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Transient Receptor Potential Ankyrin 1 Mediates Hypoxic Responses in Mice.

Frontiers in physiology

Chen S, Takahashi N, Chen C, Pauli JL, Kuroki C, Kaminosono J, Kashiwadani H, Kanmura Y, Mori Y, Ou S, Hao L, Kuwaki T.
PMID: 33192579
Front Physiol. 2020 Oct 22;11:576209. doi: 10.3389/fphys.2020.576209. eCollection 2020.

Transient receptor potential ankyrin 1 (TRPA1) is a non-selective cation channel that is broadly expressed in sensory pathways, such as the trigeminal and vagus nerves. It is capable of detecting various irritants in inspired gasses and is activated during...

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Chen S, Takahashi N, Chen C, et al. Transient Receptor Potential Ankyrin 1 Mediates Hypoxic Responses in Mice. Front Physiol. 2020;11:576209doi: 10.3389/fphys.2020.576209.
Chen, S., Takahashi, N., Chen, C., Pauli, J. L., Kuroki, C., Kaminosono, J., Kashiwadani, H., Kanmura, Y., Mori, Y., Ou, S., Hao, L., & Kuwaki, T. (2020). Transient Receptor Potential Ankyrin 1 Mediates Hypoxic Responses in Mice. Frontiers in physiology, 11576209. https://doi.org/10.3389/fphys.2020.576209
Chen, Sichong, et al. "Transient Receptor Potential Ankyrin 1 Mediates Hypoxic Responses in Mice." Frontiers in physiology vol. 11 (2020): 576209. doi: https://doi.org/10.3389/fphys.2020.576209
Chen S, Takahashi N, Chen C, Pauli JL, Kuroki C, Kaminosono J, Kashiwadani H, Kanmura Y, Mori Y, Ou S, Hao L, Kuwaki T. Transient Receptor Potential Ankyrin 1 Mediates Hypoxic Responses in Mice. Front Physiol. 2020 Oct 22;11:576209. doi: 10.3389/fphys.2020.576209. eCollection 2020. PMID: 33192579; PMCID: PMC7642990.
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ST2 gene products critically contribute to cellular transformation caused by an oncogenic Ras mutant.

Heliyon

Tago K, Ohta S, Kashiwada M, Funakoshi-Tago M, Matsugi J, Tominaga SI, Yanagisawa K.
PMID: 29226265
Heliyon. 2017 Oct 31;3(10):e00436. doi: 10.1016/j.heliyon.2017.e00436. eCollection 2017 Oct.

The ST2 gene was originally identified as a primary responsive gene, and the expressions of its gene products are induced by stimulation with growth factors and by oncogenic stresses. In this study, we observed that oncogenic Ras mutant induced...

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Tago K, Ohta S, Kashiwada M, et al. ST2 gene products critically contribute to cellular transformation caused by an oncogenic Ras mutant. Heliyon. 2017;3(10):e00436doi: 10.1016/j.heliyon.2017.e00436.
Tago, K., Ohta, S., Kashiwada, M., Funakoshi-Tago, M., Matsugi, J., Tominaga, S. I., & Yanagisawa, K. (2017). ST2 gene products critically contribute to cellular transformation caused by an oncogenic Ras mutant. Heliyon, 3(10), e00436. https://doi.org/10.1016/j.heliyon.2017.e00436
Tago, Kenji, et al. "ST2 gene products critically contribute to cellular transformation caused by an oncogenic Ras mutant." Heliyon vol. 3,10 (2017): e00436. doi: https://doi.org/10.1016/j.heliyon.2017.e00436
Tago K, Ohta S, Kashiwada M, Funakoshi-Tago M, Matsugi J, Tominaga SI, Yanagisawa K. ST2 gene products critically contribute to cellular transformation caused by an oncogenic Ras mutant. Heliyon. 2017 Oct 31;3(10):e00436. doi: 10.1016/j.heliyon.2017.e00436. eCollection 2017 Oct. PMID: 29226265; PMCID: PMC5714553.
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Linalool Odor-Induced Anxiolytic Effects in Mice.

Frontiers in behavioral neuroscience

Harada H, Kashiwadani H, Kanmura Y, Kuwaki T.
PMID: 30405369
Front Behav Neurosci. 2018 Oct 23;12:241. doi: 10.3389/fnbeh.2018.00241. eCollection 2018.

In folk medicine, it has long been believed that odorous compounds derived from plant extracts can have anxiolytic effects. Among them, linalool, one of the terpene alcohols in lavender extracts, has been reported to have the anxiolytic effects. However,...

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Harada H, Kashiwadani H, Kanmura Y, et al. Linalool Odor-Induced Anxiolytic Effects in Mice. Front Behav Neurosci. 2018;12:241doi: 10.3389/fnbeh.2018.00241.
Harada, H., Kashiwadani, H., Kanmura, Y., & Kuwaki, T. (2018). Linalool Odor-Induced Anxiolytic Effects in Mice. Frontiers in behavioral neuroscience, 12241. https://doi.org/10.3389/fnbeh.2018.00241
Harada, Hiroki, et al. "Linalool Odor-Induced Anxiolytic Effects in Mice." Frontiers in behavioral neuroscience vol. 12 (2018): 241. doi: https://doi.org/10.3389/fnbeh.2018.00241
Harada H, Kashiwadani H, Kanmura Y, Kuwaki T. Linalool Odor-Induced Anxiolytic Effects in Mice. Front Behav Neurosci. 2018 Oct 23;12:241. doi: 10.3389/fnbeh.2018.00241. eCollection 2018. PMID: 30405369; PMCID: PMC6206409.
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Linalool odor-induced analgesia is triggered by TRPA1-independent pathway in mice.

Behavioral and brain functions : BBF

Kashiwadani H, Higa Y, Sugimura M, Kuwaki T.
PMID: 33902628
Behav Brain Funct. 2021 Apr 26;17(1):3. doi: 10.1186/s12993-021-00176-y.

We had recently reported that linalool odor exposure induced significant analgesic effects in mice and that the effects were disappeared in olfactory-deprived mice in which the olfactory epithelium was damaged, thus indicating that the effects were triggered by chemical...

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Kashiwadani H, Higa Y, Sugimura M, et al. Linalool odor-induced analgesia is triggered by TRPA1-independent pathway in mice. Behav Brain Funct. 2021;17(1):3doi: 10.1186/s12993-021-00176-y.
Kashiwadani, H., Higa, Y., Sugimura, M., & Kuwaki, T. (2021). Linalool odor-induced analgesia is triggered by TRPA1-independent pathway in mice. Behavioral and brain functions : BBF, 17(1), 3. https://doi.org/10.1186/s12993-021-00176-y
Kashiwadani, Hideki, et al. "Linalool odor-induced analgesia is triggered by TRPA1-independent pathway in mice." Behavioral and brain functions : BBF vol. 17,1 (2021): 3. doi: https://doi.org/10.1186/s12993-021-00176-y
Kashiwadani H, Higa Y, Sugimura M, Kuwaki T. Linalool odor-induced analgesia is triggered by TRPA1-independent pathway in mice. Behav Brain Funct. 2021 Apr 26;17(1):3. doi: 10.1186/s12993-021-00176-y. PMID: 33902628; PMCID: PMC8077846.
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Longitudinal changes and body composition assessment using bioelectrical impedance in elderly patients with mild disequilibrium and different care needs.

Journal of physical therapy science

Goto K, Yamamoto T, Kashiwazaki M, Miura K, Hayashi S, Kubo A.
PMID: 30568337
J Phys Ther Sci. 2018 Dec;30(12):1473-1476. doi: 10.1589/jpts.30.1473. Epub 2018 Dec 03.

[Purpose] This study involved performing longitudinal measurements of muscle mass in elderly patients with mild disequilibrium using a body composition meter. The rate of change and characteristics were determined according to the level of care needed. [Participants and Methods]...

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Goto K, Yamamoto T, Kashiwazaki M, et al. Longitudinal changes and body composition assessment using bioelectrical impedance in elderly patients with mild disequilibrium and different care needs. J Phys Ther Sci. 2018;30(12):1473-1476doi: 10.1589/jpts.30.1473.
Goto, K., Yamamoto, T., Kashiwazaki, M., Miura, K., Hayashi, S., & Kubo, A. (2018). Longitudinal changes and body composition assessment using bioelectrical impedance in elderly patients with mild disequilibrium and different care needs. Journal of physical therapy science, 30(12), 1473-1476. https://doi.org/10.1589/jpts.30.1473
Goto, Kazuya, et al. "Longitudinal changes and body composition assessment using bioelectrical impedance in elderly patients with mild disequilibrium and different care needs." Journal of physical therapy science vol. 30,12 (2018): 1473-1476. doi: https://doi.org/10.1589/jpts.30.1473
Goto K, Yamamoto T, Kashiwazaki M, Miura K, Hayashi S, Kubo A. Longitudinal changes and body composition assessment using bioelectrical impedance in elderly patients with mild disequilibrium and different care needs. J Phys Ther Sci. 2018 Dec;30(12):1473-1476. doi: 10.1589/jpts.30.1473. Epub 2018 Dec 03. PMID: 30568337; PMCID: PMC6279705.
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Metformin treatment ameliorates diabetes-associated decline in hippocampal neurogenesis and memory via phosphorylation of insulin receptor substrate 1.

FEBS open bio

Tanokashira D, Kurata E, Fukuokaya W, Kawabe K, Kashiwada M, Takeuchi H, Nakazato M, Taguchi A.
PMID: 29988567
FEBS Open Bio. 2018 May 18;8(7):1104-1118. doi: 10.1002/2211-5463.12436. eCollection 2018 Jul.

Age-related reduction in adult hippocampal neurogenesis is correlated with cognitive impairment. Diabetes is a chronic systemic disease that negatively affects adult neural stem cells and memory functions in the hippocampus. Despite growing concern regarding the potential role of diabetic...

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Tanokashira D, Kurata E, Fukuokaya W, et al. Metformin treatment ameliorates diabetes-associated decline in hippocampal neurogenesis and memory via phosphorylation of insulin receptor substrate 1. FEBS Open Bio. 2018;8(7):1104-1118doi: 10.1002/2211-5463.12436.
Tanokashira, D., Kurata, E., Fukuokaya, W., Kawabe, K., Kashiwada, M., Takeuchi, H., Nakazato, M., & Taguchi, A. (2018). Metformin treatment ameliorates diabetes-associated decline in hippocampal neurogenesis and memory via phosphorylation of insulin receptor substrate 1. FEBS open bio, 8(7), 1104-1118. https://doi.org/10.1002/2211-5463.12436
Tanokashira, Daisuke, et al. "Metformin treatment ameliorates diabetes-associated decline in hippocampal neurogenesis and memory via phosphorylation of insulin receptor substrate 1." FEBS open bio vol. 8,7 (2018): 1104-1118. doi: https://doi.org/10.1002/2211-5463.12436
Tanokashira D, Kurata E, Fukuokaya W, Kawabe K, Kashiwada M, Takeuchi H, Nakazato M, Taguchi A. Metformin treatment ameliorates diabetes-associated decline in hippocampal neurogenesis and memory via phosphorylation of insulin receptor substrate 1. FEBS Open Bio. 2018 May 18;8(7):1104-1118. doi: 10.1002/2211-5463.12436. eCollection 2018 Jul. PMID: 29988567; PMCID: PMC6026705.
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Showing 1 to 6 of 6 entries