Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 585 entries
Sorted by: Best Match Show Resources per page
Coherent storage of photoexcited triplet states using 29Si nuclear spins in silicon.

Physical review letters

Akhtar W, Filidou V, Sekiguchi T, Kawakami E, Itahashi T, Vlasenko L, Morton JJ, Itoh KM.
PMID: 22463668
Phys Rev Lett. 2012 Mar 02;108(9):097601. doi: 10.1103/PhysRevLett.108.097601. Epub 2012 Feb 27.

Pulsed electron paramagnetic resonance spectroscopy of the photoexcited, metastable triplet state of the oxygen-vacancy center in silicon reveals that the lifetime of the m(s)=±1 sublevels differs significantly from that of the m(s)=0 state. We exploit this significant difference in...

Cite
Akhtar W, Filidou V, Sekiguchi T, et al. Coherent storage of photoexcited triplet states using 29Si nuclear spins in silicon. Phys Rev Lett. 2012;108(9):097601doi: 10.1103/PhysRevLett.108.097601.
Akhtar, W., Filidou, V., Sekiguchi, T., Kawakami, E., Itahashi, T., Vlasenko, L., Morton, J. J., & Itoh, K. M. (2012). Coherent storage of photoexcited triplet states using 29Si nuclear spins in silicon. Physical review letters, 108(9), 097601. https://doi.org/10.1103/PhysRevLett.108.097601
Akhtar, Waseem, et al. "Coherent storage of photoexcited triplet states using 29Si nuclear spins in silicon." Physical review letters vol. 108,9 (2012): 097601. doi: https://doi.org/10.1103/PhysRevLett.108.097601
Akhtar W, Filidou V, Sekiguchi T, Kawakami E, Itahashi T, Vlasenko L, Morton JJ, Itoh KM. Coherent storage of photoexcited triplet states using 29Si nuclear spins in silicon. Phys Rev Lett. 2012 Mar 02;108(9):097601. doi: 10.1103/PhysRevLett.108.097601. Epub 2012 Feb 27. PMID: 22463668.
Copy Download .nbib Format: NLM
Draft Genome Sequences of Six Nontypeable Haemophilus influenzae Strains That Establish Bacteremia in the Infant Rat Model of Invasive Disease.

Genome announcements

VanWagoner TM, Morton DJ, Seale TW, Mussa HJ, Cole BK, Whitby PW, Stull TL.
PMID: 26404588
Genome Announc. 2015 Sep 24;3(5). doi: 10.1128/genomeA.00899-15.

Haemophilus influenzae is an important cause of invasive disease. The infant rat is the accepted model of invasive H. influenzae disease. Here, we report the genome sequences of six nontypeable H. influenzae strains that establish bacteremia in the infant...

Cite
VanWagoner TM, Morton DJ, Seale TW, et al. Draft Genome Sequences of Six Nontypeable Haemophilus influenzae Strains That Establish Bacteremia in the Infant Rat Model of Invasive Disease. Genome Announc. 2015;3(5)doi: 10.1128/genomeA.00899-15.
VanWagoner, T. M., Morton, D. J., Seale, T. W., Mussa, H. J., Cole, B. K., Whitby, P. W., & Stull, T. L. (2015). Draft Genome Sequences of Six Nontypeable Haemophilus influenzae Strains That Establish Bacteremia in the Infant Rat Model of Invasive Disease. Genome announcements, 3(5), . https://doi.org/10.1128/genomeA.00899-15
VanWagoner, Timothy M, et al. "Draft Genome Sequences of Six Nontypeable Haemophilus influenzae Strains That Establish Bacteremia in the Infant Rat Model of Invasive Disease." Genome announcements vol. 3,5 (2015). doi: https://doi.org/10.1128/genomeA.00899-15
VanWagoner TM, Morton DJ, Seale TW, Mussa HJ, Cole BK, Whitby PW, Stull TL. Draft Genome Sequences of Six Nontypeable Haemophilus influenzae Strains That Establish Bacteremia in the Infant Rat Model of Invasive Disease. Genome Announc. 2015 Sep 24;3(5). doi: 10.1128/genomeA.00899-15. PMID: 26404588; PMCID: PMC4582564.
Copy Download .nbib Format: NLM
A model of chronic hepatitis in mice expressing a truncated XRCC1 protein.

Pathobiology of aging & age related diseases

Ge X, Pettan-Brewer C, Morton J, Ladiges WC.
PMID: 25787941
Pathobiol Aging Age Relat Dis. 2015 Mar 17;5:27703. doi: 10.3402/pba.v5.27703. eCollection 2015.

No abstract available.

Cite
Ge X, Pettan-Brewer C, Morton J, et al. A model of chronic hepatitis in mice expressing a truncated XRCC1 protein. Pathobiol Aging Age Relat Dis. 2015;5:27703doi: 10.3402/pba.v5.27703.
Ge, X., Pettan-Brewer, C., Morton, J., & Ladiges, W. C. (2015). A model of chronic hepatitis in mice expressing a truncated XRCC1 protein. Pathobiology of aging & age related diseases, 527703. https://doi.org/10.3402/pba.v5.27703
Ge, Xuan, et al. "A model of chronic hepatitis in mice expressing a truncated XRCC1 protein." Pathobiology of aging & age related diseases vol. 5 (2015): 27703. doi: https://doi.org/10.3402/pba.v5.27703
Ge X, Pettan-Brewer C, Morton J, Ladiges WC. A model of chronic hepatitis in mice expressing a truncated XRCC1 protein. Pathobiol Aging Age Relat Dis. 2015 Mar 17;5:27703. doi: 10.3402/pba.v5.27703. eCollection 2015. PMID: 25787941; PMCID: PMC4365138.
Copy Download .nbib Format: NLM
Glucose intolerance induced by blockade of central FGF receptors is linked to an acute stress response.

Molecular metabolism

Rojas JM, Matsen ME, Mundinger TO, Morton GJ, Stefanovski D, Bergman RN, Kaiyala KJ, Taborsky GJ, Schwartz MW.
PMID: 26266088
Mol Metab. 2015 Jun 04;4(8):561-8. doi: 10.1016/j.molmet.2015.05.005. eCollection 2015 Aug.

OBJECTIVE: Central administration of ligands for fibroblast growth factor receptors (FGFRs) such as fibroblast growth factor-19 (FGF19) and FGF21 exert glucose-lowering effects in rodent models of obesity and type 2 diabetes (T2D). Conversely, intracerebroventricular (icv) administration of the non-selective...

Cite
Rojas JM, Matsen ME, Mundinger TO, et al. Glucose intolerance induced by blockade of central FGF receptors is linked to an acute stress response. Mol Metab. 2015;4(8):561-8doi: 10.1016/j.molmet.2015.05.005.
Rojas, J. M., Matsen, M. E., Mundinger, T. O., Morton, G. J., Stefanovski, D., Bergman, R. N., Kaiyala, K. J., Taborsky, G. J., & Schwartz, M. W. (2015). Glucose intolerance induced by blockade of central FGF receptors is linked to an acute stress response. Molecular metabolism, 4(8), 561-8. https://doi.org/10.1016/j.molmet.2015.05.005
Rojas, Jennifer M, et al. "Glucose intolerance induced by blockade of central FGF receptors is linked to an acute stress response." Molecular metabolism vol. 4,8 (2015): 561-8. doi: https://doi.org/10.1016/j.molmet.2015.05.005
Rojas JM, Matsen ME, Mundinger TO, Morton GJ, Stefanovski D, Bergman RN, Kaiyala KJ, Taborsky GJ, Schwartz MW. Glucose intolerance induced by blockade of central FGF receptors is linked to an acute stress response. Mol Metab. 2015 Jun 04;4(8):561-8. doi: 10.1016/j.molmet.2015.05.005. eCollection 2015 Aug. PMID: 26266088; PMCID: PMC4529498.
Copy Download .nbib Format: NLM
Coherent storage of microwave excitations in rare-earth nuclear spins.

Physical review letters

Wolfowicz G, Maier-Flaig H, Marino R, Ferrier A, Vezin H, Morton JJ, Goldner P.
PMID: 25978214
Phys Rev Lett. 2015 May 01;114(17):170503. doi: 10.1103/PhysRevLett.114.170503. Epub 2015 Apr 30.

Interfacing between various elements of a computer--from memory to processors to long range communication--will be as critical for quantum computers as it is for classical computers today. Paramagnetic rare-earth doped crystals, such as Nd(3+):Y2SiO5(YSO), are excellent candidates for such...

Cite
Wolfowicz G, Maier-Flaig H, Marino R, et al. Coherent storage of microwave excitations in rare-earth nuclear spins. Phys Rev Lett. 2015;114(17):170503doi: 10.1103/PhysRevLett.114.170503.
Wolfowicz, G., Maier-Flaig, H., Marino, R., Ferrier, A., Vezin, H., Morton, J. J., & Goldner, P. (2015). Coherent storage of microwave excitations in rare-earth nuclear spins. Physical review letters, 114(17), 170503. https://doi.org/10.1103/PhysRevLett.114.170503
Wolfowicz, Gary, et al. "Coherent storage of microwave excitations in rare-earth nuclear spins." Physical review letters vol. 114,17 (2015): 170503. doi: https://doi.org/10.1103/PhysRevLett.114.170503
Wolfowicz G, Maier-Flaig H, Marino R, Ferrier A, Vezin H, Morton JJ, Goldner P. Coherent storage of microwave excitations in rare-earth nuclear spins. Phys Rev Lett. 2015 May 01;114(17):170503. doi: 10.1103/PhysRevLett.114.170503. Epub 2015 Apr 30. PMID: 25978214.
Copy Download .nbib Format: NLM
IgA nephropathy in identical twins: an excellent donor and recipient outcome 11 years after transplantation.

Clinical kidney journal

Irvine J, Cross N, Lynn K, Robson R, Searle M, Morton J, Armstrong S, Roake J.
PMID: 26069772
Clin Kidney J. 2012 Jun;5(3):232-3. doi: 10.1093/ckj/sfs022. Epub 2012 Mar 26.

We present a case of IgA nephropathy (IgAN) in monozygotic twins with excellent donor and recipient outcome 11 years after transplantation. The recipient developed IgAN at 26 years and progressed to end-stage renal failure 10 years later. His identical...

Cite
Irvine J, Cross N, Lynn K, et al. IgA nephropathy in identical twins: an excellent donor and recipient outcome 11 years after transplantation. Clin Kidney J. 2012;5(3):232-3doi: 10.1093/ckj/sfs022.
Irvine, J., Cross, N., Lynn, K., Robson, R., Searle, M., Morton, J., Armstrong, S., & Roake, J. (2012). IgA nephropathy in identical twins: an excellent donor and recipient outcome 11 years after transplantation. Clinical kidney journal, 5(3), 232-3. https://doi.org/10.1093/ckj/sfs022
Irvine, John, et al. "IgA nephropathy in identical twins: an excellent donor and recipient outcome 11 years after transplantation." Clinical kidney journal vol. 5,3 (2012): 232-3. doi: https://doi.org/10.1093/ckj/sfs022
Irvine J, Cross N, Lynn K, Robson R, Searle M, Morton J, Armstrong S, Roake J. IgA nephropathy in identical twins: an excellent donor and recipient outcome 11 years after transplantation. Clin Kidney J. 2012 Jun;5(3):232-3. doi: 10.1093/ckj/sfs022. Epub 2012 Mar 26. PMID: 26069772; PMCID: PMC4400500.
Copy Download .nbib Format: NLM
Draft Genome Sequences of Eight Nontypeable Haemophilus influenzae Strains Previously Characterized Using an Electrophoretic Typing Scheme.

Genome announcements

Mussa HJ, VanWagoner TM, Morton DJ, Seale TW, Whitby PW, Stull TL.
PMID: 26607889
Genome Announc. 2015 Nov 25;3(6). doi: 10.1128/genomeA.01374-15.

Nontypeable Haemophilus influenzae is an important cause of human disease. Strains were selected for genome sequencing to represent the breadth of nontypeable strains within the species, as previously defined by the electrophoretic mobility of 16 metabolic enzymes.

Cite
Mussa HJ, VanWagoner TM, Morton DJ, et al. Draft Genome Sequences of Eight Nontypeable Haemophilus influenzae Strains Previously Characterized Using an Electrophoretic Typing Scheme. Genome Announc. 2015;3(6)doi: 10.1128/genomeA.01374-15.
Mussa, H. J., VanWagoner, T. M., Morton, D. J., Seale, T. W., Whitby, P. W., & Stull, T. L. (2015). Draft Genome Sequences of Eight Nontypeable Haemophilus influenzae Strains Previously Characterized Using an Electrophoretic Typing Scheme. Genome announcements, 3(6), . https://doi.org/10.1128/genomeA.01374-15
Mussa, Huda J, et al. "Draft Genome Sequences of Eight Nontypeable Haemophilus influenzae Strains Previously Characterized Using an Electrophoretic Typing Scheme." Genome announcements vol. 3,6 (2015). doi: https://doi.org/10.1128/genomeA.01374-15
Mussa HJ, VanWagoner TM, Morton DJ, Seale TW, Whitby PW, Stull TL. Draft Genome Sequences of Eight Nontypeable Haemophilus influenzae Strains Previously Characterized Using an Electrophoretic Typing Scheme. Genome Announc. 2015 Nov 25;3(6). doi: 10.1128/genomeA.01374-15. PMID: 26607889; PMCID: PMC4661308.
Copy Download .nbib Format: NLM
Deletion of P58(IPK), the Cellular Inhibitor of the Protein Kinases PKR and PERK, Causes Bone Changes and Joint Degeneration in Mice.

Frontiers in endocrinology

Gilbert SJ, Meakin LB, Bonnet CS, Nowell MA, Ladiges WC, Morton J, Duance VC, Mason DJ.
PMID: 25368604
Front Endocrinol (Lausanne). 2014 Oct 17;5:174. doi: 10.3389/fendo.2014.00174. eCollection 2014.

OBJECTIVE: Protein kinase-like endoplasmic reticulum kinase (PERK) and protein kinase R (PKR) are implicated in endoplasmic reticulum stress-induced arthritis and pro-inflammatory cytokine-mediated cartilage degradation in vitro, respectively. We determined whether knockout of the cellular inhibitor of PERK and PKR,...

Cite
Gilbert SJ, Meakin LB, Bonnet CS, et al. Deletion of P58(IPK), the Cellular Inhibitor of the Protein Kinases PKR and PERK, Causes Bone Changes and Joint Degeneration in Mice. Front Endocrinol (Lausanne). 2014;5:174doi: 10.3389/fendo.2014.00174.
Gilbert, S. J., Meakin, L. B., Bonnet, C. S., Nowell, M. A., Ladiges, W. C., Morton, J., Duance, V. C., & Mason, D. J. (2014). Deletion of P58(IPK), the Cellular Inhibitor of the Protein Kinases PKR and PERK, Causes Bone Changes and Joint Degeneration in Mice. Frontiers in endocrinology, 5174. https://doi.org/10.3389/fendo.2014.00174
Gilbert, Sophie J, et al. "Deletion of P58(IPK), the Cellular Inhibitor of the Protein Kinases PKR and PERK, Causes Bone Changes and Joint Degeneration in Mice." Frontiers in endocrinology vol. 5 (2014): 174. doi: https://doi.org/10.3389/fendo.2014.00174
Gilbert SJ, Meakin LB, Bonnet CS, Nowell MA, Ladiges WC, Morton J, Duance VC, Mason DJ. Deletion of P58(IPK), the Cellular Inhibitor of the Protein Kinases PKR and PERK, Causes Bone Changes and Joint Degeneration in Mice. Front Endocrinol (Lausanne). 2014 Oct 17;5:174. doi: 10.3389/fendo.2014.00174. eCollection 2014. PMID: 25368604; PMCID: PMC4201149.
Copy Download .nbib Format: NLM
Integrative veterinary medical education and consensus guidelines for an integrative veterinary medicine curriculum within veterinary colleges.

Open veterinary journal

Memon MA, Shmalberg J, Adair HS, Allweiler S, Bryan JN, Cantwell S, Carr E, Chrisman C, Egger CM, Greene S, Haussler KK, Hershey B, Holyoak GR, Johnson M, Jeune SL, Looney A, McConnico RS, Medina C, Morton AJ, Munsterman A, Nie GJ, Park N, Parsons-Doherty M, Perdrizet JA, Peyton JL, Raditic D, Ramirez HP, Saik J, Robertson S, Sleeper M, Dyke JV, Wakshlag J.
PMID: 27200270
Open Vet J. 2016;6(1):44-56. doi: 10.4314/ovj.v6i1.7. Epub 2016 Mar 28.

Integrative veterinary medicine (IVM) describes the combination of complementary and alternative therapies with conventional care and is guided by the best available evidence. Veterinarians frequently encounter questions about complementary and alternative veterinary medicine (CAVM) in practice, and the general...

Cite
Memon MA, Shmalberg J, Adair HS, et al. Integrative veterinary medical education and consensus guidelines for an integrative veterinary medicine curriculum within veterinary colleges. Open Vet J. 2016;6(1):44-56doi: 10.4314/ovj.v6i1.7.
Memon, M. A., Shmalberg, J., Adair, H. S., Allweiler, S., Bryan, J. N., Cantwell, S., Carr, E., Chrisman, C., Egger, C. M., Greene, S., Haussler, K. K., Hershey, B., Holyoak, G. R., Johnson, M., Jeune, S. L., Looney, A., McConnico, R. S., Medina, C., Morton, A. J., Munsterman, A., Nie, G. J., Park, N., Parsons-Doherty, M., Perdrizet, J. A., Peyton, J. L., Raditic, D., Ramirez, H. P., Saik, J., Robertson, S., Sleeper, M., Dyke, J. V., & Wakshlag, J. (2016). Integrative veterinary medical education and consensus guidelines for an integrative veterinary medicine curriculum within veterinary colleges. Open veterinary journal, 6(1), 44-56. https://doi.org/10.4314/ovj.v6i1.7
Memon, M A, et al. "Integrative veterinary medical education and consensus guidelines for an integrative veterinary medicine curriculum within veterinary colleges." Open veterinary journal vol. 6,1 (2016): 44-56. doi: https://doi.org/10.4314/ovj.v6i1.7
Memon MA, Shmalberg J, Adair HS, Allweiler S, Bryan JN, Cantwell S, Carr E, Chrisman C, Egger CM, Greene S, Haussler KK, Hershey B, Holyoak GR, Johnson M, Jeune SL, Looney A, McConnico RS, Medina C, Morton AJ, Munsterman A, Nie GJ, Park N, Parsons-Doherty M, Perdrizet JA, Peyton JL, Raditic D, Ramirez HP, Saik J, Robertson S, Sleeper M, Dyke JV, Wakshlag J. Integrative veterinary medical education and consensus guidelines for an integrative veterinary medicine curriculum within veterinary colleges. Open Vet J. 2016;6(1):44-56. doi: 10.4314/ovj.v6i1.7. Epub 2016 Mar 28. PMID: 27200270; PMCID: PMC4824037.
Copy Download .nbib Format: NLM
The Investigation of Unexpected Arsenic Compounds Observed in Routine Biological Monitoring Urinary Speciation Analysis.

Toxics

Leese E, Clench M, Morton J, Gardiner PHE, Carolan VA.
PMID: 29051444
Toxics. 2017 May 20;5(2). doi: 10.3390/toxics5020012.

This study investigates the identity of two unexpected arsenic species found separately in a number of urine samples sent to the Health and Safety Executive's Health and Safety Laboratory for arsenic speciation (arsenobetaine, AB; arsenite, As

Cite
Leese E, Clench M, Morton J, et al. The Investigation of Unexpected Arsenic Compounds Observed in Routine Biological Monitoring Urinary Speciation Analysis. Toxics. 2017;5(2)doi: 10.3390/toxics5020012.
Leese, E., Clench, M., Morton, J., Gardiner, P. H. E., & Carolan, V. A. (2017). The Investigation of Unexpected Arsenic Compounds Observed in Routine Biological Monitoring Urinary Speciation Analysis. Toxics, 5(2), . https://doi.org/10.3390/toxics5020012
Leese, Elizabeth, et al. "The Investigation of Unexpected Arsenic Compounds Observed in Routine Biological Monitoring Urinary Speciation Analysis." Toxics vol. 5,2 (2017). doi: https://doi.org/10.3390/toxics5020012
Leese E, Clench M, Morton J, Gardiner PHE, Carolan VA. The Investigation of Unexpected Arsenic Compounds Observed in Routine Biological Monitoring Urinary Speciation Analysis. Toxics. 2017 May 20;5(2). doi: 10.3390/toxics5020012. PMID: 29051444; PMCID: PMC5606668.
Copy Download .nbib Format: NLM
Determinants of Cat Choice and Outcomes for Adult Cats and Kittens Adopted from an Australian Animal Shelter.

Animals : an open access journal from MDPI

Zito S, Paterson M, Vankan D, Morton J, Bennett P, Phillips C.
PMID: 26479236
Animals (Basel). 2015 Apr 29;5(2):276-314. doi: 10.3390/ani5020276.

The percentage of adult cats euthanized in animal shelters is greater than that of kittens because adult cats are less likely to be adopted. This study aimed to provide evidence to inform the design of strategies to encourage adult...

Cite
Zito S, Paterson M, Vankan D, et al. Determinants of Cat Choice and Outcomes for Adult Cats and Kittens Adopted from an Australian Animal Shelter. Animals (Basel). 2015;5(2):276-314doi: 10.3390/ani5020276.
Zito, S., Paterson, M., Vankan, D., Morton, J., Bennett, P., & Phillips, C. (2015). Determinants of Cat Choice and Outcomes for Adult Cats and Kittens Adopted from an Australian Animal Shelter. Animals : an open access journal from MDPI, 5(2), 276-314. https://doi.org/10.3390/ani5020276
Zito, Sarah, et al. "Determinants of Cat Choice and Outcomes for Adult Cats and Kittens Adopted from an Australian Animal Shelter." Animals : an open access journal from MDPI vol. 5,2 (2015): 276-314. doi: https://doi.org/10.3390/ani5020276
Zito S, Paterson M, Vankan D, Morton J, Bennett P, Phillips C. Determinants of Cat Choice and Outcomes for Adult Cats and Kittens Adopted from an Australian Animal Shelter. Animals (Basel). 2015 Apr 29;5(2):276-314. doi: 10.3390/ani5020276. PMID: 26479236; PMCID: PMC4494418.
Copy Download .nbib Format: NLM
Hematopoietic Stem Cell Transplantation for Severe Combined Immunodeficiency.

Current pediatrics reports

Wahlstrom JT, Dvorak CC, Cowan MJ.
PMID: 25821657
Curr Pediatr Rep. 2015 Mar 01;3(1):1-10. doi: 10.1007/s40124-014-0071-7.

Hematopoietic stem cell transplantation (HSCT) is an effective approach for the treatment of severe combined immunodeficiency (SCID). However, SCID is not a homogeneous disease, and the treatment required for successful transplantation varies significantly between SCID subtypes and the degree...

Cite
Wahlstrom JT, Dvorak CC, Cowan MJ. Hematopoietic Stem Cell Transplantation for Severe Combined Immunodeficiency. Curr Pediatr Rep. 2015;3(1):1-10doi: 10.1007/s40124-014-0071-7.
Wahlstrom, J. T., Dvorak, C. C., & Cowan, M. J. (2015). Hematopoietic Stem Cell Transplantation for Severe Combined Immunodeficiency. Current pediatrics reports, 3(1), 1-10. https://doi.org/10.1007/s40124-014-0071-7
Wahlstrom, Justin T, et al. "Hematopoietic Stem Cell Transplantation for Severe Combined Immunodeficiency." Current pediatrics reports vol. 3,1 (2015): 1-10. doi: https://doi.org/10.1007/s40124-014-0071-7
Wahlstrom JT, Dvorak CC, Cowan MJ. Hematopoietic Stem Cell Transplantation for Severe Combined Immunodeficiency. Curr Pediatr Rep. 2015 Mar 01;3(1):1-10. doi: 10.1007/s40124-014-0071-7. PMID: 25821657; PMCID: PMC4371740.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 585 entries