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MicroRNA-302b targets Mcl-1 and inhibits cell proliferation and induces apoptosis in malignant pleural mesothelioma cells.

American journal of cancer research

Khodayari N, Mohammed KA, Lee H, Kaye F, Nasreen N.
PMID: 27725905
Am J Cancer Res. 2016 Sep 01;6(9):1996-2009. eCollection 2016.

MicroRNAs belonging to the miR-302 family are emerging as key players in the control of cell growth, and maintaining pluripotency during cell fate determination and differentiation in embryonic stem cells. However, the mechanisms whereby ephA2/ephirnA1 signaling regulates miR-302b expression...

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Khodayari N, Mohammed KA, Lee H, et al. MicroRNA-302b targets Mcl-1 and inhibits cell proliferation and induces apoptosis in malignant pleural mesothelioma cells. Am J Cancer Res. 2016;6(9):1996-2009
Khodayari, N., Mohammed, K. A., Lee, H., Kaye, F., & Nasreen, N. (2016). MicroRNA-302b targets Mcl-1 and inhibits cell proliferation and induces apoptosis in malignant pleural mesothelioma cells. American journal of cancer research, 6(9), 1996-2009.
Khodayari, Nazli, et al. "MicroRNA-302b targets Mcl-1 and inhibits cell proliferation and induces apoptosis in malignant pleural mesothelioma cells." American journal of cancer research vol. 6,9 (2016): 1996-2009.
Khodayari N, Mohammed KA, Lee H, Kaye F, Nasreen N. MicroRNA-302b targets Mcl-1 and inhibits cell proliferation and induces apoptosis in malignant pleural mesothelioma cells. Am J Cancer Res. 2016 Sep 01;6(9):1996-2009. eCollection 2016. PMID: 27725905; PMCID: PMC5043109.
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Effective Tobacco Cessation via Health Coaching: An Institutional Case Report.

Global advances in health and medicine

Sforzo GA, Kaye M, Ayers GD, Talbert B, Hill M.
PMID: 25568823
Glob Adv Health Med. 2014 Sep;3(5):37-44. doi: 10.7453/gahmj.2014.029.

BACKGROUND: Tobacco abuse is a well-recognized scourge on health and healthcare costs. Attempts to facilitate tobacco cessation are rarely better than marginally effective.PRIMARY OBJECTIVE: To describe an observational trial of an existing and highly successful tobacco cessation program featuring...

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Sforzo GA, Kaye M, Ayers GD, et al. Effective Tobacco Cessation via Health Coaching: An Institutional Case Report. Glob Adv Health Med. 2014;3(5):37-44doi: 10.7453/gahmj.2014.029.
Sforzo, G. A., Kaye, M., Ayers, G. D., Talbert, B., & Hill, M. (2014). Effective Tobacco Cessation via Health Coaching: An Institutional Case Report. Global advances in health and medicine, 3(5), 37-44. https://doi.org/10.7453/gahmj.2014.029
Sforzo, Gary A, et al. "Effective Tobacco Cessation via Health Coaching: An Institutional Case Report." Global advances in health and medicine vol. 3,5 (2014): 37-44. doi: https://doi.org/10.7453/gahmj.2014.029
Sforzo GA, Kaye M, Ayers GD, Talbert B, Hill M. Effective Tobacco Cessation via Health Coaching: An Institutional Case Report. Glob Adv Health Med. 2014 Sep;3(5):37-44. doi: 10.7453/gahmj.2014.029. PMID: 25568823; PMCID: PMC4268607.
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The effect of performing a dual task on postural control in children with autism.

ISRN neuroscience

Bucci MP, Doyen C, Contenjean Y, Kaye K.
PMID: 24959567
ISRN Neurosci. 2013 Oct 30;2013:796174. doi: 10.1155/2013/796174. eCollection 2013.

The aim of the study was to explore the effect of eye movements (saccades and pursuits) on postural stability in children with autism versus typically developing children of comparable age. Postural stability was recorded with a platform (Techno Concept)...

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Bucci MP, Doyen C, Contenjean Y, et al. The effect of performing a dual task on postural control in children with autism. ISRN Neurosci. 2013;2013:796174doi: 10.1155/2013/796174.
Bucci, M. P., Doyen, C., Contenjean, Y., & Kaye, K. (2013). The effect of performing a dual task on postural control in children with autism. ISRN neuroscience, 2013796174. https://doi.org/10.1155/2013/796174
Bucci, Maria Pia, et al. "The effect of performing a dual task on postural control in children with autism." ISRN neuroscience vol. 2013 (2013): 796174. doi: https://doi.org/10.1155/2013/796174
Bucci MP, Doyen C, Contenjean Y, Kaye K. The effect of performing a dual task on postural control in children with autism. ISRN Neurosci. 2013 Oct 30;2013:796174. doi: 10.1155/2013/796174. eCollection 2013. PMID: 24959567; PMCID: PMC4045564.
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Corrigendum: Landscape of the mitochondrial Hsp90 metabolome in tumours.

Nature communications

Chae YC, Angelin A, Lisanti S, Kossenkov AV, Speicher KD, Wang H, Powers JF, Tischler AS, Pacak K, Fliedner S, Michalek RD, Karoly ED, Wallace DC, Languino LR, Speicher DW, Altieri DC.
PMID: 26085380
Nat Commun. 2015 Jun 18;6:7605. doi: 10.1038/ncomms8605.

No abstract available.

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Chae YC, Angelin A, Lisanti S, et al. Corrigendum: Landscape of the mitochondrial Hsp90 metabolome in tumours. Nat Commun. 2015;6:7605doi: 10.1038/ncomms8605.
Chae, Y. C., Angelin, A., Lisanti, S., Kossenkov, A. V., Speicher, K. D., Wang, H., Powers, J. F., Tischler, A. S., Pacak, K., Fliedner, S., Michalek, R. D., Karoly, E. D., Wallace, D. C., Languino, L. R., Speicher, D. W., & Altieri, D. C. (2015). Corrigendum: Landscape of the mitochondrial Hsp90 metabolome in tumours. Nature communications, 67605. https://doi.org/10.1038/ncomms8605
Chae, Young Chan, et al. "Corrigendum: Landscape of the mitochondrial Hsp90 metabolome in tumours." Nature communications vol. 6 (2015): 7605. doi: https://doi.org/10.1038/ncomms8605
Chae YC, Angelin A, Lisanti S, Kossenkov AV, Speicher KD, Wang H, Powers JF, Tischler AS, Pacak K, Fliedner S, Michalek RD, Karoly ED, Wallace DC, Languino LR, Speicher DW, Altieri DC. Corrigendum: Landscape of the mitochondrial Hsp90 metabolome in tumours. Nat Commun. 2015 Jun 18;6:7605. doi: 10.1038/ncomms8605. PMID: 26085380; PMCID: PMC4750482.
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Anti-adaptors use distinct modes of binding to inhibit the RssB-dependent turnover of RpoS (σ(S)) by ClpXP.

Frontiers in molecular biosciences

Micevski D, Zammit JE, Truscott KN, Dougan DA.
PMID: 25988182
Front Mol Biosci. 2015 Apr 23;2:15. doi: 10.3389/fmolb.2015.00015. eCollection 2015.

In Escherichia coli, σ(S) is the master regulator of the general stress response. The level of σ(S) changes in response to multiple stress conditions and it is regulated at many levels including protein turnover. In the absence of stress,...

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Micevski D, Zammit JE, Truscott KN, et al. Anti-adaptors use distinct modes of binding to inhibit the RssB-dependent turnover of RpoS (σ(S)) by ClpXP. Front Mol Biosci. 2015;2:15doi: 10.3389/fmolb.2015.00015.
Micevski, D., Zammit, J. E., Truscott, K. N., & Dougan, D. A. (2015). Anti-adaptors use distinct modes of binding to inhibit the RssB-dependent turnover of RpoS (σ(S)) by ClpXP. Frontiers in molecular biosciences, 215. https://doi.org/10.3389/fmolb.2015.00015
Micevski, Dimce, et al. "Anti-adaptors use distinct modes of binding to inhibit the RssB-dependent turnover of RpoS (σ(S)) by ClpXP." Frontiers in molecular biosciences vol. 2 (2015): 15. doi: https://doi.org/10.3389/fmolb.2015.00015
Micevski D, Zammit JE, Truscott KN, Dougan DA. Anti-adaptors use distinct modes of binding to inhibit the RssB-dependent turnover of RpoS (σ(S)) by ClpXP. Front Mol Biosci. 2015 Apr 23;2:15. doi: 10.3389/fmolb.2015.00015. eCollection 2015. PMID: 25988182; PMCID: PMC4428439.
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High resolution corneal and single pulse imaging with line field spectral domain optical coherence tomography.

Optics express

Lawman S, Dong Y, Williams BM, Romano V, Kaye S, Harding SP, Willoughby C, Shen YC, Zheng Y.
PMID: 27410154
Opt Express. 2016 May 30;24(11):12395-405. doi: 10.1364/OE.24.012395.

We report the development of a Spectral Domain Line Field Optical Coherence Tomography (LF-OCT) system, using a broad bandwidth and spatial coherent Super-Continuum (SC) source. With conventional quasi-Continuous Wave (CW) setup we achieve axial resolutions up to 2.1 μm...

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Lawman S, Dong Y, Williams BM, et al. High resolution corneal and single pulse imaging with line field spectral domain optical coherence tomography. Opt Express. 2016;24(11):12395-405doi: 10.1364/OE.24.012395.
Lawman, S., Dong, Y., Williams, B. M., Romano, V., Kaye, S., Harding, S. P., Willoughby, C., Shen, Y. C., & Zheng, Y. (2016). High resolution corneal and single pulse imaging with line field spectral domain optical coherence tomography. Optics express, 24(11), 12395-405. https://doi.org/10.1364/OE.24.012395
Lawman, Samuel, et al. "High resolution corneal and single pulse imaging with line field spectral domain optical coherence tomography." Optics express vol. 24,11 (2016): 12395-405. doi: https://doi.org/10.1364/OE.24.012395
Lawman S, Dong Y, Williams BM, Romano V, Kaye S, Harding SP, Willoughby C, Shen YC, Zheng Y. High resolution corneal and single pulse imaging with line field spectral domain optical coherence tomography. Opt Express. 2016 May 30;24(11):12395-405. doi: 10.1364/OE.24.012395. PMID: 27410154.
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Prickle1 mutation causes planar cell polarity and directional cell migration defects associated with cardiac outflow tract anomalies and other structural birth defects.

Biology open

Gibbs BC, Damerla RR, Vladar EK, Chatterjee B, Wan Y, Liu X, Cui C, Gabriel GC, Zahid M, Yagi H, Szabo-Rogers HL, Suyama KL, Axelrod JD, Lo CW.
PMID: 26883626
Biol Open. 2016 Feb 16;5(3):323-35. doi: 10.1242/bio.015750.

Planar cell polarity (PCP) is controlled by a conserved pathway that regulates directional cell behavior. Here, we show that mutant mice harboring a newly described mutation termed Beetlejuice (Bj) in Prickle1 (Pk1), a PCP component, exhibit developmental phenotypes involving...

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Gibbs BC, Damerla RR, Vladar EK, et al. Prickle1 mutation causes planar cell polarity and directional cell migration defects associated with cardiac outflow tract anomalies and other structural birth defects. Biol Open. 2016;5(3):323-35doi: 10.1242/bio.015750.
Gibbs, B. C., Damerla, R. R., Vladar, E. K., Chatterjee, B., Wan, Y., Liu, X., Cui, C., Gabriel, G. C., Zahid, M., Yagi, H., Szabo-Rogers, H. L., Suyama, K. L., Axelrod, J. D., & Lo, C. W. (2016). Prickle1 mutation causes planar cell polarity and directional cell migration defects associated with cardiac outflow tract anomalies and other structural birth defects. Biology open, 5(3), 323-35. https://doi.org/10.1242/bio.015750
Gibbs, Brian C, et al. "Prickle1 mutation causes planar cell polarity and directional cell migration defects associated with cardiac outflow tract anomalies and other structural birth defects." Biology open vol. 5,3 (2016): 323-35. doi: https://doi.org/10.1242/bio.015750
Gibbs BC, Damerla RR, Vladar EK, Chatterjee B, Wan Y, Liu X, Cui C, Gabriel GC, Zahid M, Yagi H, Szabo-Rogers HL, Suyama KL, Axelrod JD, Lo CW. Prickle1 mutation causes planar cell polarity and directional cell migration defects associated with cardiac outflow tract anomalies and other structural birth defects. Biol Open. 2016 Feb 16;5(3):323-35. doi: 10.1242/bio.015750. PMID: 26883626; PMCID: PMC4810743.
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Asymmetric localization of natural antisense RNA of neuropeptide sensorin in Aplysia sensory neurons during aging and activity.

Frontiers in genetics

Kadakkuzha BM, Liu XA, Narvaez M, Kaye A, Akhmedov K, Puthanveettil SV.
PMID: 24795747
Front Genet. 2014 Apr 22;5:84. doi: 10.3389/fgene.2014.00084. eCollection 2014.

Despite the advances in our understanding of transcriptome, regulation and function of its non-coding components continue to be poorly understood. Here we searched for natural antisense transcript for sensorin (NAT-SRN), a neuropeptide expressed in the presynaptic sensory neurons of...

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Kadakkuzha BM, Liu XA, Narvaez M, et al. Asymmetric localization of natural antisense RNA of neuropeptide sensorin in Aplysia sensory neurons during aging and activity. Front Genet. 2014;5:84doi: 10.3389/fgene.2014.00084.
Kadakkuzha, B. M., Liu, X. A., Narvaez, M., Kaye, A., Akhmedov, K., & Puthanveettil, S. V. (2014). Asymmetric localization of natural antisense RNA of neuropeptide sensorin in Aplysia sensory neurons during aging and activity. Frontiers in genetics, 584. https://doi.org/10.3389/fgene.2014.00084
Kadakkuzha, Beena M, et al. "Asymmetric localization of natural antisense RNA of neuropeptide sensorin in Aplysia sensory neurons during aging and activity." Frontiers in genetics vol. 5 (2014): 84. doi: https://doi.org/10.3389/fgene.2014.00084
Kadakkuzha BM, Liu XA, Narvaez M, Kaye A, Akhmedov K, Puthanveettil SV. Asymmetric localization of natural antisense RNA of neuropeptide sensorin in Aplysia sensory neurons during aging and activity. Front Genet. 2014 Apr 22;5:84. doi: 10.3389/fgene.2014.00084. eCollection 2014. PMID: 24795747; PMCID: PMC4001032.
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The emerging need for family-centric initiatives for obtaining consent in personal genome research.

Genome medicine

Minari J, Teare H, Mitchell C, Kaye J, Kato K.
PMID: 25593596
Genome Med. 2014 Dec 17;6(12):118. doi: 10.1186/s13073-014-0118-y. eCollection 2014.

The use of information and communication technology can offer a novel way to promote family-centric initiatives for informed consent, and can address associated ethical challenges in personal genome research.

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Minari J, Teare H, Mitchell C, et al. The emerging need for family-centric initiatives for obtaining consent in personal genome research. Genome Med. 2014;6(12):118doi: 10.1186/s13073-014-0118-y.
Minari, J., Teare, H., Mitchell, C., Kaye, J., & Kato, K. (2014). The emerging need for family-centric initiatives for obtaining consent in personal genome research. Genome medicine, 6(12), 118. https://doi.org/10.1186/s13073-014-0118-y
Minari, Jusaku, et al. "The emerging need for family-centric initiatives for obtaining consent in personal genome research." Genome medicine vol. 6,12 (2014): 118. doi: https://doi.org/10.1186/s13073-014-0118-y
Minari J, Teare H, Mitchell C, Kaye J, Kato K. The emerging need for family-centric initiatives for obtaining consent in personal genome research. Genome Med. 2014 Dec 17;6(12):118. doi: 10.1186/s13073-014-0118-y. eCollection 2014. PMID: 25593596; PMCID: PMC4295276.
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Multidrug-resistant Pathogens: Mechanisms of Resistance and Epidemiology.

Current infectious disease reports

Kaye KS, Kaye D.
PMID: 11095883
Curr Infect Dis Rep. 2000 Oct;2(5):391-398. doi: 10.1007/s11908-000-0065-1.

Resistance to antimicrobial agents among bacteria and fungi is a persistent problem complicating the management of critically ill patients. To understand the issues involved in resistance in critical care, it is essential to understand the epidemiology and mechanisms of...

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Kaye KS, Kaye D. Multidrug-resistant Pathogens: Mechanisms of Resistance and Epidemiology. Curr Infect Dis Rep. 2000;2(5):391-398doi: 10.1007/s11908-000-0065-1.
Kaye, K. S., & Kaye, D. (2000). Multidrug-resistant Pathogens: Mechanisms of Resistance and Epidemiology. Current infectious disease reports, 2(5), 391-398. https://doi.org/10.1007/s11908-000-0065-1
Kaye, and Kaye. "Multidrug-resistant Pathogens: Mechanisms of Resistance and Epidemiology." Current infectious disease reports vol. 2,5 (2000): 391-398. doi: https://doi.org/10.1007/s11908-000-0065-1
Kaye KS, Kaye D. Multidrug-resistant Pathogens: Mechanisms of Resistance and Epidemiology. Curr Infect Dis Rep. 2000 Oct;2(5):391-398. doi: 10.1007/s11908-000-0065-1. PMID: 11095883.
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Endemic influenza. II. The nature of the disease in the post-pandemic period.

The American review of respiratory disease

KAYE D, ROSENBLUTH M, HOOK EW, KILBOURNE ED.
PMID: 14454673
Am Rev Respir Dis. 1962 Jan;85:9-21. doi: 10.1164/arrd.1962.85.1.9.

No abstract available.

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KAYE D, ROSENBLUTH M, HOOK EW, et al. Endemic influenza. II. The nature of the disease in the post-pandemic period. Am Rev Respir Dis. 1962;85:9-21doi: 10.1164/arrd.1962.85.1.9.
KAYE, D., ROSENBLUTH, M., HOOK, E. W., & KILBOURNE, E. D. (1962). Endemic influenza. II. The nature of the disease in the post-pandemic period. The American review of respiratory disease, 859-21. https://doi.org/10.1164/arrd.1962.85.1.9
KAYE, D, et al. "Endemic influenza. II. The nature of the disease in the post-pandemic period." The American review of respiratory disease vol. 85 (1962): 9-21. doi: https://doi.org/10.1164/arrd.1962.85.1.9
KAYE D, ROSENBLUTH M, HOOK EW, KILBOURNE ED. Endemic influenza. II. The nature of the disease in the post-pandemic period. Am Rev Respir Dis. 1962 Jan;85:9-21. doi: 10.1164/arrd.1962.85.1.9. PMID: 14454673.
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ras and p53 in the prediction of survival in Dukes' stage B colorectal carcinoma.

Clinical molecular pathology

Bennett MA, Kay EW, Mulcahy H, O'flaherty L, O'donoghue DP, Leader M, Croke DT.
PMID: 16696029
Clin Mol Pathol. 1995 Dec;48(6):M310-5. doi: 10.1136/mp.48.6.m310.

Aims-To determine possible associations between p53 allelic deletion, c-Ki-ras mutational activation, immunohistochemical detection of p53 and ras proteins, various clinicopathological variables, and patient outcome in 168 Dukes' stage B colorectal carcinomas.Methods-Allelic deletion at the p53 tumour suppressor gene locus...

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Bennett MA, Kay EW, Mulcahy H, et al. ras and p53 in the prediction of survival in Dukes' stage B colorectal carcinoma. Clin Mol Pathol. 1995;48(6):M310-5doi: 10.1136/mp.48.6.m310.
Bennett, M. A., Kay, E. W., Mulcahy, H., O'flaherty, L., O'donoghue, D. P., Leader, M., & Croke, D. T. (1995). ras and p53 in the prediction of survival in Dukes' stage B colorectal carcinoma. Clinical molecular pathology, 48(6), M310-5. https://doi.org/10.1136/mp.48.6.m310
Bennett, M A, et al. "ras and p53 in the prediction of survival in Dukes' stage B colorectal carcinoma." Clinical molecular pathology vol. 48,6 (1995): M310-5. doi: https://doi.org/10.1136/mp.48.6.m310
Bennett MA, Kay EW, Mulcahy H, O'flaherty L, O'donoghue DP, Leader M, Croke DT. ras and p53 in the prediction of survival in Dukes' stage B colorectal carcinoma. Clin Mol Pathol. 1995 Dec;48(6):M310-5. doi: 10.1136/mp.48.6.m310. PMID: 16696029; PMCID: PMC407996.
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Showing 1 to 12 of 504 entries