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Showing 1 to 12 of 83 entries
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The impact of herbivores on nitrogen mineralization rate: consequences for salt-marsh succession.

Oecologia

van Wijnen HJ, van der Wal R, Bakker JP.
PMID: 28307698
Oecologia. 1999 Feb;118(2):225-231. doi: 10.1007/s004420050722.

Soil net N-mineralization rate was measured along a successional gradient in salt-marsh sites that were grazed by vertebrate herbivores, and in 5-year-old exclosures from which the animals were excluded. Mineralization rate was significantly higher at ungrazed than at grazed...

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van Wijnen HJ, van der Wal R, Bakker JP. The impact of herbivores on nitrogen mineralization rate: consequences for salt-marsh succession. Oecologia. 1999;118(2):225-231doi: 10.1007/s004420050722.
van Wijnen, H. J., van der Wal, R., & Bakker, J. P. (1999). The impact of herbivores on nitrogen mineralization rate: consequences for salt-marsh succession. Oecologia, 118(2), 225-231. https://doi.org/10.1007/s004420050722
van Wijnen, Harm J, et al. "The impact of herbivores on nitrogen mineralization rate: consequences for salt-marsh succession." Oecologia vol. 118,2 (1999): 225-231. doi: https://doi.org/10.1007/s004420050722
van Wijnen HJ, van der Wal R, Bakker JP. The impact of herbivores on nitrogen mineralization rate: consequences for salt-marsh succession. Oecologia. 1999 Feb;118(2):225-231. doi: 10.1007/s004420050722. PMID: 28307698.
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hsa-mir-30c promotes the invasive phenotype of metastatic breast cancer cells by targeting NOV/CCN3.

Cancer cell international

Dobson JR, Taipaleenmäki H, Hu YJ, Hong D, van Wijnen AJ, Stein JL, Stein GS, Lian JB, Pratap J.
PMID: 25120384
Cancer Cell Int. 2014 Aug 02;14:73. doi: 10.1186/s12935-014-0073-0. eCollection 2014.

BACKGROUND: For treatment and prevention of metastatic disease, one of the premier challenges is the identification of pathways and proteins to target for clinical intervention. Micro RNAs (miRNAs) are short, non-coding RNAs, which regulate cellular activities by either mRNA...

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Dobson JR, Taipaleenmäki H, Hu YJ, et al. hsa-mir-30c promotes the invasive phenotype of metastatic breast cancer cells by targeting NOV/CCN3. Cancer Cell Int. 2014;14:73doi: 10.1186/s12935-014-0073-0.
Dobson, J. R., Taipaleenmäki, H., Hu, Y. J., Hong, D., van Wijnen, A. J., Stein, J. L., Stein, G. S., Lian, J. B., & Pratap, J. (2014). hsa-mir-30c promotes the invasive phenotype of metastatic breast cancer cells by targeting NOV/CCN3. Cancer cell international, 1473. https://doi.org/10.1186/s12935-014-0073-0
Dobson, Jason R, et al. "hsa-mir-30c promotes the invasive phenotype of metastatic breast cancer cells by targeting NOV/CCN3." Cancer cell international vol. 14 (2014): 73. doi: https://doi.org/10.1186/s12935-014-0073-0
Dobson JR, Taipaleenmäki H, Hu YJ, Hong D, van Wijnen AJ, Stein JL, Stein GS, Lian JB, Pratap J. hsa-mir-30c promotes the invasive phenotype of metastatic breast cancer cells by targeting NOV/CCN3. Cancer Cell Int. 2014 Aug 02;14:73. doi: 10.1186/s12935-014-0073-0. eCollection 2014. PMID: 25120384; PMCID: PMC4129468.
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The Role of RUNX2 in Osteosarcoma Oncogenesis.

Sarcoma

Martin JW, Zielenska M, Stein GS, van Wijnen AJ, Squire JA.
PMID: 21197465
Sarcoma. 2011;2011:282745. doi: 10.1155/2011/282745. Epub 2010 Dec 09.

Osteosarcoma is an aggressive but ill-understood cancer of bone that predominantly affects adolescents. Its rarity and biological heterogeneity have limited studies of its molecular basis. In recent years, an important role has emerged for the RUNX2 "platform protein" in...

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Martin JW, Zielenska M, Stein GS, et al. The Role of RUNX2 in Osteosarcoma Oncogenesis. Sarcoma. 2010;2011:282745doi: 10.1155/2011/282745.
Martin, J. W., Zielenska, M., Stein, G. S., van Wijnen, A. J., & Squire, J. A. (2011). The Role of RUNX2 in Osteosarcoma Oncogenesis. Sarcoma, 2011282745. https://doi.org/10.1155/2011/282745
Martin, J W, et al. "The Role of RUNX2 in Osteosarcoma Oncogenesis." Sarcoma vol. 2011 (2011): 282745. doi: https://doi.org/10.1155/2011/282745
Martin JW, Zielenska M, Stein GS, van Wijnen AJ, Squire JA. The Role of RUNX2 in Osteosarcoma Oncogenesis. Sarcoma. 2011;2011:282745. doi: 10.1155/2011/282745. Epub 2010 Dec 09. PMID: 21197465; PMCID: PMC3005824.
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Molecular Validation of Chondrogenic Differentiation and Hypoxia Responsiveness of Platelet-Lysate Expanded Adipose Tissue-Derived Human Mesenchymal Stromal Cells.

Cartilage

Galeano-Garces C, Camilleri ET, Riester SM, Dudakovic A, Larson DR, Qu W, Smith J, Dietz AB, Im HJ, Krych AJ, Larson AN, Karperien M, van Wijnen AJ.
PMID: 28618870
Cartilage. 2017 Jul;8(3):283-299. doi: 10.1177/1947603516659344. Epub 2016 Jul 21.

OBJECTIVE: To determine the optimal environmental conditions for chondrogenic differentiation of human adipose tissue-derived mesenchymal stromal/stem cells (AMSCs). In this investigation we specifically investigate the role of oxygen tension and 3-dimensional (3D) culture systems.DESIGN: Both AMSCs and primary human...

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Galeano-Garces C, Camilleri ET, Riester SM, et al. Molecular Validation of Chondrogenic Differentiation and Hypoxia Responsiveness of Platelet-Lysate Expanded Adipose Tissue-Derived Human Mesenchymal Stromal Cells. Cartilage. 2016;8(3):283-299doi: 10.1177/1947603516659344.
Galeano-Garces, C., Camilleri, E. T., Riester, S. M., Dudakovic, A., Larson, D. R., Qu, W., Smith, J., Dietz, A. B., Im, H. J., Krych, A. J., Larson, A. N., Karperien, M., & van Wijnen, A. J. (2017). Molecular Validation of Chondrogenic Differentiation and Hypoxia Responsiveness of Platelet-Lysate Expanded Adipose Tissue-Derived Human Mesenchymal Stromal Cells. Cartilage, 8(3), 283-299. https://doi.org/10.1177/1947603516659344
Galeano-Garces, Catalina, et al. "Molecular Validation of Chondrogenic Differentiation and Hypoxia Responsiveness of Platelet-Lysate Expanded Adipose Tissue-Derived Human Mesenchymal Stromal Cells." Cartilage vol. 8,3 (2017): 283-299. doi: https://doi.org/10.1177/1947603516659344
Galeano-Garces C, Camilleri ET, Riester SM, Dudakovic A, Larson DR, Qu W, Smith J, Dietz AB, Im HJ, Krych AJ, Larson AN, Karperien M, van Wijnen AJ. Molecular Validation of Chondrogenic Differentiation and Hypoxia Responsiveness of Platelet-Lysate Expanded Adipose Tissue-Derived Human Mesenchymal Stromal Cells. Cartilage. 2017 Jul;8(3):283-299. doi: 10.1177/1947603516659344. Epub 2016 Jul 21. PMID: 28618870; PMCID: PMC5625857.
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Modelling global river export of microplastics to the marine environment: Sources and future trends.

The Science of the total environment

van Wijnen J, Ragas AMJ, Kroeze C.
PMID: 30991329
Sci Total Environ. 2019 Jul 10;673:392-401. doi: 10.1016/j.scitotenv.2019.04.078. Epub 2019 Apr 08.

Microplastics, transported by rivers to oceans, are triggering environmental concern. This study aims to better understand river export of microplastics from land to sea. We developed the Global Riverine Export of Microplastics into Seas (GREMiS) model, a global, spatially...

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van Wijnen J, Ragas AMJ, Kroeze C. Modelling global river export of microplastics to the marine environment: Sources and future trends. Sci Total Environ. 2019;673:392-401doi: 10.1016/j.scitotenv.2019.04.078.
van Wijnen, J., Ragas, A. M. J., & Kroeze, C. (2019). Modelling global river export of microplastics to the marine environment: Sources and future trends. The Science of the total environment, 673392-401. https://doi.org/10.1016/j.scitotenv.2019.04.078
van Wijnen, Jikke, et al. "Modelling global river export of microplastics to the marine environment: Sources and future trends." The Science of the total environment vol. 673 (2019): 392-401. doi: https://doi.org/10.1016/j.scitotenv.2019.04.078
van Wijnen J, Ragas AMJ, Kroeze C. Modelling global river export of microplastics to the marine environment: Sources and future trends. Sci Total Environ. 2019 Jul 10;673:392-401. doi: 10.1016/j.scitotenv.2019.04.078. Epub 2019 Apr 08. PMID: 30991329.
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Multimodal Quantitative MRI Reveals No Evidence for Tissue Pathology in Idiopathic Cervical Dystonia.

Frontiers in neurology

Gracien RM, Petrov F, Hok P, van Wijnen A, Maiworm M, Seiler A, Deichmann R, Baudrexel S.
PMID: 31507518
Front Neurol. 2019 Aug 27;10:914. doi: 10.3389/fneur.2019.00914. eCollection 2019.

No abstract available.

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Gracien RM, Petrov F, Hok P, et al. Multimodal Quantitative MRI Reveals No Evidence for Tissue Pathology in Idiopathic Cervical Dystonia. Front Neurol. 2019;10:914doi: 10.3389/fneur.2019.00914.
Gracien, R. M., Petrov, F., Hok, P., van Wijnen, A., Maiworm, M., Seiler, A., Deichmann, R., & Baudrexel, S. (2019). Multimodal Quantitative MRI Reveals No Evidence for Tissue Pathology in Idiopathic Cervical Dystonia. Frontiers in neurology, 10914. https://doi.org/10.3389/fneur.2019.00914
Gracien, René-Maxime, et al. "Multimodal Quantitative MRI Reveals No Evidence for Tissue Pathology in Idiopathic Cervical Dystonia." Frontiers in neurology vol. 10 (2019): 914. doi: https://doi.org/10.3389/fneur.2019.00914
Gracien RM, Petrov F, Hok P, van Wijnen A, Maiworm M, Seiler A, Deichmann R, Baudrexel S. Multimodal Quantitative MRI Reveals No Evidence for Tissue Pathology in Idiopathic Cervical Dystonia. Front Neurol. 2019 Aug 27;10:914. doi: 10.3389/fneur.2019.00914. eCollection 2019. PMID: 31507518; PMCID: PMC6719627.
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Autophagy Is Involved in Mesenchymal Stem Cell Death in Coculture with Chondrocytes.

Cartilage

Paggi CA, Dudakovic A, Fu Y, Garces CG, Hevesi M, Galeano Garces D, Dietz AB, van Wijnen AJ, Karperien M.
PMID: 32693629
Cartilage. 2021 Dec;13(2):969S-979S. doi: 10.1177/1947603520941227. Epub 2020 Jul 22.

OBJECTIVE: Cartilage formation is stimulated in mixtures of chondrocytes and human adipose-derived mesenchymal stromal cells (MSCs) both METHODS: Human primary chondrocytes were cocultured with human MSCs derived from 3 donors. The cells were cultured in monoculture or coculture (20%...

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Paggi CA, Dudakovic A, Fu Y, et al. Autophagy Is Involved in Mesenchymal Stem Cell Death in Coculture with Chondrocytes. Cartilage. 2020;13(2):969S-979Sdoi: 10.1177/1947603520941227.
Paggi, C. A., Dudakovic, A., Fu, Y., Garces, C. G., Hevesi, M., Galeano Garces, D., Dietz, A. B., van Wijnen, A. J., & Karperien, M. (2021). Autophagy Is Involved in Mesenchymal Stem Cell Death in Coculture with Chondrocytes. Cartilage, 13(2), 969S-979S. https://doi.org/10.1177/1947603520941227
Paggi, Carlo Alberto, et al. "Autophagy Is Involved in Mesenchymal Stem Cell Death in Coculture with Chondrocytes." Cartilage vol. 13,2 (2021): 969S-979S. doi: https://doi.org/10.1177/1947603520941227
Paggi CA, Dudakovic A, Fu Y, Garces CG, Hevesi M, Galeano Garces D, Dietz AB, van Wijnen AJ, Karperien M. Autophagy Is Involved in Mesenchymal Stem Cell Death in Coculture with Chondrocytes. Cartilage. 2021 Dec;13(2):969S-979S. doi: 10.1177/1947603520941227. Epub 2020 Jul 22. PMID: 32693629; PMCID: PMC8721613.
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Modernizing Storage Conditions for Fresh Osteochondral Allografts by Optimizing Viability at Physiologic Temperatures and Conditions.

Cartilage

Denbeigh JM, Hevesi M, Paggi CA, Resch ZT, Bagheri L, Mara K, Arani A, Zhang C, Larson AN, Saris DBF, Krych AJ, van Wijnen AJ.
PMID: 31777278
Cartilage. 2019 Nov 28;1947603519888798. doi: 10.1177/1947603519888798. Epub 2019 Nov 28.

No abstract available.

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Denbeigh JM, Hevesi M, Paggi CA, et al. Modernizing Storage Conditions for Fresh Osteochondral Allografts by Optimizing Viability at Physiologic Temperatures and Conditions. Cartilage. 2019;1947603519888798doi: 10.1177/1947603519888798.
Denbeigh, J. M., Hevesi, M., Paggi, C. A., Resch, Z. T., Bagheri, L., Mara, K., Arani, A., Zhang, C., Larson, A. N., Saris, D. B. F., Krych, A. J., & van Wijnen, A. J. (2019). Modernizing Storage Conditions for Fresh Osteochondral Allografts by Optimizing Viability at Physiologic Temperatures and Conditions. Cartilage, 1947603519888798. https://doi.org/10.1177/1947603519888798
Denbeigh, Janet M, et al. "Modernizing Storage Conditions for Fresh Osteochondral Allografts by Optimizing Viability at Physiologic Temperatures and Conditions." Cartilage vol. (2019): 1947603519888798. doi: https://doi.org/10.1177/1947603519888798
Denbeigh JM, Hevesi M, Paggi CA, Resch ZT, Bagheri L, Mara K, Arani A, Zhang C, Larson AN, Saris DBF, Krych AJ, van Wijnen AJ. Modernizing Storage Conditions for Fresh Osteochondral Allografts by Optimizing Viability at Physiologic Temperatures and Conditions. Cartilage. 2019 Nov 28;1947603519888798. doi: 10.1177/1947603519888798. Epub 2019 Nov 28. PMID: 31777278.
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Autophagy Is Involved in Mesenchymal Stem Cell Death in Coculture with Chondrocytes.

Cartilage

Paggi CA, Dudakovic A, Fu Y, Garces CG, Hevesi M, Galeano Garces D, Dietz AB, van Wijnen AJ, Karperien M.
PMID: 32693629
Cartilage. 2021 Dec;13(2):969S-979S. doi: 10.1177/1947603520941227. Epub 2020 Jul 22.

OBJECTIVE: Cartilage formation is stimulated in mixtures of chondrocytes and human adipose-derived mesenchymal stromal cells (MSCs) both METHODS: Human primary chondrocytes were cocultured with human MSCs derived from 3 donors. The cells were cultured in monoculture or coculture (20%...

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Paggi CA, Dudakovic A, Fu Y, et al. Autophagy Is Involved in Mesenchymal Stem Cell Death in Coculture with Chondrocytes. Cartilage. 2020;13(2):969S-979Sdoi: 10.1177/1947603520941227.
Paggi, C. A., Dudakovic, A., Fu, Y., Garces, C. G., Hevesi, M., Galeano Garces, D., Dietz, A. B., van Wijnen, A. J., & Karperien, M. (2021). Autophagy Is Involved in Mesenchymal Stem Cell Death in Coculture with Chondrocytes. Cartilage, 13(2), 969S-979S. https://doi.org/10.1177/1947603520941227
Paggi, Carlo Alberto, et al. "Autophagy Is Involved in Mesenchymal Stem Cell Death in Coculture with Chondrocytes." Cartilage vol. 13,2 (2021): 969S-979S. doi: https://doi.org/10.1177/1947603520941227
Paggi CA, Dudakovic A, Fu Y, Garces CG, Hevesi M, Galeano Garces D, Dietz AB, van Wijnen AJ, Karperien M. Autophagy Is Involved in Mesenchymal Stem Cell Death in Coculture with Chondrocytes. Cartilage. 2021 Dec;13(2):969S-979S. doi: 10.1177/1947603520941227. Epub 2020 Jul 22. PMID: 32693629.
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Brd4 Inactivation Increases Adenoviral Delivery of BMP2 for Paracrine Stimulation of Osteogenic Differentiation as a Gene Therapeutic Concept to Enhance Bone Healing.

JBMR plus

Paradise CR, De La Vega RE, Galvan ML, Carrasco ME, Thaler R, van Wijnen AJ, Dudakovic A.
PMID: 34693189
JBMR Plus. 2021 Jun 23;5(10):e10520. doi: 10.1002/jbm4.10520. eCollection 2021 Oct.

Bromodomain (BRD) proteins are histone code interpreters that recognize acetylated lysines and link the dynamic state of chromatin with the transcriptional machinery. Here, we demonstrate that ablation of the Brd4 gene in primary mouse bone marrow-derived mesenchymal stem cells...

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Paradise CR, De La Vega RE, Galvan ML, et al. Brd4 Inactivation Increases Adenoviral Delivery of BMP2 for Paracrine Stimulation of Osteogenic Differentiation as a Gene Therapeutic Concept to Enhance Bone Healing. JBMR Plus. 2021;5(10):e10520doi: 10.1002/jbm4.10520.
Paradise, C. R., De La Vega, R. E., Galvan, M. L., Carrasco, M. E., Thaler, R., van Wijnen, A. J., & Dudakovic, A. (2021). Brd4 Inactivation Increases Adenoviral Delivery of BMP2 for Paracrine Stimulation of Osteogenic Differentiation as a Gene Therapeutic Concept to Enhance Bone Healing. JBMR plus, 5(10), e10520. https://doi.org/10.1002/jbm4.10520
Paradise, Christopher R, et al. "Brd4 Inactivation Increases Adenoviral Delivery of BMP2 for Paracrine Stimulation of Osteogenic Differentiation as a Gene Therapeutic Concept to Enhance Bone Healing." JBMR plus vol. 5,10 (2021): e10520. doi: https://doi.org/10.1002/jbm4.10520
Paradise CR, De La Vega RE, Galvan ML, Carrasco ME, Thaler R, van Wijnen AJ, Dudakovic A. Brd4 Inactivation Increases Adenoviral Delivery of BMP2 for Paracrine Stimulation of Osteogenic Differentiation as a Gene Therapeutic Concept to Enhance Bone Healing. JBMR Plus. 2021 Jun 23;5(10):e10520. doi: 10.1002/jbm4.10520. eCollection 2021 Oct. PMID: 34693189; PMCID: PMC8520065.
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Elevated Expression of Plasminogen Activator Inhibitor (PAI-1/SERPINE1) is Independent from rs1799889 Genotypes in Arthrofibrosis.

Meta gene

Bayram B, Owen AR, Dudakovic A, Bettencourt JW, Limberg AK, Morrey ME, Sanchez-Sotelo J, Berry DJ, Kocher JP, van Wijnen AJ, Abdel MP.
PMID: 33816122
Meta Gene. 2021 Jun;28. doi: 10.1016/j.mgene.2021.100877. Epub 2021 Mar 05.

Arthrofibrosis is characterized by excessive extracellular matrix deposition in patients with total knee arthroplasties (TKAs) and causes undesirable joint stiffness. The pathogenesis of arthrofibrosis remains elusive and currently there are no diagnostic biomarkers for the pathological formation of this...

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Bayram B, Owen AR, Dudakovic A, et al. Elevated Expression of Plasminogen Activator Inhibitor (PAI-1/SERPINE1) is Independent from rs1799889 Genotypes in Arthrofibrosis. Meta Gene. 2021;28doi: 10.1016/j.mgene.2021.100877.
Bayram, B., Owen, A. R., Dudakovic, A., Bettencourt, J. W., Limberg, A. K., Morrey, M. E., Sanchez-Sotelo, J., Berry, D. J., Kocher, J. P., van Wijnen, A. J., & Abdel, M. P. (2021). Elevated Expression of Plasminogen Activator Inhibitor (PAI-1/SERPINE1) is Independent from rs1799889 Genotypes in Arthrofibrosis. Meta gene, 28. https://doi.org/10.1016/j.mgene.2021.100877
Bayram, Banu, et al. "Elevated Expression of Plasminogen Activator Inhibitor (PAI-1/SERPINE1) is Independent from rs1799889 Genotypes in Arthrofibrosis." Meta gene vol. 28 (2021). doi: https://doi.org/10.1016/j.mgene.2021.100877
Bayram B, Owen AR, Dudakovic A, Bettencourt JW, Limberg AK, Morrey ME, Sanchez-Sotelo J, Berry DJ, Kocher JP, van Wijnen AJ, Abdel MP. Elevated Expression of Plasminogen Activator Inhibitor (PAI-1/SERPINE1) is Independent from rs1799889 Genotypes in Arthrofibrosis. Meta Gene. 2021 Jun;28. doi: 10.1016/j.mgene.2021.100877. Epub 2021 Mar 05. PMID: 33816122; PMCID: PMC8011541.
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Global epigenetic alterations of mesenchymal stem cells in obesity: the role of vitamin C reprogramming.

Epigenetics

Afarideh M, Thaler R, Khani F, Tang H, Jordan KL, Conley SM, Saadiq IM, Obeidat Y, Pawar AS, Eirin A, Zhu XY, Lerman A, van Wijnen AJ, Lerman LO.
PMID: 32893712
Epigenetics. 2021 Jun-Jul;16(7):705-717. doi: 10.1080/15592294.2020.1819663. Epub 2020 Sep 20.

Obesity promotes dysfunction and impairs the reparative capacity of mesenchymal stem/stromal cells (MSCs), and alters their transcription, protein content, and paracrine function. Whether these adverse effects are mediated by chromatin-modifying epigenetic changes remains unclear. We tested the hypothesis that...

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Afarideh M, Thaler R, Khani F, et al. Global epigenetic alterations of mesenchymal stem cells in obesity: the role of vitamin C reprogramming. Epigenetics. 2020;16(7):705-717doi: 10.1080/15592294.2020.1819663.
Afarideh, M., Thaler, R., Khani, F., Tang, H., Jordan, K. L., Conley, S. M., Saadiq, I. M., Obeidat, Y., Pawar, A. S., Eirin, A., Zhu, X. Y., Lerman, A., van Wijnen, A. J., & Lerman, L. O. (2021). Global epigenetic alterations of mesenchymal stem cells in obesity: the role of vitamin C reprogramming. Epigenetics, 16(7), 705-717. https://doi.org/10.1080/15592294.2020.1819663
Afarideh, Mohsen, et al. "Global epigenetic alterations of mesenchymal stem cells in obesity: the role of vitamin C reprogramming." Epigenetics vol. 16,7 (2021): 705-717. doi: https://doi.org/10.1080/15592294.2020.1819663
Afarideh M, Thaler R, Khani F, Tang H, Jordan KL, Conley SM, Saadiq IM, Obeidat Y, Pawar AS, Eirin A, Zhu XY, Lerman A, van Wijnen AJ, Lerman LO. Global epigenetic alterations of mesenchymal stem cells in obesity: the role of vitamin C reprogramming. Epigenetics. 2021 Jun-Jul;16(7):705-717. doi: 10.1080/15592294.2020.1819663. Epub 2020 Sep 20. PMID: 32893712; PMCID: PMC8216191.
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