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Hyperuricemia in Kidney Disease: A Major Risk Factor for Cardiovascular Events, Vascular Calcification, and Renal Damage.

Seminars in nephrology

Ejaz AA, Nakagawa T, Kanbay M, Kuwabara M, Kumar A, Garcia Arroyo FE, Roncal-Jimenez C, Sasai F, Kang DH, Jensen T, Hernando AA, Rodriguez-Iturbe B, Garcia G, Tolan DR, Sanchez-Lozada LG, Lanaspa MA, Johnson RJ.
PMID: 33678312
Semin Nephrol. 2020 Nov;40(6):574-585. doi: 10.1016/j.semnephrol.2020.12.004.

Kidney disease, especially when it is associated with a reduction in estimated glomerular filtration rate, can be associated with an increase in serum urate (uric acid), suggesting that hyperuricemia in subjects with kidney disease may be a strictly secondary...

Cite
Ejaz AA, Nakagawa T, Kanbay M, et al. Hyperuricemia in Kidney Disease: A Major Risk Factor for Cardiovascular Events, Vascular Calcification, and Renal Damage. Semin Nephrol. 2020;40(6):574-585doi: 10.1016/j.semnephrol.2020.12.004.
Ejaz, A. A., Nakagawa, T., Kanbay, M., Kuwabara, M., Kumar, A., Garcia Arroyo, F. E., Roncal-Jimenez, C., Sasai, F., Kang, D. H., Jensen, T., Hernando, A. A., Rodriguez-Iturbe, B., Garcia, G., Tolan, D. R., Sanchez-Lozada, L. G., Lanaspa, M. A., & Johnson, R. J. (2020). Hyperuricemia in Kidney Disease: A Major Risk Factor for Cardiovascular Events, Vascular Calcification, and Renal Damage. Seminars in nephrology, 40(6), 574-585. https://doi.org/10.1016/j.semnephrol.2020.12.004
Ejaz, Abutaleb Ahsan, et al. "Hyperuricemia in Kidney Disease: A Major Risk Factor for Cardiovascular Events, Vascular Calcification, and Renal Damage." Seminars in nephrology vol. 40,6 (2020): 574-585. doi: https://doi.org/10.1016/j.semnephrol.2020.12.004
Ejaz AA, Nakagawa T, Kanbay M, Kuwabara M, Kumar A, Garcia Arroyo FE, Roncal-Jimenez C, Sasai F, Kang DH, Jensen T, Hernando AA, Rodriguez-Iturbe B, Garcia G, Tolan DR, Sanchez-Lozada LG, Lanaspa MA, Johnson RJ. Hyperuricemia in Kidney Disease: A Major Risk Factor for Cardiovascular Events, Vascular Calcification, and Renal Damage. Semin Nephrol. 2020 Nov;40(6):574-585. doi: 10.1016/j.semnephrol.2020.12.004. PMID: 33678312; PMCID: PMC7951176.
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Mini Review: Reappraisal of Uric Acid in Chronic Kidney Disease.

American journal of nephrology

Goldberg A, Garcia-Arroyo F, Sasai F, Rodriguez-Iturbe B, Sanchez-Lozada LG, Lanaspa MA, Johnson RJ.
PMID: 34673651
Am J Nephrol. 2021;52(10):837-844. doi: 10.1159/000519491. Epub 2021 Oct 21.

Hyperuricemia predicts the development of chronic kidney disease (CKD) and metabolic complications, but whether it has a causal role has been controversial. This is especially true given the 2 recently conducted randomized controlled trials that failed to show a...

Cite
Goldberg A, Garcia-Arroyo F, Sasai F, et al. Mini Review: Reappraisal of Uric Acid in Chronic Kidney Disease. Am J Nephrol. 2021;52(10):837-844doi: 10.1159/000519491.
Goldberg, A., Garcia-Arroyo, F., Sasai, F., Rodriguez-Iturbe, B., Sanchez-Lozada, L. G., Lanaspa, M. A., & Johnson, R. J. (2021). Mini Review: Reappraisal of Uric Acid in Chronic Kidney Disease. American journal of nephrology, 52(10), 837-844. https://doi.org/10.1159/000519491
Goldberg, Avi, et al. "Mini Review: Reappraisal of Uric Acid in Chronic Kidney Disease." American journal of nephrology vol. 52,10 (2021): 837-844. doi: https://doi.org/10.1159/000519491
Goldberg A, Garcia-Arroyo F, Sasai F, Rodriguez-Iturbe B, Sanchez-Lozada LG, Lanaspa MA, Johnson RJ. Mini Review: Reappraisal of Uric Acid in Chronic Kidney Disease. Am J Nephrol. 2021;52(10):837-844. doi: 10.1159/000519491. Epub 2021 Oct 21. PMID: 34673651; PMCID: PMC8665019.
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A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation.

Life sciences

Rojas-Morales P, León-Contreras JC, Sánchez-Tapia M, Silva-Palacios A, Cano-Martínez A, González-Reyes S, Jiménez-Osorio AS, Hernández-Pando R, Osorio-Alonso H, Sánchez-Lozada LG, Tovar AR, Pedraza-Chaverri J, Tapia E.
PMID: 34921866
Life Sci. 2021 Dec 16;289:120227. doi: 10.1016/j.lfs.2021.120227. Epub 2021 Dec 16.

BACKGROUND: Ischemic kidney injury is a common clinical condition resulting from transient interruption of the kidney's normal blood flow, leading to oxidative stress, inflammation, and kidney dysfunction. The ketogenic diet (KD), a low-carbohydrate, high-fat diet that stimulates endogenous ketone...

Cite
Rojas-Morales P, León-Contreras JC, Sánchez-Tapia M, et al. A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation. Life Sci. 2021;289:120227doi: 10.1016/j.lfs.2021.120227.
Rojas-Morales, P., León-Contreras, J. C., Sánchez-Tapia, M., Silva-Palacios, A., Cano-Martínez, A., González-Reyes, S., Jiménez-Osorio, A. S., Hernández-Pando, R., Osorio-Alonso, H., Sánchez-Lozada, L. G., Tovar, A. R., Pedraza-Chaverri, J., & Tapia, E. (2021). A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation. Life sciences, 289120227. https://doi.org/10.1016/j.lfs.2021.120227
Rojas-Morales, Pedro, et al. "A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation." Life sciences vol. 289 (2021): 120227. doi: https://doi.org/10.1016/j.lfs.2021.120227
Rojas-Morales P, León-Contreras JC, Sánchez-Tapia M, Silva-Palacios A, Cano-Martínez A, González-Reyes S, Jiménez-Osorio AS, Hernández-Pando R, Osorio-Alonso H, Sánchez-Lozada LG, Tovar AR, Pedraza-Chaverri J, Tapia E. A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation. Life Sci. 2021 Dec 16;289:120227. doi: 10.1016/j.lfs.2021.120227. Epub 2021 Dec 16. PMID: 34921866.
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Corrigendum to "Antioxidant supplements as a novel mean for blocking recurrent heat stress-induced kidney damage following rehydration with fructose-containing beverages" [Free Radic. Biol. Med. 141 (2019) 182-191].

Free radical biology & medicine

García-Arroyo FE, Gonzaga G, Muñoz-Jiménez I, Osorio-Alonso H, Iroz A, Vecchio M, Tapia E, Roncal-Jiménez CA, Johnson RJ, Sánchez-Lozada LG.
PMID: 31813623
Free Radic Biol Med. 2020 May 20;152:865-866. doi: 10.1016/j.freeradbiomed.2019.11.035. Epub 2019 Dec 06.

No abstract available.

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García-Arroyo FE, Gonzaga G, Muñoz-Jiménez I, et al. Corrigendum to "Antioxidant supplements as a novel mean for blocking recurrent heat stress-induced kidney damage following rehydration with fructose-containing beverages" [Free Radic. Biol. Med. 141 (2019) 182-191]. Free Radic Biol Med. 2019;152:865-866doi: 10.1016/j.freeradbiomed.2019.11.035.
García-Arroyo, F. E., Gonzaga, G., Muñoz-Jiménez, I., Osorio-Alonso, H., Iroz, A., Vecchio, M., Tapia, E., Roncal-Jiménez, C. A., Johnson, R. J., & Sánchez-Lozada, L. G. (2020). Corrigendum to "Antioxidant supplements as a novel mean for blocking recurrent heat stress-induced kidney damage following rehydration with fructose-containing beverages" [Free Radic. Biol. Med. 141 (2019) 182-191]. Free radical biology & medicine, 152865-866. https://doi.org/10.1016/j.freeradbiomed.2019.11.035
García-Arroyo, Fernando E, et al. "Corrigendum to "Antioxidant supplements as a novel mean for blocking recurrent heat stress-induced kidney damage following rehydration with fructose-containing beverages" [Free Radic. Biol. Med. 141 (2019) 182-191]." Free radical biology & medicine vol. 152 (2020): 865-866. doi: https://doi.org/10.1016/j.freeradbiomed.2019.11.035
García-Arroyo FE, Gonzaga G, Muñoz-Jiménez I, Osorio-Alonso H, Iroz A, Vecchio M, Tapia E, Roncal-Jiménez CA, Johnson RJ, Sánchez-Lozada LG. Corrigendum to "Antioxidant supplements as a novel mean for blocking recurrent heat stress-induced kidney damage following rehydration with fructose-containing beverages" [Free Radic. Biol. Med. 141 (2019) 182-191]. Free Radic Biol Med. 2020 May 20;152:865-866. doi: 10.1016/j.freeradbiomed.2019.11.035. Epub 2019 Dec 06. PMID: 31813623.
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Brief report: The uricase mutation in humans increases our risk for cancer growth.

Cancer & metabolism

Fini MA, Lanaspa MA, Gaucher EA, Boutwell B, Nakagawa T, Wright RM, Sanchez-Lozada LG, Andrews P, Stenmark KR, Johnson RJ.
PMID: 34526149
Cancer Metab. 2021 Sep 15;9(1):32. doi: 10.1186/s40170-021-00268-3.

BACKGROUND: Recent studies suggest that fructose, as well as its metabolite, uric acid, have been associated with increased risk for both cancer incidence and growth. Both substances are known to cause oxidative stress to mitochondria and to reduce adenosine...

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Fini MA, Lanaspa MA, Gaucher EA, et al. Brief report: The uricase mutation in humans increases our risk for cancer growth. Cancer Metab. 2021;9(1):32doi: 10.1186/s40170-021-00268-3.
Fini, M. A., Lanaspa, M. A., Gaucher, E. A., Boutwell, B., Nakagawa, T., Wright, R. M., Sanchez-Lozada, L. G., Andrews, P., Stenmark, K. R., & Johnson, R. J. (2021). Brief report: The uricase mutation in humans increases our risk for cancer growth. Cancer & metabolism, 9(1), 32. https://doi.org/10.1186/s40170-021-00268-3
Fini, Mehdi A, et al. "Brief report: The uricase mutation in humans increases our risk for cancer growth." Cancer & metabolism vol. 9,1 (2021): 32. doi: https://doi.org/10.1186/s40170-021-00268-3
Fini MA, Lanaspa MA, Gaucher EA, Boutwell B, Nakagawa T, Wright RM, Sanchez-Lozada LG, Andrews P, Stenmark KR, Johnson RJ. Brief report: The uricase mutation in humans increases our risk for cancer growth. Cancer Metab. 2021 Sep 15;9(1):32. doi: 10.1186/s40170-021-00268-3. PMID: 34526149; PMCID: PMC8444362.
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Cerebral Fructose Metabolism as a Potential Mechanism Driving Alzheimer's Disease.

Frontiers in aging neuroscience

Johnson RJ, Gomez-Pinilla F, Nagel M, Nakagawa T, Rodriguez-Iturbe B, Sanchez-Lozada LG, Tolan DR, Lanaspa MA.
PMID: 33024433
Front Aging Neurosci. 2020 Sep 11;12:560865. doi: 10.3389/fnagi.2020.560865. eCollection 2020.

The loss of cognitive function in Alzheimer's disease is pathologically linked with neurofibrillary tangles, amyloid deposition, and loss of neuronal communication. Cerebral insulin resistance and mitochondrial dysfunction have emerged as important contributors to pathogenesis supporting our hypothesis that cerebral...

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Johnson RJ, Gomez-Pinilla F, Nagel M, et al. Cerebral Fructose Metabolism as a Potential Mechanism Driving Alzheimer's Disease. Front Aging Neurosci. 2020;12:560865doi: 10.3389/fnagi.2020.560865.
Johnson, R. J., Gomez-Pinilla, F., Nagel, M., Nakagawa, T., Rodriguez-Iturbe, B., Sanchez-Lozada, L. G., Tolan, D. R., & Lanaspa, M. A. (2020). Cerebral Fructose Metabolism as a Potential Mechanism Driving Alzheimer's Disease. Frontiers in aging neuroscience, 12560865. https://doi.org/10.3389/fnagi.2020.560865
Johnson, Richard J, et al. "Cerebral Fructose Metabolism as a Potential Mechanism Driving Alzheimer's Disease." Frontiers in aging neuroscience vol. 12 (2020): 560865. doi: https://doi.org/10.3389/fnagi.2020.560865
Johnson RJ, Gomez-Pinilla F, Nagel M, Nakagawa T, Rodriguez-Iturbe B, Sanchez-Lozada LG, Tolan DR, Lanaspa MA. Cerebral Fructose Metabolism as a Potential Mechanism Driving Alzheimer's Disease. Front Aging Neurosci. 2020 Sep 11;12:560865. doi: 10.3389/fnagi.2020.560865. eCollection 2020. PMID: 33024433; PMCID: PMC7516162.
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A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation.

Life sciences

Rojas-Morales P, León-Contreras JC, Sánchez-Tapia M, Silva-Palacios A, Cano-Martínez A, González-Reyes S, Jiménez-Osorio AS, Hernández-Pando R, Osorio-Alonso H, Sánchez-Lozada LG, Tovar AR, Pedraza-Chaverri J, Tapia E.
PMID: 34921866
Life Sci. 2021 Dec 16;289:120227. doi: 10.1016/j.lfs.2021.120227. Epub 2021 Dec 16.

BACKGROUND: Ischemic kidney injury is a common clinical condition resulting from transient interruption of the kidney's normal blood flow, leading to oxidative stress, inflammation, and kidney dysfunction. The ketogenic diet (KD), a low-carbohydrate, high-fat diet that stimulates endogenous ketone...

Cite
Rojas-Morales P, León-Contreras JC, Sánchez-Tapia M, et al. A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation. Life Sci. 2021;289:120227doi: 10.1016/j.lfs.2021.120227.
Rojas-Morales, P., León-Contreras, J. C., Sánchez-Tapia, M., Silva-Palacios, A., Cano-Martínez, A., González-Reyes, S., Jiménez-Osorio, A. S., Hernández-Pando, R., Osorio-Alonso, H., Sánchez-Lozada, L. G., Tovar, A. R., Pedraza-Chaverri, J., & Tapia, E. (2021). A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation. Life sciences, 289120227. https://doi.org/10.1016/j.lfs.2021.120227
Rojas-Morales, Pedro, et al. "A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation." Life sciences vol. 289 (2021): 120227. doi: https://doi.org/10.1016/j.lfs.2021.120227
Rojas-Morales P, León-Contreras JC, Sánchez-Tapia M, Silva-Palacios A, Cano-Martínez A, González-Reyes S, Jiménez-Osorio AS, Hernández-Pando R, Osorio-Alonso H, Sánchez-Lozada LG, Tovar AR, Pedraza-Chaverri J, Tapia E. A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation. Life Sci. 2021 Dec 16;289:120227. doi: 10.1016/j.lfs.2021.120227. Epub 2021 Dec 16. PMID: 34921866.
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High fructose exposure modifies the amount of adipocyte-secreted microRNAs into extracellular vesicles in supernatants and plasma.

PeerJ

Hernández-Díazcouder A, González-Ramírez J, Giacoman-Martínez A, Cardoso-Saldaña G, Martínez-Martínez E, Osorio-Alonso H, Márquez-Velasco R, Sánchez-Gloria JL, Juárez-Vicuña Y, Gonzaga G, Sánchez-Lozada LG, Almanza-Pérez JC, Sánchez-Muñoz F.
PMID: 34055478
PeerJ. 2021 May 19;9:e11305. doi: 10.7717/peerj.11305. eCollection 2021.

BACKGROUND: High fructose exposure induces metabolic and endocrine responses in adipose tissue. Recent evidence suggests that microRNAs in extracellular vesicles are endocrine signals secreted by adipocytes. Fructose exposure on the secretion of microRNA by tissues and cells is poorly...

Cite
Hernández-Díazcouder A, González-Ramírez J, Giacoman-Martínez A, et al. High fructose exposure modifies the amount of adipocyte-secreted microRNAs into extracellular vesicles in supernatants and plasma. PeerJ. 2021;9:e11305doi: 10.7717/peerj.11305.
Hernández-Díazcouder, A., González-Ramírez, J., Giacoman-Martínez, A., Cardoso-Saldaña, G., Martínez-Martínez, E., Osorio-Alonso, H., Márquez-Velasco, R., Sánchez-Gloria, J. L., Juárez-Vicuña, Y., Gonzaga, G., Sánchez-Lozada, L. G., Almanza-Pérez, J. C., & Sánchez-Muñoz, F. (2021). High fructose exposure modifies the amount of adipocyte-secreted microRNAs into extracellular vesicles in supernatants and plasma. PeerJ, 9e11305. https://doi.org/10.7717/peerj.11305
Hernández-Díazcouder, Adrián, et al. "High fructose exposure modifies the amount of adipocyte-secreted microRNAs into extracellular vesicles in supernatants and plasma." PeerJ vol. 9 (2021): e11305. doi: https://doi.org/10.7717/peerj.11305
Hernández-Díazcouder A, González-Ramírez J, Giacoman-Martínez A, Cardoso-Saldaña G, Martínez-Martínez E, Osorio-Alonso H, Márquez-Velasco R, Sánchez-Gloria JL, Juárez-Vicuña Y, Gonzaga G, Sánchez-Lozada LG, Almanza-Pérez JC, Sánchez-Muñoz F. High fructose exposure modifies the amount of adipocyte-secreted microRNAs into extracellular vesicles in supernatants and plasma. PeerJ. 2021 May 19;9:e11305. doi: 10.7717/peerj.11305. eCollection 2021. PMID: 34055478; PMCID: PMC8140597.
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A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation.

Life sciences

Rojas-Morales P, León-Contreras JC, Sánchez-Tapia M, Silva-Palacios A, Cano-Martínez A, González-Reyes S, Jiménez-Osorio AS, Hernández-Pando R, Osorio-Alonso H, Sánchez-Lozada LG, Tovar AR, Pedraza-Chaverri J, Tapia E.
PMID: 34921866
Life Sci. 2022 Jan 15;289:120227. doi: 10.1016/j.lfs.2021.120227. Epub 2021 Dec 16.

BACKGROUND: Ischemic kidney injury is a common clinical condition resulting from transient interruption of the kidney's normal blood flow, leading to oxidative stress, inflammation, and kidney dysfunction. The ketogenic diet (KD), a low-carbohydrate, high-fat diet that stimulates endogenous ketone...

Cite
Rojas-Morales P, León-Contreras JC, Sánchez-Tapia M, et al. A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation. Life Sci. 2021;289:120227doi: 10.1016/j.lfs.2021.120227.
Rojas-Morales, P., León-Contreras, J. C., Sánchez-Tapia, M., Silva-Palacios, A., Cano-Martínez, A., González-Reyes, S., Jiménez-Osorio, A. S., Hernández-Pando, R., Osorio-Alonso, H., Sánchez-Lozada, L. G., Tovar, A. R., Pedraza-Chaverri, J., & Tapia, E. (2022). A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation. Life sciences, 289120227. https://doi.org/10.1016/j.lfs.2021.120227
Rojas-Morales, Pedro, et al. "A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation." Life sciences vol. 289 (2022): 120227. doi: https://doi.org/10.1016/j.lfs.2021.120227
Rojas-Morales P, León-Contreras JC, Sánchez-Tapia M, Silva-Palacios A, Cano-Martínez A, González-Reyes S, Jiménez-Osorio AS, Hernández-Pando R, Osorio-Alonso H, Sánchez-Lozada LG, Tovar AR, Pedraza-Chaverri J, Tapia E. A ketogenic diet attenuates acute and chronic ischemic kidney injury and reduces markers of oxidative stress and inflammation. Life Sci. 2022 Jan 15;289:120227. doi: 10.1016/j.lfs.2021.120227. Epub 2021 Dec 16. PMID: 34921866.
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Hyperuricemia and chronic kidney disease: to treat or not to treat.

Jornal brasileiro de nefrologia : 'orgao oficial de Sociedades Brasileira e Latino-Americana de Nefrologia

Piani F, Sasai F, Bjornstad P, Borghi C, Yoshimura A, Sanchez-Lozada LG, Roncal-Jimenez C, Garcia GE, Hernando AA, Fuentes GC, Rodriguez-Iturbe B, Lanaspa MA, Johnson RJ.
PMID: 33704350
J Bras Nefrol. 2021 Mar 05; doi: 10.1590/2175-8239-JBN-2020-U002. Epub 2021 Mar 05.

Hyperuricemia is common in chronic kidney disease (CKD) and may be present in 50% of patients presenting for dialysis. Hyperuricemia can be secondary to impaired glomerular filtration rate (GFR) that occurs in CKD. However, hyperuricemia can also precede the...

Cite
Piani F, Sasai F, Bjornstad P, et al. Hyperuricemia and chronic kidney disease: to treat or not to treat. J Bras Nefrol. 2021;doi: 10.1590/2175-8239-JBN-2020-U002.
Piani, F., Sasai, F., Bjornstad, P., Borghi, C., Yoshimura, A., Sanchez-Lozada, L. G., Roncal-Jimenez, C., Garcia, G. E., Hernando, A. A., Fuentes, G. C., Rodriguez-Iturbe, B., Lanaspa, M. A., & Johnson, R. J. (2021). Hyperuricemia and chronic kidney disease: to treat or not to treat. Jornal brasileiro de nefrologia : 'orgao oficial de Sociedades Brasileira e Latino-Americana de Nefrologia, . https://doi.org/10.1590/2175-8239-JBN-2020-U002
Piani, Federica, et al. "Hyperuricemia and chronic kidney disease: to treat or not to treat." Jornal brasileiro de nefrologia : 'orgao oficial de Sociedades Brasileira e Latino-Americana de Nefrologia vol. (2021). doi: https://doi.org/10.1590/2175-8239-JBN-2020-U002
Piani F, Sasai F, Bjornstad P, Borghi C, Yoshimura A, Sanchez-Lozada LG, Roncal-Jimenez C, Garcia GE, Hernando AA, Fuentes GC, Rodriguez-Iturbe B, Lanaspa MA, Johnson RJ. Hyperuricemia and chronic kidney disease: to treat or not to treat. J Bras Nefrol. 2021 Mar 05; doi: 10.1590/2175-8239-JBN-2020-U002. Epub 2021 Mar 05. Portuguese. PMID: 33704350.
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Progressive Reduction in Mitochondrial Mass Is Triggered by Alterations in Mitochondrial Biogenesis and Dynamics in Chronic Kidney Disease Induced by 5/6 Nephrectomy.

Biology

Prieto-Carrasco R, García-Arroyo FE, Aparicio-Trejo OE, Rojas-Morales P, León-Contreras JC, Hernández-Pando R, Sánchez-Lozada LG, Tapia E, Pedraza-Chaverri J.
PMID: 33919054
Biology (Basel). 2021 Apr 21;10(5). doi: 10.3390/biology10050349.

The five-sixth nephrectomy (5/6Nx) model is widely used to study the mechanisms involved in chronic kidney disease (CKD) progression. Mitochondrial impairment is a critical mechanism that favors CKD progression. However, until now, there are no temporal studies of the...

Cite
Prieto-Carrasco R, García-Arroyo FE, Aparicio-Trejo OE, et al. Progressive Reduction in Mitochondrial Mass Is Triggered by Alterations in Mitochondrial Biogenesis and Dynamics in Chronic Kidney Disease Induced by 5/6 Nephrectomy. Biology (Basel). 2021;10(5)doi: 10.3390/biology10050349.
Prieto-Carrasco, R., García-Arroyo, F. E., Aparicio-Trejo, O. E., Rojas-Morales, P., León-Contreras, J. C., Hernández-Pando, R., Sánchez-Lozada, L. G., Tapia, E., & Pedraza-Chaverri, J. (2021). Progressive Reduction in Mitochondrial Mass Is Triggered by Alterations in Mitochondrial Biogenesis and Dynamics in Chronic Kidney Disease Induced by 5/6 Nephrectomy. Biology, 10(5), . https://doi.org/10.3390/biology10050349
Prieto-Carrasco, Rodrigo, et al. "Progressive Reduction in Mitochondrial Mass Is Triggered by Alterations in Mitochondrial Biogenesis and Dynamics in Chronic Kidney Disease Induced by 5/6 Nephrectomy." Biology vol. 10,5 (2021). doi: https://doi.org/10.3390/biology10050349
Prieto-Carrasco R, García-Arroyo FE, Aparicio-Trejo OE, Rojas-Morales P, León-Contreras JC, Hernández-Pando R, Sánchez-Lozada LG, Tapia E, Pedraza-Chaverri J. Progressive Reduction in Mitochondrial Mass Is Triggered by Alterations in Mitochondrial Biogenesis and Dynamics in Chronic Kidney Disease Induced by 5/6 Nephrectomy. Biology (Basel). 2021 Apr 21;10(5). doi: 10.3390/biology10050349. PMID: 33919054; PMCID: PMC8143166.
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Hyperuricemia and chronic kidney disease: to treat or not to treat.

Jornal brasileiro de nefrologia : 'orgao oficial de Sociedades Brasileira e Latino-Americana de Nefrologia

Piani F, Sasai F, Bjornstad P, Borghi C, Yoshimura A, Sanchez-Lozada LG, Roncal-Jimenez C, Garcia GE, Hernando AA, Fuentes GC, Rodriguez-Iturbe B, Lanaspa MA, Johnson RJ.
PMID: 33704350
J Bras Nefrol. 2021 Oct-Dec;43(4):572-579. doi: 10.1590/2175-8239-JBN-2020-U002.

Hyperuricemia is common in chronic kidney disease (CKD) and may be present in 50% of patients presenting for dialysis. Hyperuricemia can be secondary to impaired glomerular filtration rate (GFR) that occurs in CKD. However, hyperuricemia can also precede the...

Cite
Piani F, Sasai F, Bjornstad P, et al. Hyperuricemia and chronic kidney disease: to treat or not to treat. J Bras Nefrol. 2021;43(4):572-579doi: 10.1590/2175-8239-JBN-2020-U002.
Piani, F., Sasai, F., Bjornstad, P., Borghi, C., Yoshimura, A., Sanchez-Lozada, L. G., Roncal-Jimenez, C., Garcia, G. E., Hernando, A. A., Fuentes, G. C., Rodriguez-Iturbe, B., Lanaspa, M. A., & Johnson, R. J. (2021). Hyperuricemia and chronic kidney disease: to treat or not to treat. Jornal brasileiro de nefrologia : 'orgao oficial de Sociedades Brasileira e Latino-Americana de Nefrologia, 43(4), 572-579. https://doi.org/10.1590/2175-8239-JBN-2020-U002
Piani, Federica, et al. "Hyperuricemia and chronic kidney disease: to treat or not to treat." Jornal brasileiro de nefrologia : 'orgao oficial de Sociedades Brasileira e Latino-Americana de Nefrologia vol. 43,4 (2021): 572-579. doi: https://doi.org/10.1590/2175-8239-JBN-2020-U002
Piani F, Sasai F, Bjornstad P, Borghi C, Yoshimura A, Sanchez-Lozada LG, Roncal-Jimenez C, Garcia GE, Hernando AA, Fuentes GC, Rodriguez-Iturbe B, Lanaspa MA, Johnson RJ. Hyperuricemia and chronic kidney disease: to treat or not to treat. J Bras Nefrol. 2021 Oct-Dec;43(4):572-579. doi: 10.1590/2175-8239-JBN-2020-U002. Portuguese. PMID: 33704350.
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