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Knock-in Luciferase Reporter Mice for In Vivo Monitoring of CREB Activity.

PloS one

Akhmedov D, Rajendran K, Mendoza-Rodriguez MG, Berdeaux R.
PMID: 27336479
PLoS One. 2016 Jun 23;11(6):e0158274. doi: 10.1371/journal.pone.0158274. eCollection 2016.

The cAMP response element binding protein (CREB) is induced during fasting in the liver, where it stimulates transcription of rate-limiting gluconeogenic genes to maintain metabolic homeostasis. Adenoviral and transgenic CREB reporters have been used to monitor hepatic CREB activity...

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Akhmedov D, Rajendran K, Mendoza-Rodriguez MG, et al. Knock-in Luciferase Reporter Mice for In Vivo Monitoring of CREB Activity. PLoS One. 2016;11(6):e0158274doi: 10.1371/journal.pone.0158274.
Akhmedov, D., Rajendran, K., Mendoza-Rodriguez, M. G., & Berdeaux, R. (2016). Knock-in Luciferase Reporter Mice for In Vivo Monitoring of CREB Activity. PloS one, 11(6), e0158274. https://doi.org/10.1371/journal.pone.0158274
Akhmedov, Dmitry, et al. "Knock-in Luciferase Reporter Mice for In Vivo Monitoring of CREB Activity." PloS one vol. 11,6 (2016): e0158274. doi: https://doi.org/10.1371/journal.pone.0158274
Akhmedov D, Rajendran K, Mendoza-Rodriguez MG, Berdeaux R. Knock-in Luciferase Reporter Mice for In Vivo Monitoring of CREB Activity. PLoS One. 2016 Jun 23;11(6):e0158274. doi: 10.1371/journal.pone.0158274. eCollection 2016. PMID: 27336479; PMCID: PMC4940169.
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The effects of obesity on skeletal muscle regeneration.

Frontiers in physiology

Akhmedov D, Berdeaux R.
PMID: 24381559
Front Physiol. 2013 Dec 17;4:371. doi: 10.3389/fphys.2013.00371.

Obesity and metabolic disorders such as type 2 diabetes mellitus are accompanied by increased lipid deposition in adipose and non-adipose tissues including liver, pancreas, heart and skeletal muscle. Recent publications report impaired regenerative capacity of skeletal muscle following injury...

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Akhmedov D, Berdeaux R. The effects of obesity on skeletal muscle regeneration. Front Physiol. 2013;4:371doi: 10.3389/fphys.2013.00371.
Akhmedov, D., & Berdeaux, R. (2013). The effects of obesity on skeletal muscle regeneration. Frontiers in physiology, 4371. https://doi.org/10.3389/fphys.2013.00371
Akhmedov, Dmitry, and Berdeaux, Rebecca. "The effects of obesity on skeletal muscle regeneration." Frontiers in physiology vol. 4 (2013): 371. doi: https://doi.org/10.3389/fphys.2013.00371
Akhmedov D, Berdeaux R. The effects of obesity on skeletal muscle regeneration. Front Physiol. 2013 Dec 17;4:371. doi: 10.3389/fphys.2013.00371. PMID: 24381559; PMCID: PMC3865699.
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[The problem of activation of EEGs in psychiatry].

Archivio internazionale di studi neurologici

VERDEAUX G, VERDEAUX J.
PMID: 13008580
Arch Internazionale Studi Neurol. 1952 Sep;1(4):512-22.

No abstract available.

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VERDEAUX G, VERDEAUX J. Le problème de l'activation des E.E.G. en psychiatrie. [The problem of activation of EEGs in psychiatry]. Arch Internazionale Studi Neurol. 1952;1(4):512-22
VERDEAUX, G., & VERDEAUX, J. (1952). Le problème de l'activation des E.E.G. en psychiatrie. [The problem of activation of EEGs in psychiatry]. Archivio internazionale di studi neurologici, 1(4), 512-22.
VERDEAUX, G, and VERDEAUX, J. "Le problème de l'activation des E.E.G. en psychiatrie." [The problem of activation of EEGs in psychiatry]. Archivio internazionale di studi neurologici vol. 1,4 (1952): 512-22.
VERDEAUX G, VERDEAUX J. Le problème de l'activation des E.E.G. en psychiatrie. [The problem of activation of EEGs in psychiatry]. Arch Internazionale Studi Neurol. 1952 Sep;1(4):512-22. Undetermined. PMID: 13008580.
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[Epileptic seizures followed by sleep; activation of E.E.G. by hyperpnoea and sleep].

Revue neurologique

LEROY R, VERDEAUX G.
PMID: 14786995
Rev Neurol (Paris). 1950 Jun;82(6):558-9.

No abstract available.

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LEROY R, VERDEAUX G. Crises comitiales avec sommeil posteritique. Activation de l'E.E.G. par l'hyperpnée et le sommeil. [Epileptic seizures followed by sleep; activation of E.E.G. by hyperpnoea and sleep]. Rev Neurol (Paris). 1950;82(6):558-9
LEROY, R., & VERDEAUX, G. (1950). Crises comitiales avec sommeil posteritique. Activation de l'E.E.G. par l'hyperpnée et le sommeil. [Epileptic seizures followed by sleep; activation of E.E.G. by hyperpnoea and sleep]. Revue neurologique, 82(6), 558-9.
LEROY, R, and VERDEAUX, G. "Crises comitiales avec sommeil posteritique. Activation de l'E.E.G. par l'hyperpnée et le sommeil." [Epileptic seizures followed by sleep; activation of E.E.G. by hyperpnoea and sleep]. Revue neurologique vol. 82,6 (1950): 558-9.
LEROY R, VERDEAUX G. Crises comitiales avec sommeil posteritique. Activation de l'E.E.G. par l'hyperpnée et le sommeil. [Epileptic seizures followed by sleep; activation of E.E.G. by hyperpnoea and sleep]. Rev Neurol (Paris). 1950 Jun;82(6):558-9. Undetermined. PMID: 14786995.
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The epidemiology, economics and quality of life burden of age-related macular degeneration in France, Germany, Italy and the United Kingdom.

The European journal of health economics : HEPAC : health economics in prevention and care

Bonastre J, Le Pen C, Anderson P, Ganz A, Berto P, Berdeaux G.
PMID: 15609135
Eur J Health Econ. 2002;3(2):94-102. doi: 10.1007/s10198-002-0104-y.

Age-related macular degeneration (AMD) is a major public health issue, but little is known about the economics of the disease. This contribution describes the epidemiology and the economics of AMD in four European countries: France, Germany, Italy and the...

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Bonastre J, Le Pen C, Anderson P, et al. The epidemiology, economics and quality of life burden of age-related macular degeneration in France, Germany, Italy and the United Kingdom. Eur J Health Econ. 2002;3(2):94-102doi: 10.1007/s10198-002-0104-y.
Bonastre, J., Le Pen, C., Anderson, P., Ganz, A., Berto, P., & Berdeaux, G. (2002). The epidemiology, economics and quality of life burden of age-related macular degeneration in France, Germany, Italy and the United Kingdom. The European journal of health economics : HEPAC : health economics in prevention and care, 3(2), 94-102. https://doi.org/10.1007/s10198-002-0104-y
Bonastre, J, et al. "The epidemiology, economics and quality of life burden of age-related macular degeneration in France, Germany, Italy and the United Kingdom." The European journal of health economics : HEPAC : health economics in prevention and care vol. 3,2 (2002): 94-102. doi: https://doi.org/10.1007/s10198-002-0104-y
Bonastre J, Le Pen C, Anderson P, Ganz A, Berto P, Berdeaux G. The epidemiology, economics and quality of life burden of age-related macular degeneration in France, Germany, Italy and the United Kingdom. Eur J Health Econ. 2002;3(2):94-102. doi: 10.1007/s10198-002-0104-y. PMID: 15609135.
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Treatment persistence and cost-effectiveness of latanoprost/latanoprost-timolol, bimatoprost/bimatoprost-timolol, and travoprost/travoprost-timolol in glaucoma: an analysis based on the United Kingdom general practitioner research database.

Clinical ophthalmology (Auckland, N.Z.)

Lafuma A, Salmon JF, Robert J, Berdeaux G.
PMID: 21468347
Clin Ophthalmol. 2011;5:361-7. doi: 10.2147/OPTH.S16888. Epub 2011 Mar 14.

OBJECTIVE: To compare treatment persistence and costs with 3 glaucoma treatment sequences (first-line/second-line): latanoprost/latanoprost-timolol (LLT), bimatoprost/bimatoprost-timolol (BBT), and travoprost/travoprost-timolol (TTT), derived from the UK General Practitioner Research Database (UK-GPRD).METHODS: Patient records referring to ocular hypertension, topical glaucoma treatment, surgery,...

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Lafuma A, Salmon JF, Robert J, et al. Treatment persistence and cost-effectiveness of latanoprost/latanoprost-timolol, bimatoprost/bimatoprost-timolol, and travoprost/travoprost-timolol in glaucoma: an analysis based on the United Kingdom general practitioner research database. Clin Ophthalmol. 2011;5:361-7doi: 10.2147/OPTH.S16888.
Lafuma, A., Salmon, J. F., Robert, J., & Berdeaux, G. (2011). Treatment persistence and cost-effectiveness of latanoprost/latanoprost-timolol, bimatoprost/bimatoprost-timolol, and travoprost/travoprost-timolol in glaucoma: an analysis based on the United Kingdom general practitioner research database. Clinical ophthalmology (Auckland, N.Z.), 5361-7. https://doi.org/10.2147/OPTH.S16888
Lafuma, Antoine, et al. "Treatment persistence and cost-effectiveness of latanoprost/latanoprost-timolol, bimatoprost/bimatoprost-timolol, and travoprost/travoprost-timolol in glaucoma: an analysis based on the United Kingdom general practitioner research database." Clinical ophthalmology (Auckland, N.Z.) vol. 5 (2011): 361-7. doi: https://doi.org/10.2147/OPTH.S16888
Lafuma A, Salmon JF, Robert J, Berdeaux G. Treatment persistence and cost-effectiveness of latanoprost/latanoprost-timolol, bimatoprost/bimatoprost-timolol, and travoprost/travoprost-timolol in glaucoma: an analysis based on the United Kingdom general practitioner research database. Clin Ophthalmol. 2011;5:361-7. doi: 10.2147/OPTH.S16888. Epub 2011 Mar 14. PMID: 21468347; PMCID: PMC3065581.
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Loss and gain of function of Grp75 or mitofusin 2 distinctly alter cholesterol metabolism, but all promote triglyceride accumulation in hepatocytes.

Biochimica et biophysica acta. Molecular and cell biology of lipids

Bassot A, Prip-Buus C, Alves A, Berdeaux O, Perrier J, Lenoir V, Ji-Cao J, Berger MA, Loizon E, Cabaret S, Panthu B, Rieusset J, Morio B.
PMID: 34419589
Biochim Biophys Acta Mol Cell Biol Lipids. 2021 Dec;1866(12):159030. doi: 10.1016/j.bbalip.2021.159030. Epub 2021 Aug 20.

In the liver, contact sites between the endoplasmic reticulum (ER) and mitochondria (named MAMs) may be crucial hubs for the regulation of lipid metabolism, thus contributing to the exacerbation or prevention of fatty liver. We hypothesized that tether proteins...

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Bassot A, Prip-Buus C, Alves A, et al. Loss and gain of function of Grp75 or mitofusin 2 distinctly alter cholesterol metabolism, but all promote triglyceride accumulation in hepatocytes. Biochim Biophys Acta Mol Cell Biol Lipids. 2021;1866(12):159030doi: 10.1016/j.bbalip.2021.159030.
Bassot, A., Prip-Buus, C., Alves, A., Berdeaux, O., Perrier, J., Lenoir, V., Ji-Cao, J., Berger, M. A., Loizon, E., Cabaret, S., Panthu, B., Rieusset, J., & Morio, B. (2021). Loss and gain of function of Grp75 or mitofusin 2 distinctly alter cholesterol metabolism, but all promote triglyceride accumulation in hepatocytes. Biochimica et biophysica acta. Molecular and cell biology of lipids, 1866(12), 159030. https://doi.org/10.1016/j.bbalip.2021.159030
Bassot, Arthur, et al. "Loss and gain of function of Grp75 or mitofusin 2 distinctly alter cholesterol metabolism, but all promote triglyceride accumulation in hepatocytes." Biochimica et biophysica acta. Molecular and cell biology of lipids vol. 1866,12 (2021): 159030. doi: https://doi.org/10.1016/j.bbalip.2021.159030
Bassot A, Prip-Buus C, Alves A, Berdeaux O, Perrier J, Lenoir V, Ji-Cao J, Berger MA, Loizon E, Cabaret S, Panthu B, Rieusset J, Morio B. Loss and gain of function of Grp75 or mitofusin 2 distinctly alter cholesterol metabolism, but all promote triglyceride accumulation in hepatocytes. Biochim Biophys Acta Mol Cell Biol Lipids. 2021 Dec;1866(12):159030. doi: 10.1016/j.bbalip.2021.159030. Epub 2021 Aug 20. PMID: 34419589.
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Loss and gain of function of Grp75 or mitofusin 2 distinctly alter cholesterol metabolism, but all promote triglyceride accumulation in hepatocytes.

Biochimica et biophysica acta. Molecular and cell biology of lipids

Bassot A, Prip-Buus C, Alves A, Berdeaux O, Perrier J, Lenoir V, Ji-Cao J, Berger MA, Loizon E, Cabaret S, Panthu B, Rieusset J, Morio B.
PMID: 34419589
Biochim Biophys Acta Mol Cell Biol Lipids. 2021 Dec;1866(12):159030. doi: 10.1016/j.bbalip.2021.159030. Epub 2021 Aug 20.

In the liver, contact sites between the endoplasmic reticulum (ER) and mitochondria (named MAMs) may be crucial hubs for the regulation of lipid metabolism, thus contributing to the exacerbation or prevention of fatty liver. We hypothesized that tether proteins...

Cite
Bassot A, Prip-Buus C, Alves A, et al. Loss and gain of function of Grp75 or mitofusin 2 distinctly alter cholesterol metabolism, but all promote triglyceride accumulation in hepatocytes. Biochim Biophys Acta Mol Cell Biol Lipids. 2021;1866(12):159030doi: 10.1016/j.bbalip.2021.159030.
Bassot, A., Prip-Buus, C., Alves, A., Berdeaux, O., Perrier, J., Lenoir, V., Ji-Cao, J., Berger, M. A., Loizon, E., Cabaret, S., Panthu, B., Rieusset, J., & Morio, B. (2021). Loss and gain of function of Grp75 or mitofusin 2 distinctly alter cholesterol metabolism, but all promote triglyceride accumulation in hepatocytes. Biochimica et biophysica acta. Molecular and cell biology of lipids, 1866(12), 159030. https://doi.org/10.1016/j.bbalip.2021.159030
Bassot, Arthur, et al. "Loss and gain of function of Grp75 or mitofusin 2 distinctly alter cholesterol metabolism, but all promote triglyceride accumulation in hepatocytes." Biochimica et biophysica acta. Molecular and cell biology of lipids vol. 1866,12 (2021): 159030. doi: https://doi.org/10.1016/j.bbalip.2021.159030
Bassot A, Prip-Buus C, Alves A, Berdeaux O, Perrier J, Lenoir V, Ji-Cao J, Berger MA, Loizon E, Cabaret S, Panthu B, Rieusset J, Morio B. Loss and gain of function of Grp75 or mitofusin 2 distinctly alter cholesterol metabolism, but all promote triglyceride accumulation in hepatocytes. Biochim Biophys Acta Mol Cell Biol Lipids. 2021 Dec;1866(12):159030. doi: 10.1016/j.bbalip.2021.159030. Epub 2021 Aug 20. PMID: 34419589.
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Clenbuterol exerts antidiabetic activity through metabolic reprogramming of skeletal muscle cells.

Nature communications

Meister J, Bone DBJ, Knudsen JR, Barella LF, Velenosi TJ, Akhmedov D, Lee RJ, Cohen AH, Gavrilova O, Cui Y, Karsenty G, Chen M, Weinstein LS, Kleinert M, Berdeaux R, Jensen TE, Richter EA, Wess J.
PMID: 35013148
Nat Commun. 2022 Jan 10;13(1):22. doi: 10.1038/s41467-021-27540-w.

Activation of the sympathetic nervous system causes pronounced metabolic changes that are mediated by multiple adrenergic receptor subtypes. Systemic treatment with β

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Meister J, Bone DBJ, Knudsen JR, et al. Clenbuterol exerts antidiabetic activity through metabolic reprogramming of skeletal muscle cells. Nat Commun. 2022;13(1):22doi: 10.1038/s41467-021-27540-w.
Meister, J., Bone, D. B. J., Knudsen, J. R., Barella, L. F., Velenosi, T. J., Akhmedov, D., Lee, R. J., Cohen, A. H., Gavrilova, O., Cui, Y., Karsenty, G., Chen, M., Weinstein, L. S., Kleinert, M., Berdeaux, R., Jensen, T. E., Richter, E. A., & Wess, J. (2022). Clenbuterol exerts antidiabetic activity through metabolic reprogramming of skeletal muscle cells. Nature communications, 13(1), 22. https://doi.org/10.1038/s41467-021-27540-w
Meister, Jaroslawna, et al. "Clenbuterol exerts antidiabetic activity through metabolic reprogramming of skeletal muscle cells." Nature communications vol. 13,1 (2022): 22. doi: https://doi.org/10.1038/s41467-021-27540-w
Meister J, Bone DBJ, Knudsen JR, Barella LF, Velenosi TJ, Akhmedov D, Lee RJ, Cohen AH, Gavrilova O, Cui Y, Karsenty G, Chen M, Weinstein LS, Kleinert M, Berdeaux R, Jensen TE, Richter EA, Wess J. Clenbuterol exerts antidiabetic activity through metabolic reprogramming of skeletal muscle cells. Nat Commun. 2022 Jan 10;13(1):22. doi: 10.1038/s41467-021-27540-w. PMID: 35013148.
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Fatty acid composition and conjugated linoleic acid content of different tissues in rats fed individual conjugated linoleic acid isomers given as triacylglycerols small star, filled.

The Journal of nutritional biochemistry

Alasnier C, Berdeaux O, Chardigny JM, Sébédio JL.
PMID: 12088799
J Nutr Biochem. 2002 Jun;13(6):337-345. doi: 10.1016/s0955-2863(02)00176-6.

HEAT TREATMENT OF VEGETABLE OILS GAVE RISE TO FOUR MAIN CONJUGATED LINOLEIC ACID (CLA) ISOMERS : the 9c,11t, 9t,11t, 10t,12c and 10t,12t. The diet of male Wistar rats was supplemented with 150 mg/day either 9c,11t-, 9t,11t-, 10t,12c- or 10t,12t...

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Alasnier C, Berdeaux O, Chardigny JM, et al. Fatty acid composition and conjugated linoleic acid content of different tissues in rats fed individual conjugated linoleic acid isomers given as triacylglycerols small star, filled. J Nutr Biochem. 2002;13(6):337-345doi: 10.1016/s0955-2863(02)00176-6.
Alasnier, C., Berdeaux, O., Chardigny, J. M., & Sébédio, J. L. (2002). Fatty acid composition and conjugated linoleic acid content of different tissues in rats fed individual conjugated linoleic acid isomers given as triacylglycerols small star, filled. The Journal of nutritional biochemistry, 13(6), 337-345. https://doi.org/10.1016/s0955-2863(02)00176-6
Alasnier, Corinne, et al. "Fatty acid composition and conjugated linoleic acid content of different tissues in rats fed individual conjugated linoleic acid isomers given as triacylglycerols small star, filled." The Journal of nutritional biochemistry vol. 13,6 (2002): 337-345. doi: https://doi.org/10.1016/s0955-2863(02)00176-6
Alasnier C, Berdeaux O, Chardigny JM, Sébédio JL. Fatty acid composition and conjugated linoleic acid content of different tissues in rats fed individual conjugated linoleic acid isomers given as triacylglycerols small star, filled. J Nutr Biochem. 2002 Jun;13(6):337-345. doi: 10.1016/s0955-2863(02)00176-6. PMID: 12088799.
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Red blood cell β-adrenergic receptors contribute to diet-induced energy expenditure by increasing O2 supply.

JCI insight

Kim ER, Fan S, Akhmedov D, Sun K, Lim H, O'Brien W, Xu Y, Mangieri LR, Zhu Y, Lee CC, Chung Y, Xia Y, Xu Y, Li F, Sun K, Berdeaux R, Tong Q.
PMID: 28724789
JCI Insight. 2017 Jul 20;2(14). doi: 10.1172/jci.insight.93367. eCollection 2017 Jul 20.

Diet-induced obesity (DIO) represents the major cause for the current obesity epidemic, but the mechanism underlying DIO is unclear. β-Adrenergic receptors (β-ARs) play a major role in sympathetic nervous system-mediated (SNS-mediated) diet-induced energy expenditure (EE). Rbc express abundant β-ARs;...

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Kim ER, Fan S, Akhmedov D, et al. Red blood cell β-adrenergic receptors contribute to diet-induced energy expenditure by increasing O2 supply. JCI Insight. 2017;2(14)doi: 10.1172/jci.insight.93367.
Kim, E. R., Fan, S., Akhmedov, D., Sun, K., Lim, H., O'Brien, W., Xu, Y., Mangieri, L. R., Zhu, Y., Lee, C. C., Chung, Y., Xia, Y., Xu, Y., Li, F., Sun, K., Berdeaux, R., & Tong, Q. (2017). Red blood cell β-adrenergic receptors contribute to diet-induced energy expenditure by increasing O2 supply. JCI insight, 2(14), . https://doi.org/10.1172/jci.insight.93367
Kim, Eun Ran, et al. "Red blood cell β-adrenergic receptors contribute to diet-induced energy expenditure by increasing O2 supply." JCI insight vol. 2,14 (2017). doi: https://doi.org/10.1172/jci.insight.93367
Kim ER, Fan S, Akhmedov D, Sun K, Lim H, O'Brien W, Xu Y, Mangieri LR, Zhu Y, Lee CC, Chung Y, Xia Y, Xu Y, Li F, Sun K, Berdeaux R, Tong Q. Red blood cell β-adrenergic receptors contribute to diet-induced energy expenditure by increasing O2 supply. JCI Insight. 2017 Jul 20;2(14). doi: 10.1172/jci.insight.93367. eCollection 2017 Jul 20. PMID: 28724789; PMCID: PMC5518553.
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Hypothermic total liquid ventilation after experimental aspiration-associated acute respiratory distress syndrome.

Annals of intensive care

Rambaud J, Lidouren F, Sage M, Kohlhauer M, Nadeau M, Fortin-Pellerin É, Micheau P, Zilberstein L, Mongardon N, Ricard JD, Terada M, Bruneval P, Berdeaux A, Ghaleh B, Walti H, Tissier R.
PMID: 29721820
Ann Intensive Care. 2018 May 02;8(1):57. doi: 10.1186/s13613-018-0404-8.

BACKGROUND: Ultrafast cooling by total liquid ventilation (TLV) provides potent cardio- and neuroprotection after experimental cardiac arrest. However, this was evaluated in animals with no initial lung injury, whereas out-of-hospital cardiac arrest is frequently associated with early-onset pneumonia, which...

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Rambaud J, Lidouren F, Sage M, et al. Hypothermic total liquid ventilation after experimental aspiration-associated acute respiratory distress syndrome. Ann Intensive Care. 2018;8(1):57doi: 10.1186/s13613-018-0404-8.
Rambaud, J., Lidouren, F., Sage, M., Kohlhauer, M., Nadeau, M., Fortin-Pellerin, �. �., Micheau, P., Zilberstein, L., Mongardon, N., Ricard, J. D., Terada, M., Bruneval, P., Berdeaux, A., Ghaleh, B., Walti, H., & Tissier, R. (2018). Hypothermic total liquid ventilation after experimental aspiration-associated acute respiratory distress syndrome. Annals of intensive care, 8(1), 57. https://doi.org/10.1186/s13613-018-0404-8
Rambaud, Jérôme, et al. "Hypothermic total liquid ventilation after experimental aspiration-associated acute respiratory distress syndrome." Annals of intensive care vol. 8,1 (2018): 57. doi: https://doi.org/10.1186/s13613-018-0404-8
Rambaud J, Lidouren F, Sage M, Kohlhauer M, Nadeau M, Fortin-Pellerin É, Micheau P, Zilberstein L, Mongardon N, Ricard JD, Terada M, Bruneval P, Berdeaux A, Ghaleh B, Walti H, Tissier R. Hypothermic total liquid ventilation after experimental aspiration-associated acute respiratory distress syndrome. Ann Intensive Care. 2018 May 02;8(1):57. doi: 10.1186/s13613-018-0404-8. PMID: 29721820; PMCID: PMC5931951.
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