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Translating medical school social missions to student experiences.

Medical education

Ellaway RH, Van Roy K, Preston R, Greenhill J, Clithero A, Elsanousi S, Richards J, Labarda C, Graves L, Mammen M, Assayed AA, Willems S.
PMID: 28949033
Med Educ. 2018 Feb;52(2):171-181. doi: 10.1111/medu.13417. Epub 2017 Sep 26.

CONTEXT: There is a growing focus on the social missions of medical schools as a way of expressing an institutional commitment to service, responsibility and accountability. However, there has been little exploration of how a social mission translates to...

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Ellaway RH, Van Roy K, Preston R, et al. Translating medical school social missions to student experiences. Med Educ. 2017;52(2):171-181doi: 10.1111/medu.13417.
Ellaway, R. H., Van Roy, K., Preston, R., Greenhill, J., Clithero, A., Elsanousi, S., Richards, J., Labarda, C., Graves, L., Mammen, M., Assayed, A. A., & Willems, S. (2018). Translating medical school social missions to student experiences. Medical education, 52(2), 171-181. https://doi.org/10.1111/medu.13417
Ellaway, Rachel H, et al. "Translating medical school social missions to student experiences." Medical education vol. 52,2 (2018): 171-181. doi: https://doi.org/10.1111/medu.13417
Ellaway RH, Van Roy K, Preston R, Greenhill J, Clithero A, Elsanousi S, Richards J, Labarda C, Graves L, Mammen M, Assayed AA, Willems S. Translating medical school social missions to student experiences. Med Educ. 2018 Feb;52(2):171-181. doi: 10.1111/medu.13417. Epub 2017 Sep 26. PMID: 28949033.
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TALEN-mediated apc mutation in Xenopus tropicalis phenocopies familial adenomatous polyposis.

Oncoscience

Van Nieuwenhuysen T, Naert T, Tran HT, Van Imschoot G, Geurs S, Sanders E, Creytens D, Van Roy F, Vleminckx K.
PMID: 26097888
Oncoscience. 2015 May 19;2(5):555-66. doi: 10.18632/oncoscience.166. eCollection 2015.

Truncating mutations in the tumor suppressor gene adenomatous polyposis coli (APC) are the initiating step in the vast majority of sporadic colorectal cancers, and they underlie familial adenomatous polyposis (FAP) syndromes. Modeling of APC- driven tumor formation in the...

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Van Nieuwenhuysen T, Naert T, Tran HT, et al. TALEN-mediated apc mutation in Xenopus tropicalis phenocopies familial adenomatous polyposis. Oncoscience. 2015;2(5):555-66doi: 10.18632/oncoscience.166.
Van Nieuwenhuysen, T., Naert, T., Tran, H. T., Van Imschoot, G., Geurs, S., Sanders, E., Creytens, D., Van Roy, F., & Vleminckx, K. (2015). TALEN-mediated apc mutation in Xenopus tropicalis phenocopies familial adenomatous polyposis. Oncoscience, 2(5), 555-66. https://doi.org/10.18632/oncoscience.166
Van Nieuwenhuysen, Tom, et al. "TALEN-mediated apc mutation in Xenopus tropicalis phenocopies familial adenomatous polyposis." Oncoscience vol. 2,5 (2015): 555-66. doi: https://doi.org/10.18632/oncoscience.166
Van Nieuwenhuysen T, Naert T, Tran HT, Van Imschoot G, Geurs S, Sanders E, Creytens D, Van Roy F, Vleminckx K. TALEN-mediated apc mutation in Xenopus tropicalis phenocopies familial adenomatous polyposis. Oncoscience. 2015 May 19;2(5):555-66. doi: 10.18632/oncoscience.166. eCollection 2015. PMID: 26097888; PMCID: PMC4468341.
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Tumour invasion: effects of cell adhesion and motility.

Trends in cell biology

Van Roy F, Mareel M.
PMID: 14731985
Trends Cell Biol. 1992 Jun;2(6):163-9. doi: 10.1016/0962-8924(92)90035-l.

Metastasis is the major cause of failure in cancer therapy. Recent studies of the molecular cell biology of the metastatic process have provided new insights into the mechanisms of cell-cell adhesion, cell-substrate adhesion and cell motility that underly invasion...

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Van Roy F, Mareel M. Tumour invasion: effects of cell adhesion and motility. Trends Cell Biol. 1992;2(6):163-9doi: 10.1016/0962-8924(92)90035-l.
Van Roy, F., & Mareel, M. (1992). Tumour invasion: effects of cell adhesion and motility. Trends in cell biology, 2(6), 163-9. https://doi.org/10.1016/0962-8924(92)90035-l
Van Roy, F, and Mareel, M. "Tumour invasion: effects of cell adhesion and motility." Trends in cell biology vol. 2,6 (1992): 163-9. doi: https://doi.org/10.1016/0962-8924(92)90035-l
Van Roy F, Mareel M. Tumour invasion: effects of cell adhesion and motility. Trends Cell Biol. 1992 Jun;2(6):163-9. doi: 10.1016/0962-8924(92)90035-l. PMID: 14731985.
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Spatial structure of an individual Mn acceptor in GaAs.

Physical review letters

Yakunin AM, Silov AY, Koenraad PM, Wolter JH, Van Roy W, De Boeck J, Tang JM, Flatté ME.
PMID: 15245308
Phys Rev Lett. 2004 May 28;92(21):216806. doi: 10.1103/PhysRevLett.92.216806. Epub 2004 May 28.

The wave function of a hole bound to an individual Mn acceptor in GaAs is spatially mapped by scanning tunneling microscopy at room temperature and an anisotropic, crosslike shape is observed. The spatial structure is compared with that from...

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Yakunin AM, Silov AY, Koenraad PM, et al. Spatial structure of an individual Mn acceptor in GaAs. Phys Rev Lett. 2004;92(21):216806doi: 10.1103/PhysRevLett.92.216806.
Yakunin, A. M., Silov, A. Y., Koenraad, P. M., Wolter, J. H., Van Roy, W., De Boeck, J., Tang, J. M., & Flatté, M. E. (2004). Spatial structure of an individual Mn acceptor in GaAs. Physical review letters, 92(21), 216806. https://doi.org/10.1103/PhysRevLett.92.216806
Yakunin, A M, et al. "Spatial structure of an individual Mn acceptor in GaAs." Physical review letters vol. 92,21 (2004): 216806. doi: https://doi.org/10.1103/PhysRevLett.92.216806
Yakunin AM, Silov AY, Koenraad PM, Wolter JH, Van Roy W, De Boeck J, Tang JM, Flatté ME. Spatial structure of an individual Mn acceptor in GaAs. Phys Rev Lett. 2004 May 28;92(21):216806. doi: 10.1103/PhysRevLett.92.216806. Epub 2004 May 28. PMID: 15245308.
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The emerging molecular pathogenesis of neuroblastoma: implications for improved risk assessment and targeted therapy.

Genome medicine

Van Roy N, De Preter K, Hoebeeck J, Van Maerken T, Pattyn F, Mestdagh P, Vermeulen J, Vandesompele J, Speleman F.
PMID: 19638189
Genome Med. 2009 Jul 27;1(7):74. doi: 10.1186/gm74.

Neuroblastoma is one of the most common solid tumors of childhood, arising from immature sympathetic nervous system cells. The clinical course of patients with neuroblastoma is highly variable, ranging from spontaneous regression to widespread metastatic disease. Although the outcome...

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Van Roy N, De Preter K, Hoebeeck J, et al. The emerging molecular pathogenesis of neuroblastoma: implications for improved risk assessment and targeted therapy. Genome Med. 2009;1(7):74doi: 10.1186/gm74.
Van Roy, N., De Preter, K., Hoebeeck, J., Van Maerken, T., Pattyn, F., Mestdagh, P., Vermeulen, J., Vandesompele, J., & Speleman, F. (2009). The emerging molecular pathogenesis of neuroblastoma: implications for improved risk assessment and targeted therapy. Genome medicine, 1(7), 74. https://doi.org/10.1186/gm74
Van Roy, Nadine, et al. "The emerging molecular pathogenesis of neuroblastoma: implications for improved risk assessment and targeted therapy." Genome medicine vol. 1,7 (2009): 74. doi: https://doi.org/10.1186/gm74
Van Roy N, De Preter K, Hoebeeck J, Van Maerken T, Pattyn F, Mestdagh P, Vermeulen J, Vandesompele J, Speleman F. The emerging molecular pathogenesis of neuroblastoma: implications for improved risk assessment and targeted therapy. Genome Med. 2009 Jul 27;1(7):74. doi: 10.1186/gm74. PMID: 19638189; PMCID: PMC2717400.
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αT-catenin in restricted brain cell types and its potential connection to autism.

Journal of molecular psychiatry

Folmsbee SS, Wilcox DR, Tyberghein K, De Bleser P, Tourtellotte WG, van Hengel J, van Roy F, Gottardi CJ.
PMID: 27330745
J Mol Psychiatry. 2016 Jun 21;4:2. doi: 10.1186/s40303-016-0017-9. eCollection 2016.

BACKGROUND: Recent genetic association studies have linked the cadherin-based adherens junction protein alpha-T-catenin (αT-cat, CTNNA3) with the development of autism. Where αT-cat is expressed in the brain, and how its loss could contribute to this disorder, are entirely unknown.METHODS:...

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Folmsbee SS, Wilcox DR, Tyberghein K, et al. αT-catenin in restricted brain cell types and its potential connection to autism. J Mol Psychiatry. 2016;4:2doi: 10.1186/s40303-016-0017-9.
Folmsbee, S. S., Wilcox, D. R., Tyberghein, K., De Bleser, P., Tourtellotte, W. G., van Hengel, J., van Roy, F., & Gottardi, C. J. (2016). αT-catenin in restricted brain cell types and its potential connection to autism. Journal of molecular psychiatry, 42. https://doi.org/10.1186/s40303-016-0017-9
Folmsbee, Stephen Sai, et al. "αT-catenin in restricted brain cell types and its potential connection to autism." Journal of molecular psychiatry vol. 4 (2016): 2. doi: https://doi.org/10.1186/s40303-016-0017-9
Folmsbee SS, Wilcox DR, Tyberghein K, De Bleser P, Tourtellotte WG, van Hengel J, van Roy F, Gottardi CJ. αT-catenin in restricted brain cell types and its potential connection to autism. J Mol Psychiatry. 2016 Jun 21;4:2. doi: 10.1186/s40303-016-0017-9. eCollection 2016. PMID: 27330745; PMCID: PMC4915096.
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Network of networks: preliminary lessons from the Antwerp Port Authority on crisis management and network governance to deal with the COVID-19 pandemic.

Public administration review

van den Oord S, Vanlaer N, Marynissen H, Brugghemans B, Van Roey J, Albers S, Cambré B, Kenis P.
PMID: 32836461
Public Adm Rev. 2020 Jun 02; doi: 10.1111/puar.13256. Epub 2020 Jun 02.

In this article we describe and illustrate what we call a network of networks perspective and map the development of a Lead network of the Antwerp Port Authority that governs various organizations and networks in the port community before...

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van den Oord S, Vanlaer N, Marynissen H, et al. Network of networks: preliminary lessons from the Antwerp Port Authority on crisis management and network governance to deal with the COVID-19 pandemic. Public Adm Rev. 2020;doi: 10.1111/puar.13256.
van den Oord, S., Vanlaer, N., Marynissen, H., Brugghemans, B., Van Roey, J., Albers, S., Cambré, B., & Kenis, P. (2020). Network of networks: preliminary lessons from the Antwerp Port Authority on crisis management and network governance to deal with the COVID-19 pandemic. Public administration review, . https://doi.org/10.1111/puar.13256
van den Oord, Steven, et al. "Network of networks: preliminary lessons from the Antwerp Port Authority on crisis management and network governance to deal with the COVID-19 pandemic." Public administration review vol. (2020). doi: https://doi.org/10.1111/puar.13256
van den Oord S, Vanlaer N, Marynissen H, Brugghemans B, Van Roey J, Albers S, Cambré B, Kenis P. Network of networks: preliminary lessons from the Antwerp Port Authority on crisis management and network governance to deal with the COVID-19 pandemic. Public Adm Rev. 2020 Jun 02; doi: 10.1111/puar.13256. Epub 2020 Jun 02. PMID: 32836461; PMCID: PMC7300886.
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Plakophilin3 loss leads to increased adenoma formation and rectal prolapse in APC.

Biochemical and biophysical research communications

Raghavan R, Koyande N, Beher R, Chetlangia N, Ramadwar M, Pawade S, Thorat R, van Hengel J, Sklyarova T, van Roy F, Dalal SN.
PMID: 34823217
Biochem Biophys Res Commun. 2021 Nov 19;586:14-19. doi: 10.1016/j.bbrc.2021.11.071. Epub 2021 Nov 19.

Plakophilin3 (PKP3) loss leads to tumor progression and metastasis of colon cancer cells. The goal of this report was to determine if PKP3 loss led to increased disease progression in mice. We generated a colonocyte-specific knockout of PKP3 in...

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Raghavan R, Koyande N, Beher R, et al. Plakophilin3 loss leads to increased adenoma formation and rectal prolapse in APC. Biochem Biophys Res Commun. 2021;586:14-19doi: 10.1016/j.bbrc.2021.11.071.
Raghavan, R., Koyande, N., Beher, R., Chetlangia, N., Ramadwar, M., Pawade, S., Thorat, R., van Hengel, J., Sklyarova, T., van Roy, F., & Dalal, S. N. (2021). Plakophilin3 loss leads to increased adenoma formation and rectal prolapse in APC. Biochemical and biophysical research communications, 58614-19. https://doi.org/10.1016/j.bbrc.2021.11.071
Raghavan, Rahul, et al. "Plakophilin3 loss leads to increased adenoma formation and rectal prolapse in APC." Biochemical and biophysical research communications vol. 586 (2021): 14-19. doi: https://doi.org/10.1016/j.bbrc.2021.11.071
Raghavan R, Koyande N, Beher R, Chetlangia N, Ramadwar M, Pawade S, Thorat R, van Hengel J, Sklyarova T, van Roy F, Dalal SN. Plakophilin3 loss leads to increased adenoma formation and rectal prolapse in APC. Biochem Biophys Res Commun. 2021 Nov 19;586:14-19. doi: 10.1016/j.bbrc.2021.11.071. Epub 2021 Nov 19. PMID: 34823217.
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Extrauterine Mesonephric-like Neoplasms: Expanding the Morphologic Spectrum.

The American journal of surgical pathology

Deolet E, Arora I, Van Dorpe J, Van der Meulen J, Desai S, Van Roy N, Kaur B, Van de Vijver K, McCluggage WG.
PMID: 34183523
Am J Surg Pathol. 2022 Jan 01;46(1):124-133. doi: 10.1097/PAS.0000000000001766.

Mesonephric-like adenocarcinomas (MLA) are rare neoplasms arising in the uterine corpus and ovary which have been added to the recent 2020 World Health Organization Classification of Female Genital Tumors. They have similar morphology and immunophenotype and exhibit molecular aberrations...

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Deolet E, Arora I, Van Dorpe J, et al. Extrauterine Mesonephric-like Neoplasms: Expanding the Morphologic Spectrum. Am J Surg Pathol. 2022;46(1):124-133doi: 10.1097/PAS.0000000000001766.
Deolet, E., Arora, I., Van Dorpe, J., Van der Meulen, J., Desai, S., Van Roy, N., Kaur, B., Van de Vijver, K., & McCluggage, W. G. (2022). Extrauterine Mesonephric-like Neoplasms: Expanding the Morphologic Spectrum. The American journal of surgical pathology, 46(1), 124-133. https://doi.org/10.1097/PAS.0000000000001766
Deolet, Ellen, et al. "Extrauterine Mesonephric-like Neoplasms: Expanding the Morphologic Spectrum." The American journal of surgical pathology vol. 46,1 (2022): 124-133. doi: https://doi.org/10.1097/PAS.0000000000001766
Deolet E, Arora I, Van Dorpe J, Van der Meulen J, Desai S, Van Roy N, Kaur B, Van de Vijver K, McCluggage WG. Extrauterine Mesonephric-like Neoplasms: Expanding the Morphologic Spectrum. Am J Surg Pathol. 2022 Jan 01;46(1):124-133. doi: 10.1097/PAS.0000000000001766. PMID: 34183523.
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Selectivity in the Addition of Electron Deficient Radicals to the .

European journal of organic chemistry

Li Y, Vaz RJ, Olson SH, Munson M, Paras NA, Conrad J.
PMID: 33692651
European J Org Chem. 2020 Sep 30;2020(36):5828-5832. doi: 10.1002/ejoc.201901784. Epub 2020 Aug 18.

The addition of electron deficient radicals to the

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Li Y, Vaz RJ, Olson SH, et al. Selectivity in the Addition of Electron Deficient Radicals to the . European J Org Chem. 2020;2020(36):5828-5832doi: 10.1002/ejoc.201901784.
Li, Y., Vaz, R. J., Olson, S. H., Munson, M., Paras, N. A., & Conrad, J. (2020). Selectivity in the Addition of Electron Deficient Radicals to the . European journal of organic chemistry, 2020(36), 5828-5832. https://doi.org/10.1002/ejoc.201901784
Li, Yi, et al. "Selectivity in the Addition of Electron Deficient Radicals to the ." European journal of organic chemistry vol. 2020,36 (2020): 5828-5832. doi: https://doi.org/10.1002/ejoc.201901784
Li Y, Vaz RJ, Olson SH, Munson M, Paras NA, Conrad J. Selectivity in the Addition of Electron Deficient Radicals to the . European J Org Chem. 2020 Sep 30;2020(36):5828-5832. doi: 10.1002/ejoc.201901784. Epub 2020 Aug 18. PMID: 33692651; PMCID: PMC7939109.
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From DNA Copy Number Gains and Tumor Dependencies to Novel Therapeutic Targets for High-Risk Neuroblastoma.

Journal of personalized medicine

Decaesteker B, Durinck K, Van Roy N, De Wilde B, Van Neste C, Van Haver S, Roberts S, De Preter K, Vermeirssen V, Speleman F.
PMID: 34945759
J Pers Med. 2021 Dec 03;11(12). doi: 10.3390/jpm11121286.

Neuroblastoma is a pediatric tumor arising from the sympatho-adrenal lineage and a worldwide leading cause of childhood cancer-related deaths. About half of high-risk patients die from the disease while survivors suffer from multiple therapy-related side-effects. While neuroblastomas present with...

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Decaesteker B, Durinck K, Van Roy N, et al. From DNA Copy Number Gains and Tumor Dependencies to Novel Therapeutic Targets for High-Risk Neuroblastoma. J Pers Med. 2021;11(12)doi: 10.3390/jpm11121286.
Decaesteker, B., Durinck, K., Van Roy, N., De Wilde, B., Van Neste, C., Van Haver, S., Roberts, S., De Preter, K., Vermeirssen, V., & Speleman, F. (2021). From DNA Copy Number Gains and Tumor Dependencies to Novel Therapeutic Targets for High-Risk Neuroblastoma. Journal of personalized medicine, 11(12), . https://doi.org/10.3390/jpm11121286
Decaesteker, Bieke, et al. "From DNA Copy Number Gains and Tumor Dependencies to Novel Therapeutic Targets for High-Risk Neuroblastoma." Journal of personalized medicine vol. 11,12 (2021). doi: https://doi.org/10.3390/jpm11121286
Decaesteker B, Durinck K, Van Roy N, De Wilde B, Van Neste C, Van Haver S, Roberts S, De Preter K, Vermeirssen V, Speleman F. From DNA Copy Number Gains and Tumor Dependencies to Novel Therapeutic Targets for High-Risk Neuroblastoma. J Pers Med. 2021 Dec 03;11(12). doi: 10.3390/jpm11121286. PMID: 34945759; PMCID: PMC8707517.
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Association of unbalanced translocation der(1;7) with germline GATA2 mutations.

Blood

Kozyra EJ, Göhring G, Hickstein DD, Calvo KR, DiNardo CD, Dworzak M, de Haas V, Starý J, Hasle H, Shimamura A, Fleming MD, Inaba H, Lewis S, Hsu AP, Holland SM, Arnold DE, Mecucci C, Keel SB, Bertuch AA, Tawana K, Barzilai S, Hirabayashi S, Onozawa M, Lei S, Alaiz H, Andrikovics H, Betts D, Beverloo BH, Buechner J, Čermák M, Cervera J, Haus O, Jahnukainen K, Manola KN, Nebral K, Pasquali F, Tchinda J, Turkiewicz D, Van Roy N, Zemanova Z, Pastor VB, Strahm B, Noellke P, Niemeyer CM, Schlegelberger B, Yoshimi A, Wlodarski MW.
PMID: 34469508
Blood. 2021 Dec 09;138(23):2441-2445. doi: 10.1182/blood.2021012781.

No abstract available.

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Kozyra EJ, Göhring G, Hickstein DD, et al. Association of unbalanced translocation der(1;7) with germline GATA2 mutations. Blood. 2021;138(23):2441-2445doi: 10.1182/blood.2021012781.
Kozyra, E. J., Göhring, G., Hickstein, D. D., Calvo, K. R., DiNardo, C. D., Dworzak, M., de Haas, V., Starý, J., Hasle, H., Shimamura, A., Fleming, M. D., Inaba, H., Lewis, S., Hsu, A. P., Holland, S. M., Arnold, D. E., Mecucci, C., Keel, S. B., Bertuch, A. A., Tawana, K., Barzilai, S., Hirabayashi, S., Onozawa, M., Lei, S., Alaiz, H., Andrikovics, H., Betts, D., Beverloo, B. H., Buechner, J., Čermák, M., Cervera, J., Haus, O., Jahnukainen, K., Manola, K. N., Nebral, K., Pasquali, F., Tchinda, J., Turkiewicz, D., Van Roy, N., Zemanova, Z., Pastor, V. B., Strahm, B., Noellke, P., Niemeyer, C. M., Schlegelberger, B., Yoshimi, A., & Wlodarski, M. W. (2021). Association of unbalanced translocation der(1;7) with germline GATA2 mutations. Blood, 138(23), 2441-2445. https://doi.org/10.1182/blood.2021012781
Kozyra, Emilia J, et al. "Association of unbalanced translocation der(1;7) with germline GATA2 mutations." Blood vol. 138,23 (2021): 2441-2445. doi: https://doi.org/10.1182/blood.2021012781
Kozyra EJ, Göhring G, Hickstein DD, Calvo KR, DiNardo CD, Dworzak M, de Haas V, Starý J, Hasle H, Shimamura A, Fleming MD, Inaba H, Lewis S, Hsu AP, Holland SM, Arnold DE, Mecucci C, Keel SB, Bertuch AA, Tawana K, Barzilai S, Hirabayashi S, Onozawa M, Lei S, Alaiz H, Andrikovics H, Betts D, Beverloo BH, Buechner J, Čermák M, Cervera J, Haus O, Jahnukainen K, Manola KN, Nebral K, Pasquali F, Tchinda J, Turkiewicz D, Van Roy N, Zemanova Z, Pastor VB, Strahm B, Noellke P, Niemeyer CM, Schlegelberger B, Yoshimi A, Wlodarski MW. Association of unbalanced translocation der(1;7) with germline GATA2 mutations. Blood. 2021 Dec 09;138(23):2441-2445. doi: 10.1182/blood.2021012781. PMID: 34469508; PMCID: PMC8662074.
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