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DNA methylation signature in peripheral blood reveals distinct characteristics of human X chromosome numerical aberrations.

Clinical epigenetics

Sharma A, Jamil MA, Nuesgen N, Schreiner F, Priebe L, Hoffmann P, Herns S, Nöthen MM, Fröhlich H, Oldenburg J, Woelfle J, El-Maarri O.
PMID: 26221191
Clin Epigenetics. 2015 Jul 28;7:76. doi: 10.1186/s13148-015-0112-2. eCollection 2015.

BACKGROUND: Abnormal sex chromosome numbers in humans are observed in Turner (45,X) and Klinefelter (47,XXY) syndromes. Both syndromes are associated with several clinical phenotypes, whose molecular mechanisms are obscure, and show a range of inter-individual penetrance. In order to...

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Sharma A, Jamil MA, Nuesgen N, et al. DNA methylation signature in peripheral blood reveals distinct characteristics of human X chromosome numerical aberrations. Clin Epigenetics. 2015;7:76doi: 10.1186/s13148-015-0112-2.
Sharma, A., Jamil, M. A., Nuesgen, N., Schreiner, F., Priebe, L., Hoffmann, P., Herns, S., Nöthen, M. M., Fröhlich, H., Oldenburg, J., Woelfle, J., & El-Maarri, O. (2015). DNA methylation signature in peripheral blood reveals distinct characteristics of human X chromosome numerical aberrations. Clinical epigenetics, 776. https://doi.org/10.1186/s13148-015-0112-2
Sharma, Amit, et al. "DNA methylation signature in peripheral blood reveals distinct characteristics of human X chromosome numerical aberrations." Clinical epigenetics vol. 7 (2015): 76. doi: https://doi.org/10.1186/s13148-015-0112-2
Sharma A, Jamil MA, Nuesgen N, Schreiner F, Priebe L, Hoffmann P, Herns S, Nöthen MM, Fröhlich H, Oldenburg J, Woelfle J, El-Maarri O. DNA methylation signature in peripheral blood reveals distinct characteristics of human X chromosome numerical aberrations. Clin Epigenetics. 2015 Jul 28;7:76. doi: 10.1186/s13148-015-0112-2. eCollection 2015. PMID: 26221191; PMCID: PMC4517491.
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Identification of increased genetic risk scores for schizophrenia in treatment-resistant patients.

Molecular psychiatry

Frank J, Lang M, Witt SH, Strohmaier J, Rujescu D, Cichon S, Degenhardt F, Nöthen MM, Collier DA, Ripke S, Naber D, Rietschel M.
PMID: 25869806
Mol Psychiatry. 2015 Jul;20(7):913. doi: 10.1038/mp.2015.52. Epub 2015 Apr 14.

No abstract available.

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Frank J, Lang M, Witt SH, et al. Identification of increased genetic risk scores for schizophrenia in treatment-resistant patients. Mol Psychiatry. 2015;20(7):913doi: 10.1038/mp.2015.52.
Frank, J., Lang, M., Witt, S. H., Strohmaier, J., Rujescu, D., Cichon, S., Degenhardt, F., Nöthen, M. M., Collier, D. A., Ripke, S., Naber, D., & Rietschel, M. (2015). Identification of increased genetic risk scores for schizophrenia in treatment-resistant patients. Molecular psychiatry, 20(7), 913. https://doi.org/10.1038/mp.2015.52
Frank, J, et al. "Identification of increased genetic risk scores for schizophrenia in treatment-resistant patients." Molecular psychiatry vol. 20,7 (2015): 913. doi: https://doi.org/10.1038/mp.2015.52
Frank J, Lang M, Witt SH, Strohmaier J, Rujescu D, Cichon S, Degenhardt F, Nöthen MM, Collier DA, Ripke S, Naber D, Rietschel M. Identification of increased genetic risk scores for schizophrenia in treatment-resistant patients. Mol Psychiatry. 2015 Jul;20(7):913. doi: 10.1038/mp.2015.52. Epub 2015 Apr 14. PMID: 25869806.
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Cooperative material transport during the early stage of sintering.

Nature communications

Grupp R, Nöthe M, Kieback B, Banhart J.
PMID: 21540840
Nat Commun. 2011;2:298. doi: 10.1038/ncomms1300.

The complex transport processes contributing to sintering are not yet fully understood, partially because in-situ observations of sintering in three dimensions (3D) are very difficult. Here we report a novel experiment in which monocrystalline copper spheres are first marked...

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Grupp R, Nöthe M, Kieback B, et al. Cooperative material transport during the early stage of sintering. Nat Commun. 2011;2:298doi: 10.1038/ncomms1300.
Grupp, R., Nöthe, M., Kieback, B., & Banhart, J. (2011). Cooperative material transport during the early stage of sintering. Nature communications, 2298. https://doi.org/10.1038/ncomms1300
Grupp, R, et al. "Cooperative material transport during the early stage of sintering." Nature communications vol. 2 (2011): 298. doi: https://doi.org/10.1038/ncomms1300
Grupp R, Nöthe M, Kieback B, Banhart J. Cooperative material transport during the early stage of sintering. Nat Commun. 2011;2:298. doi: 10.1038/ncomms1300. PMID: 21540840.
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Feasible and successful: genome-wide interaction analysis involving all 1.9 x 10(11) pair-wise interaction tests.

Human heredity

Steffens M, Becker T, Sander T, Fimmers R, Herold C, Holler DA, Leu C, Herms S, Cichon S, Bohn B, Gerstner T, Griebel M, Nöthen MM, Wienker TF, Baur MP.
PMID: 20357478
Hum Hered. 2010;69(4):268-84. doi: 10.1159/000295896. Epub 2010 Mar 31.

The Genome-Wide Association Study (GWAS) is the study design of choice for detecting common genetic risk factors for multifactorial diseases. The performance of full Genome-Wide Interaction Analyses (GWIA) has always been considered computationally challenging. Two-stage strategies to reduce the...

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Steffens M, Becker T, Sander T, et al. Feasible and successful: genome-wide interaction analysis involving all 1.9 x 10(11) pair-wise interaction tests. Hum Hered. 2010;69(4):268-84doi: 10.1159/000295896.
Steffens, M., Becker, T., Sander, T., Fimmers, R., Herold, C., Holler, D. A., Leu, C., Herms, S., Cichon, S., Bohn, B., Gerstner, T., Griebel, M., Nöthen, M. M., Wienker, T. F., & Baur, M. P. (2010). Feasible and successful: genome-wide interaction analysis involving all 1.9 x 10(11) pair-wise interaction tests. Human heredity, 69(4), 268-84. https://doi.org/10.1159/000295896
Steffens, Michael, et al. "Feasible and successful: genome-wide interaction analysis involving all 1.9 x 10(11) pair-wise interaction tests." Human heredity vol. 69,4 (2010): 268-84. doi: https://doi.org/10.1159/000295896
Steffens M, Becker T, Sander T, Fimmers R, Herold C, Holler DA, Leu C, Herms S, Cichon S, Bohn B, Gerstner T, Griebel M, Nöthen MM, Wienker TF, Baur MP. Feasible and successful: genome-wide interaction analysis involving all 1.9 x 10(11) pair-wise interaction tests. Hum Hered. 2010;69(4):268-84. doi: 10.1159/000295896. Epub 2010 Mar 31. PMID: 20357478.
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Susceptibility for alcoholism: DRD4 exon III polymorphism: a case-control and a family-based association approach.

Addiction biology

Franke P, Wang T, Möthen MM, Knapp M, Neith H, Lichtermann D, Capellen KM, Sander T, Propping P, Maier W.
PMID: 20575843
Addict Biol. 2000 Jul 01;5(3):289-95. doi: 10.1111/j.1369-1600.2000.tb00193.x.

Abstract The present investigation explored whether the 7-repeat allele of the exon III polymorphism in the dopamine D4 receptor gene confers to susceptibility of alcoholism. Using a classical case-control approach we first compared DRD4 exon III VNTR frequencies between...

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Franke P, Wang T, Möthen MM, et al. Susceptibility for alcoholism: DRD4 exon III polymorphism: a case-control and a family-based association approach. Addict Biol. 2000;5(3):289-95doi: 10.1111/j.1369-1600.2000.tb00193.x.
Franke, P., Wang, T., Möthen, M. M., Knapp, M., Neith, H., Lichtermann, D., Capellen, K. M., Sander, T., Propping, P., & Maier, W. (2000). Susceptibility for alcoholism: DRD4 exon III polymorphism: a case-control and a family-based association approach. Addiction biology, 5(3), 289-95. https://doi.org/10.1111/j.1369-1600.2000.tb00193.x
Franke, P, et al. "Susceptibility for alcoholism: DRD4 exon III polymorphism: a case-control and a family-based association approach." Addiction biology vol. 5,3 (2000): 289-95. doi: https://doi.org/10.1111/j.1369-1600.2000.tb00193.x
Franke P, Wang T, Möthen MM, Knapp M, Neith H, Lichtermann D, Capellen KM, Sander T, Propping P, Maier W. Susceptibility for alcoholism: DRD4 exon III polymorphism: a case-control and a family-based association approach. Addict Biol. 2000 Jul 01;5(3):289-95. doi: 10.1111/j.1369-1600.2000.tb00193.x. PMID: 20575843.
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Author Correction: Genome-wide association study identifies susceptibility loci for B-cell childhood acute lymphoblastic leukemia.

Nature communications

Vijayakrishnan J, Studd J, Broderick P, Kinnersley B, Holroyd A, Law PJ, Kumar R, Allan JM, Harrison CJ, Moorman AV, Vora A, Roman E, Rachakonda S, Kinsey SE, Sheridan E, Thompson PD, Irving JA, Koehler R, Hoffmann P, Nöthen MM, Heilmann-Heimbach S, Jöckel KH, Easton DF, Pharaoh PDP, Dunning AM, Peto J, Canzian F, Swerdlow A, Eeles RA, Kote-Jarai Z, Muir K, Pashayan N, Greaves M, Zimmerman M, Bartram CR, Schrappe M, Stanulla M, Hemminki K, Houlston RS.
PMID: 30664635
Nat Commun. 2019 Jan 21;10(1):419. doi: 10.1038/s41467-018-08106-9.

The original version of this Article contained an error in the spelling of a member of the PRACTICAL Consortium, Manuela Gago-Dominguez, which was incorrectly given as Manuela Gago Dominguez. This has now been corrected in both the PDF and...

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Vijayakrishnan J, Studd J, Broderick P, et al. Author Correction: Genome-wide association study identifies susceptibility loci for B-cell childhood acute lymphoblastic leukemia. Nat Commun. 2019;10(1):419doi: 10.1038/s41467-018-08106-9.
Vijayakrishnan, J., Studd, J., Broderick, P., Kinnersley, B., Holroyd, A., Law, P. J., Kumar, R., Allan, J. M., Harrison, C. J., Moorman, A. V., Vora, A., Roman, E., Rachakonda, S., Kinsey, S. E., Sheridan, E., Thompson, P. D., Irving, J. A., Koehler, R., Hoffmann, P., Nöthen, M. M., Heilmann-Heimbach, S., Jöckel, K. H., Easton, D. F., Pharaoh, P. D. P., Dunning, A. M., Peto, J., Canzian, F., Swerdlow, A., Eeles, R. A., Kote-Jarai, Z., Muir, K., Pashayan, N., Greaves, M., Zimmerman, M., Bartram, C. R., Schrappe, M., Stanulla, M., Hemminki, K., & Houlston, R. S. (2019). Author Correction: Genome-wide association study identifies susceptibility loci for B-cell childhood acute lymphoblastic leukemia. Nature communications, 10(1), 419. https://doi.org/10.1038/s41467-018-08106-9
Vijayakrishnan, Jayaram, et al. "Author Correction: Genome-wide association study identifies susceptibility loci for B-cell childhood acute lymphoblastic leukemia." Nature communications vol. 10,1 (2019): 419. doi: https://doi.org/10.1038/s41467-018-08106-9
Vijayakrishnan J, Studd J, Broderick P, Kinnersley B, Holroyd A, Law PJ, Kumar R, Allan JM, Harrison CJ, Moorman AV, Vora A, Roman E, Rachakonda S, Kinsey SE, Sheridan E, Thompson PD, Irving JA, Koehler R, Hoffmann P, Nöthen MM, Heilmann-Heimbach S, Jöckel KH, Easton DF, Pharaoh PDP, Dunning AM, Peto J, Canzian F, Swerdlow A, Eeles RA, Kote-Jarai Z, Muir K, Pashayan N, Greaves M, Zimmerman M, Bartram CR, Schrappe M, Stanulla M, Hemminki K, Houlston RS. Author Correction: Genome-wide association study identifies susceptibility loci for B-cell childhood acute lymphoblastic leukemia. Nat Commun. 2019 Jan 21;10(1):419. doi: 10.1038/s41467-018-08106-9. PMID: 30664635; PMCID: PMC6341085.
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Retraction for Dixson et al., Identification of gene ontologies linked to prefrontal-hippocampal functional coupling in the human brain.

Proceedings of the National Academy of Sciences of the United States of America

Dixson L, Walter H, Schneider M, Erk S, Schäfer A, Haddad L, Grimm O, Mattheisen M, Nöthen MM, Cichon S, Witt SH, Rietschel M, Mohnke S, Seiferth N, Heinz A, Tost H, Meyer-Lindenberg A.
PMID: 25197092
Proc Natl Acad Sci U S A. 2014 Sep 16;111(37):13582. doi: 10.1073/pnas.1414905111. Epub 2014 Sep 02.

No abstract available.

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Dixson L, Walter H, Schneider M, et al. Retraction for Dixson et al., Identification of gene ontologies linked to prefrontal-hippocampal functional coupling in the human brain. Proc Natl Acad Sci U S A. 2014;111(37):13582doi: 10.1073/pnas.1414905111.
Dixson, L., Walter, H., Schneider, M., Erk, S., Schäfer, A., Haddad, L., Grimm, O., Mattheisen, M., Nöthen, M. M., Cichon, S., Witt, S. H., Rietschel, M., Mohnke, S., Seiferth, N., Heinz, A., Tost, H., & Meyer-Lindenberg, A. (2014). Retraction for Dixson et al., Identification of gene ontologies linked to prefrontal-hippocampal functional coupling in the human brain. Proceedings of the National Academy of Sciences of the United States of America, 111(37), 13582. https://doi.org/10.1073/pnas.1414905111
Dixson, Luanna, et al. "Retraction for Dixson et al., Identification of gene ontologies linked to prefrontal-hippocampal functional coupling in the human brain." Proceedings of the National Academy of Sciences of the United States of America vol. 111,37 (2014): 13582. doi: https://doi.org/10.1073/pnas.1414905111
Dixson L, Walter H, Schneider M, Erk S, Schäfer A, Haddad L, Grimm O, Mattheisen M, Nöthen MM, Cichon S, Witt SH, Rietschel M, Mohnke S, Seiferth N, Heinz A, Tost H, Meyer-Lindenberg A. Retraction for Dixson et al., Identification of gene ontologies linked to prefrontal-hippocampal functional coupling in the human brain. Proc Natl Acad Sci U S A. 2014 Sep 16;111(37):13582. doi: 10.1073/pnas.1414905111. Epub 2014 Sep 02. PMID: 25197092; PMCID: PMC4169929.
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Polygenic risk for schizophrenia and schizotypal traits in non-clinical subjects.

Psychological medicine

Nenadić I, Meller T, Schmitt S, Stein F, Brosch K, Mosebach J, Ettinger U, Grant P, Meinert S, Opel N, Lemke H, Fingas S, Förster K, Hahn T, Jansen A, Andlauer TFM, Forstner AJ, Heilmann-Heimbach S, Hall ASM, Awasthi S, Ripke S, Witt SH, Rietschel M, Müller-Myhsok B, Nöthen MM, Dannlowski U, Krug A, Streit F, Kircher T.
PMID: 32758327
Psychol Med. 2020 Aug 06;1-11. doi: 10.1017/S0033291720002822. Epub 2020 Aug 06.

BACKGROUND: Schizotypy is a putative risk phenotype for psychosis liability, but the overlap of its genetic architecture with schizophrenia is poorly understood.METHODS: We tested the hypothesis that dimensions of schizotypy (assessed with the SPQ-B) are associated with a polygenic...

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Nenadić I, Meller T, Schmitt S, et al. Polygenic risk for schizophrenia and schizotypal traits in non-clinical subjects. Psychol Med. 2020;1-11doi: 10.1017/S0033291720002822.
Nenadić, I., Meller, T., Schmitt, S., Stein, F., Brosch, K., Mosebach, J., Ettinger, U., Grant, P., Meinert, S., Opel, N., Lemke, H., Fingas, S., Förster, K., Hahn, T., Jansen, A., Andlauer, T. F. M., Forstner, A. J., Heilmann-Heimbach, S., Hall, A. S. M., Awasthi, S., Ripke, S., Witt, S. H., Rietschel, M., Müller-Myhsok, B., Nöthen, M. M., Dannlowski, U., Krug, A., Streit, F., & Kircher, T. (2020). Polygenic risk for schizophrenia and schizotypal traits in non-clinical subjects. Psychological medicine, 1-11. https://doi.org/10.1017/S0033291720002822
Nenadić, Igor, et al. "Polygenic risk for schizophrenia and schizotypal traits in non-clinical subjects." Psychological medicine vol. (2020): 1-11. doi: https://doi.org/10.1017/S0033291720002822
Nenadić I, Meller T, Schmitt S, Stein F, Brosch K, Mosebach J, Ettinger U, Grant P, Meinert S, Opel N, Lemke H, Fingas S, Förster K, Hahn T, Jansen A, Andlauer TFM, Forstner AJ, Heilmann-Heimbach S, Hall ASM, Awasthi S, Ripke S, Witt SH, Rietschel M, Müller-Myhsok B, Nöthen MM, Dannlowski U, Krug A, Streit F, Kircher T. Polygenic risk for schizophrenia and schizotypal traits in non-clinical subjects. Psychol Med. 2020 Aug 06;1-11. doi: 10.1017/S0033291720002822. Epub 2020 Aug 06. PMID: 32758327.
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Association between genetic variants of the cholinergic system and postoperative delirium and cognitive dysfunction in elderly patients.

BMC medical genomics

Heinrich M, Sieg M, Kruppa J, Nürnberg P, Schreier PH, Heilmann-Heimbach S, Hoffmann P, Nöthen MM, Janke J, Pischon T, Slooter AJC, Winterer G, Spies CD.
PMID: 34674705
BMC Med Genomics. 2021 Oct 21;14(1):248. doi: 10.1186/s12920-021-01071-1.

BACKGROUND: Postoperative delirium (POD) and postoperative cognitive dysfunction (POCD) are frequent and serious complications after surgery. We aim to investigate the association between genetic variants in cholinergic candidate genes according to the Kyoto encyclopedia of genes and genomes -...

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Heinrich M, Sieg M, Kruppa J, et al. Association between genetic variants of the cholinergic system and postoperative delirium and cognitive dysfunction in elderly patients. BMC Med Genomics. 2021;14(1):248doi: 10.1186/s12920-021-01071-1.
Heinrich, M., Sieg, M., Kruppa, J., Nürnberg, P., Schreier, P. H., Heilmann-Heimbach, S., Hoffmann, P., Nöthen, M. M., Janke, J., Pischon, T., Slooter, A. J. C., Winterer, G., & Spies, C. D. (2021). Association between genetic variants of the cholinergic system and postoperative delirium and cognitive dysfunction in elderly patients. BMC medical genomics, 14(1), 248. https://doi.org/10.1186/s12920-021-01071-1
Heinrich, Maria, et al. "Association between genetic variants of the cholinergic system and postoperative delirium and cognitive dysfunction in elderly patients." BMC medical genomics vol. 14,1 (2021): 248. doi: https://doi.org/10.1186/s12920-021-01071-1
Heinrich M, Sieg M, Kruppa J, Nürnberg P, Schreier PH, Heilmann-Heimbach S, Hoffmann P, Nöthen MM, Janke J, Pischon T, Slooter AJC, Winterer G, Spies CD. Association between genetic variants of the cholinergic system and postoperative delirium and cognitive dysfunction in elderly patients. BMC Med Genomics. 2021 Oct 21;14(1):248. doi: 10.1186/s12920-021-01071-1. PMID: 34674705; PMCID: PMC8529799.
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LAMP-Seq enables sensitive, multiplexed COVID-19 diagnostics using molecular barcoding.

Nature biotechnology

Ludwig KU, Schmithausen RM, Li D, Jacobs ML, Hollstein R, Blumenstock K, Liebing J, Słabicki M, Ben-Shmuel A, Israeli O, Weiss S, Ebert TS, Paran N, Rüdiger W, Wilbring G, Feldman D, Lippke B, Ishorst N, Hochfeld LM, Beins EC, Kaltheuner IH, Schmitz M, Wöhler A, Döhla M, Sib E, Jentzsch M, Borrajo JD, Strecker J, Reinhardt J, Cleary B, Geyer M, Hölzel M, Macrae R, Nöthen MM, Hoffmann P, Exner M, Regev A, Zhang F, Schmid-Burgk JL.
PMID: 34188222
Nat Biotechnol. 2021 Dec;39(12):1556-1562. doi: 10.1038/s41587-021-00966-9. Epub 2021 Jun 29.

Frequent testing of large population groups combined with contact tracing and isolation measures will be crucial for containing Coronavirus Disease 2019 outbreaks. Here we present LAMP-Seq, a modified, highly scalable reverse transcription loop-mediated isothermal amplification (RT-LAMP) method. Unpurified biosamples...

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Ludwig KU, Schmithausen RM, Li D, et al. LAMP-Seq enables sensitive, multiplexed COVID-19 diagnostics using molecular barcoding. Nat Biotechnol. 2021;39(12):1556-1562doi: 10.1038/s41587-021-00966-9.
Ludwig, K. U., Schmithausen, R. M., Li, D., Jacobs, M. L., Hollstein, R., Blumenstock, K., Liebing, J., Słabicki, M., Ben-Shmuel, A., Israeli, O., Weiss, S., Ebert, T. S., Paran, N., Rüdiger, W., Wilbring, G., Feldman, D., Lippke, B., Ishorst, N., Hochfeld, L. M., Beins, E. C., Kaltheuner, I. H., Schmitz, M., Wöhler, A., Döhla, M., Sib, E., Jentzsch, M., Borrajo, J. D., Strecker, J., Reinhardt, J., Cleary, B., Geyer, M., Hölzel, M., Macrae, R., Nöthen, M. M., Hoffmann, P., Exner, M., Regev, A., Zhang, F., & Schmid-Burgk, J. L. (2021). LAMP-Seq enables sensitive, multiplexed COVID-19 diagnostics using molecular barcoding. Nature biotechnology, 39(12), 1556-1562. https://doi.org/10.1038/s41587-021-00966-9
Ludwig, Kerstin U, et al. "LAMP-Seq enables sensitive, multiplexed COVID-19 diagnostics using molecular barcoding." Nature biotechnology vol. 39,12 (2021): 1556-1562. doi: https://doi.org/10.1038/s41587-021-00966-9
Ludwig KU, Schmithausen RM, Li D, Jacobs ML, Hollstein R, Blumenstock K, Liebing J, Słabicki M, Ben-Shmuel A, Israeli O, Weiss S, Ebert TS, Paran N, Rüdiger W, Wilbring G, Feldman D, Lippke B, Ishorst N, Hochfeld LM, Beins EC, Kaltheuner IH, Schmitz M, Wöhler A, Döhla M, Sib E, Jentzsch M, Borrajo JD, Strecker J, Reinhardt J, Cleary B, Geyer M, Hölzel M, Macrae R, Nöthen MM, Hoffmann P, Exner M, Regev A, Zhang F, Schmid-Burgk JL. LAMP-Seq enables sensitive, multiplexed COVID-19 diagnostics using molecular barcoding. Nat Biotechnol. 2021 Dec;39(12):1556-1562. doi: 10.1038/s41587-021-00966-9. Epub 2021 Jun 29. PMID: 34188222; PMCID: PMC8678193.
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Ventral Striatal-Hippocampus Coupling During Reward Processing as a Stratification Biomarker for Psychotic Disorders.

Biological psychiatry

Schwarz K, Moessnang C, Schweiger JI, Harneit A, Schneider M, Chen J, Cao H, Schwarz E, Witt SH, Rietschel M, Nöthen M, Degenhardt F, Wackerhagen C, Erk S, Romanczuk-Seiferth N, Walter H, Tost H, Meyer-Lindenberg A.
PMID: 34607654
Biol Psychiatry. 2021 Jul 24; doi: 10.1016/j.biopsych.2021.07.016. Epub 2021 Jul 24.

BACKGROUND: Altered ventral striatal (vST) activation to reward expectancy is a well-established intermediate phenotype for psychiatric disorders, specifically schizophrenia (SZ). Preclinical research suggests that striatal alterations are related to a reduced inhibition by the hippocampal formation, but its role...

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Schwarz K, Moessnang C, Schweiger JI, et al. Ventral Striatal-Hippocampus Coupling During Reward Processing as a Stratification Biomarker for Psychotic Disorders. Biol Psychiatry. 2021;doi: 10.1016/j.biopsych.2021.07.016.
Schwarz, K., Moessnang, C., Schweiger, J. I., Harneit, A., Schneider, M., Chen, J., Cao, H., Schwarz, E., Witt, S. H., Rietschel, M., Nöthen, M., Degenhardt, F., Wackerhagen, C., Erk, S., Romanczuk-Seiferth, N., Walter, H., Tost, H., & Meyer-Lindenberg, A. (2021). Ventral Striatal-Hippocampus Coupling During Reward Processing as a Stratification Biomarker for Psychotic Disorders. Biological psychiatry, . https://doi.org/10.1016/j.biopsych.2021.07.016
Schwarz, Kristina, et al. "Ventral Striatal-Hippocampus Coupling During Reward Processing as a Stratification Biomarker for Psychotic Disorders." Biological psychiatry vol. (2021). doi: https://doi.org/10.1016/j.biopsych.2021.07.016
Schwarz K, Moessnang C, Schweiger JI, Harneit A, Schneider M, Chen J, Cao H, Schwarz E, Witt SH, Rietschel M, Nöthen M, Degenhardt F, Wackerhagen C, Erk S, Romanczuk-Seiferth N, Walter H, Tost H, Meyer-Lindenberg A. Ventral Striatal-Hippocampus Coupling During Reward Processing as a Stratification Biomarker for Psychotic Disorders. Biol Psychiatry. 2021 Jul 24; doi: 10.1016/j.biopsych.2021.07.016. Epub 2021 Jul 24. PMID: 34607654.
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Publisher Correction: Mendelian randomisation study of the relationship between vitamin D and risk of glioma.

Scientific reports

Takahashi H, Cornish AJ, Sud A, Law PJ, Kinnersley B, Ostrom QT, Labreche K, Eckel-Passow JE, Armstrong GN, Claus EB, Il'yasova D, Schildkraut J, Barnholtz-Sloan JS, Olson SH, Bernstein JL, Lai RK, Schoemaker MJ, Simon M, Hoffmann P, Nöthen MM, Jöckel KH, Chanock S, Rajaraman P, Johansen C, Jenkins RB, Melin BS, Wrensch MR, Sanson M, Bondy ML, Turnbull C, Houlston RS.
PMID: 31118477
Sci Rep. 2019 May 23;9(1):7924. doi: 10.1038/s41598-019-43787-2.

A correction to this article has been published and is linked from the HTML and PDF versions of this paper. The error has been fixed in the paper.

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Takahashi H, Cornish AJ, Sud A, et al. Publisher Correction: Mendelian randomisation study of the relationship between vitamin D and risk of glioma. Sci Rep. 2019;9(1):7924doi: 10.1038/s41598-019-43787-2.
Takahashi, H., Cornish, A. J., Sud, A., Law, P. J., Kinnersley, B., Ostrom, Q. T., Labreche, K., Eckel-Passow, J. E., Armstrong, G. N., Claus, E. B., Il'yasova, D., Schildkraut, J., Barnholtz-Sloan, J. S., Olson, S. H., Bernstein, J. L., Lai, R. K., Schoemaker, M. J., Simon, M., Hoffmann, P., Nöthen, M. M., Jöckel, K. H., Chanock, S., Rajaraman, P., Johansen, C., Jenkins, R. B., Melin, B. S., Wrensch, M. R., Sanson, M., Bondy, M. L., Turnbull, C., & Houlston, R. S. (2019). Publisher Correction: Mendelian randomisation study of the relationship between vitamin D and risk of glioma. Scientific reports, 9(1), 7924. https://doi.org/10.1038/s41598-019-43787-2
Takahashi, Hannah, et al. "Publisher Correction: Mendelian randomisation study of the relationship between vitamin D and risk of glioma." Scientific reports vol. 9,1 (2019): 7924. doi: https://doi.org/10.1038/s41598-019-43787-2
Takahashi H, Cornish AJ, Sud A, Law PJ, Kinnersley B, Ostrom QT, Labreche K, Eckel-Passow JE, Armstrong GN, Claus EB, Il'yasova D, Schildkraut J, Barnholtz-Sloan JS, Olson SH, Bernstein JL, Lai RK, Schoemaker MJ, Simon M, Hoffmann P, Nöthen MM, Jöckel KH, Chanock S, Rajaraman P, Johansen C, Jenkins RB, Melin BS, Wrensch MR, Sanson M, Bondy ML, Turnbull C, Houlston RS. Publisher Correction: Mendelian randomisation study of the relationship between vitamin D and risk of glioma. Sci Rep. 2019 May 23;9(1):7924. doi: 10.1038/s41598-019-43787-2. PMID: 31118477; PMCID: PMC6531429.
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