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Hydroxycobalamin Reveals the Involvement of Hydrogen Sulfide in the Hypoxic Responses of Rat Carotid Body Chemoreceptor Cells.

Antioxidants (Basel, Switzerland)

Gallego-Martin T, Prieto-Lloret J, Aaronson PI, Rocher A, Obeso A.
PMID: 30871199
Antioxidants (Basel). 2019 Mar 13;8(3). doi: 10.3390/antiox8030062.

Carotid body (CB) chemoreceptor cells sense arterial blood PO₂, generating a neurosecretory response proportional to the intensity of hypoxia. Hydrogen sulfide (H₂S) is a physiological gaseous messenger that is proposed to act as an oxygen sensor in CBs, although...

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Gallego-Martin T, Prieto-Lloret J, Aaronson PI, et al. Hydroxycobalamin Reveals the Involvement of Hydrogen Sulfide in the Hypoxic Responses of Rat Carotid Body Chemoreceptor Cells. Antioxidants (Basel). 2019;8(3)doi: 10.3390/antiox8030062.
Gallego-Martin, T., Prieto-Lloret, J., Aaronson, P. I., Rocher, A., & Obeso, A. (2019). Hydroxycobalamin Reveals the Involvement of Hydrogen Sulfide in the Hypoxic Responses of Rat Carotid Body Chemoreceptor Cells. Antioxidants (Basel, Switzerland), 8(3), . https://doi.org/10.3390/antiox8030062
Gallego-Martin, Teresa, et al. "Hydroxycobalamin Reveals the Involvement of Hydrogen Sulfide in the Hypoxic Responses of Rat Carotid Body Chemoreceptor Cells." Antioxidants (Basel, Switzerland) vol. 8,3 (2019). doi: https://doi.org/10.3390/antiox8030062
Gallego-Martin T, Prieto-Lloret J, Aaronson PI, Rocher A, Obeso A. Hydroxycobalamin Reveals the Involvement of Hydrogen Sulfide in the Hypoxic Responses of Rat Carotid Body Chemoreceptor Cells. Antioxidants (Basel). 2019 Mar 13;8(3). doi: 10.3390/antiox8030062. PMID: 30871199; PMCID: PMC6466535.
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Guinea Pig as a Model to Study the Carotid Body Mediated Chronic Intermittent Hypoxia Effects.

Frontiers in physiology

Docio I, Olea E, Prieto-LLoret J, Gallego-Martin T, Obeso A, Gomez-Niño A, Rocher A.
PMID: 29922183
Front Physiol. 2018 Jun 05;9:694. doi: 10.3389/fphys.2018.00694. eCollection 2018.

Clinical and experimental evidence indicates a positive correlation between chronic intermittent hypoxia (CIH), increased carotid body (CB) chemosensitivity, enhanced sympatho-respiratory coupling and arterial hypertension and cardiovascular disease. Several groups have reported that both the afferent and efferent arms of...

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Docio I, Olea E, Prieto-LLoret J, et al. Guinea Pig as a Model to Study the Carotid Body Mediated Chronic Intermittent Hypoxia Effects. Front Physiol. 2018;9:694doi: 10.3389/fphys.2018.00694.
Docio, I., Olea, E., Prieto-LLoret, J., Gallego-Martin, T., Obeso, A., Gomez-Niño, A., & Rocher, A. (2018). Guinea Pig as a Model to Study the Carotid Body Mediated Chronic Intermittent Hypoxia Effects. Frontiers in physiology, 9694. https://doi.org/10.3389/fphys.2018.00694
Docio, Inmaculada, et al. "Guinea Pig as a Model to Study the Carotid Body Mediated Chronic Intermittent Hypoxia Effects." Frontiers in physiology vol. 9 (2018): 694. doi: https://doi.org/10.3389/fphys.2018.00694
Docio I, Olea E, Prieto-LLoret J, Gallego-Martin T, Obeso A, Gomez-Niño A, Rocher A. Guinea Pig as a Model to Study the Carotid Body Mediated Chronic Intermittent Hypoxia Effects. Front Physiol. 2018 Jun 05;9:694. doi: 10.3389/fphys.2018.00694. eCollection 2018. PMID: 29922183; PMCID: PMC5996279.
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l-Phenylalanine Restores Vascular Function in Spontaneously Hypertensive Rats Through Activation of the GCH1-GFRP Complex.

JACC. Basic to translational science

Heikal L, Starr A, Hussein D, Prieto-Lloret J, Aaronson P, Dailey LA, Nandi M.
PMID: 29963647
JACC Basic Transl Sci. 2018 May 30;3(3):366-377. doi: 10.1016/j.jacbts.2018.01.015. eCollection 2018 Jun.

Reduced nitric oxide (NO) bioavailability correlates with impaired cardiovascular function. NO is extremely labile and has been challenging to develop as a therapeutic agent. However, NO bioavailability could be enhanced by pharmacologically targeting endogenous NO regulatory pathways. Tetrahydrobiopterin, an...

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Heikal L, Starr A, Hussein D, et al. l-Phenylalanine Restores Vascular Function in Spontaneously Hypertensive Rats Through Activation of the GCH1-GFRP Complex. JACC Basic Transl Sci. 2018;3(3):366-377doi: 10.1016/j.jacbts.2018.01.015.
Heikal, L., Starr, A., Hussein, D., Prieto-Lloret, J., Aaronson, P., Dailey, L. A., & Nandi, M. (2018). l-Phenylalanine Restores Vascular Function in Spontaneously Hypertensive Rats Through Activation of the GCH1-GFRP Complex. JACC. Basic to translational science, 3(3), 366-377. https://doi.org/10.1016/j.jacbts.2018.01.015
Heikal, Lamia, et al. "l-Phenylalanine Restores Vascular Function in Spontaneously Hypertensive Rats Through Activation of the GCH1-GFRP Complex." JACC. Basic to translational science vol. 3,3 (2018): 366-377. doi: https://doi.org/10.1016/j.jacbts.2018.01.015
Heikal L, Starr A, Hussein D, Prieto-Lloret J, Aaronson P, Dailey LA, Nandi M. l-Phenylalanine Restores Vascular Function in Spontaneously Hypertensive Rats Through Activation of the GCH1-GFRP Complex. JACC Basic Transl Sci. 2018 May 30;3(3):366-377. doi: 10.1016/j.jacbts.2018.01.015. eCollection 2018 Jun. PMID: 29963647; PMCID: PMC6018612.
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Chronic Intermittent Hypoxia Induces Early-Stage Metabolic Dysfunction Independently of Adipose Tissue Deregulation.

Antioxidants (Basel, Switzerland)

Martins FO, Sacramento JF, Olea E, Melo BF, Prieto-Lloret J, Obeso A, Rocher A, Matafome P, Monteiro EC, Conde SV.
PMID: 34439481
Antioxidants (Basel). 2021 Jul 30;10(8). doi: 10.3390/antiox10081233.

Several studies demonstrated a link between obstructive sleep apnea (OSA) and the development of insulin resistance. However, the main event triggering insulin resistance in OSA remains to be clarified. Herein, we investigated the effect of mild and severe chronic...

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Martins FO, Sacramento JF, Olea E, et al. Chronic Intermittent Hypoxia Induces Early-Stage Metabolic Dysfunction Independently of Adipose Tissue Deregulation. Antioxidants (Basel). 2021;10(8)doi: 10.3390/antiox10081233.
Martins, F. O., Sacramento, J. F., Olea, E., Melo, B. F., Prieto-Lloret, J., Obeso, A., Rocher, A., Matafome, P., Monteiro, E. C., & Conde, S. V. (2021). Chronic Intermittent Hypoxia Induces Early-Stage Metabolic Dysfunction Independently of Adipose Tissue Deregulation. Antioxidants (Basel, Switzerland), 10(8), . https://doi.org/10.3390/antiox10081233
Martins, Fátima O, et al. "Chronic Intermittent Hypoxia Induces Early-Stage Metabolic Dysfunction Independently of Adipose Tissue Deregulation." Antioxidants (Basel, Switzerland) vol. 10,8 (2021). doi: https://doi.org/10.3390/antiox10081233
Martins FO, Sacramento JF, Olea E, Melo BF, Prieto-Lloret J, Obeso A, Rocher A, Matafome P, Monteiro EC, Conde SV. Chronic Intermittent Hypoxia Induces Early-Stage Metabolic Dysfunction Independently of Adipose Tissue Deregulation. Antioxidants (Basel). 2021 Jul 30;10(8). doi: 10.3390/antiox10081233. PMID: 34439481; PMCID: PMC8388878.
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Showing 1 to 4 of 4 entries