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The trajectory of gray matter development in Broca's area is abnormal in people who stutter.

Frontiers in human neuroscience

Beal DS, Lerch JP, Cameron B, Henderson R, Gracco VL, De Nil LF.
PMID: 25784869
Front Hum Neurosci. 2015 Mar 03;9:89. doi: 10.3389/fnhum.2015.00089. eCollection 2015.

The acquisition and mastery of speech-motor control requires years of practice spanning the course of development. People who stutter often perform poorly on speech-motor tasks thereby calling into question their ability to establish the stable neural motor programs required...

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Beal DS, Lerch JP, Cameron B, et al. The trajectory of gray matter development in Broca's area is abnormal in people who stutter. Front Hum Neurosci. 2015;9:89doi: 10.3389/fnhum.2015.00089.
Beal, D. S., Lerch, J. P., Cameron, B., Henderson, R., Gracco, V. L., & De Nil, L. F. (2015). The trajectory of gray matter development in Broca's area is abnormal in people who stutter. Frontiers in human neuroscience, 989. https://doi.org/10.3389/fnhum.2015.00089
Beal, Deryk S, et al. "The trajectory of gray matter development in Broca's area is abnormal in people who stutter." Frontiers in human neuroscience vol. 9 (2015): 89. doi: https://doi.org/10.3389/fnhum.2015.00089
Beal DS, Lerch JP, Cameron B, Henderson R, Gracco VL, De Nil LF. The trajectory of gray matter development in Broca's area is abnormal in people who stutter. Front Hum Neurosci. 2015 Mar 03;9:89. doi: 10.3389/fnhum.2015.00089. eCollection 2015. PMID: 25784869; PMCID: PMC4347452.
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Is metabotropic glutamate receptor 5 upregulated in prefrontal cortex in fragile X syndrome?.

Molecular autism

Lohith TG, Osterweil EK, Fujita M, Jenko KJ, Bear MF, Innis RB.
PMID: 23706040
Mol Autism. 2013 May 24;4(1):15. doi: 10.1186/2040-2392-4-15.

BACKGROUND: Fragile X syndrome (FXS) is a common inherited form of intellectual disability caused by loss of function of the fragile X mental retardation protein. Recent animal studies suggest that upregulated downstream signaling by metabotropic glutamate receptor 5 (mGluR5)...

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Lohith TG, Osterweil EK, Fujita M, et al. Is metabotropic glutamate receptor 5 upregulated in prefrontal cortex in fragile X syndrome?. Mol Autism. 2013;4(1):15doi: 10.1186/2040-2392-4-15.
Lohith, T. G., Osterweil, E. K., Fujita, M., Jenko, K. J., Bear, M. F., & Innis, R. B. (2013). Is metabotropic glutamate receptor 5 upregulated in prefrontal cortex in fragile X syndrome?. Molecular autism, 4(1), 15. https://doi.org/10.1186/2040-2392-4-15
Lohith, Talakad G, et al. "Is metabotropic glutamate receptor 5 upregulated in prefrontal cortex in fragile X syndrome?." Molecular autism vol. 4,1 (2013): 15. doi: https://doi.org/10.1186/2040-2392-4-15
Lohith TG, Osterweil EK, Fujita M, Jenko KJ, Bear MF, Innis RB. Is metabotropic glutamate receptor 5 upregulated in prefrontal cortex in fragile X syndrome?. Mol Autism. 2013 May 24;4(1):15. doi: 10.1186/2040-2392-4-15. PMID: 23706040; PMCID: PMC3679853.
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Laparoscopic hepatectomy for hepatocellular carcinoma: are oncologic outcomes truly superior to an open approach?.

Hepatobiliary surgery and nutrition

Moris D, Burkhart RA, Beal EW, Pawlik TM.
PMID: 28653005
Hepatobiliary Surg Nutr. 2017 Jun;6(3):200-202. doi: 10.21037/hbsn.2017.03.09.

No abstract available.

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Moris D, Burkhart RA, Beal EW, et al. Laparoscopic hepatectomy for hepatocellular carcinoma: are oncologic outcomes truly superior to an open approach?. Hepatobiliary Surg Nutr. 2017;6(3):200-202doi: 10.21037/hbsn.2017.03.09.
Moris, D., Burkhart, R. A., Beal, E. W., & Pawlik, T. M. (2017). Laparoscopic hepatectomy for hepatocellular carcinoma: are oncologic outcomes truly superior to an open approach?. Hepatobiliary surgery and nutrition, 6(3), 200-202. https://doi.org/10.21037/hbsn.2017.03.09
Moris, Dimitrios, et al. "Laparoscopic hepatectomy for hepatocellular carcinoma: are oncologic outcomes truly superior to an open approach?." Hepatobiliary surgery and nutrition vol. 6,3 (2017): 200-202. doi: https://doi.org/10.21037/hbsn.2017.03.09
Moris D, Burkhart RA, Beal EW, Pawlik TM. Laparoscopic hepatectomy for hepatocellular carcinoma: are oncologic outcomes truly superior to an open approach?. Hepatobiliary Surg Nutr. 2017 Jun;6(3):200-202. doi: 10.21037/hbsn.2017.03.09. PMID: 28653005; PMCID: PMC5474454.
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Profile of collagen gene expression in the glenohumeral capsule of patients with traumatic anterior instability of the shoulder.

Revista brasileira de ortopedia

Belangero PS, Leal MF, de Castro Pochini A, Andreoli CV, Ejnisman B, Cohen M.
PMID: 26229875
Rev Bras Ortop. 2014 Oct 23;49(6):642-6. doi: 10.1016/j.rboe.2014.10.008. eCollection 2014.

OBJECTIVE: To evaluate the expression of the genes COL1A1, COL1A2, COL3A1 and COL5A1 in the glenohumeral capsule of patients with traumatic anterior instability of the shoulder.METHODS: Samples from the glenohumeral capsule of 18 patients with traumatic anterior instability of...

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Belangero PS, Leal MF, de Castro Pochini A, et al. Profile of collagen gene expression in the glenohumeral capsule of patients with traumatic anterior instability of the shoulder. Rev Bras Ortop. 2014;49(6):642-6doi: 10.1016/j.rboe.2014.10.008.
Belangero, P. S., Leal, M. F., de Castro Pochini, A., Andreoli, C. V., Ejnisman, B., & Cohen, M. (2014). Profile of collagen gene expression in the glenohumeral capsule of patients with traumatic anterior instability of the shoulder. Revista brasileira de ortopedia, 49(6), 642-6. https://doi.org/10.1016/j.rboe.2014.10.008
Belangero, Paulo Santoro, et al. "Profile of collagen gene expression in the glenohumeral capsule of patients with traumatic anterior instability of the shoulder." Revista brasileira de ortopedia vol. 49,6 (2014): 642-6. doi: https://doi.org/10.1016/j.rboe.2014.10.008
Belangero PS, Leal MF, de Castro Pochini A, Andreoli CV, Ejnisman B, Cohen M. Profile of collagen gene expression in the glenohumeral capsule of patients with traumatic anterior instability of the shoulder. Rev Bras Ortop. 2014 Oct 23;49(6):642-6. doi: 10.1016/j.rboe.2014.10.008. eCollection 2014. PMID: 26229875; PMCID: PMC4487492.
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Response:Model of Huntington's disease.

Science (New York, N.Y.)

Beal MF, Kowall NW, Swartz KJ, Ferrante RJ, Martin JB.
PMID: 17792612
Science. 1988 Jul 22;241(4864):475. doi: 10.1126/science.241.4864.475.

No abstract available.

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Beal MF, Kowall NW, Swartz KJ, et al. Response:Model of Huntington's disease. Science. 1988;241(4864):475doi: 10.1126/science.241.4864.475.
Beal, M. F., Kowall, N. W., Swartz, K. J., Ferrante, R. J., & Martin, J. B. (1988). Response:Model of Huntington's disease. Science (New York, N.Y.), 241(4864), 475. https://doi.org/10.1126/science.241.4864.475
Beal, M F, et al. "Response:Model of Huntington's disease." Science (New York, N.Y.) vol. 241,4864 (1988): 475. doi: https://doi.org/10.1126/science.241.4864.475
Beal MF, Kowall NW, Swartz KJ, Ferrante RJ, Martin JB. Response:Model of Huntington's disease. Science. 1988 Jul 22;241(4864):475. doi: 10.1126/science.241.4864.475. PMID: 17792612.
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Prognostic import of a large Q3 deflection: a mortality study.

Transactions of the Association of Life Insurance Medical Directors of America

BOLT W, BELL MF.
PMID: 14835766
Trans Assoc Life Insur Med Dir Am. 1950;34:87-98.

No abstract available.

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BOLT W, BELL MF. Prognostic import of a large Q3 deflection: a mortality study. Trans Assoc Life Insur Med Dir Am. 1950;34:87-98
BOLT, W., & BELL, M. F. (1950). Prognostic import of a large Q3 deflection: a mortality study. Transactions of the Association of Life Insurance Medical Directors of America, 3487-98.
BOLT, W, and BELL, M F. "Prognostic import of a large Q3 deflection: a mortality study." Transactions of the Association of Life Insurance Medical Directors of America vol. 34 (1950): 87-98.
BOLT W, BELL MF. Prognostic import of a large Q3 deflection: a mortality study. Trans Assoc Life Insur Med Dir Am. 1950;34:87-98. PMID: 14835766.
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Drug delivery to the cochlea after implantation: consideration of the risk factors.

Cochlear implants international

Garnham C, Reetz G, Jolly C, Miller J, Salt A, Beal F.
PMID: 18792345
Cochlear Implants Int. 2005 Sep;6:12-4. doi: 10.1179/cim.2005.6.Supplement-1.12.

No abstract available.

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Garnham C, Reetz G, Jolly C, et al. Drug delivery to the cochlea after implantation: consideration of the risk factors. Cochlear Implants Int. 2005;6:12-4doi: 10.1179/cim.2005.6.Supplement-1.12.
Garnham, C., Reetz, G., Jolly, C., Miller, J., Salt, A., & Beal, F. (2005). Drug delivery to the cochlea after implantation: consideration of the risk factors. Cochlear implants international, 612-4. https://doi.org/10.1179/cim.2005.6.Supplement-1.12
Garnham, C, et al. "Drug delivery to the cochlea after implantation: consideration of the risk factors." Cochlear implants international vol. 6 (2005): 12-4. doi: https://doi.org/10.1179/cim.2005.6.Supplement-1.12
Garnham C, Reetz G, Jolly C, Miller J, Salt A, Beal F. Drug delivery to the cochlea after implantation: consideration of the risk factors. Cochlear Implants Int. 2005 Sep;6:12-4. doi: 10.1179/cim.2005.6.Supplement-1.12. PMID: 18792345.
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Excitotoxicity.

Handbook of clinical neurology

Henchcliffe C, Beal MF.
PMID: 18808934
Handb Clin Neurol. 2007;83:553-69. doi: 10.1016/S0072-9752(07)83027-2.

No abstract available.

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Henchcliffe C, Beal MF. Excitotoxicity. Handb Clin Neurol. 2007;83:553-69doi: 10.1016/S0072-9752(07)83027-2.
Henchcliffe, C., & Beal, M. F. (2007). Excitotoxicity. Handbook of clinical neurology, 83553-69. https://doi.org/10.1016/S0072-9752(07)83027-2
Henchcliffe, Claire, and Beal, M Flint. "Excitotoxicity." Handbook of clinical neurology vol. 83 (2007): 553-69. doi: https://doi.org/10.1016/S0072-9752(07)83027-2
Henchcliffe C, Beal MF. Excitotoxicity. Handb Clin Neurol. 2007;83:553-69. doi: 10.1016/S0072-9752(07)83027-2. PMID: 18808934.
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Hunting-ton for new proteases: MMPs as the new target?.

Neuron

Johri A, Beal MF.
PMID: 20670824
Neuron. 2010 Jul 29;67(2):171-3. doi: 10.1016/j.neuron.2010.07.011.

Mutant huntingtin proteolysis mediated by various proteases plays a key role in Huntington's disease (HD) pathogenesis. In this issue of Neuron, Miller et al. have identified 11 proteases, including matrix metalloproteinases (MMPs), that when inhibited reduce huntingtin proteolysis and...

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Johri A, Beal MF. Hunting-ton for new proteases: MMPs as the new target?. Neuron. 2010;67(2):171-3doi: 10.1016/j.neuron.2010.07.011.
Johri, A., & Beal, M. F. (2010). Hunting-ton for new proteases: MMPs as the new target?. Neuron, 67(2), 171-3. https://doi.org/10.1016/j.neuron.2010.07.011
Johri, Ashu, and Beal, M Flint. "Hunting-ton for new proteases: MMPs as the new target?." Neuron vol. 67,2 (2010): 171-3. doi: https://doi.org/10.1016/j.neuron.2010.07.011
Johri A, Beal MF. Hunting-ton for new proteases: MMPs as the new target?. Neuron. 2010 Jul 29;67(2):171-3. doi: 10.1016/j.neuron.2010.07.011. PMID: 20670824; PMCID: PMC3094731.
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Metabolomic analysis and signatures in motor neuron disease.

Metabolomics : Official journal of the Metabolomic Society

Rozen S, Cudkowicz ME, Bogdanov M, Matson WR, Kristal BS, Beecher C, Harrison S, Vouros P, Flarakos J, Vigneau-Callahan K, Matson TD, Newhall KM, Beal MF, Brown RH, Kaddurah-Daouk R.
PMID: 18820733
Metabolomics. 2005;1(2):101-108. doi: 10.1007/s11306-005-4810-1.

Motor neuron diseases (MND) are a heterogeneous group of disorders that includes amyotrophic lateral sclerosis (ALS) and result in death of motor neurons. These diseases may produce characteristic perturbations of the metabolome, the collection of small-molecules (metabolites) present in...

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Rozen S, Cudkowicz ME, Bogdanov M, et al. Metabolomic analysis and signatures in motor neuron disease. Metabolomics. 2005;1(2):101-108doi: 10.1007/s11306-005-4810-1.
Rozen, S., Cudkowicz, M. E., Bogdanov, M., Matson, W. R., Kristal, B. S., Beecher, C., Harrison, S., Vouros, P., Flarakos, J., Vigneau-Callahan, K., Matson, T. D., Newhall, K. M., Beal, M. F., Brown, R. H., & Kaddurah-Daouk, R. (2005). Metabolomic analysis and signatures in motor neuron disease. Metabolomics : Official journal of the Metabolomic Society, 1(2), 101-108. https://doi.org/10.1007/s11306-005-4810-1
Rozen, Steve, et al. "Metabolomic analysis and signatures in motor neuron disease." Metabolomics : Official journal of the Metabolomic Society vol. 1,2 (2005): 101-108. doi: https://doi.org/10.1007/s11306-005-4810-1
Rozen S, Cudkowicz ME, Bogdanov M, Matson WR, Kristal BS, Beecher C, Harrison S, Vouros P, Flarakos J, Vigneau-Callahan K, Matson TD, Newhall KM, Beal MF, Brown RH, Kaddurah-Daouk R. Metabolomic analysis and signatures in motor neuron disease. Metabolomics. 2005;1(2):101-108. doi: 10.1007/s11306-005-4810-1. PMID: 18820733; PMCID: PMC2553219.
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Muscling in on PGC-1α for improved quality of life in ALS.

Cell metabolism

Johri A, Beal MF.
PMID: 22560208
Cell Metab. 2012 May 02;15(5):567-9. doi: 10.1016/j.cmet.2012.04.015.

Impaired activity of peroxisome proliferator-activated receptor (PPAR)-γ coactivator (PGC)-1α has been implicated in the pathophysiology of several neurodegenerative disorders. In this issue, Da Cruz et al. (2012) show improved muscle function, but not survival, with increased PGC-1α activity in...

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Johri A, Beal MF. Muscling in on PGC-1α for improved quality of life in ALS. Cell Metab. 2012;15(5):567-9doi: 10.1016/j.cmet.2012.04.015.
Johri, A., & Beal, M. F. (2012). Muscling in on PGC-1α for improved quality of life in ALS. Cell metabolism, 15(5), 567-9. https://doi.org/10.1016/j.cmet.2012.04.015
Johri, Ashu, and Beal, M Flint. "Muscling in on PGC-1α for improved quality of life in ALS." Cell metabolism vol. 15,5 (2012): 567-9. doi: https://doi.org/10.1016/j.cmet.2012.04.015
Johri A, Beal MF. Muscling in on PGC-1α for improved quality of life in ALS. Cell Metab. 2012 May 02;15(5):567-9. doi: 10.1016/j.cmet.2012.04.015. PMID: 22560208.
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Diffusion imaging of cerebral white matter in persons who stutter: evidence for network-level anomalies.

Frontiers in human neuroscience

Cai S, Tourville JA, Beal DS, Perkell JS, Guenther FH, Ghosh SS.
PMID: 24611042
Front Hum Neurosci. 2014 Feb 11;8:54. doi: 10.3389/fnhum.2014.00054. eCollection 2014.

Deficits in brain white matter have been a main focus of recent neuroimaging studies on stuttering. However, no prior study has examined brain connectivity on the global level of the cerebral cortex in persons who stutter (PWS). In the...

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Cai S, Tourville JA, Beal DS, et al. Diffusion imaging of cerebral white matter in persons who stutter: evidence for network-level anomalies. Front Hum Neurosci. 2014;8:54doi: 10.3389/fnhum.2014.00054.
Cai, S., Tourville, J. A., Beal, D. S., Perkell, J. S., Guenther, F. H., & Ghosh, S. S. (2014). Diffusion imaging of cerebral white matter in persons who stutter: evidence for network-level anomalies. Frontiers in human neuroscience, 854. https://doi.org/10.3389/fnhum.2014.00054
Cai, Shanqing, et al. "Diffusion imaging of cerebral white matter in persons who stutter: evidence for network-level anomalies." Frontiers in human neuroscience vol. 8 (2014): 54. doi: https://doi.org/10.3389/fnhum.2014.00054
Cai S, Tourville JA, Beal DS, Perkell JS, Guenther FH, Ghosh SS. Diffusion imaging of cerebral white matter in persons who stutter: evidence for network-level anomalies. Front Hum Neurosci. 2014 Feb 11;8:54. doi: 10.3389/fnhum.2014.00054. eCollection 2014. PMID: 24611042; PMCID: PMC3920071.
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Showing 1 to 12 of 128 entries