Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 290 entries
Sorted by: Best Match Show Resources per page
MicroRNAs as regulators of drug abuse and immunity.

Central-European journal of immunology

Zhang K, Jing X, Wang G.
PMID: 28450806
Cent Eur J Immunol. 2016;41(4):426-434. doi: 10.5114/ceji.2016.65142. Epub 2017 Jan 24.

MicroRNAs (miRNAs) are 20-22 nucleotide non-coding RNAs that participate in gene regulation. They bind to 3'-untranslated regions of their mRNA targets, inhibiting the transcripts' translation and/or destabilizing them. Chronic drug abuse induces changes of miRNAs expression in the brain,...

Cite
Zhang K, Jing X, Wang G. MicroRNAs as regulators of drug abuse and immunity. Cent Eur J Immunol. 2017;41(4):426-434doi: 10.5114/ceji.2016.65142.
Zhang, K., Jing, X., & Wang, G. (2016). MicroRNAs as regulators of drug abuse and immunity. Central-European journal of immunology, 41(4), 426-434. https://doi.org/10.5114/ceji.2016.65142
Zhang, Kai, et al. "MicroRNAs as regulators of drug abuse and immunity." Central-European journal of immunology vol. 41,4 (2016): 426-434. doi: https://doi.org/10.5114/ceji.2016.65142
Zhang K, Jing X, Wang G. MicroRNAs as regulators of drug abuse and immunity. Cent Eur J Immunol. 2016;41(4):426-434. doi: 10.5114/ceji.2016.65142. Epub 2017 Jan 24. PMID: 28450806; PMCID: PMC5382888.
Copy Download .nbib Format: NLM
MicroRNA binding site polymorphisms as biomarkers in cancer management and research.

Pharmacogenomics and personalized medicine

Cipollini M, Landi S, Gemignani F.
PMID: 25114582
Pharmgenomics Pers Med. 2014 Jul 23;7:173-91. doi: 10.2147/PGPM.S61693. eCollection 2014.

MicroRNAs (miRNAs) are important regulators of eukaryotic gene expression. They have been implicated in a broad range of biological processes, and miRNA-related genetic alterations probably underlie several human diseases. Single nucleotide polymorphisms of transcripts may modulate the posttranscriptional regulation...

Cite
Cipollini M, Landi S, Gemignani F. MicroRNA binding site polymorphisms as biomarkers in cancer management and research. Pharmgenomics Pers Med. 2014;7:173-91doi: 10.2147/PGPM.S61693.
Cipollini, M., Landi, S., & Gemignani, F. (2014). MicroRNA binding site polymorphisms as biomarkers in cancer management and research. Pharmacogenomics and personalized medicine, 7173-91. https://doi.org/10.2147/PGPM.S61693
Cipollini, Monica, et al. "MicroRNA binding site polymorphisms as biomarkers in cancer management and research." Pharmacogenomics and personalized medicine vol. 7 (2014): 173-91. doi: https://doi.org/10.2147/PGPM.S61693
Cipollini M, Landi S, Gemignani F. MicroRNA binding site polymorphisms as biomarkers in cancer management and research. Pharmgenomics Pers Med. 2014 Jul 23;7:173-91. doi: 10.2147/PGPM.S61693. eCollection 2014. PMID: 25114582; PMCID: PMC4126202.
Copy Download .nbib Format: NLM
Selective repression of the oncogene cyclin D1 by the tumor suppressor miR-206 in cancers.

Oncogenesis

Elliman SJ, Howley BV, Mehta DS, Fearnhead HO, Kemp DM, Barkley LR.
PMID: 25111862
Oncogenesis. 2014 Aug 11;3:e113. doi: 10.1038/oncsis.2014.26.

MicroRNAs (miRNAs) are deregulated in cancer and have been shown to exhibit both oncogenic and tumor suppressive functions. Although the functional effects of several miRNAs have been elucidated, those of many remain to be discovered. In silico analysis identified...

Cite
Elliman SJ, Howley BV, Mehta DS, et al. Selective repression of the oncogene cyclin D1 by the tumor suppressor miR-206 in cancers. Oncogenesis. 2014;3:e113doi: 10.1038/oncsis.2014.26.
Elliman, S. J., Howley, B. V., Mehta, D. S., Fearnhead, H. O., Kemp, D. M., & Barkley, L. R. (2014). Selective repression of the oncogene cyclin D1 by the tumor suppressor miR-206 in cancers. Oncogenesis, 3e113. https://doi.org/10.1038/oncsis.2014.26
Elliman, S J, et al. "Selective repression of the oncogene cyclin D1 by the tumor suppressor miR-206 in cancers." Oncogenesis vol. 3 (2014): e113. doi: https://doi.org/10.1038/oncsis.2014.26
Elliman SJ, Howley BV, Mehta DS, Fearnhead HO, Kemp DM, Barkley LR. Selective repression of the oncogene cyclin D1 by the tumor suppressor miR-206 in cancers. Oncogenesis. 2014 Aug 11;3:e113. doi: 10.1038/oncsis.2014.26. PMID: 25111862; PMCID: PMC5189965.
Copy Download .nbib Format: NLM
Evaluation of miR-29c inhibits endotheliocyte migration and angiogenesis of human endothelial cells by suppressing the insulin like growth factor 1.

American journal of translational research

Hu Y, Deng F, Song J, Lin J, Li X, Tang Y, Zhou J, Tang T, Zheng L.
PMID: 26045889
Am J Transl Res. 2015 Mar 15;7(3):489-501. eCollection 2015.

MicroRNAs, a class of 22-nucleotide non-coding RNAs, modulate gene expression by associating with the 3'-untranslated regions (3'-UTRs) of messenger RNAs (mRNAs). Although multiple miRNAs are known to be regulated during angiogenesis, their individual roles in blood vessel development are...

Cite
Hu Y, Deng F, Song J, et al. Evaluation of miR-29c inhibits endotheliocyte migration and angiogenesis of human endothelial cells by suppressing the insulin like growth factor 1. Am J Transl Res. 2015;7(3):489-501
Hu, Y., Deng, F., Song, J., Lin, J., Li, X., Tang, Y., Zhou, J., Tang, T., & Zheng, L. (2015). Evaluation of miR-29c inhibits endotheliocyte migration and angiogenesis of human endothelial cells by suppressing the insulin like growth factor 1. American journal of translational research, 7(3), 489-501.
Hu, Yun, et al. "Evaluation of miR-29c inhibits endotheliocyte migration and angiogenesis of human endothelial cells by suppressing the insulin like growth factor 1." American journal of translational research vol. 7,3 (2015): 489-501.
Hu Y, Deng F, Song J, Lin J, Li X, Tang Y, Zhou J, Tang T, Zheng L. Evaluation of miR-29c inhibits endotheliocyte migration and angiogenesis of human endothelial cells by suppressing the insulin like growth factor 1. Am J Transl Res. 2015 Mar 15;7(3):489-501. eCollection 2015. PMID: 26045889; PMCID: PMC4448189.
Copy Download .nbib Format: NLM
Isolation and cDNA characteristics of .

Biotechnology, biotechnological equipment

Sun Y, Zhang X, Xi D, Li G, Wang L, Zheng H, Du M, Gu Z, Yang Y, Yang Y.
PMID: 26019617
Biotechnol Biotechnol Equip. 2015 Jan 02;29(1):33-39. doi: 10.1080/13102818.2014.986128. Epub 2014 Dec 16.

The mammalian major histocompatibility complex (MHC) plays important roles in pathogen recognition and disease resistance. In the present study, the coding sequence and the 5'- and 3'-untranslated regions of MHC class II DR alpha chain (the DRA gene) from...

Cite
Sun Y, Zhang X, Xi D, et al. Isolation and cDNA characteristics of . Biotechnol Biotechnol Equip. 2014;29(1):33-39doi: 10.1080/13102818.2014.986128.
Sun, Y., Zhang, X., Xi, D., Li, G., Wang, L., Zheng, H., Du, M., Gu, Z., Yang, Y., & Yang, Y. (2015). Isolation and cDNA characteristics of . Biotechnology, biotechnological equipment, 29(1), 33-39. https://doi.org/10.1080/13102818.2014.986128
Sun, Yongke, et al. "Isolation and cDNA characteristics of ." Biotechnology, biotechnological equipment vol. 29,1 (2015): 33-39. doi: https://doi.org/10.1080/13102818.2014.986128
Sun Y, Zhang X, Xi D, Li G, Wang L, Zheng H, Du M, Gu Z, Yang Y, Yang Y. Isolation and cDNA characteristics of . Biotechnol Biotechnol Equip. 2015 Jan 02;29(1):33-39. doi: 10.1080/13102818.2014.986128. Epub 2014 Dec 16. PMID: 26019617; PMCID: PMC4434052.
Copy Download .nbib Format: NLM
Electroacupuncture Alleviates Surgical Trauma-Induced Hypothalamus Pituitary Adrenal Axis Hyperactivity Via microRNA-142.

Frontiers in molecular neuroscience

Zhu J, Chen Z, Meng Z, Ju M, Zhang M, Wu G, Guo H, Tian Z.
PMID: 29021740
Front Mol Neurosci. 2017 Sep 27;10:308. doi: 10.3389/fnmol.2017.00308. eCollection 2017.

Electroacupuncture (EA) could improve the hyperactivity of the hypothalamus pituitary adrenal (HPA) axis induced by hepatectomy. However, its underlying mechanism still remains largely unclear. Here, we found that hypothalamic corticotrophin releasing hormone (CRH) modulates the function of the HPA...

Cite
Zhu J, Chen Z, Meng Z, et al. Electroacupuncture Alleviates Surgical Trauma-Induced Hypothalamus Pituitary Adrenal Axis Hyperactivity Via microRNA-142. Front Mol Neurosci. 2017;10:308doi: 10.3389/fnmol.2017.00308.
Zhu, J., Chen, Z., Meng, Z., Ju, M., Zhang, M., Wu, G., Guo, H., & Tian, Z. (2017). Electroacupuncture Alleviates Surgical Trauma-Induced Hypothalamus Pituitary Adrenal Axis Hyperactivity Via microRNA-142. Frontiers in molecular neuroscience, 10308. https://doi.org/10.3389/fnmol.2017.00308
Zhu, Jing, et al. "Electroacupuncture Alleviates Surgical Trauma-Induced Hypothalamus Pituitary Adrenal Axis Hyperactivity Via microRNA-142." Frontiers in molecular neuroscience vol. 10 (2017): 308. doi: https://doi.org/10.3389/fnmol.2017.00308
Zhu J, Chen Z, Meng Z, Ju M, Zhang M, Wu G, Guo H, Tian Z. Electroacupuncture Alleviates Surgical Trauma-Induced Hypothalamus Pituitary Adrenal Axis Hyperactivity Via microRNA-142. Front Mol Neurosci. 2017 Sep 27;10:308. doi: 10.3389/fnmol.2017.00308. eCollection 2017. PMID: 29021740; PMCID: PMC5623716.
Copy Download .nbib Format: NLM
Pedigree based DNA sequencing pipeline for germline genomes of cancer families.

Hereditary cancer in clinical practice

Försti A, Kumar A, Paramasivam N, Schlesner M, Catalano C, Dymerska D, Lubinski J, Eils R, Hemminki K.
PMID: 27508007
Hered Cancer Clin Pract. 2016 Aug 09;14:16. doi: 10.1186/s13053-016-0058-1. eCollection 2016.

BACKGROUND: In the course of our whole-genome sequencing efforts, we have developed a pipeline for analyzing germline genomes from Mendelian types of cancer pedigrees (familial cancer variant prioritization pipeline, FCVPP).RESULTS: The variant calling step distinguishes two types of genomic...

Cite
Försti A, Kumar A, Paramasivam N, et al. Pedigree based DNA sequencing pipeline for germline genomes of cancer families. Hered Cancer Clin Pract. 2016;14:16doi: 10.1186/s13053-016-0058-1.
Försti, A., Kumar, A., Paramasivam, N., Schlesner, M., Catalano, C., Dymerska, D., Lubinski, J., Eils, R., & Hemminki, K. (2016). Pedigree based DNA sequencing pipeline for germline genomes of cancer families. Hereditary cancer in clinical practice, 1416. https://doi.org/10.1186/s13053-016-0058-1
Försti, Asta, et al. "Pedigree based DNA sequencing pipeline for germline genomes of cancer families." Hereditary cancer in clinical practice vol. 14 (2016): 16. doi: https://doi.org/10.1186/s13053-016-0058-1
Försti A, Kumar A, Paramasivam N, Schlesner M, Catalano C, Dymerska D, Lubinski J, Eils R, Hemminki K. Pedigree based DNA sequencing pipeline for germline genomes of cancer families. Hered Cancer Clin Pract. 2016 Aug 09;14:16. doi: 10.1186/s13053-016-0058-1. eCollection 2016. PMID: 27508007; PMCID: PMC4977614.
Copy Download .nbib Format: NLM
Dysregulation of Alternative Poly-adenylation as a Potential Player in Autism Spectrum Disorder.

Frontiers in molecular neuroscience

Szkop KJ, Cooke PIC, Humphries JA, Kalna V, Moss DS, Schuster EF, Nobeli I.
PMID: 28955198
Front Mol Neurosci. 2017 Sep 13;10:279. doi: 10.3389/fnmol.2017.00279. eCollection 2017.

We present here the hypothesis that alternative poly-adenylation (APA) is dysregulated in the brains of individuals affected by Autism Spectrum Disorder (ASD), due to disruptions in the calcium signaling networks. APA, the process of selecting different poly-adenylation sites on...

Cite
Szkop KJ, Cooke PIC, Humphries JA, et al. Dysregulation of Alternative Poly-adenylation as a Potential Player in Autism Spectrum Disorder. Front Mol Neurosci. 2017;10:279doi: 10.3389/fnmol.2017.00279.
Szkop, K. J., Cooke, P. I. C., Humphries, J. A., Kalna, V., Moss, D. S., Schuster, E. F., & Nobeli, I. (2017). Dysregulation of Alternative Poly-adenylation as a Potential Player in Autism Spectrum Disorder. Frontiers in molecular neuroscience, 10279. https://doi.org/10.3389/fnmol.2017.00279
Szkop, Krzysztof J, et al. "Dysregulation of Alternative Poly-adenylation as a Potential Player in Autism Spectrum Disorder." Frontiers in molecular neuroscience vol. 10 (2017): 279. doi: https://doi.org/10.3389/fnmol.2017.00279
Szkop KJ, Cooke PIC, Humphries JA, Kalna V, Moss DS, Schuster EF, Nobeli I. Dysregulation of Alternative Poly-adenylation as a Potential Player in Autism Spectrum Disorder. Front Mol Neurosci. 2017 Sep 13;10:279. doi: 10.3389/fnmol.2017.00279. eCollection 2017. PMID: 28955198; PMCID: PMC5601403.
Copy Download .nbib Format: NLM
Association of the Serotonin Receptor 3E Gene as a Functional Variant in the MicroRNA-510 Target Site with Diarrhea Predominant Irritable Bowel Syndrome in Chinese Women.

Journal of neurogastroenterology and motility

Zhang Y, Li Y, Hao Z, Li X, Bo P, Gong W.
PMID: 26787495
J Neurogastroenterol Motil. 2016 Apr 30;22(2):272-81. doi: 10.5056/jnm15138.

BACKGROUND/AIMS: The functional variant (rs56109847) in the 3'-untranslated regions (3'-UTR) of the serotonin receptor 3E (HTR3E) gene is associated with female diarrhea predominant irritable bowel syndrome (IBS-D) in British populations. However, the relationship of the polymorphism both to HTR3E...

Cite
Zhang Y, Li Y, Hao Z, et al. Association of the Serotonin Receptor 3E Gene as a Functional Variant in the MicroRNA-510 Target Site with Diarrhea Predominant Irritable Bowel Syndrome in Chinese Women. J Neurogastroenterol Motil. 2016;22(2):272-81doi: 10.5056/jnm15138.
Zhang, Y., Li, Y., Hao, Z., Li, X., Bo, P., & Gong, W. (2016). Association of the Serotonin Receptor 3E Gene as a Functional Variant in the MicroRNA-510 Target Site with Diarrhea Predominant Irritable Bowel Syndrome in Chinese Women. Journal of neurogastroenterology and motility, 22(2), 272-81. https://doi.org/10.5056/jnm15138
Zhang, Yu, et al. "Association of the Serotonin Receptor 3E Gene as a Functional Variant in the MicroRNA-510 Target Site with Diarrhea Predominant Irritable Bowel Syndrome in Chinese Women." Journal of neurogastroenterology and motility vol. 22,2 (2016): 272-81. doi: https://doi.org/10.5056/jnm15138
Zhang Y, Li Y, Hao Z, Li X, Bo P, Gong W. Association of the Serotonin Receptor 3E Gene as a Functional Variant in the MicroRNA-510 Target Site with Diarrhea Predominant Irritable Bowel Syndrome in Chinese Women. J Neurogastroenterol Motil. 2016 Apr 30;22(2):272-81. doi: 10.5056/jnm15138. PMID: 26787495; PMCID: PMC4819866.
Copy Download .nbib Format: NLM
Molecular Cloning and Characterization of Rice Pollen Profilin.

Sheng wu hua xue yu sheng wu wu li xue bao Acta biochimica et biophysica Sinica

Ye Q, Xu Y, Yan F, Tang L, Chen F.
PMID: 12040422
Sheng Wu Hua Xue Yu Sheng Wu Wu Li Xue Bao (Shanghai). 2001;33(4):452-456.

Profilin is a low molecular weight protein and is identified as an actin-binding protein in higher plants. Two pollen-specific cDNA clones of rice, RproA and RproB, were isolated from a cDNA library of rice mature pollen. The two cDNA...

Cite
Ye Q, Xu Y, Yan F, et al. Molecular Cloning and Characterization of Rice Pollen Profilin. Sheng Wu Hua Xue Yu Sheng Wu Wu Li Xue Bao (Shanghai). 2001;33(4):452-456
Ye, Q., Xu, Y., Yan, F., Tang, L., & Chen, F. (2001). Molecular Cloning and Characterization of Rice Pollen Profilin. Sheng wu hua xue yu sheng wu wu li xue bao Acta biochimica et biophysica Sinica, 33(4), 452-456.
Ye, Qiu, et al. "Molecular Cloning and Characterization of Rice Pollen Profilin." Sheng wu hua xue yu sheng wu wu li xue bao Acta biochimica et biophysica Sinica vol. 33,4 (2001): 452-456.
Ye Q, Xu Y, Yan F, Tang L, Chen F. Molecular Cloning and Characterization of Rice Pollen Profilin. Sheng Wu Hua Xue Yu Sheng Wu Wu Li Xue Bao (Shanghai). 2001;33(4):452-456. PMID: 12040422.
Copy Download .nbib Format: NLM
C4 GENE EXPRESSION.

Annual review of plant physiology and plant molecular biology

Sheen J.
PMID: 15012208
Annu Rev Plant Physiol Plant Mol Biol. 1999 Jun;50:187-217. doi: 10.1146/annurev.arplant.50.1.187.

C4 plants, including maize, Flaveria, amaranth, sorghum, and an amphibious sedge Eleocharis vivipara, have been employed to elucidate the molecular mechanisms and signaling pathways that control C4 photosynthesis gene expression. Current evidence suggests that pre-existing genes were recruited for...

Cite
Sheen J. C4 GENE EXPRESSION. Annu Rev Plant Physiol Plant Mol Biol. 1999;50:187-217doi: 10.1146/annurev.arplant.50.1.187.
Sheen, J. (1999). C4 GENE EXPRESSION. Annual review of plant physiology and plant molecular biology, 50187-217. https://doi.org/10.1146/annurev.arplant.50.1.187
Sheen, Jen. "C4 GENE EXPRESSION." Annual review of plant physiology and plant molecular biology vol. 50 (1999): 187-217. doi: https://doi.org/10.1146/annurev.arplant.50.1.187
Sheen J. C4 GENE EXPRESSION. Annu Rev Plant Physiol Plant Mol Biol. 1999 Jun;50:187-217. doi: 10.1146/annurev.arplant.50.1.187. PMID: 15012208.
Copy Download .nbib Format: NLM
Searching for potential microRNA-binding site mutations amongst known disease-associated 3' UTR variants.

Genomic medicine

Chuzhanova N, Cooper DN, Férec C, Chen JM.
PMID: 18923926
Genomic Med. 2007;1(1):29-33. doi: 10.1007/s11568-006-9000-3. Epub 2007 Jan 30.

The 3' untranslated regions (3' UTRs) of human protein-coding genes play a pivotal role in the regulation of mRNA 3' end formation, stability/degradation, nuclear export, subcellular localisation and translation, and hence are particularly rich in cis-acting regulatory elements. One...

Cite
Chuzhanova N, Cooper DN, Férec C, et al. Searching for potential microRNA-binding site mutations amongst known disease-associated 3' UTR variants. Genomic Med. 2007;1(1):29-33doi: 10.1007/s11568-006-9000-3.
Chuzhanova, N., Cooper, D. N., Férec, C., & Chen, J. M. (2007). Searching for potential microRNA-binding site mutations amongst known disease-associated 3' UTR variants. Genomic medicine, 1(1), 29-33. https://doi.org/10.1007/s11568-006-9000-3
Chuzhanova, Nadia, et al. "Searching for potential microRNA-binding site mutations amongst known disease-associated 3' UTR variants." Genomic medicine vol. 1,1 (2007): 29-33. doi: https://doi.org/10.1007/s11568-006-9000-3
Chuzhanova N, Cooper DN, Férec C, Chen JM. Searching for potential microRNA-binding site mutations amongst known disease-associated 3' UTR variants. Genomic Med. 2007;1(1):29-33. doi: 10.1007/s11568-006-9000-3. Epub 2007 Jan 30. PMID: 18923926; PMCID: PMC2276887.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 290 entries