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Showing 1 to 12 of 112 entries
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MICROTRANSPLANTATION OF NIGRAL DOPAMINE NEURONS IN A RAT MODEL OF PARKINSON'S DISEASE.

Cell transplantation

Borlongan CV, Sanberg PR.
PMID: 28148360
Cell Transplant. 1995 May 01;4(3):347-348. doi: 10.1177/096368979500400314.

No abstract available.

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Borlongan CV, Sanberg PR. MICROTRANSPLANTATION OF NIGRAL DOPAMINE NEURONS IN A RAT MODEL OF PARKINSON'S DISEASE. Cell Transplant. 1995;4(3):347-348doi: 10.1177/096368979500400314.
Borlongan, C. V., & Sanberg, P. R. (1995). MICROTRANSPLANTATION OF NIGRAL DOPAMINE NEURONS IN A RAT MODEL OF PARKINSON'S DISEASE. Cell transplantation, 4(3), 347-348. https://doi.org/10.1177/096368979500400314
Borlongan, Cesario V, and Sanberg, Paul R. "MICROTRANSPLANTATION OF NIGRAL DOPAMINE NEURONS IN A RAT MODEL OF PARKINSON'S DISEASE." Cell transplantation vol. 4,3 (1995): 347-348. doi: https://doi.org/10.1177/096368979500400314
Borlongan CV, Sanberg PR. MICROTRANSPLANTATION OF NIGRAL DOPAMINE NEURONS IN A RAT MODEL OF PARKINSON'S DISEASE. Cell Transplant. 1995 May 01;4(3):347-348. doi: 10.1177/096368979500400314. PMID: 28148360.
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Article Commentary: G-CSF-Mobilized Human Peripheral Blood for Transplantation Therapy in Stroke.

Cell transplantation

Borlongan CV, Hess DC.
PMID: 28876090
Cell Transplant. 2003 May;12(4):447-448. doi: 10.3727/000000003108746876.

No abstract available.

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Borlongan CV, Hess DC. Article Commentary: G-CSF-Mobilized Human Peripheral Blood for Transplantation Therapy in Stroke. Cell Transplant. 2003;12(4):447-448doi: 10.3727/000000003108746876.
Borlongan, C. V., & Hess, D. C. (2003). Article Commentary: G-CSF-Mobilized Human Peripheral Blood for Transplantation Therapy in Stroke. Cell transplantation, 12(4), 447-448. https://doi.org/10.3727/000000003108746876
Borlongan, Cesar V, and Hess, David C. "Article Commentary: G-CSF-Mobilized Human Peripheral Blood for Transplantation Therapy in Stroke." Cell transplantation vol. 12,4 (2003): 447-448. doi: https://doi.org/10.3727/000000003108746876
Borlongan CV, Hess DC. Article Commentary: G-CSF-Mobilized Human Peripheral Blood for Transplantation Therapy in Stroke. Cell Transplant. 2003 May;12(4):447-448. doi: 10.3727/000000003108746876. PMID: 28876090.
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Human Umbilical Cord Blood for Transplantation Therapy in Myocardial Infarction.

Journal of stem cell research & therapy

Acosta SA, Franzese N, Staples M, Weinbren NL, Babilonia M, Patel J, Merchant N, Simancas AJ, Slakter A, Caputo M, Patel M, Franyuti G, Franzblau MH, Suarez L, Gonzales-Portillo C, Diamandis T, Shinozuka K, Tajiri N, Sanberg PR, Kaneko Y, Miller LW, Borlongan CV.
PMID: 24307973
J Stem Cell Res Ther. 2013 Jul 01;

Cell-based therapy is a promising therapy for myocardial infarction. Endogenous repair of the heart muscle after myocardial infarction is a challenge because adult cardiomyocytes have a limited capacity to proliferate and replace damaged cells. Pre-clinical and clinical evidence has...

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Acosta SA, Franzese N, Staples M, et al. Human Umbilical Cord Blood for Transplantation Therapy in Myocardial Infarction. J Stem Cell Res Ther. 2013;
Acosta, S. A., Franzese, N., Staples, M., Weinbren, N. L., Babilonia, M., Patel, J., Merchant, N., Simancas, A. J., Slakter, A., Caputo, M., Patel, M., Franyuti, G., Franzblau, M. H., Suarez, L., Gonzales-Portillo, C., Diamandis, T., Shinozuka, K., Tajiri, N., Sanberg, P. R., Kaneko, Y., Miller, L. W., & Borlongan, C. V. (2013). Human Umbilical Cord Blood for Transplantation Therapy in Myocardial Infarction. Journal of stem cell research & therapy, .
Acosta, Sandra A, et al. "Human Umbilical Cord Blood for Transplantation Therapy in Myocardial Infarction." Journal of stem cell research & therapy vol. (2013).
Acosta SA, Franzese N, Staples M, Weinbren NL, Babilonia M, Patel J, Merchant N, Simancas AJ, Slakter A, Caputo M, Patel M, Franyuti G, Franzblau MH, Suarez L, Gonzales-Portillo C, Diamandis T, Shinozuka K, Tajiri N, Sanberg PR, Kaneko Y, Miller LW, Borlongan CV. Human Umbilical Cord Blood for Transplantation Therapy in Myocardial Infarction. J Stem Cell Res Ther. 2013 Jul 01; PMID: 24307973; PMCID: PMC3845524.
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Asymmetrical behavior in rats following striatal lesions and fetal transplants: the elevated body swing test.

Restorative neurology and neuroscience

Borlongan CV, Cahill DW, Sanberg PR.
PMID: 21551830
Restor Neurol Neurosci. 1995 Jan 01;9(1):15-9. doi: 10.3233/RNN-1995-9103.

Animals with unilateral neurotoxic lesions in the striatum exhibit a stereotypical biased rotational behavior in response to dopamine agonists. We recently argued that the rotational test may be subject to sensitization effects of the drug. Accordingly, we proposed the...

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Borlongan CV, Cahill DW, Sanberg PR. Asymmetrical behavior in rats following striatal lesions and fetal transplants: the elevated body swing test. Restor Neurol Neurosci. 1995;9(1):15-9doi: 10.3233/RNN-1995-9103.
Borlongan, C. V., Cahill, D. W., & Sanberg, P. R. (1995). Asymmetrical behavior in rats following striatal lesions and fetal transplants: the elevated body swing test. Restorative neurology and neuroscience, 9(1), 15-9. https://doi.org/10.3233/RNN-1995-9103
Borlongan, C V, et al. "Asymmetrical behavior in rats following striatal lesions and fetal transplants: the elevated body swing test." Restorative neurology and neuroscience vol. 9,1 (1995): 15-9. doi: https://doi.org/10.3233/RNN-1995-9103
Borlongan CV, Cahill DW, Sanberg PR. Asymmetrical behavior in rats following striatal lesions and fetal transplants: the elevated body swing test. Restor Neurol Neurosci. 1995 Jan 01;9(1):15-9. doi: 10.3233/RNN-1995-9103. PMID: 21551830.
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Age-dependent neurobehavioral responses by young and mature adult rats to systemic kainic acid.

Restorative neurology and neuroscience

Borlongan CV, Bjugstad KB, Stahl CE, Ross SD, Arendash GW, Cahill DW, Sanberg PR.
PMID: 21551859
Restor Neurol Neurosci. 1996 Jan 01;10(2):103-8. doi: 10.3233/RNN-1996-10206.

Neurobehavioral effects caused by the excitotoxin kainic acid (KA) have been characterized by convulsions including 'wet dog shakes' (WDS) with accompanying hippocampal degeneration in experimental animals. Accordingly, this model has been proposed for putative excitotoxin-mediated disorders, such as the...

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Borlongan CV, Bjugstad KB, Stahl CE, et al. Age-dependent neurobehavioral responses by young and mature adult rats to systemic kainic acid. Restor Neurol Neurosci. 1996;10(2):103-8doi: 10.3233/RNN-1996-10206.
Borlongan, C. V., Bjugstad, K. B., Stahl, C. E., Ross, S. D., Arendash, G. W., Cahill, D. W., & Sanberg, P. R. (1996). Age-dependent neurobehavioral responses by young and mature adult rats to systemic kainic acid. Restorative neurology and neuroscience, 10(2), 103-8. https://doi.org/10.3233/RNN-1996-10206
Borlongan, C V, et al. "Age-dependent neurobehavioral responses by young and mature adult rats to systemic kainic acid." Restorative neurology and neuroscience vol. 10,2 (1996): 103-8. doi: https://doi.org/10.3233/RNN-1996-10206
Borlongan CV, Bjugstad KB, Stahl CE, Ross SD, Arendash GW, Cahill DW, Sanberg PR. Age-dependent neurobehavioral responses by young and mature adult rats to systemic kainic acid. Restor Neurol Neurosci. 1996 Jan 01;10(2):103-8. doi: 10.3233/RNN-1996-10206. PMID: 21551859.
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Detrimental effects of physical inactivity on neurogenesis.

Brain circulation

Lippert T, Watson N, Ji X, Yasuhara T, Date I, Kaneko Y, Tajiri N, Borlongan CV.
PMID: 30276277
Brain Circ. 2016 Apr-Jun;2(2):80-85. doi: 10.4103/2394-8108.186278. Epub 2016 Jul 13.

Patients diagnosed with neurological disorders exhibit a variety of physical and psychiatric symptoms, including muscle atrophy, general immobility, and depression. Patients who participate in physical rehabilitation at times show unexpected clinical improvement, which includes diminished depression and other stress-related...

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Lippert T, Watson N, Ji X, et al. Detrimental effects of physical inactivity on neurogenesis. Brain Circ. 2016;2(2):80-85doi: 10.4103/2394-8108.186278.
Lippert, T., Watson, N., Ji, X., Yasuhara, T., Date, I., Kaneko, Y., Tajiri, N., & Borlongan, C. V. (2016). Detrimental effects of physical inactivity on neurogenesis. Brain circulation, 2(2), 80-85. https://doi.org/10.4103/2394-8108.186278
Lippert, Trenton, et al. "Detrimental effects of physical inactivity on neurogenesis." Brain circulation vol. 2,2 (2016): 80-85. doi: https://doi.org/10.4103/2394-8108.186278
Lippert T, Watson N, Ji X, Yasuhara T, Date I, Kaneko Y, Tajiri N, Borlongan CV. Detrimental effects of physical inactivity on neurogenesis. Brain Circ. 2016 Apr-Jun;2(2):80-85. doi: 10.4103/2394-8108.186278. Epub 2016 Jul 13. PMID: 30276277; PMCID: PMC6126252.
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Transplants of Encapsulated Rat Choroid Plexus Cells Exert Neuroprotection in a Rodent Model of Huntington's Disease.

Cell transplantation

Borlongan CV, Thanos CG, Skinner SJM, Geaney M, Emerich DF.
PMID: 28866919
Cell Transplant. 2007 Nov;16(10):987-992. doi: 10.3727/000000007783472426.

Choroid plexus (CP) epithelial cells secrete several neurotrophic factors and have been used in transplantation studies designed to impart neuroprotection against central nervous system (CNS) trauma. In the present study, CP was isolated from adult rats, encapsulated within alginate...

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Borlongan CV, Thanos CG, Skinner SJM, et al. Transplants of Encapsulated Rat Choroid Plexus Cells Exert Neuroprotection in a Rodent Model of Huntington's Disease. Cell Transplant. 2007;16(10):987-992doi: 10.3727/000000007783472426.
Borlongan, C. V., Thanos, C. G., Skinner, S. J. M., Geaney, M., & Emerich, D. F. (2007). Transplants of Encapsulated Rat Choroid Plexus Cells Exert Neuroprotection in a Rodent Model of Huntington's Disease. Cell transplantation, 16(10), 987-992. https://doi.org/10.3727/000000007783472426
Borlongan, Cesario V, et al. "Transplants of Encapsulated Rat Choroid Plexus Cells Exert Neuroprotection in a Rodent Model of Huntington's Disease." Cell transplantation vol. 16,10 (2007): 987-992. doi: https://doi.org/10.3727/000000007783472426
Borlongan CV, Thanos CG, Skinner SJM, Geaney M, Emerich DF. Transplants of Encapsulated Rat Choroid Plexus Cells Exert Neuroprotection in a Rodent Model of Huntington's Disease. Cell Transplant. 2007 Nov;16(10):987-992. doi: 10.3727/000000007783472426. PMID: 28866919.
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Advancing stem cells: New therapeutic strategies for treating central nervous system disorders.

Brain circulation

Russo E, Lippert T, Tuazon JP, Borlongan CV.
PMID: 30450412
Brain Circ. 2018 Jul-Sep;4(3):81-83. doi: 10.4103/bc.bc_22_18. Epub 2018 Oct 09.

No abstract available.

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Russo E, Lippert T, Tuazon JP, et al. Advancing stem cells: New therapeutic strategies for treating central nervous system disorders. Brain Circ. 2018;4(3):81-83doi: 10.4103/bc.bc_22_18.
Russo, E., Lippert, T., Tuazon, J. P., & Borlongan, C. V. (2018). Advancing stem cells: New therapeutic strategies for treating central nervous system disorders. Brain circulation, 4(3), 81-83. https://doi.org/10.4103/bc.bc_22_18
Russo, Eleonora, et al. "Advancing stem cells: New therapeutic strategies for treating central nervous system disorders." Brain circulation vol. 4,3 (2018): 81-83. doi: https://doi.org/10.4103/bc.bc_22_18
Russo E, Lippert T, Tuazon JP, Borlongan CV. Advancing stem cells: New therapeutic strategies for treating central nervous system disorders. Brain Circ. 2018 Jul-Sep;4(3):81-83. doi: 10.4103/bc.bc_22_18. Epub 2018 Oct 09. PMID: 30450412; PMCID: PMC6187938.
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Mitochondrial targeting as a novel therapy for stroke.

Brain circulation

Russo E, Nguyen H, Lippert T, Tuazon J, Borlongan CV, Napoli E.
PMID: 30450413
Brain Circ. 2018 Jul-Sep;4(3):84-94. doi: 10.4103/bc.bc_14_18. Epub 2018 Oct 09.

Stroke is a main cause of mortality and morbidity worldwide. Despite the increasing development of innovative treatments for stroke, most are unsuccessful in clinical trials. In recent years, an encouraging strategy for stroke therapy has been identified in stem...

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Russo E, Nguyen H, Lippert T, et al. Mitochondrial targeting as a novel therapy for stroke. Brain Circ. 2018;4(3):84-94doi: 10.4103/bc.bc_14_18.
Russo, E., Nguyen, H., Lippert, T., Tuazon, J., Borlongan, C. V., & Napoli, E. (2018). Mitochondrial targeting as a novel therapy for stroke. Brain circulation, 4(3), 84-94. https://doi.org/10.4103/bc.bc_14_18
Russo, Eleonora, et al. "Mitochondrial targeting as a novel therapy for stroke." Brain circulation vol. 4,3 (2018): 84-94. doi: https://doi.org/10.4103/bc.bc_14_18
Russo E, Nguyen H, Lippert T, Tuazon J, Borlongan CV, Napoli E. Mitochondrial targeting as a novel therapy for stroke. Brain Circ. 2018 Jul-Sep;4(3):84-94. doi: 10.4103/bc.bc_14_18. Epub 2018 Oct 09. PMID: 30450413; PMCID: PMC6187947.
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Combination therapy for ischemic stroke: Novel approaches to lengthen therapeutic window of tissue plasminogen activator.

Brain circulation

Knecht T, Borlongan C, Dela Peña I.
PMID: 30450415
Brain Circ. 2018 Jul-Sep;4(3):99-108. doi: 10.4103/bc.bc_21_18. Epub 2018 Oct 09.

Tissue plasminogen activator (tPA) thrombolysis continues to be the gold standard therapy for ischemic stroke. Due to the time-limited treatment window, within 4.5 h of stroke onset, and a variety of potentially deadly complications related to delayed administration, particularly...

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Knecht T, Borlongan C, Dela Peña I. Combination therapy for ischemic stroke: Novel approaches to lengthen therapeutic window of tissue plasminogen activator. Brain Circ. 2018;4(3):99-108doi: 10.4103/bc.bc_21_18.
Knecht, T., Borlongan, C., & Dela Peña, I. (2018). Combination therapy for ischemic stroke: Novel approaches to lengthen therapeutic window of tissue plasminogen activator. Brain circulation, 4(3), 99-108. https://doi.org/10.4103/bc.bc_21_18
Knecht, Talia, et al. "Combination therapy for ischemic stroke: Novel approaches to lengthen therapeutic window of tissue plasminogen activator." Brain circulation vol. 4,3 (2018): 99-108. doi: https://doi.org/10.4103/bc.bc_21_18
Knecht T, Borlongan C, Dela Peña I. Combination therapy for ischemic stroke: Novel approaches to lengthen therapeutic window of tissue plasminogen activator. Brain Circ. 2018 Jul-Sep;4(3):99-108. doi: 10.4103/bc.bc_21_18. Epub 2018 Oct 09. PMID: 30450415; PMCID: PMC6187940.
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Brief overview: Protective roles of astrocyte-derived pentraxin-3 in blood-brain barrier integrity.

Brain circulation

Bonsack B, Borlongan MC, Lo EH, Arai K.
PMID: 31620663
Brain Circ. 2019 Sep 30;5(3):145-149. doi: 10.4103/bc.bc_37_19. eCollection 2019.

Stroke is one of the world's leading causes of mortality and morbidity. Greater understanding is required of the underlying relationships in ischemic brains in order to prevent stroke or to develop effective treatment. This review highlights new findings about...

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Bonsack B, Borlongan MC, Lo EH, et al. Brief overview: Protective roles of astrocyte-derived pentraxin-3 in blood-brain barrier integrity. Brain Circ. 2019;5(3):145-149doi: 10.4103/bc.bc_37_19.
Bonsack, B., Borlongan, M. C., Lo, E. H., & Arai, K. (2019). Brief overview: Protective roles of astrocyte-derived pentraxin-3 in blood-brain barrier integrity. Brain circulation, 5(3), 145-149. https://doi.org/10.4103/bc.bc_37_19
Bonsack, Brooke, et al. "Brief overview: Protective roles of astrocyte-derived pentraxin-3 in blood-brain barrier integrity." Brain circulation vol. 5,3 (2019): 145-149. doi: https://doi.org/10.4103/bc.bc_37_19
Bonsack B, Borlongan MC, Lo EH, Arai K. Brief overview: Protective roles of astrocyte-derived pentraxin-3 in blood-brain barrier integrity. Brain Circ. 2019 Sep 30;5(3):145-149. doi: 10.4103/bc.bc_37_19. eCollection 2019. PMID: 31620663; PMCID: PMC6785941.
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Correction to: High‑Mobility Group Box‑1‑Induced Angiogenesis After Indirect Bypass Surgery in a Chronic Cerebral Hypoperfusion Model.

Neuromolecular medicine

Nishihiro S, Hishikawa T, Hiramatsu M, Kidani N, Takahashi Y, Murai S, Sugiu K, Higaki Y, Yasuhara T, Borlongan CV, Date I.
PMID: 32103453
Neuromolecular Med. 2020 Jun;22(2):332-333. doi: 10.1007/s12017-020-08594-3.

The original version of this article unfortunately contained a mistake. Figure 5a, b were incorrect. The correct figures are given below.

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Nishihiro S, Hishikawa T, Hiramatsu M, et al. Correction to: High‑Mobility Group Box‑1‑Induced Angiogenesis After Indirect Bypass Surgery in a Chronic Cerebral Hypoperfusion Model. Neuromolecular Med. 2020;22(2):332-333doi: 10.1007/s12017-020-08594-3.
Nishihiro, S., Hishikawa, T., Hiramatsu, M., Kidani, N., Takahashi, Y., Murai, S., Sugiu, K., Higaki, Y., Yasuhara, T., Borlongan, C. V., & Date, I. (2020). Correction to: High‑Mobility Group Box‑1‑Induced Angiogenesis After Indirect Bypass Surgery in a Chronic Cerebral Hypoperfusion Model. Neuromolecular medicine, 22(2), 332-333. https://doi.org/10.1007/s12017-020-08594-3
Nishihiro, Shingo, et al. "Correction to: High‑Mobility Group Box‑1‑Induced Angiogenesis After Indirect Bypass Surgery in a Chronic Cerebral Hypoperfusion Model." Neuromolecular medicine vol. 22,2 (2020): 332-333. doi: https://doi.org/10.1007/s12017-020-08594-3
Nishihiro S, Hishikawa T, Hiramatsu M, Kidani N, Takahashi Y, Murai S, Sugiu K, Higaki Y, Yasuhara T, Borlongan CV, Date I. Correction to: High‑Mobility Group Box‑1‑Induced Angiogenesis After Indirect Bypass Surgery in a Chronic Cerebral Hypoperfusion Model. Neuromolecular Med. 2020 Jun;22(2):332-333. doi: 10.1007/s12017-020-08594-3. PMID: 32103453; PMCID: PMC7645495.
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Showing 1 to 12 of 112 entries