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Showing 1 to 12 of 27 entries
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Interactions between PPAR Gamma and the Canonical Wnt/Beta-Catenin Pathway in Type 2 Diabetes and Colon Cancer.

PPAR research

Lecarpentier Y, Claes V, Vallée A, Hébert JL.
PMID: 28298922
PPAR Res. 2017;2017:5879090. doi: 10.1155/2017/5879090. Epub 2017 Feb 19.

In both colon cancer and type 2 diabetes, metabolic changes induced by upregulation of the Wnt/beta-catenin signaling and downregulation of peroxisome proliferator-activated receptor gamma (PPAR gamma) may help account for the frequent association of these two diseases. In both...

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Lecarpentier Y, Claes V, Vallée A, et al. Interactions between PPAR Gamma and the Canonical Wnt/Beta-Catenin Pathway in Type 2 Diabetes and Colon Cancer. PPAR Res. 2017;2017:5879090doi: 10.1155/2017/5879090.
Lecarpentier, Y., Claes, V., Vallée, A., & Hébert, J. L. (2017). Interactions between PPAR Gamma and the Canonical Wnt/Beta-Catenin Pathway in Type 2 Diabetes and Colon Cancer. PPAR research, 20175879090. https://doi.org/10.1155/2017/5879090
Lecarpentier, Yves, et al. "Interactions between PPAR Gamma and the Canonical Wnt/Beta-Catenin Pathway in Type 2 Diabetes and Colon Cancer." PPAR research vol. 2017 (2017): 5879090. doi: https://doi.org/10.1155/2017/5879090
Lecarpentier Y, Claes V, Vallée A, Hébert JL. Interactions between PPAR Gamma and the Canonical Wnt/Beta-Catenin Pathway in Type 2 Diabetes and Colon Cancer. PPAR Res. 2017;2017:5879090. doi: 10.1155/2017/5879090. Epub 2017 Feb 19. PMID: 28298922; PMCID: PMC5337359.
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Mechanics of Relaxation of the Human Heart.

News in physiological sciences : an international journal of physiology produced jointly by the International Union of Physiological Sciences and the American Physiological Society

Chemla D, Coirault C, Hébert JL, Lecarpentier Y.
PMID: 11390883
News Physiol Sci. 2000 Apr;15:78-83. doi: 10.1152/physiologyonline.2000.15.2.78.

Rapid and complete relaxation is a prerequisite for cardiac output adaptation to changes in loading conditions, inotropic stimulation, and heart rate. In the healthy human heart, the rate and extent of relaxation depend mainly on actomyosin cross bridge dissociation...

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Chemla D, Coirault C, Hébert JL, et al. Mechanics of Relaxation of the Human Heart. News Physiol Sci. 2000;15:78-83doi: 10.1152/physiologyonline.2000.15.2.78.
Chemla, D., Coirault, C., Hébert, J. L., & Lecarpentier, Y. (2000). Mechanics of Relaxation of the Human Heart. News in physiological sciences : an international journal of physiology produced jointly by the International Union of Physiological Sciences and the American Physiological Society, 1578-83. https://doi.org/10.1152/physiologyonline.2000.15.2.78
Chemla, Denis, et al. "Mechanics of Relaxation of the Human Heart." News in physiological sciences : an international journal of physiology produced jointly by the International Union of Physiological Sciences and the American Physiological Society vol. 15 (2000): 78-83. doi: https://doi.org/10.1152/physiologyonline.2000.15.2.78
Chemla D, Coirault C, Hébert JL, Lecarpentier Y. Mechanics of Relaxation of the Human Heart. News Physiol Sci. 2000 Apr;15:78-83. doi: 10.1152/physiologyonline.2000.15.2.78. PMID: 11390883.
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Opposed Interplay between IDH1 Mutations and the WNT/β-Catenin Pathway: Added Information for Glioma Classification.

Biomedicines

Vallée A, Lecarpentier Y, Vallée JN.
PMID: 34070746
Biomedicines. 2021 May 30;9(6). doi: 10.3390/biomedicines9060619.

Gliomas are the main common primary intraparenchymal brain tumor in the central nervous system (CNS), with approximately 7% of the death caused by cancers. In the WHO 2016 classification, molecular dysregulations are part of the definition of particular brain...

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Vallée A, Lecarpentier Y, Vallée JN. Opposed Interplay between IDH1 Mutations and the WNT/β-Catenin Pathway: Added Information for Glioma Classification. Biomedicines. 2021;9(6)doi: 10.3390/biomedicines9060619.
Vallée, A., Lecarpentier, Y., & Vallée, J. N. (2021). Opposed Interplay between IDH1 Mutations and the WNT/β-Catenin Pathway: Added Information for Glioma Classification. Biomedicines, 9(6), . https://doi.org/10.3390/biomedicines9060619
Vallée, Alexandre, et al. "Opposed Interplay between IDH1 Mutations and the WNT/β-Catenin Pathway: Added Information for Glioma Classification." Biomedicines vol. 9,6 (2021). doi: https://doi.org/10.3390/biomedicines9060619
Vallée A, Lecarpentier Y, Vallée JN. Opposed Interplay between IDH1 Mutations and the WNT/β-Catenin Pathway: Added Information for Glioma Classification. Biomedicines. 2021 May 30;9(6). doi: 10.3390/biomedicines9060619. PMID: 34070746; PMCID: PMC8229353.
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Key Roles of RGD-Recognizing Integrins During Cardiac Development, on Cardiac Cells, and After Myocardial Infarction.

Journal of cardiovascular translational research

Schussler O, Chachques JC, Alifano M, Lecarpentier Y.
PMID: 34342855
J Cardiovasc Transl Res. 2021 Aug 03; doi: 10.1007/s12265-021-10154-4. Epub 2021 Aug 03.

Cardiac cells interact with the extracellular matrix (ECM) proteins through integrin mechanoreceptors that control many cellular events such as cell survival, apoptosis, differentiation, migration, and proliferation. Integrins play a crucial role in cardiac development as well as in cardiac...

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Schussler O, Chachques JC, Alifano M, et al. Key Roles of RGD-Recognizing Integrins During Cardiac Development, on Cardiac Cells, and After Myocardial Infarction. J Cardiovasc Transl Res. 2021;doi: 10.1007/s12265-021-10154-4.
Schussler, O., Chachques, J. C., Alifano, M., & Lecarpentier, Y. (2021). Key Roles of RGD-Recognizing Integrins During Cardiac Development, on Cardiac Cells, and After Myocardial Infarction. Journal of cardiovascular translational research, . https://doi.org/10.1007/s12265-021-10154-4
Schussler, Olivier, et al. "Key Roles of RGD-Recognizing Integrins During Cardiac Development, on Cardiac Cells, and After Myocardial Infarction." Journal of cardiovascular translational research vol. (2021). doi: https://doi.org/10.1007/s12265-021-10154-4
Schussler O, Chachques JC, Alifano M, Lecarpentier Y. Key Roles of RGD-Recognizing Integrins During Cardiac Development, on Cardiac Cells, and After Myocardial Infarction. J Cardiovasc Transl Res. 2021 Aug 03; doi: 10.1007/s12265-021-10154-4. Epub 2021 Aug 03. PMID: 34342855.
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Possible Treatment of Myocardial Infarct Based on Tissue Engineering Using a Cellularized Solid Collagen Scaffold Functionalized with Arg-Glyc-Asp (RGD) Peptide.

International journal of molecular sciences

Schussler O, Falcoz PE, Chachques JC, Alifano M, Lecarpentier Y.
PMID: 34830447
Int J Mol Sci. 2021 Nov 22;22(22). doi: 10.3390/ijms222212563.

Currently, the clinical impact of cell therapy after a myocardial infarction (MI) is limited by low cell engraftment due to low cell retention, cell death in inflammatory and poor angiogenic infarcted areas, secondary migration. Cells interact with their microenvironment...

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Schussler O, Falcoz PE, Chachques JC, et al. Possible Treatment of Myocardial Infarct Based on Tissue Engineering Using a Cellularized Solid Collagen Scaffold Functionalized with Arg-Glyc-Asp (RGD) Peptide. Int J Mol Sci. 2021;22(22)doi: 10.3390/ijms222212563.
Schussler, O., Falcoz, P. E., Chachques, J. C., Alifano, M., & Lecarpentier, Y. (2021). Possible Treatment of Myocardial Infarct Based on Tissue Engineering Using a Cellularized Solid Collagen Scaffold Functionalized with Arg-Glyc-Asp (RGD) Peptide. International journal of molecular sciences, 22(22), . https://doi.org/10.3390/ijms222212563
Schussler, Olivier, et al. "Possible Treatment of Myocardial Infarct Based on Tissue Engineering Using a Cellularized Solid Collagen Scaffold Functionalized with Arg-Glyc-Asp (RGD) Peptide." International journal of molecular sciences vol. 22,22 (2021). doi: https://doi.org/10.3390/ijms222212563
Schussler O, Falcoz PE, Chachques JC, Alifano M, Lecarpentier Y. Possible Treatment of Myocardial Infarct Based on Tissue Engineering Using a Cellularized Solid Collagen Scaffold Functionalized with Arg-Glyc-Asp (RGD) Peptide. Int J Mol Sci. 2021 Nov 22;22(22). doi: 10.3390/ijms222212563. PMID: 34830447; PMCID: PMC8620820.
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Circadian rhythms, Wnt/beta-catenin pathway and PPAR alpha/gamma profiles in diseases with primary or secondary cardiac dysfunction.

Frontiers in physiology

Lecarpentier Y, Claes V, Duthoit G, Hébert JL.
PMID: 25414671
Front Physiol. 2014 Nov 04;5:429. doi: 10.3389/fphys.2014.00429. eCollection 2014.

Circadian clock mechanisms are far-from-equilibrium dissipative structures. Peroxisome proliferator-activated receptors (PPAR alpha, beta/delta, and gamma) play a key role in metabolic regulatory processes, particularly in heart muscle. Links between circadian rhythms (CRs) and PPARs have been established. Mammalian CRs...

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Lecarpentier Y, Claes V, Duthoit G, et al. Circadian rhythms, Wnt/beta-catenin pathway and PPAR alpha/gamma profiles in diseases with primary or secondary cardiac dysfunction. Front Physiol. 2014;5:429doi: 10.3389/fphys.2014.00429.
Lecarpentier, Y., Claes, V., Duthoit, G., & Hébert, J. L. (2014). Circadian rhythms, Wnt/beta-catenin pathway and PPAR alpha/gamma profiles in diseases with primary or secondary cardiac dysfunction. Frontiers in physiology, 5429. https://doi.org/10.3389/fphys.2014.00429
Lecarpentier, Yves, et al. "Circadian rhythms, Wnt/beta-catenin pathway and PPAR alpha/gamma profiles in diseases with primary or secondary cardiac dysfunction." Frontiers in physiology vol. 5 (2014): 429. doi: https://doi.org/10.3389/fphys.2014.00429
Lecarpentier Y, Claes V, Duthoit G, Hébert JL. Circadian rhythms, Wnt/beta-catenin pathway and PPAR alpha/gamma profiles in diseases with primary or secondary cardiac dysfunction. Front Physiol. 2014 Nov 04;5:429. doi: 10.3389/fphys.2014.00429. eCollection 2014. PMID: 25414671; PMCID: PMC4220097.
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TGF-β in fibrosis by acting as a conductor for contractile properties of myofibroblasts.

Cell & bioscience

Vallée A, Lecarpentier Y.
PMID: 31827764
Cell Biosci. 2019 Dec 09;9:98. doi: 10.1186/s13578-019-0362-3. eCollection 2019.

Myofibroblasts are non-muscle contractile cells that play a key physiologically role in organs such as the stem villi of the human placenta during physiological pregnancy. They are able to contract and relax in response to changes in the volume...

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Vallée A, Lecarpentier Y. TGF-β in fibrosis by acting as a conductor for contractile properties of myofibroblasts. Cell Biosci. 2019;9:98doi: 10.1186/s13578-019-0362-3.
Vallée, A., & Lecarpentier, Y. (2019). TGF-β in fibrosis by acting as a conductor for contractile properties of myofibroblasts. Cell & bioscience, 998. https://doi.org/10.1186/s13578-019-0362-3
Vallée, Alexandre, and Lecarpentier, Yves. "TGF-β in fibrosis by acting as a conductor for contractile properties of myofibroblasts." Cell & bioscience vol. 9 (2019): 98. doi: https://doi.org/10.1186/s13578-019-0362-3
Vallée A, Lecarpentier Y. TGF-β in fibrosis by acting as a conductor for contractile properties of myofibroblasts. Cell Biosci. 2019 Dec 09;9:98. doi: 10.1186/s13578-019-0362-3. eCollection 2019. PMID: 31827764; PMCID: PMC6902440.
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Opposite Interplay between PPAR Gamma and Canonical Wnt/Beta-Catenin Pathway in Amyotrophic Lateral Sclerosis.

Frontiers in neurology

Lecarpentier Y, Vallée A.
PMID: 27445967
Front Neurol. 2016 Jun 28;7:100. doi: 10.3389/fneur.2016.00100. eCollection 2016.

The opposite interplay between peroxisome proliferator-activated receptor gamma (PPAR gamma) and Wnt/beta-catenin signaling has led to the categorization of neurodegenerative diseases (NDs) as either NDs in which PPAR gamma is downregulated while the canonical Wnt/beta-catenin pathway is upregulated [amyotrophic...

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Lecarpentier Y, Vallée A. Opposite Interplay between PPAR Gamma and Canonical Wnt/Beta-Catenin Pathway in Amyotrophic Lateral Sclerosis. Front Neurol. 2016;7:100doi: 10.3389/fneur.2016.00100.
Lecarpentier, Y., & Vallée, A. (2016). Opposite Interplay between PPAR Gamma and Canonical Wnt/Beta-Catenin Pathway in Amyotrophic Lateral Sclerosis. Frontiers in neurology, 7100. https://doi.org/10.3389/fneur.2016.00100
Lecarpentier, Yves, and Vallée, Alexandre. "Opposite Interplay between PPAR Gamma and Canonical Wnt/Beta-Catenin Pathway in Amyotrophic Lateral Sclerosis." Frontiers in neurology vol. 7 (2016): 100. doi: https://doi.org/10.3389/fneur.2016.00100
Lecarpentier Y, Vallée A. Opposite Interplay between PPAR Gamma and Canonical Wnt/Beta-Catenin Pathway in Amyotrophic Lateral Sclerosis. Front Neurol. 2016 Jun 28;7:100. doi: 10.3389/fneur.2016.00100. eCollection 2016. PMID: 27445967; PMCID: PMC4923074.
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HIP/PAP prevents excitotoxic neuronal death and promotes plasticity.

Annals of clinical and translational neurology

Haldipur P, Dupuis N, Degos V, Moniaux N, Chhor V, Rasika S, Schwendimann L, le Charpentier T, Rougier E, Amouyal P, Amouyal G, Dournaud P, Bréchot C, El Ghouzzi V, Faivre J, Fleiss B, Mani S, Gressens P.
PMID: 25493266
Ann Clin Transl Neurol. 2014 Oct;1(10):739-54. doi: 10.1002/acn3.127. Epub 2014 Oct 09.

OBJECTIVES: Excitotoxicity plays a significant role in the pathogenesis of perinatal brain injuries. Among the consequences of excessive activation of the N-methyl-d-aspartate (NMDA)-type glutamate are oxidative stress caused by free radical release from damaged mitochondria, neuronal death and subsequent...

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Haldipur P, Dupuis N, Degos V, et al. HIP/PAP prevents excitotoxic neuronal death and promotes plasticity. Ann Clin Transl Neurol. 2014;1(10):739-54doi: 10.1002/acn3.127.
Haldipur, P., Dupuis, N., Degos, V., Moniaux, N., Chhor, V., Rasika, S., Schwendimann, L., le Charpentier, T., Rougier, E., Amouyal, P., Amouyal, G., Dournaud, P., Bréchot, C., El Ghouzzi, V., Faivre, J., Fleiss, B., Mani, S., & Gressens, P. (2014). HIP/PAP prevents excitotoxic neuronal death and promotes plasticity. Annals of clinical and translational neurology, 1(10), 739-54. https://doi.org/10.1002/acn3.127
Haldipur, Parthiv, et al. "HIP/PAP prevents excitotoxic neuronal death and promotes plasticity." Annals of clinical and translational neurology vol. 1,10 (2014): 739-54. doi: https://doi.org/10.1002/acn3.127
Haldipur P, Dupuis N, Degos V, Moniaux N, Chhor V, Rasika S, Schwendimann L, le Charpentier T, Rougier E, Amouyal P, Amouyal G, Dournaud P, Bréchot C, El Ghouzzi V, Faivre J, Fleiss B, Mani S, Gressens P. HIP/PAP prevents excitotoxic neuronal death and promotes plasticity. Ann Clin Transl Neurol. 2014 Oct;1(10):739-54. doi: 10.1002/acn3.127. Epub 2014 Oct 09. PMID: 25493266; PMCID: PMC4241802.
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Necrotizing Enterocolitis: LPS/TLR4-Induced Crosstalk Between Canonical TGF-β/Wnt/β-Catenin Pathways and PPARγ.

Frontiers in pediatrics

Gomart A, Vallée A, Lecarpentier Y.
PMID: 34712628
Front Pediatr. 2021 Oct 12;9:713344. doi: 10.3389/fped.2021.713344. eCollection 2021.

Necrotizing enterocolitis (NEC) represents one of the major causes of morbidity and mortality in premature infants. Several recent studies, however, have contributed to a better understanding of the pathophysiology of this dreadful disease. Numerous intracellular pathways play a key...

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Gomart A, Vallée A, Lecarpentier Y. Necrotizing Enterocolitis: LPS/TLR4-Induced Crosstalk Between Canonical TGF-β/Wnt/β-Catenin Pathways and PPARγ. Front Pediatr. 2021;9:713344doi: 10.3389/fped.2021.713344.
Gomart, A., Vallée, A., & Lecarpentier, Y. (2021). Necrotizing Enterocolitis: LPS/TLR4-Induced Crosstalk Between Canonical TGF-β/Wnt/β-Catenin Pathways and PPARγ. Frontiers in pediatrics, 9713344. https://doi.org/10.3389/fped.2021.713344
Gomart, Alexia, et al. "Necrotizing Enterocolitis: LPS/TLR4-Induced Crosstalk Between Canonical TGF-β/Wnt/β-Catenin Pathways and PPARγ." Frontiers in pediatrics vol. 9 (2021): 713344. doi: https://doi.org/10.3389/fped.2021.713344
Gomart A, Vallée A, Lecarpentier Y. Necrotizing Enterocolitis: LPS/TLR4-Induced Crosstalk Between Canonical TGF-β/Wnt/β-Catenin Pathways and PPARγ. Front Pediatr. 2021 Oct 12;9:713344. doi: 10.3389/fped.2021.713344. eCollection 2021. PMID: 34712628; PMCID: PMC8547806.
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In vitro demonstration of the antidotal efficacy of hydroxocobalamin in cyanide poisoning.

Journal of neurosurgical anesthesiology

Riou B, Baud FJ, Astier A, Barriot P, Lecarpentier Y.
PMID: 15815367
J Neurosurg Anesthesiol. 1990 Dec;2(4):296-304. doi: 10.1097/00008506-199012000-00008.

The effects of sodium cyanide (1 mM) and the antidotal action of hydroxocobalamin (1 mM) were studied on rat cardiac papillary muscle. A 10-min period of exposure to cyanide induced a marked decrease in inotropy as shown by a...

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Riou B, Baud FJ, Astier A, et al. In vitro demonstration of the antidotal efficacy of hydroxocobalamin in cyanide poisoning. J Neurosurg Anesthesiol. 1990;2(4):296-304doi: 10.1097/00008506-199012000-00008.
Riou, B., Baud, F. J., Astier, A., Barriot, P., & Lecarpentier, Y. (1990). In vitro demonstration of the antidotal efficacy of hydroxocobalamin in cyanide poisoning. Journal of neurosurgical anesthesiology, 2(4), 296-304. https://doi.org/10.1097/00008506-199012000-00008
Riou, B, et al. "In vitro demonstration of the antidotal efficacy of hydroxocobalamin in cyanide poisoning." Journal of neurosurgical anesthesiology vol. 2,4 (1990): 296-304. doi: https://doi.org/10.1097/00008506-199012000-00008
Riou B, Baud FJ, Astier A, Barriot P, Lecarpentier Y. In vitro demonstration of the antidotal efficacy of hydroxocobalamin in cyanide poisoning. J Neurosurg Anesthesiol. 1990 Dec;2(4):296-304. doi: 10.1097/00008506-199012000-00008. PMID: 15815367.
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Thermodynamics in cancers: opposing interactions between PPAR gamma and the canonical WNT/beta-catenin pathway.

Clinical and translational medicine

Lecarpentier Y, Claes V, Vallée A, Hébert JL.
PMID: 28405929
Clin Transl Med. 2017 Dec;6(1):14. doi: 10.1186/s40169-017-0144-7. Epub 2017 Apr 12.

Cancer cells are the site of numerous metabolic and thermodynamic abnormalities. We focus this review on the interactions between the canonical WNT/beta-catenin pathway and peroxisome proliferator-activated receptor gamma (PPAR gamma) in cancers and their implications from an energetic and...

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Lecarpentier Y, Claes V, Vallée A, et al. Thermodynamics in cancers: opposing interactions between PPAR gamma and the canonical WNT/beta-catenin pathway. Clin Transl Med. 2017;6(1):14doi: 10.1186/s40169-017-0144-7.
Lecarpentier, Y., Claes, V., Vallée, A., & Hébert, J. L. (2017). Thermodynamics in cancers: opposing interactions between PPAR gamma and the canonical WNT/beta-catenin pathway. Clinical and translational medicine, 6(1), 14. https://doi.org/10.1186/s40169-017-0144-7
Lecarpentier, Yves, et al. "Thermodynamics in cancers: opposing interactions between PPAR gamma and the canonical WNT/beta-catenin pathway." Clinical and translational medicine vol. 6,1 (2017): 14. doi: https://doi.org/10.1186/s40169-017-0144-7
Lecarpentier Y, Claes V, Vallée A, Hébert JL. Thermodynamics in cancers: opposing interactions between PPAR gamma and the canonical WNT/beta-catenin pathway. Clin Transl Med. 2017 Dec;6(1):14. doi: 10.1186/s40169-017-0144-7. Epub 2017 Apr 12. PMID: 28405929; PMCID: PMC5389954.
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Showing 1 to 12 of 27 entries