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Sociotechnical Systems View of Catalysts that Enable Underrepresented Minority Student Success

Minority STEM

Bisantz A, Pennathur P.
GSID: zqoglEL43kYJ
P Pennathur, B Quiroz, A Bisantz… - Proceedings of the …, 2022 - journals.sagepub.com

… Educational institutions in the US have continued to devote attention and efforts as part of recent DEI initiatives to increasing the participation of URM students in STEM disciplines. …

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Pennathur, P., Quiroz, B., Bisantz, A., Everett, L., & Pennathur, A. (2022, September). Sociotechnical Systems View of Catalysts that Enable Underrepresented Minority Student Success. In Proceedings of the Human Factors and Ergonomics Society Annual Meeting (Vol. 66, No. 1, pp. 126-129). Sage CA: Los Angeles, CA: SAGE Publications.
Pennathur, Priyadarshini, et al. "Sociotechnical Systems View of Catalysts that Enable Underrepresented Minority Student Success." Proceedings of the Human Factors and Ergonomics Society Annual Meeting. Vol. 66. No. 1. Sage CA: Los Angeles, CA: SAGE Publications, 2022.
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Lipidomic Signature of Progression of Chronic Kidney Disease in the Chronic Renal Insufficiency Cohort.

Kidney international reports

Afshinnia F, Rajendiran TM, Karnovsky A, Soni T, Wang X, Xie D, Yang W, Shafi T, Weir MR, He J, Brecklin CS, Rhee EP, Schelling JR, Ojo A, Feldman H, Michailidis G, Pennathur S.
PMID: 28451650
Kidney Int Rep. 2016 Nov;1(4):256-268. doi: 10.1016/j.ekir.2016.08.007. Epub 2016 Aug 18.

INTRODUCTION: Human studies report conflicting results on the predictive power of serum lipids on progression of chronic kidney disease (CKD). We aimed to systematically identify the lipids that predict progression to end-stage kidney disease.METHODS: From the Chronic Renal Insufficiency...

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Afshinnia F, Rajendiran TM, Karnovsky A, et al. Lipidomic Signature of Progression of Chronic Kidney Disease in the Chronic Renal Insufficiency Cohort. Kidney Int Rep. 2016;1(4):256-268doi: 10.1016/j.ekir.2016.08.007.
Afshinnia, F., Rajendiran, T. M., Karnovsky, A., Soni, T., Wang, X., Xie, D., Yang, W., Shafi, T., Weir, M. R., He, J., Brecklin, C. S., Rhee, E. P., Schelling, J. R., Ojo, A., Feldman, H., Michailidis, G., & Pennathur, S. (2016). Lipidomic Signature of Progression of Chronic Kidney Disease in the Chronic Renal Insufficiency Cohort. Kidney international reports, 1(4), 256-268. https://doi.org/10.1016/j.ekir.2016.08.007
Afshinnia, Farsad, et al. "Lipidomic Signature of Progression of Chronic Kidney Disease in the Chronic Renal Insufficiency Cohort." Kidney international reports vol. 1,4 (2016): 256-268. doi: https://doi.org/10.1016/j.ekir.2016.08.007
Afshinnia F, Rajendiran TM, Karnovsky A, Soni T, Wang X, Xie D, Yang W, Shafi T, Weir MR, He J, Brecklin CS, Rhee EP, Schelling JR, Ojo A, Feldman H, Michailidis G, Pennathur S. Lipidomic Signature of Progression of Chronic Kidney Disease in the Chronic Renal Insufficiency Cohort. Kidney Int Rep. 2016 Nov;1(4):256-268. doi: 10.1016/j.ekir.2016.08.007. Epub 2016 Aug 18. PMID: 28451650; PMCID: PMC5402253.
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Targeted Lipidomic and Transcriptomic Analysis Identifies Dysregulated Renal Ceramide Metabolism in a Mouse Model of Diabetic Kidney Disease.

Journal of proteomics & bioinformatics

Sas KM, Nair V, Byun J, Kayampilly P, Zhang H, Saha J, Brosius FC, Kretzler M, Pennathur S.
PMID: 26778897
J Proteomics Bioinform. 2015 Oct; doi: 10.4172/jpb.S14-002. Epub 2015 May 18.

Both type 1 and type 2 diabetes are associated with altered lipid metabolism, which might in part contribute to debilitating complications such as diabetic kidney disease (DKD). Ceramides are bioactive sphingolipids that have been implicated in a variety of...

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Sas KM, Nair V, Byun J, et al. Targeted Lipidomic and Transcriptomic Analysis Identifies Dysregulated Renal Ceramide Metabolism in a Mouse Model of Diabetic Kidney Disease. J Proteomics Bioinform. 2015;doi: 10.4172/jpb.S14-002.
Sas, K. M., Nair, V., Byun, J., Kayampilly, P., Zhang, H., Saha, J., Brosius, F. C., Kretzler, M., & Pennathur, S. (2015). Targeted Lipidomic and Transcriptomic Analysis Identifies Dysregulated Renal Ceramide Metabolism in a Mouse Model of Diabetic Kidney Disease. Journal of proteomics & bioinformatics, . https://doi.org/10.4172/jpb.S14-002
Sas, Kelli M, et al. "Targeted Lipidomic and Transcriptomic Analysis Identifies Dysregulated Renal Ceramide Metabolism in a Mouse Model of Diabetic Kidney Disease." Journal of proteomics & bioinformatics vol. (2015). doi: https://doi.org/10.4172/jpb.S14-002
Sas KM, Nair V, Byun J, Kayampilly P, Zhang H, Saha J, Brosius FC, Kretzler M, Pennathur S. Targeted Lipidomic and Transcriptomic Analysis Identifies Dysregulated Renal Ceramide Metabolism in a Mouse Model of Diabetic Kidney Disease. J Proteomics Bioinform. 2015 Oct; doi: 10.4172/jpb.S14-002. Epub 2015 May 18. PMID: 26778897; PMCID: PMC4712744.
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Automation photometer of Hitachi U-2000 spectrophotometer with RS-232C-based computer.

The Journal of automatic chemistry

Kumar KS, Lakshmi BS, Pennathur G.
PMID: 18924834
J Automat Chem. 1998;20(6):189-93. doi: 10.1155/S1463924698000248.

The interfacing of a commonly used spectrophotometer, the Hitachi U2000, through its RS-232C port to a IBM compatible computer is described. The hardware for data acquisation was designed by suitably modifying readily available materials, and the software was written...

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Kumar KS, Lakshmi BS, Pennathur G. Automation photometer of Hitachi U-2000 spectrophotometer with RS-232C-based computer. J Automat Chem. 1998;20(6):189-93doi: 10.1155/S1463924698000248.
Kumar, K. S., Lakshmi, B. S., & Pennathur, G. (1998). Automation photometer of Hitachi U-2000 spectrophotometer with RS-232C-based computer. The Journal of automatic chemistry, 20(6), 189-93. https://doi.org/10.1155/S1463924698000248
Kumar, K S, et al. "Automation photometer of Hitachi U-2000 spectrophotometer with RS-232C-based computer." The Journal of automatic chemistry vol. 20,6 (1998): 189-93. doi: https://doi.org/10.1155/S1463924698000248
Kumar KS, Lakshmi BS, Pennathur G. Automation photometer of Hitachi U-2000 spectrophotometer with RS-232C-based computer. J Automat Chem. 1998;20(6):189-93. doi: 10.1155/S1463924698000248. PMID: 18924834; PMCID: PMC2548161.
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A novel phosphoprotein analysis scheme for assessing changes in premalignant and malignant breast cell lines using 2D liquid separations, protein microarrays and tandem mass spectrometry.

Proteomics. Clinical applications

Patwa TH, Wang Y, Miller FR, Goodison S, Pennathur S, Barder TJ, Lubman DM.
PMID: 19194518
Proteomics Clin Appl. 2008;3(1):51-66. doi: 10.1002/prca.200800097.

An analysis of phosphorylation changes that occur during cancer progression would provide insights into the molecular pathways responsible for a malignant phenotype. In this study we employed a novel coupling of 2D-liquid separations and protein microarray technology to reveal...

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Patwa TH, Wang Y, Miller FR, et al. A novel phosphoprotein analysis scheme for assessing changes in premalignant and malignant breast cell lines using 2D liquid separations, protein microarrays and tandem mass spectrometry. Proteomics Clin Appl. 2008;3(1):51-66doi: 10.1002/prca.200800097.
Patwa, T. H., Wang, Y., Miller, F. R., Goodison, S., Pennathur, S., Barder, T. J., & Lubman, D. M. (2008). A novel phosphoprotein analysis scheme for assessing changes in premalignant and malignant breast cell lines using 2D liquid separations, protein microarrays and tandem mass spectrometry. Proteomics. Clinical applications, 3(1), 51-66. https://doi.org/10.1002/prca.200800097
Patwa, Tasneem H, et al. "A novel phosphoprotein analysis scheme for assessing changes in premalignant and malignant breast cell lines using 2D liquid separations, protein microarrays and tandem mass spectrometry." Proteomics. Clinical applications vol. 3,1 (2008): 51-66. doi: https://doi.org/10.1002/prca.200800097
Patwa TH, Wang Y, Miller FR, Goodison S, Pennathur S, Barder TJ, Lubman DM. A novel phosphoprotein analysis scheme for assessing changes in premalignant and malignant breast cell lines using 2D liquid separations, protein microarrays and tandem mass spectrometry. Proteomics Clin Appl. 2008;3(1):51-66. doi: 10.1002/prca.200800097. PMID: 19194518; PMCID: PMC2633720.
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Interfacial Polarization and Ionic Structure at the Ionic Liquid-Metal Interface Studied by Vibrational Spectroscopy and Molecular Dynamics Simulations.

The journal of physical chemistry. B

Voegtle MJ, Pal T, Pennathur AK, Menachekanian S, Patrow JG, Sarkar S, Cui Q, Dawlaty JM.
PMID: 33689335
J Phys Chem B. 2021 Mar 18;125(10):2741-2753. doi: 10.1021/acs.jpcb.0c11232. Epub 2021 Mar 09.

Ionic liquids (ILs) have both fundamental and practical value in interfacial science and electrochemistry. However, understanding their behavior near a surface is challenging because of strong Coulomb interactions and large and irregular ionic sizes, which affect both their structure...

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Voegtle MJ, Pal T, Pennathur AK, et al. Interfacial Polarization and Ionic Structure at the Ionic Liquid-Metal Interface Studied by Vibrational Spectroscopy and Molecular Dynamics Simulations. J Phys Chem B. 2021;125(10):2741-2753doi: 10.1021/acs.jpcb.0c11232.
Voegtle, M. J., Pal, T., Pennathur, A. K., Menachekanian, S., Patrow, J. G., Sarkar, S., Cui, Q., & Dawlaty, J. M. (2021). Interfacial Polarization and Ionic Structure at the Ionic Liquid-Metal Interface Studied by Vibrational Spectroscopy and Molecular Dynamics Simulations. The journal of physical chemistry. B, 125(10), 2741-2753. https://doi.org/10.1021/acs.jpcb.0c11232
Voegtle, Matthew J, et al. "Interfacial Polarization and Ionic Structure at the Ionic Liquid-Metal Interface Studied by Vibrational Spectroscopy and Molecular Dynamics Simulations." The journal of physical chemistry. B vol. 125,10 (2021): 2741-2753. doi: https://doi.org/10.1021/acs.jpcb.0c11232
Voegtle MJ, Pal T, Pennathur AK, Menachekanian S, Patrow JG, Sarkar S, Cui Q, Dawlaty JM. Interfacial Polarization and Ionic Structure at the Ionic Liquid-Metal Interface Studied by Vibrational Spectroscopy and Molecular Dynamics Simulations. J Phys Chem B. 2021 Mar 18;125(10):2741-2753. doi: 10.1021/acs.jpcb.0c11232. Epub 2021 Mar 09. PMID: 33689335.
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Oncogenic Activity of Solute Carrier Family 45 Member 2 and Alpha-Methylacyl-Coenzyme A Racemase Gene Fusion Is Mediated by Mitogen-Activated Protein Kinase.

Hepatology communications

Zuo ZH, Yu YP, Ren BG, Liu S, Nelson J, Wang Z, Tao J, Pradhan-Sundd T, Bhargava R, Michalopoulos G, Chen Q, Zhang J, Ma D, Pennathur A, Luketich J, Satdarshan Monga P, Nalesnik M, Luo JH.
PMID: 34505419
Hepatol Commun. 2022 Jan;6(1):209-222. doi: 10.1002/hep4.1724. Epub 2021 Sep 10.

Chromosome rearrangement is one of the hallmarks of human malignancies. Gene fusion is one of the consequences of chromosome rearrangements. In this report, we show that gene fusion between solute carrier family 45 member 2 (SLC45A2) and alpha-methylacyl-coenzyme A...

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Zuo ZH, Yu YP, Ren BG, et al. Oncogenic Activity of Solute Carrier Family 45 Member 2 and Alpha-Methylacyl-Coenzyme A Racemase Gene Fusion Is Mediated by Mitogen-Activated Protein Kinase. Hepatol Commun. 2021;6(1):209-222doi: 10.1002/hep4.1724.
Zuo, Z. H., Yu, Y. P., Ren, B. G., Liu, S., Nelson, J., Wang, Z., Tao, J., Pradhan-Sundd, T., Bhargava, R., Michalopoulos, G., Chen, Q., Zhang, J., Ma, D., Pennathur, A., Luketich, J., Satdarshan Monga, P., Nalesnik, M., & Luo, J. H. (2022). Oncogenic Activity of Solute Carrier Family 45 Member 2 and Alpha-Methylacyl-Coenzyme A Racemase Gene Fusion Is Mediated by Mitogen-Activated Protein Kinase. Hepatology communications, 6(1), 209-222. https://doi.org/10.1002/hep4.1724
Zuo, Ze-Hua, et al. "Oncogenic Activity of Solute Carrier Family 45 Member 2 and Alpha-Methylacyl-Coenzyme A Racemase Gene Fusion Is Mediated by Mitogen-Activated Protein Kinase." Hepatology communications vol. 6,1 (2022): 209-222. doi: https://doi.org/10.1002/hep4.1724
Zuo ZH, Yu YP, Ren BG, Liu S, Nelson J, Wang Z, Tao J, Pradhan-Sundd T, Bhargava R, Michalopoulos G, Chen Q, Zhang J, Ma D, Pennathur A, Luketich J, Satdarshan Monga P, Nalesnik M, Luo JH. Oncogenic Activity of Solute Carrier Family 45 Member 2 and Alpha-Methylacyl-Coenzyme A Racemase Gene Fusion Is Mediated by Mitogen-Activated Protein Kinase. Hepatol Commun. 2022 Jan;6(1):209-222. doi: 10.1002/hep4.1724. Epub 2021 Sep 10. PMID: 34505419; PMCID: PMC8710797.
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Quantitative analysis of γ-glutamylisoleucine, γ-glutamylthreonine, and γ-glutamylvaline in HeLa cells using UHPLC-MS/MS.

Journal of separation science

Thacker JB, He C, Pennathur S.
PMID: 34042281
J Sep Sci. 2021 Aug;44(15):2898-2907. doi: 10.1002/jssc.202001266. Epub 2021 Jun 13.

γ-Glutamylpeptides have been identified as potential biomarkers for a number of diseases including cancer, diabetes, and liver disease. In this study, we developed and validated a novel quantitative analytical strategy for measuring γ-glutamylisoleucine, γ-glutamylthreonine, and γ-glutamylvaline, all of which...

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Thacker JB, He C, Pennathur S. Quantitative analysis of γ-glutamylisoleucine, γ-glutamylthreonine, and γ-glutamylvaline in HeLa cells using UHPLC-MS/MS. J Sep Sci. 2021;44(15):2898-2907doi: 10.1002/jssc.202001266.
Thacker, J. B., He, C., & Pennathur, S. (2021). Quantitative analysis of γ-glutamylisoleucine, γ-glutamylthreonine, and γ-glutamylvaline in HeLa cells using UHPLC-MS/MS. Journal of separation science, 44(15), 2898-2907. https://doi.org/10.1002/jssc.202001266
Thacker, Jonathan B, et al. "Quantitative analysis of γ-glutamylisoleucine, γ-glutamylthreonine, and γ-glutamylvaline in HeLa cells using UHPLC-MS/MS." Journal of separation science vol. 44,15 (2021): 2898-2907. doi: https://doi.org/10.1002/jssc.202001266
Thacker JB, He C, Pennathur S. Quantitative analysis of γ-glutamylisoleucine, γ-glutamylthreonine, and γ-glutamylvaline in HeLa cells using UHPLC-MS/MS. J Sep Sci. 2021 Aug;44(15):2898-2907. doi: 10.1002/jssc.202001266. Epub 2021 Jun 13. PMID: 34042281; PMCID: PMC8363571.
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Short- and long-term effects of perinatal phthalate exposures on metabolic pathways in the mouse liver.

Environmental epigenetics

Neier K, Montrose L, Chen K, Malloy MA, Jones TR, Svoboda LK, Harris C, Song PXK, Pennathur S, Sartor MA, Dolinoy DC.
PMID: 33391822
Environ Epigenet. 2020 Dec 23;6(1):dvaa017. doi: 10.1093/eep/dvaa017. eCollection 2020.

Phthalates have been demonstrated to interfere with metabolism, presumably by interacting with peroxisome proliferator-activated receptors (PPARs). However, mechanisms linking developmental phthalate exposures to long-term metabolic effects have not yet been elucidated. We investigated the hypothesis that developmental phthalate exposure...

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Neier K, Montrose L, Chen K, et al. Short- and long-term effects of perinatal phthalate exposures on metabolic pathways in the mouse liver. Environ Epigenet. 2020;6(1):dvaa017doi: 10.1093/eep/dvaa017.
Neier, K., Montrose, L., Chen, K., Malloy, M. A., Jones, T. R., Svoboda, L. K., Harris, C., Song, P. X. K., Pennathur, S., Sartor, M. A., & Dolinoy, D. C. (2020). Short- and long-term effects of perinatal phthalate exposures on metabolic pathways in the mouse liver. Environmental epigenetics, 6(1), dvaa017. https://doi.org/10.1093/eep/dvaa017
Neier, Kari, et al. "Short- and long-term effects of perinatal phthalate exposures on metabolic pathways in the mouse liver." Environmental epigenetics vol. 6,1 (2020): dvaa017. doi: https://doi.org/10.1093/eep/dvaa017
Neier K, Montrose L, Chen K, Malloy MA, Jones TR, Svoboda LK, Harris C, Song PXK, Pennathur S, Sartor MA, Dolinoy DC. Short- and long-term effects of perinatal phthalate exposures on metabolic pathways in the mouse liver. Environ Epigenet. 2020 Dec 23;6(1):dvaa017. doi: 10.1093/eep/dvaa017. eCollection 2020. PMID: 33391822; PMCID: PMC7757125.
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Oncogenic Activity of Solute Carrier Family 45 Member 2 and Alpha-Methylacyl-Coenzyme A Racemase Gene Fusion Is Mediated by Mitogen-Activated Protein Kinase.

Hepatology communications

Zuo ZH, Yu YP, Ren BG, Liu S, Nelson J, Wang Z, Tao J, Pradhan-Sundd T, Bhargava R, Michalopoulos G, Chen Q, Zhang J, Ma D, Pennathur A, Luketich J, Satdarshan Monga P, Nalesnik M, Luo JH.
PMID: 34505419
Hepatol Commun. 2022 Jan;6(1):209-222. doi: 10.1002/hep4.1724. Epub 2021 Sep 10.

Chromosome rearrangement is one of the hallmarks of human malignancies. Gene fusion is one of the consequences of chromosome rearrangements. In this report, we show that gene fusion between solute carrier family 45 member 2 (SLC45A2) and alpha-methylacyl-coenzyme A...

Cite
Zuo ZH, Yu YP, Ren BG, et al. Oncogenic Activity of Solute Carrier Family 45 Member 2 and Alpha-Methylacyl-Coenzyme A Racemase Gene Fusion Is Mediated by Mitogen-Activated Protein Kinase. Hepatol Commun. 2021;6(1):209-222doi: 10.1002/hep4.1724.
Zuo, Z. H., Yu, Y. P., Ren, B. G., Liu, S., Nelson, J., Wang, Z., Tao, J., Pradhan-Sundd, T., Bhargava, R., Michalopoulos, G., Chen, Q., Zhang, J., Ma, D., Pennathur, A., Luketich, J., Satdarshan Monga, P., Nalesnik, M., & Luo, J. H. (2022). Oncogenic Activity of Solute Carrier Family 45 Member 2 and Alpha-Methylacyl-Coenzyme A Racemase Gene Fusion Is Mediated by Mitogen-Activated Protein Kinase. Hepatology communications, 6(1), 209-222. https://doi.org/10.1002/hep4.1724
Zuo, Ze-Hua, et al. "Oncogenic Activity of Solute Carrier Family 45 Member 2 and Alpha-Methylacyl-Coenzyme A Racemase Gene Fusion Is Mediated by Mitogen-Activated Protein Kinase." Hepatology communications vol. 6,1 (2022): 209-222. doi: https://doi.org/10.1002/hep4.1724
Zuo ZH, Yu YP, Ren BG, Liu S, Nelson J, Wang Z, Tao J, Pradhan-Sundd T, Bhargava R, Michalopoulos G, Chen Q, Zhang J, Ma D, Pennathur A, Luketich J, Satdarshan Monga P, Nalesnik M, Luo JH. Oncogenic Activity of Solute Carrier Family 45 Member 2 and Alpha-Methylacyl-Coenzyme A Racemase Gene Fusion Is Mediated by Mitogen-Activated Protein Kinase. Hepatol Commun. 2022 Jan;6(1):209-222. doi: 10.1002/hep4.1724. Epub 2021 Sep 10. PMID: 34505419.
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How lapse and slip errors influence head-of-bed angle compliance rates as measured by a portable, wireless data collection system.

IIE transactions on healthcare systems engineering

Thomas GW, Pennathur P, Falk DM, Myers J, Ayres B, Polgreen PM.
PMID: 31168335
IIE Trans Healthc Syst Eng. 2015;5(1):1-13. doi: 10.1080/19488300.2014.993005. Epub 2015 Mar 16.

The recommended protocols to prevent ventilator-associated pneumonia include keeping ventilated patients' head and upper body elevated to an angle between 30 and 45 degrees. These recommendations are largely based on a study that has been difficult to replicate, because...

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Thomas GW, Pennathur P, Falk DM, et al. How lapse and slip errors influence head-of-bed angle compliance rates as measured by a portable, wireless data collection system. IIE Trans Healthc Syst Eng. 2015;5(1):1-13doi: 10.1080/19488300.2014.993005.
Thomas, G. W., Pennathur, P., Falk, D. M., Myers, J., Ayres, B., & Polgreen, P. M. (2015). How lapse and slip errors influence head-of-bed angle compliance rates as measured by a portable, wireless data collection system. IIE transactions on healthcare systems engineering, 5(1), 1-13. https://doi.org/10.1080/19488300.2014.993005
Thomas, Geb W, et al. "How lapse and slip errors influence head-of-bed angle compliance rates as measured by a portable, wireless data collection system." IIE transactions on healthcare systems engineering vol. 5,1 (2015): 1-13. doi: https://doi.org/10.1080/19488300.2014.993005
Thomas GW, Pennathur P, Falk DM, Myers J, Ayres B, Polgreen PM. How lapse and slip errors influence head-of-bed angle compliance rates as measured by a portable, wireless data collection system. IIE Trans Healthc Syst Eng. 2015;5(1):1-13. doi: 10.1080/19488300.2014.993005. Epub 2015 Mar 16. PMID: 31168335; PMCID: PMC6546299.
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Altered HDL proteome predicts incident CVD in chronic kidney disease patients.

Journal of lipid research

Shao B, Mathew AV, Thornock C, Pennathur S.
PMID: 34634315
J Lipid Res. 2021;62:100135. doi: 10.1016/j.jlr.2021.100135. Epub 2021 Oct 09.

Patients with chronic kidney disease (CKD) are at high risk for CVD. However, traditional lipid risk factors, including low HDL levels, cannot completely explain the increased risk. Altered HDL proteome is linked with both CVD and CKD, but the...

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Shao B, Mathew AV, Thornock C, et al. Altered HDL proteome predicts incident CVD in chronic kidney disease patients. J Lipid Res. 2021;62:100135doi: 10.1016/j.jlr.2021.100135.
Shao, B., Mathew, A. V., Thornock, C., Pennathur, S. (2021). Altered HDL proteome predicts incident CVD in chronic kidney disease patients. Journal of lipid research, 62100135. https://doi.org/10.1016/j.jlr.2021.100135
Shao, Baohai, et al. "Altered HDL proteome predicts incident CVD in chronic kidney disease patients." Journal of lipid research vol. 62 (2021): 100135. doi: https://doi.org/10.1016/j.jlr.2021.100135
Shao B, Mathew AV, Thornock C, Pennathur S. Altered HDL proteome predicts incident CVD in chronic kidney disease patients. J Lipid Res. 2021;62:100135. doi: 10.1016/j.jlr.2021.100135. Epub 2021 Oct 09. PMID: 34634315; PMCID: PMC8566900.
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