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Comparative cost effectiveness of Coflex® interlaminar stabilization versus instrumented posterolateral lumbar fusion for the treatment of lumbar spinal stenosis and spondylolisthesis.

ClinicoEconomics and outcomes research : CEOR

Schmier JK, Halevi M, Maislin G, Ong K.
PMID: 24672250
Clinicoecon Outcomes Res. 2014 Mar 18;6:125-31. doi: 10.2147/CEOR.S59194. eCollection 2014.

INTRODUCTION: Symptomatic chronic low back and leg pain resulting from lumbar spinal stenosis is expensive to treat and manage. A randomized, controlled, multicenter US Food and Drug Administration Investigational Device Exemption clinical trial assessed treatment-related patient outcomes comparing the...

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Schmier JK, Halevi M, Maislin G, et al. Comparative cost effectiveness of Coflex® interlaminar stabilization versus instrumented posterolateral lumbar fusion for the treatment of lumbar spinal stenosis and spondylolisthesis. Clinicoecon Outcomes Res. 2014;6:125-31doi: 10.2147/CEOR.S59194.
Schmier, J. K., Halevi, M., Maislin, G., & Ong, K. (2014). Comparative cost effectiveness of Coflex® interlaminar stabilization versus instrumented posterolateral lumbar fusion for the treatment of lumbar spinal stenosis and spondylolisthesis. ClinicoEconomics and outcomes research : CEOR, 6125-31. https://doi.org/10.2147/CEOR.S59194
Schmier, Jordana Kate, et al. "Comparative cost effectiveness of Coflex® interlaminar stabilization versus instrumented posterolateral lumbar fusion for the treatment of lumbar spinal stenosis and spondylolisthesis." ClinicoEconomics and outcomes research : CEOR vol. 6 (2014): 125-31. doi: https://doi.org/10.2147/CEOR.S59194
Schmier JK, Halevi M, Maislin G, Ong K. Comparative cost effectiveness of Coflex® interlaminar stabilization versus instrumented posterolateral lumbar fusion for the treatment of lumbar spinal stenosis and spondylolisthesis. Clinicoecon Outcomes Res. 2014 Mar 18;6:125-31. doi: 10.2147/CEOR.S59194. eCollection 2014. PMID: 24672250; PMCID: PMC3964032.
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Mutation in .

World journal of medical genetics

Kaufman CS, Butler MG.
PMID: 28344932
World J Med Genet. 2016 May 27;6(2):17-21. doi: 10.5496/wjmg.v6.i2.17.

We report a 28-year-old female who presented with severe joint pain, chronic muscle weakness, Raynaud's phenomenon, and hypermobility. She was found to have a 6074A > T nucleotide transition in the

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Kaufman CS, Butler MG. Mutation in . World J Med Genet. 2016;6(2):17-21doi: 10.5496/wjmg.v6.i2.17.
Kaufman, C. S., & Butler, M. G. (2016). Mutation in . World journal of medical genetics, 6(2), 17-21. https://doi.org/10.5496/wjmg.v6.i2.17
Kaufman, Carolyn S, and Butler, Merlin G. "Mutation in ." World journal of medical genetics vol. 6,2 (2016): 17-21. doi: https://doi.org/10.5496/wjmg.v6.i2.17
Kaufman CS, Butler MG. Mutation in . World J Med Genet. 2016 May 27;6(2):17-21. doi: 10.5496/wjmg.v6.i2.17. PMID: 28344932; PMCID: PMC5363719.
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Duplication of 19p13.3 in 11-Year-Old Male Patient with Dysmorphic Features and Intellectual Disability: A Review.

Journal of pediatric genetics

Novikova I, Sen P, Manzardo A, Butler MG.
PMID: 29142765
J Pediatr Genet. 2017 Dec;6(4):227-233. doi: 10.1055/s-0037-1603650. Epub 2017 Jun 02.

We present a clinical report of an 11-year-old male patient with an interstitial duplication of 19p13.3 (829 kb in size) at genomic coordinates 3,804,495-4,033,722 bp (hg19) identified by chromosomal microarray analysis and review the literature from nine published reports...

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Novikova I, Sen P, Manzardo A, et al. Duplication of 19p13.3 in 11-Year-Old Male Patient with Dysmorphic Features and Intellectual Disability: A Review. J Pediatr Genet. 2017;6(4):227-233doi: 10.1055/s-0037-1603650.
Novikova, I., Sen, P., Manzardo, A., & Butler, M. G. (2017). Duplication of 19p13.3 in 11-Year-Old Male Patient with Dysmorphic Features and Intellectual Disability: A Review. Journal of pediatric genetics, 6(4), 227-233. https://doi.org/10.1055/s-0037-1603650
Novikova, Irina, et al. "Duplication of 19p13.3 in 11-Year-Old Male Patient with Dysmorphic Features and Intellectual Disability: A Review." Journal of pediatric genetics vol. 6,4 (2017): 227-233. doi: https://doi.org/10.1055/s-0037-1603650
Novikova I, Sen P, Manzardo A, Butler MG. Duplication of 19p13.3 in 11-Year-Old Male Patient with Dysmorphic Features and Intellectual Disability: A Review. J Pediatr Genet. 2017 Dec;6(4):227-233. doi: 10.1055/s-0037-1603650. Epub 2017 Jun 02. PMID: 29142765; PMCID: PMC5683958.
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Effects of the nephrilin peptide on post-burn glycemic control, renal function, fat and lean body mass, and wound healing.

International journal of burns and trauma

Mascarenhas DD, Ayadi AE, Wetzel M, Prasai A, Mifflin R, Jay J, Herndon DN, Finnerty CC.
PMID: 28078180
Int J Burns Trauma. 2016 Nov 30;6(3):44-50. eCollection 2016.

The mechanisms underlying the effects of severe burn trauma are not well understood. We previously demonstrated the ability of nephrilin peptide (an iron-binding peptide believed to enter cells through iron-uptake pathways) to suppress aspects of the neuroinflammatory response in...

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Mascarenhas DD, Ayadi AE, Wetzel M, et al. Effects of the nephrilin peptide on post-burn glycemic control, renal function, fat and lean body mass, and wound healing. Int J Burns Trauma. 2016;6(3):44-50
Mascarenhas, D. D., Ayadi, A. E., Wetzel, M., Prasai, A., Mifflin, R., Jay, J., Herndon, D. N., & Finnerty, C. C. (2016). Effects of the nephrilin peptide on post-burn glycemic control, renal function, fat and lean body mass, and wound healing. International journal of burns and trauma, 6(3), 44-50.
Mascarenhas, Desmond D, et al. "Effects of the nephrilin peptide on post-burn glycemic control, renal function, fat and lean body mass, and wound healing." International journal of burns and trauma vol. 6,3 (2016): 44-50.
Mascarenhas DD, Ayadi AE, Wetzel M, Prasai A, Mifflin R, Jay J, Herndon DN, Finnerty CC. Effects of the nephrilin peptide on post-burn glycemic control, renal function, fat and lean body mass, and wound healing. Int J Burns Trauma. 2016 Nov 30;6(3):44-50. eCollection 2016. PMID: 28078180; PMCID: PMC5218868.
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Nutritional control of storage-protein synthesis in developing grain of barley (Hordeum vulgare L.).

Planta

Rahman S, Shewry PR, Forde BG, Kreis M, Miflin BJ.
PMID: 24258235
Planta. 1983 Nov;159(4):366-72. doi: 10.1007/BF00393176.

Sulphur starvation of barley results in decreased accumulation of the "sulphur-rich" B-hordein polypeptides, with little or no effect on the 'sulphur-poor' C hordeins. The populations of mRNAs for C hordeins and the two major subfamilies of B-hordein polypeptides have...

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Rahman S, Shewry PR, Forde BG, et al. Nutritional control of storage-protein synthesis in developing grain of barley (Hordeum vulgare L.). Planta. 1983;159(4):366-72doi: 10.1007/BF00393176.
Rahman, S., Shewry, P. R., Forde, B. G., Kreis, M., & Miflin, B. J. (1983). Nutritional control of storage-protein synthesis in developing grain of barley (Hordeum vulgare L.). Planta, 159(4), 366-72. https://doi.org/10.1007/BF00393176
Rahman, S, et al. "Nutritional control of storage-protein synthesis in developing grain of barley (Hordeum vulgare L.)." Planta vol. 159,4 (1983): 366-72. doi: https://doi.org/10.1007/BF00393176
Rahman S, Shewry PR, Forde BG, Kreis M, Miflin BJ. Nutritional control of storage-protein synthesis in developing grain of barley (Hordeum vulgare L.). Planta. 1983 Nov;159(4):366-72. doi: 10.1007/BF00393176. PMID: 24258235.
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Lysine metabolism in a barley mutant resistant to S(2-aminoethyl)cysteine.

Planta

Bright SW, Featherstone LC, Miflin BJ.
PMID: 24318338
Planta. 1979 Oct;146(5):629-33. doi: 10.1007/BF00388843.

Lysine and S(2-aminoethyl)cysteine (AEC) metabolism were investigated in normal barley (Hordeum vulgare L. cv. Bomi) and a hemozygous recessive AEC-resistant mutant (R906). Feedback regulation of lysine and threonine synthesis from [(14)C] acetate was unimpaired in plants of the mutant...

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Bright SW, Featherstone LC, Miflin BJ. Lysine metabolism in a barley mutant resistant to S(2-aminoethyl)cysteine. Planta. 1979;146(5):629-33doi: 10.1007/BF00388843.
Bright, S. W., Featherstone, L. C., & Miflin, B. J. (1979). Lysine metabolism in a barley mutant resistant to S(2-aminoethyl)cysteine. Planta, 146(5), 629-33. https://doi.org/10.1007/BF00388843
Bright, S W, et al. "Lysine metabolism in a barley mutant resistant to S(2-aminoethyl)cysteine." Planta vol. 146,5 (1979): 629-33. doi: https://doi.org/10.1007/BF00388843
Bright SW, Featherstone LC, Miflin BJ. Lysine metabolism in a barley mutant resistant to S(2-aminoethyl)cysteine. Planta. 1979 Oct;146(5):629-33. doi: 10.1007/BF00388843. PMID: 24318338.
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Gene expression in pediatric heart disease with emphasis on conotruncal defects.

Progress in pediatric cardiology

Bittel DC, Kibiryeva N, O'Brien JE, Lofland GK, Butler MG.
PMID: 28529438
Prog Pediatr Cardiol. 2005 Jul;20(2):127-141. doi: 10.1016/j.ppedcard.2005.04.004. Epub 2005 Jun 09.

Developmental abnormalities of the heart are the underlying cause of many congenital heart malformations. The embryological development of the integrated cardiovascular tissue is the result of multiple tissue and cell-to-cell interactions involving temporal and spatial events under genetic control....

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Bittel DC, Kibiryeva N, O'Brien JE, et al. Gene expression in pediatric heart disease with emphasis on conotruncal defects. Prog Pediatr Cardiol. 2005;20(2):127-141doi: 10.1016/j.ppedcard.2005.04.004.
Bittel, D. C., Kibiryeva, N., O'Brien, J. E., Lofland, G. K., & Butler, M. G. (2005). Gene expression in pediatric heart disease with emphasis on conotruncal defects. Progress in pediatric cardiology, 20(2), 127-141. https://doi.org/10.1016/j.ppedcard.2005.04.004
Bittel, Douglas C, et al. "Gene expression in pediatric heart disease with emphasis on conotruncal defects." Progress in pediatric cardiology vol. 20,2 (2005): 127-141. doi: https://doi.org/10.1016/j.ppedcard.2005.04.004
Bittel DC, Kibiryeva N, O'Brien JE, Lofland GK, Butler MG. Gene expression in pediatric heart disease with emphasis on conotruncal defects. Prog Pediatr Cardiol. 2005 Jul;20(2):127-141. doi: 10.1016/j.ppedcard.2005.04.004. Epub 2005 Jun 09. PMID: 28529438; PMCID: PMC5436709.
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Sportsmedicine forum.

The Physician and sportsmedicine

Yessis M, Nedzbala RM, Mirkin G, Wilmore JH, Levy AM.
PMID: 27457002
Phys Sportsmed. 1987 Jan;15(1):58-61. doi: 10.1080/00913847.1987.11709251.

A Forum For Our Readers Sportsmedicine Forum is intended to provide a sounding board for our readers. Perhaps you have a special way to treat a common medical problem, or you may want to air your views on a...

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Yessis M, Nedzbala RM, Mirkin G, et al. Sportsmedicine forum. Phys Sportsmed. 1987;15(1):58-61doi: 10.1080/00913847.1987.11709251.
Yessis, M., Nedzbala, R. M., Mirkin, G., Wilmore, J. H., & Levy, A. M. (1987). Sportsmedicine forum. The Physician and sportsmedicine, 15(1), 58-61. https://doi.org/10.1080/00913847.1987.11709251
Yessis, M, et al. "Sportsmedicine forum." The Physician and sportsmedicine vol. 15,1 (1987): 58-61. doi: https://doi.org/10.1080/00913847.1987.11709251
Yessis M, Nedzbala RM, Mirkin G, Wilmore JH, Levy AM. Sportsmedicine forum. Phys Sportsmed. 1987 Jan;15(1):58-61. doi: 10.1080/00913847.1987.11709251. PMID: 27457002.
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INTRAFAMILIAL AND MIDPARENTAL-CHILD CORRELATIONS AND HERITABILITY ESTIMATES OF ANTHROPOMETRIC MEASUREMENTS IN PRADER-WILLI SYNDROME FAMILIES.

Dysmorphology and clinical genetics : official publication of the Center for Birth Defects Information Services, Inc

Butler MG, Haynes JL, Meaney FJ.
PMID: 25505360
Dysmorphol Clin Genet. 1990;4(1):2-6.

To determine the effects of familial background on anthropometric dimensions in Prader-Willi syndrome (PWS), we measured weight; height; sitting height; longitude and breadth of the head, hands, and feet; head, arm, and calf circumferences; and triceps and subscapular skinfolds...

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Butler MG, Haynes JL, Meaney FJ. INTRAFAMILIAL AND MIDPARENTAL-CHILD CORRELATIONS AND HERITABILITY ESTIMATES OF ANTHROPOMETRIC MEASUREMENTS IN PRADER-WILLI SYNDROME FAMILIES. Dysmorphol Clin Genet. 1990;4(1):2-6
Butler, M. G., Haynes, J. L., & Meaney, F. J. (1990). INTRAFAMILIAL AND MIDPARENTAL-CHILD CORRELATIONS AND HERITABILITY ESTIMATES OF ANTHROPOMETRIC MEASUREMENTS IN PRADER-WILLI SYNDROME FAMILIES. Dysmorphology and clinical genetics : official publication of the Center for Birth Defects Information Services, Inc, 4(1), 2-6.
Butler, Merlin G, et al. "INTRAFAMILIAL AND MIDPARENTAL-CHILD CORRELATIONS AND HERITABILITY ESTIMATES OF ANTHROPOMETRIC MEASUREMENTS IN PRADER-WILLI SYNDROME FAMILIES." Dysmorphology and clinical genetics : official publication of the Center for Birth Defects Information Services, Inc vol. 4,1 (1990): 2-6.
Butler MG, Haynes JL, Meaney FJ. INTRAFAMILIAL AND MIDPARENTAL-CHILD CORRELATIONS AND HERITABILITY ESTIMATES OF ANTHROPOMETRIC MEASUREMENTS IN PRADER-WILLI SYNDROME FAMILIES. Dysmorphol Clin Genet. 1990;4(1):2-6. PMID: 25505360; PMCID: PMC4259252.
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Distribution of the Enzymes of Nitrogen Assimilation within the Pea Leaf Cell.

Plant physiology

Wallsgrove RM, Lea PJ, Miflin BJ.
PMID: 16660703
Plant Physiol. 1979 Feb;63(2):232-6. doi: 10.1104/pp.63.2.232.

Protoplasts obtained from expanded leaves of Pisum sativum have been used for the isolation of cell organelles and the subsequent study of the intracellular distribution of the enzymes of nitrate assimilation. The protoplasts were ruptured in a suitable medium...

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Wallsgrove RM, Lea PJ, Miflin BJ. Distribution of the Enzymes of Nitrogen Assimilation within the Pea Leaf Cell. Plant Physiol. 1979;63(2):232-6doi: 10.1104/pp.63.2.232.
Wallsgrove, R. M., Lea, P. J., & Miflin, B. J. (1979). Distribution of the Enzymes of Nitrogen Assimilation within the Pea Leaf Cell. Plant physiology, 63(2), 232-6. https://doi.org/10.1104/pp.63.2.232
Wallsgrove, R M, et al. "Distribution of the Enzymes of Nitrogen Assimilation within the Pea Leaf Cell." Plant physiology vol. 63,2 (1979): 232-6. doi: https://doi.org/10.1104/pp.63.2.232
Wallsgrove RM, Lea PJ, Miflin BJ. Distribution of the Enzymes of Nitrogen Assimilation within the Pea Leaf Cell. Plant Physiol. 1979 Feb;63(2):232-6. doi: 10.1104/pp.63.2.232. PMID: 16660703; PMCID: PMC542804.
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Properties and Regulation of Aspartate Kinase from Barley Seedlings (Hordeum vulgare L.).

Plant physiology

Shewry PR, Miflin BJ.
PMID: 16659791
Plant Physiol. 1977 Jan;59(1):69-73. doi: 10.1104/pp.59.1.69.

Aspartate kinase (EC 2.7.2.4) has been purified 8-fold and characterized from germinating barley (Hordeum vulgare) seedlings. The enzyme is inhibited 50% by 0.7 mm l-lysine and almost completely at 5 mm. l-Methionine does not affect the enzyme on its...

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Shewry PR, Miflin BJ. Properties and Regulation of Aspartate Kinase from Barley Seedlings (Hordeum vulgare L.). Plant Physiol. 1977;59(1):69-73doi: 10.1104/pp.59.1.69.
Shewry, P. R., & Miflin, B. J. (1977). Properties and Regulation of Aspartate Kinase from Barley Seedlings (Hordeum vulgare L.). Plant physiology, 59(1), 69-73. https://doi.org/10.1104/pp.59.1.69
Shewry, P R, and Miflin, B J. "Properties and Regulation of Aspartate Kinase from Barley Seedlings (Hordeum vulgare L.)." Plant physiology vol. 59,1 (1977): 69-73. doi: https://doi.org/10.1104/pp.59.1.69
Shewry PR, Miflin BJ. Properties and Regulation of Aspartate Kinase from Barley Seedlings (Hordeum vulgare L.). Plant Physiol. 1977 Jan;59(1):69-73. doi: 10.1104/pp.59.1.69. PMID: 16659791; PMCID: PMC542332.
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Regulatory isoenzymes of aspartate kinase and the control of lysine and threonine biosynthesis in carrot cell suspension culture.

Plant physiology

Davies HM, Miflin BJ.
PMID: 16660554
Plant Physiol. 1978 Oct;62(4):536-41. doi: 10.1104/pp.62.4.536.

Aspartate kinase (EC 2.7.2.4) from carrot (Daucus carota L.) cell suspension culture has been partially resolved into lysine-sensitive and threonine-sensitive components by gel filtration chromatography. The yield of lysine-sensitive aspartate kinase changed independently of the yield of the threonine-sensitive...

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Davies HM, Miflin BJ. Regulatory isoenzymes of aspartate kinase and the control of lysine and threonine biosynthesis in carrot cell suspension culture. Plant Physiol. 1978;62(4):536-41doi: 10.1104/pp.62.4.536.
Davies, H. M., & Miflin, B. J. (1978). Regulatory isoenzymes of aspartate kinase and the control of lysine and threonine biosynthesis in carrot cell suspension culture. Plant physiology, 62(4), 536-41. https://doi.org/10.1104/pp.62.4.536
Davies, H M, and Miflin, B J. "Regulatory isoenzymes of aspartate kinase and the control of lysine and threonine biosynthesis in carrot cell suspension culture." Plant physiology vol. 62,4 (1978): 536-41. doi: https://doi.org/10.1104/pp.62.4.536
Davies HM, Miflin BJ. Regulatory isoenzymes of aspartate kinase and the control of lysine and threonine biosynthesis in carrot cell suspension culture. Plant Physiol. 1978 Oct;62(4):536-41. doi: 10.1104/pp.62.4.536. PMID: 16660554; PMCID: PMC1092166.
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Showing 1 to 12 of 236 entries