Dec;36(12):2945-2950. doi: 10.1002/mds.28779. Epub 2021 Sep 04.

BACKGROUND: Neurofilament light chain protein (NfL) is a promising biomarker of neurodegeneration.OBJECTIVES: To determine whether plasma and CSF NfL (1) associate with motor or cognitive status in Parkinson's disease (PD) and (2) predict future motor or cognitive decline in...

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Aamodt WW, Waligorska T, Shen J, et al. Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease. Mov Disord. 2021;36(12):2945-2950doi: 10.1002/mds.28779.
Aamodt, W. W., Waligorska, T., Shen, J., Tropea, T. F., Siderowf, A., Weintraub, D., Grossman, M., Irwin, D., Wolk, D. A., Xie, S. X., Trojanowski, J. Q., Shaw, L. M., & Chen-Plotkin, A. S. (2021). Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease. Movement disorders : official journal of the Movement Disorder Society, 36(12), 2945-2950. https://doi.org/10.1002/mds.28779
Aamodt, Whitley W, et al. "Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease." Movement disorders : official journal of the Movement Disorder Society vol. 36,12 (2021): 2945-2950. doi: https://doi.org/10.1002/mds.28779
Aamodt WW, Waligorska T, Shen J, Tropea TF, Siderowf A, Weintraub D, Grossman M, Irwin D, Wolk DA, Xie SX, Trojanowski JQ, Shaw LM, Chen-Plotkin AS. Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease. Mov Disord. 2021 Dec;36(12):2945-2950. doi: 10.1002/mds.28779. Epub 2021 Sep 04. PMID: 34480363.
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Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease.

Movement disorders : official journal of the Movement Disorder Society

Aamodt WW, Waligorska T, Shen J, Tropea TF, Siderowf A, Weintraub D, Grossman M, Irwin D, Wolk DA, Xie SX, Trojanowski JQ, Shaw LM, Chen-Plotkin AS.
PMID: 34480363
Mov Disord. 2021 Sep 04; doi: 10.1002/mds.28779. Epub 2021 Sep 04.

BACKGROUND: Neurofilament light chain protein (NfL) is a promising biomarker of neurodegeneration.OBJECTIVES: To determine whether plasma and CSF NfL (1) associate with motor or cognitive status in Parkinson's disease (PD) and (2) predict future motor or cognitive decline in...

Cite
Aamodt WW, Waligorska T, Shen J, et al. Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease. Mov Disord. 2021;doi: 10.1002/mds.28779.
Aamodt, W. W., Waligorska, T., Shen, J., Tropea, T. F., Siderowf, A., Weintraub, D., Grossman, M., Irwin, D., Wolk, D. A., Xie, S. X., Trojanowski, J. Q., Shaw, L. M., & Chen-Plotkin, A. S. (2021). Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease. Movement disorders : official journal of the Movement Disorder Society, . https://doi.org/10.1002/mds.28779
Aamodt, Whitley W, et al. "Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease." Movement disorders : official journal of the Movement Disorder Society vol. (2021). doi: https://doi.org/10.1002/mds.28779
Aamodt WW, Waligorska T, Shen J, Tropea TF, Siderowf A, Weintraub D, Grossman M, Irwin D, Wolk DA, Xie SX, Trojanowski JQ, Shaw LM, Chen-Plotkin AS. Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease. Mov Disord. 2021 Sep 04; doi: 10.1002/mds.28779. Epub 2021 Sep 04. PMID: 34480363.
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Klotho expression and nodal involvement as predictive factors for large cell lung carcinoma.

Archives of medical science : AMS

Brominska B, Gabryel P, Jarmołowska-Jurczyszyn D, Janicka-Jedyńska M, Kluk A, Trojanowski M, Brajer-Luftmann B, Woliński K, Czepczyński R, Gut P, Bromiński G, Majewski P, Dyszkiewicz W, Ruchała M.
PMID: 31360195
Arch Med Sci. 2019 Jul;15(4):1010-1016. doi: 10.5114/aoms.2018.75889. Epub 2018 Jun 01.

INTRODUCTION: Klotho has been recently described as a carcinogenesis suppressor. Large cell neuroendocrine lung carcinoma (LCNEC) is a rare, highly malignant neoplasm. In the light of increasing incidence of neuroendocrine tumours, biomarkers predicting survival are needed. We consider that...

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Brominska B, Gabryel P, Jarmołowska-Jurczyszyn D, et al. Klotho expression and nodal involvement as predictive factors for large cell lung carcinoma. Arch Med Sci. 2018;15(4):1010-1016doi: 10.5114/aoms.2018.75889.
Brominska, B., Gabryel, P., Jarmołowska-Jurczyszyn, D., Janicka-Jedyńska, M., Kluk, A., Trojanowski, M., Brajer-Luftmann, B., Woliński, K., Czepczyński, R., Gut, P., Bromiński, G., Majewski, P., Dyszkiewicz, W., & Ruchała, M. (2019). Klotho expression and nodal involvement as predictive factors for large cell lung carcinoma. Archives of medical science : AMS, 15(4), 1010-1016. https://doi.org/10.5114/aoms.2018.75889
Brominska, Barbara, et al. "Klotho expression and nodal involvement as predictive factors for large cell lung carcinoma." Archives of medical science : AMS vol. 15,4 (2019): 1010-1016. doi: https://doi.org/10.5114/aoms.2018.75889
Brominska B, Gabryel P, Jarmołowska-Jurczyszyn D, Janicka-Jedyńska M, Kluk A, Trojanowski M, Brajer-Luftmann B, Woliński K, Czepczyński R, Gut P, Bromiński G, Majewski P, Dyszkiewicz W, Ruchała M. Klotho expression and nodal involvement as predictive factors for large cell lung carcinoma. Arch Med Sci. 2019 Jul;15(4):1010-1016. doi: 10.5114/aoms.2018.75889. Epub 2018 Jun 01. PMID: 31360195; PMCID: PMC6657266.
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Co-occurrence of birds and bats in natural nest-holes.

The Ibis

Myczko Ł, Dylewski Ł, Sparks TH, Łochyński M, Tryjanowski P.
PMID: 32336773
Ibis (Lond 1859). 2017 Jan;159(1):235-237. doi: 10.1111/ibi.12434. Epub 2016 Dec 09.

No abstract available.

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Myczko Ł, Dylewski Ł, Sparks TH, et al. Co-occurrence of birds and bats in natural nest-holes. Ibis (Lond 1859). 2016;159(1):235-237doi: 10.1111/ibi.12434.
Myczko, �. �., Dylewski, �. �., Sparks, T. H., Łochyński, M., & Tryjanowski, P. (2017). Co-occurrence of birds and bats in natural nest-holes. The Ibis, 159(1), 235-237. https://doi.org/10.1111/ibi.12434
Myczko, Łukasz, et al. "Co-occurrence of birds and bats in natural nest-holes." The Ibis vol. 159,1 (2017): 235-237. doi: https://doi.org/10.1111/ibi.12434
Myczko Ł, Dylewski Ł, Sparks TH, Łochyński M, Tryjanowski P. Co-occurrence of birds and bats in natural nest-holes. Ibis (Lond 1859). 2017 Jan;159(1):235-237. doi: 10.1111/ibi.12434. Epub 2016 Dec 09. PMID: 32336773; PMCID: PMC7165987.
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Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease.

Movement disorders : official journal of the Movement Disorder Society

Aamodt WW, Waligorska T, Shen J, Tropea TF, Siderowf A, Weintraub D, Grossman M, Irwin D, Wolk DA, Xie SX, Trojanowski JQ, Shaw LM, Chen-Plotkin AS.
PMID: 34480363
Mov Disord. 2021 Dec;36(12):2945-2950. doi: 10.1002/mds.28779. Epub 2021 Sep 04.

BACKGROUND: Neurofilament light chain protein (NfL) is a promising biomarker of neurodegeneration.OBJECTIVES: To determine whether plasma and CSF NfL (1) associate with motor or cognitive status in Parkinson's disease (PD) and (2) predict future motor or cognitive decline in...

Cite
Aamodt WW, Waligorska T, Shen J, et al. Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease. Mov Disord. 2021;36(12):2945-2950doi: 10.1002/mds.28779.
Aamodt, W. W., Waligorska, T., Shen, J., Tropea, T. F., Siderowf, A., Weintraub, D., Grossman, M., Irwin, D., Wolk, D. A., Xie, S. X., Trojanowski, J. Q., Shaw, L. M., & Chen-Plotkin, A. S. (2021). Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease. Movement disorders : official journal of the Movement Disorder Society, 36(12), 2945-2950. https://doi.org/10.1002/mds.28779
Aamodt, Whitley W, et al. "Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease." Movement disorders : official journal of the Movement Disorder Society vol. 36,12 (2021): 2945-2950. doi: https://doi.org/10.1002/mds.28779
Aamodt WW, Waligorska T, Shen J, Tropea TF, Siderowf A, Weintraub D, Grossman M, Irwin D, Wolk DA, Xie SX, Trojanowski JQ, Shaw LM, Chen-Plotkin AS. Neurofilament Light Chain as a Biomarker for Cognitive Decline in Parkinson Disease. Mov Disord. 2021 Dec;36(12):2945-2950. doi: 10.1002/mds.28779. Epub 2021 Sep 04. PMID: 34480363; PMCID: PMC8688198.
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