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Response.

Clinical medicine (London, England)

Maurice JB, Manousou P.
PMID: 30287450
Clin Med (Lond). 2018 Oct;18(5):440. doi: 10.7861/clinmedicine.18-5-440.

No abstract available.

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Maurice JB, Manousou P. Response. Clin Med (Lond). 2018;18(5):440doi: 10.7861/clinmedicine.18-5-440.
Maurice, J. B., & Manousou, P. (2018). Response. Clinical medicine (London, England), 18(5), 440. https://doi.org/10.7861/clinmedicine.18-5-440
Maurice, James B, and Manousou, Pinelopi. "Response." Clinical medicine (London, England) vol. 18,5 (2018): 440. doi: https://doi.org/10.7861/clinmedicine.18-5-440
Maurice JB, Manousou P. Response. Clin Med (Lond). 2018 Oct;18(5):440. doi: 10.7861/clinmedicine.18-5-440. PMID: 30287450; PMCID: PMC6334119.
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Association of the leptin-to-adiponectin ratio with metabolic syndrome in a sub-Saharan African population.

Diabetology & metabolic syndrome

Ayina CNA, Endomba FTA, Mandengue SH, Noubiap JJN, Ngoa LSE, Boudou P, Gautier JF, Mbanya JC, Sobngwi E.
PMID: 28878827
Diabetol Metab Syndr. 2017 Sep 05;9:66. doi: 10.1186/s13098-017-0265-6. eCollection 2017.

BACKGROUND: Worldwide there is an increased prevalence of metabolic syndrome mainly due to life-style modifications, and Africans are not saved of this situation. Many markers have been studied to predict the risk of this syndrome but the most used...

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Ayina CNA, Endomba FTA, Mandengue SH, et al. Association of the leptin-to-adiponectin ratio with metabolic syndrome in a sub-Saharan African population. Diabetol Metab Syndr. 2017;9:66doi: 10.1186/s13098-017-0265-6.
Ayina, C. N. A., Endomba, F. T. A., Mandengue, S. H., Noubiap, J. J. N., Ngoa, L. S. E., Boudou, P., Gautier, J. F., Mbanya, J. C., & Sobngwi, E. (2017). Association of the leptin-to-adiponectin ratio with metabolic syndrome in a sub-Saharan African population. Diabetology & metabolic syndrome, 966. https://doi.org/10.1186/s13098-017-0265-6
Ayina, Clarisse Noël A, et al. "Association of the leptin-to-adiponectin ratio with metabolic syndrome in a sub-Saharan African population." Diabetology & metabolic syndrome vol. 9 (2017): 66. doi: https://doi.org/10.1186/s13098-017-0265-6
Ayina CNA, Endomba FTA, Mandengue SH, Noubiap JJN, Ngoa LSE, Boudou P, Gautier JF, Mbanya JC, Sobngwi E. Association of the leptin-to-adiponectin ratio with metabolic syndrome in a sub-Saharan African population. Diabetol Metab Syndr. 2017 Sep 05;9:66. doi: 10.1186/s13098-017-0265-6. eCollection 2017. PMID: 28878827; PMCID: PMC5584041.
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Sex Difference In the Effect of Fetal Exposure to Maternal Diabetes on Insulin Secretion.

Journal of the Endocrine Society

Gautier JF, Fetita LS, Riveline JP, Ibrahim F, Porcher R, Abi Khalil C, Velho G, Choukem SP, Hadjadj S, Larger E, Roussel R, Boudou P, Marre M, Ravussin E, Mauvais-Jarvis F.
PMID: 29687090
J Endocr Soc. 2018 Mar 22;2(5):391-397. doi: 10.1210/js.2017-00482. eCollection 2018 May 01.

We previously showed that fetal exposure to maternal type 1 diabetes (T1D) is associated with altered glucose-stimulated insulin secretion in adult offspring. Here, we investigated whether this

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Gautier JF, Fetita LS, Riveline JP, et al. Sex Difference In the Effect of Fetal Exposure to Maternal Diabetes on Insulin Secretion. J Endocr Soc. 2018;2(5):391-397doi: 10.1210/js.2017-00482.
Gautier, J. F., Fetita, L. S., Riveline, J. P., Ibrahim, F., Porcher, R., Abi Khalil, C., Velho, G., Choukem, S. P., Hadjadj, S., Larger, E., Roussel, R., Boudou, P., Marre, M., Ravussin, E., & Mauvais-Jarvis, F. (2018). Sex Difference In the Effect of Fetal Exposure to Maternal Diabetes on Insulin Secretion. Journal of the Endocrine Society, 2(5), 391-397. https://doi.org/10.1210/js.2017-00482
Gautier, Jean-François, et al. "Sex Difference In the Effect of Fetal Exposure to Maternal Diabetes on Insulin Secretion." Journal of the Endocrine Society vol. 2,5 (2018): 391-397. doi: https://doi.org/10.1210/js.2017-00482
Gautier JF, Fetita LS, Riveline JP, Ibrahim F, Porcher R, Abi Khalil C, Velho G, Choukem SP, Hadjadj S, Larger E, Roussel R, Boudou P, Marre M, Ravussin E, Mauvais-Jarvis F. Sex Difference In the Effect of Fetal Exposure to Maternal Diabetes on Insulin Secretion. J Endocr Soc. 2018 Mar 22;2(5):391-397. doi: 10.1210/js.2017-00482. eCollection 2018 May 01. PMID: 29687090; PMCID: PMC5905383.
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A New Strategy to Preserve and Assess Oxygen Consumption in Murine Tissues.

International journal of molecular sciences

Kluza J, Peugnet V, Daunou B, Laine W, Kervoaze G, Rémy G, Loyens A, Maboudou P, Fovez Q, Grangette C, Wolowczuk I, Gosset P, Garçon G, Marchetti P, Pinet F, Pichavant M, Dubois-Deruy E.
PMID: 35008535
Int J Mol Sci. 2021 Dec 22;23(1). doi: 10.3390/ijms23010109.

Mitochondrial dysfunctions are implicated in several pathologies, such as metabolic, cardiovascular, respiratory, and neurological diseases, as well as in cancer and aging. These metabolic alterations are usually assessed in human or murine samples by mitochondrial respiratory chain enzymatic assays,...

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Kluza J, Peugnet V, Daunou B, et al. A New Strategy to Preserve and Assess Oxygen Consumption in Murine Tissues. Int J Mol Sci. 2021;23(1)doi: 10.3390/ijms23010109.
Kluza, J., Peugnet, V., Daunou, B., Laine, W., Kervoaze, G., Rémy, G., Loyens, A., Maboudou, P., Fovez, Q., Grangette, C., Wolowczuk, I., Gosset, P., Garçon, G., Marchetti, P., Pinet, F., Pichavant, M., & Dubois-Deruy, E. (2021). A New Strategy to Preserve and Assess Oxygen Consumption in Murine Tissues. International journal of molecular sciences, 23(1), . https://doi.org/10.3390/ijms23010109
Kluza, Jerome, et al. "A New Strategy to Preserve and Assess Oxygen Consumption in Murine Tissues." International journal of molecular sciences vol. 23,1 (2021). doi: https://doi.org/10.3390/ijms23010109
Kluza J, Peugnet V, Daunou B, Laine W, Kervoaze G, Rémy G, Loyens A, Maboudou P, Fovez Q, Grangette C, Wolowczuk I, Gosset P, Garçon G, Marchetti P, Pinet F, Pichavant M, Dubois-Deruy E. A New Strategy to Preserve and Assess Oxygen Consumption in Murine Tissues. Int J Mol Sci. 2021 Dec 22;23(1). doi: 10.3390/ijms23010109. PMID: 35008535.
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Addition of Regular Insulin to Ternary Parenteral Nutrition: A Stability Study.

Pharmaceutics

Henry H, Lannoy D, Maboudou P, Seguy D, Dine T, Pigny P, Odou P.
PMID: 33801784
Pharmaceutics. 2021 Mar 27;13(4). doi: 10.3390/pharmaceutics13040458.

BACKGROUND: Parenteral nutrition (PN) is a complex medium in which added insulin can become unstable. The aim of this study is, therefore, to evaluate the stability of insulin in PN and to identify influencing factors.METHODS: A total of 20...

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Henry H, Lannoy D, Maboudou P, et al. Addition of Regular Insulin to Ternary Parenteral Nutrition: A Stability Study. Pharmaceutics. 2021;13(4)doi: 10.3390/pharmaceutics13040458.
Henry, H., Lannoy, D., Maboudou, P., Seguy, D., Dine, T., Pigny, P., & Odou, P. (2021). Addition of Regular Insulin to Ternary Parenteral Nutrition: A Stability Study. Pharmaceutics, 13(4), . https://doi.org/10.3390/pharmaceutics13040458
Henry, Heloise, et al. "Addition of Regular Insulin to Ternary Parenteral Nutrition: A Stability Study." Pharmaceutics vol. 13,4 (2021). doi: https://doi.org/10.3390/pharmaceutics13040458
Henry H, Lannoy D, Maboudou P, Seguy D, Dine T, Pigny P, Odou P. Addition of Regular Insulin to Ternary Parenteral Nutrition: A Stability Study. Pharmaceutics. 2021 Mar 27;13(4). doi: 10.3390/pharmaceutics13040458. PMID: 33801784; PMCID: PMC8066181.
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Author Correction: The Wolbachia mobilome in Culex pipiens includes a putative plasmid.

Nature communications

Reveillaud J, Bordenstein SR, Cruaud C, Shaiber A, Esen ÖC, Weill M, Makoundou P, Lolans K, Watson AR, Rakotoarivony I, Bordenstein SR, Eren AM.
PMID: 31300646
Nat Commun. 2019 Jul 12;10(1):3153. doi: 10.1038/s41467-019-11234-5.

An amendment to this paper has been published and can be accessed via a link at the top of the paper.

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Reveillaud J, Bordenstein SR, Cruaud C, et al. Author Correction: The Wolbachia mobilome in Culex pipiens includes a putative plasmid. Nat Commun. 2019;10(1):3153doi: 10.1038/s41467-019-11234-5.
Reveillaud, J., Bordenstein, S. R., Cruaud, C., Shaiber, A., Esen, �. �. C., Weill, M., Makoundou, P., Lolans, K., Watson, A. R., Rakotoarivony, I., Bordenstein, S. R., & Eren, A. M. (2019). Author Correction: The Wolbachia mobilome in Culex pipiens includes a putative plasmid. Nature communications, 10(1), 3153. https://doi.org/10.1038/s41467-019-11234-5
Reveillaud, Julie, et al. "Author Correction: The Wolbachia mobilome in Culex pipiens includes a putative plasmid." Nature communications vol. 10,1 (2019): 3153. doi: https://doi.org/10.1038/s41467-019-11234-5
Reveillaud J, Bordenstein SR, Cruaud C, Shaiber A, Esen ÖC, Weill M, Makoundou P, Lolans K, Watson AR, Rakotoarivony I, Bordenstein SR, Eren AM. Author Correction: The Wolbachia mobilome in Culex pipiens includes a putative plasmid. Nat Commun. 2019 Jul 12;10(1):3153. doi: 10.1038/s41467-019-11234-5. PMID: 31300646; PMCID: PMC6626035.
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Antimetabolic cooperativity with the clinically approved l-asparaginase and tyrosine kinase inhibitors to eradicate CML stem cells.

Molecular metabolism

Trinh A, Khamari R, Fovez Q, Mahon FX, Turcq B, Bouscary D, Maboudou P, Joncquel M, Coiteux V, Germain N, Laine W, Dekiouk S, Jean-Pierre S, Maguer-Satta V, Ghesquiere B, Idziorek T, Quesnel B, Kluza J, Marchetti P.
PMID: 34863941
Mol Metab. 2021 Dec 01;55:101410. doi: 10.1016/j.molmet.2021.101410. Epub 2021 Dec 01.

OBJECTIVE: Long-term treatment with tyrosine kinase inhibitors (TKI) represents an effective cure for chronic myeloid leukemia (CML) patients and discontinuation of TKI therapy is now proposed to patient with deep molecular responses. However, evidence demonstrating that TKI are unable...

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Trinh A, Khamari R, Fovez Q, et al. Antimetabolic cooperativity with the clinically approved l-asparaginase and tyrosine kinase inhibitors to eradicate CML stem cells. Mol Metab. 2021;55:101410doi: 10.1016/j.molmet.2021.101410.
Trinh, A., Khamari, R., Fovez, Q., Mahon, F. X., Turcq, B., Bouscary, D., Maboudou, P., Joncquel, M., Coiteux, V., Germain, N., Laine, W., Dekiouk, S., Jean-Pierre, S., Maguer-Satta, V., Ghesquiere, B., Idziorek, T., Quesnel, B., Kluza, J., & Marchetti, P. (2021). Antimetabolic cooperativity with the clinically approved l-asparaginase and tyrosine kinase inhibitors to eradicate CML stem cells. Molecular metabolism, 55101410. https://doi.org/10.1016/j.molmet.2021.101410
Trinh, Anne, et al. "Antimetabolic cooperativity with the clinically approved l-asparaginase and tyrosine kinase inhibitors to eradicate CML stem cells." Molecular metabolism vol. 55 (2021): 101410. doi: https://doi.org/10.1016/j.molmet.2021.101410
Trinh A, Khamari R, Fovez Q, Mahon FX, Turcq B, Bouscary D, Maboudou P, Joncquel M, Coiteux V, Germain N, Laine W, Dekiouk S, Jean-Pierre S, Maguer-Satta V, Ghesquiere B, Idziorek T, Quesnel B, Kluza J, Marchetti P. Antimetabolic cooperativity with the clinically approved l-asparaginase and tyrosine kinase inhibitors to eradicate CML stem cells. Mol Metab. 2021 Dec 01;55:101410. doi: 10.1016/j.molmet.2021.101410. Epub 2021 Dec 01. PMID: 34863941; PMCID: PMC8732793.
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ANTIMETABOLIC COOPERATIVITY WITH THE CLINICALLY-APPROVED L-ASPARAGINASE AND TYROSINE KINASE INHIBITORS TO ERADICATE CML STEM CELLS.

Molecular metabolism

Trinh A, Khamari R, Fovez Q, Mahon FX, Turcq B, Bouscary D, Maboudou P, Joncquel M, Coiteux V, Germain N, Laine W, Dekiouk S, Jean-Pierre S, Maguer-Satta V, Ghesquiere B, Idziorek T, Quesnel B, Kluza J, Marchetti P.
PMID: 34863941
Mol Metab. 2021 Dec 01;101410. doi: 10.1016/j.molmet.2021.101410. Epub 2021 Dec 01.

Long-term treatment with tyrosine kinase inhibitors (TKI) represents an effective treatment for chronic myeloid leukemia (CML) and discontinuation of TKI therapy is now being proposed to patients with deep molecular responses. However, evidence demonstrating that TKI are unable to...

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Trinh A, Khamari R, Fovez Q, et al. ANTIMETABOLIC COOPERATIVITY WITH THE CLINICALLY-APPROVED L-ASPARAGINASE AND TYROSINE KINASE INHIBITORS TO ERADICATE CML STEM CELLS. Mol Metab. 2021;101410doi: 10.1016/j.molmet.2021.101410.
Trinh, A., Khamari, R., Fovez, Q., Mahon, F. X., Turcq, B., Bouscary, D., Maboudou, P., Joncquel, M., Coiteux, V., Germain, N., Laine, W., Dekiouk, S., Jean-Pierre, S., Maguer-Satta, V., Ghesquiere, B., Idziorek, T., Quesnel, B., Kluza, J., & Marchetti, P. (2021). ANTIMETABOLIC COOPERATIVITY WITH THE CLINICALLY-APPROVED L-ASPARAGINASE AND TYROSINE KINASE INHIBITORS TO ERADICATE CML STEM CELLS. Molecular metabolism, 101410. https://doi.org/10.1016/j.molmet.2021.101410
Trinh, Anne, et al. "ANTIMETABOLIC COOPERATIVITY WITH THE CLINICALLY-APPROVED L-ASPARAGINASE AND TYROSINE KINASE INHIBITORS TO ERADICATE CML STEM CELLS." Molecular metabolism vol. (2021): 101410. doi: https://doi.org/10.1016/j.molmet.2021.101410
Trinh A, Khamari R, Fovez Q, Mahon FX, Turcq B, Bouscary D, Maboudou P, Joncquel M, Coiteux V, Germain N, Laine W, Dekiouk S, Jean-Pierre S, Maguer-Satta V, Ghesquiere B, Idziorek T, Quesnel B, Kluza J, Marchetti P. ANTIMETABOLIC COOPERATIVITY WITH THE CLINICALLY-APPROVED L-ASPARAGINASE AND TYROSINE KINASE INHIBITORS TO ERADICATE CML STEM CELLS. Mol Metab. 2021 Dec 01;101410. doi: 10.1016/j.molmet.2021.101410. Epub 2021 Dec 01. PMID: 34863941.
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Variability of vitamins B1, B2 and minerals content in baobab (Adansonia digitata) leaves in East and West Africa.

Food science & nutrition

Hyacinthe T, Charles P, Adama K, Diarra CS, Dicko MH, Svejgaard JJ, Diawara B.
PMID: 25649547
Food Sci Nutr. 2015 Jan;3(1):17-24. doi: 10.1002/fsn3.184. Epub 2014 Nov 17.

The regional variability and age-age correlation on vitamin B1, vitamin B2 and minerals (Ca, Mg, P, K, Cu, Fe, Mn, Na, and Zn) concentration in baobab leaves were investigated. Baobab was cultivated from seeds from 11 countries including Benin,...

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Hyacinthe T, Charles P, Adama K, et al. Variability of vitamins B1, B2 and minerals content in baobab (Adansonia digitata) leaves in East and West Africa. Food Sci Nutr. 2014;3(1):17-24doi: 10.1002/fsn3.184.
Hyacinthe, T., Charles, P., Adama, K., Diarra, C. S., Dicko, M. H., Svejgaard, J. J., & Diawara, B. (2015). Variability of vitamins B1, B2 and minerals content in baobab (Adansonia digitata) leaves in East and West Africa. Food science & nutrition, 3(1), 17-24. https://doi.org/10.1002/fsn3.184
Hyacinthe, Traoré, et al. "Variability of vitamins B1, B2 and minerals content in baobab (Adansonia digitata) leaves in East and West Africa." Food science & nutrition vol. 3,1 (2015): 17-24. doi: https://doi.org/10.1002/fsn3.184
Hyacinthe T, Charles P, Adama K, Diarra CS, Dicko MH, Svejgaard JJ, Diawara B. Variability of vitamins B1, B2 and minerals content in baobab (Adansonia digitata) leaves in East and West Africa. Food Sci Nutr. 2015 Jan;3(1):17-24. doi: 10.1002/fsn3.184. Epub 2014 Nov 17. PMID: 25649547; PMCID: PMC4304558.
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Hematocrit alterations and its effects in naturally infected indigenous cattle breeds due to Trypanosoma spp. on the Adamawa Plateau - Cameroon.

Veterinary world

Mamoudou A, Payne VK, Sevidzem SL.
PMID: 27047158
Vet World. 2015 Jun;8(6):813-8. doi: 10.14202/vetworld.2015.813-818. Epub 2015 Jun 30.

AIM: An experimental study was carried out on 148 naturally infected indigenous cattle breeds with either single or mix infections of various species of trypanosomes. The objectives of this study were to determine the species of trypanosomes, observe their...

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Mamoudou A, Payne VK, Sevidzem SL. Hematocrit alterations and its effects in naturally infected indigenous cattle breeds due to Trypanosoma spp. on the Adamawa Plateau - Cameroon. Vet World. 2015;8(6):813-8doi: 10.14202/vetworld.2015.813-818.
Mamoudou, A., Payne, V. K., & Sevidzem, S. L. (2015). Hematocrit alterations and its effects in naturally infected indigenous cattle breeds due to Trypanosoma spp. on the Adamawa Plateau - Cameroon. Veterinary world, 8(6), 813-8. https://doi.org/10.14202/vetworld.2015.813-818
Mamoudou, A, et al. "Hematocrit alterations and its effects in naturally infected indigenous cattle breeds due to Trypanosoma spp. on the Adamawa Plateau - Cameroon." Veterinary world vol. 8,6 (2015): 813-8. doi: https://doi.org/10.14202/vetworld.2015.813-818
Mamoudou A, Payne VK, Sevidzem SL. Hematocrit alterations and its effects in naturally infected indigenous cattle breeds due to Trypanosoma spp. on the Adamawa Plateau - Cameroon. Vet World. 2015 Jun;8(6):813-8. doi: 10.14202/vetworld.2015.813-818. Epub 2015 Jun 30. PMID: 27047158; PMCID: PMC4779167.
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Adaptive deletion in resistance gene duplications in the malaria vector .

Evolutionary applications

Assogba BS, Alout H, Koffi A, Penetier C, Djogbénou LS, Makoundou P, Weill M, Labbé P.
PMID: 30151037
Evol Appl. 2018 Mar 25;11(8):1245-1256. doi: 10.1111/eva.12619. eCollection 2018 Sep.

While gene copy-number variations play major roles in long-term evolution, their early dynamics remains largely unknown. However, examples of their role in short-term adaptation are accumulating: identical repetitions of a locus (homogeneous duplications) can provide a quantitative advantage, while...

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Assogba BS, Alout H, Koffi A, et al. Adaptive deletion in resistance gene duplications in the malaria vector . Evol Appl. 2018;11(8):1245-1256doi: 10.1111/eva.12619.
Assogba, B. S., Alout, H., Koffi, A., Penetier, C., Djogbénou, L. S., Makoundou, P., Weill, M., & Labbé, P. (2018). Adaptive deletion in resistance gene duplications in the malaria vector . Evolutionary applications, 11(8), 1245-1256. https://doi.org/10.1111/eva.12619
Assogba, Benoît S, et al. "Adaptive deletion in resistance gene duplications in the malaria vector ." Evolutionary applications vol. 11,8 (2018): 1245-1256. doi: https://doi.org/10.1111/eva.12619
Assogba BS, Alout H, Koffi A, Penetier C, Djogbénou LS, Makoundou P, Weill M, Labbé P. Adaptive deletion in resistance gene duplications in the malaria vector . Evol Appl. 2018 Mar 25;11(8):1245-1256. doi: 10.1111/eva.12619. eCollection 2018 Sep. PMID: 30151037; PMCID: PMC6099818.
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Osteoprotegerin in relation to insulin resistance and blood lipids in sub-Saharan African women with and without abdominal obesity.

Diabetology & metabolic syndrome

Ayina Ayina CN, Sobngwi E, Essouma M, Noubiap JJ, Boudou P, Etoundi Ngoa LS, Gautier JF.
PMID: 26034511
Diabetol Metab Syndr. 2015 May 23;7:47. doi: 10.1186/s13098-015-0042-3. eCollection 2015.

BACKGROUND: Osteoprotegerin (OPG), a soluble member of the tumor necrosis factor receptor superfamily that inhibits bone resorption, has been suggested as a potential marker of cardiovascular risk. This study aimed to assess the relationship between insulin resistance, lipid profile...

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Ayina Ayina CN, Sobngwi E, Essouma M, et al. Osteoprotegerin in relation to insulin resistance and blood lipids in sub-Saharan African women with and without abdominal obesity. Diabetol Metab Syndr. 2015;7:47doi: 10.1186/s13098-015-0042-3.
Ayina Ayina, C. N., Sobngwi, E., Essouma, M., Noubiap, J. J., Boudou, P., Etoundi Ngoa, L. S., & Gautier, J. F. (2015). Osteoprotegerin in relation to insulin resistance and blood lipids in sub-Saharan African women with and without abdominal obesity. Diabetology & metabolic syndrome, 747. https://doi.org/10.1186/s13098-015-0042-3
Ayina Ayina, Clarisse Noël, et al. "Osteoprotegerin in relation to insulin resistance and blood lipids in sub-Saharan African women with and without abdominal obesity." Diabetology & metabolic syndrome vol. 7 (2015): 47. doi: https://doi.org/10.1186/s13098-015-0042-3
Ayina Ayina CN, Sobngwi E, Essouma M, Noubiap JJ, Boudou P, Etoundi Ngoa LS, Gautier JF. Osteoprotegerin in relation to insulin resistance and blood lipids in sub-Saharan African women with and without abdominal obesity. Diabetol Metab Syndr. 2015 May 23;7:47. doi: 10.1186/s13098-015-0042-3. eCollection 2015. PMID: 26034511; PMCID: PMC4450452.
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