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Showing 1 to 12 of 565 entries
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Applications of Mesenchymal Stem Cells and Neural Crest Cells in Craniofacial Skeletal Research.

Stem cells international

Morikawa S, Ouchi T, Shibata S, Fujimura T, Kawana H, Okano H, Nakagawa T.
PMID: 27006661
Stem Cells Int. 2016;2016:2849879. doi: 10.1155/2016/2849879. Epub 2016 Feb 24.

Craniofacial skeletal tissues are composed of tooth and bone, together with nerves and blood vessels. This composite material is mainly derived from neural crest cells (NCCs). The neural crest is transient embryonic tissue present during neural tube formation whose...

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Morikawa S, Ouchi T, Shibata S, et al. Applications of Mesenchymal Stem Cells and Neural Crest Cells in Craniofacial Skeletal Research. Stem Cells Int. 2016;2016:2849879doi: 10.1155/2016/2849879.
Morikawa, S., Ouchi, T., Shibata, S., Fujimura, T., Kawana, H., Okano, H., & Nakagawa, T. (2016). Applications of Mesenchymal Stem Cells and Neural Crest Cells in Craniofacial Skeletal Research. Stem cells international, 20162849879. https://doi.org/10.1155/2016/2849879
Morikawa, Satoru, et al. "Applications of Mesenchymal Stem Cells and Neural Crest Cells in Craniofacial Skeletal Research." Stem cells international vol. 2016 (2016): 2849879. doi: https://doi.org/10.1155/2016/2849879
Morikawa S, Ouchi T, Shibata S, Fujimura T, Kawana H, Okano H, Nakagawa T. Applications of Mesenchymal Stem Cells and Neural Crest Cells in Craniofacial Skeletal Research. Stem Cells Int. 2016;2016:2849879. doi: 10.1155/2016/2849879. Epub 2016 Feb 24. PMID: 27006661; PMCID: PMC4783549.
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Plexiform Fibrohistiocytic Tumor on the Ear: Case Report and Immunohistochemical Investigation of Stromal Factor.

Case reports in dermatology

Shido K, Fujimura T, Kakizaki A, Furudate S, Asano M, Aiba S.
PMID: 27293390
Case Rep Dermatol. 2016 Feb 05;8(1):26-30. doi: 10.1159/000444045. eCollection 2016.

Plexiform fibrohistiocytic tumor (PFT) is a rare mesenchymal neoplasm of intermediate malignant potential with a high local recurrence rate. In this report, we describe a case of PFT on the ear, which showed a dense deposition of periostin (POSTN)...

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Shido K, Fujimura T, Kakizaki A, et al. Plexiform Fibrohistiocytic Tumor on the Ear: Case Report and Immunohistochemical Investigation of Stromal Factor. Case Rep Dermatol. 2016;8(1):26-30doi: 10.1159/000444045.
Shido, K., Fujimura, T., Kakizaki, A., Furudate, S., Asano, M., & Aiba, S. (2016). Plexiform Fibrohistiocytic Tumor on the Ear: Case Report and Immunohistochemical Investigation of Stromal Factor. Case reports in dermatology, 8(1), 26-30. https://doi.org/10.1159/000444045
Shido, Kosuke, et al. "Plexiform Fibrohistiocytic Tumor on the Ear: Case Report and Immunohistochemical Investigation of Stromal Factor." Case reports in dermatology vol. 8,1 (2016): 26-30. doi: https://doi.org/10.1159/000444045
Shido K, Fujimura T, Kakizaki A, Furudate S, Asano M, Aiba S. Plexiform Fibrohistiocytic Tumor on the Ear: Case Report and Immunohistochemical Investigation of Stromal Factor. Case Rep Dermatol. 2016 Feb 05;8(1):26-30. doi: 10.1159/000444045. eCollection 2016. PMID: 27293390; PMCID: PMC4899637.
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Intermittent docetaxel chemotherapy is feasible for castration-resistant prostate cancer.

Molecular and clinical oncology

Kume H, Kawai T, Nagata M, Azuma T, Miyazaki H, Suzuki M, Fujimura T, Nakagawa T, Fukuhara H, Homma Y.
PMID: 25798258
Mol Clin Oncol. 2015 Mar;3(2):303-307. doi: 10.3892/mco.2014.469. Epub 2014 Dec 01.

This study was conducted with the aim to investigate the feasibility of intermittent treatment with docetaxel chemotherapy for castration-resistant prostate cancer (CRPC). A total of 51 men with CRPC received docetaxel at 75 mg/m

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Kume H, Kawai T, Nagata M, et al. Intermittent docetaxel chemotherapy is feasible for castration-resistant prostate cancer. Mol Clin Oncol. 2014;3(2):303-307doi: 10.3892/mco.2014.469.
Kume, H., Kawai, T., Nagata, M., Azuma, T., Miyazaki, H., Suzuki, M., Fujimura, T., Nakagawa, T., Fukuhara, H., & Homma, Y. (2015). Intermittent docetaxel chemotherapy is feasible for castration-resistant prostate cancer. Molecular and clinical oncology, 3(2), 303-307. https://doi.org/10.3892/mco.2014.469
Kume, Haruki, et al. "Intermittent docetaxel chemotherapy is feasible for castration-resistant prostate cancer." Molecular and clinical oncology vol. 3,2 (2015): 303-307. doi: https://doi.org/10.3892/mco.2014.469
Kume H, Kawai T, Nagata M, Azuma T, Miyazaki H, Suzuki M, Fujimura T, Nakagawa T, Fukuhara H, Homma Y. Intermittent docetaxel chemotherapy is feasible for castration-resistant prostate cancer. Mol Clin Oncol. 2015 Mar;3(2):303-307. doi: 10.3892/mco.2014.469. Epub 2014 Dec 01. PMID: 25798258; PMCID: PMC4360872.
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Human retinal pigment epithelial cell proliferation by the combined stimulation of hydroquinone and advanced glycation end-products via up-regulation of .

Biochemistry and biophysics reports

Tsujinaka H, Itaya-Hironaka A, Yamauchi A, Sakuramoto-Tsuchida S, Ota H, Takeda M, Fujimura T, Takasawa S, Ogata N.
PMID: 29124153
Biochem Biophys Rep. 2015 May 29;2:123-131. doi: 10.1016/j.bbrep.2015.05.005. eCollection 2015 Jul.

Although recent research showed that advanced glycation endproduct (AGE) and hydroquinone (HQ) are related to the pathogenesis of age-related macular degeneration (AMD), the mechanism how AGE and HQ induce or accelerate AMD remains elusive. In the present study, we...

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Tsujinaka H, Itaya-Hironaka A, Yamauchi A, et al. Human retinal pigment epithelial cell proliferation by the combined stimulation of hydroquinone and advanced glycation end-products via up-regulation of . Biochem Biophys Rep. 2015;2:123-131doi: 10.1016/j.bbrep.2015.05.005.
Tsujinaka, H., Itaya-Hironaka, A., Yamauchi, A., Sakuramoto-Tsuchida, S., Ota, H., Takeda, M., Fujimura, T., Takasawa, S., & Ogata, N. (2015). Human retinal pigment epithelial cell proliferation by the combined stimulation of hydroquinone and advanced glycation end-products via up-regulation of . Biochemistry and biophysics reports, 2123-131. https://doi.org/10.1016/j.bbrep.2015.05.005
Tsujinaka, Hiroki, et al. "Human retinal pigment epithelial cell proliferation by the combined stimulation of hydroquinone and advanced glycation end-products via up-regulation of ." Biochemistry and biophysics reports vol. 2 (2015): 123-131. doi: https://doi.org/10.1016/j.bbrep.2015.05.005
Tsujinaka H, Itaya-Hironaka A, Yamauchi A, Sakuramoto-Tsuchida S, Ota H, Takeda M, Fujimura T, Takasawa S, Ogata N. Human retinal pigment epithelial cell proliferation by the combined stimulation of hydroquinone and advanced glycation end-products via up-regulation of . Biochem Biophys Rep. 2015 May 29;2:123-131. doi: 10.1016/j.bbrep.2015.05.005. eCollection 2015 Jul. PMID: 29124153; PMCID: PMC5668646.
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Marked Elevation of Plasma Levels of Oxidative Stress-Responsive Apoptosis-Inducing Protein in Dialysis Patients.

Kidney international reports

Tanaka K, Yao T, Fujimura T, Murayama K, Fukuda S, Okumura K, Seko Y.
PMID: 29142934
Kidney Int Rep. 2016 Aug 24;1(4):321-324. doi: 10.1016/j.ekir.2016.08.011. eCollection 2016 Nov.

No abstract available.

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Tanaka K, Yao T, Fujimura T, et al. Marked Elevation of Plasma Levels of Oxidative Stress-Responsive Apoptosis-Inducing Protein in Dialysis Patients. Kidney Int Rep. 2016;1(4):321-324doi: 10.1016/j.ekir.2016.08.011.
Tanaka, K., Yao, T., Fujimura, T., Murayama, K., Fukuda, S., Okumura, K., & Seko, Y. (2016). Marked Elevation of Plasma Levels of Oxidative Stress-Responsive Apoptosis-Inducing Protein in Dialysis Patients. Kidney international reports, 1(4), 321-324. https://doi.org/10.1016/j.ekir.2016.08.011
Tanaka, Kentaro, et al. "Marked Elevation of Plasma Levels of Oxidative Stress-Responsive Apoptosis-Inducing Protein in Dialysis Patients." Kidney international reports vol. 1,4 (2016): 321-324. doi: https://doi.org/10.1016/j.ekir.2016.08.011
Tanaka K, Yao T, Fujimura T, Murayama K, Fukuda S, Okumura K, Seko Y. Marked Elevation of Plasma Levels of Oxidative Stress-Responsive Apoptosis-Inducing Protein in Dialysis Patients. Kidney Int Rep. 2016 Aug 24;1(4):321-324. doi: 10.1016/j.ekir.2016.08.011. eCollection 2016 Nov. PMID: 29142934; PMCID: PMC5678848.
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Complete mitochondrial genome sequences of .

Breeding science

Hatono S, Nishimura K, Murakami Y, Tsujimura M, Yamagishi H.
PMID: 29085245
Breed Sci. 2017 Sep;67(4):357-362. doi: 10.1270/jsbbs.17023. Epub 2017 Jul 28.

The complete sequence of the mitochondrial genome was determined for two cultivars of

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Hatono S, Nishimura K, Murakami Y, et al. Complete mitochondrial genome sequences of . Breed Sci. 2017;67(4):357-362doi: 10.1270/jsbbs.17023.
Hatono, S., Nishimura, K., Murakami, Y., Tsujimura, M., & Yamagishi, H. (2017). Complete mitochondrial genome sequences of . Breeding science, 67(4), 357-362. https://doi.org/10.1270/jsbbs.17023
Hatono, Saki, et al. "Complete mitochondrial genome sequences of ." Breeding science vol. 67,4 (2017): 357-362. doi: https://doi.org/10.1270/jsbbs.17023
Hatono S, Nishimura K, Murakami Y, Tsujimura M, Yamagishi H. Complete mitochondrial genome sequences of . Breed Sci. 2017 Sep;67(4):357-362. doi: 10.1270/jsbbs.17023. Epub 2017 Jul 28. PMID: 29085245; PMCID: PMC5654463.
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Interleukin-6/STAT pathway is responsible for the induction of gene expression of REG Iα, a new auto-antigen in Sjögren׳s syndrome patients, in salivary duct epithelial cells.

Biochemistry and biophysics reports

Fujimura T, Fujimoto T, Itaya-Hironaka A, Miyaoka T, Yoshimoto K, Yamauchi A, Sakuramoto-Tsuchida S, Kondo S, Takeda M, Tsujinaka H, Azuma M, Tanaka Y, Takasawa S.
PMID: 29124146
Biochem Biophys Rep. 2015 May 30;2:69-74. doi: 10.1016/j.bbrep.2015.05.006. eCollection 2015 Jul.

The regenerating gene,

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Fujimura T, Fujimoto T, Itaya-Hironaka A, et al. Interleukin-6/STAT pathway is responsible for the induction of gene expression of REG Iα, a new auto-antigen in Sjögren׳s syndrome patients, in salivary duct epithelial cells. Biochem Biophys Rep. 2015;2:69-74doi: 10.1016/j.bbrep.2015.05.006.
Fujimura, T., Fujimoto, T., Itaya-Hironaka, A., Miyaoka, T., Yoshimoto, K., Yamauchi, A., Sakuramoto-Tsuchida, S., Kondo, S., Takeda, M., Tsujinaka, H., Azuma, M., Tanaka, Y., & Takasawa, S. (2015). Interleukin-6/STAT pathway is responsible for the induction of gene expression of REG Iα, a new auto-antigen in Sjögren׳s syndrome patients, in salivary duct epithelial cells. Biochemistry and biophysics reports, 269-74. https://doi.org/10.1016/j.bbrep.2015.05.006
Fujimura, Takanori, et al. "Interleukin-6/STAT pathway is responsible for the induction of gene expression of REG Iα, a new auto-antigen in Sjögren׳s syndrome patients, in salivary duct epithelial cells." Biochemistry and biophysics reports vol. 2 (2015): 69-74. doi: https://doi.org/10.1016/j.bbrep.2015.05.006
Fujimura T, Fujimoto T, Itaya-Hironaka A, Miyaoka T, Yoshimoto K, Yamauchi A, Sakuramoto-Tsuchida S, Kondo S, Takeda M, Tsujinaka H, Azuma M, Tanaka Y, Takasawa S. Interleukin-6/STAT pathway is responsible for the induction of gene expression of REG Iα, a new auto-antigen in Sjögren׳s syndrome patients, in salivary duct epithelial cells. Biochem Biophys Rep. 2015 May 30;2:69-74. doi: 10.1016/j.bbrep.2015.05.006. eCollection 2015 Jul. PMID: 29124146; PMCID: PMC5668644.
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Photophysical properties of three coordinated copper(i) complexes bearing 1,10-phenanthroline and a monodentate phosphine ligand.

Dalton transactions (Cambridge, England : 2003)

Kakizoe D, Nishikawa M, Fujii Y, Tsubomura T.
PMID: 29039865
Dalton Trans. 2017 Nov 07;46(43):14804-14811. doi: 10.1039/c7dt02938a.

We report the synthesis and photophysical properties of Cu(i) complexes with 1,10-phenanthroline (phen) and monodentate phosphine ligands. Single crystal X-ray structural analysis revealed that these have three-coordinated trigonal planar geometries. We also found that one of them, [Cu(phen)(Johnphos)]BF

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Kakizoe D, Nishikawa M, Fujii Y, et al. Photophysical properties of three coordinated copper(i) complexes bearing 1,10-phenanthroline and a monodentate phosphine ligand. Dalton Trans. 2017;46(43):14804-14811doi: 10.1039/c7dt02938a.
Kakizoe, D., Nishikawa, M., Fujii, Y., & Tsubomura, T. (2017). Photophysical properties of three coordinated copper(i) complexes bearing 1,10-phenanthroline and a monodentate phosphine ligand. Dalton transactions (Cambridge, England : 2003), 46(43), 14804-14811. https://doi.org/10.1039/c7dt02938a
Kakizoe, Daichi, et al. "Photophysical properties of three coordinated copper(i) complexes bearing 1,10-phenanthroline and a monodentate phosphine ligand." Dalton transactions (Cambridge, England : 2003) vol. 46,43 (2017): 14804-14811. doi: https://doi.org/10.1039/c7dt02938a
Kakizoe D, Nishikawa M, Fujii Y, Tsubomura T. Photophysical properties of three coordinated copper(i) complexes bearing 1,10-phenanthroline and a monodentate phosphine ligand. Dalton Trans. 2017 Nov 07;46(43):14804-14811. doi: 10.1039/c7dt02938a. PMID: 29039865.
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Dermoscopy findings of hidroacanthoma simplex.

Case reports in dermatology

Sato Y, Fujimura T, Tamabuchi E, Haga T, Aiba S.
PMID: 24987351
Case Rep Dermatol. 2014 May 23;6(2):154-8. doi: 10.1159/000363369. eCollection 2014 May.

Hidroacanthoma simplex (HAS), also known as intraepidermal eccrine poroma, is a rare eccrine adnexal tumor that tends to be misdiagnosed as other types of benign skin tumor, including clonal seborrheic keratosis. Notably, HAS is sometimes misdiagnosed and treated by...

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Sato Y, Fujimura T, Tamabuchi E, et al. Dermoscopy findings of hidroacanthoma simplex. Case Rep Dermatol. 2014;6(2):154-8doi: 10.1159/000363369.
Sato, Y., Fujimura, T., Tamabuchi, E., Haga, T., & Aiba, S. (2014). Dermoscopy findings of hidroacanthoma simplex. Case reports in dermatology, 6(2), 154-8. https://doi.org/10.1159/000363369
Sato, Yota, et al. "Dermoscopy findings of hidroacanthoma simplex." Case reports in dermatology vol. 6,2 (2014): 154-8. doi: https://doi.org/10.1159/000363369
Sato Y, Fujimura T, Tamabuchi E, Haga T, Aiba S. Dermoscopy findings of hidroacanthoma simplex. Case Rep Dermatol. 2014 May 23;6(2):154-8. doi: 10.1159/000363369. eCollection 2014 May. PMID: 24987351; PMCID: PMC4067718.
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Multifocal BRAF(V600E)-Mutated Melanoma in situ on the Foot.

Case reports in dermatology

Iwama E, Fujimura T, Kusakari Y, Haga T, Aiba S.
PMID: 26594172
Case Rep Dermatol. 2015 Oct 30;7(3):322-7. doi: 10.1159/000441601. eCollection 2015.

Melanoma is an aggressive skin cancer that originates from melanocytes, and about one half of melanoma cases possess a BRAF mutation. Together with PD-L1 expression, the BRAF(V600E) mutation is one of the optimal therapeutic targets for the treatment of...

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Iwama E, Fujimura T, Kusakari Y, et al. Multifocal BRAF(V600E)-Mutated Melanoma in situ on the Foot. Case Rep Dermatol. 2015;7(3):322-7doi: 10.1159/000441601.
Iwama, E., Fujimura, T., Kusakari, Y., Haga, T., & Aiba, S. (2015). Multifocal BRAF(V600E)-Mutated Melanoma in situ on the Foot. Case reports in dermatology, 7(3), 322-7. https://doi.org/10.1159/000441601
Iwama, Eimei, et al. "Multifocal BRAF(V600E)-Mutated Melanoma in situ on the Foot." Case reports in dermatology vol. 7,3 (2015): 322-7. doi: https://doi.org/10.1159/000441601
Iwama E, Fujimura T, Kusakari Y, Haga T, Aiba S. Multifocal BRAF(V600E)-Mutated Melanoma in situ on the Foot. Case Rep Dermatol. 2015 Oct 30;7(3):322-7. doi: 10.1159/000441601. eCollection 2015. PMID: 26594172; PMCID: PMC4650989.
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Psoriasiform Drug Eruption Caused by Abatacept: Immunohistochemical Investigation of STAT Signaling.

Case reports in dermatology

Tanita K, Fujimura T, Kakizaki A, Furudate S, Kusakari Y, Aiba S.
PMID: 26351425
Case Rep Dermatol. 2015 Jul 24;7(2):166-70. doi: 10.1159/000437415. eCollection 2015.

Abatacept is a biological immune modifier that is used for the treatment of rheumatoid arthritis. Although psoriasiform drug eruption is reported as one of the cutaneous adverse effects of abatacept, the precise mechanisms are not fully understood. In this...

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Tanita K, Fujimura T, Kakizaki A, et al. Psoriasiform Drug Eruption Caused by Abatacept: Immunohistochemical Investigation of STAT Signaling. Case Rep Dermatol. 2015;7(2):166-70doi: 10.1159/000437415.
Tanita, K., Fujimura, T., Kakizaki, A., Furudate, S., Kusakari, Y., & Aiba, S. (2015). Psoriasiform Drug Eruption Caused by Abatacept: Immunohistochemical Investigation of STAT Signaling. Case reports in dermatology, 7(2), 166-70. https://doi.org/10.1159/000437415
Tanita, Kayo, et al. "Psoriasiform Drug Eruption Caused by Abatacept: Immunohistochemical Investigation of STAT Signaling." Case reports in dermatology vol. 7,2 (2015): 166-70. doi: https://doi.org/10.1159/000437415
Tanita K, Fujimura T, Kakizaki A, Furudate S, Kusakari Y, Aiba S. Psoriasiform Drug Eruption Caused by Abatacept: Immunohistochemical Investigation of STAT Signaling. Case Rep Dermatol. 2015 Jul 24;7(2):166-70. doi: 10.1159/000437415. eCollection 2015. PMID: 26351425; PMCID: PMC4560327.
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Pravastatin reduces radiation-induced damage in normal tissues.

Experimental and therapeutic medicine

Doi H, Matsumoto S, Odawara S, Shikata T, Kitajima K, Tanooka M, Takada Y, Tsujimura T, Kamikonya N, Hirota S.
PMID: 28565765
Exp Ther Med. 2017 May;13(5):1765-1772. doi: 10.3892/etm.2017.4192. Epub 2017 Mar 08.

Pravastatin is an inhibitor of 3-hydroxy-3-methyl- glutaryl-coenzyme A reductase that has been reported to have therapeutic applications in a range of inflammatory conditions. The aim of the present study was to assess the radioprotective effects of pravastatin in an...

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Doi H, Matsumoto S, Odawara S, et al. Pravastatin reduces radiation-induced damage in normal tissues. Exp Ther Med. 2017;13(5):1765-1772doi: 10.3892/etm.2017.4192.
Doi, H., Matsumoto, S., Odawara, S., Shikata, T., Kitajima, K., Tanooka, M., Takada, Y., Tsujimura, T., Kamikonya, N., & Hirota, S. (2017). Pravastatin reduces radiation-induced damage in normal tissues. Experimental and therapeutic medicine, 13(5), 1765-1772. https://doi.org/10.3892/etm.2017.4192
Doi, Hiroshi, et al. "Pravastatin reduces radiation-induced damage in normal tissues." Experimental and therapeutic medicine vol. 13,5 (2017): 1765-1772. doi: https://doi.org/10.3892/etm.2017.4192
Doi H, Matsumoto S, Odawara S, Shikata T, Kitajima K, Tanooka M, Takada Y, Tsujimura T, Kamikonya N, Hirota S. Pravastatin reduces radiation-induced damage in normal tissues. Exp Ther Med. 2017 May;13(5):1765-1772. doi: 10.3892/etm.2017.4192. Epub 2017 Mar 08. PMID: 28565765; PMCID: PMC5443166.
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Showing 1 to 12 of 565 entries