Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 14 entries
Sorted by: Best Match Show Resources per page
CAND3: A ubiquitously expressed gene immediately adjacent and in opposite transcriptional orientation to the ATM gene at 1lq23.1.

Mammalian genome : official journal of the International Mammalian Genome Society

Chen X, Yang L, Udar N, Liang T, Uhrhammer N, Xu S, Bay JO, Wang Z, Dandakar S, Chiplunkar S, Klisak I, Telatar M, Yang H, Concannon P, Gatti RA.
PMID: 27518307
Mamm Genome. 1997 Feb;8(2):129-33. doi: 10.1007/s003359900371.

Using a magnetic beads-mediated cDNA selection procedure and a fetal brain expression library, we identified a transcriptional unit within a cosmid positive for the marker D11S384. Pursuit of its full-length cDNA led to the cloning of the third candidate...

Cite
Chen X, Yang L, Udar N, et al. CAND3: A ubiquitously expressed gene immediately adjacent and in opposite transcriptional orientation to the ATM gene at 1lq23.1. Mamm Genome. 1997;8(2):129-33doi: 10.1007/s003359900371.
Chen, X., Yang, L., Udar, N., Liang, T., Uhrhammer, N., Xu, S., Bay, J. O., Wang, Z., Dandakar, S., Chiplunkar, S., Klisak, I., Telatar, M., Yang, H., Concannon, P., & Gatti, R. A. (1997). CAND3: A ubiquitously expressed gene immediately adjacent and in opposite transcriptional orientation to the ATM gene at 1lq23.1. Mammalian genome : official journal of the International Mammalian Genome Society, 8(2), 129-33. https://doi.org/10.1007/s003359900371
Chen, X, et al. "CAND3: A ubiquitously expressed gene immediately adjacent and in opposite transcriptional orientation to the ATM gene at 1lq23.1." Mammalian genome : official journal of the International Mammalian Genome Society vol. 8,2 (1997): 129-33. doi: https://doi.org/10.1007/s003359900371
Chen X, Yang L, Udar N, Liang T, Uhrhammer N, Xu S, Bay JO, Wang Z, Dandakar S, Chiplunkar S, Klisak I, Telatar M, Yang H, Concannon P, Gatti RA. CAND3: A ubiquitously expressed gene immediately adjacent and in opposite transcriptional orientation to the ATM gene at 1lq23.1. Mamm Genome. 1997 Feb;8(2):129-33. doi: 10.1007/s003359900371. PMID: 27518307.
Copy Download .nbib Format: NLM
Identification of a novel pathogenic variant in .

Journal of genomics

Laraqui A, Cavaillé M, Uhrhammer N, ElBiad O, Bidet Y, El Rhaffouli H, El Anaz H, Rahali DM, Kouach J, Guelzim K, Badaoui B, AlBouzidi A, Oukabli M, Tanz R, Sbitti Y, Ichou M, Ennibi K, Sekhsokh Y, Bignon YJ.
PMID: 34646395
J Genomics. 2021 Sep 18;9:43-54. doi: 10.7150/jgen.61713. eCollection 2021.

Pathogenic variants (PVs) in

Cite
Laraqui A, Cavaillé M, Uhrhammer N, et al. Identification of a novel pathogenic variant in . J Genomics. 2021;9:43-54doi: 10.7150/jgen.61713.
Laraqui, A., Cavaillé, M., Uhrhammer, N., ElBiad, O., Bidet, Y., El Rhaffouli, H., El Anaz, H., Rahali, D. M., Kouach, J., Guelzim, K., Badaoui, B., AlBouzidi, A., Oukabli, M., Tanz, R., Sbitti, Y., Ichou, M., Ennibi, K., Sekhsokh, Y., & Bignon, Y. J. (2021). Identification of a novel pathogenic variant in . Journal of genomics, 943-54. https://doi.org/10.7150/jgen.61713
Laraqui, Abdelilah, et al. "Identification of a novel pathogenic variant in ." Journal of genomics vol. 9 (2021): 43-54. doi: https://doi.org/10.7150/jgen.61713
Laraqui A, Cavaillé M, Uhrhammer N, ElBiad O, Bidet Y, El Rhaffouli H, El Anaz H, Rahali DM, Kouach J, Guelzim K, Badaoui B, AlBouzidi A, Oukabli M, Tanz R, Sbitti Y, Ichou M, Ennibi K, Sekhsokh Y, Bignon YJ. Identification of a novel pathogenic variant in . J Genomics. 2021 Sep 18;9:43-54. doi: 10.7150/jgen.61713. eCollection 2021. PMID: 34646395; PMCID: PMC8490085.
Copy Download .nbib Format: NLM
5' Region Large Genomic Rearrangements in the .

Cancers

Caputo SM, Telly D, Briaux A, Sesen J, Ceppi M, Bonnet F, Bourdon V, Coulet F, Castera L, Delnatte C, Hardouin A, Mazoyer S, Schultz I, Sevenet N, Uhrhammer N, Bonnet C, Tilkin-Mariamé AF, Houdayer C, Moncoutier V, Andrieu C, French Covar Group Collaborators, Bièche I, Stern MH, Stoppa-Lyonnet D, Lidereau R, Toulas C, Rouleau E.
PMID: 34202044
Cancers (Basel). 2021 Jun 25;13(13). doi: 10.3390/cancers13133171.

BACKGROUND: Large genomic rearrangements (LGR) in METHODS: DNA from 20 families with one apparent duplication and nine deletions was analyzed with a dedicated comparative genomic hybridization (CGH) array, high-resolution BRCA1 Genomic Morse Codes analysis and Sanger sequencing.RESULTS: The apparent...

Cite
Caputo SM, Telly D, Briaux A, et al. 5' Region Large Genomic Rearrangements in the . Cancers (Basel). 2021;13(13)doi: 10.3390/cancers13133171.
Caputo, S. M., Telly, D., Briaux, A., Sesen, J., Ceppi, M., Bonnet, F., Bourdon, V., Coulet, F., Castera, L., Delnatte, C., Hardouin, A., Mazoyer, S., Schultz, I., Sevenet, N., Uhrhammer, N., Bonnet, C., Tilkin-Mariamé, A. F., Houdayer, C., Moncoutier, V., Andrieu, C., French Covar Group Collaborators, Bièche, I., Stern, M. H., Stoppa-Lyonnet, D., Lidereau, R., Toulas, C., & Rouleau, E. (2021). 5' Region Large Genomic Rearrangements in the . Cancers, 13(13), . https://doi.org/10.3390/cancers13133171
Caputo, Sandrine M, et al. "5' Region Large Genomic Rearrangements in the ." Cancers vol. 13,13 (2021). doi: https://doi.org/10.3390/cancers13133171
Caputo SM, Telly D, Briaux A, Sesen J, Ceppi M, Bonnet F, Bourdon V, Coulet F, Castera L, Delnatte C, Hardouin A, Mazoyer S, Schultz I, Sevenet N, Uhrhammer N, Bonnet C, Tilkin-Mariamé AF, Houdayer C, Moncoutier V, Andrieu C, French Covar Group Collaborators, Bièche I, Stern MH, Stoppa-Lyonnet D, Lidereau R, Toulas C, Rouleau E. 5' Region Large Genomic Rearrangements in the . Cancers (Basel). 2021 Jun 25;13(13). doi: 10.3390/cancers13133171. PMID: 34202044; PMCID: PMC8268747.
Copy Download .nbib Format: NLM
Use of gene expression profiles of peripheral blood lymphocytes to distinguish BRCA1 mutation carriers in high risk breast cancer families.

Cancer informatics

Vuillaume ML, Uhrhammer N, Vidal V, Vidal VS, Chabaud V, Jesson B, Kwiatkowski F, Bignon YJ.
PMID: 19352458
Cancer Inform. 2009;7:41-56. doi: 10.4137/cin.s931. Epub 2009 Mar 02.

Mutations in two major genes, BRCA1 and BRCA2, account for up to 30% of families with hereditary breast cancer. Unfortunately, in most families there is little to indicate which gene should be targeted first for mutation screening, which is...

Cite
Vuillaume ML, Uhrhammer N, Vidal V, et al. Use of gene expression profiles of peripheral blood lymphocytes to distinguish BRCA1 mutation carriers in high risk breast cancer families. Cancer Inform. 2009;7:41-56doi: 10.4137/cin.s931.
Vuillaume, M. L., Uhrhammer, N., Vidal, V., Vidal, V. S., Chabaud, V., Jesson, B., Kwiatkowski, F., & Bignon, Y. J. (2009). Use of gene expression profiles of peripheral blood lymphocytes to distinguish BRCA1 mutation carriers in high risk breast cancer families. Cancer informatics, 741-56. https://doi.org/10.4137/cin.s931
Vuillaume, Marie-Laure, et al. "Use of gene expression profiles of peripheral blood lymphocytes to distinguish BRCA1 mutation carriers in high risk breast cancer families." Cancer informatics vol. 7 (2009): 41-56. doi: https://doi.org/10.4137/cin.s931
Vuillaume ML, Uhrhammer N, Vidal V, Vidal VS, Chabaud V, Jesson B, Kwiatkowski F, Bignon YJ. Use of gene expression profiles of peripheral blood lymphocytes to distinguish BRCA1 mutation carriers in high risk breast cancer families. Cancer Inform. 2009;7:41-56. doi: 10.4137/cin.s931. Epub 2009 Mar 02. PMID: 19352458; PMCID: PMC2664702.
Copy Download .nbib Format: NLM
Prevalance of BRCA1 and BRCA2 mutations in familial breast cancer patients in Lebanon.

Hereditary cancer in clinical practice

Jalkh N, Nassar-Slaba J, Chouery E, Salem N, Uhrchammer N, Golmard L, Stoppa-Lyonnet D, Bignon YJ, Mégarbané A.
PMID: 22713736
Hered Cancer Clin Pract. 2012 Jun 19;10(1):7. doi: 10.1186/1897-4287-10-7.

Breast cancer is the most prevalent malignancy in women in Western countries, currently accounting for one third of all female cancers. Familial aggregation is thought to account for 5-10 % of all BC cases, and germline mutations in BRCA1...

Cite
Jalkh N, Nassar-Slaba J, Chouery E, et al. Prevalance of BRCA1 and BRCA2 mutations in familial breast cancer patients in Lebanon. Hered Cancer Clin Pract. 2012;10(1):7doi: 10.1186/1897-4287-10-7.
Jalkh, N., Nassar-Slaba, J., Chouery, E., Salem, N., Uhrchammer, N., Golmard, L., Stoppa-Lyonnet, D., Bignon, Y. J., & Mégarbané, A. (2012). Prevalance of BRCA1 and BRCA2 mutations in familial breast cancer patients in Lebanon. Hereditary cancer in clinical practice, 10(1), 7. https://doi.org/10.1186/1897-4287-10-7
Jalkh, Nadine, et al. "Prevalance of BRCA1 and BRCA2 mutations in familial breast cancer patients in Lebanon." Hereditary cancer in clinical practice vol. 10,1 (2012): 7. doi: https://doi.org/10.1186/1897-4287-10-7
Jalkh N, Nassar-Slaba J, Chouery E, Salem N, Uhrchammer N, Golmard L, Stoppa-Lyonnet D, Bignon YJ, Mégarbané A. Prevalance of BRCA1 and BRCA2 mutations in familial breast cancer patients in Lebanon. Hered Cancer Clin Pract. 2012 Jun 19;10(1):7. doi: 10.1186/1897-4287-10-7. PMID: 22713736; PMCID: PMC3441239.
Copy Download .nbib Format: NLM
DNA repair genes implicated in triple negative familial non-BRCA1/2 breast cancer predisposition.

American journal of cancer research

Ollier M, Radosevic-Robin N, Kwiatkowski F, Ponelle F, Viala S, Privat M, Uhrhammer N, Bernard-Gallon D, Penault-Llorca F, Bignon YJ, Bidet Y.
PMID: 26328243
Am J Cancer Res. 2015 Jun 15;5(7):2113-26. eCollection 2015.

Among breast cancers, 10 to 15% of cases would be due to hereditary risk. In these familial cases, mutations in BRCA1 and BRCA2 are found in only 15% to 20%, meaning that new susceptibility genes remain to be found....

Cite
Ollier M, Radosevic-Robin N, Kwiatkowski F, et al. DNA repair genes implicated in triple negative familial non-BRCA1/2 breast cancer predisposition. Am J Cancer Res. 2015;5(7):2113-26
Ollier, M., Radosevic-Robin, N., Kwiatkowski, F., Ponelle, F., Viala, S., Privat, M., Uhrhammer, N., Bernard-Gallon, D., Penault-Llorca, F., Bignon, Y. J., & Bidet, Y. (2015). DNA repair genes implicated in triple negative familial non-BRCA1/2 breast cancer predisposition. American journal of cancer research, 5(7), 2113-26.
Ollier, Marie, et al. "DNA repair genes implicated in triple negative familial non-BRCA1/2 breast cancer predisposition." American journal of cancer research vol. 5,7 (2015): 2113-26.
Ollier M, Radosevic-Robin N, Kwiatkowski F, Ponelle F, Viala S, Privat M, Uhrhammer N, Bernard-Gallon D, Penault-Llorca F, Bignon YJ, Bidet Y. DNA repair genes implicated in triple negative familial non-BRCA1/2 breast cancer predisposition. Am J Cancer Res. 2015 Jun 15;5(7):2113-26. eCollection 2015. PMID: 26328243; PMCID: PMC4548324.
Copy Download .nbib Format: NLM
Analysis of 11 candidate genes in 849 adult patients with suspected hereditary cancer predisposition.

Genes, chromosomes & cancer

Cavaillé M, Uhrhammer N, Privat M, Ponelle-Chachuat F, Gay-Bellile M, Lepage M, Molnar I, Viala S, Bidet Y, Bignon YJ.
PMID: 33099839
Genes Chromosomes Cancer. 2021 Feb;60(2):73-78. doi: 10.1002/gcc.22911. Epub 2020 Nov 10.

Hereditary predisposition to cancer concerns between 5% and 10% of cancers. The main genes involved in the most frequent syndromes (hereditary breast and ovarian cancer syndrome, hereditary nonpolyposis colorectal cancer syndrome) were identified in the 1990s. Exploration of their...

Cite
Cavaillé M, Uhrhammer N, Privat M, et al. Analysis of 11 candidate genes in 849 adult patients with suspected hereditary cancer predisposition. Genes Chromosomes Cancer. 2020;60(2):73-78doi: 10.1002/gcc.22911.
Cavaillé, M., Uhrhammer, N., Privat, M., Ponelle-Chachuat, F., Gay-Bellile, M., Lepage, M., Molnar, I., Viala, S., Bidet, Y., & Bignon, Y. J. (2021). Analysis of 11 candidate genes in 849 adult patients with suspected hereditary cancer predisposition. Genes, chromosomes & cancer, 60(2), 73-78. https://doi.org/10.1002/gcc.22911
Cavaillé, Mathias, et al. "Analysis of 11 candidate genes in 849 adult patients with suspected hereditary cancer predisposition." Genes, chromosomes & cancer vol. 60,2 (2021): 73-78. doi: https://doi.org/10.1002/gcc.22911
Cavaillé M, Uhrhammer N, Privat M, Ponelle-Chachuat F, Gay-Bellile M, Lepage M, Molnar I, Viala S, Bidet Y, Bignon YJ. Analysis of 11 candidate genes in 849 adult patients with suspected hereditary cancer predisposition. Genes Chromosomes Cancer. 2021 Feb;60(2):73-78. doi: 10.1002/gcc.22911. Epub 2020 Nov 10. PMID: 33099839; PMCID: PMC7756731.
Copy Download .nbib Format: NLM
Early Onset Multiple Primary Tumors in Atypical Presentation of Cowden Syndrome Identified by Whole-Exome-Sequencing.

Frontiers in genetics

Cavaillé M, Ponelle-Chachuat F, Uhrhammer N, Viala S, Gay-Bellile M, Privat M, Bidet Y, Bignon YJ.
PMID: 30233642
Front Genet. 2018 Aug 31;9:353. doi: 10.3389/fgene.2018.00353. eCollection 2018.

A family with an aggregation of rare early onset multiple primary tumors has been managed in our oncogenetics department: the proband developed four early onset carcinomas between ages 31 and 33 years, including acral melanoma, bilateral clear cell renal...

Cite
Cavaillé M, Ponelle-Chachuat F, Uhrhammer N, et al. Early Onset Multiple Primary Tumors in Atypical Presentation of Cowden Syndrome Identified by Whole-Exome-Sequencing. Front Genet. 2018;9:353doi: 10.3389/fgene.2018.00353.
Cavaillé, M., Ponelle-Chachuat, F., Uhrhammer, N., Viala, S., Gay-Bellile, M., Privat, M., Bidet, Y., & Bignon, Y. J. (2018). Early Onset Multiple Primary Tumors in Atypical Presentation of Cowden Syndrome Identified by Whole-Exome-Sequencing. Frontiers in genetics, 9353. https://doi.org/10.3389/fgene.2018.00353
Cavaillé, Mathias, et al. "Early Onset Multiple Primary Tumors in Atypical Presentation of Cowden Syndrome Identified by Whole-Exome-Sequencing." Frontiers in genetics vol. 9 (2018): 353. doi: https://doi.org/10.3389/fgene.2018.00353
Cavaillé M, Ponelle-Chachuat F, Uhrhammer N, Viala S, Gay-Bellile M, Privat M, Bidet Y, Bignon YJ. Early Onset Multiple Primary Tumors in Atypical Presentation of Cowden Syndrome Identified by Whole-Exome-Sequencing. Front Genet. 2018 Aug 31;9:353. doi: 10.3389/fgene.2018.00353. eCollection 2018. PMID: 30233642; PMCID: PMC6127642.
Copy Download .nbib Format: NLM
Full in-frame exon 3 skipping of .

Oncotarget

Caputo SM, Léone M, Damiola F, Ehlen A, Carreira A, Gaidrat P, Martins A, Brandão RD, Peixoto A, Vega A, Houdayer C, Delnatte C, Bronner M, Muller D, Castera L, Guillaud-Bataille M, Søkilde I, Uhrhammer N, Demontety S, Tubeuf H, Castelain G, Jensen UB, Petitalot A, Krieger S, Lefol C, Moncoutier V, Boutry-Kryza N, Nielsen HR, Sinilnikova O, Stoppa-Lyonnet D, Spurdle AB, Teixeira MR, Coulet F, Thomassen M, Rouleau E.
PMID: 29707112
Oncotarget. 2018 Apr 03;9(25):17334-17348. doi: 10.18632/oncotarget.24671. eCollection 2018 Apr 03.

Germline pathogenic variants in the

Cite
Caputo SM, Léone M, Damiola F, et al. Full in-frame exon 3 skipping of . Oncotarget. 2018;9(25):17334-17348doi: 10.18632/oncotarget.24671.
Caputo, S. M., Léone, M., Damiola, F., Ehlen, A., Carreira, A., Gaidrat, P., Martins, A., Brandão, R. D., Peixoto, A., Vega, A., Houdayer, C., Delnatte, C., Bronner, M., Muller, D., Castera, L., Guillaud-Bataille, M., Søkilde, I., Uhrhammer, N., Demontety, S., Tubeuf, H., Castelain, G., Jensen, U. B., Petitalot, A., Krieger, S., Lefol, C., Moncoutier, V., Boutry-Kryza, N., Nielsen, H. R., Sinilnikova, O., Stoppa-Lyonnet, D., Spurdle, A. B., Teixeira, M. R., Coulet, F., Thomassen, M., & Rouleau, E. (2018). Full in-frame exon 3 skipping of . Oncotarget, 9(25), 17334-17348. https://doi.org/10.18632/oncotarget.24671
Caputo, Sandrine M, et al. "Full in-frame exon 3 skipping of ." Oncotarget vol. 9,25 (2018): 17334-17348. doi: https://doi.org/10.18632/oncotarget.24671
Caputo SM, Léone M, Damiola F, Ehlen A, Carreira A, Gaidrat P, Martins A, Brandão RD, Peixoto A, Vega A, Houdayer C, Delnatte C, Bronner M, Muller D, Castera L, Guillaud-Bataille M, Søkilde I, Uhrhammer N, Demontety S, Tubeuf H, Castelain G, Jensen UB, Petitalot A, Krieger S, Lefol C, Moncoutier V, Boutry-Kryza N, Nielsen HR, Sinilnikova O, Stoppa-Lyonnet D, Spurdle AB, Teixeira MR, Coulet F, Thomassen M, Rouleau E. Full in-frame exon 3 skipping of . Oncotarget. 2018 Apr 03;9(25):17334-17348. doi: 10.18632/oncotarget.24671. eCollection 2018 Apr 03. PMID: 29707112; PMCID: PMC5915120.
Copy Download .nbib Format: NLM
Improved Efficiency and Reliability of NGS Amplicon Sequencing Data Analysis for Genetic Diagnostic Procedures Using AGSA Software.

BioMed research international

Poulet A, Privat M, Ponelle F, Viala S, Decousus S, Perin A, Lafarge L, Ollier M, El Saghir NS, Uhrhammer N, Bignon YJ, Bidet Y.
PMID: 27656653
Biomed Res Int. 2016;2016:5623089. doi: 10.1155/2016/5623089. Epub 2016 Aug 30.

Screening for

Cite
Poulet A, Privat M, Ponelle F, et al. Improved Efficiency and Reliability of NGS Amplicon Sequencing Data Analysis for Genetic Diagnostic Procedures Using AGSA Software. Biomed Res Int. 2016;2016:5623089doi: 10.1155/2016/5623089.
Poulet, A., Privat, M., Ponelle, F., Viala, S., Decousus, S., Perin, A., Lafarge, L., Ollier, M., El Saghir, N. S., Uhrhammer, N., Bignon, Y. J., & Bidet, Y. (2016). Improved Efficiency and Reliability of NGS Amplicon Sequencing Data Analysis for Genetic Diagnostic Procedures Using AGSA Software. BioMed research international, 20165623089. https://doi.org/10.1155/2016/5623089
Poulet, Axel, et al. "Improved Efficiency and Reliability of NGS Amplicon Sequencing Data Analysis for Genetic Diagnostic Procedures Using AGSA Software." BioMed research international vol. 2016 (2016): 5623089. doi: https://doi.org/10.1155/2016/5623089
Poulet A, Privat M, Ponelle F, Viala S, Decousus S, Perin A, Lafarge L, Ollier M, El Saghir NS, Uhrhammer N, Bignon YJ, Bidet Y. Improved Efficiency and Reliability of NGS Amplicon Sequencing Data Analysis for Genetic Diagnostic Procedures Using AGSA Software. Biomed Res Int. 2016;2016:5623089. doi: 10.1155/2016/5623089. Epub 2016 Aug 30. PMID: 27656653; PMCID: PMC5021467.
Copy Download .nbib Format: NLM
Dynamic exciton localisation in a pyrene-BODIPY-pyrene dye conjugate.

Physical chemistry chemical physics : PCCP

Auerhammer N, Schulz A, Schmiedel A, Holzapfel M, Hoche J, Röhr MIS, Mitric R, Lambert C.
PMID: 30931442
Phys Chem Chem Phys. 2019 May 08;21(18):9013-9025. doi: 10.1039/c9cp00908f.

The photophysics of a molecular triad consisting of a BODIPY dye and two pyrene chromophores attached in 2-position are investigated by steady state and fs-time resolved transient absorption spectroscopy as well as by field induced surface hopping (FISH) simulations....

Cite
Auerhammer N, Schulz A, Schmiedel A, et al. Dynamic exciton localisation in a pyrene-BODIPY-pyrene dye conjugate. Phys Chem Chem Phys. 2019;21(18):9013-9025doi: 10.1039/c9cp00908f.
Auerhammer, N., Schulz, A., Schmiedel, A., Holzapfel, M., Hoche, J., Röhr, M. I. S., Mitric, R., & Lambert, C. (2019). Dynamic exciton localisation in a pyrene-BODIPY-pyrene dye conjugate. Physical chemistry chemical physics : PCCP, 21(18), 9013-9025. https://doi.org/10.1039/c9cp00908f
Auerhammer, Nina, et al. "Dynamic exciton localisation in a pyrene-BODIPY-pyrene dye conjugate." Physical chemistry chemical physics : PCCP vol. 21,18 (2019): 9013-9025. doi: https://doi.org/10.1039/c9cp00908f
Auerhammer N, Schulz A, Schmiedel A, Holzapfel M, Hoche J, Röhr MIS, Mitric R, Lambert C. Dynamic exciton localisation in a pyrene-BODIPY-pyrene dye conjugate. Phys Chem Chem Phys. 2019 May 08;21(18):9013-9025. doi: 10.1039/c9cp00908f. PMID: 30931442.
Copy Download .nbib Format: NLM
Modulation of molybdenum-centered redox potentials and electron-transfer rates by sulfur versus oxygen ligation.

Inorganic chemistry

Uhrhammer D, Schultz FA.
PMID: 15530089
Inorg Chem. 2004 Nov 15;43(23):7389-95. doi: 10.1021/ic040082i.

Temperature-dependent measurements of potential, E degrees', and electron-transfer rate constant, k(s,h), are reported for electrochemical reduction (in 0.3 M TBAPF(6)/CH(3)CN) of a series of oxomolybdenum(V) complexes, [(Tp)MoO(X,Y)], where Tp = hydrotris(3,5-dimethyl-1-pyrazolyl)borate and X,Y is a series of bidentate 1,2-disubstituted...

Cite
Uhrhammer D, Schultz FA. Modulation of molybdenum-centered redox potentials and electron-transfer rates by sulfur versus oxygen ligation. Inorg Chem. 2004;43(23):7389-95doi: 10.1021/ic040082i.
Uhrhammer, D., & Schultz, F. A. (2004). Modulation of molybdenum-centered redox potentials and electron-transfer rates by sulfur versus oxygen ligation. Inorganic chemistry, 43(23), 7389-95. https://doi.org/10.1021/ic040082i
Uhrhammer, Darrell, and Schultz, Franklin A. "Modulation of molybdenum-centered redox potentials and electron-transfer rates by sulfur versus oxygen ligation." Inorganic chemistry vol. 43,23 (2004): 7389-95. doi: https://doi.org/10.1021/ic040082i
Uhrhammer D, Schultz FA. Modulation of molybdenum-centered redox potentials and electron-transfer rates by sulfur versus oxygen ligation. Inorg Chem. 2004 Nov 15;43(23):7389-95. doi: 10.1021/ic040082i. PMID: 15530089.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 14 entries