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How to manage TB in children? Problems and solutions in four cases.

Open medicine (Warsaw, Poland)

Buonsenso D, Serranti D, Focarelli B, Chiaretti A, Calzedda R, Valentini P.
PMID: 28352679
Open Med (Wars). 2014 Nov 03;10(1):63-69. doi: 10.1515/med-2015-0012. eCollection 2015.

Children bear a substantial part of the tuberculosis (TB) epidemic worldwide, and it is estimated that there were ≅ 500.000 childhood TB cases globally in 2010, although accurate data are problematic to obtain given the many difficulties associated with...

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Buonsenso D, Serranti D, Focarelli B, et al. How to manage TB in children? Problems and solutions in four cases. Open Med (Wars). 2014;10(1):63-69doi: 10.1515/med-2015-0012.
Buonsenso, D., Serranti, D., Focarelli, B., Chiaretti, A., Calzedda, R., & Valentini, P. (2015). How to manage TB in children? Problems and solutions in four cases. Open medicine (Warsaw, Poland), 10(1), 63-69. https://doi.org/10.1515/med-2015-0012
Buonsenso, Danilo, et al. "How to manage TB in children? Problems and solutions in four cases." Open medicine (Warsaw, Poland) vol. 10,1 (2015): 63-69. doi: https://doi.org/10.1515/med-2015-0012
Buonsenso D, Serranti D, Focarelli B, Chiaretti A, Calzedda R, Valentini P. How to manage TB in children? Problems and solutions in four cases. Open Med (Wars). 2014 Nov 03;10(1):63-69. doi: 10.1515/med-2015-0012. eCollection 2015. PMID: 28352679; PMCID: PMC5152955.
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The GABAergic Hypothesis for Cognitive Disabilities in Down Syndrome.

Frontiers in cellular neuroscience

Contestabile A, Magara S, Cancedda L.
PMID: 28326014
Front Cell Neurosci. 2017 Mar 07;11:54. doi: 10.3389/fncel.2017.00054. eCollection 2017.

Down syndrome (DS) is a genetic disorder caused by the presence of a third copy of chromosome 21. DS affects multiple organs, but it invariably results in altered brain development and diverse degrees of intellectual disability. A large body...

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Contestabile A, Magara S, Cancedda L. The GABAergic Hypothesis for Cognitive Disabilities in Down Syndrome. Front Cell Neurosci. 2017;11:54doi: 10.3389/fncel.2017.00054.
Contestabile, A., Magara, S., & Cancedda, L. (2017). The GABAergic Hypothesis for Cognitive Disabilities in Down Syndrome. Frontiers in cellular neuroscience, 1154. https://doi.org/10.3389/fncel.2017.00054
Contestabile, Andrea, et al. "The GABAergic Hypothesis for Cognitive Disabilities in Down Syndrome." Frontiers in cellular neuroscience vol. 11 (2017): 54. doi: https://doi.org/10.3389/fncel.2017.00054
Contestabile A, Magara S, Cancedda L. The GABAergic Hypothesis for Cognitive Disabilities in Down Syndrome. Front Cell Neurosci. 2017 Mar 07;11:54. doi: 10.3389/fncel.2017.00054. eCollection 2017. PMID: 28326014; PMCID: PMC5339239.
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Static and dynamic properties of 1,1'-bi-2-naphthol and its conjugated acids and bases.

Chemistry (Weinheim an der Bergstrasse, Germany)

Alkorta I, Cancedda C, Cocinero EJ, Dávalos JZ, Ecija P, Elguero J, González J, Lesarri A, Ramos R, Reviriego F, Roussel C, Uriarte I, Vanthuyne N.
PMID: 25234135
Chemistry. 2014 Nov 03;20(45):14816-25. doi: 10.1002/chem.201402686. Epub 2014 Sep 18.

Several convergent techniques were used to characterize 1,1'-bi-2-naphthol (BINOL) and some of its properties. Its acidity in the gas-phase, from neutral species to monoanion, was measured by mass spectrometry. The conformation and structure of BINOL in the gas phase...

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Alkorta I, Cancedda C, Cocinero EJ, et al. Static and dynamic properties of 1,1'-bi-2-naphthol and its conjugated acids and bases. Chemistry. 2014;20(45):14816-25doi: 10.1002/chem.201402686.
Alkorta, I., Cancedda, C., Cocinero, E. J., Dávalos, J. Z., Ecija, P., Elguero, J., González, J., Lesarri, A., Ramos, R., Reviriego, F., Roussel, C., Uriarte, I., & Vanthuyne, N. (2014). Static and dynamic properties of 1,1'-bi-2-naphthol and its conjugated acids and bases. Chemistry (Weinheim an der Bergstrasse, Germany), 20(45), 14816-25. https://doi.org/10.1002/chem.201402686
Alkorta, Ibon, et al. "Static and dynamic properties of 1,1'-bi-2-naphthol and its conjugated acids and bases." Chemistry (Weinheim an der Bergstrasse, Germany) vol. 20,45 (2014): 14816-25. doi: https://doi.org/10.1002/chem.201402686
Alkorta I, Cancedda C, Cocinero EJ, Dávalos JZ, Ecija P, Elguero J, González J, Lesarri A, Ramos R, Reviriego F, Roussel C, Uriarte I, Vanthuyne N. Static and dynamic properties of 1,1'-bi-2-naphthol and its conjugated acids and bases. Chemistry. 2014 Nov 03;20(45):14816-25. doi: 10.1002/chem.201402686. Epub 2014 Sep 18. PMID: 25234135.
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Metformin inhibition of neuroblastoma cell proliferation is differently modulated by cell differentiation induced by retinoic acid or overexpression of NDM29 non-coding RNA.

Cancer cell international

Costa D, Gigoni A, Würth R, Cancedda R, Florio T, Pagano A.
PMID: 25120382
Cancer Cell Int. 2014 Jul 02;14:59. doi: 10.1186/1475-2867-14-59. eCollection 2014.

BACKGROUND: Metformin is a widely used oral hypoglycemizing agent recently proposed as potential anti-cancer drug. In this study we report the antiproliferative effect of metformin treatment in a high risk neuroblastoma cell model, focusing on possible effects associated to...

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Costa D, Gigoni A, Würth R, et al. Metformin inhibition of neuroblastoma cell proliferation is differently modulated by cell differentiation induced by retinoic acid or overexpression of NDM29 non-coding RNA. Cancer Cell Int. 2014;14:59doi: 10.1186/1475-2867-14-59.
Costa, D., Gigoni, A., Würth, R., Cancedda, R., Florio, T., & Pagano, A. (2014). Metformin inhibition of neuroblastoma cell proliferation is differently modulated by cell differentiation induced by retinoic acid or overexpression of NDM29 non-coding RNA. Cancer cell international, 1459. https://doi.org/10.1186/1475-2867-14-59
Costa, Delfina, et al. "Metformin inhibition of neuroblastoma cell proliferation is differently modulated by cell differentiation induced by retinoic acid or overexpression of NDM29 non-coding RNA." Cancer cell international vol. 14 (2014): 59. doi: https://doi.org/10.1186/1475-2867-14-59
Costa D, Gigoni A, Würth R, Cancedda R, Florio T, Pagano A. Metformin inhibition of neuroblastoma cell proliferation is differently modulated by cell differentiation induced by retinoic acid or overexpression of NDM29 non-coding RNA. Cancer Cell Int. 2014 Jul 02;14:59. doi: 10.1186/1475-2867-14-59. eCollection 2014. PMID: 25120382; PMCID: PMC4128937.
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Metabolism of human erythrocyte glucose-6-phosphate dehydrogenase. III - isolation of a protein involved in the 6-phosphogluconate-induced inactivation.

FEBS letters

Bonsignore A, De Flora A, Lorenzoni I, Mangiarotti MA, Cancedda R, Dina D.
PMID: 11945287
FEBS Lett. 1968 Aug;1(3):176-178. doi: 10.1016/0014-5793(68)80053-5.

No abstract available.

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Bonsignore A, De Flora A, Lorenzoni I, et al. Metabolism of human erythrocyte glucose-6-phosphate dehydrogenase. III - isolation of a protein involved in the 6-phosphogluconate-induced inactivation. FEBS Lett. 1968;1(3):176-178doi: 10.1016/0014-5793(68)80053-5.
Bonsignore, A., De Flora, A., Lorenzoni, I., Mangiarotti, M. A., Cancedda, R., & Dina, D. (1968). Metabolism of human erythrocyte glucose-6-phosphate dehydrogenase. III - isolation of a protein involved in the 6-phosphogluconate-induced inactivation. FEBS letters, 1(3), 176-178. https://doi.org/10.1016/0014-5793(68)80053-5
Bonsignore, A, et al. "Metabolism of human erythrocyte glucose-6-phosphate dehydrogenase. III - isolation of a protein involved in the 6-phosphogluconate-induced inactivation." FEBS letters vol. 1,3 (1968): 176-178. doi: https://doi.org/10.1016/0014-5793(68)80053-5
Bonsignore A, De Flora A, Lorenzoni I, Mangiarotti MA, Cancedda R, Dina D. Metabolism of human erythrocyte glucose-6-phosphate dehydrogenase. III - isolation of a protein involved in the 6-phosphogluconate-induced inactivation. FEBS Lett. 1968 Aug;1(3):176-178. doi: 10.1016/0014-5793(68)80053-5. PMID: 11945287.
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Expression of anchorin CII mRNA by cultured chondrocytes.

Cytotechnology

Castagnola P, Cancedda R.
PMID: 22358950
Cytotechnology. 1991 Feb;5:41-4. doi: 10.1007/BF00736804.

The establishment of a cell culture system promoting chondrocyte differentiation has been utilized to better characterize phenotypic stages of chondrogenesis at the cellular level. Although the expression of the type II collagen gene has been studied during "in vitro"...

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Castagnola P, Cancedda R. Expression of anchorin CII mRNA by cultured chondrocytes. Cytotechnology. 1991;5:41-4doi: 10.1007/BF00736804.
Castagnola, P., & Cancedda, R. (1991). Expression of anchorin CII mRNA by cultured chondrocytes. Cytotechnology, 541-4. https://doi.org/10.1007/BF00736804
Castagnola, P, and Cancedda, R. "Expression of anchorin CII mRNA by cultured chondrocytes." Cytotechnology vol. 5 (1991): 41-4. doi: https://doi.org/10.1007/BF00736804
Castagnola P, Cancedda R. Expression of anchorin CII mRNA by cultured chondrocytes. Cytotechnology. 1991 Feb;5:41-4. doi: 10.1007/BF00736804. PMID: 22358950.
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Learning from Mother Nature: Innovative Tools to Boost Endogenous Repair of Critical or Difficult-to-Heal Large Tissue Defects.

Frontiers in bioengineering and biotechnology

Cancedda R, Bollini S, Descalzi F, Mastrogiacomo M, Tasso R.
PMID: 28503549
Front Bioeng Biotechnol. 2017 Apr 28;5:28. doi: 10.3389/fbioe.2017.00028. eCollection 2017.

For repair of chronic or difficult-to-heal tissue lesions and defects, major constraints exist to a broad application of cell therapy and tissue engineering approaches, i.e., transplantation of "

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Cancedda R, Bollini S, Descalzi F, et al. Learning from Mother Nature: Innovative Tools to Boost Endogenous Repair of Critical or Difficult-to-Heal Large Tissue Defects. Front Bioeng Biotechnol. 2017;5:28doi: 10.3389/fbioe.2017.00028.
Cancedda, R., Bollini, S., Descalzi, F., Mastrogiacomo, M., & Tasso, R. (2017). Learning from Mother Nature: Innovative Tools to Boost Endogenous Repair of Critical or Difficult-to-Heal Large Tissue Defects. Frontiers in bioengineering and biotechnology, 528. https://doi.org/10.3389/fbioe.2017.00028
Cancedda, Ranieri, et al. "Learning from Mother Nature: Innovative Tools to Boost Endogenous Repair of Critical or Difficult-to-Heal Large Tissue Defects." Frontiers in bioengineering and biotechnology vol. 5 (2017): 28. doi: https://doi.org/10.3389/fbioe.2017.00028
Cancedda R, Bollini S, Descalzi F, Mastrogiacomo M, Tasso R. Learning from Mother Nature: Innovative Tools to Boost Endogenous Repair of Critical or Difficult-to-Heal Large Tissue Defects. Front Bioeng Biotechnol. 2017 Apr 28;5:28. doi: 10.3389/fbioe.2017.00028. eCollection 2017. PMID: 28503549; PMCID: PMC5408079.
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Gene Editing Preserves Visual Functions in a Mouse Model of Retinal Degeneration.

Frontiers in neuroscience

Vagni P, Perlini LE, Chenais NAL, Marchetti T, Parrini M, Contestabile A, Cancedda L, Ghezzi D.
PMID: 31551698
Front Neurosci. 2019 Sep 10;13:945. doi: 10.3389/fnins.2019.00945. eCollection 2019.

Inherited retinal dystrophies (IRDs) are a large and heterogeneous group of degenerative diseases caused by mutations in various genes. Given the favorable anatomical and immunological characteristics of the eye, gene therapy holds great potential for their treatment. Our goal...

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Vagni P, Perlini LE, Chenais NAL, et al. Gene Editing Preserves Visual Functions in a Mouse Model of Retinal Degeneration. Front Neurosci. 2019;13:945doi: 10.3389/fnins.2019.00945.
Vagni, P., Perlini, L. E., Chenais, N. A. L., Marchetti, T., Parrini, M., Contestabile, A., Cancedda, L., & Ghezzi, D. (2019). Gene Editing Preserves Visual Functions in a Mouse Model of Retinal Degeneration. Frontiers in neuroscience, 13945. https://doi.org/10.3389/fnins.2019.00945
Vagni, Paola, et al. "Gene Editing Preserves Visual Functions in a Mouse Model of Retinal Degeneration." Frontiers in neuroscience vol. 13 (2019): 945. doi: https://doi.org/10.3389/fnins.2019.00945
Vagni P, Perlini LE, Chenais NAL, Marchetti T, Parrini M, Contestabile A, Cancedda L, Ghezzi D. Gene Editing Preserves Visual Functions in a Mouse Model of Retinal Degeneration. Front Neurosci. 2019 Sep 10;13:945. doi: 10.3389/fnins.2019.00945. eCollection 2019. PMID: 31551698; PMCID: PMC6748340.
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Skin physiology in microgravity: a 3-month stay aboard ISS induces dermal atrophy and affects cutaneous muscle and hair follicles cycling in mice.

NPJ microgravity

Neutelings T, Nusgens BV, Liu Y, Tavella S, Ruggiu A, Cancedda R, Gabriel M, Colige A, Lambert C.
PMID: 28725708
NPJ Microgravity. 2015 May 27;1:15002. doi: 10.1038/npjmgrav.2015.2. eCollection 2015.

AIMS: The Mice Drawer System (MDS) Tissue Sharing program was the longest rodent space mission ever performed. It provided 20 research teams with organs and tissues collected from mice having spent 3 months on the International Space Station (ISS)....

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Neutelings T, Nusgens BV, Liu Y, et al. Skin physiology in microgravity: a 3-month stay aboard ISS induces dermal atrophy and affects cutaneous muscle and hair follicles cycling in mice. NPJ Microgravity. 2015;1:15002doi: 10.1038/npjmgrav.2015.2.
Neutelings, T., Nusgens, B. V., Liu, Y., Tavella, S., Ruggiu, A., Cancedda, R., Gabriel, M., Colige, A., & Lambert, C. (2015). Skin physiology in microgravity: a 3-month stay aboard ISS induces dermal atrophy and affects cutaneous muscle and hair follicles cycling in mice. NPJ microgravity, 115002. https://doi.org/10.1038/npjmgrav.2015.2
Neutelings, Thibaut, et al. "Skin physiology in microgravity: a 3-month stay aboard ISS induces dermal atrophy and affects cutaneous muscle and hair follicles cycling in mice." NPJ microgravity vol. 1 (2015): 15002. doi: https://doi.org/10.1038/npjmgrav.2015.2
Neutelings T, Nusgens BV, Liu Y, Tavella S, Ruggiu A, Cancedda R, Gabriel M, Colige A, Lambert C. Skin physiology in microgravity: a 3-month stay aboard ISS induces dermal atrophy and affects cutaneous muscle and hair follicles cycling in mice. NPJ Microgravity. 2015 May 27;1:15002. doi: 10.1038/npjmgrav.2015.2. eCollection 2015. PMID: 28725708; PMCID: PMC5515501.
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Growth Factors Delivery System for Skin Regeneration: An Advanced Wound Dressing.

Pharmaceutics

Nardini M, Perteghella S, Mastracci L, Grillo F, Marrubini G, Bari E, Formica M, Gentili C, Cancedda R, Torre ML, Mastrogiacomo M.
PMID: 32028579
Pharmaceutics. 2020 Feb 03;12(2). doi: 10.3390/pharmaceutics12020120.

Standard treatments of chronic skin ulcers based on the direct application of dressings still present several limits with regard to a complete tissue regeneration. Innovative strategies in tissue engineering offer materials that can tune cell behavior and promote growth...

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Nardini M, Perteghella S, Mastracci L, et al. Growth Factors Delivery System for Skin Regeneration: An Advanced Wound Dressing. Pharmaceutics. 2020;12(2)doi: 10.3390/pharmaceutics12020120.
Nardini, M., Perteghella, S., Mastracci, L., Grillo, F., Marrubini, G., Bari, E., Formica, M., Gentili, C., Cancedda, R., Torre, M. L., & Mastrogiacomo, M. (2020). Growth Factors Delivery System for Skin Regeneration: An Advanced Wound Dressing. Pharmaceutics, 12(2), . https://doi.org/10.3390/pharmaceutics12020120
Nardini, Marta, et al. "Growth Factors Delivery System for Skin Regeneration: An Advanced Wound Dressing." Pharmaceutics vol. 12,2 (2020). doi: https://doi.org/10.3390/pharmaceutics12020120
Nardini M, Perteghella S, Mastracci L, Grillo F, Marrubini G, Bari E, Formica M, Gentili C, Cancedda R, Torre ML, Mastrogiacomo M. Growth Factors Delivery System for Skin Regeneration: An Advanced Wound Dressing. Pharmaceutics. 2020 Feb 03;12(2). doi: 10.3390/pharmaceutics12020120. PMID: 32028579; PMCID: PMC7076531.
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It is time for academic institutions to align their strategies and priorities with the Sustainable Development Goals.

BMJ global health

Cancedda C, Binagwaho A, Kerry V.
PMID: 30613425
BMJ Glob Health. 2018 Dec 06;3(6):e001067. doi: 10.1136/bmjgh-2018-001067. eCollection 2018.

No abstract available.

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Cancedda C, Binagwaho A, Kerry V. It is time for academic institutions to align their strategies and priorities with the Sustainable Development Goals. BMJ Glob Health. 2018;3(6):e001067doi: 10.1136/bmjgh-2018-001067.
Cancedda, C., Binagwaho, A., & Kerry, V. (2018). It is time for academic institutions to align their strategies and priorities with the Sustainable Development Goals. BMJ global health, 3(6), e001067. https://doi.org/10.1136/bmjgh-2018-001067
Cancedda, Corrado, et al. "It is time for academic institutions to align their strategies and priorities with the Sustainable Development Goals." BMJ global health vol. 3,6 (2018): e001067. doi: https://doi.org/10.1136/bmjgh-2018-001067
Cancedda C, Binagwaho A, Kerry V. It is time for academic institutions to align their strategies and priorities with the Sustainable Development Goals. BMJ Glob Health. 2018 Dec 06;3(6):e001067. doi: 10.1136/bmjgh-2018-001067. eCollection 2018. PMID: 30613425; PMCID: PMC6304100.
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Adopting locally the lessons learned globally: a call to action for academic medical centres in the USA.

BMJ global health

Cancedda C, Adams LV, Powderly W.
PMID: 32399257
BMJ Glob Health. 2020 Apr 20;5(4):e002238. doi: 10.1136/bmjgh-2019-002238. eCollection 2020.

No abstract available.

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Cancedda C, Adams LV, Powderly W. Adopting locally the lessons learned globally: a call to action for academic medical centres in the USA. BMJ Glob Health. 2020;5(4):e002238doi: 10.1136/bmjgh-2019-002238.
Cancedda, C., Adams, L. V., & Powderly, W. (2020). Adopting locally the lessons learned globally: a call to action for academic medical centres in the USA. BMJ global health, 5(4), e002238. https://doi.org/10.1136/bmjgh-2019-002238
Cancedda, Corrado, et al. "Adopting locally the lessons learned globally: a call to action for academic medical centres in the USA." BMJ global health vol. 5,4 (2020): e002238. doi: https://doi.org/10.1136/bmjgh-2019-002238
Cancedda C, Adams LV, Powderly W. Adopting locally the lessons learned globally: a call to action for academic medical centres in the USA. BMJ Glob Health. 2020 Apr 20;5(4):e002238. doi: 10.1136/bmjgh-2019-002238. eCollection 2020. PMID: 32399257; PMCID: PMC7204932.
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Showing 1 to 12 of 45 entries