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Showing 1 to 12 of 15 entries
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Epithelial-to-Mesenchymal Transition and MicroRNAs in Lung Cancer.

Cancers

Legras A, Pécuchet N, Imbeaud S, Pallier K, Didelot A, Roussel H, Gibault L, Fabre E, Le Pimpec-Barthes F, Laurent-Puig P, Blons H.
PMID: 28771186
Cancers (Basel). 2017 Aug 03;9(8). doi: 10.3390/cancers9080101.

Despite major advances, non-small cell lung cancer (NSCLC) remains the major cause of cancer-related death in developed countries. Metastasis and drug resistance are the main factors contributing to relapse and death. Epithelial-to-mesenchymal transition (EMT) is a complex molecular and...

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Legras A, Pécuchet N, Imbeaud S, et al. Epithelial-to-Mesenchymal Transition and MicroRNAs in Lung Cancer. Cancers (Basel). 2017;9(8)doi: 10.3390/cancers9080101.
Legras, A., Pécuchet, N., Imbeaud, S., Pallier, K., Didelot, A., Roussel, H., Gibault, L., Fabre, E., Le Pimpec-Barthes, F., Laurent-Puig, P., & Blons, H. (2017). Epithelial-to-Mesenchymal Transition and MicroRNAs in Lung Cancer. Cancers, 9(8), . https://doi.org/10.3390/cancers9080101
Legras, Antoine, et al. "Epithelial-to-Mesenchymal Transition and MicroRNAs in Lung Cancer." Cancers vol. 9,8 (2017). doi: https://doi.org/10.3390/cancers9080101
Legras A, Pécuchet N, Imbeaud S, Pallier K, Didelot A, Roussel H, Gibault L, Fabre E, Le Pimpec-Barthes F, Laurent-Puig P, Blons H. Epithelial-to-Mesenchymal Transition and MicroRNAs in Lung Cancer. Cancers (Basel). 2017 Aug 03;9(8). doi: 10.3390/cancers9080101. PMID: 28771186; PMCID: PMC5575604.
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Retrovesical hydatic cyst with spontaneous fistulisation in the bladder.

BMJ case reports

Mérida-Rodrigo L, Dueñas AE, Badía CM, Cabeza LR, Calero CL, Palliser JP.
PMID: 21686519
BMJ Case Rep. 2009;2009. doi: 10.1136/bcr.09.2008.0991. Epub 2009 Jun 01.

No abstract available.

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Mérida-Rodrigo L, Dueñas AE, Badía CM, et al. Retrovesical hydatic cyst with spontaneous fistulisation in the bladder. BMJ Case Rep. 2009;2009doi: 10.1136/bcr.09.2008.0991.
Mérida-Rodrigo, L., Dueñas, A. E., Badía, C. M., Cabeza, L. R., Calero, C. L., & Palliser, J. P. (2009). Retrovesical hydatic cyst with spontaneous fistulisation in the bladder. BMJ case reports, 2009. https://doi.org/10.1136/bcr.09.2008.0991
Mérida-Rodrigo, Luis, et al. "Retrovesical hydatic cyst with spontaneous fistulisation in the bladder." BMJ case reports vol. 2009 (2009). doi: https://doi.org/10.1136/bcr.09.2008.0991
Mérida-Rodrigo L, Dueñas AE, Badía CM, Cabeza LR, Calero CL, Palliser JP. Retrovesical hydatic cyst with spontaneous fistulisation in the bladder. BMJ Case Rep. 2009;2009. doi: 10.1136/bcr.09.2008.0991. Epub 2009 Jun 01. PMID: 21686519; PMCID: PMC3027747.
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Systematics of cluster-radioactivity-decay constants as suggested by microscopic calculations.

Physical review letters

Blendowske R, Walliser H.
PMID: 10038935
Phys Rev Lett. 1988 Oct 24;61(17):1930-1933. doi: 10.1103/PhysRevLett.61.1930.

No abstract available.

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Blendowske R, Walliser H. Systematics of cluster-radioactivity-decay constants as suggested by microscopic calculations. Phys Rev Lett. 1988;61(17):1930-1933doi: 10.1103/PhysRevLett.61.1930.
Blendowske, R., & Walliser, H. (1988). Systematics of cluster-radioactivity-decay constants as suggested by microscopic calculations. Physical review letters, 61(17), 1930-1933. https://doi.org/10.1103/PhysRevLett.61.1930
Blendowske, and Walliser. "Systematics of cluster-radioactivity-decay constants as suggested by microscopic calculations." Physical review letters vol. 61,17 (1988): 1930-1933. doi: https://doi.org/10.1103/PhysRevLett.61.1930
Blendowske R, Walliser H. Systematics of cluster-radioactivity-decay constants as suggested by microscopic calculations. Phys Rev Lett. 1988 Oct 24;61(17):1930-1933. doi: 10.1103/PhysRevLett.61.1930. PMID: 10038935.
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Effects of prenatal stress on behavioural and neurodevelopmental outcomes are altered by maternal separation in the neonatal period.

Psychoneuroendocrinology

Crombie GK, Palliser HK, Shaw JC, Hodgson DM, Walker DW, Hirst JJ.
PMID: 33333379
Psychoneuroendocrinology. 2021 Feb;124:105060. doi: 10.1016/j.psyneuen.2020.105060. Epub 2020 Nov 26.

BACKGROUND: Chronic psychosocial stress during pregnancy and/or after birth, and the associated elevation in cortisol, is linked with the onset of behavioural disorders in childhood. Previously, prenatal stress has been shown to reduce neurosteroid pathways in the fetus and...

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Crombie GK, Palliser HK, Shaw JC, et al. Effects of prenatal stress on behavioural and neurodevelopmental outcomes are altered by maternal separation in the neonatal period. Psychoneuroendocrinology. 2020;124:105060doi: 10.1016/j.psyneuen.2020.105060.
Crombie, G. K., Palliser, H. K., Shaw, J. C., Hodgson, D. M., Walker, D. W., & Hirst, J. J. (2021). Effects of prenatal stress on behavioural and neurodevelopmental outcomes are altered by maternal separation in the neonatal period. Psychoneuroendocrinology, 124105060. https://doi.org/10.1016/j.psyneuen.2020.105060
Crombie, Gabrielle K, et al. "Effects of prenatal stress on behavioural and neurodevelopmental outcomes are altered by maternal separation in the neonatal period." Psychoneuroendocrinology vol. 124 (2021): 105060. doi: https://doi.org/10.1016/j.psyneuen.2020.105060
Crombie GK, Palliser HK, Shaw JC, Hodgson DM, Walker DW, Hirst JJ. Effects of prenatal stress on behavioural and neurodevelopmental outcomes are altered by maternal separation in the neonatal period. Psychoneuroendocrinology. 2021 Feb;124:105060. doi: 10.1016/j.psyneuen.2020.105060. Epub 2020 Nov 26. PMID: 33333379.
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Comment on "Reverse Brazil nut problem: competition between percolation and condensation".

Physical review letters

Walliser H.
PMID: 12398647
Phys Rev Lett. 2002 Oct 28;89(18):189603; author reply 189604. doi: 10.1103/PhysRevLett.89.189603. Epub 2002 Oct 09.

No abstract available.

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Walliser H. Comment on "Reverse Brazil nut problem: competition between percolation and condensation". Phys Rev Lett. 2002;89(18):189603; author reply 189604doi: 10.1103/PhysRevLett.89.189603.
Walliser, H. (2002). Comment on "Reverse Brazil nut problem: competition between percolation and condensation". Physical review letters, 89(18), 189603; author reply 189604. https://doi.org/10.1103/PhysRevLett.89.189603
Walliser, H. "Comment on "Reverse Brazil nut problem: competition between percolation and condensation"." Physical review letters vol. 89,18 (2002): 189603; author reply 189604. doi: https://doi.org/10.1103/PhysRevLett.89.189603
Walliser H. Comment on "Reverse Brazil nut problem: competition between percolation and condensation". Phys Rev Lett. 2002 Oct 28;89(18):189603; author reply 189604. doi: 10.1103/PhysRevLett.89.189603. Epub 2002 Oct 09. PMID: 12398647.
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Impaired Oligodendrocyte Development Following Preterm Birth: Promoting GABAergic Action to Improve Outcomes.

Frontiers in pediatrics

Shaw JC, Crombie GK, Palliser HK, Hirst JJ.
PMID: 33634057
Front Pediatr. 2021 Feb 04;9:618052. doi: 10.3389/fped.2021.618052. eCollection 2021.

Preterm birth is associated with poor long-term neurodevelopmental and behavioral outcomes, even in the absence of obvious brain injury at the time of birth. In particular, behavioral disorders characterized by inattention, social difficulties and anxiety are common among children...

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Shaw JC, Crombie GK, Palliser HK, et al. Impaired Oligodendrocyte Development Following Preterm Birth: Promoting GABAergic Action to Improve Outcomes. Front Pediatr. 2021;9:618052doi: 10.3389/fped.2021.618052.
Shaw, J. C., Crombie, G. K., Palliser, H. K., & Hirst, J. J. (2021). Impaired Oligodendrocyte Development Following Preterm Birth: Promoting GABAergic Action to Improve Outcomes. Frontiers in pediatrics, 9618052. https://doi.org/10.3389/fped.2021.618052
Shaw, Julia C, et al. "Impaired Oligodendrocyte Development Following Preterm Birth: Promoting GABAergic Action to Improve Outcomes." Frontiers in pediatrics vol. 9 (2021): 618052. doi: https://doi.org/10.3389/fped.2021.618052
Shaw JC, Crombie GK, Palliser HK, Hirst JJ. Impaired Oligodendrocyte Development Following Preterm Birth: Promoting GABAergic Action to Improve Outcomes. Front Pediatr. 2021 Feb 04;9:618052. doi: 10.3389/fped.2021.618052. eCollection 2021. PMID: 33634057; PMCID: PMC7901941.
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Genome wide SNP comparative analysis between EGFR and KRAS mutated NSCLC and characterization of two models of oncogenic cooperation in non-small cell lung carcinoma.

BMC medical genomics

Blons H, Pallier K, Le Corre D, Danel C, Tremblay-Gravel M, Houdayer C, Fabre-Guillevin E, Riquet M, Dessen P, Laurent-Puig P.
PMID: 18549475
BMC Med Genomics. 2008 Jun 12;1:25. doi: 10.1186/1755-8794-1-25.

BACKGROUND: Lung cancer with EGFR mutation was shown to be a specific clinical entity. In order to better understand the biology behind this disease we used a genome wide characterization of loss of heterozygosity and amplification by Single Nucleotide...

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Blons H, Pallier K, Le Corre D, et al. Genome wide SNP comparative analysis between EGFR and KRAS mutated NSCLC and characterization of two models of oncogenic cooperation in non-small cell lung carcinoma. BMC Med Genomics. 2008;1:25doi: 10.1186/1755-8794-1-25.
Blons, H., Pallier, K., Le Corre, D., Danel, C., Tremblay-Gravel, M., Houdayer, C., Fabre-Guillevin, E., Riquet, M., Dessen, P., & Laurent-Puig, P. (2008). Genome wide SNP comparative analysis between EGFR and KRAS mutated NSCLC and characterization of two models of oncogenic cooperation in non-small cell lung carcinoma. BMC medical genomics, 125. https://doi.org/10.1186/1755-8794-1-25
Blons, Hélène, et al. "Genome wide SNP comparative analysis between EGFR and KRAS mutated NSCLC and characterization of two models of oncogenic cooperation in non-small cell lung carcinoma." BMC medical genomics vol. 1 (2008): 25. doi: https://doi.org/10.1186/1755-8794-1-25
Blons H, Pallier K, Le Corre D, Danel C, Tremblay-Gravel M, Houdayer C, Fabre-Guillevin E, Riquet M, Dessen P, Laurent-Puig P. Genome wide SNP comparative analysis between EGFR and KRAS mutated NSCLC and characterization of two models of oncogenic cooperation in non-small cell lung carcinoma. BMC Med Genomics. 2008 Jun 12;1:25. doi: 10.1186/1755-8794-1-25. PMID: 18549475; PMCID: PMC2527324.
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Reduced Neurosteroid Exposure Following Preterm Birth and Its' Contribution to Neurological Impairment: A Novel Avenue for Preventative Therapies.

Frontiers in physiology

Shaw JC, Berry MJ, Dyson RM, Crombie GK, Hirst JJ, Palliser HK.
PMID: 31156466
Front Physiol. 2019 May 15;10:599. doi: 10.3389/fphys.2019.00599. eCollection 2019.

Children born preterm are at an increased risk of developing cognitive problems and neuro-behavioral disorders such as attention deficit hyperactivity disorder (ADHD) and anxiety. Whilst neonates born at all gestational ages, even at term, can experience poor cognitive outcomes...

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Shaw JC, Berry MJ, Dyson RM, et al. Reduced Neurosteroid Exposure Following Preterm Birth and Its' Contribution to Neurological Impairment: A Novel Avenue for Preventative Therapies. Front Physiol. 2019;10:599doi: 10.3389/fphys.2019.00599.
Shaw, J. C., Berry, M. J., Dyson, R. M., Crombie, G. K., Hirst, J. J., & Palliser, H. K. (2019). Reduced Neurosteroid Exposure Following Preterm Birth and Its' Contribution to Neurological Impairment: A Novel Avenue for Preventative Therapies. Frontiers in physiology, 10599. https://doi.org/10.3389/fphys.2019.00599
Shaw, Julia C, et al. "Reduced Neurosteroid Exposure Following Preterm Birth and Its' Contribution to Neurological Impairment: A Novel Avenue for Preventative Therapies." Frontiers in physiology vol. 10 (2019): 599. doi: https://doi.org/10.3389/fphys.2019.00599
Shaw JC, Berry MJ, Dyson RM, Crombie GK, Hirst JJ, Palliser HK. Reduced Neurosteroid Exposure Following Preterm Birth and Its' Contribution to Neurological Impairment: A Novel Avenue for Preventative Therapies. Front Physiol. 2019 May 15;10:599. doi: 10.3389/fphys.2019.00599. eCollection 2019. PMID: 31156466; PMCID: PMC6529563.
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High-density substrate and activation mapping of epicardial ventricular tachycardia during left ventricular assist device implant.

HeartRhythm case reports

Kushnir A, Pallister KH, Chaudhary SB, Cevasco M, Naka Y, Saluja D.
PMID: 33101933
HeartRhythm Case Rep. 2020 Jul 04;6(10):690-693. doi: 10.1016/j.hrcr.2020.06.023. eCollection 2020 Oct.

No abstract available.

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Kushnir A, Pallister KH, Chaudhary SB, et al. High-density substrate and activation mapping of epicardial ventricular tachycardia during left ventricular assist device implant. HeartRhythm Case Rep. 2020;6(10):690-693doi: 10.1016/j.hrcr.2020.06.023.
Kushnir, A., Pallister, K. H., Chaudhary, S. B., Cevasco, M., Naka, Y., & Saluja, D. (2020). High-density substrate and activation mapping of epicardial ventricular tachycardia during left ventricular assist device implant. HeartRhythm case reports, 6(10), 690-693. https://doi.org/10.1016/j.hrcr.2020.06.023
Kushnir, Alexander, et al. "High-density substrate and activation mapping of epicardial ventricular tachycardia during left ventricular assist device implant." HeartRhythm case reports vol. 6,10 (2020): 690-693. doi: https://doi.org/10.1016/j.hrcr.2020.06.023
Kushnir A, Pallister KH, Chaudhary SB, Cevasco M, Naka Y, Saluja D. High-density substrate and activation mapping of epicardial ventricular tachycardia during left ventricular assist device implant. HeartRhythm Case Rep. 2020 Jul 04;6(10):690-693. doi: 10.1016/j.hrcr.2020.06.023. eCollection 2020 Oct. PMID: 33101933; PMCID: PMC7573345.
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Early microvascular changes in the preterm neonate: a comparative study of the human and guinea pig.

Physiological reports

Dyson RM, Palliser HK, Lakkundi A, de Waal K, Latter JL, Clifton VL, Wright IM.
PMID: 25350751
Physiol Rep. 2014 Sep 17;2(9). doi: 10.14814/phy2.12145.

Dysfunction of the transition from fetal to neonatal circulatory systems may be a major contributor to poor outcome following preterm birth. Evidence exists in the human for both a period of low flow between 5 and 11 h and...

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Dyson RM, Palliser HK, Lakkundi A, et al. Early microvascular changes in the preterm neonate: a comparative study of the human and guinea pig. Physiol Rep. 2014;2(9)doi: 10.14814/phy2.12145.
Dyson, R. M., Palliser, H. K., Lakkundi, A., de Waal, K., Latter, J. L., Clifton, V. L., & Wright, I. M. (2014). Early microvascular changes in the preterm neonate: a comparative study of the human and guinea pig. Physiological reports, 2(9), . https://doi.org/10.14814/phy2.12145
Dyson, Rebecca M, et al. "Early microvascular changes in the preterm neonate: a comparative study of the human and guinea pig." Physiological reports vol. 2,9 (2014). doi: https://doi.org/10.14814/phy2.12145
Dyson RM, Palliser HK, Lakkundi A, de Waal K, Latter JL, Clifton VL, Wright IM. Early microvascular changes in the preterm neonate: a comparative study of the human and guinea pig. Physiol Rep. 2014 Sep 17;2(9). doi: 10.14814/phy2.12145. PMID: 25350751; PMCID: PMC4270232.
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Evolutionarily conserved genetic interactions with budding and fission yeast MutS identify orthologous relationships in mismatch repair-deficient cancer cells.

Genome medicine

Tosti E, Katakowski JA, Schaetzlein S, Kim HS, Ryan CJ, Shales M, Roguev A, Krogan NJ, Palliser D, Keogh MC, Edelmann W.
PMID: 25302077
Genome Med. 2014 Sep 17;6(9):68. doi: 10.1186/s13073-014-0068-4. eCollection 2014.

BACKGROUND: The evolutionarily conserved DNA mismatch repair (MMR) system corrects base-substitution and insertion-deletion mutations generated during erroneous replication. The mutation or inactivation of many MMR factors strongly predisposes to cancer, where the resulting tumors often display resistance to standard...

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Tosti E, Katakowski JA, Schaetzlein S, et al. Evolutionarily conserved genetic interactions with budding and fission yeast MutS identify orthologous relationships in mismatch repair-deficient cancer cells. Genome Med. 2014;6(9):68doi: 10.1186/s13073-014-0068-4.
Tosti, E., Katakowski, J. A., Schaetzlein, S., Kim, H. S., Ryan, C. J., Shales, M., Roguev, A., Krogan, N. J., Palliser, D., Keogh, M. C., & Edelmann, W. (2014). Evolutionarily conserved genetic interactions with budding and fission yeast MutS identify orthologous relationships in mismatch repair-deficient cancer cells. Genome medicine, 6(9), 68. https://doi.org/10.1186/s13073-014-0068-4
Tosti, Elena, et al. "Evolutionarily conserved genetic interactions with budding and fission yeast MutS identify orthologous relationships in mismatch repair-deficient cancer cells." Genome medicine vol. 6,9 (2014): 68. doi: https://doi.org/10.1186/s13073-014-0068-4
Tosti E, Katakowski JA, Schaetzlein S, Kim HS, Ryan CJ, Shales M, Roguev A, Krogan NJ, Palliser D, Keogh MC, Edelmann W. Evolutionarily conserved genetic interactions with budding and fission yeast MutS identify orthologous relationships in mismatch repair-deficient cancer cells. Genome Med. 2014 Sep 17;6(9):68. doi: 10.1186/s13073-014-0068-4. eCollection 2014. PMID: 25302077; PMCID: PMC4189729.
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How apoptosis and epithelial-to-mesenchymal transition are nested in EGFR inhibitors resistance in lung cancer.

Journal of thoracic disease

Garinet S, Didelot A, Garelli E, Pallier K, Blons H, Legras A.
PMID: 30863568
J Thorac Dis. 2019 Jan;11(1):47-49. doi: 10.21037/jtd.2018.12.117.

No abstract available.

Cite
Garinet S, Didelot A, Garelli E, et al. How apoptosis and epithelial-to-mesenchymal transition are nested in EGFR inhibitors resistance in lung cancer. J Thorac Dis. 2019;11(1):47-49doi: 10.21037/jtd.2018.12.117.
Garinet, S., Didelot, A., Garelli, E., Pallier, K., Blons, H., & Legras, A. (2019). How apoptosis and epithelial-to-mesenchymal transition are nested in EGFR inhibitors resistance in lung cancer. Journal of thoracic disease, 11(1), 47-49. https://doi.org/10.21037/jtd.2018.12.117
Garinet, Simon, et al. "How apoptosis and epithelial-to-mesenchymal transition are nested in EGFR inhibitors resistance in lung cancer." Journal of thoracic disease vol. 11,1 (2019): 47-49. doi: https://doi.org/10.21037/jtd.2018.12.117
Garinet S, Didelot A, Garelli E, Pallier K, Blons H, Legras A. How apoptosis and epithelial-to-mesenchymal transition are nested in EGFR inhibitors resistance in lung cancer. J Thorac Dis. 2019 Jan;11(1):47-49. doi: 10.21037/jtd.2018.12.117. PMID: 30863568; PMCID: PMC6384333.
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Showing 1 to 12 of 15 entries