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Melting of Pb Charge Glass and Simultaneous Pb-Cr Charge Transfer in PbCrO3 as the Origin of Volume Collapse.

Journal of the American Chemical Society

Yu R, Hojo H, Watanuki T, Mizumaki M, Mizokawa T, Oka K, Kim H, Machida A, Sakaki K, Nakamura Y, Agui A, Mori D, Inaguma Y, Schlipf M, Rushchanskii KZ, Ležaić M, Matsuda M, Ma J, Calder S, Isobe M, Ikuhara Y, Azuma M.
PMID: 26374486
J Am Chem Soc. 2015 Oct 07;137(39):12719-28. doi: 10.1021/jacs.5b08216. Epub 2015 Sep 25.

A metal to insulator transition in integer or half integer charge systems can be regarded as crystallization of charges. The insulating state tends to have a glassy nature when randomness or geometrical frustration exists. We report that the charge...

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Yu R, Hojo H, Watanuki T, et al. Melting of Pb Charge Glass and Simultaneous Pb-Cr Charge Transfer in PbCrO3 as the Origin of Volume Collapse. J Am Chem Soc. 2015;137(39):12719-28doi: 10.1021/jacs.5b08216.
Yu, R., Hojo, H., Watanuki, T., Mizumaki, M., Mizokawa, T., Oka, K., Kim, H., Machida, A., Sakaki, K., Nakamura, Y., Agui, A., Mori, D., Inaguma, Y., Schlipf, M., Rushchanskii, K. Z., Ležaić, M., Matsuda, M., Ma, J., Calder, S., Isobe, M., Ikuhara, Y., & Azuma, M. (2015). Melting of Pb Charge Glass and Simultaneous Pb-Cr Charge Transfer in PbCrO3 as the Origin of Volume Collapse. Journal of the American Chemical Society, 137(39), 12719-28. https://doi.org/10.1021/jacs.5b08216
Yu, Runze, et al. "Melting of Pb Charge Glass and Simultaneous Pb-Cr Charge Transfer in PbCrO3 as the Origin of Volume Collapse." Journal of the American Chemical Society vol. 137,39 (2015): 12719-28. doi: https://doi.org/10.1021/jacs.5b08216
Yu R, Hojo H, Watanuki T, Mizumaki M, Mizokawa T, Oka K, Kim H, Machida A, Sakaki K, Nakamura Y, Agui A, Mori D, Inaguma Y, Schlipf M, Rushchanskii KZ, Ležaić M, Matsuda M, Ma J, Calder S, Isobe M, Ikuhara Y, Azuma M. Melting of Pb Charge Glass and Simultaneous Pb-Cr Charge Transfer in PbCrO3 as the Origin of Volume Collapse. J Am Chem Soc. 2015 Oct 07;137(39):12719-28. doi: 10.1021/jacs.5b08216. Epub 2015 Sep 25. PMID: 26374486.
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OFF-ON-OFF fluorescent response of N,N,N',N'-tetrakis(1-isoquinolylmethyl)-2-hydroxy-1,3-propanediamine (1-isoHTQHPN) toward Zn(2.).

Dalton transactions (Cambridge, England : 2003)

Mikata Y, Ohnishi R, Ugai A, Konno H, Nakata Y, Hamagami I, Sato S.
PMID: 27007647
Dalton Trans. 2016 Apr 25;45(17):7250-7. doi: 10.1039/c6dt00506c.

An isoquinoline-based ligand, N,N,N',N'-tetrakis(1-isoquinolylmethyl)-2-hydroxy-1,3-propanediamine (1-isoHTQHPN), exhibits a fluorescence increase at 475 nm upon addition of 1 equiv. of Zn(2+) (IZn/I0 = 12, ϕZn = 0.023). This fluorescence enhancement turns and then decreases sharply after the addition of more than...

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Mikata Y, Ohnishi R, Ugai A, et al. OFF-ON-OFF fluorescent response of N,N,N',N'-tetrakis(1-isoquinolylmethyl)-2-hydroxy-1,3-propanediamine (1-isoHTQHPN) toward Zn(2.). Dalton Trans. 2016;45(17):7250-7doi: 10.1039/c6dt00506c.
Mikata, Y., Ohnishi, R., Ugai, A., Konno, H., Nakata, Y., Hamagami, I., & Sato, S. (2016). OFF-ON-OFF fluorescent response of N,N,N',N'-tetrakis(1-isoquinolylmethyl)-2-hydroxy-1,3-propanediamine (1-isoHTQHPN) toward Zn(2.). Dalton transactions (Cambridge, England : 2003), 45(17), 7250-7. https://doi.org/10.1039/c6dt00506c
Mikata, Yuji, et al. "OFF-ON-OFF fluorescent response of N,N,N',N'-tetrakis(1-isoquinolylmethyl)-2-hydroxy-1,3-propanediamine (1-isoHTQHPN) toward Zn(2.)." Dalton transactions (Cambridge, England : 2003) vol. 45,17 (2016): 7250-7. doi: https://doi.org/10.1039/c6dt00506c
Mikata Y, Ohnishi R, Ugai A, Konno H, Nakata Y, Hamagami I, Sato S. OFF-ON-OFF fluorescent response of N,N,N',N'-tetrakis(1-isoquinolylmethyl)-2-hydroxy-1,3-propanediamine (1-isoHTQHPN) toward Zn(2.). Dalton Trans. 2016 Apr 25;45(17):7250-7. doi: 10.1039/c6dt00506c. PMID: 27007647.
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Synthesis and Structure-Activity Relationship Study of 1-Phenyl-1-(quinazolin-4-yl)ethanols as Anticancer Agents.

ACS medicinal chemistry letters

Kuroiwa K, Ishii H, Matsuno K, Asai A, Suzuki Y.
PMID: 25815147
ACS Med Chem Lett. 2015 Jan 10;6(3):287-91. doi: 10.1021/ml5004684. eCollection 2015 Mar 12.

A quinazoline derivative PVHD121 (1a) was shown to have strong antiproliferative activity against various tumor-derived cell lines, including A549 (lung), NCI-H460 (lung), HCT116 (colon), MCF7 (breast), PC3 (prostate), and HeLa (cervical) cells with IC50 values from 0.1 to 0.3...

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Kuroiwa K, Ishii H, Matsuno K, et al. Synthesis and Structure-Activity Relationship Study of 1-Phenyl-1-(quinazolin-4-yl)ethanols as Anticancer Agents. ACS Med Chem Lett. 2015;6(3):287-91doi: 10.1021/ml5004684.
Kuroiwa, K., Ishii, H., Matsuno, K., Asai, A., & Suzuki, Y. (2015). Synthesis and Structure-Activity Relationship Study of 1-Phenyl-1-(quinazolin-4-yl)ethanols as Anticancer Agents. ACS medicinal chemistry letters, 6(3), 287-91. https://doi.org/10.1021/ml5004684
Kuroiwa, Kenta, et al. "Synthesis and Structure-Activity Relationship Study of 1-Phenyl-1-(quinazolin-4-yl)ethanols as Anticancer Agents." ACS medicinal chemistry letters vol. 6,3 (2015): 287-91. doi: https://doi.org/10.1021/ml5004684
Kuroiwa K, Ishii H, Matsuno K, Asai A, Suzuki Y. Synthesis and Structure-Activity Relationship Study of 1-Phenyl-1-(quinazolin-4-yl)ethanols as Anticancer Agents. ACS Med Chem Lett. 2015 Jan 10;6(3):287-91. doi: 10.1021/ml5004684. eCollection 2015 Mar 12. PMID: 25815147; PMCID: PMC4360158.
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Selective breeding of mice for different susceptibilities to high fat diet-induced glucose intolerance: Development of two novel mouse lines, Selectively bred Diet-induced Glucose intolerance-Prone and -Resistant.

Journal of diabetes investigation

Nagao M, Asai A, Kawahara M, Nakajima Y, Sato Y, Tanimura K, Okajima F, Takaya M, Sudo M, Takemitsu S, Harada T, Sugihara H, Oikawa S.
PMID: 24843572
J Diabetes Investig. 2012 Jun 06;3(3):245-51. doi: 10.1111/j.2040-1124.2011.00175.x.

UNLABELLED: Aims/Introduction:  The development of type 2 diabetes is primarily due to lifestyle and environmental factors, as well as genetics, as shown by familial clustering. To establish mouse lines for evaluating heritable factors determining susceptibility to diet-induced diabetes, we...

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Nagao M, Asai A, Kawahara M, et al. Selective breeding of mice for different susceptibilities to high fat diet-induced glucose intolerance: Development of two novel mouse lines, Selectively bred Diet-induced Glucose intolerance-Prone and -Resistant. J Diabetes Investig. 2012;3(3):245-51doi: 10.1111/j.2040-1124.2011.00175.x.
Nagao, M., Asai, A., Kawahara, M., Nakajima, Y., Sato, Y., Tanimura, K., Okajima, F., Takaya, M., Sudo, M., Takemitsu, S., Harada, T., Sugihara, H., & Oikawa, S. (2012). Selective breeding of mice for different susceptibilities to high fat diet-induced glucose intolerance: Development of two novel mouse lines, Selectively bred Diet-induced Glucose intolerance-Prone and -Resistant. Journal of diabetes investigation, 3(3), 245-51. https://doi.org/10.1111/j.2040-1124.2011.00175.x
Nagao, Mototsugu, et al. "Selective breeding of mice for different susceptibilities to high fat diet-induced glucose intolerance: Development of two novel mouse lines, Selectively bred Diet-induced Glucose intolerance-Prone and -Resistant." Journal of diabetes investigation vol. 3,3 (2012): 245-51. doi: https://doi.org/10.1111/j.2040-1124.2011.00175.x
Nagao M, Asai A, Kawahara M, Nakajima Y, Sato Y, Tanimura K, Okajima F, Takaya M, Sudo M, Takemitsu S, Harada T, Sugihara H, Oikawa S. Selective breeding of mice for different susceptibilities to high fat diet-induced glucose intolerance: Development of two novel mouse lines, Selectively bred Diet-induced Glucose intolerance-Prone and -Resistant. J Diabetes Investig. 2012 Jun 06;3(3):245-51. doi: 10.1111/j.2040-1124.2011.00175.x. PMID: 24843572; PMCID: PMC4014945.
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Student use and pedagogical impact of a mobile learning application.

Biochemistry and molecular biology education : a bimonthly publication of the International Union of Biochemistry and Molecular Biology

Teri S, Acai A, Griffith D, Mahmoud Q, Ma DW, Newton G.
PMID: 24375862
Biochem Mol Biol Educ. 2014 Mar-Apr;42(2):121-35. doi: 10.1002/bmb.20771. Epub 2013 Dec 20.

Mobile learning (m-learning) is a relevant innovation in teaching and learning in higher education. A mobile app called NutriBiochem was developed for use in biochemistry and nutrition education for students in a second year Biochemistry and Metabolism course. NutriBiochem...

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Teri S, Acai A, Griffith D, et al. Student use and pedagogical impact of a mobile learning application. Biochem Mol Biol Educ. 2013;42(2):121-35doi: 10.1002/bmb.20771.
Teri, S., Acai, A., Griffith, D., Mahmoud, Q., Ma, D. W., & Newton, G. (2014). Student use and pedagogical impact of a mobile learning application. Biochemistry and molecular biology education : a bimonthly publication of the International Union of Biochemistry and Molecular Biology, 42(2), 121-35. https://doi.org/10.1002/bmb.20771
Teri, Saskia, et al. "Student use and pedagogical impact of a mobile learning application." Biochemistry and molecular biology education : a bimonthly publication of the International Union of Biochemistry and Molecular Biology vol. 42,2 (2014): 121-35. doi: https://doi.org/10.1002/bmb.20771
Teri S, Acai A, Griffith D, Mahmoud Q, Ma DW, Newton G. Student use and pedagogical impact of a mobile learning application. Biochem Mol Biol Educ. 2014 Mar-Apr;42(2):121-35. doi: 10.1002/bmb.20771. Epub 2013 Dec 20. PMID: 24375862.
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Photodynamic therapy with di-l-arginine protoporphyrinate on WiDr human colon adenocarcinoma xenografts in athymic nude mice.

Photodiagnosis and photodynamic therapy

Bugaj A, Kwitniewski M, Iani V, Juzeniene A, Juzenas P, Ma LW, Moan J.
PMID: 25047558
Photodiagnosis Photodyn Ther. 2007 Dec;4(4):237-41. doi: 10.1016/j.pdpdt.2007.08.001. Epub 2007 Nov 08.

The fluorescence kinetics of a new photosensitizer for photodynamic therapy, di-l-arginine protoporphyrinate (PP(Arg)2), was studied in the skin of healthy mice. Furthermore, induction of necrosis in WiDr human colon adenocarcinoma xenografts in athymic nude mice was studied after photodynamic...

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Bugaj A, Kwitniewski M, Iani V, et al. Photodynamic therapy with di-l-arginine protoporphyrinate on WiDr human colon adenocarcinoma xenografts in athymic nude mice. Photodiagnosis Photodyn Ther. 2007;4(4):237-41doi: 10.1016/j.pdpdt.2007.08.001.
Bugaj, A., Kwitniewski, M., Iani, V., Juzeniene, A., Juzenas, P., Ma, L. W., & Moan, J. (2007). Photodynamic therapy with di-l-arginine protoporphyrinate on WiDr human colon adenocarcinoma xenografts in athymic nude mice. Photodiagnosis and photodynamic therapy, 4(4), 237-41. https://doi.org/10.1016/j.pdpdt.2007.08.001
Bugaj, Andrzej, et al. "Photodynamic therapy with di-l-arginine protoporphyrinate on WiDr human colon adenocarcinoma xenografts in athymic nude mice." Photodiagnosis and photodynamic therapy vol. 4,4 (2007): 237-41. doi: https://doi.org/10.1016/j.pdpdt.2007.08.001
Bugaj A, Kwitniewski M, Iani V, Juzeniene A, Juzenas P, Ma LW, Moan J. Photodynamic therapy with di-l-arginine protoporphyrinate on WiDr human colon adenocarcinoma xenografts in athymic nude mice. Photodiagnosis Photodyn Ther. 2007 Dec;4(4):237-41. doi: 10.1016/j.pdpdt.2007.08.001. Epub 2007 Nov 08. PMID: 25047558.
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p62/SQSTM1 levels predicts radiotherapy resistance in hypopharyngeal carcinomas.

American journal of cancer research

Arai A, Chano T, Ikebuchi K, Hama Y, Ochi Y, Tameno H, Shimada T.
PMID: 28469960
Am J Cancer Res. 2017 Apr 01;7(4):881-891. eCollection 2017.

Hypopharyngeal carcinoma is one of the worst prognostic malignancies among head and neck carcinomas. Therefore, a good biomarker should be identified to predict the best therapeutic option before starting the treatment. In cell models, p62/SQSTM1 levels affected the Nrf2-Keap1...

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Arai A, Chano T, Ikebuchi K, et al. p62/SQSTM1 levels predicts radiotherapy resistance in hypopharyngeal carcinomas. Am J Cancer Res. 2017;7(4):881-891
Arai, A., Chano, T., Ikebuchi, K., Hama, Y., Ochi, Y., Tameno, H., & Shimada, T. (2017). p62/SQSTM1 levels predicts radiotherapy resistance in hypopharyngeal carcinomas. American journal of cancer research, 7(4), 881-891.
Arai, Akihito, et al. "p62/SQSTM1 levels predicts radiotherapy resistance in hypopharyngeal carcinomas." American journal of cancer research vol. 7,4 (2017): 881-891.
Arai A, Chano T, Ikebuchi K, Hama Y, Ochi Y, Tameno H, Shimada T. p62/SQSTM1 levels predicts radiotherapy resistance in hypopharyngeal carcinomas. Am J Cancer Res. 2017 Apr 01;7(4):881-891. eCollection 2017. PMID: 28469960; PMCID: PMC5411795.
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[Clinical ethics education in the United States of America].

Nihon Naika Gakkai zasshi. The Journal of the Japanese Society of Internal Medicine

Asai A.
PMID: 7594960
Nihon Naika Gakkai Zasshi. 1995 Aug 10;84(8):1344-9.

No abstract available.

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Asai A. [Clinical ethics education in the United States of America]. Nihon Naika Gakkai Zasshi. 1995;84(8):1344-9
Asai, A. (1995). [Clinical ethics education in the United States of America]. Nihon Naika Gakkai zasshi. The Journal of the Japanese Society of Internal Medicine, 84(8), 1344-9.
Asai, A. "[Clinical ethics education in the United States of America]." Nihon Naika Gakkai zasshi. The Journal of the Japanese Society of Internal Medicine vol. 84,8 (1995): 1344-9.
Asai A. [Clinical ethics education in the United States of America]. Nihon Naika Gakkai Zasshi. 1995 Aug 10;84(8):1344-9. Japanese. PMID: 7594960.
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2-Phenylquinoline-Carbohydrate Hybrids: Molecular Design, Chemical Synthesis, and Evaluation of a New Family of Light-Activatable DNA-Cleaving Agents.

Angewandte Chemie (International ed. in English)

Toshima K, Takano R, Maeda Y, Suzuki M, Asai A, Matsumura S.
PMID: 10649341
Angew Chem Int Ed Engl. 1999 Dec 16;38(24):3733-3735.

Artificial intercalator-carbohydrate hybrids such as that shown in the scheme cleave DNA at the site of guanine on irradiation with UV light with a long wavelength. The hybrids also exhibit strong cytotoxicity when irradiated. The hybrid system is very...

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Toshima K, Takano R, Maeda Y, et al. 2-Phenylquinoline-Carbohydrate Hybrids: Molecular Design, Chemical Synthesis, and Evaluation of a New Family of Light-Activatable DNA-Cleaving Agents. Angew Chem Int Ed Engl. 1999;38(24):3733-3735
Toshima, K., Takano, R., Maeda, Y., Suzuki, M., Asai, A., & Matsumura, S. (1999). 2-Phenylquinoline-Carbohydrate Hybrids: Molecular Design, Chemical Synthesis, and Evaluation of a New Family of Light-Activatable DNA-Cleaving Agents. Angewandte Chemie (International ed. in English), 38(24), 3733-3735.
Toshima, et al. "2-Phenylquinoline-Carbohydrate Hybrids: Molecular Design, Chemical Synthesis, and Evaluation of a New Family of Light-Activatable DNA-Cleaving Agents." Angewandte Chemie (International ed. in English) vol. 38,24 (1999): 3733-3735.
Toshima K, Takano R, Maeda Y, Suzuki M, Asai A, Matsumura S. 2-Phenylquinoline-Carbohydrate Hybrids: Molecular Design, Chemical Synthesis, and Evaluation of a New Family of Light-Activatable DNA-Cleaving Agents. Angew Chem Int Ed Engl. 1999 Dec 16;38(24):3733-3735. PMID: 10649341.
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Time-resolved fluorescent X-ray interference.

Journal of synchrotron radiation

Sasaki YC, Suzuki Y, Yamanashi H, Arai A, Yanagihara M.
PMID: 15263750
J Synchrotron Radiat. 1998 May 01;5:1075-8. doi: 10.1107/S0909049598002787. Epub 1998 May 01.

A fluorescent X-ray interference method can effectively measure nanometer-level conformational changes for non-crystallized molecules and proteins in aqueous conditions. The time-resolved technique can be used to obtain information about the dynamics of molecules and proteins. Instrumentation for time-resolved fluorescent...

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Sasaki YC, Suzuki Y, Yamanashi H, et al. Time-resolved fluorescent X-ray interference. J Synchrotron Radiat. 1998;5:1075-8doi: 10.1107/S0909049598002787.
Sasaki, Y. C., Suzuki, Y., Yamanashi, H., Arai, A., & Yanagihara, M. (1998). Time-resolved fluorescent X-ray interference. Journal of synchrotron radiation, 51075-8. https://doi.org/10.1107/S0909049598002787
Sasaki, Y C, et al. "Time-resolved fluorescent X-ray interference." Journal of synchrotron radiation vol. 5 (1998): 1075-8. doi: https://doi.org/10.1107/S0909049598002787
Sasaki YC, Suzuki Y, Yamanashi H, Arai A, Yanagihara M. Time-resolved fluorescent X-ray interference. J Synchrotron Radiat. 1998 May 01;5:1075-8. doi: 10.1107/S0909049598002787. Epub 1998 May 01. PMID: 15263750.
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Direct-write patterning of indium-tin-oxide film by high pulse repetition frequency femtosecond laser ablation.

Applied optics

Choi HW, Farson DF, Bovatsek J, Arai A, Ashkenasi D.
PMID: 17700755
Appl Opt. 2007 Aug 10;46(23):5792-9. doi: 10.1364/ao.46.005792.

Ablation of indium oxide doped with tin oxide (ITO) from glass substrates is described. Laser pulse energy and focus spot size were varied in single-pulse, single-spot ablation tests and for ablation of linear features with scanned multiple pulses. The...

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Choi HW, Farson DF, Bovatsek J, et al. Direct-write patterning of indium-tin-oxide film by high pulse repetition frequency femtosecond laser ablation. Appl Opt. 2007;46(23):5792-9doi: 10.1364/ao.46.005792.
Choi, H. W., Farson, D. F., Bovatsek, J., Arai, A., & Ashkenasi, D. (2007). Direct-write patterning of indium-tin-oxide film by high pulse repetition frequency femtosecond laser ablation. Applied optics, 46(23), 5792-9. https://doi.org/10.1364/ao.46.005792
Choi, H W, et al. "Direct-write patterning of indium-tin-oxide film by high pulse repetition frequency femtosecond laser ablation." Applied optics vol. 46,23 (2007): 5792-9. doi: https://doi.org/10.1364/ao.46.005792
Choi HW, Farson DF, Bovatsek J, Arai A, Ashkenasi D. Direct-write patterning of indium-tin-oxide film by high pulse repetition frequency femtosecond laser ablation. Appl Opt. 2007 Aug 10;46(23):5792-9. doi: 10.1364/ao.46.005792. PMID: 17700755.
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s-Myc acts as a transcriptional activator and its sequence-specific DNA binding is required for induction of programmed cell death in glioma cells.

Cell death and differentiation

Kitanaka C, Sugiyama A, Kanazu S, Miyagi Y, Mishima K, Asai A, Kuchino Y.
PMID: 17180074
Cell Death Differ. 1995 Apr;2(2):123-31.

We previously reported that s-Myc expression effectively induces programmed cell death (PCD) by apoptosis in glioma cells that express only mutated p53. To determine the molecular mechanism of s-Myc-induced PCD, we examined the correlation between transcriptional activation of s-Myc...

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Kitanaka C, Sugiyama A, Kanazu S, et al. s-Myc acts as a transcriptional activator and its sequence-specific DNA binding is required for induction of programmed cell death in glioma cells. Cell Death Differ. 1995;2(2):123-31
Kitanaka, C., Sugiyama, A., Kanazu, S., Miyagi, Y., Mishima, K., Asai, A., & Kuchino, Y. (1995). s-Myc acts as a transcriptional activator and its sequence-specific DNA binding is required for induction of programmed cell death in glioma cells. Cell death and differentiation, 2(2), 123-31.
Kitanaka, C, et al. "s-Myc acts as a transcriptional activator and its sequence-specific DNA binding is required for induction of programmed cell death in glioma cells." Cell death and differentiation vol. 2,2 (1995): 123-31.
Kitanaka C, Sugiyama A, Kanazu S, Miyagi Y, Mishima K, Asai A, Kuchino Y. s-Myc acts as a transcriptional activator and its sequence-specific DNA binding is required for induction of programmed cell death in glioma cells. Cell Death Differ. 1995 Apr;2(2):123-31. PMID: 17180074.
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Showing 1 to 12 of 374 entries