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Anorexia nervosa depends on adrenal sympathetic hyperactivity: opposite neuroautonomic profile of hyperinsulinism syndrome.

Diabetes, metabolic syndrome and obesity : targets and therapy

Lechin F, van der Dijs B, Pardey-Maldonado B, Rivera JE, Baez S, Lechin ME.
PMID: 21437100
Diabetes Metab Syndr Obes. 2010 Sep 09;3:311-7. doi: 10.2147/DMSOTT.S10744.

OBJECTIVE: The aim of our study was to determine the central and peripheral autonomic nervous system profiles underlying anorexia nervosa (AN) syndrome, given that affected patients present with the opposite clinical profile to that seen in the hyperinsulinism syndrome.DESIGN:...

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Lechin F, van der Dijs B, Pardey-Maldonado B, et al. Anorexia nervosa depends on adrenal sympathetic hyperactivity: opposite neuroautonomic profile of hyperinsulinism syndrome. Diabetes Metab Syndr Obes. 2010;3:311-7doi: 10.2147/DMSOTT.S10744.
Lechin, F., van der Dijs, B., Pardey-Maldonado, B., Rivera, J. E., Baez, S., & Lechin, M. E. (2010). Anorexia nervosa depends on adrenal sympathetic hyperactivity: opposite neuroautonomic profile of hyperinsulinism syndrome. Diabetes, metabolic syndrome and obesity : targets and therapy, 3311-7. https://doi.org/10.2147/DMSOTT.S10744
Lechin, Fuad, et al. "Anorexia nervosa depends on adrenal sympathetic hyperactivity: opposite neuroautonomic profile of hyperinsulinism syndrome." Diabetes, metabolic syndrome and obesity : targets and therapy vol. 3 (2010): 311-7. doi: https://doi.org/10.2147/DMSOTT.S10744
Lechin F, van der Dijs B, Pardey-Maldonado B, Rivera JE, Baez S, Lechin ME. Anorexia nervosa depends on adrenal sympathetic hyperactivity: opposite neuroautonomic profile of hyperinsulinism syndrome. Diabetes Metab Syndr Obes. 2010 Sep 09;3:311-7. doi: 10.2147/DMSOTT.S10744. PMID: 21437100; PMCID: PMC3047992.
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Anorexia nervosa versus hyperinsulinism: therapeutic effects of neuropharmacological manipulation.

Therapeutics and clinical risk management

Lechin F, van der Dijs B, Pardey-Maldonado B, Baez S, Lechin ME.
PMID: 21445279
Ther Clin Risk Manag. 2011;7:53-8. doi: 10.2147/TCRM.S16958. Epub 2011 Feb 17.

BACKGROUND: We have demonstrated that anorexia nervosa is underpinned by overwhelming adrenal sympathetic activity which abolishes the neural sympathetic branch of the peripheral autonomic nervous system. This physiological disorder is responsible for gastrointestinal hypomotility, hyperglycemia, raised systolic blood pressure,...

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Lechin F, van der Dijs B, Pardey-Maldonado B, et al. Anorexia nervosa versus hyperinsulinism: therapeutic effects of neuropharmacological manipulation. Ther Clin Risk Manag. 2011;7:53-8doi: 10.2147/TCRM.S16958.
Lechin, F., van der Dijs, B., Pardey-Maldonado, B., Baez, S., & Lechin, M. E. (2011). Anorexia nervosa versus hyperinsulinism: therapeutic effects of neuropharmacological manipulation. Therapeutics and clinical risk management, 753-8. https://doi.org/10.2147/TCRM.S16958
Lechin, Fuad, et al. "Anorexia nervosa versus hyperinsulinism: therapeutic effects of neuropharmacological manipulation." Therapeutics and clinical risk management vol. 7 (2011): 53-8. doi: https://doi.org/10.2147/TCRM.S16958
Lechin F, van der Dijs B, Pardey-Maldonado B, Baez S, Lechin ME. Anorexia nervosa versus hyperinsulinism: therapeutic effects of neuropharmacological manipulation. Ther Clin Risk Manag. 2011;7:53-8. doi: 10.2147/TCRM.S16958. Epub 2011 Feb 17. PMID: 21445279; PMCID: PMC3061844.
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Effects of an oral dose of l-glutamic acid on circulating neurotransmitters: Possible roles of the C1(Ad) and the A5(NA) pontomedullary nuclei.

Journal of experimental pharmacology

Lechin F, van der Dijs B, Pardey-Maldonado B, Rivera JE, Lechin ME, Baez S.
PMID: 27186090
J Exp Pharmacol. 2010 Feb 20;2:47-53. doi: 10.2147/jep.s9410. eCollection 2010.

OBJECTIVE: Investigation of the effects of an oral administration of a small dose of l-glutamic acid on the two peripheral sympathetic branches (neural and adrenal) of the autonomic nervous system.RESEARCH DESIGN AND METHODS: Circulating neurotransmitters and cardiovascular parameters were...

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Lechin F, van der Dijs B, Pardey-Maldonado B, et al. Effects of an oral dose of l-glutamic acid on circulating neurotransmitters: Possible roles of the C1(Ad) and the A5(NA) pontomedullary nuclei. J Exp Pharmacol. 2010;2:47-53doi: 10.2147/jep.s9410.
Lechin, F., van der Dijs, B., Pardey-Maldonado, B., Rivera, J. E., Lechin, M. E., & Baez, S. (2010). Effects of an oral dose of l-glutamic acid on circulating neurotransmitters: Possible roles of the C1(Ad) and the A5(NA) pontomedullary nuclei. Journal of experimental pharmacology, 247-53. https://doi.org/10.2147/jep.s9410
Lechin, Fuad, et al. "Effects of an oral dose of l-glutamic acid on circulating neurotransmitters: Possible roles of the C1(Ad) and the A5(NA) pontomedullary nuclei." Journal of experimental pharmacology vol. 2 (2010): 47-53. doi: https://doi.org/10.2147/jep.s9410
Lechin F, van der Dijs B, Pardey-Maldonado B, Rivera JE, Lechin ME, Baez S. Effects of an oral dose of l-glutamic acid on circulating neurotransmitters: Possible roles of the C1(Ad) and the A5(NA) pontomedullary nuclei. J Exp Pharmacol. 2010 Feb 20;2:47-53. doi: 10.2147/jep.s9410. eCollection 2010. PMID: 27186090; PMCID: PMC4863285.
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Amantadine reduces glucagon and enhances insulin secretion throughout the oral glucose tolerance test: central plus peripheral nervous system mechanisms.

Diabetes, metabolic syndrome and obesity : targets and therapy

Lechin F, van der Dijs B, Pardey-Maldonado B, Rivera JE, Lechin ME, Baez S.
PMID: 21437134
Diabetes Metab Syndr Obes. 2009 Dec 02;2:203-13. doi: 10.2147/dmsott.s7606.

OBJECTIVE: The purpose of the trial was to examine the effects of amantadine, a N-methyl-D-aspartate (NMDA) antagonist, on the oral glucose tolerance test (OGTT) plus insulin, glucagon and neurotransmitters circulating levels. Previous findings showed that hyperinsulinism and type 2...

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Lechin F, van der Dijs B, Pardey-Maldonado B, et al. Amantadine reduces glucagon and enhances insulin secretion throughout the oral glucose tolerance test: central plus peripheral nervous system mechanisms. Diabetes Metab Syndr Obes. 2009;2:203-13doi: 10.2147/dmsott.s7606.
Lechin, F., van der Dijs, B., Pardey-Maldonado, B., Rivera, J. E., Lechin, M. E., & Baez, S. (2009). Amantadine reduces glucagon and enhances insulin secretion throughout the oral glucose tolerance test: central plus peripheral nervous system mechanisms. Diabetes, metabolic syndrome and obesity : targets and therapy, 2203-13. https://doi.org/10.2147/dmsott.s7606
Lechin, Fuad, et al. "Amantadine reduces glucagon and enhances insulin secretion throughout the oral glucose tolerance test: central plus peripheral nervous system mechanisms." Diabetes, metabolic syndrome and obesity : targets and therapy vol. 2 (2009): 203-13. doi: https://doi.org/10.2147/dmsott.s7606
Lechin F, van der Dijs B, Pardey-Maldonado B, Rivera JE, Lechin ME, Baez S. Amantadine reduces glucagon and enhances insulin secretion throughout the oral glucose tolerance test: central plus peripheral nervous system mechanisms. Diabetes Metab Syndr Obes. 2009 Dec 02;2:203-13. doi: 10.2147/dmsott.s7606. PMID: 21437134; PMCID: PMC3048001.
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Showing 1 to 4 of 4 entries