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Titer-plate formatted continuous flow thermal reactors: Design and performance of a nanoliter reactor.

Sensors and actuators. B, Chemical

Chen PC, Park DS, You BH, Kim N, Park T, Soper SA, Nikitopoulos DE, Murphy MC.
PMID: 20871807
Sens Actuators B Chem. 2010 Aug 06;149(1):291-300. doi: 10.1016/j.snb.2010.05.068.

Arrays of continuous flow thermal reactors were designed, configured, and fabricated in a 96-device (12 × 8) titer-plate format with overall dimensions of 120 mm × 96 mm, with each reactor confined to a 8 mm × 8 mm...

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Chen PC, Park DS, You BH, et al. Titer-plate formatted continuous flow thermal reactors: Design and performance of a nanoliter reactor. Sens Actuators B Chem. 2010;149(1):291-300doi: 10.1016/j.snb.2010.05.068.
Chen, P. C., Park, D. S., You, B. H., Kim, N., Park, T., Soper, S. A., Nikitopoulos, D. E., & Murphy, M. C. (2010). Titer-plate formatted continuous flow thermal reactors: Design and performance of a nanoliter reactor. Sensors and actuators. B, Chemical, 149(1), 291-300. https://doi.org/10.1016/j.snb.2010.05.068
Chen, Pin-Chuan, et al. "Titer-plate formatted continuous flow thermal reactors: Design and performance of a nanoliter reactor." Sensors and actuators. B, Chemical vol. 149,1 (2010): 291-300. doi: https://doi.org/10.1016/j.snb.2010.05.068
Chen PC, Park DS, You BH, Kim N, Park T, Soper SA, Nikitopoulos DE, Murphy MC. Titer-plate formatted continuous flow thermal reactors: Design and performance of a nanoliter reactor. Sens Actuators B Chem. 2010 Aug 06;149(1):291-300. doi: 10.1016/j.snb.2010.05.068. PMID: 20871807; PMCID: PMC2944033.
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Follow-up care after out-of-hospital cardiac arrest: A pilot study of survivors and families' experiences and recommendations.

Resuscitation plus

Mion M, Case R, Smith K, Lilja G, Blennow Nordström E, Swindell P, Nikolopoulou E, Davis J, Farrell K, Gudde E, Karamasis GV, Davies JR, Toff WD, Abella BS, Keeble TR.
PMID: 34386781
Resusc Plus. 2021 Jul 29;7:100154. doi: 10.1016/j.resplu.2021.100154. eCollection 2021 Sep.

BACKGROUND AND OBJECTIVES: Cognitive and physical difficulties are common in survivors of out-of-hospital cardiac arrest (OHCA); both survivors and close family members are also at risk of developing mood disorders. In the UK, dedicated follow-up pathways for OHCA survivors...

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Mion M, Case R, Smith K, et al. Follow-up care after out-of-hospital cardiac arrest: A pilot study of survivors and families' experiences and recommendations. Resusc Plus. 2021;7:100154doi: 10.1016/j.resplu.2021.100154.
Mion, M., Case, R., Smith, K., Lilja, G., Blennow Nordström, E., Swindell, P., Nikolopoulou, E., Davis, J., Farrell, K., Gudde, E., Karamasis, G. V., Davies, J. R., Toff, W. D., Abella, B. S., & Keeble, T. R. (2021). Follow-up care after out-of-hospital cardiac arrest: A pilot study of survivors and families' experiences and recommendations. Resuscitation plus, 7100154. https://doi.org/10.1016/j.resplu.2021.100154
Mion, Marco, et al. "Follow-up care after out-of-hospital cardiac arrest: A pilot study of survivors and families' experiences and recommendations." Resuscitation plus vol. 7 (2021): 100154. doi: https://doi.org/10.1016/j.resplu.2021.100154
Mion M, Case R, Smith K, Lilja G, Blennow Nordström E, Swindell P, Nikolopoulou E, Davis J, Farrell K, Gudde E, Karamasis GV, Davies JR, Toff WD, Abella BS, Keeble TR. Follow-up care after out-of-hospital cardiac arrest: A pilot study of survivors and families' experiences and recommendations. Resusc Plus. 2021 Jul 29;7:100154. doi: 10.1016/j.resplu.2021.100154. eCollection 2021 Sep. PMID: 34386781; PMCID: PMC8342775.
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Convergent somatic mutations in metabolism genes in chronic liver disease.

Nature

Ng SWK, Rouhani FJ, Brunner SF, Brzozowska N, Aitken SJ, Yang M, Abascal F, Moore L, Nikitopoulou E, Chappell L, Leongamornlert D, Ivovic A, Robinson P, Butler T, Sanders MA, Williams N, Coorens THH, Teague J, Raine K, Butler AP, Hooks Y, Wilson B, Birtchnell N, Naylor H, Davies SE, Stratton MR, Martincorena I, Rahbari R, Frezza C, Hoare M, Campbell PJ.
PMID: 34646017
Nature. 2021 Oct;598(7881):473-478. doi: 10.1038/s41586-021-03974-6. Epub 2021 Oct 13.

The progression of chronic liver disease to hepatocellular carcinoma is caused by the acquisition of somatic mutations that affect 20-30 cancer genes

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Ng SWK, Rouhani FJ, Brunner SF, et al. Convergent somatic mutations in metabolism genes in chronic liver disease. Nature. 2021;598(7881):473-478doi: 10.1038/s41586-021-03974-6.
Ng, S. W. K., Rouhani, F. J., Brunner, S. F., Brzozowska, N., Aitken, S. J., Yang, M., Abascal, F., Moore, L., Nikitopoulou, E., Chappell, L., Leongamornlert, D., Ivovic, A., Robinson, P., Butler, T., Sanders, M. A., Williams, N., Coorens, T. H. H., Teague, J., Raine, K., Butler, A. P., Hooks, Y., Wilson, B., Birtchnell, N., Naylor, H., Davies, S. E., Stratton, M. R., Martincorena, I., Rahbari, R., Frezza, C., Hoare, M., & Campbell, P. J. (2021). Convergent somatic mutations in metabolism genes in chronic liver disease. Nature, 598(7881), 473-478. https://doi.org/10.1038/s41586-021-03974-6
Ng, Stanley W K, et al. "Convergent somatic mutations in metabolism genes in chronic liver disease." Nature vol. 598,7881 (2021): 473-478. doi: https://doi.org/10.1038/s41586-021-03974-6
Ng SWK, Rouhani FJ, Brunner SF, Brzozowska N, Aitken SJ, Yang M, Abascal F, Moore L, Nikitopoulou E, Chappell L, Leongamornlert D, Ivovic A, Robinson P, Butler T, Sanders MA, Williams N, Coorens THH, Teague J, Raine K, Butler AP, Hooks Y, Wilson B, Birtchnell N, Naylor H, Davies SE, Stratton MR, Martincorena I, Rahbari R, Frezza C, Hoare M, Campbell PJ. Convergent somatic mutations in metabolism genes in chronic liver disease. Nature. 2021 Oct;598(7881):473-478. doi: 10.1038/s41586-021-03974-6. Epub 2021 Oct 13. PMID: 34646017.
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Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice.

Pulmonary circulation

Nikitopoulou I, Manitsopoulos N, Kotanidou A, Tian X, Petrovic A, Magkou C, Ninou I, Aidinis V, Schermuly RT, Kosanovic D, Orfanos SE.
PMID: 31819797
Pulm Circ. 2019 Nov 15;9(4):2045894019881954. doi: 10.1177/2045894019881954. eCollection 2019.

Pulmonary fibrosis is a progressive disease characterized by disruption of lung architecture and deregulation of the pulmonary function. Prostacyclin, a metabolite of arachidonic acid, is a potential disease mediator since it exerts anti-inflammatory and anti-fibrotic actions. We investigated the...

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Nikitopoulou I, Manitsopoulos N, Kotanidou A, et al. Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice. Pulm Circ. 2019;9(4):2045894019881954doi: 10.1177/2045894019881954.
Nikitopoulou, I., Manitsopoulos, N., Kotanidou, A., Tian, X., Petrovic, A., Magkou, C., Ninou, I., Aidinis, V., Schermuly, R. T., Kosanovic, D., & Orfanos, S. E. (2019). Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice. Pulmonary circulation, 9(4), 2045894019881954. https://doi.org/10.1177/2045894019881954
Nikitopoulou, Ioanna, et al. "Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice." Pulmonary circulation vol. 9,4 (2019): 2045894019881954. doi: https://doi.org/10.1177/2045894019881954
Nikitopoulou I, Manitsopoulos N, Kotanidou A, Tian X, Petrovic A, Magkou C, Ninou I, Aidinis V, Schermuly RT, Kosanovic D, Orfanos SE. Orotracheal treprostinil administration attenuates bleomycin-induced lung injury, vascular remodeling, and fibrosis in mice. Pulm Circ. 2019 Nov 15;9(4):2045894019881954. doi: 10.1177/2045894019881954. eCollection 2019. PMID: 31819797; PMCID: PMC6883672.
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Five-year prospective multi-center cohort study of patients with giant cell arteritis in Greece.

Mediterranean journal of rheumatology

Tsalapaki C, Nikitopoulou E, Boki KA, Boumpas D, Sfikakis PP, Vosvotekas G, Voulgari PV, Vassilopoulos D.
PMID: 32185310
Mediterr J Rheumatol. 2018 Jun 29;29(2):103-105. doi: 10.31138/mjr.29.2.103. eCollection 2018 Jun.

Giant cell arteritis (GCA) is the most common systemic vasculitis in the aged population associated with significant morbidity due to the long term administration of corticosteroids and the presence of various comorbidities. Data regarding its current treatment modalities, comorbidities,...

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Tsalapaki C, Nikitopoulou E, Boki KA, et al. Five-year prospective multi-center cohort study of patients with giant cell arteritis in Greece. Mediterr J Rheumatol. 2018;29(2):103-105doi: 10.31138/mjr.29.2.103.
Tsalapaki, C., Nikitopoulou, E., Boki, K. A., Boumpas, D., Sfikakis, P. P., Vosvotekas, G., Voulgari, P. V., & Vassilopoulos, D. (2018). Five-year prospective multi-center cohort study of patients with giant cell arteritis in Greece. Mediterranean journal of rheumatology, 29(2), 103-105. https://doi.org/10.31138/mjr.29.2.103
Tsalapaki, Christina, et al. "Five-year prospective multi-center cohort study of patients with giant cell arteritis in Greece." Mediterranean journal of rheumatology vol. 29,2 (2018): 103-105. doi: https://doi.org/10.31138/mjr.29.2.103
Tsalapaki C, Nikitopoulou E, Boki KA, Boumpas D, Sfikakis PP, Vosvotekas G, Voulgari PV, Vassilopoulos D. Five-year prospective multi-center cohort study of patients with giant cell arteritis in Greece. Mediterr J Rheumatol. 2018 Jun 29;29(2):103-105. doi: 10.31138/mjr.29.2.103. eCollection 2018 Jun. PMID: 32185310; PMCID: PMC7046071.
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Optical Coherence Tomography Angiography Evolution of Choroidal Neovascular Membrane in Choroidal Rupture Managed by Intravitreal Bevacizumab.

Case reports in ophthalmological medicine

Lorusso M, Micelli Ferrari L, Nikolopoulou E, Micelli Ferrari T.
PMID: 30723562
Case Rep Ophthalmol Med. 2019 Jan 06;2019:5241573. doi: 10.1155/2019/5241573. eCollection 2019.

PURPOSE: To describe a case of a 25-year-old man with choroidal neovascularization (CNV) secondary to traumatic choroidal rupture treated with intravitreal bevacizumab and to evaluate the vascular structure of the area near the traumatic choroidal rupture.METHODS: The patient underwent...

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Lorusso M, Micelli Ferrari L, Nikolopoulou E, et al. Optical Coherence Tomography Angiography Evolution of Choroidal Neovascular Membrane in Choroidal Rupture Managed by Intravitreal Bevacizumab. Case Rep Ophthalmol Med. 2019;2019:5241573doi: 10.1155/2019/5241573.
Lorusso, M., Micelli Ferrari, L., Nikolopoulou, E., & Micelli Ferrari, T. (2019). Optical Coherence Tomography Angiography Evolution of Choroidal Neovascular Membrane in Choroidal Rupture Managed by Intravitreal Bevacizumab. Case reports in ophthalmological medicine, 20195241573. https://doi.org/10.1155/2019/5241573
Lorusso, Massimo, et al. "Optical Coherence Tomography Angiography Evolution of Choroidal Neovascular Membrane in Choroidal Rupture Managed by Intravitreal Bevacizumab." Case reports in ophthalmological medicine vol. 2019 (2019): 5241573. doi: https://doi.org/10.1155/2019/5241573
Lorusso M, Micelli Ferrari L, Nikolopoulou E, Micelli Ferrari T. Optical Coherence Tomography Angiography Evolution of Choroidal Neovascular Membrane in Choroidal Rupture Managed by Intravitreal Bevacizumab. Case Rep Ophthalmol Med. 2019 Jan 06;2019:5241573. doi: 10.1155/2019/5241573. eCollection 2019. PMID: 30723562; PMCID: PMC6339766.
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Spontaneous resolution of optic pit maculopathy: an OCT report.

Therapeutic advances in ophthalmology

Lorusso M, Zito R, Micelli Ferrari L, Nikolopoulou E, Cicinelli MV, Borrelli E, Querques G, Micelli Ferrari T.
PMID: 32923940
Ther Adv Ophthalmol. 2020 Aug 26;12:2515841420950843. doi: 10.1177/2515841420950843. eCollection 2020.

The separation of the vitreous from the optic nerve head and the macula plays a primary role in the spontaneous resolution of optic disc pit (ODP) maculopathy. Optical coherence tomography (OCT) helps in the non-invasive monitoring of this condition,...

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Lorusso M, Zito R, Micelli Ferrari L, et al. Spontaneous resolution of optic pit maculopathy: an OCT report. Ther Adv Ophthalmol. 2020;12:2515841420950843doi: 10.1177/2515841420950843.
Lorusso, M., Zito, R., Micelli Ferrari, L., Nikolopoulou, E., Cicinelli, M. V., Borrelli, E., Querques, G., & Micelli Ferrari, T. (2020). Spontaneous resolution of optic pit maculopathy: an OCT report. Therapeutic advances in ophthalmology, 122515841420950843. https://doi.org/10.1177/2515841420950843
Lorusso, Massimo, et al. "Spontaneous resolution of optic pit maculopathy: an OCT report." Therapeutic advances in ophthalmology vol. 12 (2020): 2515841420950843. doi: https://doi.org/10.1177/2515841420950843
Lorusso M, Zito R, Micelli Ferrari L, Nikolopoulou E, Cicinelli MV, Borrelli E, Querques G, Micelli Ferrari T. Spontaneous resolution of optic pit maculopathy: an OCT report. Ther Adv Ophthalmol. 2020 Aug 26;12:2515841420950843. doi: 10.1177/2515841420950843. eCollection 2020. PMID: 32923940; PMCID: PMC7453435.
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Succinate Anaplerosis Has an Onco-Driving Potential in Prostate Cancer Cells.

Cancers

Sant'Anna-Silva ACB, Perez-Valencia JA, Sciacovelli M, Lalou C, Sarlak S, Tronci L, Nikitopoulou E, Meszaros AT, Frezza C, Rossignol R, Gnaiger E, Klocker H.
PMID: 33917317
Cancers (Basel). 2021 Apr 06;13(7). doi: 10.3390/cancers13071727.

Tumor cells display metabolic alterations when compared to non-transformed cells. These characteristics are crucial for tumor development, maintenance and survival providing energy supplies and molecular precursors. Anaplerosis is the property of replenishing the TCA cycle, the hub of carbon...

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Sant'Anna-Silva ACB, Perez-Valencia JA, Sciacovelli M, et al. Succinate Anaplerosis Has an Onco-Driving Potential in Prostate Cancer Cells. Cancers (Basel). 2021;13(7)doi: 10.3390/cancers13071727.
Sant'Anna-Silva, A. C. B., Perez-Valencia, J. A., Sciacovelli, M., Lalou, C., Sarlak, S., Tronci, L., Nikitopoulou, E., Meszaros, A. T., Frezza, C., Rossignol, R., Gnaiger, E., & Klocker, H. (2021). Succinate Anaplerosis Has an Onco-Driving Potential in Prostate Cancer Cells. Cancers, 13(7), . https://doi.org/10.3390/cancers13071727
Sant'Anna-Silva, Ana Carolina B, et al. "Succinate Anaplerosis Has an Onco-Driving Potential in Prostate Cancer Cells." Cancers vol. 13,7 (2021). doi: https://doi.org/10.3390/cancers13071727
Sant'Anna-Silva ACB, Perez-Valencia JA, Sciacovelli M, Lalou C, Sarlak S, Tronci L, Nikitopoulou E, Meszaros AT, Frezza C, Rossignol R, Gnaiger E, Klocker H. Succinate Anaplerosis Has an Onco-Driving Potential in Prostate Cancer Cells. Cancers (Basel). 2021 Apr 06;13(7). doi: 10.3390/cancers13071727. PMID: 33917317; PMCID: PMC8038717.
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Disruption of the TCA cycle reveals an ATF4-dependent integration of redox and amino acid metabolism.

eLife

Ryan DG, Yang M, Prag HA, Blanco GR, Nikitopoulou E, Segarra-Mondejar M, Powell CA, Young T, Burger N, Miljkovic JL, Minczuk M, Murphy MP, von Kriegsheim A, Frezza C.
PMID: 34939929
Elife. 2021 Dec 23;10. doi: 10.7554/eLife.72593. Epub 2021 Dec 23.

The Tricarboxylic Acid Cycle (TCA) cycle is arguably the most critical metabolic cycle in physiology and exists as an essential interface coordinating cellular metabolism, bioenergetics, and redox homeostasis. Despite decades of research, a comprehensive investigation into the consequences of...

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Ryan DG, Yang M, Prag HA, et al. Disruption of the TCA cycle reveals an ATF4-dependent integration of redox and amino acid metabolism. Elife. 2021;10doi: 10.7554/eLife.72593.
Ryan, D. G., Yang, M., Prag, H. A., Blanco, G. R., Nikitopoulou, E., Segarra-Mondejar, M., Powell, C. A., Young, T., Burger, N., Miljkovic, J. L., Minczuk, M., Murphy, M. P., von Kriegsheim, A., & Frezza, C. (2021). Disruption of the TCA cycle reveals an ATF4-dependent integration of redox and amino acid metabolism. eLife, 10. https://doi.org/10.7554/eLife.72593
Ryan, Dylan Gerard, et al. "Disruption of the TCA cycle reveals an ATF4-dependent integration of redox and amino acid metabolism." eLife vol. 10 (2021). doi: https://doi.org/10.7554/eLife.72593
Ryan DG, Yang M, Prag HA, Blanco GR, Nikitopoulou E, Segarra-Mondejar M, Powell CA, Young T, Burger N, Miljkovic JL, Minczuk M, Murphy MP, von Kriegsheim A, Frezza C. Disruption of the TCA cycle reveals an ATF4-dependent integration of redox and amino acid metabolism. Elife. 2021 Dec 23;10. doi: 10.7554/eLife.72593. Epub 2021 Dec 23. PMID: 34939929.
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Disruption of the TCA cycle reveals an ATF4-dependent integration of redox and amino acid metabolism.

eLife

Ryan DG, Yang M, Prag HA, Blanco GR, Nikitopoulou E, Segarra-Mondejar M, Powell CA, Young T, Burger N, Miljkovic JL, Minczuk M, Murphy MP, von Kriegsheim A, Frezza C.
PMID: 34939929
Elife. 2021 Dec 23;10. doi: 10.7554/eLife.72593.

The Tricarboxylic Acid (TCA) Cycle is arguably the most critical metabolic cycle in physiology and exists as an essential interface coordinating cellular metabolism, bioenergetics, and redox homeostasis. Despite decades of research, a comprehensive investigation into the consequences of TCA...

Cite
Ryan DG, Yang M, Prag HA, et al. Disruption of the TCA cycle reveals an ATF4-dependent integration of redox and amino acid metabolism. Elife. 2021;10doi: 10.7554/eLife.72593.
Ryan, D. G., Yang, M., Prag, H. A., Blanco, G. R., Nikitopoulou, E., Segarra-Mondejar, M., Powell, C. A., Young, T., Burger, N., Miljkovic, J. L., Minczuk, M., Murphy, M. P., von Kriegsheim, A., & Frezza, C. (2021). Disruption of the TCA cycle reveals an ATF4-dependent integration of redox and amino acid metabolism. eLife, 10. https://doi.org/10.7554/eLife.72593
Ryan, Dylan Gerard, et al. "Disruption of the TCA cycle reveals an ATF4-dependent integration of redox and amino acid metabolism." eLife vol. 10 (2021). doi: https://doi.org/10.7554/eLife.72593
Ryan DG, Yang M, Prag HA, Blanco GR, Nikitopoulou E, Segarra-Mondejar M, Powell CA, Young T, Burger N, Miljkovic JL, Minczuk M, Murphy MP, von Kriegsheim A, Frezza C. Disruption of the TCA cycle reveals an ATF4-dependent integration of redox and amino acid metabolism. Elife. 2021 Dec 23;10. doi: 10.7554/eLife.72593. PMID: 34939929; PMCID: PMC8735863.
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Lung tumor growth promotion by tobacco-specific nitrosamines involves the β2-adrenergic receptors-dependent stimulation of mitochondrial REDOX signaling.

Antioxidants & redox signaling

Sarlak S, Lalou C, Bastos Sant'Anna-Silva AC, Mahfouf W, De Luise M, Rousseau B, Izotte J, Claverol S, Lacombe D, Nikitopoulou E, Yang M, Oliveira M, Frezza C, Gasparre G, Rezvani HR, Amoedo ND, Rossignol R.
PMID: 34715750
Antioxid Redox Signal. 2021 Oct 29; doi: 10.1089/ars.2020.8259. Epub 2021 Oct 29.

AIMS: Lung cancer is the leading cause of cancer death worldwide, and tobacco smoking is a recognized major risk factor for lung tumor development. We analyzed the effect of tobacco-specific nitrosamines (TSNAs) on human lung adenocarcinoma metabolic reprogramming, an...

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Sarlak S, Lalou C, Bastos Sant'Anna-Silva AC, et al. Lung tumor growth promotion by tobacco-specific nitrosamines involves the β2-adrenergic receptors-dependent stimulation of mitochondrial REDOX signaling. Antioxid Redox Signal. 2021;doi: 10.1089/ars.2020.8259.
Sarlak, S., Lalou, C., Bastos Sant'Anna-Silva, A. C., Mahfouf, W., De Luise, M., Rousseau, B., Izotte, J., Claverol, S., Lacombe, D., Nikitopoulou, E., Yang, M., Oliveira, M., Frezza, C., Gasparre, G., Rezvani, H. R., Amoedo, N. D., & Rossignol, R. (2021). Lung tumor growth promotion by tobacco-specific nitrosamines involves the β2-adrenergic receptors-dependent stimulation of mitochondrial REDOX signaling. Antioxidants & redox signaling, . https://doi.org/10.1089/ars.2020.8259
Sarlak, Saharnaz, et al. "Lung tumor growth promotion by tobacco-specific nitrosamines involves the β2-adrenergic receptors-dependent stimulation of mitochondrial REDOX signaling." Antioxidants & redox signaling vol. (2021). doi: https://doi.org/10.1089/ars.2020.8259
Sarlak S, Lalou C, Bastos Sant'Anna-Silva AC, Mahfouf W, De Luise M, Rousseau B, Izotte J, Claverol S, Lacombe D, Nikitopoulou E, Yang M, Oliveira M, Frezza C, Gasparre G, Rezvani HR, Amoedo ND, Rossignol R. Lung tumor growth promotion by tobacco-specific nitrosamines involves the β2-adrenergic receptors-dependent stimulation of mitochondrial REDOX signaling. Antioxid Redox Signal. 2021 Oct 29; doi: 10.1089/ars.2020.8259. Epub 2021 Oct 29. PMID: 34715750.
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Modeling of misalignment effects in microfluidic interconnects for modular bio-analytical chip applications.

Electrophoresis

Rani SD, Park T, You BH, Soper SA, Murphy MC, Nikitopoulos DE.
PMID: 23893860
Electrophoresis. 2013 Nov;34(20):2988-95. doi: 10.1002/elps.201300110. Epub 2013 Oct 09.

Minimizing misalignments during the interconnection of microfluidic modules is extremely critical to develop a fully integrated microfluidic device. Misalignments arising during chip-to-chip or world-to-chip interconnections can be greatly detrimental to efficient functioning of microfluidic devices. To address this problem,...

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Rani SD, Park T, You BH, et al. Modeling of misalignment effects in microfluidic interconnects for modular bio-analytical chip applications. Electrophoresis. 2013;34(20):2988-95doi: 10.1002/elps.201300110.
Rani, S. D., Park, T., You, B. H., Soper, S. A., Murphy, M. C., & Nikitopoulos, D. E. (2013). Modeling of misalignment effects in microfluidic interconnects for modular bio-analytical chip applications. Electrophoresis, 34(20), 2988-95. https://doi.org/10.1002/elps.201300110
Rani, Sudheer D, et al. "Modeling of misalignment effects in microfluidic interconnects for modular bio-analytical chip applications." Electrophoresis vol. 34,20 (2013): 2988-95. doi: https://doi.org/10.1002/elps.201300110
Rani SD, Park T, You BH, Soper SA, Murphy MC, Nikitopoulos DE. Modeling of misalignment effects in microfluidic interconnects for modular bio-analytical chip applications. Electrophoresis. 2013 Nov;34(20):2988-95. doi: 10.1002/elps.201300110. Epub 2013 Oct 09. PMID: 23893860; PMCID: PMC4477268.
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