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Showing 1 to 12 of 97 entries
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Caspase-6 promotes activation of the caspase-11-NLRP3 inflammasome during gram-negative bacterial infections.

The Journal of biological chemistry

Zheng M, Karki R, Kancharana B, Berns H, Pruett-Miller SM, Kanneganti TD.
PMID: 34740613
J Biol Chem. 2021 Nov 02;297(6):101379. doi: 10.1016/j.jbc.2021.101379. Epub 2021 Nov 02.

The innate immune system acts as the first line of defense against infection. One key component of the innate immune response to gram-negative bacterial infections is inflammasome activation. The caspase-11 (CASP11)-nucleotide-binding oligomerization domain-like receptor pyrin domain-containing 3 (NLRP3) inflammasome...

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Zheng M, Karki R, Kancharana B, et al. Caspase-6 promotes activation of the caspase-11-NLRP3 inflammasome during gram-negative bacterial infections. J Biol Chem. 2021;297(6):101379doi: 10.1016/j.jbc.2021.101379.
Zheng, M., Karki, R., Kancharana, B., Berns, H., Pruett-Miller, S. M., & Kanneganti, T. D. (2021). Caspase-6 promotes activation of the caspase-11-NLRP3 inflammasome during gram-negative bacterial infections. The Journal of biological chemistry, 297(6), 101379. https://doi.org/10.1016/j.jbc.2021.101379
Zheng, Min, et al. "Caspase-6 promotes activation of the caspase-11-NLRP3 inflammasome during gram-negative bacterial infections." The Journal of biological chemistry vol. 297,6 (2021): 101379. doi: https://doi.org/10.1016/j.jbc.2021.101379
Zheng M, Karki R, Kancharana B, Berns H, Pruett-Miller SM, Kanneganti TD. Caspase-6 promotes activation of the caspase-11-NLRP3 inflammasome during gram-negative bacterial infections. J Biol Chem. 2021 Nov 02;297(6):101379. doi: 10.1016/j.jbc.2021.101379. Epub 2021 Nov 02. PMID: 34740613; PMCID: PMC8633687.
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Inflammasome-mediated GSDMD activation facilitates escape of Candida albicans from macrophages.

Nature communications

Ding X, Kambara H, Guo R, Kanneganti A, Acosta-Zaldívar M, Li J, Liu F, Bei T, Qi W, Xie X, Han W, Liu N, Zhang C, Zhang X, Yu H, Zhao L, Ma F, Köhler JR, Luo HR.
PMID: 34795266
Nat Commun. 2021 Nov 18;12(1):6699. doi: 10.1038/s41467-021-27034-9.

Candida albicans is the most common cause of fungal sepsis. Inhibition of inflammasome activity confers resistance to polymicrobial and LPS-induced sepsis; however, inflammasome signaling appears to protect against C. albicans infection, so inflammasome inhibitors are not clinically useful for...

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Ding X, Kambara H, Guo R, et al. Inflammasome-mediated GSDMD activation facilitates escape of Candida albicans from macrophages. Nat Commun. 2021;12(1):6699doi: 10.1038/s41467-021-27034-9.
Ding, X., Kambara, H., Guo, R., Kanneganti, A., Acosta-Zaldívar, M., Li, J., Liu, F., Bei, T., Qi, W., Xie, X., Han, W., Liu, N., Zhang, C., Zhang, X., Yu, H., Zhao, L., Ma, F., Köhler, J. R., & Luo, H. R. (2021). Inflammasome-mediated GSDMD activation facilitates escape of Candida albicans from macrophages. Nature communications, 12(1), 6699. https://doi.org/10.1038/s41467-021-27034-9
Ding, Xionghui, et al. "Inflammasome-mediated GSDMD activation facilitates escape of Candida albicans from macrophages." Nature communications vol. 12,1 (2021): 6699. doi: https://doi.org/10.1038/s41467-021-27034-9
Ding X, Kambara H, Guo R, Kanneganti A, Acosta-Zaldívar M, Li J, Liu F, Bei T, Qi W, Xie X, Han W, Liu N, Zhang C, Zhang X, Yu H, Zhao L, Ma F, Köhler JR, Luo HR. Inflammasome-mediated GSDMD activation facilitates escape of Candida albicans from macrophages. Nat Commun. 2021 Nov 18;12(1):6699. doi: 10.1038/s41467-021-27034-9. PMID: 34795266; PMCID: PMC8602704.
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TLR and IKK Complex-Mediated Innate Immune Signaling Inhibits Stress Granule Assembly.

Journal of immunology (Baltimore, Md. : 1950)

Samir P, Place DE, Malireddi RKS, Kanneganti TD.
PMID: 34145059
J Immunol. 2021 Jul 01;207(1):115-124. doi: 10.4049/jimmunol.2100115. Epub 2021 Jun 18.

Cellular stress can induce cytoplasmic ribonucleoprotein complexes called stress granules that allow the cells to survive. Stress granules are also central to cellular responses to infections, in which they can act as platforms for viral sensing or modulate innate...

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Samir P, Place DE, Malireddi RKS, et al. TLR and IKK Complex-Mediated Innate Immune Signaling Inhibits Stress Granule Assembly. J Immunol. 2021;207(1):115-124doi: 10.4049/jimmunol.2100115.
Samir, P., Place, D. E., Malireddi, R. K. S., & Kanneganti, T. D. (2021). TLR and IKK Complex-Mediated Innate Immune Signaling Inhibits Stress Granule Assembly. Journal of immunology (Baltimore, Md. : 1950), 207(1), 115-124. https://doi.org/10.4049/jimmunol.2100115
Samir, Parimal, et al. "TLR and IKK Complex-Mediated Innate Immune Signaling Inhibits Stress Granule Assembly." Journal of immunology (Baltimore, Md. : 1950) vol. 207,1 (2021): 115-124. doi: https://doi.org/10.4049/jimmunol.2100115
Samir P, Place DE, Malireddi RKS, Kanneganti TD. TLR and IKK Complex-Mediated Innate Immune Signaling Inhibits Stress Granule Assembly. J Immunol. 2021 Jul 01;207(1):115-124. doi: 10.4049/jimmunol.2100115. Epub 2021 Jun 18. PMID: 34145059.
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ADAR1 restricts ZBP1-mediated immune response and PANoptosis to promote tumorigenesis.

Cell reports

Karki R, Sundaram B, Sharma BR, Lee S, Malireddi RKS, Nguyen LN, Christgen S, Zheng M, Wang Y, Samir P, Neale G, Vogel P, Kanneganti TD.
PMID: 34686350
Cell Rep. 2021 Oct 19;37(3):109858. doi: 10.1016/j.celrep.2021.109858.

Cell death provides host defense and maintains homeostasis. Zα-containing molecules are essential for these processes. Z-DNA binding protein 1 (ZBP1) activates inflammatory cell death, PANoptosis, whereas adenosine deaminase acting on RNA 1 (ADAR1) serves as an RNA editor to...

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Karki R, Sundaram B, Sharma BR, et al. ADAR1 restricts ZBP1-mediated immune response and PANoptosis to promote tumorigenesis. Cell Rep. 2021;37(3):109858doi: 10.1016/j.celrep.2021.109858.
Karki, R., Sundaram, B., Sharma, B. R., Lee, S., Malireddi, R. K. S., Nguyen, L. N., Christgen, S., Zheng, M., Wang, Y., Samir, P., Neale, G., Vogel, P., & Kanneganti, T. D. (2021). ADAR1 restricts ZBP1-mediated immune response and PANoptosis to promote tumorigenesis. Cell reports, 37(3), 109858. https://doi.org/10.1016/j.celrep.2021.109858
Karki, Rajendra, et al. "ADAR1 restricts ZBP1-mediated immune response and PANoptosis to promote tumorigenesis." Cell reports vol. 37,3 (2021): 109858. doi: https://doi.org/10.1016/j.celrep.2021.109858
Karki R, Sundaram B, Sharma BR, Lee S, Malireddi RKS, Nguyen LN, Christgen S, Zheng M, Wang Y, Samir P, Neale G, Vogel P, Kanneganti TD. ADAR1 restricts ZBP1-mediated immune response and PANoptosis to promote tumorigenesis. Cell Rep. 2021 Oct 19;37(3):109858. doi: 10.1016/j.celrep.2021.109858. PMID: 34686350.
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Clinical and Laboratory Markers of Brain Abscess in Tetralogy of Fallot ('BA-TOF' Score): Results of a Case-Control Study and Implications for Community Surveillance.

Journal of neurosciences in rural practice

Kanneganti V, Thakar S, Aryan S, Kini P, Mohan D, Hegde AS.
PMID: 33935447
J Neurosci Rural Pract. 2021 Apr;12(2):302-307. doi: 10.1055/s-0041-1722819. Epub 2021 Mar 15.

No abstract available.

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Kanneganti V, Thakar S, Aryan S, et al. Clinical and Laboratory Markers of Brain Abscess in Tetralogy of Fallot ('BA-TOF' Score): Results of a Case-Control Study and Implications for Community Surveillance. J Neurosci Rural Pract. 2021;12(2):302-307doi: 10.1055/s-0041-1722819.
Kanneganti, V., Thakar, S., Aryan, S., Kini, P., Mohan, D., & Hegde, A. S. (2021). Clinical and Laboratory Markers of Brain Abscess in Tetralogy of Fallot ('BA-TOF' Score): Results of a Case-Control Study and Implications for Community Surveillance. Journal of neurosciences in rural practice, 12(2), 302-307. https://doi.org/10.1055/s-0041-1722819
Kanneganti, Vidyasagar, et al. "Clinical and Laboratory Markers of Brain Abscess in Tetralogy of Fallot ('BA-TOF' Score): Results of a Case-Control Study and Implications for Community Surveillance." Journal of neurosciences in rural practice vol. 12,2 (2021): 302-307. doi: https://doi.org/10.1055/s-0041-1722819
Kanneganti V, Thakar S, Aryan S, Kini P, Mohan D, Hegde AS. Clinical and Laboratory Markers of Brain Abscess in Tetralogy of Fallot ('BA-TOF' Score): Results of a Case-Control Study and Implications for Community Surveillance. J Neurosci Rural Pract. 2021 Apr;12(2):302-307. doi: 10.1055/s-0041-1722819. Epub 2021 Mar 15. PMID: 33935447; PMCID: PMC8079174.
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Decoding the vital segments in human ATP-dependent RNA helicase.

Bioinformation

Kamjula V, Kanneganti A, Metla R, Nidamanuri K, Idupulapati S, Runthala A.
PMID: 32405168
Bioinformation. 2020 Feb 29;16(2):160-170. doi: 10.6026/97320630016160. eCollection 2020.

An analysis of the ATP-dependent RNA helicase using known functionally close analogs helps disclose the structural and functional information of the enzyme. The enzyme plays several interlinked biological functions and there is an urgent need to interpret its key...

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Kamjula V, Kanneganti A, Metla R, et al. Decoding the vital segments in human ATP-dependent RNA helicase. Bioinformation. 2020;16(2):160-170doi: 10.6026/97320630016160.
Kamjula, V., Kanneganti, A., Metla, R., Nidamanuri, K., Idupulapati, S., & Runthala, A. (2020). Decoding the vital segments in human ATP-dependent RNA helicase. Bioinformation, 16(2), 160-170. https://doi.org/10.6026/97320630016160
Kamjula, Vandana, et al. "Decoding the vital segments in human ATP-dependent RNA helicase." Bioinformation vol. 16,2 (2020): 160-170. doi: https://doi.org/10.6026/97320630016160
Kamjula V, Kanneganti A, Metla R, Nidamanuri K, Idupulapati S, Runthala A. Decoding the vital segments in human ATP-dependent RNA helicase. Bioinformation. 2020 Feb 29;16(2):160-170. doi: 10.6026/97320630016160. eCollection 2020. PMID: 32405168; PMCID: PMC7196165.
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Fostering experimental and computational synergy to modulate hyperinflammation.

Trends in immunology

Potapov I, Kanneganti TD, Del Sol A.
PMID: 34844849
Trends Immunol. 2022 Jan;43(1):4-7. doi: 10.1016/j.it.2021.11.004. Epub 2021 Nov 26.

The molecular underpinnings of the uncontrolled release of proinflammatory cytokines and chemokines ('cytokine storm'), which can cause organ damage and even mortality, are not completely understood. Furthermore, targeted therapeutic options to dampen such hyperinflammation are scarce. Here, we highlight...

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Potapov I, Kanneganti TD, Del Sol A. Fostering experimental and computational synergy to modulate hyperinflammation. Trends Immunol. 2021;43(1):4-7doi: 10.1016/j.it.2021.11.004.
Potapov, I., Kanneganti, T. D., & Del Sol, A. (2022). Fostering experimental and computational synergy to modulate hyperinflammation. Trends in immunology, 43(1), 4-7. https://doi.org/10.1016/j.it.2021.11.004
Potapov, Ilya, et al. "Fostering experimental and computational synergy to modulate hyperinflammation." Trends in immunology vol. 43,1 (2022): 4-7. doi: https://doi.org/10.1016/j.it.2021.11.004
Potapov I, Kanneganti TD, Del Sol A. Fostering experimental and computational synergy to modulate hyperinflammation. Trends Immunol. 2022 Jan;43(1):4-7. doi: 10.1016/j.it.2021.11.004. Epub 2021 Nov 26. PMID: 34844849.
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Synergism of TNF-α and IFN-γ triggers inflammatory cell death, tissue damage, and mortality in SARS-CoV-2 infection and cytokine shock syndromes.

bioRxiv : the preprint server for biology

Karki R, Sharma BR, Tuladhar S, Williams EP, Zalduondo L, Samir P, Zheng M, Sundaram B, Banoth B, Malireddi RKS, Schreiner P, Neale G, Vogel P, Webby R, Jonsson CB, Kanneganti TD.
PMID: 33140051
bioRxiv. 2020 Nov 13; doi: 10.1101/2020.10.29.361048.

The COVID-19 pandemic has caused significant morbidity and mortality. Currently, there is a critical shortage of proven treatment options and an urgent need to understand the pathogenesis of multi-organ failure and lung damage. Cytokine storm is associated with severe...

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Karki R, Sharma BR, Tuladhar S, et al. Synergism of TNF-α and IFN-γ triggers inflammatory cell death, tissue damage, and mortality in SARS-CoV-2 infection and cytokine shock syndromes. bioRxiv. 2020;doi: 10.1101/2020.10.29.361048.
Karki, R., Sharma, B. R., Tuladhar, S., Williams, E. P., Zalduondo, L., Samir, P., Zheng, M., Sundaram, B., Banoth, B., Malireddi, R. K. S., Schreiner, P., Neale, G., Vogel, P., Webby, R., Jonsson, C. B., & Kanneganti, T. D. (2020). Synergism of TNF-α and IFN-γ triggers inflammatory cell death, tissue damage, and mortality in SARS-CoV-2 infection and cytokine shock syndromes. bioRxiv : the preprint server for biology, . https://doi.org/10.1101/2020.10.29.361048
Karki, Rajendra, et al. "Synergism of TNF-α and IFN-γ triggers inflammatory cell death, tissue damage, and mortality in SARS-CoV-2 infection and cytokine shock syndromes." bioRxiv : the preprint server for biology vol. (2020). doi: https://doi.org/10.1101/2020.10.29.361048
Karki R, Sharma BR, Tuladhar S, Williams EP, Zalduondo L, Samir P, Zheng M, Sundaram B, Banoth B, Malireddi RKS, Schreiner P, Neale G, Vogel P, Webby R, Jonsson CB, Kanneganti TD. Synergism of TNF-α and IFN-γ triggers inflammatory cell death, tissue damage, and mortality in SARS-CoV-2 infection and cytokine shock syndromes. bioRxiv. 2020 Nov 13; doi: 10.1101/2020.10.29.361048. PMID: 33140051; PMCID: PMC7605562.
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Assessment of sleep quality and its predictors among newly diagnosed psychiatric patients.

Journal of basic and clinical physiology and pharmacology

Singh H, Shreyash G, Ramappa SA, Kanneganti SP, Subeesh V.
PMID: 33866696
J Basic Clin Physiol Pharmacol. 2021 Apr 19; doi: 10.1515/jbcpp-2020-0319. Epub 2021 Apr 19.

OBJECTIVES: Poor sleep is a vital symptom observed in many psychiatric conditions and is the most neglected and underdiagnosed. The current study aims at assessment of sleep quality among psychiatric patients using the Pittsburgh Sleep Quality Index (PSQI) scale...

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Singh H, Shreyash G, Ramappa SA, et al. Assessment of sleep quality and its predictors among newly diagnosed psychiatric patients. J Basic Clin Physiol Pharmacol. 2021;doi: 10.1515/jbcpp-2020-0319.
Singh, H., Shreyash, G., Ramappa, S. A., Kanneganti, S. P., & Subeesh, V. (2021). Assessment of sleep quality and its predictors among newly diagnosed psychiatric patients. Journal of basic and clinical physiology and pharmacology, . https://doi.org/10.1515/jbcpp-2020-0319
Singh, Hemendra, et al. "Assessment of sleep quality and its predictors among newly diagnosed psychiatric patients." Journal of basic and clinical physiology and pharmacology vol. (2021). doi: https://doi.org/10.1515/jbcpp-2020-0319
Singh H, Shreyash G, Ramappa SA, Kanneganti SP, Subeesh V. Assessment of sleep quality and its predictors among newly diagnosed psychiatric patients. J Basic Clin Physiol Pharmacol. 2021 Apr 19; doi: 10.1515/jbcpp-2020-0319. Epub 2021 Apr 19. PMID: 33866696.
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Are Two-Dimensional Morphometric Measures Reflective of Disease Severity in Adult Chiari I Malformation?.

World neurosurgery

Thakar S, Kanneganti V, Talla Nwotchouang BS, Salem SJ, Eppelheimer M, Loth F, Allen PA, Aryan S, Hegde AS.
PMID: 34710575
World Neurosurg. 2022 Jan;157:e497-e505. doi: 10.1016/j.wneu.2021.10.138. Epub 2021 Oct 25.

BACKGROUND: Two-dimensional (2D) morphometric measures have been previously used to diagnose Chiari I malformation (CMI) and distinguish patients from healthy subjects. There is, however, a paucity of literature regarding whether morphometric differences exist across the range of CMI disease...

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Thakar S, Kanneganti V, Talla Nwotchouang BS, et al. Are Two-Dimensional Morphometric Measures Reflective of Disease Severity in Adult Chiari I Malformation?. World Neurosurg. 2021;157:e497-e505doi: 10.1016/j.wneu.2021.10.138.
Thakar, S., Kanneganti, V., Talla Nwotchouang, B. S., Salem, S. J., Eppelheimer, M., Loth, F., Allen, P. A., Aryan, S., & Hegde, A. S. (2022). Are Two-Dimensional Morphometric Measures Reflective of Disease Severity in Adult Chiari I Malformation?. World neurosurgery, 157e497-e505. https://doi.org/10.1016/j.wneu.2021.10.138
Thakar, Sumit, et al. "Are Two-Dimensional Morphometric Measures Reflective of Disease Severity in Adult Chiari I Malformation?." World neurosurgery vol. 157 (2022): e497-e505. doi: https://doi.org/10.1016/j.wneu.2021.10.138
Thakar S, Kanneganti V, Talla Nwotchouang BS, Salem SJ, Eppelheimer M, Loth F, Allen PA, Aryan S, Hegde AS. Are Two-Dimensional Morphometric Measures Reflective of Disease Severity in Adult Chiari I Malformation?. World Neurosurg. 2022 Jan;157:e497-e505. doi: 10.1016/j.wneu.2021.10.138. Epub 2021 Oct 25. PMID: 34710575.
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Intestinal tête-à-tête: helminths blunt immunity against flaviviruses.

Cell research

Place DE, Kanneganti TD.
PMID: 33846568
Cell Res. 2021 Jul;31(7):723-724. doi: 10.1038/s41422-021-00505-w.

No abstract available.

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Place DE, Kanneganti TD. Intestinal tête-à-tête: helminths blunt immunity against flaviviruses. Cell Res. 2021;31(7):723-724doi: 10.1038/s41422-021-00505-w.
Place, D. E., & Kanneganti, T. D. (2021). Intestinal tête-à-tête: helminths blunt immunity against flaviviruses. Cell research, 31(7), 723-724. https://doi.org/10.1038/s41422-021-00505-w
Place, David E, and Kanneganti, Thirumala-Devi. "Intestinal tête-à-tête: helminths blunt immunity against flaviviruses." Cell research vol. 31,7 (2021): 723-724. doi: https://doi.org/10.1038/s41422-021-00505-w
Place DE, Kanneganti TD. Intestinal tête-à-tête: helminths blunt immunity against flaviviruses. Cell Res. 2021 Jul;31(7):723-724. doi: 10.1038/s41422-021-00505-w. PMID: 33846568; PMCID: PMC8249402.
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Comparative effect of implant-abutment connections, abutment angulations, and screw lengths on preloaded abutment screw using three-dimensional finite element analysis: An .

Journal of Indian Prosthodontic Society

Kanneganti KC, Vinnakota DN, Pottem SR, Pulagam M.
PMID: 29692570
J Indian Prosthodont Soc. 2018 Apr-Jun;18(2):161-167. doi: 10.4103/jips.jips_219_17.

PURPOSE: The purpose of this study is to compare the effect of implant-abutment connections, abutment angulations, and screw lengths on screw loosening (SL) of preloaded abutment using three dimensional (3D) finite element analysis.MATERIALS AND METHODS: 3D models of implants...

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Kanneganti KC, Vinnakota DN, Pottem SR, et al. Comparative effect of implant-abutment connections, abutment angulations, and screw lengths on preloaded abutment screw using three-dimensional finite element analysis: An . J Indian Prosthodont Soc. 2018;18(2):161-167doi: 10.4103/jips.jips_219_17.
Kanneganti, K. C., Vinnakota, D. N., Pottem, S. R., & Pulagam, M. (2018). Comparative effect of implant-abutment connections, abutment angulations, and screw lengths on preloaded abutment screw using three-dimensional finite element analysis: An . Journal of Indian Prosthodontic Society, 18(2), 161-167. https://doi.org/10.4103/jips.jips_219_17
Kanneganti, Krishna Chaitanya, et al. "Comparative effect of implant-abutment connections, abutment angulations, and screw lengths on preloaded abutment screw using three-dimensional finite element analysis: An ." Journal of Indian Prosthodontic Society vol. 18,2 (2018): 161-167. doi: https://doi.org/10.4103/jips.jips_219_17
Kanneganti KC, Vinnakota DN, Pottem SR, Pulagam M. Comparative effect of implant-abutment connections, abutment angulations, and screw lengths on preloaded abutment screw using three-dimensional finite element analysis: An . J Indian Prosthodont Soc. 2018 Apr-Jun;18(2):161-167. doi: 10.4103/jips.jips_219_17. PMID: 29692570; PMCID: PMC5903180.
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Showing 1 to 12 of 97 entries