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Showing 1 to 12 of 24 entries
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P24. Study of C677T methylene tetrahydrofolate reductase (MTHFR) polymorphism in preeclampsia.

Pregnancy hypertension

Matos A, Ferreira J, Portelinha A, Cerdeira AS, Braga J, Patrício B, Rebelo I, Bicho M, Marinho C.
PMID: 26009115
Pregnancy Hypertens. 2011 Jul-Oct;1(3):284. doi: 10.1016/j.preghy.2011.08.085. Epub 2011 Sep 29.

No abstract available.

Cite
Matos A, Ferreira J, Portelinha A, et al. P24. Study of C677T methylene tetrahydrofolate reductase (MTHFR) polymorphism in preeclampsia. Pregnancy Hypertens. 2011;1(3):284doi: 10.1016/j.preghy.2011.08.085.
Matos, A., Ferreira, J., Portelinha, A., Cerdeira, A. S., Braga, J., Patrício, B., Rebelo, I., Bicho, M., & Marinho, C. (2011). P24. Study of C677T methylene tetrahydrofolate reductase (MTHFR) polymorphism in preeclampsia. Pregnancy hypertension, 1(3), 284. https://doi.org/10.1016/j.preghy.2011.08.085
Matos, Andreia, et al. "P24. Study of C677T methylene tetrahydrofolate reductase (MTHFR) polymorphism in preeclampsia." Pregnancy hypertension vol. 1,3 (2011): 284. doi: https://doi.org/10.1016/j.preghy.2011.08.085
Matos A, Ferreira J, Portelinha A, Cerdeira AS, Braga J, Patrício B, Rebelo I, Bicho M, Marinho C. P24. Study of C677T methylene tetrahydrofolate reductase (MTHFR) polymorphism in preeclampsia. Pregnancy Hypertens. 2011 Jul-Oct;1(3):284. doi: 10.1016/j.preghy.2011.08.085. Epub 2011 Sep 29. PMID: 26009115.
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PP004. The polymorphism C677T of methylenetetrahydrofolate reductase (MTHFR), may increase risk for future higher blood pressure in women with previous hypertension in pregnancy.

Pregnancy hypertension

Matos A, da Silva AP, Maia H, Ferreira J, Clode N, Areias MJ, Bicho MC, Bicho M, Rebelo I.
PMID: 26105861
Pregnancy Hypertens. 2013 Apr;3(2):69. doi: 10.1016/j.preghy.2013.04.032. Epub 2013 Jun 06.

INTRODUCTION: The MTHFR is a key enzyme in the folate cycle involved in homocysteine remethylation. The T allele of MTHFR C677T polymorphism is associated with lower activity inhibiting the DNA methylation and protecting from oxidative stress.OBJECTIVES: To evaluate the...

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Matos A, da Silva AP, Maia H, et al. PP004. The polymorphism C677T of methylenetetrahydrofolate reductase (MTHFR), may increase risk for future higher blood pressure in women with previous hypertension in pregnancy. Pregnancy Hypertens. 2013;3(2):69doi: 10.1016/j.preghy.2013.04.032.
Matos, A., da Silva, A. P., Maia, H., Ferreira, J., Clode, N., Areias, M. J., Bicho, M. C., Bicho, M., & Rebelo, I. (2013). PP004. The polymorphism C677T of methylenetetrahydrofolate reductase (MTHFR), may increase risk for future higher blood pressure in women with previous hypertension in pregnancy. Pregnancy hypertension, 3(2), 69. https://doi.org/10.1016/j.preghy.2013.04.032
Matos, Andreia, et al. "PP004. The polymorphism C677T of methylenetetrahydrofolate reductase (MTHFR), may increase risk for future higher blood pressure in women with previous hypertension in pregnancy." Pregnancy hypertension vol. 3,2 (2013): 69. doi: https://doi.org/10.1016/j.preghy.2013.04.032
Matos A, da Silva AP, Maia H, Ferreira J, Clode N, Areias MJ, Bicho MC, Bicho M, Rebelo I. PP004. The polymorphism C677T of methylenetetrahydrofolate reductase (MTHFR), may increase risk for future higher blood pressure in women with previous hypertension in pregnancy. Pregnancy Hypertens. 2013 Apr;3(2):69. doi: 10.1016/j.preghy.2013.04.032. Epub 2013 Jun 06. PMID: 26105861.
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Molding of hydrogel microstructures to create multiphenotype cell microarrays.

Analytical chemistry

Koh WG, Itle LJ, Pishko MV.
PMID: 14588018
Anal Chem. 2003 Nov 01;75(21):5783-9. doi: 10.1021/ac034773s.

The fabrication of mammalian cell-containing poly(ethylene glycol) (PEG) hydrogel microstructures on glass and silicon substrates is described. Using photoreaction injection molding in poly(dimethylsiloxane) microfluidic channels, three-dimensional hydrogel microstructures encapsulating cells (fibroblasts, hepatocytes, macrophage) were fabricated with cells uniformly distributed...

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Koh WG, Itle LJ, Pishko MV. Molding of hydrogel microstructures to create multiphenotype cell microarrays. Anal Chem. 2003;75(21):5783-9doi: 10.1021/ac034773s.
Koh, W. G., Itle, L. J., & Pishko, M. V. (2003). Molding of hydrogel microstructures to create multiphenotype cell microarrays. Analytical chemistry, 75(21), 5783-9. https://doi.org/10.1021/ac034773s
Koh, Won-Gun, et al. "Molding of hydrogel microstructures to create multiphenotype cell microarrays." Analytical chemistry vol. 75,21 (2003): 5783-9. doi: https://doi.org/10.1021/ac034773s
Koh WG, Itle LJ, Pishko MV. Molding of hydrogel microstructures to create multiphenotype cell microarrays. Anal Chem. 2003 Nov 01;75(21):5783-9. doi: 10.1021/ac034773s. PMID: 14588018.
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8H-Chromeno[2',3':4,5]imidazo[2,1-a]isoquinoline.

Acta crystallographica. Section E, Structure reports online

Safarov S, Voskressensky LG, Bizhko OV, Kulikova LN, Khrustalev VN.
PMID: 21580448
Acta Crystallogr Sect E Struct Rep Online. 2010 Feb 27;66:o710. doi: 10.1107/S1600536810006744.

The title compound, C(18)H(12)N(2)O, comprises two aromatic fragments, viz., imidazo[2,1-a]isoquinoline and benzene, linked by oxygen and methyl-ene bridges. Despite the absence of a common conjugative system within the mol-ecule, it adopts an essentially planar conformation with an r.m.s. deviation...

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Safarov S, Voskressensky LG, Bizhko OV, et al. 8H-Chromeno[2',3':4,5]imidazo[2,1-a]isoquinoline. Acta Crystallogr Sect E Struct Rep Online. 2010;66:o710doi: 10.1107/S1600536810006744.
Safarov, S., Voskressensky, L. G., Bizhko, O. V., Kulikova, L. N., & Khrustalev, V. N. (2010). 8H-Chromeno[2',3':4,5]imidazo[2,1-a]isoquinoline. Acta crystallographica. Section E, Structure reports online, 66o710. https://doi.org/10.1107/S1600536810006744
Safarov, Saifidin, et al. "8H-Chromeno[2',3':4,5]imidazo[2,1-a]isoquinoline." Acta crystallographica. Section E, Structure reports online vol. 66 (2010): o710. doi: https://doi.org/10.1107/S1600536810006744
Safarov S, Voskressensky LG, Bizhko OV, Kulikova LN, Khrustalev VN. 8H-Chromeno[2',3':4,5]imidazo[2,1-a]isoquinoline. Acta Crystallogr Sect E Struct Rep Online. 2010 Feb 27;66:o710. doi: 10.1107/S1600536810006744. PMID: 21580448; PMCID: PMC2983581.
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Encapsulation of a Concanavalin A/dendrimer glucose sensing assay within microporated poly (ethylene glycol) microspheres.

Biomedical optics express

Cummin BM, Lim J, Simanek EE, Pishko MV, Coté GL.
PMID: 21559135
Biomed Opt Express. 2011 Apr 18;2(5):1243-57. doi: 10.1364/BOE.2.001243.

Proper management of diabetes requires the frequent measurement of a patient's blood glucose level. To create a long-term, minimally-invasive sensor that is sensitive to physiological concentrations of glucose a fluorescent glucose sensing assay using a competitive binding approach between...

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Cummin BM, Lim J, Simanek EE, et al. Encapsulation of a Concanavalin A/dendrimer glucose sensing assay within microporated poly (ethylene glycol) microspheres. Biomed Opt Express. 2011;2(5):1243-57doi: 10.1364/BOE.2.001243.
Cummin, B. M., Lim, J., Simanek, E. E., Pishko, M. V., & Coté, G. L. (2011). Encapsulation of a Concanavalin A/dendrimer glucose sensing assay within microporated poly (ethylene glycol) microspheres. Biomedical optics express, 2(5), 1243-57. https://doi.org/10.1364/BOE.2.001243
Cummin, Brian M, et al. "Encapsulation of a Concanavalin A/dendrimer glucose sensing assay within microporated poly (ethylene glycol) microspheres." Biomedical optics express vol. 2,5 (2011): 1243-57. doi: https://doi.org/10.1364/BOE.2.001243
Cummin BM, Lim J, Simanek EE, Pishko MV, Coté GL. Encapsulation of a Concanavalin A/dendrimer glucose sensing assay within microporated poly (ethylene glycol) microspheres. Biomed Opt Express. 2011 Apr 18;2(5):1243-57. doi: 10.1364/BOE.2.001243. PMID: 21559135; PMCID: PMC3087580.
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Insulin activation of NADH ferricyanide reductase in human erythrocytes is mediated by the insulin receptor tyrosine kinase: a comparative study in normal and diabetic states.

Redox report : communications in free radical research

Marques F, Crespo ME, Pantaleão O, Bicho M.
PMID: 27406671
Redox Rep. 1996 Dec;2(6):373-8. doi: 10.1080/13510002.1996.11747077.

We have previously reported that NADH ferricyanide reductase in human erythrocytes is stimulated by insulin. Hormone-stimulated activities are attenuated in the presence of glycolytic inhibitors like vanadate, indicating the involvement of glycolysis in the mechanism by which insulin stimulates...

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Marques F, Crespo ME, Pantaleão O, et al. Insulin activation of NADH ferricyanide reductase in human erythrocytes is mediated by the insulin receptor tyrosine kinase: a comparative study in normal and diabetic states. Redox Rep. 1996;2(6):373-8doi: 10.1080/13510002.1996.11747077.
Marques, F., Crespo, M. E., Pantaleão, O., & Bicho, M. (1996). Insulin activation of NADH ferricyanide reductase in human erythrocytes is mediated by the insulin receptor tyrosine kinase: a comparative study in normal and diabetic states. Redox report : communications in free radical research, 2(6), 373-8. https://doi.org/10.1080/13510002.1996.11747077
Marques, F, et al. "Insulin activation of NADH ferricyanide reductase in human erythrocytes is mediated by the insulin receptor tyrosine kinase: a comparative study in normal and diabetic states." Redox report : communications in free radical research vol. 2,6 (1996): 373-8. doi: https://doi.org/10.1080/13510002.1996.11747077
Marques F, Crespo ME, Pantaleão O, Bicho M. Insulin activation of NADH ferricyanide reductase in human erythrocytes is mediated by the insulin receptor tyrosine kinase: a comparative study in normal and diabetic states. Redox Rep. 1996 Dec;2(6):373-8. doi: 10.1080/13510002.1996.11747077. PMID: 27406671.
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Commentary: The saphenous vein in coronary artery bypass grafting: Optimizing our workhorse.

JTCVS techniques

Buchko MT, Ruel M.
PMID: 34984369
JTCVS Tech. 2021 Oct 19;10:110-111. doi: 10.1016/j.xjtc.2021.10.020. eCollection 2021 Dec.

No abstract available.

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Buchko MT, Ruel M. Commentary: The saphenous vein in coronary artery bypass grafting: Optimizing our workhorse. JTCVS Tech. 2021;10:110-111doi: 10.1016/j.xjtc.2021.10.020.
Buchko, M. T., & Ruel, M. (2021). Commentary: The saphenous vein in coronary artery bypass grafting: Optimizing our workhorse. JTCVS techniques, 10110-111. https://doi.org/10.1016/j.xjtc.2021.10.020
Buchko, Max T, and Ruel, Marc. "Commentary: The saphenous vein in coronary artery bypass grafting: Optimizing our workhorse." JTCVS techniques vol. 10 (2021): 110-111. doi: https://doi.org/10.1016/j.xjtc.2021.10.020
Buchko MT, Ruel M. Commentary: The saphenous vein in coronary artery bypass grafting: Optimizing our workhorse. JTCVS Tech. 2021 Oct 19;10:110-111. doi: 10.1016/j.xjtc.2021.10.020. eCollection 2021 Dec. PMID: 34984369; PMCID: PMC8691916.
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Visualizing the drug target landscape.

Drug discovery today

Campbell SJ, Gaulton A, Marshall J, Bichko D, Martin S, Brouwer C, Harland L.
PMID: 22178891
Drug Discov Today. 2012 Feb;17:S3-15. doi: 10.1016/j.drudis.2011.12.005. Epub 2011 Dec 07.

Generating new therapeutic hypotheses for human disease requires the analysis and interpretation of many different experimental datasets. Assembling a holistic picture of the current landscape of drug discovery activity remains a challenge, however, because of the lack of integration...

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Campbell SJ, Gaulton A, Marshall J, et al. Visualizing the drug target landscape. Drug Discov Today. 2011;17:S3-15doi: 10.1016/j.drudis.2011.12.005.
Campbell, S. J., Gaulton, A., Marshall, J., Bichko, D., Martin, S., Brouwer, C., & Harland, L. (2012). Visualizing the drug target landscape. Drug discovery today, 17S3-15. https://doi.org/10.1016/j.drudis.2011.12.005
Campbell, Stephen J, et al. "Visualizing the drug target landscape." Drug discovery today vol. 17 (2012): S3-15. doi: https://doi.org/10.1016/j.drudis.2011.12.005
Campbell SJ, Gaulton A, Marshall J, Bichko D, Martin S, Brouwer C, Harland L. Visualizing the drug target landscape. Drug Discov Today. 2012 Feb;17:S3-15. doi: 10.1016/j.drudis.2011.12.005. Epub 2011 Dec 07. PMID: 22178891.
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In women with previous pregnancy hypertension, levels of cardiovascular risk biomarkers may be modulated by haptoglobin polymorphism.

Obstetrics and gynecology international

Matos A, Pereira da Silva A, Clara Bicho M, Afonso C, Areias MJ, Rebelo I, Bicho M.
PMID: 25101128
Obstet Gynecol Int. 2014;2014:361727. doi: 10.1155/2014/361727. Epub 2014 Jul 02.

Preeclampsia (PE) may affect the risk for future cardiovascular disease. Haptoglobin (Hp), an acute phase protein with functional genetic polymorphism, synthesized in the hepatocyte and in many peripheral tissues secondary of oxidative stress of PE, may modulate that risk...

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Matos A, Pereira da Silva A, Clara Bicho M, et al. In women with previous pregnancy hypertension, levels of cardiovascular risk biomarkers may be modulated by haptoglobin polymorphism. Obstet Gynecol Int. 2014;2014:361727doi: 10.1155/2014/361727.
Matos, A., Pereira da Silva, A., Clara Bicho, M., Afonso, C., Areias, M. J., Rebelo, I., & Bicho, M. (2014). In women with previous pregnancy hypertension, levels of cardiovascular risk biomarkers may be modulated by haptoglobin polymorphism. Obstetrics and gynecology international, 2014361727. https://doi.org/10.1155/2014/361727
Matos, Andreia, et al. "In women with previous pregnancy hypertension, levels of cardiovascular risk biomarkers may be modulated by haptoglobin polymorphism." Obstetrics and gynecology international vol. 2014 (2014): 361727. doi: https://doi.org/10.1155/2014/361727
Matos A, Pereira da Silva A, Clara Bicho M, Afonso C, Areias MJ, Rebelo I, Bicho M. In women with previous pregnancy hypertension, levels of cardiovascular risk biomarkers may be modulated by haptoglobin polymorphism. Obstet Gynecol Int. 2014;2014:361727. doi: 10.1155/2014/361727. Epub 2014 Jul 02. PMID: 25101128; PMCID: PMC4102073.
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The main autoimmune and nonautoimmune etiologies of endogenous hyperthyroidism do not seem to influence the increased prevalence of morphometric vertebral fractures and osteoporosis in Portuguese men.

Osteoporosis and sarcopenia

Barbosa AP, Mascarenhas MR, Bicho M, Janeiro J, Oliveira AG.
PMID: 30775520
Osteoporos Sarcopenia. 2017 Sep;3(3):149-154. doi: 10.1016/j.afos.2017.08.099. Epub 2017 Sep 27.

OBJECTIVES: The purpose of this study was to evaluate the effects of hyperthyroidism and their etiology on bone mineral density (BMD), on body soft tissue composition, on the prevalence of vertebral fractures detected by vertebral fracture assessment (VFA) and...

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Barbosa AP, Mascarenhas MR, Bicho M, et al. The main autoimmune and nonautoimmune etiologies of endogenous hyperthyroidism do not seem to influence the increased prevalence of morphometric vertebral fractures and osteoporosis in Portuguese men. Osteoporos Sarcopenia. 2017;3(3):149-154doi: 10.1016/j.afos.2017.08.099.
Barbosa, A. P., Mascarenhas, M. R., Bicho, M., Janeiro, J., & Oliveira, A. G. (2017). The main autoimmune and nonautoimmune etiologies of endogenous hyperthyroidism do not seem to influence the increased prevalence of morphometric vertebral fractures and osteoporosis in Portuguese men. Osteoporosis and sarcopenia, 3(3), 149-154. https://doi.org/10.1016/j.afos.2017.08.099
Barbosa, Ana Paula, et al. "The main autoimmune and nonautoimmune etiologies of endogenous hyperthyroidism do not seem to influence the increased prevalence of morphometric vertebral fractures and osteoporosis in Portuguese men." Osteoporosis and sarcopenia vol. 3,3 (2017): 149-154. doi: https://doi.org/10.1016/j.afos.2017.08.099
Barbosa AP, Mascarenhas MR, Bicho M, Janeiro J, Oliveira AG. The main autoimmune and nonautoimmune etiologies of endogenous hyperthyroidism do not seem to influence the increased prevalence of morphometric vertebral fractures and osteoporosis in Portuguese men. Osteoporos Sarcopenia. 2017 Sep;3(3):149-154. doi: 10.1016/j.afos.2017.08.099. Epub 2017 Sep 27. PMID: 30775520; PMCID: PMC6372816.
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Psychosocial and Neurocognitive Factors Associated With Hepatitis C - Implications for Future Health and Wellbeing.

Frontiers in psychology

Barreira DP, Marinho RT, Bicho M, Fialho R, Ouakinin SRS.
PMID: 30687151
Front Psychol. 2019 Jan 09;9:2666. doi: 10.3389/fpsyg.2018.02666. eCollection 2018.

No abstract available.

Cite
Barreira DP, Marinho RT, Bicho M, et al. Psychosocial and Neurocognitive Factors Associated With Hepatitis C - Implications for Future Health and Wellbeing. Front Psychol. 2019;9:2666doi: 10.3389/fpsyg.2018.02666.
Barreira, D. P., Marinho, R. T., Bicho, M., Fialho, R., & Ouakinin, S. R. S. (2018). Psychosocial and Neurocognitive Factors Associated With Hepatitis C - Implications for Future Health and Wellbeing. Frontiers in psychology, 92666. https://doi.org/10.3389/fpsyg.2018.02666
Barreira, David Pires, et al. "Psychosocial and Neurocognitive Factors Associated With Hepatitis C - Implications for Future Health and Wellbeing." Frontiers in psychology vol. 9 (2018): 2666. doi: https://doi.org/10.3389/fpsyg.2018.02666
Barreira DP, Marinho RT, Bicho M, Fialho R, Ouakinin SRS. Psychosocial and Neurocognitive Factors Associated With Hepatitis C - Implications for Future Health and Wellbeing. Front Psychol. 2019 Jan 09;9:2666. doi: 10.3389/fpsyg.2018.02666. eCollection 2018. PMID: 30687151; PMCID: PMC6333630.
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Arginine and Arginases Modulate Metabolism, Tumor Microenvironment and Prostate Cancer Progression.

Nutrients

Matos A, Carvalho M, Bicho M, Ribeiro R.
PMID: 34960055
Nutrients. 2021 Dec 16;13(12). doi: 10.3390/nu13124503.

Arginine availability and activation of arginine-related pathways at cancer sites have profound effects on the tumor microenvironment, far beyond their well-known role in the hepatic urea cycle. Arginine metabolism impacts not only malignant cells but also the surrounding immune...

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Matos A, Carvalho M, Bicho M, et al. Arginine and Arginases Modulate Metabolism, Tumor Microenvironment and Prostate Cancer Progression. Nutrients. 2021;13(12)doi: 10.3390/nu13124503.
Matos, A., Carvalho, M., Bicho, M., & Ribeiro, R. (2021). Arginine and Arginases Modulate Metabolism, Tumor Microenvironment and Prostate Cancer Progression. Nutrients, 13(12), . https://doi.org/10.3390/nu13124503
Matos, Andreia, et al. "Arginine and Arginases Modulate Metabolism, Tumor Microenvironment and Prostate Cancer Progression." Nutrients vol. 13,12 (2021). doi: https://doi.org/10.3390/nu13124503
Matos A, Carvalho M, Bicho M, Ribeiro R. Arginine and Arginases Modulate Metabolism, Tumor Microenvironment and Prostate Cancer Progression. Nutrients. 2021 Dec 16;13(12). doi: 10.3390/nu13124503. PMID: 34960055; PMCID: PMC8704013.
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