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VEGFR‐3 in adult angiogenesis

adult

Dai J.
GSID: RqEaTtFBoWYJ
AN Witmer, BC Van Blijswijk, J Dai… - The Journal of …, 2001 - Wiley Online Library

… the adult, VEGFR-3 expression is lost from vascular endothelial cells but is retained on lymphatic endothelial cells3, 9, 11. It was therefore suggested that in the adult… a role in adult blood …

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Witmer, A. N., Van Blijswijk, B. C., Dai, J., Hofman, P., Partanen, T. A., Vrensen, G. F. J. M., & Schlingemann, R. O. (2001). VEGFR‐3 in adult angiogenesis. The Journal of Pathology: A Journal of the Pathological Society of Great Britain and Ireland, 195(4), 490-497.
Witmer, A. N., et al. "VEGFR‐3 in adult angiogenesis." The Journal of Pathology: A Journal of the Pathological Society of Great Britain and Ireland 195.4 (2001): 490-497.
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Rapid tumor necrosis and massive hemorrhage induced by bevacizumab and paclitaxel combination therapy in a case of advanced breast cancer.

OncoTargets and therapy

Ono M, Ito T, Kanai T, Murayama K, Koyama H, Maeno K, Mochizuki Y, Iesato A, Hanamura T, Okada T, Watanabe T, Ito K.
PMID: 24124381
Onco Targets Ther. 2013 Oct 07;6:1393-8. doi: 10.2147/OTT.S51164. eCollection 2013.

Bevacizumab when combined with chemotherapy exerts significant activity against many solid tumors through tumor angiogenesis inhibition; however, it can induce severe side effects. We report the rare case of a 27-year-old premenopausal woman with locally advanced breast cancer that...

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Ono M, Ito T, Kanai T, et al. Rapid tumor necrosis and massive hemorrhage induced by bevacizumab and paclitaxel combination therapy in a case of advanced breast cancer. Onco Targets Ther. 2013;6:1393-8doi: 10.2147/OTT.S51164.
Ono, M., Ito, T., Kanai, T., Murayama, K., Koyama, H., Maeno, K., Mochizuki, Y., Iesato, A., Hanamura, T., Okada, T., Watanabe, T., & Ito, K. (2013). Rapid tumor necrosis and massive hemorrhage induced by bevacizumab and paclitaxel combination therapy in a case of advanced breast cancer. OncoTargets and therapy, 61393-8. https://doi.org/10.2147/OTT.S51164
Ono, Mayu, et al. "Rapid tumor necrosis and massive hemorrhage induced by bevacizumab and paclitaxel combination therapy in a case of advanced breast cancer." OncoTargets and therapy vol. 6 (2013): 1393-8. doi: https://doi.org/10.2147/OTT.S51164
Ono M, Ito T, Kanai T, Murayama K, Koyama H, Maeno K, Mochizuki Y, Iesato A, Hanamura T, Okada T, Watanabe T, Ito K. Rapid tumor necrosis and massive hemorrhage induced by bevacizumab and paclitaxel combination therapy in a case of advanced breast cancer. Onco Targets Ther. 2013 Oct 07;6:1393-8. doi: 10.2147/OTT.S51164. eCollection 2013. PMID: 24124381; PMCID: PMC3794845.
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Histamine and histamine receptor regulation of gastrointestinal cancers.

Translational gastrointestinal cancer

Kennedy L, Hodges K, Meng F, Alpini G, Francis H.
PMID: 24639917
Transl Gastrointest Cancer. 2012 Oct;1(3):215-227.

Histamine is a neurotransmitter released throughout the body that regulates multiple physiological responses. Primarily histamine is acknowledged for its role in inflammatory reactions to foreign pathogens that enter the body. Aside from inflammatory responses, histamine expression and synthesis has...

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Kennedy L, Hodges K, Meng F, et al. Histamine and histamine receptor regulation of gastrointestinal cancers. Transl Gastrointest Cancer. 2012;1(3):215-227
Kennedy, L., Hodges, K., Meng, F., Alpini, G., & Francis, H. (2012). Histamine and histamine receptor regulation of gastrointestinal cancers. Translational gastrointestinal cancer, 1(3), 215-227.
Kennedy, Lindsey, et al. "Histamine and histamine receptor regulation of gastrointestinal cancers." Translational gastrointestinal cancer vol. 1,3 (2012): 215-227.
Kennedy L, Hodges K, Meng F, Alpini G, Francis H. Histamine and histamine receptor regulation of gastrointestinal cancers. Transl Gastrointest Cancer. 2012 Oct;1(3):215-227. PMID: 24639917; PMCID: PMC3955103.
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PLDing a case for angiogenesis.

Blood

Hamik A, Jain MK.
PMID: 20798242
Blood. 2010 Aug 26;116(8):1194-6. doi: 10.1182/blood-2010-05-284117.

No abstract available.

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Hamik A, Jain MK. PLDing a case for angiogenesis. Blood. 2010;116(8):1194-6doi: 10.1182/blood-2010-05-284117.
Hamik, A., & Jain, M. K. (2010). PLDing a case for angiogenesis. Blood, 116(8), 1194-6. https://doi.org/10.1182/blood-2010-05-284117
Hamik, Anne, and Jain, Mukesh K. "PLDing a case for angiogenesis." Blood vol. 116,8 (2010): 1194-6. doi: https://doi.org/10.1182/blood-2010-05-284117
Hamik A, Jain MK. PLDing a case for angiogenesis. Blood. 2010 Aug 26;116(8):1194-6. doi: 10.1182/blood-2010-05-284117. PMID: 20798242.
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Notch modulates VEGF action in endothelial cells by inducing Matrix Metalloprotease activity.

Vascular cell

Funahashi Y, Shawber CJ, Sharma A, Kanamaru E, Choi YK, Kitajewski J.
PMID: 21349159
Vasc Cell. 2011 Jan 18;3(1):2. doi: 10.1186/2045-824X-3-2.

BACKGROUND: In the vasculature, Notch signaling functions as a downstream effecter of Vascular Endothelial Growth Factor (VEGF) signaling. VEGF regulates sprouting angiogenesis in part by inducing and activating matrix metalloproteases (MMPs). This study sought to determine if VEGF regulation...

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Funahashi Y, Shawber CJ, Sharma A, et al. Notch modulates VEGF action in endothelial cells by inducing Matrix Metalloprotease activity. Vasc Cell. 2011;3(1):2doi: 10.1186/2045-824X-3-2.
Funahashi, Y., Shawber, C. J., Sharma, A., Kanamaru, E., Choi, Y. K., & Kitajewski, J. (2011). Notch modulates VEGF action in endothelial cells by inducing Matrix Metalloprotease activity. Vascular cell, 3(1), 2. https://doi.org/10.1186/2045-824X-3-2
Funahashi, Yasuhiro, et al. "Notch modulates VEGF action in endothelial cells by inducing Matrix Metalloprotease activity." Vascular cell vol. 3,1 (2011): 2. doi: https://doi.org/10.1186/2045-824X-3-2
Funahashi Y, Shawber CJ, Sharma A, Kanamaru E, Choi YK, Kitajewski J. Notch modulates VEGF action in endothelial cells by inducing Matrix Metalloprotease activity. Vasc Cell. 2011 Jan 18;3(1):2. doi: 10.1186/2045-824X-3-2. PMID: 21349159; PMCID: PMC3039832.
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Effect of DAPT, a gamma secretase inhibitor, on tumor angiogenesis in control mice.

Advanced biomedical research

Kalantari E, Saeidi H, Kia NS, Tahergorabi Z, Rashidi B, Dana N, Khazaei M.
PMID: 24520550
Adv Biomed Res. 2013 Nov 30;2:83. doi: 10.4103/2277-9175.122498. eCollection 2013.

BACKGROUND: Notch signaling is a key factor for angiogenesis in physiological and pathological condition and γ-secretase is the regulator of Notch signaling. The main goal of this study was to assess the effect of (N-[N-(3,5-Diflurophenaacetyl-L-alanyl)]-S-phenylglycine t-Butyl Ester) DAPT, a...

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Kalantari E, Saeidi H, Kia NS, et al. Effect of DAPT, a gamma secretase inhibitor, on tumor angiogenesis in control mice. Adv Biomed Res. 2013;2:83doi: 10.4103/2277-9175.122498.
Kalantari, E., Saeidi, H., Kia, N. S., Tahergorabi, Z., Rashidi, B., Dana, N., & Khazaei, M. (2013). Effect of DAPT, a gamma secretase inhibitor, on tumor angiogenesis in control mice. Advanced biomedical research, 283. https://doi.org/10.4103/2277-9175.122498
Kalantari, Elmira, et al. "Effect of DAPT, a gamma secretase inhibitor, on tumor angiogenesis in control mice." Advanced biomedical research vol. 2 (2013): 83. doi: https://doi.org/10.4103/2277-9175.122498
Kalantari E, Saeidi H, Kia NS, Tahergorabi Z, Rashidi B, Dana N, Khazaei M. Effect of DAPT, a gamma secretase inhibitor, on tumor angiogenesis in control mice. Adv Biomed Res. 2013 Nov 30;2:83. doi: 10.4103/2277-9175.122498. eCollection 2013. PMID: 24520550; PMCID: PMC3908495.
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ARF: a versatile DNA damage response ally at the crossroads of development and tumorigenesis.

Frontiers in genetics

Kotsinas A, Papanagnou P, Evangelou K, Trigas GC, Kostourou V, Townsend P, Gorgoulis VG.
PMID: 25101116
Front Genet. 2014 Jul 22;5:236. doi: 10.3389/fgene.2014.00236. eCollection 2014.

Alternative reading frame (ARF) is a tumor suppressor protein that senses oncogenic and other stressogenic signals. It can trigger p53-dependent and -independent responses with cell cycle arrest and apoptosis induction being the most prominent ones. Other ARF activities, particularly...

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Kotsinas A, Papanagnou P, Evangelou K, et al. ARF: a versatile DNA damage response ally at the crossroads of development and tumorigenesis. Front Genet. 2014;5:236doi: 10.3389/fgene.2014.00236.
Kotsinas, A., Papanagnou, P., Evangelou, K., Trigas, G. C., Kostourou, V., Townsend, P., & Gorgoulis, V. G. (2014). ARF: a versatile DNA damage response ally at the crossroads of development and tumorigenesis. Frontiers in genetics, 5236. https://doi.org/10.3389/fgene.2014.00236
Kotsinas, Athanassios, et al. "ARF: a versatile DNA damage response ally at the crossroads of development and tumorigenesis." Frontiers in genetics vol. 5 (2014): 236. doi: https://doi.org/10.3389/fgene.2014.00236
Kotsinas A, Papanagnou P, Evangelou K, Trigas GC, Kostourou V, Townsend P, Gorgoulis VG. ARF: a versatile DNA damage response ally at the crossroads of development and tumorigenesis. Front Genet. 2014 Jul 22;5:236. doi: 10.3389/fgene.2014.00236. eCollection 2014. PMID: 25101116; PMCID: PMC4106421.
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Extracellular vesicles - biomarkers and effectors of the cellular interactome in cancer.

Frontiers in pharmacology

Rak J.
PMID: 23508692
Front Pharmacol. 2013 Mar 06;4:21. doi: 10.3389/fphar.2013.00021. eCollection 2013.

In multicellular organisms both health and disease are defined by patterns of communication between the constituent cells. In addition to networks of soluble mediators, cells are also programed to exchange complex messages pre-assembled as multimolecular cargo of membraneous structures...

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Rak J. Extracellular vesicles - biomarkers and effectors of the cellular interactome in cancer. Front Pharmacol. 2013;4:21doi: 10.3389/fphar.2013.00021.
Rak, J. (2013). Extracellular vesicles - biomarkers and effectors of the cellular interactome in cancer. Frontiers in pharmacology, 421. https://doi.org/10.3389/fphar.2013.00021
Rak, Janusz. "Extracellular vesicles - biomarkers and effectors of the cellular interactome in cancer." Frontiers in pharmacology vol. 4 (2013): 21. doi: https://doi.org/10.3389/fphar.2013.00021
Rak J. Extracellular vesicles - biomarkers and effectors of the cellular interactome in cancer. Front Pharmacol. 2013 Mar 06;4:21. doi: 10.3389/fphar.2013.00021. eCollection 2013. PMID: 23508692; PMCID: PMC3589665.
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Mandibular reconstruction using an axially vascularized tissue-engineered construct.

Annals of surgical innovation and research

Eweida AM, Nabawi AS, Marei MK, Khalil MR, Elhammady HA.
PMID: 21418603
Ann Surg Innov Res. 2011 Mar 20;5:2. doi: 10.1186/1750-1164-5-2.

BACKGROUND: Current reconstructive techniques for continuity defects of the mandible include the use of free flaps, bone grafts, and alloplastic materials. New methods of regenerative medicine designed to restore tissues depend mainly on the so-called extrinsic neovascularization, where the...

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Eweida AM, Nabawi AS, Marei MK, et al. Mandibular reconstruction using an axially vascularized tissue-engineered construct. Ann Surg Innov Res. 2011;5:2doi: 10.1186/1750-1164-5-2.
Eweida, A. M., Nabawi, A. S., Marei, M. K., Khalil, M. R., & Elhammady, H. A. (2011). Mandibular reconstruction using an axially vascularized tissue-engineered construct. Annals of surgical innovation and research, 52. https://doi.org/10.1186/1750-1164-5-2
Eweida, Ahmad M, et al. "Mandibular reconstruction using an axially vascularized tissue-engineered construct." Annals of surgical innovation and research vol. 5 (2011): 2. doi: https://doi.org/10.1186/1750-1164-5-2
Eweida AM, Nabawi AS, Marei MK, Khalil MR, Elhammady HA. Mandibular reconstruction using an axially vascularized tissue-engineered construct. Ann Surg Innov Res. 2011 Mar 20;5:2. doi: 10.1186/1750-1164-5-2. PMID: 21418603; PMCID: PMC3069948.
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Peroxisome Proliferator-Activated Receptor γ Regulates the Expression of Lipid Phosphate Phosphohydrolase 1 in Human Vascular Endothelial Cells.

PPAR research

Huang Y, Zhao B, Liu Y, Wang N.
PMID: 24955089
PPAR Res. 2014;2014:740121. doi: 10.1155/2014/740121. Epub 2014 May 12.

Lipid phosphate phosphohydrolase 1 (LPP1), a membrane ectophosphohydrolase regulating the availability of bioactive lipid phosphates, plays important roles in cellular signaling and physiological processes such as angiogenesis and endothelial migration. However, the regulated expression of LPP1 remains largely unknown....

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Huang Y, Zhao B, Liu Y, et al. Peroxisome Proliferator-Activated Receptor γ Regulates the Expression of Lipid Phosphate Phosphohydrolase 1 in Human Vascular Endothelial Cells. PPAR Res. 2014;2014:740121doi: 10.1155/2014/740121.
Huang, Y., Zhao, B., Liu, Y., & Wang, N. (2014). Peroxisome Proliferator-Activated Receptor γ Regulates the Expression of Lipid Phosphate Phosphohydrolase 1 in Human Vascular Endothelial Cells. PPAR research, 2014740121. https://doi.org/10.1155/2014/740121
Huang, Yazi, et al. "Peroxisome Proliferator-Activated Receptor γ Regulates the Expression of Lipid Phosphate Phosphohydrolase 1 in Human Vascular Endothelial Cells." PPAR research vol. 2014 (2014): 740121. doi: https://doi.org/10.1155/2014/740121
Huang Y, Zhao B, Liu Y, Wang N. Peroxisome Proliferator-Activated Receptor γ Regulates the Expression of Lipid Phosphate Phosphohydrolase 1 in Human Vascular Endothelial Cells. PPAR Res. 2014;2014:740121. doi: 10.1155/2014/740121. Epub 2014 May 12. PMID: 24955089; PMCID: PMC4052932.
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The role of angiogenesis in the pathology of multiple sclerosis.

Vascular cell

Lengfeld J, Cutforth T, Agalliu D.
PMID: 25473485
Vasc Cell. 2014 Nov 28;6(1):23. doi: 10.1186/s13221-014-0023-6. eCollection 2014.

Angiogenesis, or the growth of new blood vessels from existing vasculature, is critical for the proper development of many organs. This process is inhibited and tightly regulated in adults, once endothelial cells have acquired organ-specific properties. Within the central...

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Lengfeld J, Cutforth T, Agalliu D. The role of angiogenesis in the pathology of multiple sclerosis. Vasc Cell. 2014;6(1):23doi: 10.1186/s13221-014-0023-6.
Lengfeld, J., Cutforth, T., & Agalliu, D. (2014). The role of angiogenesis in the pathology of multiple sclerosis. Vascular cell, 6(1), 23. https://doi.org/10.1186/s13221-014-0023-6
Lengfeld, Justin, et al. "The role of angiogenesis in the pathology of multiple sclerosis." Vascular cell vol. 6,1 (2014): 23. doi: https://doi.org/10.1186/s13221-014-0023-6
Lengfeld J, Cutforth T, Agalliu D. The role of angiogenesis in the pathology of multiple sclerosis. Vasc Cell. 2014 Nov 28;6(1):23. doi: 10.1186/s13221-014-0023-6. eCollection 2014. PMID: 25473485; PMCID: PMC4253611.
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Effect of NF- κ B inhibitor PDTC on VEGF and endostatin expression of mice with Lewis lung cancer.

Asian Pacific journal of tropical medicine

Gao P, Gao YJ, Liang HL.
PMID: 25902165
Asian Pac J Trop Med. 2015 Mar;8(3):220-4. doi: 10.1016/S1995-7645(14)60319-9.

OBJECTIVE: To investigate the effects of NF- κ B inhibitor pyrrolidine dithiocarbamate hydrochloride (PDTC) on vascular endothelial growth factor (VEGF) and endostatin expression in mice with Lewis lung cance; and its mechanism.METHODS: Mice survival rate and anti-tumor effects were...

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Gao P, Gao YJ, Liang HL. Effect of NF- κ B inhibitor PDTC on VEGF and endostatin expression of mice with Lewis lung cancer. Asian Pac J Trop Med. 2015;8(3):220-4doi: 10.1016/S1995-7645(14)60319-9.
Gao, P., Gao, Y. J., & Liang, H. L. (2015). Effect of NF- κ B inhibitor PDTC on VEGF and endostatin expression of mice with Lewis lung cancer. Asian Pacific journal of tropical medicine, 8(3), 220-4. https://doi.org/10.1016/S1995-7645(14)60319-9
Gao, Ping, et al. "Effect of NF- κ B inhibitor PDTC on VEGF and endostatin expression of mice with Lewis lung cancer." Asian Pacific journal of tropical medicine vol. 8,3 (2015): 220-4. doi: https://doi.org/10.1016/S1995-7645(14)60319-9
Gao P, Gao YJ, Liang HL. Effect of NF- κ B inhibitor PDTC on VEGF and endostatin expression of mice with Lewis lung cancer. Asian Pac J Trop Med. 2015 Mar;8(3):220-4. doi: 10.1016/S1995-7645(14)60319-9. PMID: 25902165.
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Showing 1 to 12 of 15765 entries